Personality disorder ppt MENTAL HEALTH NURSINGvihang tayde
Most definition of normal personality includes some or all of the following features,
Present since adolescence.
Stable overtime despite fluctuations in mood.
Manifest in different environment.
Recognizable to friends and acquaintance.
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Personality disorder ppt MENTAL HEALTH NURSINGvihang tayde
Most definition of normal personality includes some or all of the following features,
Present since adolescence.
Stable overtime despite fluctuations in mood.
Manifest in different environment.
Recognizable to friends and acquaintance.
hii guys this is my ongoing presentation from my speciality class i hope u guys lije that please so i hope it is been useful for u in ur specialities by getting little help with that
Delirium is a syndrome not a disease and it has many causes. it is an acute organic mental disorder characterised by impairment of consciousness, disorientation and disturbances in perception and restlessness.
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Tom Selleck Health: A Comprehensive Look at the Iconic Actor’s Wellness Journeygreendigital
Tom Selleck, an enduring figure in Hollywood. has captivated audiences for decades with his rugged charm, iconic moustache. and memorable roles in television and film. From his breakout role as Thomas Magnum in Magnum P.I. to his current portrayal of Frank Reagan in Blue Bloods. Selleck's career has spanned over 50 years. But beyond his professional achievements. fans have often been curious about Tom Selleck Health. especially as he has aged in the public eye.
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Introduction
Many have been interested in Tom Selleck health. not only because of his enduring presence on screen but also because of the challenges. and lifestyle choices he has faced and made over the years. This article delves into the various aspects of Tom Selleck health. exploring his fitness regimen, diet, mental health. and the challenges he has encountered as he ages. We'll look at how he maintains his well-being. the health issues he has faced, and his approach to ageing .
Early Life and Career
Childhood and Athletic Beginnings
Tom Selleck was born on January 29, 1945, in Detroit, Michigan, and grew up in Sherman Oaks, California. From an early age, he was involved in sports, particularly basketball. which played a significant role in his physical development. His athletic pursuits continued into college. where he attended the University of Southern California (USC) on a basketball scholarship. This early involvement in sports laid a strong foundation for his physical health and disciplined lifestyle.
Transition to Acting
Selleck's transition from an athlete to an actor came with its physical demands. His first significant role in "Magnum P.I." required him to perform various stunts and maintain a fit appearance. This role, which he played from 1980 to 1988. necessitated a rigorous fitness routine to meet the show's demands. setting the stage for his long-term commitment to health and wellness.
Fitness Regimen
Workout Routine
Tom Selleck health and fitness regimen has evolved. adapting to his changing roles and age. During his "Magnum, P.I." days. Selleck's workouts were intense and focused on building and maintaining muscle mass. His routine included weightlifting, cardiovascular exercises. and specific training for the stunts he performed on the show.
Selleck adjusted his fitness routine as he aged to suit his body's needs. Today, his workouts focus on maintaining flexibility, strength, and cardiovascular health. He incorporates low-impact exercises such as swimming, walking, and light weightlifting. This balanced approach helps him stay fit without putting undue strain on his joints and muscles.
Importance of Flexibility and Mobility
In recent years, Selleck has emphasized the importance of flexibility and mobility in his fitness regimen. Understanding the natural decline in muscle mass and joint flexibility with age. he includes stretching and yoga in his routine. These practices help prevent injuries, improve posture, and maintain mobilit
These lecture slides, by Dr Sidra Arshad, offer a quick overview of physiological basis of a normal electrocardiogram.
Learning objectives:
1. Define an electrocardiogram (ECG) and electrocardiography
2. Describe how dipoles generated by the heart produce the waveforms of the ECG
3. Describe the components of a normal electrocardiogram of a typical bipolar leads (limb II)
4. Differentiate between intervals and segments
5. Enlist some common indications for obtaining an ECG
Study Resources:
1. Chapter 11, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 9, Human Physiology - From Cells to Systems, Lauralee Sherwood, 9th edition
3. Chapter 29, Ganong’s Review of Medical Physiology, 26th edition
4. Electrocardiogram, StatPearls - https://www.ncbi.nlm.nih.gov/books/NBK549803/
5. ECG in Medical Practice by ABM Abdullah, 4th edition
6. ECG Basics, http://www.nataliescasebook.com/tag/e-c-g-basics
Pulmonary Thromboembolism - etilogy, types, medical- Surgical and nursing man...VarunMahajani
Disruption of blood supply to lung alveoli due to blockage of one or more pulmonary blood vessels is called as Pulmonary thromboembolism. In this presentation we will discuss its causes, types and its management in depth.
Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
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New Directions in Targeted Therapeutic Approaches for Older Adults With Mantl...i3 Health
i3 Health is pleased to make the speaker slides from this activity available for use as a non-accredited self-study or teaching resource.
This slide deck presented by Dr. Kami Maddocks, Professor-Clinical in the Division of Hematology and
Associate Division Director for Ambulatory Operations
The Ohio State University Comprehensive Cancer Center, will provide insight into new directions in targeted therapeutic approaches for older adults with mantle cell lymphoma.
STATEMENT OF NEED
Mantle cell lymphoma (MCL) is a rare, aggressive B-cell non-Hodgkin lymphoma (NHL) accounting for 5% to 7% of all lymphomas. Its prognosis ranges from indolent disease that does not require treatment for years to very aggressive disease, which is associated with poor survival (Silkenstedt et al, 2021). Typically, MCL is diagnosed at advanced stage and in older patients who cannot tolerate intensive therapy (NCCN, 2022). Although recent advances have slightly increased remission rates, recurrence and relapse remain very common, leading to a median overall survival between 3 and 6 years (LLS, 2021). Though there are several effective options, progress is still needed towards establishing an accepted frontline approach for MCL (Castellino et al, 2022). Treatment selection and management of MCL are complicated by the heterogeneity of prognosis, advanced age and comorbidities of patients, and lack of an established standard approach for treatment, making it vital that clinicians be familiar with the latest research and advances in this area. In this activity chaired by Michael Wang, MD, Professor in the Department of Lymphoma & Myeloma at MD Anderson Cancer Center, expert faculty will discuss prognostic factors informing treatment, the promising results of recent trials in new therapeutic approaches, and the implications of treatment resistance in therapeutic selection for MCL.
Target Audience
Hematology/oncology fellows, attending faculty, and other health care professionals involved in the treatment of patients with mantle cell lymphoma (MCL).
Learning Objectives
1.) Identify clinical and biological prognostic factors that can guide treatment decision making for older adults with MCL
2.) Evaluate emerging data on targeted therapeutic approaches for treatment-naive and relapsed/refractory MCL and their applicability to older adults
3.) Assess mechanisms of resistance to targeted therapies for MCL and their implications for treatment selection
Flu Vaccine Alert in Bangalore Karnatakaaddon Scans
As flu season approaches, health officials in Bangalore, Karnataka, are urging residents to get their flu vaccinations. The seasonal flu, while common, can lead to severe health complications, particularly for vulnerable populations such as young children, the elderly, and those with underlying health conditions.
Dr. Vidisha Kumari, a leading epidemiologist in Bangalore, emphasizes the importance of getting vaccinated. "The flu vaccine is our best defense against the influenza virus. It not only protects individuals but also helps prevent the spread of the virus in our communities," he says.
This year, the flu season is expected to coincide with a potential increase in other respiratory illnesses. The Karnataka Health Department has launched an awareness campaign highlighting the significance of flu vaccinations. They have set up multiple vaccination centers across Bangalore, making it convenient for residents to receive their shots.
To encourage widespread vaccination, the government is also collaborating with local schools, workplaces, and community centers to facilitate vaccination drives. Special attention is being given to ensuring that the vaccine is accessible to all, including marginalized communities who may have limited access to healthcare.
Residents are reminded that the flu vaccine is safe and effective. Common side effects are mild and may include soreness at the injection site, mild fever, or muscle aches. These side effects are generally short-lived and far less severe than the flu itself.
Healthcare providers are also stressing the importance of continuing COVID-19 precautions. Wearing masks, practicing good hand hygiene, and maintaining social distancing are still crucial, especially in crowded places.
Protect yourself and your loved ones by getting vaccinated. Together, we can help keep Bangalore healthy and safe this flu season. For more information on vaccination centers and schedules, residents can visit the Karnataka Health Department’s official website or follow their social media pages.
Stay informed, stay safe, and get your flu shot today!
