Oral epithelial tumor
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The document describes various oral lesions including papillomas, warts, condyloma, focal epithelial hyperplasia, carcinoma in situ extending into salivary ducts, several cases of squamous cell carcinoma in various oral locations, verrucous carcinoma, erythroleukoplakia, leukoplakia, epithelial dysplasia, ductal dysplasia, and basal cell carcinoma. Photomicrographs show features of many of these lesions at both low and high magnification.
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Mucocele and Renula
Mucocele and ranula are lesions caused by the extravasation of mucus from salivary glands into surrounding tissues. Mucoceles are commonly caused by trauma that severs or obstructs salivary ducts, allowing mucus to pool in surrounding tissues. Ranulas specifically occur on the floor of the mouth associated with sublingual or submandibular gland ducts. Histologically, they consist of mucus-filled cavities surrounded by granulation tissue and inflammatory cells. Treatment involves complete surgical excision to prevent recurrence.
Cysts of oral and maxillofacial region by dr. maryam salman
this lecture is about etiology , diagnosis (histopathological and clinical diagnosis) and management of cysts of oral and maxillofacial region
Cystic lesions in oral cavity
Based on the information provided, the key differentials would be:
- Radicular cyst: Most common cyst in jaws, associated with non-vital tooth. Location and association with tooth fits.
- Dentigerous cyst: Second most common, associated with crown of unerupted tooth. Location fits.
- Odontogenic keratocyst: Aggressive cyst, often multilocular radiolucency. Less likely based on description.
- Aneurysmal bone cyst: Often multilocular "soap bubble" appearance. Less likely based on description.
- Traumatic bone cyst: Often interradicular in location. Possible based on location described.
Further investigation with tooth
Odontogenic cysts
A cyst is an epithelium-lined sac containing fluid or semisolid material. In the formation of a cyst, the epithelial cells first proliferate and later undergo degeneration and liquefaction. The liquefied material exerts equal pressure on the walls of the cyst from within. Cysts grow by expansion and thus displace the adjacent teeth by pressure. May can produce expansion of the cortical bone. On a radiograph, the radiolucency of a cyst is usually bordered by a radiopaque periphery of dense sclerotic bone. The radiolucency may be unilocular or multilocular. Odontogenic cysts are those which arise from the epithelium associated with the development of teeth. The source of epithelium is from the enamel organ, the reduced enamel epithelium, the cell rests of Malassez or the remnants of the dental lamina.
Salivary gland tumors
This document provides an overview of salivary gland tumors. It discusses that salivary gland tumors are heterogeneous and most are benign. The majority originate in the parotid glands. Pleomorphic adenoma is the most common benign tumor and occurs most often in the parotid glands. The document describes the histopathology and classification of various salivary gland tumors including pleomorphic adenoma, adenoid cystic carcinoma, and mucoepidermoid carcinoma. It also discusses the genetics and hypothesized cells of origin for different salivary gland neoplasms.
Mucoepidermoid carcinoma
Mucoepidermoid carcinoma is a malignant glandular epithelial tumor that contains both epidermoid and mucus-secreting cells. It most commonly arises in the parotid gland but can also affect minor salivary glands. Histologically, it is graded based on the degree of cyst formation, cellular atypia, and relative numbers of the three cell types. Low grade tumors have many mucus cells and cysts while high grade tumors are more solid with necrosis and invasion. Treatment involves surgical excision with radiation and chemotherapy possibly added for advanced or high grade disease. Prognosis depends on grade, with low grade having over 90% 5-year survival versus intermediate and high grade around 50%.
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The document discusses Mucoepidermoid carcinoma, which is the most common malignant salivary gland tumor. It arises from the ductal system of major and minor salivary glands. The document describes the clinical features, histopathological features, grading, variants, differential diagnosis and management of Mucoepidermoid carcinoma. It is graded as low, intermediate or high grade based on histopathological characteristics like presence of cystic spaces, cellular atypia and proportion of cell types. Low grade tumors have a better prognosis compared to intermediate and high grade tumors.Tumor of oral cavity
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Mucocele and ranula are lesions caused by the extravasation of mucus from salivary glands into surrounding tissues. Mucoceles are commonly caused by trauma that severs or obstructs salivary ducts, allowing mucus to pool in surrounding tissues. Ranulas specifically occur on the floor of the mouth associated with sublingual or submandibular gland ducts. Histologically, they consist of mucus-filled cavities surrounded by granulation tissue and inflammatory cells. Treatment involves complete surgical excision to prevent recurrence.
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Cystic lesions in oral cavity
Based on the information provided, the key differentials would be:
- Radicular cyst: Most common cyst in jaws, associated with non-vital tooth. Location and association with tooth fits.
- Dentigerous cyst: Second most common, associated with crown of unerupted tooth. Location fits.
- Odontogenic keratocyst: Aggressive cyst, often multilocular radiolucency. Less likely based on description.
