The document discusses several types of odontogenic tumors that can occur in the jaws. It focuses on describing ameloblastoma, adenomatoid tumor, and calcifying epithelial odontogenic tumor. Ameloblastoma is a benign but locally aggressive tumor arising from odontogenic epithelium. It commonly presents as a painless swelling in the mandible and radiographs show multilocular radiolucency. Histologically there are follicular or plexiform patterns. Adenomatoid tumor is a rare benign tumor associated with impacted teeth. Calcifying epithelial odontogenic tumor is a rare, locally aggressive tumor that can be mistaken for carcinoma, presenting with calcified masses visible on radiographs.
This document discusses odontogenic tumors, specifically ameloblastoma. It provides details on the classification, clinical features, histologic features, treatment and prognosis of ameloblastoma. Key points include:
- Ameloblastoma is the most common odontogenic tumor and occurs most often in the mandible. It is typically benign but locally invasive.
- Radiographically, it appears as a well-circumscribed radiolucent lesion that can be unilocular or multilocular.
- Treatment options range from curettage to marginal resection, with the goal of obtaining clear margins of at least 1cm. Wide excision is necessary in the maxilla due to risk of local invasion.
Odontogenic keratocyst- A case presentationBinaya Subedi
This case presentation describes a 74-year-old female patient with a swelling in the lower front region of her jaw. Radiographs and biopsy revealed an odontogenic keratocyst. Odontogenic keratocysts are developmental cysts that arise from cell rests of the dental lamina and have a high recurrence rate due to their thin lining. Treatment options considered for this patient's odontogenic keratocyst include curettage with or without peripheral osteotomy or segmental resection of the mandible.
The document discusses different types of cysts that can occur in the oral and maxillofacial region. It defines cysts and classifies them based on their origin and location. It provides details on the pathogenesis, clinical features, radiographic appearance and histology of specific cysts such as dentigerous cysts and odontogenic keratocysts. Dentigerous cysts are defined as cysts originating from the separation of the dental follicle from around the crown of an unerupted tooth. Odontogenic keratocysts are distinctive cysts that arise from cell rests of the dental lamina and have more aggressive behavior than other cysts. Complications of cysts include recurrence, development of
Central Giant Cell Granuloma :
WHO has defined it as an intraosseous lesion consisting of cellular and fibrous tissue that contains multiple foci of hemorrhage, aggregation of multinucleated giant cells and occasionally trabaculae of woven bone
Etiology JAFFE (1953): considered this lesion to be a local reparative reaction of bone, possibly to intramedullary hemorrhage or trauma, hence the term reparative giant cell granuloma was accepted.
Charles A Waldron & W G Shafer (1966) suggested trauma be an important etiological factor in the initiation of the CGCG of the jaws
Thoma K H (1986) suggested that the lesion may be due to capillary injury caused by defective wall due to some type of trauma
J V Soames and J C Southam (1997) suggested that it could be a reaction to some form of hemodynamic disturbance in bone marrow, perhaps associated with trauma and hemorrhage REGEZI AND SCIUBBA(1999) :
Suggested that
Response to previous traumatic or inflammatory episodes.
This lesion is charecterised by proliferation of fibroblasts and multinucleated giant cells, in a densely packed stromaThe CGCG is a benign process that occurs almost exclusively within the jaw bones
CLINICAL PRESENTATION
Found predominantly in children and young adult
It has a female predilection (2:1)
Most commonly affected site is the anterior portion of the jaws, with an increased frequency of occurrence in the mandible
Majority of the CGCG of jaws are painless, expansion of bone is detected on routine examination
Few cases may be associated with pain, paresthesia or perforation of a cortical bone plate, occasionally resulting in the ulceration of the mucosal surface by the underlying lesion
Radiographic featuresCentral giant cell lesions present as radiolucent defects. Which may be unilocular or multilocular.
The defect is usually well delineated
The lesion may vary from a 5×5mm incidental radiographic findings to a destructive lesion greater than 10cm in size.
radiographic findings
A small unilocular lesion may be confused with periapical granuloma or cysts.
multilocular giant cell lesions cannot be radiographically distinguished from ameloblastomas or other multilocular lesions. Based on clinical and radiological features CGCG may be divided into two categories
- Non-aggressive lesion
- Aggressive lesion
The non aggressive lesion makes up most cases and exhibit few or no symptoms, they demonstrate slow growth and do not show cortical perforation or root resorption of teeth involved in the lesion. The aggressive lesions are characterized by pain, rapid growth, cortical perforation and root resorption and show marked tendency to recur when compared with non aggressive typeSoft spongy, brownish to reddish friable tissue of various size.
A specimen is usually coated with fresh or coagulated blood. Giant cell lesions of the jaws show a variety of features. Common to all is the presence of few to many multinucleated
Indian Dental Academy is a leader in continuing dental education, providing both online and offline courses. The document discusses ameloblastoma, a type of odontogenic tumor. It defines ameloblastoma, provides its history and classifications including clinical, radiological, and histopathological. Treatment options discussed include medical therapy, radiotherapy, and various surgical treatments such as enucleation, marsupialization, and curettage. Radiographs, biopsy, CT, and MRI are investigated for ameloblastoma.
This document discusses various non-odontogenic tumors classified according to their origin. It covers giant cell lesions including central giant cell granuloma and brown tumor of hyperparathyroidism. Vascular lesions such as hemangiomas, vascular malformations, and neurogenic tumors including neurofibromas are also discussed. For each type of lesion, the document provides information on classification, clinical features, radiographic findings, diagnosis, and treatment options.
The document discusses several types of odontogenic tumors that can occur in the jaws. It focuses on describing ameloblastoma, adenomatoid tumor, and calcifying epithelial odontogenic tumor. Ameloblastoma is a benign but locally aggressive tumor arising from odontogenic epithelium. It commonly presents as a painless swelling in the mandible and radiographs show multilocular radiolucency. Histologically there are follicular or plexiform patterns. Adenomatoid tumor is a rare benign tumor associated with impacted teeth. Calcifying epithelial odontogenic tumor is a rare, locally aggressive tumor that can be mistaken for carcinoma, presenting with calcified masses visible on radiographs.
This document discusses odontogenic tumors, specifically ameloblastoma. It provides details on the classification, clinical features, histologic features, treatment and prognosis of ameloblastoma. Key points include:
- Ameloblastoma is the most common odontogenic tumor and occurs most often in the mandible. It is typically benign but locally invasive.
- Radiographically, it appears as a well-circumscribed radiolucent lesion that can be unilocular or multilocular.
- Treatment options range from curettage to marginal resection, with the goal of obtaining clear margins of at least 1cm. Wide excision is necessary in the maxilla due to risk of local invasion.
Odontogenic keratocyst- A case presentationBinaya Subedi
This case presentation describes a 74-year-old female patient with a swelling in the lower front region of her jaw. Radiographs and biopsy revealed an odontogenic keratocyst. Odontogenic keratocysts are developmental cysts that arise from cell rests of the dental lamina and have a high recurrence rate due to their thin lining. Treatment options considered for this patient's odontogenic keratocyst include curettage with or without peripheral osteotomy or segmental resection of the mandible.
