Tips on using my ppt.
1. You can freely download, edit, modify and put your
name etc.
2. Don’t be concerned about number of slides. Half the
slides are blanks except for the title.
3. First show the blank slides (eg. Aetiology ) > Ask
students what they already know about ethology of
today's topic. > Then show next slide which enumerates
aetiologies.
4. At the end rerun the show – show blank> ask questions >
show next slide.
5. This will be an ACTIVE LEARNING SESSION x
three revisions.
6. Good for self study also.
7. See notes for bibliography.
Format
Format
1. Introduction & History
2. Relevant Anatomy, Physiology
3. Aetiology
4. Pathophysiology
5. Pathology
6. Classification
7. Clinical Features
8. Investigations
9. Management
10.Prevention
Introduction & History.
Introduction & History.
• Rectal prolapse occurs when a mucosal or
full-thickness layer of rectal tissue protrudes
through the anal orifice.
• Three different clinical entities are often
combined under the umbrella term rectal
prolapse:
1. Full-thickness rectal prolapse
2. Mucosal prolapse
3. Internal prolapse (internal intussusception)
Introduction & History.
• Full-thickness rectal is defined as protrusion of
the full thickness of the rectal wall through the
anus; it is the most commonly recognized type.
• Mucosal prolapse, in contrast, is defined as
protrusion of only the rectal mucosa (not the entire
wall) from the anus.
• Internal intussusception may be a full-thickness
or a partial rectal wall disorder,
• Today we shall focus on full-thickness rectal
prolapse, which will be referred to as rectal
prolapse.
Relevant Anatomy
Relevant Anatomy
Common anatomic features-
• Patulous or weak anal sphincter
• Levator diastasis,
• deep anterior Douglas cul-de-sac,
• poor posterior rectal fixation with a long
rectal mesentery,
• Redundant rectosigmoid.
Whether these anatomic features are the cause
or result of the prolapsing rectum is not
known.
Relevant Anatomy
Common anatomic features in children-
• vertical orientation of the rectum,
• the mobility of the sigmoid colon,
• the relative weakness of the pelvic floor
muscle
• mucosa that is poorly fixed to submucosa
• Redundant rectal mucosa..
Aetiology
• Idiopathic
• Congenital/ Genetic
• Nutritional Deficiency/excess
• Traumatic
• Infections /Infestation
• Autoimmune
• Neoplastic (Benign/Malignant)
• Degenerative
• Iatrogenic
• Psychosomatic
Associated conditions-
• chronic straining with
defecation and
constipation.
• Pregnancy
• Previous surgery
• Diarrhea
• Benign prostatic
hypertrophy
• Chronic obstructive
pulmonary disease
(COPD)
•
• Cystic fibrosis
• Pertussis (ie,
whooping cough)
• Pelvic floor
dysfunction
• Parasitic infections –
Amebiasis,
schistosomiasis
• Disordered defecation
(eg, stool withholding)
• Neurologic disorders -
Associated conditions-
• Neurologic disorders - Previous lower
backor pelvic trauma/lumbar disk disease,
cauda equina syndrome, spinal tumors,
multiple sclerosis
Pathophysiology
Pathophysiology
• The pathophysiology of rectal prolapse is not
completely understood or agreed upon.
• The first theory postulates that rectal prolapse is a
sliding hernia through a defect in the pelvic fascia.
• The second theory holds that rectal prolapse starts
as a circumferential internal intussusception of the
rectum beginning 6-8 cm proximal to the anal
verge.
• Often, prolapse begins with an internal prolapse of
the anterior rectal wall and progresses to full
prolapse.
Clinical Features
Clinical Features
• Demography
• Symptoms
• Signs
• Prognosis
• Complications
Demography
Demography
• The annual incidence 2.5 per 100,000
• peak incidences are observed in the fourth
and seventh decades of life.
• Pediatric patients usually are affected when
younger than 3 years with the peak
incidence in the first year of life.
• The incidence of prolapsed rectum in
children with cystic fibrosis approaches
20%.
Demography
• In the adult population, the male-to-female
ratio is 1:6.
• pediatric population: equal
Symptoms
Symptoms
• Mass protruding through the anus
• Pain
• Some 10-25% of patients also have uterine
or bladder prolapse,
• and 35% may have an associated cystocele.
• Constipation occurs in 15-65% of cases.
• There may also be rectal bleeding.
