Radiographs play an important role in the diagnosis and treatment of periodontal diseases. They provide important information regarding the anatomical structures and periodontal bone loss.
RADIOGRAPHIC AIDS IN THE DIAGNOSIS OF PERIODONTAL DISEASERupal Patle
The document discusses the radiographic evaluation of periodontal disease. It begins by stating that radiographs are useful for diagnosis, prognosis, and treatment evaluation but are an adjunct to clinical examination. Radiographs reveal changes to calcified tissues from past cellular activity but not current activity. Interdental septa and the lamina dura normally appear as thin radiopaque borders and variations in technique can distort radiographic findings. Early signs of periodontal disease on radiographs include fuzziness or breaks in the lamina dura continuity. Progressive bone destruction appears as wedge-shaped radiolucencies and reduced crest height. Furcation involvement and abscesses may also be visualized but radiographs have limitations. Clinical probing with radiopa
advanced diagnostic aids in periodonticsMehul Shinde
Advanced diagnostic aids provide more precise tools and technologies for diagnosis. New probes allow for controlled pressure and automated measurement. Digital radiography provides advantages like reduced radiation dose and immediate imaging. Techniques like digital subtraction radiography and cone-beam computed tomography improve detection of bone changes over time. Overall, advances in clinical, radiographic, microbiological and host-response assessments enhance diagnosis of disease presence, type and progression.
Furcation involvement is a common sequela of severe chronic periodontal disease. Its effective management has a profound influence on the outcome of periodontal therapy.
This document discusses furcation involvement in multi-rooted teeth. It begins with definitions of anatomical terms related to furcations. It then discusses various classifications of furcation involvement, ranging from initial/incipient involvement to more advanced through-and-through defects. Epidemiology, etiology, diagnosis and factors affecting treatment outcomes are also covered. The document concludes with an overview of management approaches, which include maintaining the furcation, increasing access, removing the furcation, or closing it with new attachment.
4.furcation involvement and its treatmentpunitnaidu07
This document discusses furcation involvement in multi-rooted teeth. It begins with introductions and definitions, then describes the anatomy of furcated teeth. Several classifications of furcation involvement are presented based on horizontal and vertical bone loss. Potential etiologies include dental plaque, local anatomic factors like furcation dimensions and root concavities, developmental anomalies, trauma, caries, and pulpal pathology. Diagnosis and various treatment options are also covered, along with prognostic factors and conclusions.
Refractory periodontitis refers to chronic periodontal disease that responds poorly to conventional treatment such as scaling and root planing. About 10-15% of patients have refractory periodontitis. Several studies examined clinical, microbiological, and immunological parameters to better diagnose and treat refractory periodontitis. One study found that levels of certain bacterial species, percentage of sites with deep pockets, and number of bacterial species with high antibody levels could predict refractory cases. Another study found elevated antibody levels to specific bacteria correlated with refractory cases. Molecular studies identified higher expression of certain genes involved in inflammation and bone resorption in refractory patients. Microarray analysis found refractory patients had persistent pathogenic bacteria after treatment. Combin
Phase I periodontal therapy is the first in the chronologic sequence of procedures that constitute periodontal treatment. It is also referred to as cause related therapy or non-surgical periodontal therapy.
RADIOGRAPHIC AIDS IN THE DIAGNOSIS OF PERIODONTAL DISEASERupal Patle
The document discusses the radiographic evaluation of periodontal disease. It begins by stating that radiographs are useful for diagnosis, prognosis, and treatment evaluation but are an adjunct to clinical examination. Radiographs reveal changes to calcified tissues from past cellular activity but not current activity. Interdental septa and the lamina dura normally appear as thin radiopaque borders and variations in technique can distort radiographic findings. Early signs of periodontal disease on radiographs include fuzziness or breaks in the lamina dura continuity. Progressive bone destruction appears as wedge-shaped radiolucencies and reduced crest height. Furcation involvement and abscesses may also be visualized but radiographs have limitations. Clinical probing with radiopa
advanced diagnostic aids in periodonticsMehul Shinde
Advanced diagnostic aids provide more precise tools and technologies for diagnosis. New probes allow for controlled pressure and automated measurement. Digital radiography provides advantages like reduced radiation dose and immediate imaging. Techniques like digital subtraction radiography and cone-beam computed tomography improve detection of bone changes over time. Overall, advances in clinical, radiographic, microbiological and host-response assessments enhance diagnosis of disease presence, type and progression.
Furcation involvement is a common sequela of severe chronic periodontal disease. Its effective management has a profound influence on the outcome of periodontal therapy.
This document discusses furcation involvement in multi-rooted teeth. It begins with definitions of anatomical terms related to furcations. It then discusses various classifications of furcation involvement, ranging from initial/incipient involvement to more advanced through-and-through defects. Epidemiology, etiology, diagnosis and factors affecting treatment outcomes are also covered. The document concludes with an overview of management approaches, which include maintaining the furcation, increasing access, removing the furcation, or closing it with new attachment.
4.furcation involvement and its treatmentpunitnaidu07
This document discusses furcation involvement in multi-rooted teeth. It begins with introductions and definitions, then describes the anatomy of furcated teeth. Several classifications of furcation involvement are presented based on horizontal and vertical bone loss. Potential etiologies include dental plaque, local anatomic factors like furcation dimensions and root concavities, developmental anomalies, trauma, caries, and pulpal pathology. Diagnosis and various treatment options are also covered, along with prognostic factors and conclusions.
