This document discusses conductive hearing loss, which occurs when sound is not properly conducted from the outer ear to the inner ear. Common causes of conductive hearing loss include cerumen impaction, middle ear fluid, and fixation of the small bones in the ear. The document describes various causes of conductive hearing loss affecting the outer ear, such as infections, trauma, tumors, and congenital abnormalities, as well as middle ear causes like otitis media, cholesteatoma, and otosclerosis. Diagnostic tests for conductive hearing loss include the Weber test, Rinne test, tympanometry, and audiometry to measure air and bone conduction thresholds.
Any deviation in the normal nasal septum is called DNS (Deviated Nasal Septum).
Deviated Nasal Septum may be caused by mechanical trauma and may be
associated with some developmental defects.
Conductive hearing loss, sensorineural hearing loss causes, of hearing, heari...Pauline Howard
Conductive Hearing Loss, Sensorineural Hearing Loss Causes, Of Hearing, Hearing Loss Diseases
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Hearing Loss Fix Found In Mom's Betty Crocker Cookbook.
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Recently, a retired Aerospace Engineer found, an old faded recipe that was tucked away in a Betty Crocker Cookbook in his basement.
The recipe was given to his great grandmother, from a Navajo Medicine Man or Hatałii many years ago.
The recipe was a remedy that restores hearing loss.
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Conductive Hearing Loss, Sensorineural Hearing Loss Causes, Of Hearing, Hearing Loss Diseases,
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Any deviation in the normal nasal septum is called DNS (Deviated Nasal Septum).
Deviated Nasal Septum may be caused by mechanical trauma and may be
associated with some developmental defects.
Conductive hearing loss, sensorineural hearing loss causes, of hearing, heari...Pauline Howard
Conductive Hearing Loss, Sensorineural Hearing Loss Causes, Of Hearing, Hearing Loss Diseases
http://weird-2-minute-trick-restores-hearing.info-pro.co
Hearing Loss Fix Found In Mom's Betty Crocker Cookbook.
Discover The WEIRD But 100% Scientifically Proven Trick, To Achieving Perfect Hearing In As Little As 3 Weeks.
Naturally! No Surgery and No Hearing Devices!
No matter what sort of hearing condition you have, this recent Breakthrough will have you hearing everything Crystal Clear in no time!
How is this possible?
Recently, a retired Aerospace Engineer found, an old faded recipe that was tucked away in a Betty Crocker Cookbook in his basement.
The recipe was given to his great grandmother, from a Navajo Medicine Man or Hatałii many years ago.
The recipe was a remedy that restores hearing loss.
Over 33,477 people from all around the world, with a vast variety of hearing issues have drastically improved their hearing with the remedy.
So if you or someone, you love is suffering with hearing loss, follow the link below to find out more.
Get The Details About The Scientifically Supported Navajo Hearing Loss Remedy click here.
http://weird-2-minute-trick-restores-hearing.info-pro.co
Click The Link Below To Check It Out
http://weird-2-minute-trick-restores-hearing.info-pro.co
Subscribe To Our Channel
https://www.youtube.com/watch?v=ydBaQhBaO_A
Conductive Hearing Loss, Sensorineural Hearing Loss Causes, Of Hearing, Hearing Loss Diseases,
wikipedia hearing loss,
hearing loss symptoms in children,
newborn hearing screening,
causes for conductive hearing loss,
my hearing aids,
cookie bite hearing loss,
beethoven hearing loss,
hearing aid history,
a hearing,
about hearing aids,
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action for hearing loss
Chronic otitis media effusion, also known as chronic middle ear fluid, is a condition characterized by the long-term presence of fluid in the middle ear. The middle ear is the space behind the eardrum that contains the tiny bones responsible for transmitting sound vibrations to the inner ear
Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
Tom Selleck Health: A Comprehensive Look at the Iconic Actor’s Wellness Journeygreendigital
Tom Selleck, an enduring figure in Hollywood. has captivated audiences for decades with his rugged charm, iconic moustache. and memorable roles in television and film. From his breakout role as Thomas Magnum in Magnum P.I. to his current portrayal of Frank Reagan in Blue Bloods. Selleck's career has spanned over 50 years. But beyond his professional achievements. fans have often been curious about Tom Selleck Health. especially as he has aged in the public eye.