• fecal incontinence
Symptoms
• mass protruding through the anus
• Initially only after a bowel movement and
usually retracts when the patient stands up.
• As the disease process progresses, the mass
protrudes more often, especially with
straining and Valsalva maneuvers such as
sneezing or coughing.
• Finally, the rectum prolapses with daily
activities such as walking and may progress
to continual prolapse.
Symptoms
• As the disease progresses, the rectum no
longer spontaneously retracts, and patients
may have to replace it manually.
• may then progress to a point where the
rectum prolapses immediately after being
replaced and is continuously prolapsed.
• Rarely, the rectum becomes incarcerated,
and patients cannot replace the rectum.
Signs
Signs
• Protruding rectal mucosa
• Thick concentric mucosal ring
• Sulcus noted between anal canal and rectum
• Solitary rectal ulcer (10-25%)
• Decreased anal sphincter tone
• The patient is asked to sit on a toilet and
strain, after which the rectum should
prolapse.
• phosphate enema glycerin suppository
Signs
• The protruding mass should show
concentric rings of mucosa.
• Mucosal prolapse typically exhibits radial
folds instead of concentric rings.
Prognosis
Prognosis
• The prognosis generally is good with
appropriate treatment.
• Spontaneous resolution usually occurs in
children.
Complications
Complications
Uncommon
• incarceration and strangulation (rare).
• Bleeding
• Ulceration
• Incontinence
Investigations
Investigations
• Laboratory Studies
– Routine
– Special
• Imaging Studies
• Tissue diagnosis
– Cytology
• FNAC
– Histology
Investigations
• Laboratory Studies
– sweat chloride test for pediatric patients; as
many as 11% of children with rectal prolapse
have cystic fibrosis.
Diagnostic Studies
Diagnostic Studies
Imaging Studies
• X-Ray
• USG
• CT
• Angiography
• MRI
• Endoscopy
• Nuclear scan
Diagnostic Studies
Imaging Studies
• Rigid proctosigmoidoscopy- solitary rectal
ulcers.
• Colonoscopy
• Barium enema
• Video defecography
– internal prolapse or to distinguish rectal
prolapse from mucosal prolapse
– intussusception of proximal colon
– pelvic outlet obstruction.
Diagnostic Studies
Other Tests
• Anal-rectal manometry
• The Sitz marker study
• Pudendal nerve terminal motor latency
Differential Diagnosis
Differential Diagnosis
• Hemorrhoids
• Intussusception
• Proctitis
• Rectal polyps
• Evaluate pediatric patients for cystic
fibrosis
Management
Management
• In adult patients, treatment of rectal
prolapse is essentially surgical;
• Children, however, can usually be treated
nonsurgically.
Non Operative Therapy
Non Operative Therapy
• Reduce with gentle digital pressure
sedation,
• Field block with local anesthetic
• Sprinkling the prolapse with either salt or
sugar to decrease the edema.
• Manage constipation and diarrhea
Emergency Surgery
Emergency Surgery
• If the prolapse cannot be reduced and the
viability of the bowel is in question
• Rupture of the rectum
Operative Therapy
•
Operative Therapy
• Surgical treatments can be divided into two
categories-
1. Abdominal procedures
2. Perineal procedures.
• The abdominal procedures have a lower
recurrence rate but a higher morbidity.
• Older, debilitated patients -perineal
procedures
• Younger, healthier patients -abdominal
procedures.
Operative Therapy
Abdominal procedures
• Anterior resection
• Ripstein procedure Marlex rectopexy-
Marlex mesh or an Ivalon sponge.
• Suture rectopexy
• Laparoscopic surgical rectopexy
• Frykman-Goldberg procedure- resection
with rectopexy
Operative Therapy
Perineal procedures
• Anal encirclement Thiersch wire
• Delorme mucosal sleeve resection
• Altemeier perineal rectosigmoidectomy
• Hemorrhoidectomy-Mucosal prolapse
• Perineal stapled prolapse resection
Minimally invasive Therapy
• A laparoscopic approach to rectal prolapse
repair
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Rectal prolapse.pptx

  • 1.
    Tips on usingmy ppt. 1. You can freely download, edit, modify and put your name etc. 2. Don’t be concerned about number of slides. Half the slides are blanks except for the title. 3. First show the blank slides (eg. Aetiology ) > Ask students what they already know about ethology of today's topic. > Then show next slide which enumerates aetiologies. 4. At the end rerun the show – show blank> ask questions > show next slide. 5. This will be an ACTIVE LEARNING SESSION x three revisions. 6. Good for self study also. 7. See notes for bibliography.