Refractory periodontitis refers to chronic periodontal disease that responds poorly to conventional treatment such as scaling and root planing. About 10-15% of patients have refractory periodontitis. Several studies examined clinical, microbiological, and immunological parameters to better diagnose and treat refractory periodontitis. One study found that levels of certain bacterial species, percentage of sites with deep pockets, and number of bacterial species with high antibody levels could predict refractory cases. Another study found elevated antibody levels to specific bacteria correlated with refractory cases. Molecular studies identified higher expression of certain genes involved in inflammation and bone resorption in refractory patients. Microarray analysis found refractory patients had persistent pathogenic bacteria after treatment. Combin
Phase I periodontal therapy is the first in the chronologic sequence of procedures that constitute periodontal treatment. It is also referred to as cause related therapy or non-surgical periodontal therapy.
This document discusses periodontal regeneration and the various factors involved. It begins by defining key terminology related to grafting and regeneration. It then discusses the biology and objectives of periodontal regeneration, including the ideal outcome of new attachment formation and factors that can influence outcomes. The document outlines various techniques for periodontal regeneration including non-graft associated approaches involving removal of epithelium and surgical techniques, as well as graft-associated approaches using various graft materials. Requirements for predictable regeneration and assessment methods are also summarized.
The document discusses gingival curettage, which involves using instruments to remove diseased soft tissue from periodontal pockets. It defines curettage and provides the history of the procedure. The basic technique is described as using curettes to scrape the inner lining of the pocket to remove ulcerated epithelium and damaged connective tissue. Indications include edematous pockets aiming to reduce inflammation and shrink tissue. Healing after curettage is examined through a study showing revascularization of the wound site over time.
This document discusses gingival recession, including its definitions, classifications, etiology, factors affecting treatment outcomes, and treatments. It provides an overview of several classification systems for gingival recession, including those proposed by Sullivan and Atkins, Miller, Mahajan, Cairo, and Ashish Kumar. Miller's classification is the most widely used but has limitations, so modifications have been suggested. The document also proposes a new comprehensive classification system that aims to address the limitations of previous systems.
Periodontal medicine is the study of the relationship between periodontal health and systemic health. Periodontal disease can influence systemic health through direct effects of bacteria or indirect host-mediated inflammatory responses. Periodontitis has been linked to increased risk of cardiovascular disease, diabetes, and preterm low birth weight. Treatment of periodontal infection may help improve glycemic control in diabetic patients and reduce systemic inflammation.
This document provides an overview of guided tissue regeneration (GTR). It begins with definitions of periodontal regeneration and GTR. It then discusses the history and development of GTR from the 1970s onwards. The core concept of GTR is explained, which is based on Melcher's hypothesis that only periodontal ligament cells can regenerate the periodontal attachment apparatus. Indications, contraindications, design criteria and objectives of GTR barriers are covered. The document classifies and compares advantages and disadvantages of absorbable versus non-absorbable membranes. Key factors affecting GTR outcomes are discussed. Surgical techniques and the healing of GTR-treated defects are described. The document concludes with additional considerations like complications and the
Emdogain is a gel containing enamel matrix proteins that has been shown to regenerate hard and soft tissues lost to periodontal disease. It works by attracting mesenchymal cells to the root surface, promoting attachment, proliferation, and differentiation which results in new cementum, bone, and periodontal ligament formation. Over 20 years of clinical studies involving over 2 million patients have demonstrated its effectiveness and safety in treating intra-bony and gingival recession defects.
Dentists play an important role in the diagnosis and management of desquamative gingivitis. The importance of being able to recognise and properly diagnose this condition is accentuated by the fact that a serious and life threatening disease may initially manifest as desquamative gingivitis.
Certains medications have been associated with gingival enlargement.
the seminar gives a complete analysis of etilogy and pathogenesis involved in digo as well as sequlae of it
This document summarizes the classification of osseous defects caused by periodontal disease. It describes different types of horizontal bone loss including osseous craters and bulbous bony contours. It also discusses vertical/angular bone loss and classifications proposed by Glickman and Goldman/Cohen. Furcation involvement is classified using scales proposed by Glickman and Tarnow/Fletcher. Understanding the nature of these bone alterations is important for effective diagnosis and treatment planning.
Periodontitis is a complex infection initiated by bacteria –tissue destruction.
Host: the organism from which a parasite obtains its nourishment/ an individual who receives a graft
Modulation: the alteration of function or status of something in response to a stimulus or an altered physical or chemical environment
1. Gingival recession is the exposure of root surface caused by an apical shift in gingival position. It can be classified as visible, hidden, localized, or generalized.
2. Miller and Atkin & Sullivan classified gingival recession defects based on their location and amount of bone loss. Common causes of recession include age, faulty brushing technique, tooth malposition, gingival inflammation, abnormal frenal attachment, and masochistic habits.
3. Recession can be treated non-surgically through modifying risks or surgically through pedicle or free soft tissue grafts to cover exposed root surfaces and reduce sensitivity.
This document defines and outlines common iatrogenic (treatment-caused) factors that can contribute to periodontal disease. It discusses 10 main factors: overhanging or subgingival restoration margins, poor restoration contours, materials and procedures, partial denture design, malocclusion, orthodontic therapy, impacted tooth extractions, habits like toothbrushing, chemical injuries, radiation therapy, and laser use complications. Each factor is described in terms of how it can disrupt plaque control and the periodontal environment, leading to issues like gingivitis, recession, and bone loss. Prevention methods are also outlined.
This document discusses different types of necrotizing ulcerative periodontitis including non-AIDS type and AIDS-associated type. It also discusses refractory periodontitis caused by abnormal host response, resistant bacteria, failure to remove plaque, and smoking. Microbial complexes associated with refractory periodontitis include Porphyromonas gingivalis, Treponema denticola, and Tannerella forsythia. Treatment involves antimicrobial therapy and local drug delivery. The document also discusses periodontitis caused by systemic diseases that impair neutrophil function such as Papillon-Lefèvre syndrome, Chédiak-Higashi syndrome, and Down syndrome.
