BRONCHITIS
Presented by- P.K. PANWAR
WEB- www.pkpanwarauthor.com
Introduction
It is a condition where the lining of bronchial
tubes become inflamed or infected. It have
reduce ability to breath in air & oxygen into the
lungs, they have also heavy mucus forming in the
airways.
Incidence
The recent 'Indian Study of Asthma, Respiratory
Symptoms and Chronic Bronchitis' study of
85,105 men and 84,470 women from 12 urban
and 11 rural sites reported the incidence of
chronic bronchitis to be 3.49% (4.29% in males
and 2.7% in females) in adults > 35 years.
Normal mechanism of inflammation
Infection enter into the body
Chemical release from WBC are released into the
blood or affected tissue to protect your body from
foreign substances
This release of chemicals increases the blood flow to
the area of injury or infection
may result in redness and warmth
Some of the chemicals cause a leak of fluid into the
tissues, resulting in swelling
This protective process may stimulate nerves and
cause pain
Definition
It is an inflammation and swelling of the
bronchial tubes (bronchi), the air passage
between the lungs.
Anatomy of Bronchi
Trachea divide it form two primary bronchi
Right bronchus – this is wider, shorter &
more vertical then the left bronchus. It is
approximately 2.5 cm long. After entering
into the right lung it divide into three
branches, one to each lobe. Each branches it
subdivided into numerous smaller branches.
Left bronchus – 5 cm long, & narrower then
the right. After entering into the lung it divide into
the two branches , one to each lobe, each branch
then subdivide into the numerous smaller branches.
Structure – it lined with ciliated columnar
epithelium. The bronchi progressively subdivide into
the bronchioles, terminal bronchioles, respiratory
bronchioles, alveolar duct & finally alveoli.
In trachea & bronchi cartilages, ciliated epithelium,
goblet cells is present, at the bronchiolar level
there is no cartilage it replace with smooth muscle,
ciliated replaced with non-ciliated epithelium &
goblet cell disappear.
Types
1.
2.
Acute – it is a shorter illness that commonly
follow a cold or viral infection such as flu.
It consist of a cough with mucus, chest
discomfort, throat soreness, fever,
shortness of breath. It is usually a last a
few days or weeks (1-3 week).
Chronic – it is a serious ongoing illness, it is
a persistent, mucus producing cough that
last longer then three month. The person is
having severe breathing difficulties & it may
get worsen. It occurs with emphysema and
it may become COPD.
Etiology
1.
2.
Acute – It is caused by virus, the same
virus that cause cold & flu. It can be
caused by bacterial infection & exposure to
substance that irritate the lungs such as
tobacco smoke, dust, fumes, vapours & air
pollution.
Chronic – it is caused by repeated irritation
& damage of the lungs & airway tissue.
Smoking is most common cause with other
causes including long term exposure to air
pollution, dust & fumes from the
environment & repeated episodes of acute
bronchitis.
Etiology
•
•
•
Etiologic agent – bacteria, virus
Predisposing factor (contribute to the
problem)– smoking, long term illness,
immune deficiency and immobilization.
Precipitating factor (trigger the
problem)– hospitalization, environment,
smoking and malnutrition.
Clinical manifestation
Sign & symptoms for both acute and chronic bronchitis.
•
•
•
•
•
•
•
Persistent cough
Production of mucus which can be clear, white,
yellowish gray or green in color, rarely it may be
streaked with blood.
Crackles and Wheezing sound
Low fever, chills, Headache
Chest tightening
Sore throat, dyspnoea
Blocked nose & sinuses
Diagnostic evaluation
•
•
•
•
•
•
History
Physical examination
Chest x-ray
Sputum– for gram stain, culture and
sensitivity test may be obtained to
determine presence of bacterial infection.
Pulmonary function test by using spirometer
– to determine peak expiratory flow
(person's maximum speed of expiration)
ABG Level
Management
People with bronchitis are instruct to rest, drink
fluid, breath warm & moist air, & take OTC
cough suppressant & pain relieve in order to
manage symptoms & ease breathing.
Many case of acute condition may go away without
any specific treatment, but there is a no cure
for chronic condition.
To keep bronchitis symptoms under control &
relieve symptoms, doctor may prescribe –
Antibiotics – Azithromycin, for 7-10 days
Antitussive – Codeine for suppressing the cough
Bronchodilators – To dilate the bronchi
• Beta2-adrenergic agonist agents –
Salbutamol, Terbutaline
• Anticholinergic agents – Ipratropium bromide
• Methylxanthines – Theophylline
Mucolytics - e.g. Acetylcysteine to thin the secretions.
