continuation on the urinary tract disorders. congenital and acquired disorders well covered. pyelonephritis also forms part of the text. thanks for reading. remeber to like and follow
Please find the power point on Renal and bladder stones. I tried present it on understandable way and all the contents are reviewed by experts and from very reliable references. Thank you
Urolithiasis is a common disease that is estimated to
produce medical costs of $2.1 billion per year in the United States alone.
Renal colic affects approximately 1.2 million people
each year in USA and accounts for approximately 1% of
all hospital admissions.
Most active emergency departments (EDs) manage
patients with acute renal colic every day.
Please find the power point on Renal and bladder stones. I tried present it on understandable way and all the contents are reviewed by experts and from very reliable references. Thank you
Urolithiasis is a common disease that is estimated to
produce medical costs of $2.1 billion per year in the United States alone.
Renal colic affects approximately 1.2 million people
each year in USA and accounts for approximately 1% of
all hospital admissions.
Most active emergency departments (EDs) manage
patients with acute renal colic every day.
Please find the power point on Urinary Tract Injury (Kidney Injury). I tried to present it on understandable way and all the contents are reviewed by experts and from very reliable references. Thank you
Dear Viewers,
Greetings from “ Surgical Educator”
Today I have uploaded a video on one of the congenital causes for obstructive jaundice- Biliary Atresia. In this episode, I am discussing about the etiology, types, clinical features, investigations, treatment and surgical outcome of Biliary Atresia. I hope you will enjoy the video. You can watch all my surgical teaching video casts in the following link: surgicaleducator.blogspot.com.
definition of hydronephrosis,
causes and types of hydronephrosis
pathophysiology of hydronephrosis
clinical manifestation and diagnostic test for hydronephrosis
management
Please find the power point on Urinary Tract Injury (Kidney Injury). I tried to present it on understandable way and all the contents are reviewed by experts and from very reliable references. Thank you
Dear Viewers,
Greetings from “ Surgical Educator”
Today I have uploaded a video on one of the congenital causes for obstructive jaundice- Biliary Atresia. In this episode, I am discussing about the etiology, types, clinical features, investigations, treatment and surgical outcome of Biliary Atresia. I hope you will enjoy the video. You can watch all my surgical teaching video casts in the following link: surgicaleducator.blogspot.com.
definition of hydronephrosis,
causes and types of hydronephrosis
pathophysiology of hydronephrosis
clinical manifestation and diagnostic test for hydronephrosis
management
CHOLELITHIASIS, NEPHROLITHIASIS SECONDARY HYPERTENSION, DM TYPE 2, HYDRONEPH...Jack Frost
CHOLELITHIASIS, NEPHROLITHIASIS
SECONDARY HYPERTENSION, DM TYPE 2, HYDRONEPHROSIS
This presentation contains real names of persons involve of this particular study. This names should not be copied or rewritten. Used the data of this study as basis only. All rights reserved 2009.
Nephrolithiasis, commonly known as kidney stones, refers to the formation of hard mineral and salt deposits within the kidneys or urinary tract. These stones can vary in size, ranging from tiny grains to larger, more substantial formations. Nephrolithiasis is a relatively common condition and can affect people of all ages, although it is more prevalent in adults.
Child survival strategies- interventions that lead to a childhood mortality reduction in line with the SDG(in children under 5)
The proposed SDG target for child mortality aims to end, by 2030, preventable deaths of newborns and children under 5 years of age, with all countries aiming to reduce neonatal mortality to at least as low as 12 deaths per 1,000 live births and under-5 mortality to at least as low as 25 deaths per 1,000 live births.
the recent data on child mortality are well covered.
follow the GOBIFF for seurity of the future.
easy description of common lut disorders. improvements on the slides accepted. text includes congenital and acquired disorders. more so the causes of bladder outlet obstructions. also management of the disorders are breifly described.
ARTIFICIAL INTELLIGENCE IN HEALTHCARE.pdfAnujkumaranit
Artificial intelligence (AI) refers to the simulation of human intelligence processes by machines, especially computer systems. It encompasses tasks such as learning, reasoning, problem-solving, perception, and language understanding. AI technologies are revolutionizing various fields, from healthcare to finance, by enabling machines to perform tasks that typically require human intelligence.
