This presentation (made with the aid of Microsoft Powerpoint) depicts the morphological and clinical features, highlighting the pathology behind it.
I do not own copyrights for the images used in this video, however, kindly notify while sharing or using any material employed in this presentation. I hope you find this useful.
Endocarditis presentation to internal medicine2019hospital
This document discusses infective endocarditis, including its definition, classification, common sites of involvement, risk factors, general lesions, mortality rates, and Osler's nodes and Janeway lesions as associated findings on physical examination. Key points covered include that infective endocarditis is an infection of the endocardial surface, most commonly involving the heart valves. It can be classified as acute or subacute/chronic based on temporal factors and severity. Overall mortality is around 40% usually due to heart failure from valve dysfunction. Having a prosthetic valve or previous endocarditis are major risk factors.
Acute Tubular Necrosis | DR RAI M. AMMAR | ALL MEDICAL DATA
by DR RAI M. AMMAR
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This document provides information about a seminar on clubbing. It defines clubbing, describes its historical identification and terminology. It covers the causes, grades, clinical presentation including examination findings, and theories about the pathophysiology of clubbing. Clubbing is characterized by enlargement and curvature of the fingernails and fingertips. It most commonly occurs due to lung or heart conditions. Examination involves assessing the nail bed angle and depth, and performing Schamroth's test. The exact mechanism remains unknown but theories include neurogenic and humoral factors.
This is a lecture note for 5th-semester MBBS students. Lecture notes on hepatology, liver disease, and liver abscess. Introduction to a liver abscess, pyogenic liver abscess, causes, approach and management of liver abscess.
The document discusses two causes of obstructive uropathy: nephrolithiasis and hydronephrosis. Nephrolithiasis is the formation of urinary calculi (stones) in the kidneys or urinary tract. Calculi most commonly occur in middle-aged men and can cause pain. Hydronephrosis is the dilation of the renal pelvis and calyces due to partial or intermittent blockage of urine flow. It is usually caused by obstruction in the ureter but can also be congenital or due to conditions affecting the bladder. Advanced hydronephrosis leads to compression of the renal cortex. Both conditions are demonstrated in photographs showing enlarged kidneys with dilated pelvis and
Intestinal obstruction occurs when the normal passage of intestinal contents is blocked. It can involve the small intestine, large intestine, or both. Obstructions are classified as mechanical, which involve a physical blockage, or dynamic/adynamic, which involve ineffective motility without a blockage. Common causes include adhesions, hernias, tumors, and volvulus. Symptoms vary based on the location and severity of the obstruction but often include colicky abdominal pain, vomiting, distention, and constipation. Diagnosis involves physical exam findings like distention and hyperperistalsis as well as imaging tests showing gas/fluid levels and other signs of obstruction.
This presentation (made with the aid of Microsoft Powerpoint) depicts the morphological and clinical features, highlighting the pathology behind it.
I do not own copyrights for the images used in this video, however, kindly notify while sharing or using any material employed in this presentation. I hope you find this useful.
Endocarditis presentation to internal medicine2019hospital
This document discusses infective endocarditis, including its definition, classification, common sites of involvement, risk factors, general lesions, mortality rates, and Osler's nodes and Janeway lesions as associated findings on physical examination. Key points covered include that infective endocarditis is an infection of the endocardial surface, most commonly involving the heart valves. It can be classified as acute or subacute/chronic based on temporal factors and severity. Overall mortality is around 40% usually due to heart failure from valve dysfunction. Having a prosthetic valve or previous endocarditis are major risk factors.
Acute Tubular Necrosis | DR RAI M. AMMAR | ALL MEDICAL DATA
by DR RAI M. AMMAR
www.facebook.com/drraiammar
www.twitter.com/drraiammar
www.instagram.com/drraiammar
www.linkedin.com/in/drraiammar
www.medicall.com.pk/blog/auther/drraiammar/
For Any Book or Notes Visit Our Website:
www.allmedicaldata.wordpress.com
www.drraiammar.blogspot.com
YOUTUBE CHANNEL :
https://www.youtube.com/channel/UCu-oR9V3OdFNTJW5yqXWXxA
ANY QUESTION ??