Corticosteroids – Dexamethasone, Methylprednisolone to
relieve the inflammation
Antipyretics - for fever
Other - Oxygen therapy, Pulmonary rehabilitation program,
chest physiotherapy, nutritional therapy
Additional behaviour remedies include –
•
•
•
•
Removing the source of irritants from the
lungs
Using a humidifier – loosen mucus
Exercise
Breathing exercise – pursed lip breathing
Complication
•
•
•
Pneumonia
Asthma
COPD
1. Nursing diagnosis – ineffective airway clearance
related to thick mucus discharge as evidence by
presence of rhonchi, cough and tachypnea.
Nursing goal – Improve airway clearance
Intervention - Place the patient laterally and
recumbent to help maintain an open airway and drain
the secretion.
•
•
•
•
Provide oxygen to decrease her laboured breathing.
Observe the oxygen therapy delivery system every
hourly.
To clear the thick mucus, use humidifier and
suctioning.
Use cough medicine.
2. Nursing diagnosis – Impaired respiratory
functioning related to ineffective breathing pattern
as evidence by increased respiratory rate.
Goal – To improve the respiratory functioning
Intervention - Always stay with patient to reduce
the anxiety.
•
•
•
•
To give instruction regarding the pursed lip &
diaphragmatic breathing to assist with slowing
respiratory rate.
To provide water to drink help in loosen the secretion
& lessen the dryness in mucus membrane.
To provide oxygen therapy.
To provide suctioning help in removing the secretion.
3. Nursing diagnosis – acute pain related to
inflammation, cough as evidence by report of
discomfort and facial expression.
Goal – To relieve the pain
Intervention
•
•
•
•
Administer acetaminophen medication.
Monitor vital sign for respiratory suppression
associated with pain medication.
To apply pillow to chest while coughing.
Help the patient increase level of comfort in hospital
bed by elevating the head of the bed, to help assist
in less painful breathing.
Research study
Smoking Cessation and Lung Function in Mild-to-
Moderate Chronic Obstructive Pulmonary Disease
It was conducted at 10 North American medical centres, we
studied 3,926 smokers with mild-to-moderate airway
obstruction randomized to one of two smoking cessation
groups. We measured lung function annually for 5 yr.
Participants who stopped smoking experienced an
improvement in FEV1 (forced expiratory volume) in the
year after quitting. The subsequent rate of decline in FEV1
among sustained quitters was half the rate among
continuing smokers, 31 ± 48 versus 62 ± 55 ml (mean ± SD)
comparable to that of never-smokers. Smokers with
airflow obstruction benefit from quitting despite previous
heavy smoking.
THANK YOU

bronchitis-200424105258.pdf

  • 1.
    BRONCHITIS Presented by- P.K.PANWAR WEB- www.pkpanwarauthor.com
  • 2.
    Introduction It is acondition where the lining of bronchial tubes become inflamed or infected. It have reduce ability to breath in air & oxygen into the lungs, they have also heavy mucus forming in the airways. Incidence The recent 'Indian Study of Asthma, Respiratory Symptoms and Chronic Bronchitis' study of 85,105 men and 84,470 women from 12 urban and 11 rural sites reported the incidence of chronic bronchitis to be 3.49% (4.29% in males and 2.7% in females) in adults > 35 years.
  • 4.
    Normal mechanism ofinflammation Infection enter into the body Chemical release from WBC are released into the blood or affected tissue to protect your body from foreign substances This release of chemicals increases the blood flow to the area of injury or infection may result in redness and warmth Some of the chemicals cause a leak of fluid into the tissues, resulting in swelling This protective process may stimulate nerves and cause pain
  • 5.
    Definition It is aninflammation and swelling of the bronchial tubes (bronchi), the air passage between the lungs. Anatomy of Bronchi Trachea divide it form two primary bronchi Right bronchus – this is wider, shorter & more vertical then the left bronchus. It is approximately 2.5 cm long. After entering into the right lung it divide into three branches, one to each lobe. Each branches it subdivided into numerous smaller branches.
  • 6.
    Left bronchus –5 cm long, & narrower then the right. After entering into the lung it divide into the two branches , one to each lobe, each branch then subdivide into the numerous smaller branches. Structure – it lined with ciliated columnar epithelium. The bronchi progressively subdivide into the bronchioles, terminal bronchioles, respiratory bronchioles, alveolar duct & finally alveoli. In trachea & bronchi cartilages, ciliated epithelium, goblet cells is present, at the bronchiolar level there is no cartilage it replace with smooth muscle, ciliated replaced with non-ciliated epithelium & goblet cell disappear.
  • 9.
    Types 1. 2. Acute – itis a shorter illness that commonly follow a cold or viral infection such as flu. It consist of a cough with mucus, chest discomfort, throat soreness, fever, shortness of breath. It is usually a last a few days or weeks (1-3 week). Chronic – it is a serious ongoing illness, it is a persistent, mucus producing cough that last longer then three month. The person is having severe breathing difficulties & it may get worsen. It occurs with emphysema and it may become COPD.
