This ppt contains all the details about what is obesity, etiology, & mainly focuses on various methods of assessment of obesity from field tests to lab tests.
Prevalence of obesity.Body composition & body shape (body fat distribution ) and CVD risk .Mechanisms linking obesity with cardiovascular disease.Fat-but-Fit Paradigm and CVD,The Relationship of Metabolic Risk Factors and Cardiorespiratory Fitness. Metabolically Healthy but Obese ( MHO ) Phenotype and CVD.Obesity Paradox in Patients With CVD
to download this presentation from this link
https://mohmmed-ink.blogspot.com/2020/12/obesity.html
obesity, causes, diagnosis, complications, treatment, prevention.
This ppt contains all the details about what is obesity, etiology, & mainly focuses on various methods of assessment of obesity from field tests to lab tests.
Prevalence of obesity.Body composition & body shape (body fat distribution ) and CVD risk .Mechanisms linking obesity with cardiovascular disease.Fat-but-Fit Paradigm and CVD,The Relationship of Metabolic Risk Factors and Cardiorespiratory Fitness. Metabolically Healthy but Obese ( MHO ) Phenotype and CVD.Obesity Paradox in Patients With CVD
to download this presentation from this link
https://mohmmed-ink.blogspot.com/2020/12/obesity.html
obesity, causes, diagnosis, complications, treatment, prevention.
Nutritional Trends and Implications for Weight Loss Surgerymilfamln
Learning Objectives:
1. Describe and list the types of bariatric surgeries.
2. Identify current practice guidelines for MNT in bariatrics.
3. Identify key factors in pre-op assessments for long-term success.
The Okinawa Flat Belly Tonic is a new one of a kind weight loss “tonic” supplement. It helps men and women burn fat fast using a simple 20-second Japanese tonic. IF THAT TONIC DOES NOT WORK AS GIVEN YOUR VALUABLE MONEY WILL REFUND WITH IMMEDIATE EFFECT.
A group of physiological abnormalities such as an increase in blood pressure, diabetes, increase in cholesterol levels and obesity is known as Metabolic Syndrome. Women in their pregnancy period are highly prone to this problem. Doctors are taking the issue of metabolic syndrome in obstetric practice seriously as it may risk the pregnancy.
04 May 2015Page 1 of 28ProQuestIntegrating Fundamental Conce.docxmercysuttle
04 May 2015
Page 1 of 28
ProQuest
Integrating Fundamental Concepts of Obesity and Eating Disorders: Implications for the Obesity Epidemic
Author: Macpherson-Sánchez, Ann E, EdD, MNS
ProQuest document link
Abstract: Physiological mechanisms promote weight gain after famine. Because eating disorders, obesity, and dieting limit food intake, they are famine-like experiences. The development of the concept of meeting an ideal weight was the beginning of increasing obesity. Weight stigma, the perception of being fat, lack of understanding of normal growth and development, and increased concern about obesity on the part of health providers, parents, and caregivers have reinforced each other to promote dieting. Because weight suppression and disinhibition provoke long-term weight increase, dieting is a major factor producing the obesity epidemic. The integrated eating disorder-obesity theory included in this article emphasizes that, contrary to dieters, lifetime weight maintainers depend on physiological processes to control weight and experience minimal weight change.
Links: Linking Service
Full text: Headnote
Physiological mechanisms promote weight gain after famine. Because eating disorders, obesity, and dieting limit food intake, they are famine-like experiences. The development of the concept of meeting an ideal weight was the beginning of increasing obesity. Weight stigma, the perception of being fat, lack of understanding of normal growth and development, and increased concern about obesity on the part of health providers, parents, and caregivers have reinforced each other to promote dieting. Because weight suppression and disinhibition provoke long-term weight increase, dieting is a major factor producing the obesity epidemic. The integrated eating disorder-obesity theory included in this article emphasizes that, contrary to dieters, lifetime weight maintainers depend on physiological processes to control weight and experience minimal weight change. (Am J Public Health. 2015;105:e71-e85. doi:10. 2105/AJPH.2014.302507)
Since 1960, the Centers for Disease Control and Prevention has done periodic surveys of representative samples of the US population, which include measured heights and weights.1 From the 1960 to 1962 to the 1976 to 1980 measurement periods, there was little change in population weight. However, the next survey (1988-1994) showed increases in body mass index (BMI; defined as weight in kilograms divided by the square of height in meters [kg/m2]) that were unanticipated and inexplicable.2 Most of the increase occurred in those with BMI of 30 or greater.3 In 2006, a prominent Centers for Disease Control and Prevention researcher expressed frustration with her incapacity to explain why this happened.2
Losing weight and recuperating from that weight loss is part of the biological heritage of every human being.4-6 However, in the past 70 years, self-induced famine (dieting to achieve and maintain a lower weight)7 became the socie ...
