2. MEANINGMEANING
Results fromResults from
ischemia to a partischemia to a part
of the brain orof the brain or
hemorrhage into thehemorrhage into the
brain that results inbrain that results in
death of brain cellsdeath of brain cells..
3. ISCHEMIC STROKE:ISCHEMIC STROKE:
An ischemic stroke or brain attack is a suddenAn ischemic stroke or brain attack is a sudden
loss of function resulting from disruption of bloodloss of function resulting from disruption of blood
supply to a part of brain.supply to a part of brain.
HEMORRHAGIC STROKE:HEMORRHAGIC STROKE:
It is the bleeding in to the brain tissue,It is the bleeding in to the brain tissue,
ventricles or the subarachnoid space it is mainlyventricles or the subarachnoid space it is mainly
by the rupture of small blood vessels in the brain.by the rupture of small blood vessels in the brain.
INCIDENCE:INCIDENCE:
85% of cerebral disorders are ischemic stroke.85% of cerebral disorders are ischemic stroke.
15% of cerebral disorders are hemorrhagic15% of cerebral disorders are hemorrhagic
stroke.stroke.
Occurs in the people between the ages 50 and 70Occurs in the people between the ages 50 and 70
4. CAUSES:CAUSES:
INTRACEREBRAL BLEED:INTRACEREBRAL BLEED:
It is mainly caused by the rupture of cerebral vessel thatIt is mainly caused by the rupture of cerebral vessel that
bleeds in to the brain tissue.bleeds in to the brain tissue.
HypertensionHypertension
AtherosclerosisAtherosclerosis
Neoplasm'sNeoplasm's
Dural sinus thrombosisDural sinus thrombosis
Congenital angiomasCongenital angiomas
ATRIOVENOUS MALFORMATIONS:ATRIOVENOUS MALFORMATIONS:
Congenital abnormal arteriesCongenital abnormal arteries
Congenital abnormal veinsCongenital abnormal veins
5. INCIDENCE:INCIDENCE:
26,000 cases every new year26,000 cases every new year
4% of cases have asymptomatic4% of cases have asymptomatic
cerebral aneurysm.cerebral aneurysm.
Occurs at ages of 35 and 65 yrs.Occurs at ages of 35 and 65 yrs.
RISK FACTORS:RISK FACTORS:
HypertensionHypertension
SmokingSmoking
TraumaTrauma
Autosomal dominant diseaseAutosomal dominant disease
6.
7. TYPES OF HEMORRHAGICTYPES OF HEMORRHAGIC
STROKE:STROKE:
Intracerebral hemorrhageIntracerebral hemorrhage
Intracranial (cerebral)Intracranial (cerebral)
aneurysmaneurysm
Atriovenous malformationsAtriovenous malformations
Subarachnoid hemorrhageSubarachnoid hemorrhage
8. INTRACRANIAL (CEREBRAL)INTRACRANIAL (CEREBRAL)
ANEURYSM:ANEURYSM:
It is an abnormal dilation of the wallIt is an abnormal dilation of the wall
of the cerebral artery.of the cerebral artery.
