The document summarizes anastomotic intimal hyperplasia (AIH), which is thickening of the intimal layer at vascular graft anastomoses. AIH occurs due to endothelial damage during anastomosis construction between a graft and blood vessel. This damage allows proliferation and migration of smooth muscle cells into the intima. Growth factors released by platelets and damaged cells promote this process. AIH develops mainly at the suture line and arterial floor opposite the graft hood. Techniques using venous material in a transverse orientation may help reduce AIH by creating a gradual transition between the graft and vessel properties.