Get in touch with us at Any of the Above Social Media or Email at
drraiammar@gmail.com
allmedicaldata@gmail.com
This document provides information about a seminar on clubbing. It defines clubbing, describes its historical identification and terminology. It covers the causes, grades, clinical presentation including examination findings, and theories about the pathophysiology of clubbing. Clubbing is characterized by enlargement and curvature of the fingernails and fingertips. It most commonly occurs due to lung or heart conditions. Examination involves assessing the nail bed angle and depth, and performing Schamroth's test. The exact mechanism remains unknown but theories include neurogenic and humoral factors.
This is a lecture note for 5th-semester MBBS students. Lecture notes on hepatology, liver disease, and liver abscess. Introduction to a liver abscess, pyogenic liver abscess, causes, approach and management of liver abscess.
The document discusses two causes of obstructive uropathy: nephrolithiasis and hydronephrosis. Nephrolithiasis is the formation of urinary calculi (stones) in the kidneys or urinary tract. Calculi most commonly occur in middle-aged men and can cause pain. Hydronephrosis is the dilation of the renal pelvis and calyces due to partial or intermittent blockage of urine flow. It is usually caused by obstruction in the ureter but can also be congenital or due to conditions affecting the bladder. Advanced hydronephrosis leads to compression of the renal cortex. Both conditions are demonstrated in photographs showing enlarged kidneys with dilated pelvis and
Intestinal obstruction occurs when the normal passage of intestinal contents is blocked. It can involve the small intestine, large intestine, or both. Obstructions are classified as mechanical, which involve a physical blockage, or dynamic/adynamic, which involve ineffective motility without a blockage. Common causes include adhesions, hernias, tumors, and volvulus. Symptoms vary based on the location and severity of the obstruction but often include colicky abdominal pain, vomiting, distention, and constipation. Diagnosis involves physical exam findings like distention and hyperperistalsis as well as imaging tests showing gas/fluid levels and other signs of obstruction.
This document discusses the evaluation of lymphadenopathy. It outlines the key steps in evaluation which include determining the size and characteristics of palpable lymph nodes, identifying accompanying symptoms, examining the distribution of enlarged lymph nodes, and considering epidemiological factors. A variety of diagnostic tests are described based on the location and suspected causes of lymphadenopathy including blood tests, imaging studies, biopsies and cultures. The goal of evaluation is to arrive at an accurate diagnosis and guide further treatment.
Acute tubular necrosis (ATN) is a type of acute kidney injury where there is severe or prolonged ischemia or exposure to nephrotoxins that injure the tubular epithelium, particularly in the proximal tubule and medullary thick ascending limb. ATN is characterized by tubular cell swelling, vacuolation, necrosis and sloughing of epithelial cells. The injury is caused by mechanisms like ATP depletion, oxidative stress, calcium overload and inflammation. Clinically, ATN presents with decreased urine output and rising creatinine over days. The diagnosis is supported by urine studies showing sodium greater than 40 mmol/L and muddy brown casts. Treatment is supportive with fluid management and treating the underlying cause.
This document discusses childhood Burkitt lymphoma. It begins by describing normal lymphoid tissues and cells. It then notes that Burkitt lymphoma is a highly aggressive B cell lymphoma characterized by a translocation involving the c-MYC gene. There are three clinical forms: endemic (African), sporadic, and immunodeficiency-associated. The endemic form has the highest incidence in Africa and peaks in children ages 4-7 years. Diagnosis involves identifying a rapidly growing tumor with a "starry-sky" appearance under microscopy. Key investigations include blood work, imaging scans, and tissue biopsies.
1. The document provides guidance on evaluating a patient presenting with red or brown colored urine (hematuria). An initial workup includes a urine analysis and microscopy to determine the source and characteristics of the red blood cells.