Lung Cancer: Artificial Intelligence, Synergetics, Complex System Analysis, S...Oleg Kshivets
RESULTS: Overall life span (LS) was 2252.1±1742.5 days and cumulative 5-year survival (5YS) reached 73.2%, 10 years – 64.8%, 20 years – 42.5%. 513 LCP lived more than 5 years (LS=3124.6±1525.6 days), 148 LCP – more than 10 years (LS=5054.4±1504.1 days).199 LCP died because of LC (LS=562.7±374.5 days). 5YS of LCP after bi/lobectomies was significantly superior in comparison with LCP after pneumonectomies (78.1% vs.63.7%, P=0.00001 by log-rank test). AT significantly improved 5YS (66.3% vs. 34.8%) (P=0.00000 by log-rank test) only for LCP with N1-2. Cox modeling displayed that 5YS of LCP significantly depended on: phase transition (PT) early-invasive LC in terms of synergetics, PT N0—N12, cell ratio factors (ratio between cancer cells- CC and blood cells subpopulations), G1-3, histology, glucose, AT, blood cell circuit, prothrombin index, heparin tolerance, recalcification time (P=0.000-0.038). Neural networks, genetic algorithm selection and bootstrap simulation revealed relationships between 5YS and PT early-invasive LC (rank=1), PT N0—N12 (rank=2), thrombocytes/CC (3), erythrocytes/CC (4), eosinophils/CC (5), healthy cells/CC (6), lymphocytes/CC (7), segmented neutrophils/CC (8), stick neutrophils/CC (9), monocytes/CC (10); leucocytes/CC (11). Correct prediction of 5YS was 100% by neural networks computing (area under ROC curve=1.0; error=0.0).
CONCLUSIONS: 5YS of LCP after radical procedures significantly depended on: 1) PT early-invasive cancer; 2) PT N0--N12; 3) cell ratio factors; 4) blood cell circuit; 5) biochemical factors; 6) hemostasis system; 7) AT; 8) LC characteristics; 9) LC cell dynamics; 10) surgery type: lobectomy/pneumonectomy; 11) anthropometric data. Optimal diagnosis and treatment strategies for LC are: 1) screening and early detection of LC; 2) availability of experienced thoracic surgeons because of complexity of radical procedures; 3) aggressive en block surgery and adequate lymph node dissection for completeness; 4) precise prediction; 5) adjuvant chemoimmunoradiotherapy for LCP with unfavorable prognosis.
The prostate is an exocrine gland of the male mammalian reproductive system
It is a walnut-sized gland that forms part of the male reproductive system and is located in front of the rectum and just below the urinary bladder
Function is to store and secrete a clear, slightly alkaline fluid that constitutes 10-30% of the volume of the seminal fluid that along with the spermatozoa, constitutes semen
A healthy human prostate measures (4cm-vertical, by 3cm-horizontal, 2cm ant-post ).
It surrounds the urethra just below the urinary bladder. It has anterior, median, posterior and two lateral lobes
It’s work is regulated by androgens which are responsible for male sex characteristics
Generalised disease of the prostate due to hormonal derangement which leads to non malignant enlargement of the gland (increase in the number of epithelial cells and stromal tissue)to cause compression of the urethra leading to symptoms (LUTS
Ethanol (CH3CH2OH), or beverage alcohol, is a two-carbon alcohol
that is rapidly distributed in the body and brain. Ethanol alters many
neurochemical systems and has rewarding and addictive properties. It
is the oldest recreational drug and likely contributes to more morbidity,
mortality, and public health costs than all illicit drugs combined. The
5th edition of the Diagnostic and Statistical Manual of Mental Disorders
(DSM-5) integrates alcohol abuse and alcohol dependence into a single
disorder called alcohol use disorder (AUD), with mild, moderate,
and severe subclassifications (American Psychiatric Association, 2013).
In the DSM-5, all types of substance abuse and dependence have been
combined into a single substance use disorder (SUD) on a continuum
from mild to severe. A diagnosis of AUD requires that at least two of
the 11 DSM-5 behaviors be present within a 12-month period (mild
AUD: 2–3 criteria; moderate AUD: 4–5 criteria; severe AUD: 6–11 criteria).
The four main behavioral effects of AUD are impaired control over
drinking, negative social consequences, risky use, and altered physiological
effects (tolerance, withdrawal). This chapter presents an overview
of the prevalence and harmful consequences of AUD in the U.S.,
the systemic nature of the disease, neurocircuitry and stages of AUD,
comorbidities, fetal alcohol spectrum disorders, genetic risk factors, and
pharmacotherapies for AUD.
