Inflammatory Principles & Athletic Rehab
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The document summarizes the inflammatory process and its phases: 1) The acute/early inflammatory phase involves vasodilation, increased permeability and edema, phagocytosis by immune cells to remove debris. 2) The proliferation phase involves tissue regeneration through re-epithelialization, angiogenesis and fibroplasia to form new scar tissue. 3) During the remodeling phase lasting up to a year, collagen is reorganized and strengthened to gradually restore function.
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The document discusses various aspects of inflammation and repair. It describes the signs of acute inflammation as redness, heat, swelling, pain, and loss of function. It then covers the events in acute inflammation, including neurologic events like vasoconstriction and vasodilation, hemodynamic events such as increased permeability and slowing of blood flow, and cellular events like margination and emigration of leukocytes. Chronic inflammation and types of inflammation based on location and exudates are also summarized. The document concludes by discussing repair through granulation tissue formation, remodeling and fibrosis.
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inflammatoryprocess-121015204914-phpapp02.pdf
The inflammatory process occurs in three phases:
1) The acute/early phase involves vasodilation, chemical mediator release, swelling, and phagocytosis within the first 2-4 days.
2) The tissue formation/proliferation phase lasts 2-3 weeks as new tissue is rebuilt through granulation, collagen deposition, and vascularization.
3) The remodeling phase can last up to 1 year as the tissue is strengthened and remodeled with increased collagen cross-linking and organization.
Inflammatory process
1) Inflammation is the body's response to injury and involves redness, swelling, pain, warmth, and loss of function.
2) The inflammatory process has three phases - the acute/early phase aims to remove harmful stimuli and begin healing; the proliferation phase involves new tissue formation; and the remodeling phase adapts the new tissue.
3) Key events in the early inflammatory phase include increased blood flow due to chemical mediators, fluid leakage causing swelling, and the arrival of white blood cells to remove debris.
The inflammatory process
The document summarizes the inflammatory process in three phases:
Phase I is the acute phase lasting 1-2 weeks involving vascular responses, swelling, pain, heat, and loss of function due to chemical mediators. Phagocytosis occurs as leukocytes remove dead tissue.
Phase II is the tissue formation phase lasting 2-3 weeks involving regeneration, collagen synthesis, wound contraction, and vascularization.
Phase III is the remodeling phase lasting up to 1 year involving collagen remodeling and scar maturation to adapt to original tissue. Treatment planning focuses on controlling inflammation, encouraging repair, and functional rehabilitation.
woundhealing-190112152216 (1).pdf
1. Wound healing occurs in 3 phases: inflammatory, proliferative, and remodeling. In the inflammatory phase, hemostasis and an inflammatory response begin the healing process. In the proliferative phase, new tissue such as granulation tissue and blood vessels form. In the remodeling phase, the wound gains strength through collagen deposition and remodeling.
2. Wounds can heal through primary intention with wound edges in direct apposition or secondary intention with gap formation. Primary intention involves direct regeneration while secondary intention uses granulation tissue.
3. Factors that can influence healing include nutrition, infection, wound size/location, and metabolic status. Complications include deficient healing, excessive scarring, and exaggerated wound contraction.
Wound healing
This document provides an overview of the three phases of wound healing: inflammatory, proliferative, and remodeling. It describes the key cellular and molecular processes that occur in each phase, including hemostasis, inflammation, granulation tissue formation, angiogenesis, epithelialization, and collagen deposition/remodeling. Complications of wound healing like deficient or excessive scarring, dehiscence, and exaggerated contraction are also summarized. The roles of various growth factors like PDGF, EGF, VEGF, and cytokines in stimulating and orchestrating wound repair are highlighted. Factors that can delay or impair healing, such as infection, poor nutrition, diabetes, and large wound size/location are also reviewed.
Woundhealing drneerajjain
The document discusses wound healing and its three phases: inflammatory, proliferative, and remodeling. It defines wound healing and the two processes of tissue regeneration and scar formation. The inflammatory phase involves hemostasis, recruitment of inflammatory cells, and late events like lymphocyte and mast cell entry. The proliferative phase involves granulation tissue formation, angiogenesis, contraction, and epithelialization. Remodeling involves the formation of new collagen to increase tensile strength over months. Factors influencing healing and potential complications are also outlined.
