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CASE STUDY ON
ACUTE
GASTROENTERITIS
bigdaddy
INTRODUCTION
Acute Gastroenteritis
 Acute Gastroenteritis is inflammation of the

  gastrointestinal tract , involving both the
  stomach and the small intestine and resulting
  in acute diarrhea . The inflammation is caused
  most often by infection with certain viruses ,
  less often by bacrteria or their toxins , parasites
  or adverse reaction to something in the diet or
  medication.
   Different species of bacteria can cause
         gastroenteritis, including
    Salmonella , Shigella, Staphylococcus,
    Campylobacter jejuni, Clostridium, Escherichia
    coli, Yersinia , and others.
    Each organism causes slightly different sympto
    ms but all result in diarrhea. Colitis,
    inflammation of the large intestine, may also be
    present. Some types of acute gastroenteritis will
    not resolve without antibiotic treatment,
    especially when bacteria or exposure to
    parasites are the cause. Physicians may want to
    diagnose the cause by analyzing a stool
    sample, when stomach symptoms remain
PATIENT’S PROFILE:
   Name           : A. P.
   Age            : 26 years old
   Sex            : Male
   Nationality    : Indian
   Status         : Single
   Religion       : Hindu
   Weight         : 71Kg.
   Date Admitted : 2012/11/18
   Chief Complaint: abdominal pains, nausea
                      and vomiting, watery stools
   Admitting Diagnosis: Acute Gastroenteritis
PAST HEALTH HISTORY
 The patient use to have typical cough, colds
  and fever and never had experienced major
  illness that required hospitalization. He does
  not have any known allergies to food or drugs.
PRESENT HEALTH HISTORY
 Two days prior to admission, the patient

  experienced persistent loose watery bowel
  movement accompanied by vomiting,
  abdominal pains and fever.
ANATOMY AND PHYSIOLOGY
   The human digestive system is a complex
    series of organs and glands that processes
    food. In order to use the food we eat, our body
    has to break the food down into smaller
    molecules that it can process; it also has to
    excrete waste. Most of the digestive organs
    (like the stomach and intestines) are tube-like
    and contain the food as it makes its way
    through the body. The digestive system is
    essentially a long, twisting tube that runs from
    the mouth to the anus, plus a few other organs
    (like the liver and pancreas) that produce or
    store digestive chemicals.
The Digestive Process: The start of
  the process - the mouth:
 The digestive process begins in

  the mouth. Food is partly broken
  down by the process of chewing
  and by the chemical action of
  salivary enzymes (these
  enzymes are produced by the
  salivary glands and break down
  starches into smaller molecules).
On the way to the stomach: the esophagus
 After being chewed and swallowed, the food

  enters the esophagus. The esophagus is a
  long tube that runs from the mouth to
  the stomach. It uses rhythmic, wave-like
  muscle movements (called peristalsis) to force
  food from the throat into the stomach. This
  muscle movement gives us the ability to eat
  or drink even when we're upside-down.
In the stomach
 The stomach is a large, sack-like organ that churns
  the food and bathes it in a very strong acid (gastric
  acid). Food in the stomach that is partly digested and
  mixed with stomach acids is called chyme.
In the small intestine
 After being in the stomach, food enters

  the duodenum, the first part of the small intestine. It
  then enters the jejunum and then the ileum (the final
  part of the small intestine). In the small intestine, bile
  (produced in the liver and stored in the gall
  bladder),pancreatic enzymes, and other digestive
  enzymes produced by the inner wall of the small
  intestine help in the breakdown of food.
In the large intestine
 After passing through the small intestine, food
  passes into the large intestine. In the large intestine,
  some of the water and electrolytes (chemicals like
  sodium) are removed from the food. Many microbes
  in the large intestine help in the digestion process.
 The first part of the large intestine is called the

  cecum (the appendix is connected to the cecum).
  Food then travels upward in the ascending colon.
  The food travels across the abdomen in the
  transverse colon, goes back down the other side of
  the body in the descending colon, and then through
  the sigmoid colon.
The end of the process
 Solid waste is then stored in the rectum until it