Bone loss and patterns of bone destructionvidushiKhanna1
- introduction
- bone resorption
- factors causing bone destruction in periodontal disease
-- destruction by extension of gingival inflammation
--- histopathology
--- pathways of spread of inflammation
--- radius of action
--- periods of destruction
---- mechanism of destruction
-- bone destruction caused by TFO
-- bone destruction caused by systemic disorders
- factors determining bone morphology in periodontal disease
-- normal variation of alveolar bone
-- exostosis
-- butressing bone formation
-- food impaction
-- agressive periodontitis
- patterns of bone destruction
-- horizontal bone loss
-- vertical or angular defects
-- osseous craters
-- bulbous bone contours
-- reversed architecture
-- ledges
- furcation involvement
-- classification
-conclusion
- Trauma from occlusion occurs when occlusal forces exceed the adaptive capacity of the periodontium, causing injury. It can be acute or chronic.
- The magnitude, direction, duration, and frequency of forces impact the periodontium's ability to adapt. Excessive pressure or tension can damage tissues.
- Primary trauma from occlusion is caused by changes in occlusal forces, while secondary trauma occurs when reduced bone support impairs the tissues' resistance to normal forces.
- The periodontium responds to trauma in three stages - injury, repair through new tissue formation, and adaptive remodeling to better withstand forces. Trauma can cause reversible damage if forces are reduced, or lead to irreversible injury if
This document discusses periodontal response to external forces on teeth. It defines trauma from occlusion as injury to tissues from forces exceeding their adaptive capacity. Trauma can be acute or chronic, and primary (normal support) or secondary (reduced support). It discusses classifications and concepts around how gingival inflammation spreads to bone. Glickman's concept describes zones of irritation and co-destruction that are differentially affected. Waerhaug's concept is that bone loss is due exclusively to plaque inflammation. The stages of tissue response to increased forces are injury, repair, and adaptive remodeling.
Radiographic aids in the diagnosis of periodontal diseaseDara Ghaznavi
This document discusses how radiographs can aid in the diagnosis of periodontal disease. Radiographs reveal changes to calcified tissues but not current cellular activity. Normal interdental septa appear as thin radiopaque lines along the alveolar crest. Early periodontal disease leads to disruption of the lamina dura and crestal cortication. As the disease progresses, bone loss appears as wedge-shaped radiolucencies and reduced height of the interdental bone. Furcation involvement and periodontal abscesses can also be detected radiographically, though clinical examination is still needed.
This document discusses the use of radiographs in diagnosing periodontal disease. It begins by stating that radiographs are a valuable diagnostic tool but not a substitute for clinical examination. It then covers topics like the appearance of normal bone, different radiographic techniques, how bone destruction appears in periodontal disease, and advanced imaging modalities like CBCT. Digital radiography is also discussed as it allows for enhancement and sharing of images.
This document discusses periodontal regeneration and the various factors involved. It begins by defining key terminology related to grafting and regeneration. It then discusses the biology and objectives of periodontal regeneration, including the ideal outcome of new attachment formation and factors that can influence outcomes. The document outlines various techniques for periodontal regeneration including non-graft associated approaches involving removal of epithelium and surgical techniques, as well as graft-associated approaches using various graft materials. Requirements for predictable regeneration and assessment methods are also summarized.
The document discusses gingival curettage, which involves using instruments to remove diseased soft tissue from periodontal pockets. It defines curettage and provides the history of the procedure. The basic technique is described as using curettes to scrape the inner lining of the pocket to remove ulcerated epithelium and damaged connective tissue. Indications include edematous pockets aiming to reduce inflammation and shrink tissue. Healing after curettage is examined through a study showing revascularization of the wound site over time.
This document discusses gingival recession, including its definitions, classifications, etiology, factors affecting treatment outcomes, and treatments. It provides an overview of several classification systems for gingival recession, including those proposed by Sullivan and Atkins, Miller, Mahajan, Cairo, and Ashish Kumar. Miller's classification is the most widely used but has limitations, so modifications have been suggested. The document also proposes a new comprehensive classification system that aims to address the limitations of previous systems.
Periodontal medicine is the study of the relationship between periodontal health and systemic health. Periodontal disease can influence systemic health through direct effects of bacteria or indirect host-mediated inflammatory responses. Periodontitis has been linked to increased risk of cardiovascular disease, diabetes, and preterm low birth weight. Treatment of periodontal infection may help improve glycemic control in diabetic patients and reduce systemic inflammation.
This document provides an overview of guided tissue regeneration (GTR). It begins with definitions of periodontal regeneration and GTR. It then discusses the history and development of GTR from the 1970s onwards. The core concept of GTR is explained, which is based on Melcher's hypothesis that only periodontal ligament cells can regenerate the periodontal attachment apparatus. Indications, contraindications, design criteria and objectives of GTR barriers are covered. The document classifies and compares advantages and disadvantages of absorbable versus non-absorbable membranes. Key factors affecting GTR outcomes are discussed. Surgical techniques and the healing of GTR-treated defects are described. The document concludes with additional considerations like complications and the
Emdogain is a gel containing enamel matrix proteins that has been shown to regenerate hard and soft tissues lost to periodontal disease. It works by attracting mesenchymal cells to the root surface, promoting attachment, proliferation, and differentiation which results in new cementum, bone, and periodontal ligament formation. Over 20 years of clinical studies involving over 2 million patients have demonstrated its effectiveness and safety in treating intra-bony and gingival recession defects.
Dentists play an important role in the diagnosis and management of desquamative gingivitis. The importance of being able to recognise and properly diagnose this condition is accentuated by the fact that a serious and life threatening disease may initially manifest as desquamative gingivitis.