Inflammation lecture
Inflammation is the body's immune response to infection or injury. It involves increased blood flow, migration of white blood cells, and other vascular and cellular changes. Acute inflammation resolves quickly, while chronic inflammation persists long-term due to ongoing infection, toxin exposure, or autoimmune disease. Tissue repair after injury occurs through regeneration of original cell types or scar formation involving fibroblasts and collagen deposition. Factors like nutrition, infection, and wound location influence the healing process.
Wound healings
The document discusses the three main phases of wound healing: inflammatory, proliferative, and remodeling. In the inflammatory phase, blood vessels constrict and immune cells are recruited to fight infection. The proliferative phase involves new tissue growth through granulation, collagen deposition, angiogenesis and epithelialization. Finally, the remodeling phase lasts for months as the scar matures and collagen is reorganized. Through these coordinated phases, the body works to close and resurface wounds.
Inflammation reza2003 e video
The document defines inflammation and describes the key aspects of acute and chronic inflammation. It discusses the cardinal signs of acute inflammation, potential causes, and the main events in the inflammatory response, including vascular changes, exudation of fluid and cells, and cellular events like leukocyte extravasation and phagocytosis. Acute inflammation is a rapid local response lasting days, while chronic inflammation persists for weeks to months with simultaneous tissue destruction and healing.
INFLAMMATION.pptx
Inflammation is defined as the local response of tissues to injury or infection. It is characterized by redness, swelling, heat, pain, and loss of function. The inflammatory response involves vascular changes like increased blood flow and permeability, as well as cellular responses. White blood cells like neutrophils and macrophages migrate to the site of injury to remove infectious agents and damaged tissue through phagocytosis. Acute inflammation resolves quickly, while chronic inflammation persists long-term and can cause tissue damage.
Lect 6-wound healing
The document discusses wound healing and regeneration. It describes the three phases of wound healing as the inflammatory phase, proliferative phase, and remodeling phase. The inflammatory phase involves hemostasis, inflammation, and vasodilation. The proliferative phase involves granulation, angiogenesis, contraction, and epithelialization. Finally, the remodeling phase can last up to two years as new collagen is formed and wound strength increases.
Process of Inflammation & Repair.pptx
Inflammation is the body's protective response to injury or infection. It involves both vascular and cellular events at the site of injury. The basic mechanism of inflammation includes increased blood flow, vascular permeability, exudation of fluid and cells, and changes in other tissues. Chemical mediators like histamine, bradykinin, prostaglandins, leukotrienes, and cytokines are released during inflammation to regulate the inflammatory response. Both acute and chronic inflammation aim to eliminate the initial cause of cell injury, clear out necrotic cells and tissues from the site of injury, and initiate the repair process.
Inflammation and Repair
Inflammation is the body's response to injury or infection that involves vascular and cellular events. The cardinal signs of inflammation are redness, swelling, heat, pain, and loss of function. Inflammation can be either acute (minutes to days) or chronic (weeks to months). Chemical mediators like histamine, prostaglandins, and leukotrienes are released during inflammation and cause changes like increased vascular permeability and leukocyte migration. Repair after inflammation occurs through regeneration of tissues like skin, or through healing by scar formation. Healing involves granulation tissue formation, angiogenesis, fibroblast proliferation and extracellular matrix deposition.
INFLAMMATION & WOUD HEALING..pptx
This document discusses inflammation and wound healing. It begins by defining inflammation and describing the cardinal signs. It then discusses the causes and types of inflammation. The sequence of events in acute inflammation is explained, including vascular changes like vasodilation and increased permeability, and cellular events like leukocyte recruitment and activation. Chemical mediators are also discussed. Outcomes of acute inflammation and systemic signs are summarized. Finally, the document covers factors influencing wound healing, the process of cutaneous wound healing, and potential complications.
Inflammation and wound healing. Pathophysiology
Inflammation and healing.