2. Further evaluation depends on whether the hematuria is glomerular or extraglomerular in origin, which can be suggested by factors like RBC morphology and presence of proteinuria. Potential causes include infections, stones, tumors, and glomerular diseases.
3. If hematuria persists after initial workup, referral to nephrology or urology may be warranted for cystoscopy, imaging tests, or renal biopsy to identify the underlying cause. Thorough history and physical
Neurocysticercosis is a disease caused by the larval form of the pork tapeworm Taenia solium infecting the brain and central nervous system in humans. There are two types of cysts - Cysticercus cellulosae and Cysticercus racemose, which can lodge in different areas and cause different symptoms. Neurocysticercosis has a variety of clinical presentations depending on the location, size, and number of cysts as well as the host's immune response. Treatment approaches for neurocysticercosis are controversial, with some evidence that antiparasitic treatment may cause more harm than benefit compared to simply managing seizures with antiepileptic drugs alone.
Renal colic is a sudden, severe, dull pain that originates in the costovertebral angle and may radiate to the groin or abdomen. It is caused by obstruction of the ureter, usually by a kidney stone. Patients experience intermittent, colicky pain that is exacerbated by movement and relieved briefly by analgesics. Examination may reveal abdominal tenderness over the kidney area. Investigations include urinalysis, kidney imaging tests like ultrasound or CT scan to detect stones. Treatment focuses on pain relief, increasing fluid intake, and allowing stones to pass spontaneously when possible. Surgery is considered for larger stones or if conservative measures fail.
Hydronephrosis is the dilatation of the renal pelvis or calyces, which can be associated with obstruction. It can be unilateral or bilateral. Unilateral causes include extramural obstruction from aberrant vessels or tumors, intramural obstruction from congenital abnormalities or strictures, and intraluminal obstruction from stones. Bilateral causes are usually congenital such as valves or acquired such as prostate enlargement. Hydronephrosis is classified based on unilateral vs bilateral involvement, intermittent vs persistent, presence of hydroureter, and location within or outside the kidney.
The document provides information on disorders of the gallbladder and pancreas. It begins with learning objectives related to cholelithiasis, cholecystitis, pancreatitis, and surgical treatment of pancreatic tumors. Key topics covered include risk factors for cholelithiasis, clinical manifestations, diagnostic findings, medical and surgical management of gallbladder disorders, and types of acute and chronic pancreatitis. Nursing implications are also discussed for various diagnostic and treatment procedures.
The document discusses hematuria (blood in the urine) including its definition, classification, causes, evaluation, and management. It addresses topics such as the difference between glomerular and extraglomerular hematuria, post-streptococcal glomerulonephritis (PSGN), and lupus nephritis. The key points are:
1) Hematuria can be either microscopic or macroscopic and can have many potential causes including infections, tumors, trauma, inflammation, and structural abnormalities.
2) Evaluation of hematuria involves urine analysis, blood tests, imaging, and sometimes renal biopsy to determine the cause and guide treatment.
3) Glomerular hematuria is suggested by
The document provides guidance on performing an abdominal examination including inspection, palpation, percussion, and auscultation. Key steps are outlined for each component of the exam. Inspection involves examining the abdomen visually for shape, movements, skin features etc. Palpation is done systematically to feel for tenderness, masses and enlarged organs. Percussion helps define organ borders and detect ascites. Auscultation listens for bowel sounds and vascular bruits.
This document discusses various types of genitourinary trauma, including renal, bladder, ureteral, and scrotal trauma. It provides details on the mechanisms, classifications, imaging findings, and treatment approaches for each type of injury. Radiology plays an important role in accurately diagnosing and grading genitourinary trauma in order to guide clinical management decisions. CT is often the preferred imaging modality due to its ability to simultaneously evaluate the kidneys, bladder, and other abdominal organs.