The global prevalence of overweight and obesity as a public health concern is well established and reflects the overall lack of success in our ability to achieve and maintain healthy body weight. Being overweight and obese is associated with numerous comorbidities and is a risk factor for several of the leading causes of death, including cardiovascular disease, diabetes mellitus, and many types of cancer.
The foundation of treatment has been diet and exercise.
The effect of high-fat versus high-carb diet on body composition in strength-...RefoRefaat
Low-fat, high-carb (LFHC) and low-carb, high-fat (LCHF) diets change body composition as a consequence of the reduction of body fat of overweight persons. The
aim of this study is the assessment of the impact of LFHC and LCHF diets on body
composition of men of a healthy body mass who do strength sports while maintaining the appropriate calorific value in a diet and protein intake. The research involved
55 men aged 19–35, with an average BMI of 24.01 ± 1.17 (min. 20.1, max. 26.1). The
participants were divided into two groups following two interventional diets: highfat diet or high-carb diet, for 12 weeks. The body composition of the participants
Do fructose-containing sugars lead to adverse health consequences? Results of...Corn Refiners Association
At Experimental Biology 2014, the Sponsored Satellite Program “Sugars and Health Controversies: What Does the Science Say?” held in conjunction with the American Society for Nutrition’s Scientific Sessions took place on Saturday, April 26, 2014.
Panelist John L Sievenpiper, MD, PhD, presented science about sugars and their associated health outcomes.
Presents children at risk of developing Cholecystitis and/or Hepatomegaly, discuss laboratory values that suggest Cholecystitis and/or Hepatomegaly. lists the diference to order Liver U/S vs. HB Scan on mexican american overweight children.
HXR 2016: Which Comes First: Overeating or Obesity? -Dr. David Ludwig, Boston...HxRefactored
The conventional approach to weight loss, based on the calorie balance model, offers the simple advice, “eat less and move more.” Unfortunately, few people can maintain weight loss over the long term through calorie restriction because the body fights back, with rising hunger and slowing metabolism. An alternative approach to treatment aims to target the underlying driver of weight gain – fat cells overstimulated to hoard too many calories – leading to weight loss with less struggle.
These lecture slides, by Dr Sidra Arshad, offer a quick overview of physiological basis of a normal electrocardiogram.
Learning objectives:
1. Define an electrocardiogram (ECG) and electrocardiography
2. Describe how dipoles generated by the heart produce the waveforms of the ECG
3. Describe the components of a normal electrocardiogram of a typical bipolar leads (limb II)
4. Differentiate between intervals and segments
5. Enlist some common indications for obtaining an ECG
Study Resources:
1. Chapter 11, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 9, Human Physiology - From Cells to Systems, Lauralee Sherwood, 9th edition
3. Chapter 29, Ganong’s Review of Medical Physiology, 26th edition
4. Electrocardiogram, StatPearls - https://www.ncbi.nlm.nih.gov/books/NBK549803/
5. ECG in Medical Practice by ABM Abdullah, 4th edition
6. ECG Basics, http://www.nataliescasebook.com/tag/e-c-g-basics
Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
Explore natural remedies for syphilis treatment in Singapore. Discover alternative therapies, herbal remedies, and lifestyle changes that may complement conventional treatments. Learn about holistic approaches to managing syphilis symptoms and supporting overall health.