TYPES OF CAREBRAL ANEURYSM:TYPES OF CAREBRAL ANEURYSM:
Berry or saccular aneurysmBerry or saccular aneurysm
Giant or fusiform aneurysmGiant or fusiform aneurysm
Mycotic aneurysmMycotic aneurysm
Traumatic aneurysmTraumatic aneurysm
11. THROMBOTIC STROKETHROMBOTIC STROKE
Occlusion of largeOcclusion of large
cerebral vesselcerebral vessel
Older populationOlder population
Sleeping/restingSleeping/resting
Rapid event, butRapid event, but
slow progressionslow progression
(usually reach(usually reach
max deficit in 3max deficit in 3
days)days)
14. EMBOLIC STROKEEMBOLIC STROKE
EmbolusEmbolus
becomes lodgedbecomes lodged
in vessel andin vessel and
causes occlusioncauses occlusion
Bifurcations areBifurcations are
most commonmost common
sitesite
Sudden onsetSudden onset
with immediatewith immediate
deficitsdeficits
19. CLASSIFICATION BY SEVERITYCLASSIFICATION BY SEVERITY
Grade I â Minimal Bleed:Grade I â Minimal Bleed:
Minimal headacheMinimal headache
Nuchal rigidityNuchal rigidity
No neurologic deficitNo neurologic deficit
Grade II â Mild Bleed:Grade II â Mild Bleed:
MMild to severe headacheild to severe headache
Nuchal rigidityNuchal rigidity
Minimal neurologic deficitMinimal neurologic deficit
20. Grade III â Moderate Bleed:Grade III â Moderate Bleed:
Drowsy / confusedDrowsy / confused
Severe head acheSevere head ache
Fixed neurologic deficitFixed neurologic deficit
Grade IV â severe Bleed:Grade IV â severe Bleed:
StuporStupor
Hemi paresisHemi paresis
Deep comaDeep coma
21. PATHOPHYSIOLOGYPATHOPHYSIOLOGY
Etiological factorsEtiological factors
ââ
Increase pressure in blood vesselsIncrease pressure in blood vessels
ââ
Results in rupture and intracranial hemorrhageResults in rupture and intracranial hemorrhage
ââ
Cerebral edemaCerebral edema
ââ
Increased intracranial pressureIncreased intracranial pressure
ââ
Increased pressure over brain tissueIncreased pressure over brain tissue
ââ
Brain tissue injury and deathBrain tissue injury and death
ââ
Loss of tissue functionLoss of tissue function
30. Transcranial Doppler
(TCD)
A non invasive
ultrasound technology
used to evaluate blood
flow velocity in the
major basal intracranial
arteries.
31. Transcranial Color Coded Duplex
(TCCD)
TCCD sonography is a newer technique that permits the
real-time visualization of the intracranial vascular
anatomy and blood flow dynamics.Â
32. Magnetic resonance angiographyMagnetic resonance angiography
(MRA)(MRA) â âTime of flightâ or contrastâ âTime of flightâ or contrast
enhanced techniques are used. Itenhanced techniques are used. It
tends to overestimate the severity oftends to overestimate the severity of
stenosis. When assessing the carotidstenosis. When assessing the carotid
arteries it is best used in combinationarteries it is best used in combination
with Doppler.with Doppler.
Its non-invasive nature makes itIts non-invasive nature makes it
helpful in investigating the intracranialhelpful in investigating the intracranial
Stroke â investigations (contâd)Stroke â investigations (contâd)
35. MANAGEMENT:MANAGEMENT:
GOALS:GOALS:
Prevent further deterioration in functionPrevent further deterioration in function
Prevent complicationsPrevent complications
Prevent recovery of function.Prevent recovery of function.
PHARMACOLOGICAL MANAGEMENT:PHARMACOLOGICAL MANAGEMENT:
Anti epilepticsAnti epileptics
CorticosteroidsCorticosteroids
Anti hypertensiveAnti hypertensive
Analgesics and SedativesAnalgesics and Sedatives
Anti fibrinolytic therapyAnti fibrinolytic therapy (TpA Thrombolytics Recombinant(TpA Thrombolytics Recombinant
Alteplase (rtPA) Activase, Tissue plasminogen activator)Alteplase (rtPA) Activase, Tissue plasminogen activator)
36. ï¶Drug TherapyDrug Therapy
ï¶
Measures to prevent the development of aMeasures to prevent the development of a
thrombus or embolus for âAt Riskâ patients:thrombus or embolus for âAt Riskâ patients:
ï¶Antiplatelet AgentsAntiplatelet Agents
ï¶
AspirinAspirin
ï¶
PlavixPlavix
ï¶
CombinationCombination
ï¶Oral anticoagulation â CoumadinOral anticoagulation â Coumadin
ï¶
Treatment of choice for individuals with atrialTreatment of choice for individuals with atrial
fibrillation who have had a TIAfibrillation who have had a TIA
Goals for ManagementGoals for Management
37. Goals for ManagementGoals for Management
Immediate â assess & stabilizeImmediate â assess & stabilize
ïź
ABCs, VSABCs, VS
ïź
Neurologic screeningNeurologic screening
ïź
Oxygen if hypoxicOxygen if hypoxic
ïź
IV accessIV access
ïź
Check glucoseCheck glucose
ïź
Activate stroke team âActivate stroke team â CODE GREENCODE GREEN
ïź
12-lead EKG12-lead EKG
Immediate Neuro AssessmentImmediate Neuro Assessment
Establish symptom onsetEstablish symptom onset
ïź
Review hxReview hx
ïź
Stroke ScaleStroke Scale
ïź
Facial droop; arm drift; abnormal speechFacial droop; arm drift; abnormal speech
38.