Report Back from SGO 2024: What’s the Latest in Cervical Cancer?bkling
Are you curious about what’s new in cervical cancer research or unsure what the findings mean? Join Dr. Emily Ko, a gynecologic oncologist at Penn Medicine, to learn about the latest updates from the Society of Gynecologic Oncology (SGO) 2024 Annual Meeting on Women’s Cancer. Dr. Ko will discuss what the research presented at the conference means for you and answer your questions about the new developments.
Ozempic: Preoperative Management of Patients on GLP-1 Receptor Agonists Saeid Safari
Preoperative Management of Patients on GLP-1 Receptor Agonists like Ozempic and Semiglutide
ASA GUIDELINE
NYSORA Guideline
2 Case Reports of Gastric Ultrasound
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New Directions in Targeted Therapeutic Approaches for Older Adults With Mantl...i3 Health
i3 Health is pleased to make the speaker slides from this activity available for use as a non-accredited self-study or teaching resource.
This slide deck presented by Dr. Kami Maddocks, Professor-Clinical in the Division of Hematology and
Associate Division Director for Ambulatory Operations
The Ohio State University Comprehensive Cancer Center, will provide insight into new directions in targeted therapeutic approaches for older adults with mantle cell lymphoma.
STATEMENT OF NEED
Mantle cell lymphoma (MCL) is a rare, aggressive B-cell non-Hodgkin lymphoma (NHL) accounting for 5% to 7% of all lymphomas. Its prognosis ranges from indolent disease that does not require treatment for years to very aggressive disease, which is associated with poor survival (Silkenstedt et al, 2021). Typically, MCL is diagnosed at advanced stage and in older patients who cannot tolerate intensive therapy (NCCN, 2022). Although recent advances have slightly increased remission rates, recurrence and relapse remain very common, leading to a median overall survival between 3 and 6 years (LLS, 2021). Though there are several effective options, progress is still needed towards establishing an accepted frontline approach for MCL (Castellino et al, 2022). Treatment selection and management of MCL are complicated by the heterogeneity of prognosis, advanced age and comorbidities of patients, and lack of an established standard approach for treatment, making it vital that clinicians be familiar with the latest research and advances in this area. In this activity chaired by Michael Wang, MD, Professor in the Department of Lymphoma & Myeloma at MD Anderson Cancer Center, expert faculty will discuss prognostic factors informing treatment, the promising results of recent trials in new therapeutic approaches, and the implications of treatment resistance in therapeutic selection for MCL.
Target Audience
Hematology/oncology fellows, attending faculty, and other health care professionals involved in the treatment of patients with mantle cell lymphoma (MCL).
Learning Objectives
1.) Identify clinical and biological prognostic factors that can guide treatment decision making for older adults with MCL
2.) Evaluate emerging data on targeted therapeutic approaches for treatment-naive and relapsed/refractory MCL and their applicability to older adults
3.) Assess mechanisms of resistance to targeted therapies for MCL and their implications for treatment selection
Couples presenting to the infertility clinic- Do they really have infertility...Sujoy Dasgupta
Dr Sujoy Dasgupta presented the study on "Couples presenting to the infertility clinic- Do they really have infertility? – The unexplored stories of non-consummation" in the 13th Congress of the Asia Pacific Initiative on Reproduction (ASPIRE 2024) at Manila on 24 May, 2024.
Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
2. Definition
• Disorders of the kidneys and ureters.
• Disorders can either be congenital or acquired.
• Acquired disorders of urinary tract-
A)urinary Tract infections- pyelonephritis,
B)urological Obstructions- calculi
C) neoplasms- benign and malignant(kidney,ureter tumors)
5. Renal agenesis
Bilateral renal agenesis
• both mesonephric ducts fail to develop.
• Incompatible with life.
Unilateral renal agenesis
• the mesonephric duct fails to develop.
• Usually there is absent ureter, trigone, kidney and
(in boys) vas deferens.
• Prevalence of unilateral renal agenesis is about
1:1400 and it
is usually discovered incidentally
6. Horseshoe kidney
• two distinct functioning kidneys on each side
of the midline, connected at the lower poles by
an isthmus of functioning renal parenchyma or
fibrous tissue that crosses the midline of the
body.
• occurs in 1 per 400-800 live births
• hydronephrosis, stone formation, infection are
common
7.