  • 2.
  • 3.
    Format 1. Introduction &History 2. Relevant Anatomy, Physiology 3. Aetiology 4. Pathophysiology 5. Pathology 6. Classification 7. Clinical Features 8. Investigations 9. Management 10.Prevention
  • 4.
  • 5.
    Introduction & History. •Rectal prolapse occurs when a mucosal or full-thickness layer of rectal tissue protrudes through the anal orifice. • Three different clinical entities are often combined under the umbrella term rectal prolapse: 1. Full-thickness rectal prolapse 2. Mucosal prolapse 3. Internal prolapse (internal intussusception)
  • 6.
    Introduction & History. •Full-thickness rectal is defined as protrusion of the full thickness of the rectal wall through the anus; it is the most commonly recognized type. • Mucosal prolapse, in contrast, is defined as protrusion of only the rectal mucosa (not the entire wall) from the anus. • Internal intussusception may be a full-thickness or a partial rectal wall disorder, • Today we shall focus on full-thickness rectal prolapse, which will be referred to as rectal prolapse.
  • 9.
  • 10.
    Relevant Anatomy Common anatomicfeatures- • Patulous or weak anal sphincter • Levator diastasis, • deep anterior Douglas cul-de-sac, • poor posterior rectal fixation with a long rectal mesentery, • Redundant rectosigmoid. Whether these anatomic features are the cause or result of the prolapsing rectum is not known.
  • 11.
    Relevant Anatomy Common anatomicfeatures in children- • vertical orientation of the rectum, • the mobility of the sigmoid colon, • the relative weakness of the pelvic floor muscle • mucosa that is poorly fixed to submucosa • Redundant rectal mucosa..
  • 12.
    Aetiology • Idiopathic • Congenital/Genetic • Nutritional Deficiency/excess • Traumatic • Infections /Infestation • Autoimmune • Neoplastic (Benign/Malignant) • Degenerative • Iatrogenic • Psychosomatic
  • 13.
    Associated conditions- • chronicstraining with defecation and constipation. • Pregnancy • Previous surgery • Diarrhea • Benign prostatic hypertrophy • Chronic obstructive pulmonary disease (COPD) • • Cystic fibrosis • Pertussis (ie, whooping cough) • Pelvic floor dysfunction • Parasitic infections – Amebiasis, schistosomiasis • Disordered defecation (eg, stool withholding) • Neurologic disorders -
  • 14.
    Associated conditions- • Neurologicdisorders - Previous lower backor pelvic trauma/lumbar disk disease, cauda equina syndrome, spinal tumors, multiple sclerosis
  • 15.
  • 16.
    Pathophysiology • The pathophysiologyof rectal prolapse is not completely understood or agreed upon. • The first theory postulates that rectal prolapse is a sliding hernia through a defect in the pelvic fascia. • The second theory holds that rectal prolapse starts as a circumferential internal intussusception of the rectum beginning 6-8 cm proximal to the anal verge. • Often, prolapse begins with an internal prolapse of the anterior rectal wall and progresses to full prolapse.
  • 17.
  • 18.
    Clinical Features • Demography •Symptoms • Signs • Prognosis • Complications
  • 19.
  • 20.
    Demography • The annualincidence 2.5 per 100,000 • peak incidences are observed in the fourth and seventh decades of life. • Pediatric patients usually are affected when younger than 3 years with the peak incidence in the first year of life. • The incidence of prolapsed rectum in children with cystic fibrosis approaches 20%.
  • 21.
    Demography • In theadult population, the male-to-female ratio is 1:6. • pediatric population: equal
  • 22.
  • 23.
    Symptoms • Mass protrudingthrough the anus • Pain • Some 10-25% of patients also have uterine or bladder prolapse, • and 35% may have an associated cystocele. • Constipation occurs in 15-65% of cases. • There may also be rectal bleeding. • fecal incontinence
  • 24.
    Symptoms • mass protrudingthrough the anus • Initially only after a bowel movement and usually retracts when the patient stands up. • As the disease process progresses, the mass protrudes more often, especially with straining and Valsalva maneuvers such as sneezing or coughing. • Finally, the rectum prolapses with daily activities such as walking and may progress to continual prolapse.
  • 25.