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Introduction
Many have been interested in Tom Selleck health. not only because of his enduring presence on screen but also because of the challenges. and lifestyle choices he has faced and made over the years. This article delves into the various aspects of Tom Selleck health. exploring his fitness regimen, diet, mental health. and the challenges he has encountered as he ages. We'll look at how he maintains his well-being. the health issues he has faced, and his approach to ageing .
Early Life and Career
Childhood and Athletic Beginnings
Tom Selleck was born on January 29, 1945, in Detroit, Michigan, and grew up in Sherman Oaks, California. From an early age, he was involved in sports, particularly basketball. which played a significant role in his physical development. His athletic pursuits continued into college. where he attended the University of Southern California (USC) on a basketball scholarship. This early involvement in sports laid a strong foundation for his physical health and disciplined lifestyle.
Transition to Acting
Selleck's transition from an athlete to an actor came with its physical demands. His first significant role in "Magnum P.I." required him to perform various stunts and maintain a fit appearance. This role, which he played from 1980 to 1988. necessitated a rigorous fitness routine to meet the show's demands. setting the stage for his long-term commitment to health and wellness.
Fitness Regimen
Workout Routine
Tom Selleck health and fitness regimen has evolved. adapting to his changing roles and age. During his "Magnum, P.I." days. Selleck's workouts were intense and focused on building and maintaining muscle mass. His routine included weightlifting, cardiovascular exercises. and specific training for the stunts he performed on the show.
Selleck adjusted his fitness routine as he aged to suit his body's needs. Today, his workouts focus on maintaining flexibility, strength, and cardiovascular health. He incorporates low-impact exercises such as swimming, walking, and light weightlifting. This balanced approach helps him stay fit without putting undue strain on his joints and muscles.
Importance of Flexibility and Mobility
In recent years, Selleck has emphasized the importance of flexibility and mobility in his fitness regimen. Understanding the natural decline in muscle mass and joint flexibility with age. he includes stretching and yoga in his routine. These practices help prevent injuries, improve posture, and maintain mobilit
New Directions in Targeted Therapeutic Approaches for Older Adults With Mantl...i3 Health
i3 Health is pleased to make the speaker slides from this activity available for use as a non-accredited self-study or teaching resource.
This slide deck presented by Dr. Kami Maddocks, Professor-Clinical in the Division of Hematology and
Associate Division Director for Ambulatory Operations
The Ohio State University Comprehensive Cancer Center, will provide insight into new directions in targeted therapeutic approaches for older adults with mantle cell lymphoma.
STATEMENT OF NEED
Mantle cell lymphoma (MCL) is a rare, aggressive B-cell non-Hodgkin lymphoma (NHL) accounting for 5% to 7% of all lymphomas. Its prognosis ranges from indolent disease that does not require treatment for years to very aggressive disease, which is associated with poor survival (Silkenstedt et al, 2021). Typically, MCL is diagnosed at advanced stage and in older patients who cannot tolerate intensive therapy (NCCN, 2022). Although recent advances have slightly increased remission rates, recurrence and relapse remain very common, leading to a median overall survival between 3 and 6 years (LLS, 2021). Though there are several effective options, progress is still needed towards establishing an accepted frontline approach for MCL (Castellino et al, 2022). Treatment selection and management of MCL are complicated by the heterogeneity of prognosis, advanced age and comorbidities of patients, and lack of an established standard approach for treatment, making it vital that clinicians be familiar with the latest research and advances in this area. In this activity chaired by Michael Wang, MD, Professor in the Department of Lymphoma & Myeloma at MD Anderson Cancer Center, expert faculty will discuss prognostic factors informing treatment, the promising results of recent trials in new therapeutic approaches, and the implications of treatment resistance in therapeutic selection for MCL.
Target Audience
Hematology/oncology fellows, attending faculty, and other health care professionals involved in the treatment of patients with mantle cell lymphoma (MCL).