Certains medications have been associated with gingival enlargement.
the seminar gives a complete analysis of etilogy and pathogenesis involved in digo as well as sequlae of it
This document summarizes the classification of osseous defects caused by periodontal disease. It describes different types of horizontal bone loss including osseous craters and bulbous bony contours. It also discusses vertical/angular bone loss and classifications proposed by Glickman and Goldman/Cohen. Furcation involvement is classified using scales proposed by Glickman and Tarnow/Fletcher. Understanding the nature of these bone alterations is important for effective diagnosis and treatment planning.
Periodontitis is a complex infection initiated by bacteria –tissue destruction.
Host: the organism from which a parasite obtains its nourishment/ an individual who receives a graft
Modulation: the alteration of function or status of something in response to a stimulus or an altered physical or chemical environment
1. Gingival recession is the exposure of root surface caused by an apical shift in gingival position. It can be classified as visible, hidden, localized, or generalized.
2. Miller and Atkin & Sullivan classified gingival recession defects based on their location and amount of bone loss. Common causes of recession include age, faulty brushing technique, tooth malposition, gingival inflammation, abnormal frenal attachment, and masochistic habits.
3. Recession can be treated non-surgically through modifying risks or surgically through pedicle or free soft tissue grafts to cover exposed root surfaces and reduce sensitivity.
This document defines and outlines common iatrogenic (treatment-caused) factors that can contribute to periodontal disease. It discusses 10 main factors: overhanging or subgingival restoration margins, poor restoration contours, materials and procedures, partial denture design, malocclusion, orthodontic therapy, impacted tooth extractions, habits like toothbrushing, chemical injuries, radiation therapy, and laser use complications. Each factor is described in terms of how it can disrupt plaque control and the periodontal environment, leading to issues like gingivitis, recession, and bone loss. Prevention methods are also outlined.
This document discusses different types of necrotizing ulcerative periodontitis including non-AIDS type and AIDS-associated type. It also discusses refractory periodontitis caused by abnormal host response, resistant bacteria, failure to remove plaque, and smoking. Microbial complexes associated with refractory periodontitis include Porphyromonas gingivalis, Treponema denticola, and Tannerella forsythia. Treatment involves antimicrobial therapy and local drug delivery. The document also discusses periodontitis caused by systemic diseases that impair neutrophil function such as Papillon-Lefèvre syndrome, Chédiak-Higashi syndrome, and Down syndrome.
Bone loss and patterns of bone destructionvidushiKhanna1
- introduction
- bone resorption
- factors causing bone destruction in periodontal disease
-- destruction by extension of gingival inflammation
--- histopathology
--- pathways of spread of inflammation
--- radius of action
--- periods of destruction
---- mechanism of destruction
-- bone destruction caused by TFO
-- bone destruction caused by systemic disorders
- factors determining bone morphology in periodontal disease
-- normal variation of alveolar bone
-- exostosis
-- butressing bone formation
-- food impaction
-- agressive periodontitis
- patterns of bone destruction
-- horizontal bone loss
-- vertical or angular defects
-- osseous craters
-- bulbous bone contours
-- reversed architecture
-- ledges
- furcation involvement
-- classification
-conclusion
- Trauma from occlusion occurs when occlusal forces exceed the adaptive capacity of the periodontium, causing injury. It can be acute or chronic.
- The magnitude, direction, duration, and frequency of forces impact the periodontium's ability to adapt. Excessive pressure or tension can damage tissues.
- Primary trauma from occlusion is caused by changes in occlusal forces, while secondary trauma occurs when reduced bone support impairs the tissues' resistance to normal forces.
- The periodontium responds to trauma in three stages - injury, repair through new tissue formation, and adaptive remodeling to better withstand forces. Trauma can cause reversible damage if forces are reduced, or lead to irreversible injury if
This document discusses periodontal response to external forces on teeth. It defines trauma from occlusion as injury to tissues from forces exceeding their adaptive capacity. Trauma can be acute or chronic, and primary (normal support) or secondary (reduced support). It discusses classifications and concepts around how gingival inflammation spreads to bone. Glickman's concept describes zones of irritation and co-destruction that are differentially affected. Waerhaug's concept is that bone loss is due exclusively to plaque inflammation. The stages of tissue response to increased forces are injury, repair, and adaptive remodeling.
Radiographic aids in the diagnosis of periodontal diseaseDara Ghaznavi
This document discusses how radiographs can aid in the diagnosis of periodontal disease. Radiographs reveal changes to calcified tissues but not current cellular activity. Normal interdental septa appear as thin radiopaque lines along the alveolar crest. Early periodontal disease leads to disruption of the lamina dura and crestal cortication. As the disease progresses, bone loss appears as wedge-shaped radiolucencies and reduced height of the interdental bone. Furcation involvement and periodontal abscesses can also be detected radiographically, though clinical examination is still needed.
This document discusses the use of radiographs in diagnosing periodontal disease. It begins by stating that radiographs are a valuable diagnostic tool but not a substitute for clinical examination. It then covers topics like the appearance of normal bone, different radiographic techniques, how bone destruction appears in periodontal disease, and advanced imaging modalities like CBCT. Digital radiography is also discussed as it allows for enhancement and sharing of images.
Radiographic Aids in the Diagnosis of Periodontal Diseases.pptxRoshnaTalibMustafa
This document discusses the use of radiographs in diagnosing periodontal diseases. It begins by explaining that radiographs are valuable for diagnosis, assessing severity, prognosis, and treatment outcomes, but are an adjunct to clinical exams. It then discusses specific radiographic techniques like periapical and bitewing projections. Advanced imaging modalities like cone-beam CT are described as offering advantages over conventional radiography. The remainder describes the radiographic appearance of various periodontal conditions like chronic periodontitis, furcation involvement, abscesses, and trauma from occlusion.