Report Back from SGO 2024: What’s the Latest in Cervical Cancer?bkling
Are you curious about what’s new in cervical cancer research or unsure what the findings mean? Join Dr. Emily Ko, a gynecologic oncologist at Penn Medicine, to learn about the latest updates from the Society of Gynecologic Oncology (SGO) 2024 Annual Meeting on Women’s Cancer. Dr. Ko will discuss what the research presented at the conference means for you and answer your questions about the new developments.
4. Organic Psychosis
• An organic psychosis is an abnormal mental state
with a known physical cause characterized by an
altered perception of reality.
• American Psychiatric Association defines organic
psychosis/ organic brain syndrome as, “a mental
disorder characteristically resulting from diffuse
impairment of brain tissue function from any
cause.”
5. Suspect Organic if…
• First episode
• Sudden onset
• Older age of onset
• History of drug and alcohol use disorders
• Concurrent medical or neurological disorder
• Neurological signs or symptoms like seizure
,impairment in consciousness, head injury, sensory or
motor disturbances
• Presence of confusion , disorientation ,memory
impairment or soft neurological sign.
• Prominent visual or other non auditory hallucinations.
6. Types of Organic Mental disorders
• Delirium
• Dementia
• Organic amnestic syndrome
• Other organic mental disorders
7. Delirium
• Delirium is an acute, transient, usually reversible,
fluctuating disturbance in attention, cognition,
and consciousness level.
• A sudden and significant decline in mental
functioning not better accounted for by a
preexisting or evolving dementia
• Disturbance of consciousness with reduced ability
to focus, sustain, and shift attention.
8. Prevalence
• Delirium may occur at any age but is more common
among the elderly.
• At least 10% of elderly patients who are admitted to
the hospital have delirium; 15 to 50% experience
delirium at some time during hospitalization.
• Delirium is also common after surgery and among
nursing home residents and ICU patients.
• When delirium occurs in younger people, it is usually
due to drug use or a life-threatening systemic disorder.
9. Etiology
• I nfections
• W ithdrawal drug or alcohol
• A cute metabolic conditions
• T rauma
• C NS pathology
• H ypoxia
• D efficiencies, vitamins
• E ndocrinopathies
• A cute vascular conditions
• T oxins or drugs
• H eavy metals poisoning
10. Clinical features:
• Impairment of consciousness
• Appearance and behaviour: the patient looks unwell and
behaviour may be marked by agitation or hypoactivity,
• Mood is frequently labile, with perplexity, intermittent periods
of anxiety or depression,
• Speech: the patient may mumble and be incoherent.
• Perception: visual perception is the modality most often
affected. Illusions and misinterpretations are frequent.
• Cognition: there are abnormalities in all areas of cognitive
function. Memory registration, retention, and recall are all
affected.
• Orientation: in obvious cases, orientation in person, time, and
place will all be disturbed.
11. • Concentration is impaired
• Memory disturbances are seen, with impaired
registration (e.g. digit span), short-term recall (e.g.
name and address), and long-term recall (e.g.
current news items).
• Insight is usually impaired.
• The disturbance of sleep wake cycle most
commonly insomnia at night with day time
drowsiness
• Diurnal variation is marked usually with worsening
of symptoms in the evening and night (called sun
downing )
12. ICD 10 diagnostic criteria
• Impairment of the consciousness and attention (on a
continuum from clouding to coma, reduced ability to direct
,focus ,sustain and shift attention )
• Global disturbance of cognition ( perceptual distortions:
illusions and hallucinations most often visual ; impairment
of abstract thinking and comprehension with or with out
transient delusions ,but typically with some degree of
incoherence ,impairment of immediate recall and of recent
memory but relatively intact remote memory ;disorientation
for time as well as in more severe cases for place and
person.
13. • Psychomotor disturbances ( hypo or hyper activity and
unpredictable shifts from one to one another ;increased
reaction time increased or decreased flow of speech and
enhanced startle reactions )
• Disturbance of sleep wake cycle (insomnia or in severe cases
total sleep loss or reversal of the sleep wake cycle ;day time
drowsiness ,nocturnal worsening of symptoms ,disturbing
dream or nightmares which may continue as hallucinations
after awakening)
• Emotional disturbances .e.g. depression, anxiety or fear,
irritability ,euphoria ,apathy.
• The onset is usually rapid and the course diurnally fluctuating
and total duration of the condition much less than 6 months .