  is excreted via the anus.
Pathophysiology:
   The mechanisms potentially responsible for
    viral diarrhea include lysis of enterocytes,
    interference with the brush border function that
    leads to malabsorption of electrolytes,
    stimulation of cyclic adenosine monophosphate
    (CAMP), and carbohydrate malabsorption. For
    bacterial gastroenteritis, the pathophysiology
    involves the elaboration of toxin by
    enterotoxigenic pathogens and the invasion
    and inflammation of mucosa by invasive
    pathogens. Parasitic organisms invade
    epithelial cells and cause villus atrophy and
    eventual malabsorption.
Signs and Symptoms
 Low grade fever to 100°F (37.8°C)
 Nausea with or without vomiting

 Mild to moderate diarrhea

 Crampy and painful abdominal bloating

More serious symptoms include:
 Blood in vomit or stool

 Vomiting more than 48 hours

 Fever higher than 40°C

 Swollen abdomen or abdominal pain

 Dehydration that is manifested by weakness,
  light-headedness, decreased and concentrated
  urination, dry skin and poor turgor, and dry lips
  and mouth
Diagnostic Tests:
 Blood test

 Physical examination to rule other existing

  conditions such as appendicitis
NURSING ASSESSMENT

System Review and Nursing Assessment
 Temp : 38.6ºC  HR: 137bpm
 BP: 118/76mmHg   RR: 26cpm
 Weight: 75 kgs
INSTRUCTIONS:
Place an [X] in the area of abnormality. Comment at the space
provided. Indicate the location of the problem in the figure using
[X].
GASTROINTESTINAL TRACT:
 [ ] obese      [ X ] distention [ ] mass
   [ ] dysphagia [ ] rigidity [ X ] pain
 Assess abdomen, bowel habits, swallowing, bowel sounds,
   comfort [ ]no problem
MUSCULOSKELETAL AND SKIN:
 Watery Stools

 [ ] stiffness [ ] itching
   [ ] hot [ ] drainage [ ] prosthesis [ ] swelling
 [ ] lesion [X] poor skin turgor [ ] cool [ ] flushed
   [ ] atrophy [ ] pain
 [ ] diaphoretic/moist

- Assess mobility, motion, gait, alignment, joint function, skin
   color, texture, turgor, integrity
 [ ] no problem
LAB. TEST
 Fecal Occult Blood Test- result is negative
DRUG STUDY
1. Generic Name: Loperamide 2 mg tablet
  Brand Name: Imodium
  Classification: Antidiarrheal

 Uses:
 Acute nonspecific diarrhea, chronic diarrhea
 associated with inflammatory bowel disease,
 and to reduce fecal volume from ileostomies.
Adverse effects:
 Body Whole:Hypersensitivity (skin rash);
  fever.
  CNS: Drowsiness, fatigue, dizziness,
  CNS depression (overdosage).
  GI:Abdominal discomfort or
  pain, abdominal distention, bloating,
  constipation, nausea, vomiting, anorexia, dry
  mouth
Nursing implications:
 Monitor therapeutic effectiveness.

 Discontinue if there is no improvement after 48

  hours of therapy for acute diarrhea.
 Monitor fluid and electrolyte balance.

 Notify physician promptly if the patient

  with ulcerativecolitis develops abdominal
  distention or other GI symptoms
2. Generic Name: Hyoscine ButylBromide
  Brand Name: Buscopan
  Classification: Antispasmodic
 Uses:

  Spastic states and to prevent nausea and
  vomiting
Adverse Effects:
 Overdose may produce temporary paralysis of
  ciliary muscle; papillary dilation; tachycardia;
  palpitations; hot, dry, or flushed skin; absence
  of bowel sounds; hyperthermia; increased
  respiratory rate; EKG abnormalities; nausea;
  vomiting; rash over face or upper trunk; CNS
  stimulations; and psychosis (marked by
  agitation, restlessness, rambling speech, visual
  hallucinations, paranoid behavior, and
  delusions, followed by depression).
Nursing implications:
 Use cautiously in patients with autonomic
  neuropathy, hyperthyroidism, coronary artery
  disease, arrhythmias, heart failure,
  hypertension, hiatal hernia with reflux
  esophagitis, hepatic or renal disease, known
  as suspected GI infection, or ulcerative colitis.
 Use cautiously in children.