8. Congenital cystic kidneys (synonym:
polycystic kidneys)
• Are hereditary and potentially lethal
• transmitted by either parent as an autosomal
dominant trait. Thus, the risk of an offspring
inheriting the condition can be high
• The disease is not usually detectable on
standard imaging until the second and third
decades of life and does not usually manifest
itself clinically before the age of 30 years.
• Types- adult and infantile
9.
10. Duplication of a ureter
• a congenital condition in which the ureteric
bud splits (or arises twice), resulting in two
ureters draining a single kidney.
• occurrs in approximately 1% of the
population.
• Can be : partial and complete.
11.
12. Ureterocele
• the distal ureter balloons at its opening into the bladder.
• Treatment should be avoided unless there are symptoms
arising from infection and/or stone formation.
• Ureterocele is most common in women; occasionally, the
cyst may cause obstruction to the bladder outflow by
prolapsing into the internal urethral opening.
• Endoscopic diathermy incision is usually all that is required
for treatment of a symptomatic ureterocele, although a
micturating cystogram is advisable to detect postoperative
urinary reflux.
• In advanced unilateral cases with hydronephrosis or
pyonephrosis, nephrectomy may be appropriate.
13.
14. Pelvicureteric junction obstruction
• obstruction of the flow of urine from the renal pelvis to the proximal
ureter.
• Etiology-aperistaltic segment of ureter due to absent muscles or
crossing vessels over UPJ.
Clinical features-may present at any time (before birth, in childhood, or
in adulthood) by:
• abdominal mass.
• abdominal pain.
• Haematuria after fairly minor abdominal trauma.
Diagnostic evaluation-
• IVU - shows delay in appearance of contrast and dilated renal pelvis
and calices.
• Renal scan -shows differential renal function and confirms
obstruction
15. Management-
• Surgery is indicated for:
1. obstructive symptoms,
2. stone formation,
3. recurrent urinary infection,
4. progressive renal impairment.
• Pyeloplasty is the treatment of choice
• Nephrectomy is performed if the affected kidney is
<10% of total renal function.
16. Vesicoureteric junction reflux
• retrograde flow of urine into upper urinary
tract.
• Ureters run obliquely and run submucosally
for some distance. plus ureter's muscular
attachments.
17.
18. • Micturating cystourethrography is the gold standard for
diagnosis and evaluation of VUR grade.
•Diuretic Renal scan (DMSA) is used to visualize scarring
and quantify differential renal function
Management
•antibiotic prophylaxis is recommended for children with
reflux of grades I-II.
•Surgery (uretero - vesical reimplantation or endoscopic
injection) is recommended in reflux of grades III-V and
persistent reflux despite a trial of antibiotics.
19. Pyelonephritis
• Pyelonephritis is inflammation of the kidney,
typically due to a bacterial infection.
• Common organisms are E. coli (70–80%)
and Enterococcus faecalis.
• Risk factors …….
20. Clinical features
• Fever
• Costovertebral angle pain
• Nausea and/or vomiting
• Mental status change
• Decompensation in another organ system
• Generalized deterioration
• hematuria
21. investigations
Definative
Urinalysis - Urine specimens may be obtained by
midstream clean catch, suprapubic aspiration,
or catheterization
Dipstick testing - glucose, protein, blood, nitrite,
and leukocyte esterase
Leukocyte esterase 57-96% sensitive and 94-
98% specific for identifying pyuria
22. Nitrite tests are sensitivity and specificity of 22% and 94-100%,
respectively.
Microscopic examination:
• Pyuria : Most truly infected patients have > 10 WBCs/μL. The
presence of bacteria in the absence of pyuria:due to contamination
during sampling.
• Microscopic hematuria occurs in up to 50% of patients, but gross
hematuria is uncommon.
• WBC casts: pyelonephritis, glomerulonephritis, and noninfective
tubulointerstitial nephritis.
• Pyuria in the absence of bacteriuria and of UTI is possible, for
example, if patients have nephrolithiasis, a uroepithelial tumor,
appendicitis, or inflammatory bowel disease or if the sample is
contaminated by vaginal WBCs
23. • Complete blood count.
• HIV test
• VDRL
• Erythrocyte sedimentation rate
• U/E/C
26. Renal calculi
• Incidence- The occurrence of urinary stones occurs predominantly in the
third to fifth decades of life and affects men more than women.