- Aneurysmal bone cyst: Often multilocular "soap bubble" appearance. Less likely based on description.
- Traumatic bone cyst: Often interradicular in location. Possible based on location described.
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Odontogenic cysts
A cyst is an epithelium-lined sac containing fluid or semisolid material. In the formation of a cyst, the epithelial cells first proliferate and later undergo degeneration and liquefaction. The liquefied material exerts equal pressure on the walls of the cyst from within. Cysts grow by expansion and thus displace the adjacent teeth by pressure. May can produce expansion of the cortical bone. On a radiograph, the radiolucency of a cyst is usually bordered by a radiopaque periphery of dense sclerotic bone. The radiolucency may be unilocular or multilocular. Odontogenic cysts are those which arise from the epithelium associated with the development of teeth. The source of epithelium is from the enamel organ, the reduced enamel epithelium, the cell rests of Malassez or the remnants of the dental lamina.
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This document provides an overview of salivary gland tumors. It discusses that salivary gland tumors are heterogeneous and most are benign. The majority originate in the parotid glands. Pleomorphic adenoma is the most common benign tumor and occurs most often in the parotid glands. The document describes the histopathology and classification of various salivary gland tumors including pleomorphic adenoma, adenoid cystic carcinoma, and mucoepidermoid carcinoma. It also discusses the genetics and hypothesized cells of origin for different salivary gland neoplasms.
Mucoepidermoid carcinoma
Mucoepidermoid carcinoma is a malignant glandular epithelial tumor that contains both epidermoid and mucus-secreting cells. It most commonly arises in the parotid gland but can also affect minor salivary glands. Histologically, it is graded based on the degree of cyst formation, cellular atypia, and relative numbers of the three cell types. Low grade tumors have many mucus cells and cysts while high grade tumors are more solid with necrosis and invasion. Treatment involves surgical excision with radiation and chemotherapy possibly added for advanced or high grade disease. Prognosis depends on grade, with low grade having over 90% 5-year survival versus intermediate and high grade around 50%.
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Ameloblastoma
Dr. Ahmed M. Adawy, Professor Emeritus, Dep. Oral & Maxillofacial Surgery. Former Dean, Faculty of Dental Medicine, Al-Azhar University. Ameloblastoma is benign slow-growing but locally invasive neoplasm of odontogenic origin. In 2005, the WHO has classified ameloblastomas into multi cystic, unicystic and peripheral subtypes. The clinical picture, radiographic findings and differential diagnosis are presented. Treatment of ameloblastomas is primarily surgical. There has been some debate regarding the most appropriate method for removing. These range from conservative to radical modes. Some authors advocate conservative approach and thought that ameloblastoma are essentially benign in nature and should be treated as such. However, this conservative approach result in recurrence rates of 55% to 90%of the cases. Currently, the standard of care for ameloblastoma includes en bloc resection with 1-2 combine margin and immediate bone reconstruction. Despite the medical nature of a surgical resection, it may actually involve less morbidity than extensive hard and soft tissue resection with associated extensive morbidity that may be warranted in case of recurrence following inadequate primary treatment.
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Lect.2. salivary gland pathology
Here are the answers to your questions:
1. Common types of salivary gland benign tumors with origin of each:
- Pleomorphic adenoma - originates from the intercalated duct cells and myoepithelial cells.
- Oncocytic tumors - originate from the striated duct cells.
- Acinous cell tumors - originate from the acinar cells.
- Mucoepidermoid tumors and squamous cell carcinomas develop in the excretory duct cells.
2. The histological features of mucoepidermoid carcinoma include:
- Containing mucin-producing cells and epithelial cells of the epidermoid variety.
- Divided into
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There are several types of odontogenic cysts that can develop:
1) A periapical cyst develops from a periapical granuloma caused by degradation products from a nonvital tooth.
2) A residual cyst can develop months to years after a tooth extraction if the cyst lining is incompletely removed.
3) Dentigerous cysts develop from proliferation of the enamel organ remnant surrounding an unerupted tooth.
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This document discusses various fungal infections that can affect the oral cavity and respiratory tract, including candidiasis, aspergillosis, and zygomycosis. It describes the characteristics and morphology of fungi, including molds, yeasts, dimorphic fungi. It then covers the pathogenesis, clinical manifestations, diagnosis and treatment of the main opportunistic fungal infections.
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This document discusses several vesiculobullous diseases including pemphigus vulgaris, pemphigoid, erythema multiforme, and epidermolysis bullosa. Pemphigus vulgaris is an autoimmune disease causing fluid-filled blisters and ulcers in the mouth and skin. Pemphigoid includes bullous pemphigoid and cicatricial pemphigoid, which are autoimmune diseases affecting the elderly that cause subepithelial blistering. Erythema multiforme is often triggered by infection and causes red macules and mouth sores. Epidermolysis bullosa is a group of genetic disorders that results in easy blist
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- Precancerous lesions of the oral cavity include premalignant lesions like leukoplakia, erythroplakia, oral submucous fibrosis, and lichen planus as well as premalignant conditions.