The document discusses different types of cysts that can occur in the oral and maxillofacial region. It defines cysts and classifies them based on their origin and location. It provides details on the pathogenesis, clinical features, radiographic appearance and histology of specific cysts such as dentigerous cysts and odontogenic keratocysts. Dentigerous cysts are defined as cysts originating from the separation of the dental follicle from around the crown of an unerupted tooth. Odontogenic keratocysts are distinctive cysts that arise from cell rests of the dental lamina and have more aggressive behavior than other cysts. Complications of cysts include recurrence, development of
Central Giant Cell Granuloma :
WHO has defined it as an intraosseous lesion consisting of cellular and fibrous tissue that contains multiple foci of hemorrhage, aggregation of multinucleated giant cells and occasionally trabaculae of woven bone
Etiology JAFFE (1953): considered this lesion to be a local reparative reaction of bone, possibly to intramedullary hemorrhage or trauma, hence the term reparative giant cell granuloma was accepted.
Charles A Waldron & W G Shafer (1966) suggested trauma be an important etiological factor in the initiation of the CGCG of the jaws
Thoma K H (1986) suggested that the lesion may be due to capillary injury caused by defective wall due to some type of trauma
J V Soames and J C Southam (1997) suggested that it could be a reaction to some form of hemodynamic disturbance in bone marrow, perhaps associated with trauma and hemorrhage REGEZI AND SCIUBBA(1999) :
Suggested that
Response to previous traumatic or inflammatory episodes.
This lesion is charecterised by proliferation of fibroblasts and multinucleated giant cells, in a densely packed stromaThe CGCG is a benign process that occurs almost exclusively within the jaw bones
CLINICAL PRESENTATION
Found predominantly in children and young adult
It has a female predilection (2:1)
Most commonly affected site is the anterior portion of the jaws, with an increased frequency of occurrence in the mandible
Majority of the CGCG of jaws are painless, expansion of bone is detected on routine examination
Few cases may be associated with pain, paresthesia or perforation of a cortical bone plate, occasionally resulting in the ulceration of the mucosal surface by the underlying lesion
Radiographic featuresCentral giant cell lesions present as radiolucent defects. Which may be unilocular or multilocular.
The defect is usually well delineated
The lesion may vary from a 5×5mm incidental radiographic findings to a destructive lesion greater than 10cm in size.
radiographic findings
A small unilocular lesion may be confused with periapical granuloma or cysts.
multilocular giant cell lesions cannot be radiographically distinguished from ameloblastomas or other multilocular lesions. Based on clinical and radiological features CGCG may be divided into two categories
- Non-aggressive lesion
- Aggressive lesion
The non aggressive lesion makes up most cases and exhibit few or no symptoms, they demonstrate slow growth and do not show cortical perforation or root resorption of teeth involved in the lesion. The aggressive lesions are characterized by pain, rapid growth, cortical perforation and root resorption and show marked tendency to recur when compared with non aggressive typeSoft spongy, brownish to reddish friable tissue of various size.
A specimen is usually coated with fresh or coagulated blood. Giant cell lesions of the jaws show a variety of features. Common to all is the presence of few to many multinucleated
Indian Dental Academy is a leader in continuing dental education, providing both online and offline courses. The document discusses ameloblastoma, a type of odontogenic tumor. It defines ameloblastoma, provides its history and classifications including clinical, radiological, and histopathological. Treatment options discussed include medical therapy, radiotherapy, and various surgical treatments such as enucleation, marsupialization, and curettage. Radiographs, biopsy, CT, and MRI are investigated for ameloblastoma.
This document discusses various non-odontogenic tumors classified according to their origin. It covers giant cell lesions including central giant cell granuloma and brown tumor of hyperparathyroidism. Vascular lesions such as hemangiomas, vascular malformations, and neurogenic tumors including neurofibromas are also discussed. For each type of lesion, the document provides information on classification, clinical features, radiographic findings, diagnosis, and treatment options.
This document provides definitions and classifications of radiopaque lesions that can be seen on dental radiographs. It begins with defining normal radiopacity and listing common anatomical radiopacities seen in the jaws. Lesions are then classified as abnormalities of the teeth, developmental conditions affecting bone, inflammatory conditions, and odontogenic/non-odontogenic tumors. Specific conditions like condensing osteitis, periapical cemento-osseous dysplasia, odontomes, and cementoblastoma are described in detail with their typical radiographic features and differences.
Fibro-osseous lesions of the jaws
Fibrous dysplasia
Cemento-osseous dysplasia
Focal cemento-osseous dysplasia
Periapical cemento-osseous dysplasia
Florid cemento-osseous dysplasia
Ossifying fibroma
Juvenile aggressive ossifying fibroma
Cherubism
Fibro-osseous lesions (FOL) are characterized by replacement of normal bone architecture by collagen fibers and fibroblasts containing calcified tissue.
They include a wide variety of lesions of developmental, dysplastic and neoplastic origins with clinical and radiographic presentation and behavior.
Because of the histological similarities between diverse diseases, proper diagnosis requires correlation of history, clinical and radiographic findings.Fibrous Dysplasia
2. Reactive (dysplastic lesions arising in the tooth-bearing area (presumably of periodontal origin).
a. Periapical cemento-osseous dysplasia
b. Focal cemento-osseous dysplasia
c. Florid cemento-osseous dysplasia
3. Fibro-osseous neoplasms (widely designated as cementifying fibroma, ossifying fibroma or cemento-ossifying fibroma.Bone dysplasias
a. Fibrous dyspla i. Monostoticii. Polyostotic
iii. Polyostotic with endocrinopathy (McCune-Albright)
iv Osteofibrous dysplasia
b. Osteitis deformansc. Pagetoid heritable bone dysplasias of childhood
d. Segmental odontomaxillary dysplasia
2. Cemento-osseous dysplasias
a. Focal cemento-osseous dysplasia b. Florid cemento-osseous dysplasia
3.Inflammatory/reactive processes
a. Focal sclerosing osteomyelitisb. Diffuse sclerosing osteomyelitis
c. Proliferative periostitis
4. Metabolic Disease: hyperparathyroidism
5. Neoplastic lesions (Ossifying fibromas)
a. Ossifying fibromab. Hyperparathyroidism jaw lesion syndrome
c. Juvenile ossifying fibroma i. Trabecular typeii. Psammomatoid type
d. Gigantiform cementomas
This document discusses odontogenic keratocysts (OKCs), a type of jaw cyst. It covers the classification, causes, histopathology, clinical features, radiographic features, differential diagnosis, treatment principles, and surgical treatment options for OKCs. OKCs most commonly occur in the mandibular molar and ramus areas and are often radiolucent and multilocular in appearance on radiographs. Treatment options include wide surgical excision or marsupialization to prevent recurrence of these cysts which have a high rate of recurrence compared to other jaw cysts.
This document discusses several pathologies that can affect the jaws, including:
1. The adenomatoid odontogenic tumor, which presents as a swelling in young patients around unerupted teeth and consists of epithelial cells and calcifications.
2. The calcifying epithelial odontogenic tumor, which occurs in the mandible or maxilla as a radiolucent lesion containing radiopacities from calcification.
3. Odontomas, which are hamartomas containing dental tissues like enamel and dentin that appear as radiopaque masses and require conservative excision.
This document presents a case study of a 30-year-old female patient diagnosed with plexiform ameloblastoma based on a biopsy of her right mandible. It provides background on ameloblastoma, describing it as a benign odontogenic tumor arising from odontogenic epithelium. It discusses the various histological subtypes including follicular, plexiform, unicystic (three groups), peripheral, acanthomatous, granular, basal cell, and desmoplastic. Treatment typically involves radical excision, with curettage having the highest recurrence rates. The case study aims to educate on the clinical, radiographic, and histological features of ameloblastoma.