B.Pharm 2nd Sem Pathophysiology as per PCI.
wound healing [Autosaved].pptx
Surgical wounds can be classified based on cleanliness from clean to dirty. Wound healing involves three phases - inflammatory, proliferative, and remodeling. The inflammatory phase begins immediately after injury and involves hemostasis and inflammation. The proliferative phase lasts 2-3 weeks and involves granulation tissue formation, angiogenesis, and collagen deposition. During the remodeling phase, collagen matures and wound strength increases. Factors like nutrition, infection, and underlying illness can influence wound healing. Different tissues like bone, nerve, and tendon heal through similar phases but with tissue-specific processes like callus formation or nerve regeneration. Wound closure techniques include primary intention, secondary intention, and tertiary intention.
Inflammation
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Inflammation
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Inflammation and Cellular Responses
Inflammation is the body's protective response to injury or infection. It involves vascular changes such as increased blood flow and permeability, cellular responses like leukocyte migration, and metabolic changes. The classic signs of inflammation are redness, swelling, heat, pain, and loss of function. Mediators like cytokines and chemokines are released from injured cells to initiate cascades that promote inflammation and activate the coagulation, complement, and nitric oxide pathways. After the initial response, healing processes begin through cell proliferation, angiogenesis, phagocytosis, and tissue repair or fibrosis. Growth factors orchestrate healing by attracting cells and causing proliferation.
Inflamggvggffgggggggggggmation lecture.ppt
Inflammation is the body's protective response to eliminate the initial cause of cell injury and promote tissue repair. The classical signs of acute inflammation are heat, redness, swelling, pain, and loss of function. Acute inflammation is short-lived and involves increased blood flow, vascular permeability, and recruitment of immune cells like neutrophils to the injured site. Chronic inflammation lasts longer and is characterized by lymphocytes, plasma cells and macrophages, tissue destruction, and fibrosis. The outcome of acute inflammation is typically resolution and healing of the damaged tissues.
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Kin 188 Tissue Healing
1. The document discusses the phases of soft tissue healing (inflammatory, repair/regeneration, remodeling/maturation), fracture healing (inflammation and hematoma formation, cellular proliferation, callous formation, bony union/ossification, remodeling), and nerve healing.
2. The inflammatory phase involves redness, pain, warmth, swelling and limited range of motion as the body responds to tissue damage. The repair phase sees new blood vessel growth and fibroblast activity forming new connective tissue. Remodeling shapes the scar tissue.
3. Fracture healing similarly progresses from inflammation and clot formation to new bone growth bridging the fracture via a callous before remodeling the bone. Nerve
Kin 189 Characteristics Of A Profession, Contemporary Issues
The document discusses the characteristics of an athletic training profession and some contemporary issues in athletic training. It outlines six generally accepted characteristics of a profession including theoretical knowledge, training and education, certification, adherence to a code of ethics, service to the public good, and organization. It then provides details on how athletic training meets these characteristics through its skill-based theoretical knowledge, education and certification requirements, code of ethics, service to both traditional and new patient populations, and professional organizations. Finally, it lists some contemporary issues in athletic training such as changes in regulation, education, employment settings, and position statements on various health and safety topics.
Kin 189 Protective Equipment
This document discusses protective equipment used in sports to prevent and care for athletic injuries. It covers materials used for protective splints and braces, including properties like density, strength, rigidity, and durability. It also describes protective gear for different body parts, including the head, face, trunk, upper and lower extremities. Common protective equipment includes helmets, mouthguards, shoulder pads, shin guards, and ankle braces. Factors in choosing appropriate equipment include the type of sport, positions played, and individual anatomical considerations.
Kin 188 Wrist And Hand Evaluation And Injuries
This document provides an overview of wrist and hand anatomy, evaluation, and common injuries. It describes the bony anatomy including carpals, metacarpals, and phalanges. Joint anatomy including the radiocarpal joint and intercarpal joints is also covered. Common injuries discussed are wrist sprains, distal radius fractures, scaphoid fractures, boxer's fractures, mallet finger, and gamekeeper's thumb. The evaluation section outlines taking a history, physical exam including range of motion and special tests, and assessing neurovascular status.