- Leukoplakia presents as a white patch that cannot be scraped off. Erythroplakia appears as a bright red patch. Oral submucous fibrosis causes stiffness of the oral mucosa and trismus.
- Risk factors include tobacco use, betel nut chewing, and poor oral hygiene. Histopathological examination is needed for diagnosis. Management involves eliminating risk factors, surgical excision of high risk lesions, and close follow up
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This document discusses two types of non-epithelial tumours of the oral cavity: peripheral giant cell granuloma and central giant cell granuloma. Peripheral giant cell granuloma originates from the periodontal membrane or alveolar bone and presents as a soft tissue nodule composed of multinucleated giant cells. Central giant cell granuloma is a rare, benign, intraosseous lesion most commonly found in the mandible of young people that causes expansion of the bone and resorption of tooth roots. Both lesions contain proliferation of multinucleated giant cells and other cells and may require surgical excision, with central giant cell granuloma having a higher rate of recurrence.
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Odontogeniccysts OKC
This document discusses odontogenic keratocysts (OKCs), a type of jaw cyst. It covers the classification, causes, histopathology, clinical features, radiographic features, differential diagnosis, treatment principles, and surgical treatment options for OKCs. OKCs most commonly occur in the mandibular molar and ramus areas and are often radiolucent and multilocular in appearance on radiographs. Treatment options include wide surgical excision or marsupialization to prevent recurrence of these cysts which have a high rate of recurrence compared to other jaw cysts.
Fissural cysts of oral cavity
Fissural cysts arise along lines of fusion between embryonic processes. Nasopalatine duct cysts are the most common non-odontogenic cyst, arising from epithelial remnants of the nasopalatine duct. Median palatal cysts occur in the midline of the hard palate from entrapped epithelium. Dermoid and epidermoid cysts contain skin elements and arise from implantation of epithelium during embryonic development. These cysts are examined clinically and radiographically and often surgically removed.
ODONTOMA
This document discusses odontomas, which are benign developmental malformations of dental tissues that have the basic histological structure of teeth but in a disorganized pattern. Odontomas are classified as compound or complex. Compound odontomas consist of separate small tooth-like structures, while complex odontomas are irregular hard and soft dental tissues showing no tooth resemblance. Odontomas typically present in people aged 10-20 as painless swellings and can cause complications like eruption issues or cyst formation if not removed by conservative surgery.
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This document discusses various types of pseudocysts and true cysts found in the body. It begins by defining the key differences between a true cyst and a pseudocyst. Pseudocysts are lined by granulation and/or fibrous tissue rather than an epithelial cell layer. The document then classifies and describes different types of pseudocysts including traumatic bone cysts, aneurysmal bone cysts, and developmental cysts. It provides details on the etiology, location, patient demographics, clinical presentation, radiographic features, histopathology and treatment for each type.
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This document discusses benign epithelial tumors including squamous papilloma, squamous acanthoma, and keratocanthoma. It provides details on their classification, clinical features, histology, treatment and prognosis. Squamous papilloma is associated with HPV viruses and presents as a pink, papillary growth. Squamous acanthoma is a reactive lesion with thickened orthokeratin. Keratocanthoma appears as a crateriform nodule that heals within months. The document also covers oral nevi, noting their histologic subtypes and benign nature.
Odontogenic tumours part 1
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This document summarizes different types of jaw swellings and their characteristics. It begins by classifying jaw swellings based on their origin, such as from tooth germs, mucoperiosteum, or being osseous, inflammatory, or malignant. Specific types of swellings arising from these origins are then described in more detail such as epulis, odontogenic tumors, fibrous dysplasia, Paget's disease, and osteomyelitis. Clinical features, radiographic findings, histological characteristics, and treatment approaches are provided for many of these conditions. In particular, ameloblastoma, a benign odontogenic tumor, is discussed in depth covering its pathogenesis, clinical presentation, classification, management,
epithelial lesions
This document discusses oral squamous cell carcinoma:
- It is one of the most common oral cancers, arising from dysplastic epithelial cells and characterized by invasive islands of malignant squamous cells.
- Risk factors include tobacco, alcohol, HPV infection and poor nutrition. Clinically, it presents as ulcers or masses that are often painless.
- If left untreated, it can spread to lymph nodes and distant sites like lungs. Staging evaluates tumor size and spread. Treatment involves surgery, radiation and chemotherapy. Prognosis depends on stage at diagnosis.
premalignant lesions new.pptx
This document discusses numerous preneoplastic and neoplastic lesions across multiple organ systems. Preneoplastic lesions involve abnormal cells with an increased cancer risk, while neoplastic lesions are cancerous or precancerous. Examples mentioned include actinic keratosis and Bowen's disease of the skin; oral leukoplakia; Barrett's esophagus; colonic adenomas; cirrhosis-associated liver cell changes; asbestos-related lung changes; various breast lesions; cervical intraepithelial neoplasia; penile intraepithelial neoplasia; prostatic intraepithelial neoplasia; and monoclonal gammopathy of undetermined significance, which can progress to myeloma.