 Use cautiously in patients in hot or humid

  environments; drug can cause heat
Assessment             Nursing      Planning          Interventions         Evaluation
                        Diagnosis
Subjective:           Acute          After 4 hours of   1. Place patient on   After 4 hours of
“My abdomen is        pain related   nursing            a comfortable         nursing
very painful!” as     to abdominal   interventions,     position              interventions,
                                     the patient will                         the patient
patient claimed.      distension.                       2. Monitor and
                                     report a relief                          reported relief
-with pain scale of                  from pain as       record VS             from abdominal
6/10                                 manifested by a    3.Assess patient’s    pains
                                     calm facial        level of pain         - no facial
Objective:                           expression.        4. Provide warm       grimace
   abdominal                                            compress over the     - calm facial
   cramping                                             abdominal area        expression
    irritability                                        5. Administer         -Pain scale of
    holds                                               medications as        0/10
    abdomen                                             ordered
    facial grimace
Assessment             Nursing            Planning           Interventions            Evaluation
                        Diagnosis
Objective:          Deficient fluid       After 8 hours of    1. Establish rapport     After 8 hours of
   passage of loose volume RT excessive   nursing             2. Monitor and           nursing
  watery stool      losses through        interventions,      record VS                interventions,
                                          the patient will                             the patient
    nausea          normal routes AMB                         3.Assess patient’s
                                          report                                       reported
    Vomiting        frequent passage of   understanding of    condition                understanding of
    abdominal       loose watery stool    causative factors      4. Monitor Input      causative factors
   cramping                               for fluid volume       & Output balance      for fluid volume
   Poor skin turgor                       deficit               5. Maintain            deficit
                                                                adequate             - good skin turgor
   weakness                                                     hydration,
                                                                                     - no vomiting noted
                                                                increase fluid
                                                                intake.
                                                                6. Provide
                                                                frequent oral
                                                                care
                                                                7. Administer
                                                                Intravenous
                                                                fluids as
                                                                prescribed
                                                                8 Restrict solid
                                                                food intake, as
                                                                indicated
AGE