Epithelial dysplasia refers to disturbances in epithelial cell proliferation and differentiation seen microscopically. It is characterized by cellular atypia and graded as mild, moderate, or severe. Key features include basal cell hyperplasia, abnormal mitosis, nuclear hyperchromatism, increased nuclear-cytoplasmic ratio, dyskaryosis, poikilokaryonosis, loss of polarity, anisocytosis, koilocytosis, and individual cell keratinization.
1) Ameloblastoma is a benign, locally invasive odontogenic tumor of enamel organ-type tissue that is the second most common odontogenic tumor.
2) It typically presents as a painless swelling in the mandible and is classified based on histological and clinical features into subtypes including follicular, plexiform, unicystic, and peripheral ameloblastoma.
3) Treatment involves surgical resection such as segmental resection for large tumors due to the high recurrence risk with more conservative treatments like curettage or enucleation.
This document discusses dentigerous cysts, including their definition, pathogenesis, clinical features, radiological features, histological features, investigation, treatment, and potential complications. A dentigerous cyst is an odontogenic cyst that forms around the crown of an unerupted tooth due to fluid accumulation between the reduced enamel epithelium and enamel surface. Clinically, they usually appear as asymptomatic swellings but can cause expansion of the bone. Radiographically, they appear as well-defined radiolucencies that surround the crown of an unerupted tooth. Treatment involves surgical removal of the cyst lining either through enucleation or marsupialization along with removal of the associated tooth. Complications can include
This document discusses the history, definition, etiology, clinical characteristics, diagnostic methods, and treatment of myofascial pain dysfunction syndrome (MPDS). Some key points:
- MPDS is a pain disorder caused by trigger points in the muscles of mastication that refer pain to the head and neck. It is the most common cause of masticatory pain.
- Etiology may include occlusal factors, prosthetic problems, malocclusion, psychophysiologic factors, and trauma.
- Clinical characteristics include pain in the head/neck, limited jaw motion, joint noises, and tender muscles.
- Treatment involves a multidisciplinary approach including medications, trigger point injections, physical
1. The document classifies and describes several diseases that can affect the jaw bone, including inflammatory, hereditary, metabolic, and neoplastic diseases.
2. Several primary bone tumors are described in detail, including osteoma, osteoid osteoma, osteoblastoma, osteosarcoma, osteochondroma, chondroma, chondrosarcoma, giant cell tumor, Ewing's sarcoma, and multiple myeloma.
3. For each tumor, the clinical features, radiographic appearance, and histopathology are summarized to aid in diagnosis and classification of jaw bone diseases.
Cysts in orofacial regions were discussed. Key points include:
1. Cysts are pathological cavities lined by epithelium and filled with fluid/semi-solid material. Common types are odontogenic cysts like dentigerous and keratocysts.
2. Dentigerous cysts form between reduced enamel epithelium and tooth crown, associated with unerupted teeth. Keratocysts have high recurrence rates due to thin fragile lining.
3. Treatment options are marsupialization to shrink large cysts, and enucleation to remove the cyst lining along with the associated tooth/teeth.
This document discusses mixed odontogenic tumors and odontogenic sarcomas according to the 2005 WHO classification. It provides details on ameloblastic fibroma, its clinical features, histopathology, radiographic features and differential diagnosis. It also discusses ameloblastic fibro-odontoma and fibro-dentinoma, calcifying cystic odontogenic tumor, complex and compound odontomas, and odontoameloblastoma. The key information provided includes the definitions, epidemiology, clinical and radiographic presentation, histopathology, and differential diagnosis of these odontogenic lesions.
Odontogenic keratocyst (OKC) is the cyst arising from the cell rests of dental lamina. It can occur anywhere in the jaw, but commonly seen in the posterior part of the mandible. Radiographically, most OKCs are unilocular when presented at the periapex and can be mistaken for radicular or lateral periodontal cyst.
This document summarizes different techniques for pulp capping including direct and indirect pulp capping. It discusses the history of various materials used for pulp capping such as calcium hydroxide and mineral trioxide aggregate. Key points covered include the indications and contraindications for pulp capping, the ideal properties of capping materials, and the histological response after pulp capping including zone formation. The document provides an overview of pulp capping procedures and factors influencing their success.
This document summarizes several odontogenic tumors including: ameloblastoma (benign, locally aggressive tumor of odontogenic epithelium that commonly occurs in the mandible), adenomatoid odontogenic tumor (uncommon, nonaggressive tumor that often surrounds an unerupted tooth), calcifying epithelial odontogenic tumor (rare benign neoplasm that usually occurs in the mandible), odontoma (tumor characterized by production of dental tissues that commonly occurs in young patients), ameloblastic fibroma (benign mixed odontogenic tumor that occurs in children/adolescents), odontogenic myxoma (benign tumor arising from dental papilla mesenchyme), cementoblast
Dilaceration is a bending or curvature of a tooth root caused by either trauma that displaces the calcified portion during development or a developmental defect. It most commonly occurs in maxillary incisors and is diagnosed radiographically by observing the curved root. Management can be difficult and involves restoring a dilacerated crown to improve function and prevent dental issues, while extraction may require special care due to the root curvature.
Radicular cysts are odontogenic cysts that form from cell rests of Malassez in response to inflammation from pulp necrosis. They are commonly found in the maxillary anterior and posterior regions in people aged 20-60 years old. Radicular cysts appear radiolucent on x-rays and are associated with non-vital teeth. Treatment involves root canal therapy or extraction of the offending tooth along with surgical removal of the cyst.
This document provides an overview of fibro-osseous lesions of the jaws. It discusses the classification of these lesions, which include fibrous dysplasia, ossifying fibroma, cemento-osseous dysplasia, central giant cell granuloma, cherubism, aneurysmal bone cyst, and solitary bone cyst. It focuses on the etiology, pathophysiology, clinical features, and oral manifestations of fibrous dysplasia, including monostotic fibrous dysplasia, polyostotic fibrous dysplasia, Jaffe's lichtenstein syndrome, McCune-Albright syndrome, and craniofacial fibrous dysplasia.
This document discusses radicular cysts, which are the most common inflammatory cysts in the oral cavity. Radicular cysts arise from epithelial residues in the periodontal ligament as a result of periapical periodontitis following pulp necrosis. They are usually asymptomatic but can cause swelling and bone resorption as they expand. The cyst forms from the proliferation of epithelial cell rests in the granulation tissue surrounding the apex of an infected tooth. Histologically, they are lined by stratified squamous epithelium and surrounded by fibrous connective tissue that may contain cholesterol crystals. Treatment involves root canal therapy or extraction with curettage of the cyst lining.
Non odontogenic cyst and pseudo cyst of the jaw- seminar 2- ORIGINAL.pptxReshmaAmmu11
This document summarizes and classifies different types of cysts. It begins by defining a cyst and outlining the stages of cyst formation. It then separates cysts into two main categories: odontogenic cysts, which are derived from tooth germ remnants, and non-odontogenic cysts, which arise from epithelial remnants of embryonic structures. Several examples of developmental, inflammatory, and miscellaneous cysts are provided within each category. Specific cysts like the nasopalatine duct cyst, median palatal cyst, and antral pseudocyst are then discussed in more detail, covering their clinical features, histology, treatment and differential diagnosis.