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This document presents a case study of a 30-year-old female patient diagnosed with plexiform ameloblastoma based on a biopsy of her right mandible. It provides background on ameloblastoma, describing it as a benign odontogenic tumor arising from odontogenic epithelium. It discusses the various histological subtypes including follicular, plexiform, unicystic (three groups), peripheral, acanthomatous, granular, basal cell, and desmoplastic. Treatment typically involves radical excision, with curettage having the highest recurrence rates. The case study aims to educate on the clinical, radiographic, and histological features of ameloblastoma.
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This document discusses metastatic tumors of the jaws. It notes that metastases account for 1-1.5% of all malignant tumors, with the most common primary sites being the lungs, breast, kidney, and bone. The Batson's plexus allows tumor cells to bypass the lungs and spread to the head and neck region. Metastases most commonly occur in the mandible and attached gingiva. Clinical features can include pain, swelling, tooth mobility, and pathological fracture. Radiographs may show bone resorption or a moth-eaten appearance. Histopathology is needed for diagnosis, but there can be challenges distinguishing primary from metastatic tumors. Investigations like imaging help determine the primary site and plan treatment.
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Dr. Ahmed M. Adawy, Professor Emeritus, Dep. Oral & Maxillofacial Surgery. Former Dean, Faculty of Dental Medicine, Al-Azhar University. Ameloblastoma is benign slow-growing but locally invasive neoplasm of odontogenic origin. In 2005, the WHO has classified ameloblastomas into multi cystic, unicystic and peripheral subtypes. The clinical picture, radiographic findings and differential diagnosis are presented. Treatment of ameloblastomas is primarily surgical. There has been some debate regarding the most appropriate method for removing. These range from conservative to radical modes. Some authors advocate conservative approach and thought that ameloblastoma are essentially benign in nature and should be treated as such. However, this conservative approach result in recurrence rates of 55% to 90%of the cases. Currently, the standard of care for ameloblastoma includes en bloc resection with 1-2 combine margin and immediate bone reconstruction. Despite the medical nature of a surgical resection, it may actually involve less morbidity than extensive hard and soft tissue resection with associated extensive morbidity that may be warranted in case of recurrence following inadequate primary treatment.
Odontogenic tumours
This document summarizes several odontogenic tumors including: ameloblastoma (benign, locally aggressive tumor of odontogenic epithelium that commonly occurs in the mandible), adenomatoid odontogenic tumor (uncommon, nonaggressive tumor that often surrounds an unerupted tooth), calcifying epithelial odontogenic tumor (rare benign neoplasm that usually occurs in the mandible), odontoma (tumor characterized by production of dental tissues that commonly occurs in young patients), ameloblastic fibroma (benign mixed odontogenic tumor that occurs in children/adolescents), odontogenic myxoma (benign tumor arising from dental papilla mesenchyme), cementoblast
Lect.2. salivary gland pathology
Here are the answers to your questions:
1. Common types of salivary gland benign tumors with origin of each:
- Pleomorphic adenoma - originates from the intercalated duct cells and myoepithelial cells.
- Oncocytic tumors - originate from the striated duct cells.
- Acinous cell tumors - originate from the acinar cells.
- Mucoepidermoid tumors and squamous cell carcinomas develop in the excretory duct cells.
2. The histological features of mucoepidermoid carcinoma include:
- Containing mucin-producing cells and epithelial cells of the epidermoid variety.
- Divided into
Oral manifestations of blood disorders
This document summarizes various blood disorders and their oral manifestations. It discusses disorders of red blood cells like iron-deficiency anemia, megaloblastic anemia, pernicious anemia, and sickle cell anemia. It also covers disorders of white blood cells such as leukemia and leukopenia. For each condition, it describes the causes, clinical features, diagnosis, and potential oral signs including gingival bleeding, ulcers, and infections. In general, these blood disorders can cause oral pallor, infections, and changes in taste or tooth development.
Causes of odontogenic cyst
There are several types of odontogenic cysts that can develop:
1) A periapical cyst develops from a periapical granuloma caused by degradation products from a nonvital tooth.
2) A residual cyst can develop months to years after a tooth extraction if the cyst lining is incompletely removed.
3) Dentigerous cysts develop from proliferation of the enamel organ remnant surrounding an unerupted tooth.
