This document provides guidance on performing a newborn examination. It discusses the importance, timing, precautions, and components of the examination. The examination assesses gestational age, vital signs, general appearance, and systems. It also evaluates neonatal reflexes. The goal is to establish a baseline and ensure the baby's healthy transition to extrauterine life.
Definition of neonatal sepsis,type of neonatal sepsis ,early onset neonatal sepsis,late onset neonatal sepsis,Pathophysiology of neonatal sepsis,,sign and symptoms of neonatal sepsis, diagnosis of neonatal sepsis,management of neonatal sepsis, antibiotic used for neonatal sepsis,prevention of neonatal sepsis, prognosis of neonatal sepsis ,and A summary
Definition of neonatal sepsis,type of neonatal sepsis ,early onset neonatal sepsis,late onset neonatal sepsis,Pathophysiology of neonatal sepsis,,sign and symptoms of neonatal sepsis, diagnosis of neonatal sepsis,management of neonatal sepsis, antibiotic used for neonatal sepsis,prevention of neonatal sepsis, prognosis of neonatal sepsis ,and A summary
Febrile seizure / Pediatrics
Simple vs. Complex seizure
Possible explanation of febrile seizure
Risk Factors for Febrile Seizures
Risk Factors for Recurrence of Febrile Seizure
Risk Factors for Occurrence of Subsequent Epilepsy After a Febrile Seizure
Genetic Factors
Evaluation
Lumbar Puncture
Optional LP
Electroencephalogram
Blood Studies
Neuroimaging
TREATMENT
Febrile seizure / Pediatrics
Simple vs. Complex seizure
Possible explanation of febrile seizure
Risk Factors for Febrile Seizures
Risk Factors for Recurrence of Febrile Seizure
Risk Factors for Occurrence of Subsequent Epilepsy After a Febrile Seizure
Genetic Factors
Evaluation
Lumbar Puncture
Optional LP
Electroencephalogram
Blood Studies
Neuroimaging
TREATMENT
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New Directions in Targeted Therapeutic Approaches for Older Adults With Mantl...i3 Health
i3 Health is pleased to make the speaker slides from this activity available for use as a non-accredited self-study or teaching resource.
This slide deck presented by Dr. Kami Maddocks, Professor-Clinical in the Division of Hematology and
Associate Division Director for Ambulatory Operations
The Ohio State University Comprehensive Cancer Center, will provide insight into new directions in targeted therapeutic approaches for older adults with mantle cell lymphoma.
STATEMENT OF NEED
Mantle cell lymphoma (MCL) is a rare, aggressive B-cell non-Hodgkin lymphoma (NHL) accounting for 5% to 7% of all lymphomas. Its prognosis ranges from indolent disease that does not require treatment for years to very aggressive disease, which is associated with poor survival (Silkenstedt et al, 2021). Typically, MCL is diagnosed at advanced stage and in older patients who cannot tolerate intensive therapy (NCCN, 2022). Although recent advances have slightly increased remission rates, recurrence and relapse remain very common, leading to a median overall survival between 3 and 6 years (LLS, 2021). Though there are several effective options, progress is still needed towards establishing an accepted frontline approach for MCL (Castellino et al, 2022). Treatment selection and management of MCL are complicated by the heterogeneity of prognosis, advanced age and comorbidities of patients, and lack of an established standard approach for treatment, making it vital that clinicians be familiar with the latest research and advances in this area. In this activity chaired by Michael Wang, MD, Professor in the Department of Lymphoma & Myeloma at MD Anderson Cancer Center, expert faculty will discuss prognostic factors informing treatment, the promising results of recent trials in new therapeutic approaches, and the implications of treatment resistance in therapeutic selection for MCL.
Target Audience
Hematology/oncology fellows, attending faculty, and other health care professionals involved in the treatment of patients with mantle cell lymphoma (MCL).
Learning Objectives
1.) Identify clinical and biological prognostic factors that can guide treatment decision making for older adults with MCL
2.) Evaluate emerging data on targeted therapeutic approaches for treatment-naive and relapsed/refractory MCL and their applicability to older adults
3.) Assess mechanisms of resistance to targeted therapies for MCL and their implications for treatment selection
Flu Vaccine Alert in Bangalore Karnatakaaddon Scans
As flu season approaches, health officials in Bangalore, Karnataka, are urging residents to get their flu vaccinations. The seasonal flu, while common, can lead to severe health complications, particularly for vulnerable populations such as young children, the elderly, and those with underlying health conditions.