This document provides definitions and classifications of radiopaque lesions that can be seen on dental radiographs. It begins with defining normal radiopacity and listing common anatomical radiopacities seen in the jaws. Lesions are then classified as abnormalities of the teeth, developmental conditions affecting bone, inflammatory conditions, and odontogenic/non-odontogenic tumors. Specific conditions like condensing osteitis, periapical cemento-osseous dysplasia, odontomes, and cementoblastoma are described in detail with their typical radiographic features and differences.
Fibro-osseous lesions of the jaws
Fibrous dysplasia
Cemento-osseous dysplasia
Focal cemento-osseous dysplasia
Periapical cemento-osseous dysplasia
Florid cemento-osseous dysplasia
Ossifying fibroma
Juvenile aggressive ossifying fibroma
Cherubism
Fibro-osseous lesions (FOL) are characterized by replacement of normal bone architecture by collagen fibers and fibroblasts containing calcified tissue.
They include a wide variety of lesions of developmental, dysplastic and neoplastic origins with clinical and radiographic presentation and behavior.
Because of the histological similarities between diverse diseases, proper diagnosis requires correlation of history, clinical and radiographic findings.Fibrous Dysplasia
2. Reactive (dysplastic lesions arising in the tooth-bearing area (presumably of periodontal origin).
a. Periapical cemento-osseous dysplasia
b. Focal cemento-osseous dysplasia
c. Florid cemento-osseous dysplasia
3. Fibro-osseous neoplasms (widely designated as cementifying fibroma, ossifying fibroma or cemento-ossifying fibroma.Bone dysplasias
a. Fibrous dyspla i. Monostoticii. Polyostotic
iii. Polyostotic with endocrinopathy (McCune-Albright)
iv Osteofibrous dysplasia
b. Osteitis deformansc. Pagetoid heritable bone dysplasias of childhood
d. Segmental odontomaxillary dysplasia
2. Cemento-osseous dysplasias
a. Focal cemento-osseous dysplasia b. Florid cemento-osseous dysplasia
3.Inflammatory/reactive processes
a. Focal sclerosing osteomyelitisb. Diffuse sclerosing osteomyelitis
c. Proliferative periostitis
4. Metabolic Disease: hyperparathyroidism
5. Neoplastic lesions (Ossifying fibromas)
a. Ossifying fibromab. Hyperparathyroidism jaw lesion syndrome
c. Juvenile ossifying fibroma i. Trabecular typeii. Psammomatoid type
d. Gigantiform cementomas
This document discusses odontogenic keratocysts (OKCs), a type of jaw cyst. It covers the classification, causes, histopathology, clinical features, radiographic features, differential diagnosis, treatment principles, and surgical treatment options for OKCs. OKCs most commonly occur in the mandibular molar and ramus areas and are often radiolucent and multilocular in appearance on radiographs. Treatment options include wide surgical excision or marsupialization to prevent recurrence of these cysts which have a high rate of recurrence compared to other jaw cysts.
This document discusses several pathologies that can affect the jaws, including:
1. The adenomatoid odontogenic tumor, which presents as a swelling in young patients around unerupted teeth and consists of epithelial cells and calcifications.
2. The calcifying epithelial odontogenic tumor, which occurs in the mandible or maxilla as a radiolucent lesion containing radiopacities from calcification.
3. Odontomas, which are hamartomas containing dental tissues like enamel and dentin that appear as radiopaque masses and require conservative excision.
This document presents a case study of a 30-year-old female patient diagnosed with plexiform ameloblastoma based on a biopsy of her right mandible. It provides background on ameloblastoma, describing it as a benign odontogenic tumor arising from odontogenic epithelium. It discusses the various histological subtypes including follicular, plexiform, unicystic (three groups), peripheral, acanthomatous, granular, basal cell, and desmoplastic. Treatment typically involves radical excision, with curettage having the highest recurrence rates. The case study aims to educate on the clinical, radiographic, and histological features of ameloblastoma.
Epithelial dysplasia refers to disturbances in epithelial cell proliferation and differentiation seen microscopically. It is characterized by cellular atypia and graded as mild, moderate, or severe. Key features include basal cell hyperplasia, abnormal mitosis, nuclear hyperchromatism, increased nuclear-cytoplasmic ratio, dyskaryosis, poikilokaryonosis, loss of polarity, anisocytosis, koilocytosis, and individual cell keratinization.
1) Ameloblastoma is a benign, locally invasive odontogenic tumor of enamel organ-type tissue that is the second most common odontogenic tumor.
2) It typically presents as a painless swelling in the mandible and is classified based on histological and clinical features into subtypes including follicular, plexiform, unicystic, and peripheral ameloblastoma.
3) Treatment involves surgical resection such as segmental resection for large tumors due to the high recurrence risk with more conservative treatments like curettage or enucleation.
This document discusses dentigerous cysts, including their definition, pathogenesis, clinical features, radiological features, histological features, investigation, treatment, and potential complications. A dentigerous cyst is an odontogenic cyst that forms around the crown of an unerupted tooth due to fluid accumulation between the reduced enamel epithelium and enamel surface. Clinically, they usually appear as asymptomatic swellings but can cause expansion of the bone. Radiographically, they appear as well-defined radiolucencies that surround the crown of an unerupted tooth. Treatment involves surgical removal of the cyst lining either through enucleation or marsupialization along with removal of the associated tooth. Complications can include
This document discusses the history, definition, etiology, clinical characteristics, diagnostic methods, and treatment of myofascial pain dysfunction syndrome (MPDS). Some key points:
- MPDS is a pain disorder caused by trigger points in the muscles of mastication that refer pain to the head and neck. It is the most common cause of masticatory pain.
- Etiology may include occlusal factors, prosthetic problems, malocclusion, psychophysiologic factors, and trauma.
- Clinical characteristics include pain in the head/neck, limited jaw motion, joint noises, and tender muscles.
- Treatment involves a multidisciplinary approach including medications, trigger point injections, physical
1. The document classifies and describes several diseases that can affect the jaw bone, including inflammatory, hereditary, metabolic, and neoplastic diseases.
2. Several primary bone tumors are described in detail, including osteoma, osteoid osteoma, osteoblastoma, osteosarcoma, osteochondroma, chondroma, chondrosarcoma, giant cell tumor, Ewing's sarcoma, and multiple myeloma.
3. For each tumor, the clinical features, radiographic appearance, and histopathology are summarized to aid in diagnosis and classification of jaw bone diseases.
Cysts in orofacial regions were discussed. Key points include:
1. Cysts are pathological cavities lined by epithelium and filled with fluid/semi-solid material. Common types are odontogenic cysts like dentigerous and keratocysts.
2. Dentigerous cysts form between reduced enamel epithelium and tooth crown, associated with unerupted teeth. Keratocysts have high recurrence rates due to thin fragile lining.
3. Treatment options are marsupialization to shrink large cysts, and enucleation to remove the cyst lining along with the associated tooth/teeth.
This document discusses mixed odontogenic tumors and odontogenic sarcomas according to the 2005 WHO classification. It provides details on ameloblastic fibroma, its clinical features, histopathology, radiographic features and differential diagnosis. It also discusses ameloblastic fibro-odontoma and fibro-dentinoma, calcifying cystic odontogenic tumor, complex and compound odontomas, and odontoameloblastoma. The key information provided includes the definitions, epidemiology, clinical and radiographic presentation, histopathology, and differential diagnosis of these odontogenic lesions.