Renal colic is a type of intense pain that occurs when a urinary stone becomes lodged in the kidney or ureter. Symptoms include waves of pain in the flank, groin, or lower abdomen, as well as nausea, vomiting, blood in the urine, and difficulty urinating. Risk factors include dehydration, a high-vitamin D diet, family history of kidney stones, and urinary tract infections. Treatment depends on the size of the stone but may include shock wave lithotripsy to break up the stone, ureteroscopy to remove it, or percutaneous nephrolithotomy for large stones. Preventing dehydration and limiting foods high in oxalate can
The document provides information about peritonitis, including:
- Causes of peritonitis including bacterial, chemical, allergic, traumatic, ischemic, and miscellaneous causes.
- Acute bacterial peritonitis can be caused by gastrointestinal perforation, transmural translocation without perforation, exogenous contamination, or hematogenous spread.
- Common microorganisms involved in peritonitis include E. coli, streptococci, bacteroides, clostridium, and klebsiella from gastrointestinal sources, and other microbes from other sources.
- Defense mechanisms against peritonitis include leukocyte-attracting mechanisms, killing mechanisms, sequestration mechanisms
This document discusses intestinal obstruction, including its definition, causes, clinical features, investigations and management. Intestinal obstruction occurs when bowel contents cannot pass through normally due to a mechanical or functional blockage. Clinical features depend on the location and cause of obstruction and may include pain, vomiting, distension and constipation.
Intestinal obstruction is a blockage of the bowel that prevents contents from passing through. There are two main types: mechanical obstruction from pressure on the bowel wall, and functional obstruction where the bowel muscles cannot propel contents. Causes of small bowel obstruction include adhesions, intussusception, volvulus, and tumors. Causes of large bowel obstruction include carcinoma, diverticulitis, and inflammatory bowel disorders. Treatment involves decompressing the bowel, fluid replacement, and usually surgery to relieve the obstruction.
History taking in medicine is one of the challenge all medical students face. This brief guide was prepared based on the teachings of Prof.A.S.B.Wijekoon, Prof.I.Amarasinghe & many senior registrars/registrars met during my career. This explans the basics of what must be included in your history, how to plan your investigations/ treatment in a methodical way.
This document discusses the evaluation and differential diagnosis of dysuria (painful urination) in adults. It defines dysuria and notes that acute cystitis is a common cause. The pathophysiology involves sensory nerves in the bladder being irritated by chemical or inflammatory stimuli. Common causes of dysuria include urinary tract infections (UTIs), vaginitis, prostatitis, and STIs. The history and physical exam aim to determine timing, location, and associated symptoms. Laboratory tests like urinalysis and cultures can help diagnose a UTI as a cause. Cultures are recommended for complicated cases or when symptoms don't improve with initial treatment.
The document discusses acute pancreatitis, outlining its epidemiology, pathophysiology, etiology, clinical presentation, workup, severity scoring systems, treatment, prognosis, and complications. It defines acute pancreatitis as an acute condition presenting with abdominal pain associated with raised blood or urine pancreatic enzymes due to pancreatic inflammation. The document also classifies acute pancreatitis as mild or severe based on the presence of organ failure or local complications.
This document discusses the evaluation of lymphadenopathy. It outlines the key steps in evaluation which include determining the size and characteristics of palpable lymph nodes, identifying accompanying symptoms, examining the distribution of enlarged lymph nodes, and considering epidemiological factors. A variety of diagnostic tests are described based on the location and suspected causes of lymphadenopathy including blood tests, imaging studies, biopsies and cultures. The goal of evaluation is to arrive at an accurate diagnosis and guide further treatment.
Acute tubular necrosis (ATN) is a type of acute kidney injury where there is severe or prolonged ischemia or exposure to nephrotoxins that injure the tubular epithelium, particularly in the proximal tubule and medullary thick ascending limb. ATN is characterized by tubular cell swelling, vacuolation, necrosis and sloughing of epithelial cells. The injury is caused by mechanisms like ATP depletion, oxidative stress, calcium overload and inflammation. Clinically, ATN presents with decreased urine output and rising creatinine over days. The diagnosis is supported by urine studies showing sodium greater than 40 mmol/L and muddy brown casts. Treatment is supportive with fluid management and treating the underlying cause.