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Residents are reminded that the flu vaccine is safe and effective. Common side effects are mild and may include soreness at the injection site, mild fever, or muscle aches. These side effects are generally short-lived and far less severe than the flu itself.
Healthcare providers are also stressing the importance of continuing COVID-19 precautions. Wearing masks, practicing good hand hygiene, and maintaining social distancing are still crucial, especially in crowded places.
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Are you curious about what’s new in cervical cancer research or unsure what the findings mean? Join Dr. Emily Ko, a gynecologic oncologist at Penn Medicine, to learn about the latest updates from the Society of Gynecologic Oncology (SGO) 2024 Annual Meeting on Women’s Cancer. Dr. Ko will discuss what the research presented at the conference means for you and answer your questions about the new developments.
These simplified slides by Dr. Sidra Arshad present an overview of the non-respiratory functions of the respiratory tract.
Learning objectives:
1. Enlist the non-respiratory functions of the respiratory tract
2. Briefly explain how these functions are carried out
3. Discuss the significance of dead space
4. Differentiate between minute ventilation and alveolar ventilation
5. Describe the cough and sneeze reflexes
Study Resources:
1. Chapter 39, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 34, Ganong’s Review of Medical Physiology, 26th edition
3. Chapter 17, Human Physiology by Lauralee Sherwood, 9th edition
4. Non-respiratory functions of the lungs https://academic.oup.com/bjaed/article/13/3/98/278874
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2. Indication and importance
Maternal and prenatal history
Precautions prior to examination
Vitals and anthropometry
Assessment of gestational age
General examination
Systemic examination
Neonatal reflexes
3. To assess the baby’s transition from intrauterine life
to extrauterine existence
Earliest possible detection of deviations
Establishes a baseline for subsequent
examinations
To assess the progress of the child
Parents assurance and counseling
4.
5. Immediately after birth
Detailed examination after 24hrs
At discharge and 24hrs after discharge
6. Hand washing
Thermal environment
Good source of light
Background noise
Brief examination time
7. Check the correct name,sex
Gestational age
• FULL TERM-between 37 to 42 weeks of gestation
• PRETERM-< 37 weeks
• POST TERM->42 weeks of gestation
• POST DATED-after EDD(>40 weeks of gestation)
8.
9. APGAR scoring done at 1min,5min and 10min after
birth
Value > 7 indicate the baby’s condition is good to
excellent
Value <4 necessitate continued resuscitation
APGAR score is a good predictor of survival but
using it to predict long-term outcome is
inappropriate.
11. Measure axillary temperature with tip of the
thermometer in the axilla.
Normal axillary temp is 36.5 to 37.4
Mild Hypothermia/cold stress – 36.0 to 36.4
Moderate hypothermia – 32 to 35.9
Severe hypothermia - <32
Hyperthermia - >37.5
12. Heart rate
Normal HR: 100-160/min(during crying -180min)
Tachycardia: >180/min
Bradycardia:<100/min
Look for all the pulses(bounding pulses/feeble)
Respiratory rate
In sleep or resting state: 30-60/min
In preterms- cheyne stokes rhythm or periodic
respiration
Diaphragmatic breathing
15. Use appropriate sized cuff with cuff
width 50% of arm circumference
Measured by oscillometry using NIBP
monitors
Normal BP : 60/40mm Hg
Invasive BP monitoring in sick new borns
In right upper limb and one lower limb
ZUBROS charts
16. Saturation
By pulse oximeter
Right upper limb and any lower limb
SpO2 >95% on room air
Capillary filling time
Measured on the sternum
By pressing index finger for 5 seconds
Normal CFT <3 sec
17. Birth weight
Baby weighed in naked condition using weighing
machine with an accuracy of 5 grms.
Normal >2.5kg
Low birth weight: <2.5kg
Very low birth weight: <1.5kg
Extremely LBW : <1kg
18. Classification By Weight Percentiles
AGA - 10th – 90th percentile for GA
SGA - <10th percentile for GA
LGA - >90th percentile for GA
19. Length:
Measured by infantometer with infant supine
and knees stretched
Crown to heel measurement
Normal length: 47 -50cm
Head circumference:
Occipitofrontal
By using non stretchable tape
Preferably after 24hrs
Normal HC: 33-35cm
20.
21. Chest circumference:
Measured around chest at level of nipples
Head circumference is usually 2-3cm more
than chest circumference
Ponderal index:
To identify IUGR infants
weight(gms)
length(cms)³
× 100
25. Posture and attitude-In Term infants normal
posture is hips abducted and partially flexed,
with knees flexed.
Arms are abducted and flexed at the elbow.