Odontogenic keratocyst (OKC) is the cyst arising from the cell rests of dental lamina. It can occur anywhere in the jaw, but commonly seen in the posterior part of the mandible. Radiographically, most OKCs are unilocular when presented at the periapex and can be mistaken for radicular or lateral periodontal cyst.
This document summarizes different techniques for pulp capping including direct and indirect pulp capping. It discusses the history of various materials used for pulp capping such as calcium hydroxide and mineral trioxide aggregate. Key points covered include the indications and contraindications for pulp capping, the ideal properties of capping materials, and the histological response after pulp capping including zone formation. The document provides an overview of pulp capping procedures and factors influencing their success.
This document summarizes several odontogenic tumors including: ameloblastoma (benign, locally aggressive tumor of odontogenic epithelium that commonly occurs in the mandible), adenomatoid odontogenic tumor (uncommon, nonaggressive tumor that often surrounds an unerupted tooth), calcifying epithelial odontogenic tumor (rare benign neoplasm that usually occurs in the mandible), odontoma (tumor characterized by production of dental tissues that commonly occurs in young patients), ameloblastic fibroma (benign mixed odontogenic tumor that occurs in children/adolescents), odontogenic myxoma (benign tumor arising from dental papilla mesenchyme), cementoblast
Dilaceration is a bending or curvature of a tooth root caused by either trauma that displaces the calcified portion during development or a developmental defect. It most commonly occurs in maxillary incisors and is diagnosed radiographically by observing the curved root. Management can be difficult and involves restoring a dilacerated crown to improve function and prevent dental issues, while extraction may require special care due to the root curvature.
Radicular cysts are odontogenic cysts that form from cell rests of Malassez in response to inflammation from pulp necrosis. They are commonly found in the maxillary anterior and posterior regions in people aged 20-60 years old. Radicular cysts appear radiolucent on x-rays and are associated with non-vital teeth. Treatment involves root canal therapy or extraction of the offending tooth along with surgical removal of the cyst.
This document provides an overview of fibro-osseous lesions of the jaws. It discusses the classification of these lesions, which include fibrous dysplasia, ossifying fibroma, cemento-osseous dysplasia, central giant cell granuloma, cherubism, aneurysmal bone cyst, and solitary bone cyst. It focuses on the etiology, pathophysiology, clinical features, and oral manifestations of fibrous dysplasia, including monostotic fibrous dysplasia, polyostotic fibrous dysplasia, Jaffe's lichtenstein syndrome, McCune-Albright syndrome, and craniofacial fibrous dysplasia.
This document discusses radicular cysts, which are the most common inflammatory cysts in the oral cavity. Radicular cysts arise from epithelial residues in the periodontal ligament as a result of periapical periodontitis following pulp necrosis. They are usually asymptomatic but can cause swelling and bone resorption as they expand. The cyst forms from the proliferation of epithelial cell rests in the granulation tissue surrounding the apex of an infected tooth. Histologically, they are lined by stratified squamous epithelium and surrounded by fibrous connective tissue that may contain cholesterol crystals. Treatment involves root canal therapy or extraction with curettage of the cyst lining.
Non odontogenic cyst and pseudo cyst of the jaw- seminar 2- ORIGINAL.pptxReshmaAmmu11
This document summarizes and classifies different types of cysts. It begins by defining a cyst and outlining the stages of cyst formation. It then separates cysts into two main categories: odontogenic cysts, which are derived from tooth germ remnants, and non-odontogenic cysts, which arise from epithelial remnants of embryonic structures. Several examples of developmental, inflammatory, and miscellaneous cysts are provided within each category. Specific cysts like the nasopalatine duct cyst, median palatal cyst, and antral pseudocyst are then discussed in more detail, covering their clinical features, histology, treatment and differential diagnosis.
This document discusses and compares different types of non-odontogenic (not related to teeth) cysts. It separates them into developmental and inflammatory cysts. Developmental cysts form due to epithelial cell remnants becoming trapped during embryonic development, while inflammatory cysts form due to duct obstruction or trauma. Some examples of developmental cysts mentioned are nasopalatine duct cysts, median palatal cysts, and dermoid cysts. Inflammatory cysts include mucoceles, ranulas caused by salivary gland duct obstruction, and retention cysts of the maxillary sinus. The document provides details on pathogenesis, clinical features, histopathology, diagnosis and treatment of several of these cyst types.
Non-odontogenic cysts originate from tissues other than teeth and surrounding structures, such as the mucosal lining, salivary glands, connective tissue, or developmental remnants in the oral cavity. Unlike odontogenic cysts, which arise from dental tissues, non-odontogenic cysts emerge from various non-dental tissues and often present diverse clinical manifestations. Common examples of non-odontogenic cysts include the nasopalatine duct cyst, nasolabial cyst, and developmental cysts that form from epithelial remnants during embryonic development.
This document defines and classifies different types of cysts that can occur in the oral cavity. It discusses epithelial cysts, which make up over 50% of oral cysts and includes radicular, dentigerous, and odontogenic keratocysts. Nonepithelial cysts are also mentioned. Specific cysts like paradental, nasopalatine, and solitary bone cysts are defined. Treatment options for jaw cysts include enucleation, marsupialization, a combination of both, and enucleation with curettage.
The document discusses several types of non-odontogenic cysts that develop in the oral cavity. It describes nasopalatine duct cysts, which originate from remnants of the nasopalatine duct in the maxilla. These cysts typically appear as well-defined radiolucencies between the central incisors. Median palatal cysts and globulomaxillary cysts are also discussed, which develop from epithelial remnants during fusion of facial processes. Palatal cysts of newborns are extraosseous cysts that commonly appear on the hard palate of infants.
This document discusses various cysts that can occur in the oral and maxillofacial region. It begins by defining cysts and discussing their classification. It then focuses on specific types of cysts including dentigerous cysts, odontogenic keratocysts (also called primordial cysts), and Gorlin-Goltz syndrome, which is characterized by multiple odontogenic keratocysts. For each cyst type, the document discusses epidemiology, pathogenesis, clinical features, radiographic appearance, histopathology, treatment and other relevant details. It provides an in-depth overview of cysts that can develop in the jaw bones and soft tissues of the oral cavity and face.
This document provides information on various types of cysts that can occur in the oral cavity. It defines cysts and discusses their parts and classification. It describes the pathogenesis and factors involved in cyst initiation and enlargement. It then examines several specific cysts in more detail, including their definitions, locations, clinical and radiographic features, pathogenesis and complications. The cysts discussed include dentigerous cysts, odontogenic keratocysts, eruption cysts, calcifying odontogenic cysts, nasopalatine duct cysts and nasolabial cysts. Frequency data on common cyst types is also presented.
This seminar consists of various cysts seen in the oral cavity alonh with various classifications and added case repots for better understanding and the various treatment protocols followed for treating various cysts.
Based on the information provided, the key differentials would be:
- Radicular cyst: Most common cyst in jaws, associated with non-vital tooth. Location and association with tooth fits.
- Dentigerous cyst: Second most common, associated with crown of unerupted tooth. Location fits.
- Odontogenic keratocyst: Aggressive cyst, often multilocular radiolucency. Less likely based on description.
- Aneurysmal bone cyst: Often multilocular "soap bubble" appearance. Less likely based on description.