39. CT Scan â No hemorrhage:CT Scan â No hemorrhage:
ïź
Consider Fibrinolytic therapyConsider Fibrinolytic therapy
Check for exclusionsCheck for exclusions
tPAtPA
ïź
No anticoagulants or antiplatelet therapy for 24 hoursNo anticoagulants or antiplatelet therapy for 24 hours
ïź
If not a candidate: Antiplatelet TherapyIf not a candidate: Antiplatelet Therapy
CT Scan â Hemorrhage:CT Scan â Hemorrhage:
ïź
Neurosurgery?Neurosurgery?
ïź
If no surgery: Stroke UnitIf no surgery: Stroke Unit
Monitor BP and treat HypertensionMonitor BP and treat Hypertension
Monitor Neuro statusMonitor Neuro status
Monitor blood glucose and treat as neededMonitor blood glucose and treat as needed
Supportive therapySupportive therapy
Goals for ManagementGoals for Management
47. CEREBROVASCULAR ACCIDENTCEREBROVASCULAR ACCIDENT
Nursing GoalsNursing Goals
ï¶Maintain stable or improved LOCMaintain stable or improved LOC
ï¶Attain maximum physical functioningAttain maximum physical functioning
ï¶Attain maximum self-care activities & skillsAttain maximum self-care activities & skills
ï¶Maintain stable body functionsMaintain stable body functions
ï¶Maximize communication abilitiesMaximize communication abilities
ï¶Maintain adequate nutritionMaintain adequate nutrition
ï¶Avoid complications of strokeAvoid complications of stroke
ï¶Maintain effective personal & family copingMaintain effective personal & family coping
48. Warning Signs of StrokeWarning Signs of Stroke
ï¶Sudden weakness, paralysis, or numbnessSudden weakness, paralysis, or numbness
of the face, arm, or leg, especially on oneof the face, arm, or leg, especially on one
side of the bodyside of the body
ï¶Sudden dimness or loss of vision in one orSudden dimness or loss of vision in one or
both eyesboth eyes
ï¶Sudden loss of speech, confusion, orSudden loss of speech, confusion, or
difficulty speaking or understanding speechdifficulty speaking or understanding speech
ï¶Unexplained sudden dizziness,Unexplained sudden dizziness,
unsteadiness, loss of balance, orunsteadiness, loss of balance, or
coordinationcoordination
ï¶Sudden severe headacheSudden severe headache
CEREBROVASCULAR ACCIDENTCEREBROVASCULAR ACCIDENT
50. CEREBROVASCULAR ACCIDENTCEREBROVASCULAR ACCIDENT
ACUTE PHASEACUTE PHASE
ï¶Nsg ActionNsg Action::
ï¶
Supportive CareSupportive Care
ï¶Respiratory â spans from intubation to breathing onRespiratory â spans from intubation to breathing on
ownown
ï¶Musculoskeletal -- Positioning â side-to-side; HOBMusculoskeletal -- Positioning â side-to-side; HOB
elevated; PROM exercise; splints; shoes/footboardelevated; PROM exercise; splints; shoes/footboard
ï¶GI â enteral feedings initiallyGI â enteral feedings initially
ï¶GU â foley catheterGU â foley catheter
ï¶Skin â preventive careSkin â preventive care
ï¶Meds: anti plateletMeds: anti platelet
51. CEREBROVASCULAR ACCIDENTCEREBROVASCULAR ACCIDENT
Acute PhaseAcute Phase
ï¶Patient Education:Patient Education:
ï¶
Clear explanations for all care/treatmentsClear explanations for all care/treatments
ï¶
Focus on improvementsâregainedFocus on improvementsâregained
abilitiesabilities
ï¶
Include familyInclude family
When resting, less pressure to push thrombus thru a narrowed vessel
Pathophysiology review:
Atherosclerotic plaque damages intimal lining of arteries
Platelets can then adhere to rough surface
Release adenosine diphosphate
Clotting sequence is initiated and a thrombus forms
Thrombus can continue to grow and occlude vessel or it can break off and become and embolus