RADIOGRAPHIC EXAMINATIONS OF BONE LOSS AND PATTERN OF BONE LOSSMuhammadWasilKhan1
Radiographs are useful for diagnosing and monitoring periodontal disease. Intraoral radiographs like periapical and bitewing films can detect localized bone loss around individual teeth. Panoramic radiographs provide a comprehensive view of the jaws. Pattern of bone loss seen on radiographs is diagnostically important, as horizontal loss indicates chronic periodontitis while vertical defects may signify a more aggressive disease. While radiographs cannot replace a clinical exam, they provide valuable information about the extent and pattern of destructive changes in the alveolar bone caused by periodontal disease.
RADIOGRAPHIC AIDS IN THE DIAGNOSIS OF PERIODONTAL DISEASES.pptxEUROUNDISA
This document provides an overview of the use of radiographs in diagnosing periodontal diseases. It discusses the diagnostic requirements and techniques for assessing periodontal diseases via radiographs. Key radiographic features of a healthy periodontium and changes seen in various periodontal conditions like chronic periodontitis, aggressive periodontitis and furcation defects are described. Limitations of conventional radiographs and advanced radiographic aids like digital radiography and subtraction radiography are also outlined.
This document discusses the use of radiographs in assessing periodontal disease and bone destruction. It provides information on normal interdental bone appearance and outlines criteria for evaluating bone loss patterns seen in periodontal disease. Key findings include lamina dura disruption being an early sign of periodontitis and interdental cratering appearing as irregular reduced bone density. Furcation involvement is suggested by diminished bone trabeculae radiodensity or marked bone loss on a single root. Additional imaging techniques like CBCT can provide further detail on lesion morphology.
RADIOGRAPHIC AIDS IN THE DIAGNOSIS OF PERIODONTAL DISEASESShivangani Arya
The document discusses the use of radiographic aids in diagnosing periodontal diseases. It provides a history of x-rays and dental radiography, describing their discovery and early uses. It then discusses various intraoral and extraoral radiographic techniques used in periodontal examinations, including periapical, bitewing, occlusal and panoramic images. It outlines how these techniques help assess bone loss, detect calculus, and evaluate treatment outcomes. The document also describes how periodontal bone destruction appears radiographically, such as lamina dura disruption, widening of periodontal ligament space, and the formation of interdental craters.
radiographic diagnosis of periodontal diseaseshabeel pn
Radiographs are an essential tool for diagnosing periodontal disease by assessing bone loss. Early periodontitis appears on radiographs as localized bone erosions while advanced cases show generalized horizontal bone loss. Vertical bone defects can also be seen, appearing as widened ligament spaces or loss of cortical plates. Furcation involvement initially widens the ligament but may progress to deep vertical defects. Aggressive periodontitis in young people causes rapid, widespread bone destruction and early tooth loss. Follow-up radiographs after treatment can demonstrate bone fill-in and sharpening of bony contours.
Radiographs are useful for assessing bone loss and other factors that may contribute to periodontal disease. Bitewing radiographs are best for posterior teeth while periapical views work for anterior teeth. Bone loss is described by its pattern (horizontal or vertical), distribution (localized or generalized), and severity (mild, moderate, or severe). Predisposing factors seen on radiographs include calculus, defective restorations, and open contacts. Radiographs also allow evaluation of treatment outcomes and monitoring of disease progression over time.
Radiographs in prosthodontics/dental crown &bridge course by Indian dental ac...Indian dental academy
Dental radiographs are necessary for comprehensive patient care and enable detection of conditions that may otherwise go undetected. They are useful for diagnosing lesions, diseases, conditions of teeth and surrounding structures. Radiographs provide information during procedures and are used to evaluate growth, development, and changes from diseases. They document the condition of patients over time. Radiographs play an important role in evaluating patients for complete dentures, removable partial dentures, and fixed partial dentures by assessing bone quality and quantity, lesions, root morphology, and other diagnostic factors. They are also used in implant imaging to visualize potential implant sites and guide site selection.
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This document discusses various imaging techniques and lesions involving the maxilla and mandible. It describes intraoral and extraoral imaging techniques including periapical, panoramic, and cone beam CT. Common cysts discussed include radicular, dentigerous, odontogenic keratocysts, and nasopalatine duct cysts. Characteristics like location, relationship to teeth, borders, and effects on surrounding structures are used to differentiate these lesions on imaging.
radiographic aids in the diagnosis of periodontal disease.pptmangeshandhare1
Radiographs are a valuable aid in diagnosing and monitoring periodontal disease by revealing past alterations to calcified tissues like bone and teeth. While they do not indicate current cellular activity, they can show the extent of past periodontal disease through patterns of bone destruction. Normal variations in radiographic technique can impact how structures like the interdental septa, lamina dura, and bone levels appear. Correct technique like the long cone paralleling method produces the most accurate images. Radiographs are also useful for evaluating the amount, distribution, and pattern of bone loss associated with periodontal disease.
Inflammation from endodontic disease affects the surrounding bone and teeth, causing changes that can be seen on radiographs. Radiographs used to evaluate the periapical tissues should include the entire root tip and bone, and be well-positioned to avoid distortions. Radiographic signs of endodontic disease include loss of the lamina dura, increased width of the periodontal ligament space, periapical radiolucencies with distinct or indistinct borders indicating acute or chronic lesions, diffuse radiopacities from sclerosing osteomyelitis, and changes to the trabecular bone pattern. Internal and external root resorption as well as changes to pulp cavity size from pulpitis can also
This report, prepared by the student at the College of Dentistry, Hassan Atheed , in the third phase discusses scientific topics, but it maybe did not be 100% complete.