- Traumatic bone cyst: Often interradicular in location. Possible based on location described.
Further investigation with tooth
This document provides an overview of cysts of the oral and maxillofacial region. It defines cysts and discusses their classification, parts, pathogenesis and mechanisms of enlargement. It also describes key cysts such as dentigerous cysts, odontogenic keratocysts and eruption cysts in detail, covering their definitions, locations, clinical and radiographic features, histology, differential diagnosis and complications. Dentigerous and odontogenic keratocysts are the most common epithelial cysts of the jaws.
cysts of oral and maxillofacial region.pdfasishkp1
The document discusses various cysts that can occur in the oral and maxillofacial region. It begins by defining cysts and describing their general characteristics such as being fluid-filled cavities lined by epithelium and growing slowly by expansion. It then describes different types of cysts including true cysts lined by epithelium and pseudo cysts not lined by epithelium. The document further classifies cysts based on their location, discusses their pathogenesis, and provides details on specific cysts such as dentigerous cysts, odontogenic keratocysts, eruption cysts, and lateral periodontal cysts including their definitions, clinical features, radiographic appearances, histology, and complications.
The document discusses different types of cysts that can occur in the oral and maxillofacial region. It defines cysts and classifies them based on their origin and location. It provides details on the pathogenesis, clinical features, radiographic appearance and histology of specific cysts such as dentigerous cysts and odontogenic keratocysts. Dentigerous cysts are defined as cysts originating from the separation of the dental follicle from around the crown of an unerupted tooth. Odontogenic keratocysts are distinctive cysts that arise from cell rests of the dental lamina and have more aggressive behavior than other cysts. Complications of cysts include recurrence, development of
This document defines and classifies odontogenic cysts, which are epithelium-lined sacs that arise from odontogenic epithelium. It discusses the most common types, including periapical (radicular) cysts, dentigerous cysts, odontogenic keratocysts, glandular odontogenic cysts, and calcifying odontogenic cysts. For each cyst type, it describes characteristics such as prevalence, location, radiographic appearance, histopathology, treatment involving enucleation or marsupialization, and prognosis. It also discusses the basal cell nevus syndrome that can be associated with odontogenic keratocysts.
Developmental cysts can arise from epithelial remnants during embryonic development. The nasopalatine duct cyst arises from remnants of the nasopalatine duct in the midline maxilla. It appears on imaging as an oval radiolucency between central incisors. Histologically it is lined by respiratory, cuboidal, or squamous epithelium. Surgical enucleation is the treatment and recurrence is rare. The nasolabial cyst arises from remnants of the nasolacrimal duct in the soft tissues of the anterior maxilla. It presents as a unilateral swelling and is treated with surgical excision.
Fissural cysts arise along lines of fusion between embryonic processes. Nasopalatine duct cysts are the most common non-odontogenic cyst, arising from epithelial remnants of the nasopalatine duct. Median palatal cysts occur in the midline of the hard palate from entrapped epithelium. Dermoid and epidermoid cysts contain skin elements and arise from implantation of epithelium during embryonic development. These cysts are examined clinically and radiographically and often surgically removed.
This document discusses various developmental cysts that can occur in the oral and maxillofacial region. It provides details on the location, cause, clinical features, radiographic features, histological features, treatment and prognosis for different cysts such as palatal cyst of newborn, nasolabial cyst, globullomaxillary cyst, nasopalatine duct cyst, median palatal cyst, median mandibular cyst, epidermoid cyst, dermoid cyst, thyroglossal duct cyst, branchial cleft cyst, and oral lymphoepithelial cyst.
This document discusses non-odontogenic cysts, which are cysts that arise from epithelial entrapments during embryonic development and are not derived from odontogenic epithelium. It describes several types of non-odontogenic cysts including fissural cysts like the nasopalatine duct cyst, pseudo cysts like the traumatic bone cyst, and soft tissue cysts like the thyroglossal cyst. Radiographic appearances and locations are provided for different cyst types.
1. Vascular lesions are classified as vascular tumors (hemangiomas) or vascular malformations. Hemangiomas involve proliferation of endothelial cells while malformations involve abnormal dilation of vessels without proliferation.
2. A history and physical can often diagnose vascular lesions but imaging like MRI may be needed to determine extent and type of lesion. MRI provides better contrast and ability to distinguish lesion types compared to CT or angiography.
3. Hemangiomas are the most common tumor in childhood, often on the head or neck. They have proliferative and involution stages. Malformations are always present at birth and expand over time. Treatment depends on type, location, and stage of lesion and may include laser therapy, steroid injections,
This document discusses common medical emergencies that may occur during dental treatment. It outlines 8 potential emergencies: 1) collapse, 2) anaphylaxis, 3) cardiac arrest, 4) hypoglycemia in diabetics, 5) seizures, 6) chest pain, 7) acute severe asthma, and 8) adrenal crisis. For each emergency, it describes potential causes, signs and symptoms, diagnosis, and basic management steps to be followed. The document emphasizes that serious medical emergencies are rare but can be life-threatening, so dentists should be prepared to recognize and respond appropriately to emergencies.
The document summarizes common complications that can occur from local anesthesia in dental procedures. It discusses 9 types of local complications: 1) pain on injection, 2) failure to obtain anesthesia, 3) persistent anesthesia/paresthesia, 4) needle breakage, 5) facial nerve paralysis, 6) trismus, 7) soft tissue injury, 8) hematoma, and 9) systemic complications. For each complication, it describes potential causes and recommendations for prevention and management. The goal is to educate dental practitioners about minimizing risks associated with local anesthesia administration.
This document discusses techniques for local anesthesia in dentistry. It describes four main types of local anesthesia: topical, infiltration, field block, and regional block. Infiltration anesthesia involves depositing the anesthetic solution near terminal nerve fibers to infiltrate the tissue and anesthetize a localized area. It is commonly used for the front of the maxilla and mandible. Regional block anesthesia deposits the solution near the main nerve trunk to anesthetize a larger area. The document provides details on specific infiltration and regional block techniques for anesthetizing different areas of the maxilla and palate.
This document discusses local anesthesia equipment and techniques. It describes different types of syringes used to administer local anesthetics including non-disposable, disposable, safety, and computer-controlled syringes. It also discusses dental needles in terms of gauge, length, and potential clinical problems. The dental cartridge components and potential issues are explained. Topical anesthetics are recommended to be applied before needle penetration to minimize pain.
The document discusses the constituents and components of local anesthetic solutions used in dentistry. It describes the various local anesthetic agents, including lignocaine/lidocaine, and how they are classified. It also covers vasoconstrictors like epinephrine/adrenaline and norepinephrine/noradrenaline that are added to local anesthetics to increase their duration and effectiveness. The pharmacokinetics of local anesthetics including distribution, metabolism, excretion are summarized. Factors in selecting local anesthetics and maximum recommended doses are also provided.
This document discusses the anatomy related to local anesthesia in dentistry. It provides an overview of the nervous system, with a focus on the trigeminal nerve and its three divisions - the ophthalmic, maxillary, and mandibular nerves. It describes the branches and distribution of the trigeminal nerve, as well as related ganglia. It also discusses the relevant osteology of the maxilla and mandible, noting how bone density can impact the effectiveness of local anesthesia.