Learning Objectives
1.) Identify clinical and biological prognostic factors that can guide treatment decision making for older adults with MCL
2.) Evaluate emerging data on targeted therapeutic approaches for treatment-naive and relapsed/refractory MCL and their applicability to older adults
3.) Assess mechanisms of resistance to targeted therapies for MCL and their implications for treatment selection
Knee anatomy and clinical tests 2024.pdfvimalpl1234
This includes all relevant anatomy and clinical tests compiled from standard textbooks, Campbell,netter etc..It is comprehensive and best suited for orthopaedicians and orthopaedic residents.
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New Drug Discovery and Development .....NEHA GUPTA
The "New Drug Discovery and Development" process involves the identification, design, testing, and manufacturing of novel pharmaceutical compounds with the aim of introducing new and improved treatments for various medical conditions. This comprehensive endeavor encompasses various stages, including target identification, preclinical studies, clinical trials, regulatory approval, and post-market surveillance. It involves multidisciplinary collaboration among scientists, researchers, clinicians, regulatory experts, and pharmaceutical companies to bring innovative therapies to market and address unmet medical needs.
NVBDCP.pptx Nation vector borne disease control programSapna Thakur
NVBDCP was launched in 2003-2004 . Vector-Borne Disease: Disease that results from an infection transmitted to humans and other animals by blood-feeding arthropods, such as mosquitoes, ticks, and fleas. Examples of vector-borne diseases include Dengue fever, West Nile Virus, Lyme disease, and malaria.
These simplified slides by Dr. Sidra Arshad present an overview of the non-respiratory functions of the respiratory tract.
Learning objectives:
1. Enlist the non-respiratory functions of the respiratory tract
2. Briefly explain how these functions are carried out
3. Discuss the significance of dead space
4. Differentiate between minute ventilation and alveolar ventilation
5. Describe the cough and sneeze reflexes
Study Resources:
1. Chapter 39, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 34, Ganong’s Review of Medical Physiology, 26th edition
3. Chapter 17, Human Physiology by Lauralee Sherwood, 9th edition
4. Non-respiratory functions of the lungs https://academic.oup.com/bjaed/article/13/3/98/278874
The prostate is an exocrine gland of the male mammalian reproductive system
It is a walnut-sized gland that forms part of the male reproductive system and is located in front of the rectum and just below the urinary bladder
Function is to store and secrete a clear, slightly alkaline fluid that constitutes 10-30% of the volume of the seminal fluid that along with the spermatozoa, constitutes semen
A healthy human prostate measures (4cm-vertical, by 3cm-horizontal, 2cm ant-post ).
It surrounds the urethra just below the urinary bladder. It has anterior, median, posterior and two lateral lobes
It’s work is regulated by androgens which are responsible for male sex characteristics
Generalised disease of the prostate due to hormonal derangement which leads to non malignant enlargement of the gland (increase in the number of epithelial cells and stromal tissue)to cause compression of the urethra leading to symptoms (LUTS
Lung Cancer: Artificial Intelligence, Synergetics, Complex System Analysis, S...Oleg Kshivets
RESULTS: Overall life span (LS) was 2252.1±1742.5 days and cumulative 5-year survival (5YS) reached 73.2%, 10 years – 64.8%, 20 years – 42.5%. 513 LCP lived more than 5 years (LS=3124.6±1525.6 days), 148 LCP – more than 10 years (LS=5054.4±1504.1 days).199 LCP died because of LC (LS=562.7±374.5 days). 5YS of LCP after bi/lobectomies was significantly superior in comparison with LCP after pneumonectomies (78.1% vs.63.7%, P=0.00001 by log-rank test). AT significantly improved 5YS (66.3% vs. 34.8%) (P=0.00000 by log-rank test) only for LCP with N1-2. Cox modeling displayed that 5YS of LCP significantly depended on: phase transition (PT) early-invasive LC in terms of synergetics, PT N0—N12, cell ratio factors (ratio between cancer cells- CC and blood cells subpopulations), G1-3, histology, glucose, AT, blood cell circuit, prothrombin index, heparin tolerance, recalcification time (P=0.000-0.038). Neural networks, genetic algorithm selection and bootstrap simulation revealed relationships between 5YS and PT early-invasive LC (rank=1), PT N0—N12 (rank=2), thrombocytes/CC (3), erythrocytes/CC (4), eosinophils/CC (5), healthy cells/CC (6), lymphocytes/CC (7), segmented neutrophils/CC (8), stick neutrophils/CC (9), monocytes/CC (10); leucocytes/CC (11). Correct prediction of 5YS was 100% by neural networks computing (area under ROC curve=1.0; error=0.0).