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AGE

  • 2. INTRODUCTION Acute Gastroenteritis  Acute Gastroenteritis is inflammation of the gastrointestinal tract , involving both the stomach and the small intestine and resulting in acute diarrhea . The inflammation is caused most often by infection with certain viruses , less often by bacrteria or their toxins , parasites or adverse reaction to something in the diet or medication.
  • 3. Different species of bacteria can cause gastroenteritis, including Salmonella , Shigella, Staphylococcus, Campylobacter jejuni, Clostridium, Escherichia coli, Yersinia , and others. Each organism causes slightly different sympto ms but all result in diarrhea. Colitis, inflammation of the large intestine, may also be present. Some types of acute gastroenteritis will not resolve without antibiotic treatment, especially when bacteria or exposure to parasites are the cause. Physicians may want to diagnose the cause by analyzing a stool sample, when stomach symptoms remain
  • 4. PATIENT’S PROFILE:  Name : A. P.  Age : 26 years old  Sex : Male  Nationality : Indian  Status : Single  Religion : Hindu  Weight : 71Kg.  Date Admitted : 2012/11/18  Chief Complaint: abdominal pains, nausea and vomiting, watery stools  Admitting Diagnosis: Acute Gastroenteritis
  • 5. PAST HEALTH HISTORY  The patient use to have typical cough, colds and fever and never had experienced major illness that required hospitalization. He does not have any known allergies to food or drugs. PRESENT HEALTH HISTORY  Two days prior to admission, the patient experienced persistent loose watery bowel movement accompanied by vomiting, abdominal pains and fever.
  • 6. ANATOMY AND PHYSIOLOGY  The human digestive system is a complex series of organs and glands that processes food. In order to use the food we eat, our body has to break the food down into smaller molecules that it can process; it also has to excrete waste. Most of the digestive organs (like the stomach and intestines) are tube-like and contain the food as it makes its way through the body. The digestive system is essentially a long, twisting tube that runs from the mouth to the anus, plus a few other organs (like the liver and pancreas) that produce or store digestive chemicals.
  • 7. The Digestive Process: The start of the process - the mouth:  The digestive process begins in the mouth. Food is partly broken down by the process of chewing and by the chemical action of salivary enzymes (these enzymes are produced by the salivary glands and break down starches into smaller molecules).
  • 8. On the way to the stomach: the esophagus  After being chewed and swallowed, the food enters the esophagus. The esophagus is a long tube that runs from the mouth to the stomach. It uses rhythmic, wave-like muscle movements (called peristalsis) to force food from the throat into the stomach. This muscle movement gives us the ability to eat or drink even when we're upside-down.
  • 9. In the stomach  The stomach is a large, sack-like organ that churns the food and bathes it in a very strong acid (gastric acid). Food in the stomach that is partly digested and mixed with stomach acids is called chyme. In the small intestine  After being in the stomach, food enters the duodenum, the first part of the small intestine. It then enters the jejunum and then the ileum (the final part of the small intestine). In the small intestine, bile (produced in the liver and stored in the gall bladder),pancreatic enzymes, and other digestive enzymes produced by the inner wall of the small intestine help in the breakdown of food.
  • 10. In the large intestine  After passing through the small intestine, food passes into the large intestine. In the large intestine, some of the water and electrolytes (chemicals like sodium) are removed from the food. Many microbes in the large intestine help in the digestion process.  The first part of the large intestine is called the cecum (the appendix is connected to the cecum). Food then travels upward in the ascending colon. The food travels across the abdomen in the transverse colon, goes back down the other side of the body in the descending colon, and then through the sigmoid colon.
  • 11. The end of the process  Solid waste is then stored in the rectum until it is excreted via the anus.
  • 12. Pathophysiology:  The mechanisms potentially responsible for viral diarrhea include lysis of enterocytes, interference with the brush border function that leads to malabsorption of electrolytes, stimulation of cyclic adenosine monophosphate (CAMP), and carbohydrate malabsorption. For bacterial gastroenteritis, the pathophysiology involves the elaboration of toxin by enterotoxigenic pathogens and the invasion and inflammation of mucosa by invasive pathogens. Parasitic organisms invade epithelial cells and cause villus atrophy and eventual malabsorption.