Basal cell carcinoma of oral cavity
This document describes basal cell carcinoma (BCC), the most common type of skin cancer. It develops from stem cells in the basal layer of the epidermis or hair follicles. The main cause is ultraviolet radiation from sun exposure. BCC usually appears as a raised bump on sun-exposed skin and grows slowly, rarely spreading to other areas. If left untreated, it can cause significant tissue damage. The document outlines the clinical features and subtypes of BCC, including nodular, pigmented, cystic, superficial, micronodular, and infiltrating types. Histopathological analysis is used to diagnose and characterize BCC subtypes based on tissue appearance. Surgical excision or radiation therapy can effectively treat
Fungal infections of the oral cavity
This document discusses various fungal infections that can affect the oral cavity and respiratory tract, including candidiasis, aspergillosis, and zygomycosis. It describes the characteristics and morphology of fungi, including molds, yeasts, dimorphic fungi. It then covers the pathogenesis, clinical manifestations, diagnosis and treatment of the main opportunistic fungal infections.
Vesiculobullous diseases
This document discusses several vesiculobullous diseases including pemphigus vulgaris, pemphigoid, erythema multiforme, and epidermolysis bullosa. Pemphigus vulgaris is an autoimmune disease causing fluid-filled blisters and ulcers in the mouth and skin. Pemphigoid includes bullous pemphigoid and cicatricial pemphigoid, which are autoimmune diseases affecting the elderly that cause subepithelial blistering. Erythema multiforme is often triggered by infection and causes red macules and mouth sores. Epidermolysis bullosa is a group of genetic disorders that results in easy blist
Precancerous lesions of oral cavity
- Precancerous lesions of the oral cavity include premalignant lesions like leukoplakia, erythroplakia, oral submucous fibrosis, and lichen planus as well as premalignant conditions.
- Leukoplakia presents as a white patch that cannot be scraped off. Erythroplakia appears as a bright red patch. Oral submucous fibrosis causes stiffness of the oral mucosa and trismus.
- Risk factors include tobacco use, betel nut chewing, and poor oral hygiene. Histopathological examination is needed for diagnosis. Management involves eliminating risk factors, surgical excision of high risk lesions, and close follow up
Peripheral and central giant cell granuloma
This document discusses two types of non-epithelial tumours of the oral cavity: peripheral giant cell granuloma and central giant cell granuloma. Peripheral giant cell granuloma originates from the periodontal membrane or alveolar bone and presents as a soft tissue nodule composed of multinucleated giant cells. Central giant cell granuloma is a rare, benign, intraosseous lesion most commonly found in the mandible of young people that causes expansion of the bone and resorption of tooth roots. Both lesions contain proliferation of multinucleated giant cells and other cells and may require surgical excision, with central giant cell granuloma having a higher rate of recurrence.
Acute apical-periodontitis.25.mar.2013
Acute apical periodontitis is an inflammatory condition caused by a necrotic pulp that results in inflammation around the root apex. It is characterized by excruciating pain, a necrotic pulp, and thickening of the periodontal ligament space visible on radiographs. Treatment requires removal of the pulp or extraction of the tooth. While bacteria normally cause apical periodontitis, it can also arise from debris forced into tissues during root canal treatment. Symptoms include localized pain that can be provoked by tapping on the tooth. The condition requires extraction or root canal treatment to remove the necrotic pulp causing the inflammation.
Odontogeniccysts OKC
This document discusses odontogenic keratocysts (OKCs), a type of jaw cyst. It covers the classification, causes, histopathology, clinical features, radiographic features, differential diagnosis, treatment principles, and surgical treatment options for OKCs. OKCs most commonly occur in the mandibular molar and ramus areas and are often radiolucent and multilocular in appearance on radiographs. Treatment options include wide surgical excision or marsupialization to prevent recurrence of these cysts which have a high rate of recurrence compared to other jaw cysts.
Fissural cysts of oral cavity
Fissural cysts arise along lines of fusion between embryonic processes. Nasopalatine duct cysts are the most common non-odontogenic cyst, arising from epithelial remnants of the nasopalatine duct. Median palatal cysts occur in the midline of the hard palate from entrapped epithelium. Dermoid and epidermoid cysts contain skin elements and arise from implantation of epithelium during embryonic development. These cysts are examined clinically and radiographically and often surgically removed.
ODONTOMA
This document discusses odontomas, which are benign developmental malformations of dental tissues that have the basic histological structure of teeth but in a disorganized pattern. Odontomas are classified as compound or complex. Compound odontomas consist of separate small tooth-like structures, while complex odontomas are irregular hard and soft dental tissues showing no tooth resemblance. Odontomas typically present in people aged 10-20 as painless swellings and can cause complications like eruption issues or cyst formation if not removed by conservative surgery.
Pseudo cyst
This document discusses various types of pseudocysts and true cysts found in the body. It begins by defining the key differences between a true cyst and a pseudocyst. Pseudocysts are lined by granulation and/or fibrous tissue rather than an epithelial cell layer. The document then classifies and describes different types of pseudocysts including traumatic bone cysts, aneurysmal bone cysts, and developmental cysts. It provides details on the etiology, location, patient demographics, clinical presentation, radiographic features, histopathology and treatment for each type.