This document discusses childhood Burkitt lymphoma. It begins by describing normal lymphoid tissues and cells. It then notes that Burkitt lymphoma is a highly aggressive B cell lymphoma characterized by a translocation involving the c-MYC gene. There are three clinical forms: endemic (African), sporadic, and immunodeficiency-associated. The endemic form has the highest incidence in Africa and peaks in children ages 4-7 years. Diagnosis involves identifying a rapidly growing tumor with a "starry-sky" appearance under microscopy. Key investigations include blood work, imaging scans, and tissue biopsies.
1. The document provides guidance on evaluating a patient presenting with red or brown colored urine (hematuria). An initial workup includes a urine analysis and microscopy to determine the source and characteristics of the red blood cells.
2. Further evaluation depends on whether the hematuria is glomerular or extraglomerular in origin, which can be suggested by factors like RBC morphology and presence of proteinuria. Potential causes include infections, stones, tumors, and glomerular diseases.
3. If hematuria persists after initial workup, referral to nephrology or urology may be warranted for cystoscopy, imaging tests, or renal biopsy to identify the underlying cause. Thorough history and physical
Neurocysticercosis is a disease caused by the larval form of the pork tapeworm Taenia solium infecting the brain and central nervous system in humans. There are two types of cysts - Cysticercus cellulosae and Cysticercus racemose, which can lodge in different areas and cause different symptoms. Neurocysticercosis has a variety of clinical presentations depending on the location, size, and number of cysts as well as the host's immune response. Treatment approaches for neurocysticercosis are controversial, with some evidence that antiparasitic treatment may cause more harm than benefit compared to simply managing seizures with antiepileptic drugs alone.
Renal colic is a sudden, severe, dull pain that originates in the costovertebral angle and may radiate to the groin or abdomen. It is caused by obstruction of the ureter, usually by a kidney stone. Patients experience intermittent, colicky pain that is exacerbated by movement and relieved briefly by analgesics. Examination may reveal abdominal tenderness over the kidney area. Investigations include urinalysis, kidney imaging tests like ultrasound or CT scan to detect stones. Treatment focuses on pain relief, increasing fluid intake, and allowing stones to pass spontaneously when possible. Surgery is considered for larger stones or if conservative measures fail.
Hydronephrosis is the dilatation of the renal pelvis or calyces, which can be associated with obstruction. It can be unilateral or bilateral. Unilateral causes include extramural obstruction from aberrant vessels or tumors, intramural obstruction from congenital abnormalities or strictures, and intraluminal obstruction from stones. Bilateral causes are usually congenital such as valves or acquired such as prostate enlargement. Hydronephrosis is classified based on unilateral vs bilateral involvement, intermittent vs persistent, presence of hydroureter, and location within or outside the kidney.
The document provides information on disorders of the gallbladder and pancreas. It begins with learning objectives related to cholelithiasis, cholecystitis, pancreatitis, and surgical treatment of pancreatic tumors. Key topics covered include risk factors for cholelithiasis, clinical manifestations, diagnostic findings, medical and surgical management of gallbladder disorders, and types of acute and chronic pancreatitis. Nursing implications are also discussed for various diagnostic and treatment procedures.
The document discusses hematuria (blood in the urine) including its definition, classification, causes, evaluation, and management. It addresses topics such as the difference between glomerular and extraglomerular hematuria, post-streptococcal glomerulonephritis (PSGN), and lupus nephritis. The key points are:
1) Hematuria can be either microscopic or macroscopic and can have many potential causes including infections, tumors, trauma, inflammation, and structural abnormalities.
2) Evaluation of hematuria involves urine analysis, blood tests, imaging, and sometimes renal biopsy to determine the cause and guide treatment.