A radiograph is only one part of the diagnostic process. Usually one does NOT make a diagnosis solely from a radiograph. A diagnosis is made by the clinician once all the diagnostic information has been collected and analyzed collectively. An interpretation or a differential diagnosis is made from the radiograph.
One examines a radiograph and NOT an X-ray. Bear in mind that an X-ray cannot be seen. X-ray is a photon / beam of energy.
حسن عضيد
Radiographs are essential for endodontic diagnosis, treatment, and follow-up. The document outlines the importance and limitations of various radiographic techniques used in endodontics. It discusses radiographic interpretation of normal and abnormal findings, differential diagnosis of lesions, and new technologies like digital radiography and cone beam computed tomography. Radiographic sequence and special techniques like paralleling, bisecting, and cone shift methods are also covered to optimize imaging quality and diagnostic value.
This document provides an overview of radiographic interpretation for periapical and panoramic dental x-rays. It discusses the objectives of understanding normal dental anatomy and interpreting pathology under radiographs. Key views covered include periapical and panoramic x-rays. The document outlines the normal radiographic anatomy seen in periapical and panoramic views and provides guidance on interpreting radiographs, including steps to localize, observe, consider generally, interpret, and correlate findings. Common dental structures and conditions that can be evaluated on radiographs are described, such as caries, pulp calcification, bone loss, restorations, and lesions.
Radiographic aids in periodontal disease diagnosis part IPeriowiki.com
This document discusses the use of radiographs in diagnosing periodontal disease. It provides a brief history of using radiographs in dentistry and periodontics. Radiographs can reveal bone loss and destruction patterns but not current soft tissue changes. Standardized techniques are needed for accurate assessment over time. Early periodontitis may show fuzziness of the lamina dura or widening of the periodontal ligament space. More advanced disease appears as severe bone loss, fingerlike projections into the bone, or interdental bone craters. However, radiographs have limitations in depicting the full extent and morphology of bone defects.
This document provides an overview of radiographic interpretation for periapical and panoramic dental x-rays. It defines dental radiography and describes the main intraoral and extraoral views. Periapical radiographs show the entire tooth and surrounding structures, and are useful for detecting dental issues like caries, periapical pathology, implants and more. Panoramic radiographs provide a wide view of the jaws and are used to assess issues like gross caries, fractures, cysts and tumors. The document outlines the normal radiographic anatomy seen in these views and provides guidance on interpreting radiographs through steps like localization, observation, interpretation and correlation to arrive at a diagnosis.
Similar to Radiographic aids in Periodontal Diagnosis (20)
Host modulation therapy is recommended as an adjunct to scaling and root planing in the periodontal therapy. The basic purpose of host modulation therapy is to restore the balance between pro-inflammatory and anti-inflammatory mediators.
Periodontal pocket is a pathologically deepened gingival sulcus. There are two types of pockets - gingival pocket and periodontal pocket. The periodontal pocket formation is the first step in the periodontal destruction. It is important to understand the etiopathogenesis of the periodontal pocket formation for appropriate diagnosis and treatment planning.
Human Immunodeficiency Virus (HIV) can cause oral manifestations in 30-80% of infected individuals. Common oral issues include candidiasis, a fungal infection causing lesions; periodontitis with tissue destruction; and viral infections like herpes simplex causing vesicles and ulcers. Other conditions linked to HIV/AIDS are Kaposi's sarcoma, a cancer originating from blood vessels, and non-Hodgkin's lymphoma. Proper dental management of HIV-infected individuals includes monitoring for early signs of oral opportunistic infections and treating them promptly to support overall health.
This document discusses the effects of smoking on periodontal disease. It notes that cigarette smoke contains over 400 toxic substances and that nicotine is the main alkaloid that is responsible for the addictive potential of tobacco. It then summarizes various studies that found higher rates of periodontal disease in smokers compared to non-smokers. The document also outlines how smoking can impact clinical signs of inflammation, the gingival epithelium, gingival bleeding, the gingival crevicular fluid, and the subgingival microflora. It discusses the negative effects of smoking on periodontal treatment and healing. The importance of smoking cessation is emphasized, with models and methods for quitting smoking presented.
Dental caries is the major dental disease affecting a large population. Cariostatic efficacy of the fluorides have increased the use of fluoride agents. This presentation will enlighten us about the use of fluorides in preventive dentistry.
Halitosis is derived from a Latin word which means unpleasant breath. If not treated, it could affect your social life. Majority of the cases of halitosis have oral origin. Therefore, appropriate dental treatment eliminates the cause.
Vitamins & minerals are essential for the development and functioning of the organism. Maintaining a healthy life will help in maintaining a healthy mouth since poor health is a link to diseases.
Acute periodontal diseases are clinical conditions of rapid onset that involve the periodontium. They are characterised by discomfort or pain and infection. They require urgent attention which involves prompt diagnosis and treatment to prevent the further destruction.
Periodontal abscess is a localised purulent infection in the tissues adjacent to the periodontal pocket that may lead to the destruction of the periodontal ligament and alveolar bone. Periodontal abscess is the third most prevalent emergency infection after acute alveolar abscess and pericoronitis. It could lead to complications due to bacteremia that may cause infection at distant locations. Proper management of the abscess is crucial to alleviate pain, establish drainage and control the spread of infection.
The cementum is a specialised calcified substance covering the root of the tooth. The cementum is a part of the periodontium that attaches the teeth to the alveolar bone by anchoring the periodontal ligament. This presentation covers the anatomy and pathologies associated with the cementum.