14. Physical and Laboratory Examination
• Physical examination reveals the cause of delirium
• Laboratory work up include CBC, electrolytes, thyroid
function tests, ECG , EEG ,chest x ray ,blood ,urine ,
and CSF cultures .
• EEG: It shows a generalized slowing of activity
18. Physical Intervention
• Initial interventions include general measures to support
cerebral function, such as intravenous hydration and
appropriate nourishment.
• Supplemental oxygen has been found to be highly effective
in patients who develop delirium with pneumonia.
• Physical restraints, once a mainstay in the treatment of
delirium, are now used only when all pharmacologic and
nonpharmacologic interventions have failed.
19. Environmental Intervention
• Environmental manipulations are directed toward
providing the right amount of stimulation for the
patient, encouraging sleep, maximizing the patient's
ability to perceive the environment accurately,
maintaining safety, and achieving familiarity and
consistency for the patient.
• Over stimulation should be avoided, because it
contributes to both confusion and insomnia
• Avoid understimulation too
• Sun downing can be lessened by leaving a radio on in
the patient's room
20. • visual hallucinations by controlled visual stimuli, auditory
hallucinations by music and other meaningful external sounds, and
olfactory hallucinations by the introduction of odors or scents
• adequate daytime lighting and a night light should be provided
• Hearing aids, eyeglasses and other devices that assist sensory
perception should be used whenever possible and should not be
put away during a delirious episode
• having family members stay with the patient. Family members
should also be encouraged to bring personal effects from home,
because some patients with delirium are greatly comforted by the
presence of familiar photographs or objects.
21. Cognitive Interventions:
• Reorientation
• place a clock and a calendar where the patient can
see them easily.
• should then be verbally reoriented to time and place
several times over the course of the day.
• Repetition is recommended to compensate for
memory impairment in the delirious patient.
22. Psychologic Interventions:
• The delusions expressed by a patient should not be
directly disputed. Instead, alternative explanations of
events should be offered, and frequent reassurance
should be given.
23. Pharmacologic Interventions
• 100 mg of B1 IV for thiamine deficiency and IV fluids
for fluid and electrolyte imbalances
• Symptomatic management: as many patients are
agitated , emergency psychiatric treatments may be
needed. Small doses of benzodiazepines (lorazepam
or diazepam) or antipsychotics (haloperidol) may be
given orally or parenterally.
24. Patient Education
• Educating families and patients regarding the etiology
and course of disease is an important role for physicians.
• Educate the patient, family, and primary caregivers
about future risk factors.
• Families may worry that the patient has brain damage
or a permanent psychiatric illness. Providing
reassurance that delirium often is temporary and is the
result of a medical condition may be beneficial to both
patients and their families.
• Suggest that family members or friends visit the patient,
usually one at a time, and provide a calm and structured
environment. Encourage them to furnish some familiar
objects, such as photos or a favorite blanket, to help
reorient the patient and make the patient feel more
secure.
25. Nursing management :
• Assessment
• Client history : from the clients history ,nurses should
assess the following areas of concern.
• Type ,frequency, and severity of mood swings,
• Personality and behavioral changes
• Catastrophic emotional reactions
• Cognitive changes such as problems with attention
span ,thinking process ,problem –solving
• Language difficulties
• Orientation to person ,place, date and situation
• Appropriateness of social behavior
27. Nursing diagnoses and Intervention
• Arrange furniture and other items in the room to
accommodate clients disabilities
• Store frequently used items within easy access
• Do not keep bed in elevated position
• Assist the client with ambulation
• Keep a dim light on at night
• Frequently orient the client to place ,time and
situation
• Soft restraints may be required if client is very
disoriented and hyperactive
30. Introduction
• Dementia is not a specific disease. It is an
overall term that describes a wide range of
symptoms associated with a decline in
memory or other thinking skills severe enough
to reduce a person’s ability to perform
everyday activities.
• Dementia is a general loss of cognitive
abilities, including impairment of memory as
well as one or more of the following: aphasia,
apraxia, agnosia, or disturbed planning,
organizing, and abstract thinking abilities.
32. Alzheimer's disease (AD):
• The exact cause of Alzheimer’s disease (AD) is
unknown, but several theories have been
proposed, such as reduction in brain
acetylcholine, the formation of plaques and
tangles, serious head trauma, and genetic factors.