3) Glomerular hematuria is suggested by
The document provides guidance on performing an abdominal examination including inspection, palpation, percussion, and auscultation. Key steps are outlined for each component of the exam. Inspection involves examining the abdomen visually for shape, movements, skin features etc. Palpation is done systematically to feel for tenderness, masses and enlarged organs. Percussion helps define organ borders and detect ascites. Auscultation listens for bowel sounds and vascular bruits.
This document discusses various types of genitourinary trauma, including renal, bladder, ureteral, and scrotal trauma. It provides details on the mechanisms, classifications, imaging findings, and treatment approaches for each type of injury. Radiology plays an important role in accurately diagnosing and grading genitourinary trauma in order to guide clinical management decisions. CT is often the preferred imaging modality due to its ability to simultaneously evaluate the kidneys, bladder, and other abdominal organs.
Renal colic is a type of intense pain that occurs when a urinary stone becomes lodged in the kidney or ureter. Symptoms include waves of pain in the flank, groin, or lower abdomen, as well as nausea, vomiting, blood in the urine, and difficulty urinating. Risk factors include dehydration, a high-vitamin D diet, family history of kidney stones, and urinary tract infections. Treatment depends on the size of the stone but may include shock wave lithotripsy to break up the stone, ureteroscopy to remove it, or percutaneous nephrolithotomy for large stones. Preventing dehydration and limiting foods high in oxalate can
The document provides information about peritonitis, including:
- Causes of peritonitis including bacterial, chemical, allergic, traumatic, ischemic, and miscellaneous causes.
- Acute bacterial peritonitis can be caused by gastrointestinal perforation, transmural translocation without perforation, exogenous contamination, or hematogenous spread.
- Common microorganisms involved in peritonitis include E. coli, streptococci, bacteroides, clostridium, and klebsiella from gastrointestinal sources, and other microbes from other sources.
- Defense mechanisms against peritonitis include leukocyte-attracting mechanisms, killing mechanisms, sequestration mechanisms
This document discusses intestinal obstruction, including its definition, causes, clinical features, investigations and management. Intestinal obstruction occurs when bowel contents cannot pass through normally due to a mechanical or functional blockage. Clinical features depend on the location and cause of obstruction and may include pain, vomiting, distension and constipation.
Intestinal obstruction is a blockage of the bowel that prevents contents from passing through. There are two main types: mechanical obstruction from pressure on the bowel wall, and functional obstruction where the bowel muscles cannot propel contents. Causes of small bowel obstruction include adhesions, intussusception, volvulus, and tumors. Causes of large bowel obstruction include carcinoma, diverticulitis, and inflammatory bowel disorders. Treatment involves decompressing the bowel, fluid replacement, and usually surgery to relieve the obstruction.
History taking in medicine is one of the challenge all medical students face. This brief guide was prepared based on the teachings of Prof.A.S.B.Wijekoon, Prof.I.Amarasinghe & many senior registrars/registrars met during my career. This explans the basics of what must be included in your history, how to plan your investigations/ treatment in a methodical way.
This document discusses the evaluation and differential diagnosis of dysuria (painful urination) in adults. It defines dysuria and notes that acute cystitis is a common cause. The pathophysiology involves sensory nerves in the bladder being irritated by chemical or inflammatory stimuli. Common causes of dysuria include urinary tract infections (UTIs), vaginitis, prostatitis, and STIs. The history and physical exam aim to determine timing, location, and associated symptoms. Laboratory tests like urinalysis and cultures can help diagnose a UTI as a cause. Cultures are recommended for complicated cases or when symptoms don't improve with initial treatment.
The document discusses acute pancreatitis, outlining its epidemiology, pathophysiology, etiology, clinical presentation, workup, severity scoring systems, treatment, prognosis, and complications. It defines acute pancreatitis as an acute condition presenting with abdominal pain associated with raised blood or urine pancreatic enzymes due to pancreatic inflammation. The document also classifies acute pancreatitis as mild or severe based on the presence of organ failure or local complications.