This document discusses various types of gingival pathology including acute and chronic conditions, localized and generalized manifestations, and inflammatory and neoplastic enlargements. It reviews clinical findings such as bleeding, color changes, consistency changes, surface texture changes, position changes and contour changes. Specific acute gingival infections discussed include necrotizing ulcerative gingivitis, primary herpetic gingivostomatitis, and pericoronitis. Chronic conditions and enlargements covered include inflammatory, drug-induced, idiopathic, pregnancy-related, puberty-related, vitamin C deficiency, plasma cell, leukemia-associated, and neoplastic enlargements. Treatment approaches are presented for different conditions.
Every periodontal surgical procedure has its own indications. With proper knowledge of the etiology of the disease, correct diagnosis and treatment planning, the clinician is able to draw predictable success with periodontal flap surgery.
The future of dentistry and periodontics lies in regeneration. The goals of periodontal therapy lies in not only the arrest of periodontal disease progression but also regeneration of the lost periodontal structures. This presentation provides a review of the current understanding of the regeneration of the periodontium and the procedures involved to restore the periodontal tissues around the teeth.
The main objective of periodontal surgery is to achieve health and integrity of the periodontium by plaque removal and plaque control. Patient preparation is an important aspect of the intervention. The presentation mentions certain principles of periodontal surgery which are crucial for effective treatment of the patient.
DENTIN HYPERSENSITIVITY - ETIOLOGY, DIAGNOSIS AND TREATMENTDr.Shraddha Kode
This document discusses dentin hypersensitivity (DH), including its definition, prevalence, causes, diagnostic process, and treatment options. It notes that DH is pain from exposed dentin in response to stimuli that cannot be explained by other dental issues. It affects 20-50 year olds, especially women, and commonly occurs in canines and premolars. Treatment includes at-home options like desensitizing toothpastes and in-office options like potassium nitrate, resins, or lasers to occlude tubules or disturb nerve transmission. Newer treatments showing promise include arginine-based toothpastes and nano-hydroxyapatite due to their ability to quickly and effectively reduce DH pain.
Biologic width - Importance in Periodontal and Restorative DentistryDr.Shraddha Kode
The document discusses the biologic width, which refers to the dimensions of the soft tissue attached to the tooth coronal to the alveolar bone crest. It consists of the junctional epithelium, connective tissue attachment, and the sulcular depth, totaling approximately 2-3 mm. Violation of the biologic width by placing restorative margins too far subgingivally can lead to bone resorption, gingival recession, or hyperplasia. The document outlines techniques for assessing biologic width dimensions and various surgical or orthodontic procedures to correct violations. Maintaining at least 3 mm of distance from the restorative margin to alveolar bone is important for periodontal health.
Bone Morphogenetic Proteins - Role in Periodontal RegenerationDr.Shraddha Kode
This document discusses bone morphogenetic proteins (BMPs), which are signaling molecules that govern tissue development and regeneration. BMPs play an important role in periodontal regeneration and regeneration of other dental tissues. The document outlines the classification, signaling pathways, roles in development, and clinical applications of BMPs, particularly BMP-2 and BMP-7, which have shown efficacy in regenerating bone and periodontal tissues when delivered with appropriate carrier materials. While BMPs show promise for regenerative therapies, further research is still needed to optimize dosage, delivery methods, and carrier materials.
Nicotine Replacement Therapy (NRT) can help with the withdrawal symptoms in patients who find it difficult to quit tobacco. It is available in the form of - gums, patches, sprays, inhalers or lozenges.
Platelet Rich Fibrin (PRF) is an autologous fibrin based biomaterial derived from human blood discovered by Choukroun and coworkers in the year 2006. The future of PRF has enormous therapeutic implications. Therefore, more clinicians should adopt this technology for the benefit of the patients.
Cell Therapy Expansion and Challenges in Autoimmune DiseaseHealth Advances
There is increasing confidence that cell therapies will soon play a role in the treatment of autoimmune disorders, but the extent of this impact remains to be seen. Early readouts on autologous CAR-Ts in lupus are encouraging, but manufacturing and cost limitations are likely to restrict access to highly refractory patients. Allogeneic CAR-Ts have the potential to broaden access to earlier lines of treatment due to their inherent cost benefits, however they will need to demonstrate comparable or improved efficacy to established modalities.
In addition to infrastructure and capacity constraints, CAR-Ts face a very different risk-benefit dynamic in autoimmune compared to oncology, highlighting the need for tolerable therapies with low adverse event risk. CAR-NK and Treg-based therapies are also being developed in certain autoimmune disorders and may demonstrate favorable safety profiles. Several novel non-cell therapies such as bispecific antibodies, nanobodies, and RNAi drugs, may also offer future alternative competitive solutions with variable value propositions.
Widespread adoption of cell therapies will not only require strong efficacy and safety data, but also adapted pricing and access strategies. At oncology-based price points, CAR-Ts are unlikely to achieve broad market access in autoimmune disorders, with eligible patient populations that are potentially orders of magnitude greater than the number of currently addressable cancer patients. Developers have made strides towards reducing cell therapy COGS while improving manufacturing efficiency, but payors will inevitably restrict access until more sustainable pricing is achieved.
Despite these headwinds, industry leaders and investors remain confident that cell therapies are poised to address significant unmet need in patients suffering from autoimmune disorders. However, the extent of this impact on the treatment landscape remains to be seen, as the industry rapidly approaches an inflection point.