This document provides an introduction to local anesthesia. It discusses that dentists, not doctors, were responsible for discovering anesthesia due to their motivation to alleviate pain from dental procedures. The first two people to introduce anesthesia were dentists - Horace Wells with nitrous oxide in 1844 and William Morton with ether. Local anesthesia works by preventing the generation and conduction of nerve impulses, setting up a chemical roadblock between the source of pain and the brain. The document then discusses the mechanism of action, factors affecting local anesthetics, and uses and contraindications of local anesthesia.
This document discusses potential complications that can occur after dental extractions and their treatment. It covers hemorrhage, pain and discomfort, edema, trismus, oroantral communications, and soft tissue injuries. The key points are:
1) Initial control of bleeding is achieved by having the patient bite firmly on gauze for 30 minutes. Persistent bleeding may require additional gauze or tea bags.
2) Pain peaks at 12 hours and usually resolves within 2 days with over-the-counter analgesics like ibuprofen.
3) Swelling reaches a maximum at 2 days and usually resolves within a week with ice applications.
1. Complications from dental extractions can include failure of local anesthesia, failure to move the tooth, root fracture, injury to adjacent teeth or structures, bone fractures, root displacement, and losing the tooth in the pharynx or airway.
2. Many complications can be prevented by careful preoperative evaluation and planning, use of proper technique, and knowing the limits of one's own skill level and referring when needed.
3. Common complications are generally manageable but some situations like displaced roots or lost teeth in the airway may require specialty referral.
The document discusses the mechanical principles and steps involved in tooth extraction. It describes how levers, wedges, and the wheel and axle are used to remove teeth. The 5 steps of closed extraction are outlined: 1) loosening soft tissue, 2) luxating with an elevator, 3) adapting forceps, 4) luxating with forceps, 5) removing the tooth with traction. Controlled force and slow, steady pressure on the forceps are emphasized to expand bone and luxate the tooth.
This document describes different dental instruments used for tooth extraction including elevators, periotomes, extraction forceps, and the Physics forceps. It discusses the components and uses of elevators, how periotomes are used to sever the periodontal ligament, variations in extraction forceps designs for different tooth types, and how the Physics forceps utilizes leverage and creep to extract teeth with minimal pressure or force.
1. The document discusses indications and contraindications for tooth extractions. Common indications include severe tooth decay, non-restorable pulpal necrosis, advanced periodontal disease, orthodontic reasons, malpositioned teeth, cracked teeth, impacted teeth, supernumerary teeth, and teeth involved in pathologic lesions or jaw fractures.
2. Absolute contraindications for extraction include uncontrolled metabolic disorders like diabetes, cardiac problems, leukemia, renal failure, and liver disorders. Relative contraindications include conditions where extraction should be deferred until the underlying condition is addressed.
3. Local contraindications include a history of radiation therapy in the extraction site, teeth located near a tumor, pericoronitis, and acute dent
The document discusses principles of dental extractions. It states that the ideal extraction removes the whole tooth with minimal trauma, allowing the wound to heal properly. Extractions can be simple, removing the tooth intact, or complex, requiring the tooth to be sectioned. Clinical evaluation involves assessing access, mobility, crown condition, and root morphology via radiographs. Configuration of roots, presence of pathology, and surrounding bone density are evaluated. Proper patient and surgeon preparation, positioning, and use of instruments and assistance are also reviewed.
- Surgical supplies and equipment used in oral and maxillofacial surgery must be properly sterilized and maintained to prevent the spread of infection between patients and surgical staff. This includes using disposable materials sterilized by the manufacturer and following aseptic techniques.
- The goal of infection control procedures during surgery is to prevent microorganisms from entering the patient's wound. This involves disinfecting surfaces in the operatory, properly preparing the surgical staff, and disposing of contaminated sharps to avoid accidental needle sticks or lacerations.
- There are different levels of preparation depending on the type of procedure, with clean techniques used for basic office surgery and sterile techniques requiring more strict protocols to minimize
1) The document discusses various methods of controlling infection in surgical practice, including identifying pathogens and understanding how the oral microbiome is normally regulated.
2) It then covers specific microorganisms like hepatitis viruses, HIV, mycobacteria, and their modes of transmission.
3) Key concepts and terminology related to infection control are defined, like the differences between sterilization, sanitization, and disinfection. Various sterilization techniques using heat, gas, and chemicals are also described.
The document discusses the process of clinical examination for oral diagnosis. It describes examining the patient's general appearance and vital signs. The extraoral exam involves inspecting and palpating structures like the face, skin, skull and lymph nodes. The intraoral exam assesses the mucosa, dentition, floor of mouth and salivary glands through inspection, palpation, percussion and auscultation. Radiographs, biopsy and laboratory tests provide further diagnostic information. A provisional diagnosis is formed based on the clinical findings to guide treatment planning.
This document provides background on the history of oral surgery and dentistry. It discusses key figures like Hippocrates, Aristotle, Ambrose Pare, and Pierre Fauchard who contributed to the early development of the field. It also outlines the steps involved in making an accurate diagnosis, including taking a thorough patient history, clinical examination, radiological analysis, laboratory tests, and interpreting all findings to arrive at a final diagnosis. Specific components of an effective medical history are described, such as chief complaints, history of present illness, pain assessment using the SOCRATES mnemonic, past dental history, medical history, drug history, and personal/family history.
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3. • Cysts are the most common cause of chronic swellings of the jaws.
They are more common in the jaws than in any other bone
because of the many rests of odontogenic epithelium remaining in
the tissues
• Cyst is define as pathologic cavity within the hard or soft
tissues, lined by epithelium and contain fluid, semifluid or
gas (not created by accumulation of pus) and surrounded
by connective tissue wall or capsule.
4. THE MAIN MECHANISMS OF CYST FORMATION
INVOLVED IN VARYING DEGREES:
1.Proliferation of the epithelial lining and
connective tissue capsule
2. Accumulation of fluid within the cyst
3. the resorption of the surrounding bone
5.
6. CLINICAL FEATURES OF JAW CYST
• Most jaw cysts behave similarly and usually grow slowly
and expansively.
• They differ mainly in their relationship to a tooth, and
the radiographic features are usually a good guide as to
their nature.
• Even when it is not possible to decide the nature of a
cyst, this affects treatment only with less common types.
(keratocysts and unicystic ameloblastomas)
• Diagnosis ultimately depends on histopathology
7. KEY FEATURES OF JAW CYSTS
•Form sharply-defined radiolucencies with smooth
borders
•Fluid may be aspirated and thin-walled cysts may
be transilluminated
•Grow slowly, displacing rather than resorbing
teeth
•Symptomless unless infected and are frequently
chance radiographic findings
8.
9.
10. KEY FEATURES OF JAW CYSTS
•Rarely large enough to cause pathological fracture
•Form compressible and fluctuant swellings if
extending into soft tissues
•Appear bluish when close to the mucosal surface
•Large cysts usually deflect the neurovascular
bundle, paresthesia and anesthesia is rare
15. INTRAOSSEOUS CYST
I. Epithelial Cysts:
Non-odontogenic epithelial origin (Fissural cyst):
Median mandibular
Median palatal
Globulomaxillary
Incisive canal (nasopalatine duct or median anterior
maxillary) cyst
16. INTRAOSSEOUS CYST
II. Non-epithelial cysts: (PSUDOCYST)
Solitary bone cyst (traumatic)
Aneurysmal bone cyst
Stafne’s bone cavity
III. Cyst of the maxillary antrum:
Surgical ciliated cyst of maxilla
Benign mucosal cyst of the maxillary antrum
19. INFLAMMATORY RADICULAR CYST
• The most common of all cysts of odontogenic origin.