Hamlet is a play written by William Shakespeare between 1599-1601. It explores Hamlet's famous soliloquy about whether it is better to endure life's struggles or take arms against a sea of troubles. The story follows Hamlet's quest to avenge his father's death at the hands of his uncle, who marries Hamlet's mother.
The document appears to be notes from an English language lesson. It includes vocabulary words, sentences to match with pictures, and prompts for activities like making stories from pictures and sentences. Warmup exercises are mentioned along with resources from websites like Wikipedia and YouTube.
1. THE LIONAND THE RABBIT
A cruel Lion lived in the forest. Every day, He killed and ate a lot of animals. The other
animals were afraid the Lion would kill them all.
The animals told the Lion, "Let’s make a deal. If you promise to eat only one animal each
day, then one of us will come to you every day. Then you don’t have to hunt and kill us."
The plan sounded well thought-out to the Lion, so he agreed, but he also said, "If you don’t
come every day, I promise to kill all of you the next day!"
Each day after that, one animal went to the Lion so that the Lion could eat it. Then, all the
other animals were safe.
Finally, it was the Rabbit’s turn to go to the Lion. The Rabbit went very slowly that day, so
the Lion was angry when the Rabbit finally arrived. The Lion angrily asked the Rabbit,
"Why are you late?"
"I was hiding from another Lion in the forest. That Lion said he was the king, so I was
afraid."
The Lion told the Rabbit, "I am the only king here! Take me to that other lion, and I will kill
him."
The Rabbit replied, "I will be happy to show you where he lives."
The Rabbit led the Lion to an old well in the middle of the forest. The well was very deep
with water at the bottom. The Rabbit told the Lion, "Look in there. The lion lives at the
bottom."
When the Lion looked in the well, he could see his own face in the water. He thought that
was the other lion. Without waiting another moment, the Lion jumped into the well to
attack the other lion. He never came out.
All of the other animals in the forest were very pleased with the Rabbit’s clever trick.
2. Лев и Кролик
Жил в лесу безжалостный Лев. Каждый день он убивал и съедал множество лесных
зверей. Остальные лесные звери боялись, что Лев так вскоре и их всех убьёт.
Лесные звери обратились ко Льву: "Давай договоримся. Если ты пообещаешь съедать
только одного из нас каждый день, то один из нас сам будет приходить к тебе каждый
день. Так, тебе не придётся больше охотиться и убивать нас"
Предложение звучало заманчиво. Подумал Лев, и согласился, но поставил условие:
"Если кто-то из вас не явится ко мне хотя бы день, я обещаю, на следующий день я вас
всех убью(переем)!"
После этого каждый день, один зверь приходил ко Льву, чтобы он мог спокойно съесть
его. Но так все остальные звери были в безопасности.
Наконец, настала очередь Кролика идти ко Льву. В назначенный день Кролик шёл ко
Льву очень медленно, поэтому Лев рассердился, когда Кролик наконец пришёл к нему
Лев гневно спросил Кролика: "Почему ты так поздно?"
"Я прятался от другого Льва, что живёт в этом же лесу. Этот Лев сказал, что будто бы он
король, я очень испугался"
Лев сказал Кролику: "Здесь только Я король! Веди меня к этому льву, я убью его."
На что Кролик ответил: "Я буду счастлив, показать тебе, где он живёт"
Кролик привёл Льва к старому колодцу, что стоял посреди леса. Колодец был очень
глубоким и наполнен водой в самой нижней его части. Кролик сказал Льву: "Загляни
внутрь. Тот лев живёт в самом низу этого колодца".
Когда Лев заглянул в колодец, он увидел в воде своё отражение. Он подумал, что это
был другой лев. С яростным рёвом Лев прыгнул в колодец, чтобы атаковать другого
льва. Так он там навсегда и остался. Так мудрый кролик спас лесных жителей от
тирана.