• About half of patients with a single renal stone have another episode within
5 years.
• Most stones contain calcium or magnesium in combination with
phosphorus or oxalate.
• Most stones are radiopaque and can be detected by x-ray studies
Types of stone-
• Calcium stone
• Oxalate stone
• Cystiene stone
• Struvite stone
27. Causes and predisposing factors of renal calculi:
• Chronic dehydration, poor fluid intake, and immobility
• Living in mountainous, desert, or tropical areas
• Infection, urinary stasis, and periods of immobility
• Inflammatory bowel disease and in patients with an ileostomy or
bowel resection because these patients absorb more oxalate.
• Medications- antacids, acetazolamide (Diamox), vitamin D,
laxatives, and high doses of aspirin
Location of stones-
• Kidney
• Ureter
• Bladder
• Urethra
• Prostate
29. Diagnostic evaluation
• Urinalysis-
hematuria and pyuria
pH < 5.5 indicates uric acid stone
pH > 7.5 indicates struvite stone
urine culture and drug sensitivity studies to detect
infection.
• 24-hour urine test for measurement of calcium,
uric acid, creatinine, sodium,citrate and oxalate
30. Blood studies-
• Hyperuracemia
• Hypercalcemia
• Neutrophilia
• Elevated serum parathyroid hormone
Stone chemistry-
• Collection of stone through a strainer is useful.
• Analyze the stone chemically to find out the
composition which helps in therapeutic
management.
31. Radiographic studies-
• Kidney, ureters, and bladder radiography may
show stone.
• Intra venous urogram (intravenous pyelogram) to
determine site and evaluate degree of obstruction
• Retrograde pyelography
• Ultrasound
• Helical or axial CT Scan
32. Management
General Principles
• If small stone (< 4 mm) and able to treat as
outpatient, 80% will pass stone spontaneously
with hydration, pain control, and reassurance.
• Hospitalized for intractable pain, persistent
vomiting, high-grade fever, obstruction with
infection, and solitary kidney with obstruction.
• Medical management
• Surgical management
33. Medical management
Goal-
Immediate goal- To relieve the pain until its
causes can be eliminated.
Long term goal (basic goal)-
• To eradicate the stone
• To determine the stone type
• To prevent nephron destruction
• To control infection
• To relieve any obstruction
34. Medical management
• Opioid analgesics or NSAIDs
• NSAIDs provide specific pain relief because
they inhibit the synthesis of prostaglandin E.
• Hot baths or moist heat to the flank areas may
also be useful.
• Fluids are encouraged. This increases the
hydrostatic pressure behind the stone, assisting
it in its downward passage.
• A high, around-the-clock fluid intake reduces
the concentration of urinary crystalloids, dilutes
the urine, and ensures a high urine output
35. Calcium stone-
• Cellulose sodium phosphate (Calcibind) may be effective
in preventing calcium stones.
• it binds calcium from food in the intestinal tract, reducing
the amount of calcium absorbed into the circulation.
• restrict calcium in diet
• Therapy with thiazide diuretics may be beneficial in
reducing the calcium loss in the urine
• The urine may be acidified by use of medications such as
ammonium chloride or acetohydroxamic acid
• Sodium and protein restriction diet
36. Uric acid stone-
• low-purine diet such as shellfish, anchovies, asparagus,
mushrooms, and organ meats are avoided
• Allopurinol may be prescribed to reduce serum uric acid
levels and urinary uric acid excretion.
• Proteins may be limited in diet
Cystine stone -
• Low-protein diet
• Penicillamine is administered to reduce the amount of
cystine in the urine
• urine is alkalinized.
Oxalate stone -
• Encourage the increased fluid intake
• Avoid the food contains oxalate such as- spinach,
strawberries, tea, peanuts, wheat bran
37. Non surgical management-
• Ureteroscopy
• ESWL (Extra Corporeal Shock wave lithotripsy)
• Endoscopic procedures
• Electrohydrolic lithotripsy
• Chemolysis
Surgical management-
• Nephrolithotomy - Incision into the kidney with removal of the
stone
• Nephrectomy – removal of kidney
• Pyelolithotomy - removal of stone from renal pelvis
• Ureterolithotomy - removal of stone from ureter
• Cystostomy – removal of stone from bladder
• Cystolitholapaxy - an instrument is inserted through the urethra
into the bladder, and the stone is crushed in the jaws of this
instrument