• Pathologic changes in the brain include atrophy,
enlarged ventricles, and the presence of
numerous neurofibrillary plaques and tangles.
33.
34. • Definitive diagnosis is by biopsy or
autopsy examination of brain tissue,
although refinement of diagnostic
criteria and new diagnostic tools now
enable clinicians to use specific clinical
features to identify the disease at an
accuracy rate of 70% to 90%.
35. Vascular Dementia
• This type of dementia is caused by significant
cerebrovascular disease.
• The client suffers the equivalent of small
strokes caused by arterial hypertension or
cerebral emboli or thrombi, which destroy
many areas of the brain.
• The onset of symptoms is more abrupt than in
AD and runs a highly variable course, progressing
in steps rather than as a gradual deterioration.
36.
37. Dementia due to HIV Disease
• The immune dysfunction associated with
human immunodeficiency virus (HIV) disease
can lead to brain infections by other organisms.
• HIV also appears to cause dementia directly.
38. Lewy body Dementia
• Clinically, Lewy body disease is fairly similar to
AD; however, it tends to progress more
rapidly, and there is an earlier appearance
visual hallucinations and parkinsonian
features.
• This disorder is distinctive by the presence of
Lewy bodies—eosinophilic inclusion bodies—
seen in the cerebral cortex and brainstem
39.
40.
41. Frontotemporal Dementia
• Frontotemporal dementia (FTD) or
frontotemporal degenerations refers to a
group of disorders caused by progressive
nerve cell loss in the brain's frontal lobes (or
its temporal lobes.
42. • The nerve cell damage caused by
frontotemporal dementia leads to loss of
function in these brain regions, which variably
cause deterioration in behavior and
personality, language disturbances, or
alterations in muscle or motor functions.
• It was used to be called Picks disease.
43. Key Differences Between FTD and
Alzheimer's
• Age at diagnosis may be an important clue. Most
people with FTD are diagnosed in their 40s and
early 60s. Alzheimer's, on the other hand, grows
more common with increasing age.
• Memory loss tends to be a more prominent
symptom in early Alzheimer's than in early FTD,
although advanced FTD often causes memory
loss in addition to its more characteristic effects
on behavior and language.
44. • Behavior changes are often the first
noticeable symptoms in bvFTD, the most
common form of FTD. Behavior changes are
also common as Alzheimer's progresses, but
they tend to occur later in the disease.
• Hallucinations and delusions are relatively
common as Alzheimer's progresses, but
relatively uncommon in FTD.
45. • Problems with spatial orientation — for
example, getting lost in familiar places — are
more common in Alzheimer's than in FTD.
• Problems with speech. Although people with
Alzheimer's may have trouble thinking of the
right word or remembering names, they tend
to have less difficulty making sense when they
speak, understanding the speech of others, or
reading than those with FTD.
46. Huntington’s Dementia
• Huntington disease (HD) is a genetic,
autosomal dominant, neurodegenerative
disorder characterized clinically by disorders
of movement, progressive dementia, and
psychiatric and/or behavioral disturbance.
47. Dementia Due to Other General
Medical Conditions
• A number of other general medical conditions
can cause dementia.
• Some of these include endocrine conditions (e.g.,
hypoglycemia, hypothyroidism), pulmonary
disease, hepatic or renal failure, cardiopulmonary
insufficiency, fluid and electrolyte imbalances,
nutritional deficiencies, frontal or temporal lobe
lesions, central nervous system (CNS) or systemic
infections, uncontrolled epilepsy, and other
neurological conditions such as multiple sclerosis.
48. Substance-Induced Persisting
Dementia.
• This type of dementia is related to the
persisting effects of substances such as
alcohol, inhalants, sedatives, hypnotics,
anxiolytics, other medications, and
environmental toxins.
• The term “persisting” is used to indicate that
the dementia persists long after the effects of
substance intoxication or substance
withdrawal have subsided.
49. Causes of Dementia
• Degeneration of nerve cells
• Parkinson’s disease
• Huntington’s disease
• Infection like HIV, syphilis
• Toxic cause eg. Alcohol, carbon monoxide
• Trauma, stroke
50. Signs and symptoms of Dementia
• Memory losses.
• Impaired abstraction and planning
• Language and comprehension disturbances.