Prix Galien International 2024 Forum ProgramLevi Shapiro
June 20, 2024, Prix Galien International and Jerusalem Ethics Forum in ROME. Detailed agenda including panels:
- ADVANCES IN CARDIOLOGY: A NEW PARADIGM IS COMING
- WOMEN’S HEALTH: FERTILITY PRESERVATION
- WHAT’S NEW IN THE TREATMENT OF INFECTIOUS,
ONCOLOGICAL AND INFLAMMATORY SKIN DISEASES?
- ARTIFICIAL INTELLIGENCE AND ETHICS
- GENE THERAPY
- BEYOND BORDERS: GLOBAL INITIATIVES FOR DEMOCRATIZING LIFE SCIENCE TECHNOLOGIES AND PROMOTING ACCESS TO HEALTHCARE
- ETHICAL CHALLENGES IN LIFE SCIENCES
- Prix Galien International Awards Ceremony
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Ve...kevinkariuki227
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Verified Chapters 1 - 19, Complete Newest Version.pdf
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Verified Chapters 1 - 19, Complete Newest Version.pdf
Ozempic: Preoperative Management of Patients on GLP-1 Receptor Agonists Saeid Safari
Preoperative Management of Patients on GLP-1 Receptor Agonists like Ozempic and Semiglutide
ASA GUIDELINE
NYSORA Guideline
2 Case Reports of Gastric Ultrasound
Pulmonary Thromboembolism - etilogy, types, medical- Surgical and nursing man...VarunMahajani
Disruption of blood supply to lung alveoli due to blockage of one or more pulmonary blood vessels is called as Pulmonary thromboembolism. In this presentation we will discuss its causes, types and its management in depth.
The prostate is an exocrine gland of the male mammalian reproductive system
It is a walnut-sized gland that forms part of the male reproductive system and is located in front of the rectum and just below the urinary bladder
Function is to store and secrete a clear, slightly alkaline fluid that constitutes 10-30% of the volume of the seminal fluid that along with the spermatozoa, constitutes semen
A healthy human prostate measures (4cm-vertical, by 3cm-horizontal, 2cm ant-post ).
It surrounds the urethra just below the urinary bladder. It has anterior, median, posterior and two lateral lobes
It’s work is regulated by androgens which are responsible for male sex characteristics
Generalised disease of the prostate due to hormonal derangement which leads to non malignant enlargement of the gland (increase in the number of epithelial cells and stromal tissue)to cause compression of the urethra leading to symptoms (LUTS
8. NHANES1999 - 2000 to 2003 - 2004 Ogden, Carroll, Curtin, McDowell, Tabak and Flegal ( 2006), Prevalence of overweight and obesity in the United States, 1999 – 2004. JAMA, 295 (13) 1549 – 1555.
9. Estimated Economic Cost of Obesity in America Ogden, Carroll, Curtin, McDowell, Tabak and Flegal ( 2006), Prevalence of overweight and obesity in the United States, 1999 – 2004. JAMA, 295 (13) 1549 – 1555.
10. Obesity Potentiates the Accelerated Rate for Development of Select Health ConditionsCroghan, A. ( 2004). Nursing assessment: Gastrointestional system. In SM Lewis, MM Heitkemper, and SR Dirksen (eds). Medical surgical nursing: Assessment and management of clinical problems,6th ed. St. Louis: Mosby, Chs. 39.
11. Pathophysiology of ObesityHuether (2006). Alterations of digestive function. In KL McCance and SE Huether (eds). Pathophysiology, 5th ed. Salt Lake City: Elsevier, Chs.39.