Fists are often clenched, with the fingers
covering the thumb
Physical activity and behavior
Active and passive muscle tone
26. Colour-(normal is pink color in centre &
peripheries)
◦ Mottling/CUTIS MARMORATA– due to vasomotor instability
◦ Cyanosis(central or acrocyanosis)
◦ Pallor
◦ Icterus
◦ Edema
28. Hemangiomas
Petechiae
Extensive skin fragility
Amniotic bands
Sucking blisters or calluses
Nails and Hair(texture,distribution)
SALMON
PATCH/nevus
simplex
PORT WINE STAIN
29. ERYTHEMA TOXICUM
yellow or white papules or
pustules on erythematous
base.
On face,trunk,extremities
except palms and soles
contain eosinophils and are
sterile
Benign and self limited
BENIGN PUSTULAR
MELANOSIS OF THE
NEWBORN
Vesiculopapular eruption on
chin,neck,back,extremities
Hyperpigmented macules
Contain neutrophils
Benign and lasts for 2-3
days
30. Slate-gray to blue-black lesions
Usually over
lumbosacral area and
buttocks
Accumulation of melanocytes
within the dermis
Generally fade by the age of 7
years
APLASIA CUTIS
CONGENITA
•Focal lesion with
congenital absence of
some or all layers of skin
31. HARLEQUIN COLOR
CHANGE
Due to vasomotor
instability or immature
circulation
NEUROCUTANEOUS
MARKERS
Café-au-lait spots
Light brown macules or
patches
No pathologic significance, if
5mm in length and < 6 in
number
If >5mm or > 6 in number it may
indicate Cutaneous
Neurofibromatosis.
32. IMPETIGO NEONATORUM
Vesicular, pustular, or bullous
lesions developing as early as
day of life 2-3 up to 2 weeks of
life.
Lesions occur in moist
areas of skin
Unroofed lesions do not form
crusts
Treat with antibiotics
HSV
CONGENITAL SYPHILIS
CONGENITAL CANDIDIASIS
33. Orifice count
Skull
Moulding of the head
Size of the head-
MACROCEPHALY/MICRO
CEPHALY
Shape of the head
CRANIOTABES
34. Wide areas of fibrous tissue at junction of two
or more sutures
6 fontanelle at birth
1 anterior
1 posterior
2 anterolateral
2 posterolateral
FONTANELLE
35. CAPUT SUCCEDANEUM
– Boggy edema in
presenting part of
head
– Cross suture lines
– Disappear in few days
CEPHALHEMATOMA
– Subperiosteal
– Dose not cross
sutures
– Weeks to resolve
36. .uscneurol
premature closure of one or more cranial suture.
TYPE OF SKULL SYNOSTOSIS APPEARANCE LENGTH-WIDTH
INDEX
MESOCEPHALY normal Normal cranium 76-80
DOLICOCEPHALY/
SCAPHOCEPHALY
sagittal Long/canoe
shaped cranium
70-75
BRACHYCEPHALY coronal Short cranium 80-85
PLAGIOCEPHALY U/L coronal or
lambdoid
Oblique deformity
TRIGONOCEPHALY metopic High peaked >85
37. Look for Dysmorphic features – mongoloid facies,
potter, elfin facies
Facial asymmetry
Hypoplasia of depressor anguli oris
Milia
41. Pre auricular tags
Partial or complete absence of pinna
Deformed pinna and microtia
Low set ears
42. Nares for symmetry and patency
Flat/depressed nasal bridge
Choanal atresia
43. Microstomia/Macrostomia
Micrognathia/retrognathia
Fish like mouth appearance
Natal teeth
Complete or incomplete Cleft lip
Soft and hard palate – high arched palate,
cleft palate(submucosal left)
EPSTEIN PEARLS – small, greyish white
swellings on either side of raphe
TONGUE – size and position,
ankyloglossia, protruding tongue
44. Look for swellings – goitre, cystic hygroma,
branchial arch cysts, teratoma, hemangioma,
sternocleidomastoid tumour.
Shortening or webbing of neck
Congenital torticollis
Palpate clavicles for fractures
45. Chest asymmetry and deformities
Supernummery nipples
Inverted nipples
Widely spaced nipples – Turner syndrome
Breast hypertrophy
Milk production
No redness
Look for asymmetry, erythema, induration or
tenderness
Unilateral absence or hypoplasia of pectoralis
major-POLAND SYNDROME
49. Erb’s palsy: extended arm
and internal rotation with
limited movement
Humerous fracture
Digital abnormality
Syndactyly,
brachydactyly,
polydactyly,clinodactyly
EDEMA of hands and feet-
Turners
55. Inspection for any Distress
signs(Grunting, Tachypnea, Nasal
flaring, stridor, asymmetric chest
rise,supra-sternal, intercostal, sub costal
retraction).