BENIGN TUMORS OF EPITHELIAL ORIGIN
This document discusses benign epithelial tumors including squamous papilloma, squamous acanthoma, and keratocanthoma. It provides details on their classification, clinical features, histology, treatment and prognosis. Squamous papilloma is associated with HPV viruses and presents as a pink, papillary growth. Squamous acanthoma is a reactive lesion with thickened orthokeratin. Keratocanthoma appears as a crateriform nodule that heals within months. The document also covers oral nevi, noting their histologic subtypes and benign nature.
Odontogenic tumours part 1
Dr. Ali Tahir discusses the classification, clinical features, histopathology, and treatment of ameloblastoma, which is the most common odontogenic tumor. Ameloblastoma is a locally aggressive, epithelial benign odontogenic neoplasm that resembles the enamel organ. It has several subtypes including multicystic/solid, unicystic, and peripheral. The multicystic type is the most common and presents as multilocular radiolucencies resembling a "soap bubble" or "honeycomb" pattern. Histologically it shows islands of epithelial cells resembling the enamel organ. Treatment involves resection with adequate margins to prevent recurrence.
Swellings of the jaw
This document summarizes different types of jaw swellings and their characteristics. It begins by classifying jaw swellings based on their origin, such as from tooth germs, mucoperiosteum, or being osseous, inflammatory, or malignant. Specific types of swellings arising from these origins are then described in more detail such as epulis, odontogenic tumors, fibrous dysplasia, Paget's disease, and osteomyelitis. Clinical features, radiographic findings, histological characteristics, and treatment approaches are provided for many of these conditions. In particular, ameloblastoma, a benign odontogenic tumor, is discussed in depth covering its pathogenesis, clinical presentation, classification, management,
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- Risk factors include tobacco, alcohol, HPV infection and poor nutrition. Clinically, it presents as ulcers or masses that are often painless.
- If left untreated, it can spread to lymph nodes and distant sites like lungs. Staging evaluates tumor size and spread. Treatment involves surgery, radiation and chemotherapy. Prognosis depends on stage at diagnosis.
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This document discusses numerous preneoplastic and neoplastic lesions across multiple organ systems. Preneoplastic lesions involve abnormal cells with an increased cancer risk, while neoplastic lesions are cancerous or precancerous. Examples mentioned include actinic keratosis and Bowen's disease of the skin; oral leukoplakia; Barrett's esophagus; colonic adenomas; cirrhosis-associated liver cell changes; asbestos-related lung changes; various breast lesions; cervical intraepithelial neoplasia; penile intraepithelial neoplasia; prostatic intraepithelial neoplasia; and monoclonal gammopathy of undetermined significance, which can progress to myeloma.
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This document discusses various types of tumors that can occur in the maxillofacial area, including the lip, oral cavity, tongue, and salivary glands. It provides details on the prevalence, risk factors, clinical forms, diagnosis, and treatment of cancers in these areas. Specifically, it notes that lip cancer prevalence in Ukraine is 5 cases per 100,000 people. It also discusses pretumor diseases of the lip like leukoplakia and papilloma. For oral cavity cancer, it states the highest incidence rates are 24% in Odessa region and 5% in Ivano-Frankivsk region. Tongue cancer most often occurs in the middle of the tongue. Salivary gland tumors
SID tumors of maxillofacial areas .pdf
This document discusses tumors of the maxillofacial area, including lip cancer, oral cavity cancer, tongue cancer, and salivary gland cancer. It covers risk factors, clinical presentations, diagnostic procedures, pretumor diseases, classifications, and treatment approaches for each type of cancer. Lip cancer most commonly presents as exophytic papillary or verruciform growths. Oral cavity cancer frequently involves the tongue or oral floor. Salivary gland tumors usually originate in the parotid glands. Diagnosis involves biopsy and imaging. Treatment depends on cancer stage but commonly involves surgery, radiation therapy, and chemotherapy. Early diagnosis is important as cure rates for early stage cancers can be over 95%.
Histopathological variants of squamous cell carcinoma (Doctor Faris Alabeedi ...
Histopathological variants of squamous cell carcinoma (Doctor Faris Alabeedi ...Doctor Faris Alabeedi
This document summarizes the key histopathological variants of squamous cell carcinoma (SCC), including verrucous carcinoma, spindle cell carcinoma, adenosquamous carcinoma, adenoid SCC, pseudovascular adenoid SCC, and basaloid SCC. It provides diagnostic characteristics for each variant such as growth patterns, cell morphology, associations with other types of carcinoma, and prognosis. Images are also included to illustrate histological features of each variant.Salivary gland tumors.pptx
Salivary gland tumors can be benign or malignant. Benign tumors are more common and include pleomorphic adenomas, Warthin's tumors, and oncocytomas. These typically present as slow-growing masses in the parotid or submandibular glands. Malignant tumors are less common but can be diagnosed based on symptoms like rapid growth, pain, nerve palsies, or lymph node involvement. The most common sites for minor salivary gland tumors are the hard palate and other areas of the oral cavity. A thorough examination is needed to determine if a salivary gland tumor is benign or malignant.
