Bleeding disorders /certified fixed orthodontic courses by Indian dental aca...Indian dental academy
Welcome to Indian Dental Academy
The Indian Dental Academy is the Leader in continuing dental education , training dentists in all aspects of dentistry and offering a wide range of dental certified courses in different formats.
Indian dental academy has a unique training program & curriculum that provides students with exceptional clinical skills and enabling them to return to their office with high level confidence and start treating patients
State of the art comprehensive training-Faculty of world wide repute &Very affordable.
Platelet and coagulation post graduate lecture Monkez M Yousif
This lecture is prepared for postgraduate students in Internal medicine. It presents a physiologic and basic background of the process of homeostasis followed by a practical approach to diagnosis and brief information of different causes of bleeding disorders
Bleeding disorders /certified fixed orthodontic courses by Indian dental aca...Indian dental academy
Welcome to Indian Dental Academy
The Indian Dental Academy is the Leader in continuing dental education , training dentists in all aspects of dentistry and offering a wide range of dental certified courses in different formats.
Indian dental academy has a unique training program & curriculum that provides students with exceptional clinical skills and enabling them to return to their office with high level confidence and start treating patients
State of the art comprehensive training-Faculty of world wide repute &Very affordable.
Platelet and coagulation post graduate lecture Monkez M Yousif
This lecture is prepared for postgraduate students in Internal medicine. It presents a physiologic and basic background of the process of homeostasis followed by a practical approach to diagnosis and brief information of different causes of bleeding disorders
Investigation of bleeding disorder || bleeding disorderparveen singh
this is a topic on investigation of bleeding disorder.
This may result from:
1 Vascular disorders
a] Thrombocytopenia
2Platelet Disorder
b] Defective platelet function
3Defective coagulation
4Defective Fibrinolysis
it is due to
-Inherited bleeding disorders
-Acquired bleeding disorders
investigation include:
first line test {basic test daily done in coagulation lab}
second line test {some important test done whenever all first line investigation test are normal }
Main Credit Goes To__-----___--- nitin dudeja {senior}
The Indian Dental Academy is the Leader in continuing dental education , training dentists in all aspects of dentistry and
offering a wide range of dental certified courses in different formats.for more details please visit
www.indiandentalacademy.com
Bleeding Disorders: Causes, Types, and Diagnosis Dr Medical
https://userupload.net/wxvqfbo7ywqu
A bleeding disorder is a condition that affects the way your blood normally clots. The clotting process, also known as coagulation, changes blood from a liquid to a solid. When you’re injured, your blood normally begins to clot to prevent a massive loss of blood. Sometimes, certain conditions prevent blood from clotting properly, which can result in heavy or prolonged bleeding.
Bleeding disorders can cause abnormal bleeding both outside and inside the body. Some disorders can drastically increase the amount of blood leaving your body. Others cause bleeding to occur under the skin or in vital organs, such as the brain.
Investigation of bleeding disorder || bleeding disorderparveen singh
this is a topic on investigation of bleeding disorder.
This may result from:
1 Vascular disorders
a] Thrombocytopenia
2Platelet Disorder
b] Defective platelet function
3Defective coagulation
4Defective Fibrinolysis
it is due to
-Inherited bleeding disorders
-Acquired bleeding disorders
investigation include:
first line test {basic test daily done in coagulation lab}
second line test {some important test done whenever all first line investigation test are normal }
Main Credit Goes To__-----___--- nitin dudeja {senior}
The Indian Dental Academy is the Leader in continuing dental education , training dentists in all aspects of dentistry and
offering a wide range of dental certified courses in different formats.for more details please visit
www.indiandentalacademy.com
Bleeding Disorders: Causes, Types, and Diagnosis Dr Medical
https://userupload.net/wxvqfbo7ywqu
A bleeding disorder is a condition that affects the way your blood normally clots. The clotting process, also known as coagulation, changes blood from a liquid to a solid. When you’re injured, your blood normally begins to clot to prevent a massive loss of blood. Sometimes, certain conditions prevent blood from clotting properly, which can result in heavy or prolonged bleeding.
Bleeding disorders can cause abnormal bleeding both outside and inside the body. Some disorders can drastically increase the amount of blood leaving your body. Others cause bleeding to occur under the skin or in vital organs, such as the brain.
Cardiogenicshock by Dr.Afroza Prioty -140123092109-phpapp02Afroza Prioty
A small overview on cardiogenic shock which sometimes becomes a burning issue for the medical personnels and to combat the situation, the measures should be taken immediately and urgently.
It gives information regarding indication and different routes adopted for blood transfusion as well as merits and demerits of different routes adopted for blood transfusion in animals.
2. Hemmorhage is defined as excessiveHemmorhage is defined as excessive
escape of blood outside the blood vessel.escape of blood outside the blood vessel.