Auscultate
Air entry, symmetry
Added sounds
56. HR 100-160 beats/min
Color, perfusion, Central cyanosis
Apex: 4T
Hto 5thICS, lateral to left sternal border
Precordial pulsations
Dextrocardia- heart on right side and Displacement of apex
S2 slightly sharper and higher pitch than S1
Murmurs
Innocent murmurs in neonates
Systolic murmurs along upper or lower left sternal borders
Intensity 1 to 2/6
Ex: transient TR, closing PDA, pulmonary flow murmur
57. Inspection
Palpation; baby sucking and use warmhands
– Kidneys are normaly palpable
– Liver 2-3 cm
– Spleen tip palpable
– Umbilical vessels
• 2 artery, one vein
– Hernias ; umbilical and inguinal
– Look for cystic/solid masses
59. 1-smell of mother’s milk(not routinely done)
2-fixing at soft light, pupillary reflex, blinking reflex
3,4,6-ptosis,eye movements(spontaneous and doll’s eye
response)
5-rooting reflex, corneal reflex
7-observe child cry for facial palsy
8-response to sound(startle reflex)
9,10-suck and swallow, position of uvula during cry,
choking during feeding
11-lift head off the bed and look for SCM
12-symmetry of the tongue
60. Motor assessment-
Tone(active and passive)
Posture
Head control(pull to sit)
Spontaneous movements
Trunk(ventral suspension,vertical suspension)
Response to handling
DTRs,
Babinski reflex(normal)
61.
62. Also known as developmental, primary, or
primitive reflexes.
They consist of autonomic behaviors that
do not require higher level brain
functioning. They can provide information
about lower motor neurons and muscle
tone.
They are often protective and disappear as
higher level motor functions emerge.
63. Onset: ~28weeks GA
Well-established: 32-34 weeks GA
Disappears: around 12 months
Elicited by the examiner stroking the lips of the
infant; the infant’s mouth opens and the
examiner introduces their gloved finger and
sucking starts.
64. Onset: 28 weeks GA
Well-established: 32-34
weeks GA
Disappears: 3-4 months
Elicited by the examiner
stroking the cheek or corner of
the infant’s mouth. The infant’s
head turns toward the
stimulus and opens its mouth.
65. Onset: 28 weeks GA
Well-established: 32 weeks
GA Disappears: 2 months
Elicited by the examiner
placing his finger on the palmar
surface of the infant’s hand and
the infant’s hand grasps the
finger. Attempts to remove the
finger result in the infant
tightening the grasp.
66. Onset: 35 weeks GA
Well-established: 4 weeks PCA
Disappearance: 7 months
Elicited by rotating the infants head
from midline to one side. The infant
should respond by extending the arm
on the side to which the head is
turned and flexing the opposite arm.
The lower extremities respond
similarly.
67. The examiner holds the infant so that one hand
supports the head and the other supports the
buttocks. The reflex is elicited by the sudden
dropping of the head in hand.
The response is
Opening of hands(by 28weeks gestation)
extension and abduction of the upper extremities(by
32weeks).
anterior flexion of the upper extremities(by 37 wks)
audible cry.
Onset: 28-32 weeks GA
Well-established: 37 weeks GA
Disappearance: 6 months
68. •BILATERAL absence:
CNS depression by narcotics or anesthesia
Brain anoxia and kernicterus
Very Premature baby
ASYMMETRIC response:
Erbs palsy , fracture clavicle or humerus
PERSISTENCE beyond 6th month
CNS damage
69. Onset: 28 weeks GA
Well-established: 40 weeks GA
Disappearance: 3-4 months
The infant is held in ventral suspension with the chest in
the palm of the examiner’s hand. Firm pressure is applied
to the infant’s side parallel to the spine in the thoracic
area. The response consists of flexion of the pelvis toward
the side of the stimulus.
70. Onset: 35-36 weeks GA
Well-established: 37 weeks GA
Disappearance: 3-4 months PCA
Elicited by touching the top of the infant’s
foot to the edge of a table while the infant is
held upright. The infant makes movements
that resemble stepping.
71. When dorsum of the baby
foot touches the under surface
of the table
→ flexion then extension to
place or put his foot on the
table
72. Wash your hand prior to examination
Inspect,Inspect,Inspect,then Touch.
Neonatal reflexes
Normal variations in newborn