    Symptoms • As thedisease progresses, the rectum no longer spontaneously retracts, and patients may have to replace it manually. • may then progress to a point where the rectum prolapses immediately after being replaced and is continuously prolapsed. • Rarely, the rectum becomes incarcerated, and patients cannot replace the rectum.
  • 26.
  • 27.
    Signs • Protruding rectalmucosa • Thick concentric mucosal ring • Sulcus noted between anal canal and rectum • Solitary rectal ulcer (10-25%) • Decreased anal sphincter tone • The patient is asked to sit on a toilet and strain, after which the rectum should prolapse. • phosphate enema glycerin suppository
  • 28.
    Signs • The protrudingmass should show concentric rings of mucosa. • Mucosal prolapse typically exhibits radial folds instead of concentric rings.
  • 29.
  • 30.
    Prognosis • The prognosisgenerally is good with appropriate treatment. • Spontaneous resolution usually occurs in children.
  • 31.
  • 32.
    Complications Uncommon • incarceration andstrangulation (rare). • Bleeding • Ulceration • Incontinence
  • 33.
  • 34.
    Investigations • Laboratory Studies –Routine – Special • Imaging Studies • Tissue diagnosis – Cytology • FNAC – Histology
  • 35.
    Investigations • Laboratory Studies –sweat chloride test for pediatric patients; as many as 11% of children with rectal prolapse have cystic fibrosis.
  • 36.
  • 37.
    Diagnostic Studies Imaging Studies •X-Ray • USG • CT • Angiography • MRI • Endoscopy • Nuclear scan
  • 38.
    Diagnostic Studies Imaging Studies •Rigid proctosigmoidoscopy- solitary rectal ulcers. • Colonoscopy • Barium enema • Video defecography – internal prolapse or to distinguish rectal prolapse from mucosal prolapse – intussusception of proximal colon – pelvic outlet obstruction.
  • 39.
    Diagnostic Studies Other Tests •Anal-rectal manometry • The Sitz marker study • Pudendal nerve terminal motor latency
  • 40.
  • 41.
    Differential Diagnosis • Hemorrhoids •Intussusception • Proctitis • Rectal polyps • Evaluate pediatric patients for cystic fibrosis
  • 42.
  • 43.
    Management • In adultpatients, treatment of rectal prolapse is essentially surgical; • Children, however, can usually be treated nonsurgically.
  • 44.
  • 45.
    Non Operative Therapy •Reduce with gentle digital pressure sedation, • Field block with local anesthetic • Sprinkling the prolapse with either salt or sugar to decrease the edema. • Manage constipation and diarrhea
  • 46.
  • 47.
    Emergency Surgery • Ifthe prolapse cannot be reduced and the viability of the bowel is in question • Rupture of the rectum
  • 48.
  • 49.
    Operative Therapy • Surgicaltreatments can be divided into two categories- 1. Abdominal procedures 2. Perineal procedures. • The abdominal procedures have a lower recurrence rate but a higher morbidity. • Older, debilitated patients -perineal procedures • Younger, healthier patients -abdominal procedures.
  • 50.
    Operative Therapy Abdominal procedures •Anterior resection • Ripstein procedure Marlex rectopexy- Marlex mesh or an Ivalon sponge. • Suture rectopexy • Laparoscopic surgical rectopexy • Frykman-Goldberg procedure- resection with rectopexy
  • 51.
    Operative Therapy Perineal procedures •Anal encirclement Thiersch wire • Delorme mucosal sleeve resection • Altemeier perineal rectosigmoidectomy • Hemorrhoidectomy-Mucosal prolapse • Perineal stapled prolapse resection
  • 52.
    Minimally invasive Therapy •A laparoscopic approach to rectal prolapse repair
  • 53.
    Get this pptin mobile 1. Download Microsoft PowerPoint from play store. 2. Open Google assistant 3. Open Google lens. 4. Scan qr code from next slide.
  • 56.
    Get my pptcollection • https://www.slideshare.net/drpradeeppande/ edit_my_uploads • https://www.dropbox.com/sh/x600md3cvj8 5woy/AACVMHuQtvHvl_K8ehc3ltkEa?dl =0 • https://www.facebook.com/doctorpradeeppa nde/?ref=pages_you_manage

Editor's Notes

  • #3 https://emedicine.medscape.com/article/2026460-clinical#b3
  • #4 https://emedicine.medscape.com/article/2026460-clinical#b3
  • #56 drpradeeppande@gmail.com 7697305442