Based on vessel bleedingBased on vessel bleeding
Artery veinArtery vein
4. Primary haemorrhagePrimary haemorrhage
It refers to the escape of blood at the timeIt refers to the escape of blood at the time
of injuryof injury
Reactionary haemorrhageReactionary haemorrhage
It occurs with in first 24 hrsIt occurs with in first 24 hrs
It is due to displacement of blood clotIt is due to displacement of blood clot
5. Secondary haemorrhageSecondary haemorrhage
It occurs after 7 to 10 days after injuryIt occurs after 7 to 10 days after injury
It is due to sepsisIt is due to sepsis
acid pusacid pus
Digestion of vessel wallDigestion of vessel wall
6. Clinical signs and symptomsClinical signs and symptoms
DizzinessDizziness
BleedingBleeding
Rapid heart rate and respiratory rateRapid heart rate and respiratory rate
7. INTERNAL BLEEDING- Bleeding with inINTERNAL BLEEDING- Bleeding with in
body cavitybody cavity
EXTERNAL BLEEDING-Blood escapingEXTERNAL BLEEDING-Blood escaping
through wound in skinthrough wound in skin
SPONTANEOUS BLEEDING- patient onSPONTANEOUS BLEEDING- patient on
oral hypoglceamic agents, heriditaryoral hypoglceamic agents, heriditary
coagulopathy.coagulopathy.
8. Steps in haemostasisSteps in haemostasis
vasoconstrictionvasoconstriction
platelet plug &primary haemostasisplatelet plug &primary haemostasis
activation of clotting mechanismactivation of clotting mechanism
secondary haemostasissecondary haemostasis
fibrous organisationfibrous organisation
9. Primary haemostasisPrimary haemostasis
It is a process of platelet plug formationIt is a process of platelet plug formation
It occurs with in seconds of injuryIt occurs with in seconds of injury
important in stopping of blood from vesselimportant in stopping of blood from vessel
Release of granules and plateletRelease of granules and platelet
aggregationaggregation
10. Secondary hemostasisSecondary hemostasis
It occurs via clotting process.It occurs via clotting process.
Reaction 1. intrinsic phaseReaction 1. intrinsic phase
factors 7, 9, 11, 12,along with calcium &factors 7, 9, 11, 12,along with calcium &
plasma protein(PTT)plasma protein(PTT)
Reaction 2. release of tissue thromboplastinReaction 2. release of tissue thromboplastin
(PT)(PT)
Reaction 10. activation of factor 10.Reaction 10. activation of factor 10.
11. Reaction 4. conversion of prothrombin toReaction 4. conversion of prothrombin to
thrombin(factor 5)thrombin(factor 5)
Fibrinogen – fibrin (thrombin)Fibrinogen – fibrin (thrombin)
19. Ligation of major vesselsLigation of major vessels
External carotid and its branchesExternal carotid and its branches
Ligation of greater palatine vesselLigation of greater palatine vessel
20. shockshock
It is a pathophyisiological conditionIt is a pathophyisiological condition
,clinically recogonized as a state of in,clinically recogonized as a state of in
adequate perfusion.adequate perfusion.
24. Basic haemodynamic mechanismBasic haemodynamic mechanism
Reduced circulating blood volumeReduced circulating blood volume
Decreased venous return to heartDecreased venous return to heart
Decreased cardiac out putDecreased cardiac out put
Reduced blood flow through tissuesReduced blood flow through tissues
Tissue hypoxiaTissue hypoxia
AcidosisAcidosis
25. HYPOVOLEMIC SHOCKHYPOVOLEMIC SHOCK
in circulating blood volumein circulating blood volume
Hypovolemic shockHypovolemic shock
Haemorrhagic Non haemorrhagicHaemorrhagic Non haemorrhagic
Example (fluid loss vomiting burns)Example (fluid loss vomiting burns)
26. Inadequate cardiac outputInadequate cardiac output
Impaired OImpaired O22 delivery and reduced tissue perfusiondelivery and reduced tissue perfusion
Causes-Causes-
Loss of effective contractile function of myocardiumLoss of effective contractile function of myocardium
Reducing adequate forward outputReducing adequate forward output
Cardiogenic shockCardiogenic shock
27. Clinical feature –Clinical feature –
Increase pulmonary artery wedge pressureIncrease pulmonary artery wedge pressure
Decrease cardiac our putDecrease cardiac our put
Increase peripheral vascular resistanceIncrease peripheral vascular resistance
Less mean arterial presser (<60 mmHg)Less mean arterial presser (<60 mmHg)
Management –Management –
Identifying the causeIdentifying the cause
Dopamine (vasopressor), norepinephrine andDopamine (vasopressor), norepinephrine and
doputaminedoputamine
28. Neurogenic shockNeurogenic shock
In neurogenic shock vascular capacity increasesIn neurogenic shock vascular capacity increases
so much that even the normal amount of bloodso much that even the normal amount of blood
becomes incapable of adequately filling thebecomes incapable of adequately filling the
circulatory system.circulatory system.
29. Early Signs and symptoms –Early Signs and symptoms –
Pale skinPale skin
PerspirationPerspiration
NauseaNausea
TachycardiaTachycardia
Feeling of warmth in neck or faceFeeling of warmth in neck or face
30. Late symptoms –Late symptoms –
Coldness in hands and feetColdness in hands and feet
HypotensionHypotension
BradycardiaBradycardia
DizzinessDizziness
Visual disturbanceVisual disturbance
Pupillary dilationPupillary dilation
Hyperpnoea andHyperpnoea and
loss of consciousnessloss of consciousness
31. Septic shockSeptic shock
Due to cell membrane endothelial injuryDue to cell membrane endothelial injury
Peripheral vasodilatationPeripheral vasodilatation
Example Ecoli, klebsiellaExample Ecoli, klebsiella
33. Management –Management –
Early and effective volume replacementEarly and effective volume replacement
Restoration of tissue perfusionRestoration of tissue perfusion
Adequate OAdequate O22 supply to cellssupply to cells
AntibioticsAntibiotics