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3. “Xrays were discovered in 1895 by
professor William Conrad
Roentgen and Dr.Otto Walkhoff
is credited with the first dental
radiograph
3
4. INTRODUCTION
4
◦ Radiographs are a VALUABLE
TOOL for the diagnosis of
periodontal disease, estimation of
severity, determination of
prognosis and evaluation of
treatment outcome
◦ Radiographs are an ADJUNCT to
the clinical examination not a
substitute for it
5. RADIOGRAPHIC
TECHNIQUES
5
◦ Radiographs can be divided
into extraoral and intraoral
radiographs
◦ Intraoral – bitewing, periapical
and occlusal
◦ Intraoral periapical –
commonly done for a part of
the dentition whereas bite wing
radiographs are done for
posterior teeth
6. 6
◦ Occlusal radiographs – usually
done to find out buccolingual
or buccopalatal positions of the
impacted canines or third
molars to determine the extent
of diseases like cysts
◦ Extraoral – orthopantomogram
(OPG) – most commonly used
– complete bilateral view of
maxilla, mandible and TMJ
8. 8
Prichard established following FOUR CRITERIA to determine
adequate angulation of periapical radiographs:
1.The radiograph should show tips of molar cusps with little
or none of the occlusal surface showing
2.Enamel caps and pulp chambers should be distinct
3.Interproximal spaces should be open
4.Proximal contacts should not overlap unless teeth are out
of line automatically
9. Positioning guidelines for intraoral
radiographs
Paralleling technique:
Most accurate
Xray film is kept parallel to the teeth
Xray beam is directed at right angles
to the teeth and Xray film
Disadv – shallow palate – keeping
film parallel becomes difficult
10. Positioning guidelines for intraoral
radiographs
Bisecting angle technique:
Receptor is placed diagonally to the
teeth
Xray beam is directed at right angles
to the plane that is midway between
Xray film and teeth
Useful technique when receptor
placement cannot be achieved due to
shallow palate, pre of tori
11. STANDARDISED
RADIOGRAPHIC
TECHNIQUES
11
◦ Standardised reproducible
techniques – for pre-treatment and
post-treatment comparisons –
using position indicating device
◦ Standardisation of exposure and
development time, type of film,
Xray angulation minimises
image distortion
◦ Grid calibrated in millimeters over
the radiograph – calculate bone
levels
12. Radiographic findings of healthy
periodontal structures
Tooth is surrounded by a thin
radiolucent space which houses the pdl
Width of pdl – tooth under occlusal
overload – width is increased
Alveolar bone surrounding the tooth
root – radiopaque line just adjacent to
pdl space – lamina dura
13. 13
Radiographically it appears as a white line but in reality
it is perforated by numerous small foramina, traversed by
blood vessels, lymphatics and nerves which pass between pdl
and bone – continuity and integrity examined carefully on
radiograph
14. 14
The level of the crest of the interdental bone is parallel to the line
joining the CEJ of the adjacent teeth
Interdental bone – thin between anterior teeth due to less
interdental space and wide between posterior teeth – wide interdental
space
Bone resorption of interdental bone due to periodontal disease –
crest of the interdental bone – angulated
Most common reason for radiolucency in the apical region of the
root – endodontic involvement
15. 😉
15
Bone loss in Periodontal Disease
Minor bone loss on the buccal aspect is overlapped by
intact lingual bone, thus bone loss on one aspect may be
camouflaged by bone on the opposite side
Thus, early signs of periodontitis like deepening of
periodontal pocket or recession are best visualised
clinically
Actual severity of periodontal destruction is more than
as shown on radiograph
16. 16
The amount of bone lost is calculated
arbitarily by estimating the difference
between the physiological bone level and the
height of bone remaining
Distance between CEJ and the alveolar
crest in a healthy periodontium is 2mm
17. 17
How to assess for bone loss on radiograph????
The interproximal bone loss may be parallel to the line joining
the CEJ – Horizontal bone loss
At an angle to the line joining the CEJ of adjacent teeth –
Angular or Vertical bone loss
Topography of the bone defect cannot be accurately assessed
by the radiograph – bone destruction that occurs in the
cancellous bone is obscured by the
dense buccal and lingual/palatal
Cortical plates
18. 18
A minimum of 0.5-1mm reduction in the
level of cortical plate is required to permit the
radiographic visualisation of bone loss of
cancellous bone
The best method to check for the defect
morphology is surgical exposure of the area
19. 19
Radiographic features of bone loss in
periodontitis
Imp radiographic feature of periodontitis: fuzziness
and discontinuity of the lamina dura
Radiographic findings should not be correlated to
the clinical findings – intact lamina dura
indicates periodontal health ; discontinuity or
fuzziness of lamina dura does not indicate pre of
inflammation, BOP, periodontal pockets or loss of
attachment
20. 20
Bone resorption on lateral aspect of interdental septum
– wedge shaped radiolucent area on the mesial or
distal aspect – widening of pdl
Radiographic appearance of finger like radiolucent
projections that extend from the crest of the bone into
the septum
Localised aggressive periodontitis – vertical arc like
destructive pattern
Generalised aggressive periodontitis – severe bone loss
21. 21
Arc shaped bone lossWedge shaped
radiolucent area
Finger like radiolucent
projections
23. 23
RADIOGRAPHIC
FINDINGS OF
PERIODONTAL
ABSCESS
Acute changes cannot be visualised on
the radiograph due to minimal changes
in the alveolar bone whereas chronic
lesion can be visualised
Periodontal abscess – localised to the
soft tissue wall – less likely to produce
radiographic changes
Periodontal abscess present on lingual
and facial surfaces of teeth are obscured
by the root surface on radiograph – less
visible. Thus radiograph is not a good
indicator of periodontal abscess
25. 25
Osteosclerosis
Homogenous
radiodense areas
– mandible more
affected –
deposition of
excessive bone
during repair
Fibrous dysplasia
Finely trabeculated
radiodensity –
ground glass
appearance
Paget’s disease
Radiolucent – osteolysis dominates, middle stage: -
deposition of bone – cotton wool appearance and late
stage – osteoblastic apposition
Vestibulum nec
congue tempus
SPECIFIC
DISEASES
27. 27
CONCLUSION
Radiographs – Important role in the diagnosis of periodontal
diseases
Important information regarding anatomical structures
and periodontal bone loss