Kin 188 Thoracic And Abdominal Evaluation And Injuries
This document provides an overview of anatomy and common injuries of the thorax, abdomen, and associated organ systems. It describes the bony, muscular, respiratory, cardiovascular, digestive, genitourinary, and reproductive anatomy. Evaluation methods like history, inspection, palpation, and special tests are outlined. Common thoracic injuries discussed are rib fractures, pneumothorax, and hemothorax. Abdominal injuries covered include liver, spleen, kidney, and reproductive organ injuries. Appendicitis, testicular torsion, and conditions like the female athlete triad are also summarized.
Kin 188 Therapeutic Modalities And Rehabilitation
Therapeutic modalities and rehabilitation are used to treat athletic injuries. Modalities like ice, heat, ultrasound and electrical stimulation are applied based on the injury, severity, and doctor's prescription to reduce pain and swelling and optimize healing. Rehabilitation follows phases from immobilization and limiting activity to regaining range of motion and strength through therapeutic exercises and sport-specific drills before returning to play, with maintenance exercises after to prevent re-injury.
Kin 188 Standards Of Practice, Legal And Ethical Considerations
Standards of practice guide athletic trainers' actions and promote high ethical standards. Athletic trainers must provide competent care, respect patients' rights and privacy, and avoid conflicts of interest. They are responsible for understanding legal concepts like negligence, informed consent, and liability to minimize risks to patients and litigation risks. Documentation and safety protocols help ensure care meets the standard of care.
Kin 188 Shoulder Evaluation And Injuries
The document provides an overview of shoulder anatomy, evaluation, and common injuries. It describes the bony structures, articulations, muscles, neurological components, and vascular supply of the shoulder. Evaluation involves taking a history, visual inspection, palpation of structures, range of motion testing, and special tests. Common injuries discussed are to the sternoclavicular joint, acromioclavicular joint, glenohumeral joint, rotator cuff, biceps tendon, and fractures of the scapula, clavicle, and humerus.
Kin 188 Record Keeping
1) Medical records for student athletes must be kept confidential following HIPAA and FERPA regulations, stored securely with limited access.
2) Injury evaluations and progress notes should follow a SOAP note format and be updated at each follow up evaluation to document the treatment plan and progress.
3) Insurance billing requires accurate diagnostic and procedural codes on forms to maximize reimbursement for services provided.
Kin 188 Protective Equipment
This document discusses principles of protective equipment used in sports. It provides details on the types of materials used to manufacture protective gear and how different materials can help absorb impacts and protect the body. Guidelines are presented on properly fitting and inspecting football helmets and shoulder pads to ensure they provide maximum protection according to safety standards.
Kin 188 Pre Participation Physical Examinations
The document discusses pre-participation physical examinations (PPE) for athletes. The goals of a PPE are to evaluate an athlete's general health, detect any medical conditions that could increase injury risk, and establish baseline health measurements. A PPE typically involves collecting a medical history, conducting a physical exam checking various body systems, and clearing the athlete for participation or making recommendations. The physical exam covers vital signs, musculoskeletal/neurological functions, and screening for common health issues. Laboratory tests are not usually required but may be used if issues arise during the exam.
Kin 188 Mechanisms And Types Of Injuries
The document discusses various mechanisms and types of athletic injuries. It describes how force, torque, stress and strain can cause soft tissue injuries like contusions, strains and sprains if they exceed the tissue's tolerance. Bone injuries include fractures from excessive force or repeated stress. Nerve injuries can occur from tension or compression and range from temporary neuropraxia to complete neurotmesis.
Kin 188 Knee Injuries And Evaluation
The document discusses knee anatomy, including bony structures like the femur and tibia, ligaments like the ACL and PCL, and muscles like the quadriceps and hamstrings. It outlines the evaluation process for knee injuries, including taking a history of the mechanism of injury, inspecting for deformities or swelling, and performing range of motion and special tests. Common knee injuries are then reviewed such as ACL tears from rotational forces, PCL injuries from falls on a flexed knee, MCL sprains from valgus stress, and meniscal tears that can cause clicking or locking.