  • 13. Signs and Symptoms  Low grade fever to 100°F (37.8°C)  Nausea with or without vomiting  Mild to moderate diarrhea  Crampy and painful abdominal bloating More serious symptoms include:  Blood in vomit or stool  Vomiting more than 48 hours  Fever higher than 40°C  Swollen abdomen or abdominal pain  Dehydration that is manifested by weakness, light-headedness, decreased and concentrated urination, dry skin and poor turgor, and dry lips and mouth
  • 14. Diagnostic Tests:  Blood test  Physical examination to rule other existing conditions such as appendicitis
  • 15. NURSING ASSESSMENT System Review and Nursing Assessment  Temp : 38.6ºC HR: 137bpm  BP: 118/76mmHg RR: 26cpm  Weight: 75 kgs
  • 16. INSTRUCTIONS: Place an [X] in the area of abnormality. Comment at the space provided. Indicate the location of the problem in the figure using [X]. GASTROINTESTINAL TRACT:  [ ] obese [ X ] distention [ ] mass [ ] dysphagia [ ] rigidity [ X ] pain  Assess abdomen, bowel habits, swallowing, bowel sounds, comfort [ ]no problem MUSCULOSKELETAL AND SKIN:  Watery Stools  [ ] stiffness [ ] itching [ ] hot [ ] drainage [ ] prosthesis [ ] swelling  [ ] lesion [X] poor skin turgor [ ] cool [ ] flushed [ ] atrophy [ ] pain  [ ] diaphoretic/moist - Assess mobility, motion, gait, alignment, joint function, skin color, texture, turgor, integrity  [ ] no problem
  • 17. LAB. TEST  Fecal Occult Blood Test- result is negative
  • 18. DRUG STUDY 1. Generic Name: Loperamide 2 mg tablet Brand Name: Imodium Classification: Antidiarrheal Uses: Acute nonspecific diarrhea, chronic diarrhea associated with inflammatory bowel disease, and to reduce fecal volume from ileostomies.
  • 19. Adverse effects:  Body Whole:Hypersensitivity (skin rash); fever. CNS: Drowsiness, fatigue, dizziness, CNS depression (overdosage). GI:Abdominal discomfort or pain, abdominal distention, bloating, constipation, nausea, vomiting, anorexia, dry mouth
  • 20. Nursing implications:  Monitor therapeutic effectiveness.  Discontinue if there is no improvement after 48 hours of therapy for acute diarrhea.  Monitor fluid and electrolyte balance.  Notify physician promptly if the patient with ulcerativecolitis develops abdominal distention or other GI symptoms
  • 21. 2. Generic Name: Hyoscine ButylBromide Brand Name: Buscopan Classification: Antispasmodic  Uses: Spastic states and to prevent nausea and vomiting
  • 22. Adverse Effects:  Overdose may produce temporary paralysis of ciliary muscle; papillary dilation; tachycardia; palpitations; hot, dry, or flushed skin; absence of bowel sounds; hyperthermia; increased respiratory rate; EKG abnormalities; nausea; vomiting; rash over face or upper trunk; CNS stimulations; and psychosis (marked by agitation, restlessness, rambling speech, visual hallucinations, paranoid behavior, and delusions, followed by depression).
  • 23. Nursing implications:  Use cautiously in patients with autonomic neuropathy, hyperthyroidism, coronary artery disease, arrhythmias, heart failure, hypertension, hiatal hernia with reflux esophagitis, hepatic or renal disease, known as suspected GI infection, or ulcerative colitis.  Use cautiously in children.  Use cautiously in patients in hot or humid environments; drug can cause heat
  • 24. Assessment Nursing Planning Interventions Evaluation Diagnosis Subjective: Acute After 4 hours of 1. Place patient on After 4 hours of “My abdomen is pain related nursing a comfortable nursing very painful!” as to abdominal interventions, position interventions, the patient will the patient patient claimed. distension. 2. Monitor and report a relief reported relief -with pain scale of from pain as record VS from abdominal 6/10 manifested by a 3.Assess patient’s pains calm facial level of pain - no facial Objective: expression. 4. Provide warm grimace abdominal compress over the - calm facial cramping abdominal area expression irritability 5. Administer -Pain scale of holds medications as 0/10 abdomen ordered facial grimace
  • 25. Assessment Nursing Planning Interventions Evaluation Diagnosis Objective: Deficient fluid After 8 hours of 1. Establish rapport After 8 hours of passage of loose volume RT excessive nursing 2. Monitor and nursing watery stool losses through interventions, record VS interventions, the patient will the patient nausea normal routes AMB 3.Assess patient’s report reported Vomiting frequent passage of understanding of condition understanding of abdominal loose watery stool causative factors 4. Monitor Input causative factors cramping for fluid volume & Output balance for fluid volume Poor skin turgor deficit 5. Maintain deficit adequate - good skin turgor weakness hydration, - no vomiting noted increase fluid intake. 6. Provide frequent oral care 7. Administer Intravenous fluids as prescribed 8 Restrict solid food intake, as indicated