• Poor judgment.
• Impaired orientation ability
• Decreased attention and increased restlessness.
• Behavioral changes and psychosis.
• Wandering
• Personality Changes
51. • Impaired ability to perform motor activities
despite intact motor abilities (apraxia).
• Impairment in language ability, such as
difficulty naming objects. In some instances,
the individual may not speak at all (aphasia).
52. Additional symptoms
1. Emotional lability (marked variation in emotional
expression).
2. Catastrophic reaction (when confronted with an
assignment which is beyond the residual intellectual
capacity, patient may go into a sudden rage).
3. Thought abnormalities, e.g. perseveration,
delusions.
4. Urinary and faecal incontinence may develop in
later stages.
5. Neurological signs may or may not be present,
depending on the underlying cause.
53. Diagnosis
• Medical History
• Basic Medical Test
• Neurological Reflexes- reflexes, co-ordination
and balance, muscle tone and strength, eye
movement, speech and sensation.
• Brain imaging- EEG, CT, PECT
• Psychiatric Evaluation- Mental status
examination, mini mental status examination
55. Other Management
–Reduce environmental Confusion by-
• Approaching patient in pleasant and calm
way.
• Keep the environment simple and pleasing,
remove unwanted utensils and furniture.
• Maintain regular daily living schedule.
• Provide memory device like – list of
activities, reminding notes, label on items
etc.
56. • Increased environment cues
–Address patient by name to facilitate
orientation of self.
–Offer environmental cues to offer
orientation of time, place and person.
57. • Monitor medication regimen
– Administer drug at appropriate time and dose,
should not leave the medicine by patient’s side.
• Monitor temperature of food
– Patient is protected from burning self by warm
food.
58. –Assist in self care activities of patient as
required.
–Provide adequate rest and sleep
–Encourage visit from family and friends.
– Provide assistive device like glass, hearing
aid, walker etc. if needed.
–Caring a dementic patient is a burdensome
task for family members too, provide
support to the career too.
59. • Reminiscence Therapy
– Reminiscence therapy is defined by the American
Psychological ) as "the use of life histories –
written, oral, or both – to improve psychological
well-being.
– This form of therapeutic intervention respects the
life and experiences of the individual with the aim
to help the patient maintain good mental health.
60. • People with dementia often have difficulty
remembering what’s recently happened in
their lives.
• This can leave them feeling confused,
vulnerable and less confident.
• However, their memories from years ago often
remain detailed and intact.
61.
62. Nursing Management
• Nursing Assessment
– Assess the key areas in history, MSE and MMSE to
get probable symptoms and defect.
63. Nursing Diagnosis
• Risk for trauma
• Risk for self directed or Other directed
violence
• Chronic Confusion
• Self Care Deficit
• Disturbed Sensory Perception
• Low self Esteem
• Care giver role strain
65. Nursing Intervention
• Assess client’s level of disorientation and confusion
to determine specifi c requirements for safety.
• Institute appropriate safety measures, such as the
following:
a. Place furniture in room in an arrangement that
best accommodates client’s disabilities.
b. Observe client behaviors frequently; assign staff on
one to- one basis if condition warrants; accompany
and assist client when ambulating; use wheelchair for
transporting long distances.
66. c. Store items that client uses frequently within easy
access.
d. Remove potentially harmful articles from client’s
room: cigarettes, matches, lighters, sharp objects.
e. Remain with client while he or she smokes.
f. Pad side rails and headboard of client with seizure
disorder. Institute seizure precautions as described
in procedure manual of individual institution.
g. If client is prone to wander, provide an area
within which wandering can be carried out safely.
67. • Frequently orient client to reality and
surroundings.
• Use tranquilizing medications and soft
restraints, as prescribed by physician, for
client’s protection during periods of excessive
hyperactivity.
68. For decreasing violence
• Assess client’s level of anxiety and behaviors
that indicate the anxiety is increasing.
• Maintain low level of stimuli in client’s
environment (low lighting, few people, simple
decor, low noise level).
• Remove all potentially dangerous objects from
client’s environment.
• Have sufficient staff available to execute a
physical confrontation, if necessary.
69. • Use tranquilizing medications and soft
restraints, as prescribed by physician.
• Sit with client and provide one-to-one
observation if assessed to be actively suicidal.