Neoplasms of Salivary Glands
Salivary gland tumours are a relatively rare and morphologically diverse group of lesions. So here are slides containing information about salivary gland tumours with images.
Malignant epithelial tumors iii/prosthodontic courses
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0091-9248678078Salivary Gland Disorders
Childhood mumps, certain bacterial infections (for example, of the tonsils or teeth), and other diseases that are typically more common among adults (such as AIDS, Sjögren syndrome, diabetes mellitus, sarcoidosis, and bulimia) often cause swelling of the major salivary glands.
Tumors & tumor like conditions of nasal cavity
This document provides information on the classification, histology, and clinical features of various tumors of the nasal cavity and paranasal sinuses. It begins by describing the normal anatomy and histology of the nasal cavity. It then classifies benign and malignant tumors of the nasal cavity and paranasal sinuses into epithelial, neuroectodermal, and mesenchymal categories. Specific tumor types are further described in terms of their epidemiology, clinical presentation, gross and microscopic appearance, immunoprofile, and prognosis. These include schneiderian papilloma, squamous cell carcinoma, adenocarcinoma, undifferentiated carcinoma, hemangiomas, angiofibroma, and olfactory neuroblastoma
Lesions of oral cavity and salivary gland.
This document provides an overview of common lesions of the oral cavity and salivary glands, including inflammatory, preneoplastic, and neoplastic conditions. Key topics discussed include oral leukoplakia, squamous cell carcinoma, ameloblastoma, pleomorphic adenoma, Warthin's tumor, mucoepidermoid carcinoma, and adenoid cystic carcinoma. The document describes the etiology, pathogenesis, morphology, and clinical manifestations of these various oral and salivary gland lesions.
Histology of Oral Cancer
This document summarizes several variants of oral squamous cell carcinoma (SCC), including verrucous carcinoma, basaloid SCC, and sarcomatoid carcinoma. It describes the histological features and sites of involvement for each variant. Key microscopic findings are outlined, such as the prominent keratin layer and pushing margins seen in verrucous carcinoma. Diagnostic criteria and differential diagnoses for each variant are also provided. The document additionally reviews adenocarcinoma subtypes seen in the oral cavity and describes patterns of bone and perineural invasion associated with oral cancers.
Tumors of salivary gland
This document provides information on various types of salivary gland tumors. It begins with an overview of the major and minor salivary gland tumors that are most commonly seen in adults and children. Examples include Warthin tumor, acinic cell carcinoma, and polymorphous low-grade adenocarcinoma. The document then describes the histological features of many benign and malignant epithelial and nonepithelial salivary gland tumors at both the macroscopic and microscopic levels. Key diagnostic characteristics of tumors like pleomorphic adenoma, myoepithelioma, oncocytoma, and adenoid cystic carcinoma are highlighted.
Benign sinonasal masses presentation & management-1
This document discusses various fibro-osseous tumors of the sinonasal region, including fibrous dysplasia. Fibrous dysplasia is a benign condition caused by a defect in osteoblast differentiation and maturation, resulting in the replacement of normal bone by fibrous connective tissue. It is caused by mutations in the GNAS1 gene. Histologically, there is slow replacement of medullary bone by abnormal fibrous tissue at different stages of bone metaplasia. Fibrous dysplasia typically presents in children and adolescents, with females being affected more often than males.
Dr samreen younas
This document discusses salivary gland tumors. It begins with definitions of tumors and classifications of salivary glands and salivary gland tumors. It then covers the incidence, clinical features, histopathological features, and treatment plans for various benign and malignant salivary gland tumors. The document emphasizes that surgical resection is usually the primary treatment for salivary gland tumors, with adjuvant radiotherapy sometimes used as well.
Salivarygland neoplasm by numan h.k.d.e.t dental clg1
This document provides information on salivary gland neoplasms:
- It describes the anatomy and locations of the major salivary glands.
- The most common benign tumor is pleomorphic adenoma, while mucoepidermoid carcinoma is the most common malignant tumor.
- Other tumors discussed include adenolymphoma, oncocytoma, and adenoid cystic carcinoma.
- Surgical excision is the primary treatment for salivary gland tumors. Care must be taken during parotid surgery to preserve the facial nerve.
Salivarygland neoplasm by numan(h.k.d.e.t.dental clg)
This document provides information about salivary gland neoplasms. It discusses the anatomy of major and minor salivary glands. It describes different tumors that can occur in the parotid gland, submandibular gland and minor salivary glands. The most common benign tumor is pleomorphic adenoma, while mucoepidermoid carcinoma is the most common malignant tumor. Different tumors are classified as epithelial or connective tissue tumors. Clinical features, pathology, treatment and prognosis are outlined for various tumors including pleomorphic adenoma, adenolymphoma, oncocytoma and malignant tumors.