Kin 188 Introduction To Athletic Training
The document provides an overview of the roles and responsibilities of members of a sports medicine team, including certified athletic trainers. It discusses the history and development of the profession of athletic training and the National Athletic Trainers' Association. The document also outlines the requirements to become a certified athletic trainer and the domains of practice for athletic trainers.
Kin 188 Injury Evaluation Procedures
The document outlines the procedures for clinically evaluating athletic injuries, including obtaining a history, inspecting and observing the injured area, palpating for things like swelling or muscle spasms, and performing special tests to assess range of motion, ligament integrity, and neurovascular function. The evaluation aims to gather both subjective information from the patient and objective findings from inspection and examination. It also discusses differences between evaluations in a clinical setting versus on the field during a game or practice.
Kin 188 Head And Neck Evaluation And Injuries
This document discusses head and neck injuries, including concussions, skull fractures, cervical spine injuries, and spinal cord injuries. It covers anatomy of the head and neck, evaluation of conscious and unconscious athletes, common injuries, return to play criteria, and special tests to assess range of motion, balance, and cognition. Cervical fractures can cause quadriplegia if the spinal cord is compressed, and second impact syndrome occurs when an athlete sustains a second concussion before symptoms of the first have resolved.
Kin 188 Hip And Thigh Evaluation And Injuries
This document provides an overview of hip and thigh anatomy, evaluation, and common injuries. It describes the bony anatomy of the hip including the acetabulum, femur head, and surrounding ligaments. Evaluation involves taking a thorough history, inspecting for deformities, palpating muscles and bones, and performing range of motion and special tests. Common injuries discussed include muscle strains from overuse, stress fractures, hip dislocations, and bursitis.
Kin 188 Foot, Ankle And Leg Injuries
This document provides an overview of foot, ankle, and leg anatomy, evaluation, and common injuries for athletic trainers. It describes the bony, muscular, and ligamentous structures of the foot, ankle, and leg. Evaluation involves taking a thorough history, visual inspection, palpation, range of motion and special tests. Common foot injuries include bunions, turf toe, and stress fractures. Ankle injuries include sprains, syndesmosis sprains, and fractures. Leg injuries include compartment syndrome, Achilles tendon ruptures, muscle strains, and fractures.
Kin 188 General Medical Conditions
This document provides an overview of various medical conditions that are relevant for the prevention and care of athletic injuries, including:
1. Cardiovascular conditions like hypertension, hypotension, sudden cardiac death from conditions like hypertrophic cardiomyopathy, and non-cardiac causes like substance abuse.
2. Blood and lymph disorders such as anemia, sickle cell anemia, hemophilia, and lymphangitis.
3. Neurological conditions including headaches, seizure disorders, meningitis, and encephalitis.
4. Respiratory tract infections and conditions affecting the upper respiratory tract and lungs, like the common cold, sinusitis, bronchitis, and asthma.
Kin 188 Environmental Conditions
The document discusses various environmental conditions and injuries related to heat and cold exposure. It covers topics like heat regulation, heat illness symptoms and treatments, measuring heat stress, cold-related conditions and injuries like frostbite, and hypothermia symptoms. Prevention and management strategies are provided for conditions like heat exhaustion, heat stroke, frostbite, and hypothermia. Factors that can modify heat tolerance like acclimatization, fluid intake, electrolyte replacement, clothing, and age are also reviewed.
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Mind map of terminologies used in context of Generative AI
Mind map of terminologies used in context of Generative AI
Inflammatory Principles & Athletic Rehab
- 1. Inflammatory Process KIN 195
- 12. Phase I -Early Phase: Acute Inflammation
- 13. Inflammatory Phases Chart Designates Percent of phase over time
- 23. Phase I: Late Phase Blood Clotting Factor X Prothrombin Thrombin Fibrogen and Thrombin Meet Fibrin Monomer Fibrin Mesh Fibrin Forms Seal
- 37. Summary
- 39. Treatment Planning for Phases of Tissue Healing
- 41. Treatment Planning: Maturation Phase