  • 12.
    Etiology 1. 2. Acute – Itis caused by virus, the same virus that cause cold & flu. It can be caused by bacterial infection & exposure to substance that irritate the lungs such as tobacco smoke, dust, fumes, vapours & air pollution. Chronic – it is caused by repeated irritation & damage of the lungs & airway tissue. Smoking is most common cause with other causes including long term exposure to air pollution, dust & fumes from the environment & repeated episodes of acute bronchitis.
  • 13.
    Etiology • • • Etiologic agent –bacteria, virus Predisposing factor (contribute to the problem)– smoking, long term illness, immune deficiency and immobilization. Precipitating factor (trigger the problem)– hospitalization, environment, smoking and malnutrition.
  • 15.
    Clinical manifestation Sign &symptoms for both acute and chronic bronchitis. • • • • • • • Persistent cough Production of mucus which can be clear, white, yellowish gray or green in color, rarely it may be streaked with blood. Crackles and Wheezing sound Low fever, chills, Headache Chest tightening Sore throat, dyspnoea Blocked nose & sinuses
  • 17.
    Diagnostic evaluation • • • • • • History Physical examination Chestx-ray Sputum– for gram stain, culture and sensitivity test may be obtained to determine presence of bacterial infection. Pulmonary function test by using spirometer – to determine peak expiratory flow (person's maximum speed of expiration) ABG Level
  • 19.
    Management People with bronchitisare instruct to rest, drink fluid, breath warm & moist air, & take OTC cough suppressant & pain relieve in order to manage symptoms & ease breathing. Many case of acute condition may go away without any specific treatment, but there is a no cure for chronic condition. To keep bronchitis symptoms under control & relieve symptoms, doctor may prescribe – Antibiotics – Azithromycin, for 7-10 days Antitussive – Codeine for suppressing the cough
  • 20.
    Bronchodilators – Todilate the bronchi • Beta2-adrenergic agonist agents – Salbutamol, Terbutaline • Anticholinergic agents – Ipratropium bromide • Methylxanthines – Theophylline Mucolytics - e.g. Acetylcysteine to thin the secretions. Corticosteroids – Dexamethasone, Methylprednisolone to relieve the inflammation Antipyretics - for fever Other - Oxygen therapy, Pulmonary rehabilitation program, chest physiotherapy, nutritional therapy
  • 21.
    Additional behaviour remediesinclude – • • • • Removing the source of irritants from the lungs Using a humidifier – loosen mucus Exercise Breathing exercise – pursed lip breathing
  • 22.
  • 23.
    1. Nursing diagnosis– ineffective airway clearance related to thick mucus discharge as evidence by presence of rhonchi, cough and tachypnea. Nursing goal – Improve airway clearance Intervention - Place the patient laterally and recumbent to help maintain an open airway and drain the secretion. • • • • Provide oxygen to decrease her laboured breathing. Observe the oxygen therapy delivery system every hourly. To clear the thick mucus, use humidifier and suctioning. Use cough medicine.
  • 24.
    2. Nursing diagnosis– Impaired respiratory functioning related to ineffective breathing pattern as evidence by increased respiratory rate. Goal – To improve the respiratory functioning Intervention - Always stay with patient to reduce the anxiety. • • • • To give instruction regarding the pursed lip & diaphragmatic breathing to assist with slowing respiratory rate. To provide water to drink help in loosen the secretion & lessen the dryness in mucus membrane. To provide oxygen therapy. To provide suctioning help in removing the secretion.
  • 25.
    3. Nursing diagnosis– acute pain related to inflammation, cough as evidence by report of discomfort and facial expression. Goal – To relieve the pain Intervention • • • • Administer acetaminophen medication. Monitor vital sign for respiratory suppression associated with pain medication. To apply pillow to chest while coughing. Help the patient increase level of comfort in hospital bed by elevating the head of the bed, to help assist in less painful breathing.
  • 26.
    Research study Smoking Cessationand Lung Function in Mild-to- Moderate Chronic Obstructive Pulmonary Disease It was conducted at 10 North American medical centres, we studied 3,926 smokers with mild-to-moderate airway obstruction randomized to one of two smoking cessation groups. We measured lung function annually for 5 yr. Participants who stopped smoking experienced an improvement in FEV1 (forced expiratory volume) in the year after quitting. The subsequent rate of decline in FEV1 among sustained quitters was half the rate among continuing smokers, 31 ± 48 versus 62 ± 55 ml (mean ± SD) comparable to that of never-smokers. Smokers with airflow obstruction benefit from quitting despite previous heavy smoking.
  • 27.