Tissue Diagnosis of Salivary Gland Tumors
This document discusses various tumors of the salivary glands. It begins by describing the major salivary glands and then covers topics such as pleomorphic adenoma, carcinoma ex pleomorphic adenoma, Warthin's tumor, mucoepidermoid carcinoma, adenoid cystic carcinoma, acinic cell carcinoma, non-Hodgkin's lymphoma, and squamous cell carcinoma. Key points are that pleomorphic adenoma is the most common tumor of the major salivary glands, adenoid cystic carcinoma is most common in the minor salivary glands, and mucoepidermoid carcinoma is the most common malignant tumor overall.
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Oral epithelial tumor
- 1. Papilloma, floor of mouth.
- 3. Papilloma
- 8. Oral wart.
- 9. wart
- 10. Oral wart.
- 11. Oral wart. Immunohistochemical stain for common human papillomavirus in an oral wart. Positive, brown-staining nuclei are seen in upper level keratinocytes.
- 13. Focal epithelial hyperplasia of the lip.
- 14. Focal epithelial hyperplasia of the buccal mucosa.
- 15. Biopsy specimen showing carcinoma in situ.
- 16. in situ carcinoma extending into salivary duct.
- 17. Squamous cell carcinoma. Small, crusted ulcer of the lower lip vermilion.
- 18. Squamous cell carcinoma. Ulcerated mass of the lower lip vermilion.
- 19. Squamous cell carcinoma of the gingiva.
- 20. Squamous cell carcinoma, floor of mouth.
- 21. Advanced squamous cell carcinoma of the posterior-lateral tongue.
- 22. Squamous cell carcinoma, gingiva.
- 23. Squamous cell carcinoma of the lip.
- 24. Exophytic squamous cell carcinoma of the lip.
- 25. Squamous cell carcinoma of the lateral tongue in a 34-year-old man.
- 26. Squamous cell carcinoma of the lateral tongue.
- 27. Squamous cell carcinoma of the ventral surface of the tongue.
- 28. Early squamous cell carcinoma of the floor of mouth.
- 29. Early squamous cell carcinoma of the floor of mouth.
- 30. Squamous cell carcinoma of the floor of mouth.
- 31. Squamous cell carcinoma of the floor of mouth.
- 32. Second primary squamous cell carcinoma of the palate in a 34-year-old man.
- 33. Metastasis of squamous cell carcinoma of the tongue to a submandibular lymph node.
- 34. Squamous ce ll carcinoma. Metastatic deposits within cervical lymph nodes present as firm, painless enlargements as seen in this patient with metastasis to a superior jugular node froma posterior lateral tongue carcinoma.
- 35. Well-differentiated squamous cell carcinoma. l ow-power photomicrograph showing islands of malignant squamous epithelium invading into the lamina properia.
- 37. Well-differentiated squamous cell carcinoma. High-power view showing dysplastic epithelial cells with keratin pearl formation.
- 40. Moderately differentiated squamous cell carcinoma. Although no keratinization is seen in this medium-power view, these malignant cells are still easily recognizable as being of squamous epithelial origin.
- 43. Poorly differentiated squamous cell carcinoma. The numerous pleomorphic cells within the lamina properia represent anaplastic carcinoma.
- 48. Verru cous carcinoma. Extensive papillary, white lesion of the maxillary vestibule.
- 49. Verrucous carcinoma. Large, exophytic, papillary mass of the maxillary alveolar ridge.
- 50. Verrucous carcinoma. Low-power photomicrograph showing marked epithelial hyperplasia with a rough, papillary surface and keratin plugging.
- 51. Verrucouscarcinoma. High-power view showing bulbous rete ridges without significant dysplasia.
- 53. Erythroleukoplakia. Mixed red-an d-white lesion of the lateral borde r of the tongue. Biopsy revealed carcinoma in situ.
- 54. l eu koplakia. Extensive ventra l and lateral tongue lesion containing multiple areas representi ng various possible pha ses or clinical appearances
- 55. Moderate epithelial dysplasia. Dysplast ic changes extend to the midpo int of the ep ithelium and are character ized by nuclear hyperchromatism. pleomorphism, and ce llular crowding.
- 58. Ductal dysplasia. Salivary gland duct exhibiting squamous metaplasia and dysplasia t hat originated from an overlying surface epit helial dysplasia
- 60. Basal cell carcinoma. Noduloulcerat ive lesion of the upper lip demonstrating telangiectasia and small ulceration.
- 62. Basal cell carcinoma. Pigmented basal cell carcinoma of the cheek.
- 64. Basal cell carcinoma. Note solid tumor (left) and nested tumor (right).
- 65. Basal cell carcinom a. low- power photomicrograph showing ulceration of the epidermal surface associated with an invading tumor of hyperchromatic epithelial cells. Inset demonstratesislands of basophilic epithelium with peripheral palisading.