(65%- 70%)
• The involved tooth is non-vital.
• The incidence is highest in the anterior maxillary and the
posterior mandibular teeth.
• A round or ovoid shaped radiolucency
• A straw colored or brownish fluid and cholesterol
crystals.
• Treatment: enucleation
25. KERATOCYST
• It is about 5 to 10% of odontogenic cysts of the jaws
• tend to grow in an anteroposterior direction within the
medullary cavity and clinically observable expansion of
the bone occurs late
• The teeth adjoining the cyst are vital
• can be unilocular or multilocular
• contain cheesy like material
29. RECURRENCE OF KERATOCYST
•The recurrence rate may vary from 5-60% with
most occurring in the first 5 years.
•Some of the possible reasons reported for
recurrence are as follows:
1. Presence of satellite cysts,
2. Cystic lining is very thin and fragile,
3. portions of which may be left behind
35. DENTIGEROUS CYST
• Results because of the enlargement of the follicular
space of the whole or part of the crown of an impacted
or unerupted tooth and is attached to the neck of the
tooth.
• More common than the keratocyst but less common
than the inflammatory types
• dentigerous cysts have a higher tendency to cause root
resorption of adjacent teeth
• Treatment: marsupialization in children and enucleation
in adult
38. Gingival cyst of infants
• (Bohn nodules) up to 80% of newborn infants have small
nodules or cysts in the gingivae Most resolve spontaneously
• Cysts (Epstein's pearls) may also arise from nonodontogenic
epithelium along the midpalatine raphe. also resolve
spontaneously in a matter of months.
Gingival cysts of adults
• Gingival cysts are exceedingly rare. They usually form after
the age of approximately 40. Clinically, they form dome-
shaped swellings less than 1 cm in diameter and sometimes
erode the underlying bone. They are unlikely to recur after
enucleation
41. FISSURAL CYSTS
•Fissural cysts are non-odontogenic cysts that
arise owning to epithelial inclusions or
entrapments in the lines of fusion of the
developing facial processes during the
embryonic stage.
48. SOLITARY BONE CYST
& ANEURYSMAL BONE CYST
•Solitary bone cyst and aneurysmal bone cysts are
without epithelial lining (pseudocysts). These also
are cystic only in their radiographic appearances
•It is not confined to the jaws as similar lesions are
seen elsewhere in the skeleton
•Mainly due to trauma
•It is seen mainly in children, adolescents or young
adults
51. STAFNE BONE CAVITY
•Stafne's bone cavity is not a cyst, it is included
because of their clinical similarity to cysts of the
jaw bones
•due to failure of the normal deposition of bone
during development of the jaws
•Below the inferior alveolar canal, mainly unilateral
•No treatment
53. MUCOCELE & RANULA
• Two types of distinct entities described are, the true
retention cyst which is lined by epithelium and the other
is the mucous extravasation cyst which occurs because
of the pooling of mucus, it does not have any epithelial
lining and is surrounded by connective tissue cells
• Majority of mucocele are seen to affect the lower lip
• Ranula is variant of a mucocele that is present on the
floor of the mouth, beneath the tongue.
• Treatment: They are best treated by surgical excision
together with the associated salivary gland
61. DERMOID AND EPIDERMOID CYSTS
• The dermoid cyst is a form of cystic teratoma, which is lined
by epithelium and in addition reveals the presence of skin
appendages, e.g. hair, sebaceous glands or teeth. The
epidermoid cyst is also lined by epithelium but does not
contain any skin appendages.
• Dermoid are seen in the midline in the floor of the mouth
above or below the geniohyoid muscle
• Those above the geniohyoid muscle, elevate the tongue,
causing difficulty with mastication and speech. Those
present inferior to the geniohyoid muscle, cause a
submental swelling that has been described as a double
chin
65. THYROGLOSSAL DUCT CYST
• It can occur anywhere in the midline along the course of
the embryonic thyroglossal duct,
(which extends from the foramen caecum of the tongue
into the deep fascia near the thyroid isthmus)
• Pathognomonic is the movement of the cyst during
swallowing and protrusion of the tongue
• Complete radical surgical excision of the cyst along with
its tract is essential to prevent recurrence.
70. CYSTIC HYGROMA
• it is a developmental abnormality, in which cavernous lymphatic
spaces communicate and form large thin walled cysts
• Typically they appear in the first few months of life
• swelling that is painless and compressible, the swelling may
increase in size over several months to a year, and they may
remain static or may even regress
• overlying skin may be bluish and transillumination will be
positive
• Hygroma may suddenly enlarge when there is an upper
respiratory infection. This may cause acute respiratory
obstruction and may require an emergency tracheostomy or
surgical extirpation
74. Cysts of the jaws, may be treated by one of the following basic
methods:
1. Marsupialization (decompression)
• Partsch I
• Partsch II (combined marsupialization and encleation)
• Marsupialization by opening into nose or antrum.
2. Enucleation:
• Enucleation and packing
• Enucleation and primary closure
• Enucleation and primary closure with reconstruction/bone
grafting.
GENERAL PRINCIPLES OF TREATMENT
81. ADVANTAGES OF MARSUPIALIZATION
1. Simple procedure to perform and reduces operating
time
2. Spares vital structures and prevents oronasal and
oroantral fistulae
3. Alveolar ridge is preserved and allows eruption of
teeth
4. Helps shrinkage of cystic lining and prevents
pathological fracture
5. Allows for endosteal bone formation to take place.
82. DISADVANTAGES OF MARSUPIALIZATION
1. Pathologic tissue is left in situ
2.Histologic examination of the entire cystic lining is not
done
3. Prolonged healing time and prolonged followup visits
4. Periodic changing of pack, periodic irrigation of cavity
5. Inconvenience to the patient
6. Secondary surgery needed
7. Formation of slit-like pockets that may harbor foodstuffs
83. MODIFICATIONS OF MARSUPIALIZATION PARTSCH II
this is a two-stage technique that combines
the two standard procedures, in which, first
marsupialization is performed and at a later
stage when the cavity becomes smaller, the
procedure of enucleation is performed and
the entire tissue is examined
histopathologically
93. ENUCLEATION
Advantages:
1. Primary closure of the wound
2. Healing is rapid and postoperative care is
reduced
3. Thorough examination of the entire cystic lining
can be done.
94. ENUCLEATION
Disadvantages:
1. In young persons, the unerupted teeth in a
dentigerous cyst will be removed with the lesion
2. Removal of large cysts will weaken the mandible,
making it prone to jaw fracture
3. Damage to adjacent vital structures
4. Pulpal necrosis.
95. COMPLICATIONS OF CYSTIC LESIONS
1. Pathological fracture
2. Postoperative wound dehiscence
3. Loss of teeth vitality
4. Neuropraxia in infected cysts
5. Postoperative infection
6. Recurrence in some cysts
7. Dysplastic, neoplastic or even malignant changes
97. SUGGESTED FOLLOW-UP:
• Long-term follow-up, at least up to 8 years for keratocysts
for early detection of dealing with any recurrence
• To check postoperative vitality of teeth
• Unerupted teeth that may require orthodontic assistance for
eruption
• Orthodontic assistance for alignment of displaced teeth
• Long-term follow-up of patients with Gorlin's syndrome.