12. Obesity PathophysiologyHuether (2006). Alterations of digestive function. In KL McCance and SE Huether (eds). Pathophysiology, 5th ed. Salt Lake City: Elsevier, Chs.39
13. Obesity PathophysiologyHuether (2006). Alterations of digestive function. In KL McCance and SE Huether (eds). Pathophysiology, 5th ed. Salt Lake City: Elsevier, Chs.39
14. Obesity PathophysiologyHuether (2006). Alterations of digestive function. In KL McCance and SE Huether (eds). Pathophysiology, 5th ed. Salt Lake City: Elsevier, Chs.39
15. Obesity PathophysiologyPhysiological Contributors of Obesity Huether (2006). Alterations of digestive function. In KL McCance and SE Huether (eds). Pathophysiology, 5th ed. Salt Lake City: Elsevier, Chs.39
16. Physiological Contributors to ObesityHuether (2006). Alterations of digestive function. In KL McCance and SE Huether (eds). Pathophysiology, 5th ed. Salt Lake City: Elsevier, Chs.39
17. Physiological Contributors to ObesityHuether (2006). Alterations of digestive function. In KL McCance and SE Huether (eds). Pathophysiology, 5th ed. Salt Lake City: Elsevier, Chs.39
18. Obesity PhysiologyHuether (2006). Alterations of digestive function. In KL McCance and SE Huether (eds). Pathophysiology, 5th ed. Salt Lake City: Elsevier, Chs.39
19. Obesity PathophysiologyOrexins and AnorexinsHuether (2006). Alterations of digestive function. In KL McCance and SE Huether (eds). Pathophysiology, 5th ed. Salt Lake City: Elsevier, Chs.39
20. Obesity PhysiologyHuether (2006). Alterations of digestive function. In KL McCance and SE Huether (eds). Pathophysiology, 5th ed. Salt Lake City: Elsevier, Chs.39.
21. Obesity PhysiologyLeptin. Huether (2006). Alterations of digestive function. In KL McCance and SE Huether (eds). Pathophysiology, 5th ed. Salt Lake City: Elsevier, Chs.39
22. Obesity PhysiologyGhrelin Huether (2006). Alterations of digestive function. In KL McCance and SE Huether (eds). Pathophysiology, 5th ed. Salt Lake City: Elsevier, Chs.39
25. Obesity PathophysiologyPoirier, P.,Giles,T.,Bray, G., Hong, Y.,Stern,J. PI-Sunger, X. & Eckel, R. (2006). Obesity and cardiovascular disease,Pathophysiology,evaluaton, and effect of weight loss ,Circulation, 113, 898-918.
42. NHLBI Summary of Evidence-Based Obesity Treatment RecommendationsFrom RCTshttp://www.nhlbi.nih.gov/guidelines/obesity/sum_rec.htm
43. NHLBI Summary of Evidence-Based Obesity Treatment RecommendationsFrom RCTs
44. NHLBI Summary of Evidence-Based Obesity Treatment RecommendationsFrom RCTs
45. NHLBI Summary of Evidence-Based Obesity Treatment RecommendationsFrom RCTs
46. NHLBI Summary of Evidence-Based Obesity Treatment RecommendationsFrom RCTs
47. NHLBI Summary of Evidence-Based Obesity Treatment RecommendationsFrom RCTs
48. NHLBI Summary of Evidence-Based Obesity Treatment RecommendationsFrom RCTs
49. NHLBI Summary of Evidence-Based Obesity Treatment RecommendationsFrom RCTs
50. NHLBI Summary of Evidence-Based Obesity Treatment RecommendationsFrom RCTs
51. NHLBI Summary of Evidence-Based Obesity Treatment RecommendationsFrom RCTs
52. NHLBI Summary of Evidence-Based Obesity Treatment RecommendationsFrom RCTs
53. NHLBI Summary of Evidence-Based Obesity Treatment RecommendationsFrom RCTs
54. NHLBI Summary of Evidence-Based Obesity Treatment RecommendationsFrom RCTs
55. NHLBI Summary of Evidence-Based Obesity Treatment RecommendationsFrom RCTs
56. NHLBI Summary of Evidence-Based Obesity Treatment RecommendationsFrom RCTs
57. NHLBI Summary of Evidence-Based Obesity Treatment RecommendationsFrom RCTs
58. NHLBI Summary of Evidence-Based Obesity Treatment RecommendationsFrom RCTs
59. NHLBI Summary of Evidence-Based Obesity Treatment RecommendationsFrom RCTs Diverse Patient Populations The possibility that a standard approach to weight loss will work differently in diverse patient populations must be considered when setting expectations about treatment outcomes. Evidence Category B.
60. Recommendations for Future Obesity ResearchPoirier, P., Giles, T., Bray, G., Hong, Y., Stern, J., PI-Sunyer, X., and Eckel, R. ( 2006). Obesity and cardiovascular disease: Pathophysiology,evaluation, and effect of weight loss. Circulation, 113, 898 – 918.