CONCLUSIONS: 5YS of LCP after radical procedures significantly depended on: 1) PT early-invasive cancer; 2) PT N0--N12; 3) cell ratio factors; 4) blood cell circuit; 5) biochemical factors; 6) hemostasis system; 7) AT; 8) LC characteristics; 9) LC cell dynamics; 10) surgery type: lobectomy/pneumonectomy; 11) anthropometric data. Optimal diagnosis and treatment strategies for LC are: 1) screening and early detection of LC; 2) availability of experienced thoracic surgeons because of complexity of radical procedures; 3) aggressive en block surgery and adequate lymph node dissection for completeness; 4) precise prediction; 5) adjuvant chemoimmunoradiotherapy for LCP with unfavorable prognosis.
3. Hearing loss is a common problem that everyone experiences from time to
time. Most commonly it occurs when flying or traveling up a mountain.
Diminished hearing also may occur during an ear infection. These causes of
hearing loss are usually short-lived. The other extreme is the permanent
sensorineural hearing loss that occurs with aging, which we will all experience
to some degree.
4. EPIDEMIOLOGY :
Hearing loss is a common chronic impairment, particularly for older adults. In the
Beaver Dam cohort in the United States, the prevalence of hearing loss, defined by
audiometry, increased steadily with age :
● 3 percent ages 21 to 34
● 6 percent ages 35 to 44
● 11 percent ages 45 to 54
● 25 percent ages 55 to 64
● 43 percent ages 65 to 84
Worldwide, estimates from the World Health Organization are that hearing loss
affects 538 million people
6. Conductive Hearing Loss :
involving any cause that in some way limits the amount of external sound
from gaining access to the inner ear. Examples include cerumen impaction,
middle ear fluid, or ossicular chain fixation (lack of movement of the small
bones of the ear).
7. Causes of Conductive hearing loss :
Outer-ear causes
Congenital microtia or atresia
External otitis
Trauma
Squamous cell carcinoma
Exostosis
Osteoma
Psoriasis
Cerumen
Middle-ear causes
Congenital atresia or ossicular chain malformation
Otitis media
Cholesteatoma
Otosclerosis
Tympanic membrane perforation
Temporal bone trauma
Glomus tumors
8. OUTER EAR CAUSES :
Congenital :
The external auditory canal (EAC) develops from the 8th to the 28th
week of gestation; problems can occur anytime during this
development
al phase. Such as: microtia ,atresia,…
Infection :
Infections may lead to blockage of the EAC due to the accumulation of
debris, edema, or inflammation.
9. OUTER EAR CAUSES :
Trauma :
Penetrating trauma to the external auditory canal or meatus due to a
bullet, knife, or fracture may cause mild or profound conductive
hearing loss, depending upon the degree of EAC occlusion.
Tumor :
The most common malignant tumor of the EAC is squamous cell
carcinoma. This and other tumors of the EAC, such as basal cell
carcinoma and melanoma, typically cause conductive hearing loss due
to occlusion of the canal.
10. OUTER EAR CAUSES :
Exostosis :
Exostosis or multiple benign bony growths of the EAC most
commonly occur in individuals who have had repeated
exposure to cold water.
Osteoma :
Osteoma is a solitary bony growth that is most commonly attached to the
tympanosquamous suture line.
Similar to exostoses, osteomas are not treated until they become so large that they
affect hearing by occlusion or repeated infections because debris cannot exit the
EAC.
11. OUTER EAR CAUSES :
Benign polyps :
Benign polyps may occur as a result of other otologic
conditions, such as chronic ear infections or
cholesteatoma.
Systemic disease :
Diabetes mellitus and other immunocompromised states can
predispose to developing necrotizing otitis externa, which in turn can
cause conductive hearing loss.
12. OUTER EAR CAUSES :
Dermatologic :
Certain skin diseases, such as psoriasis, may cause scaling and edema of the EAC
and meatus. Treatment is usually with steroid ointments or creams.
Cerumen :
Some patients are not able to clear cerumen on their own or use Q-tips that push
the cerumen down the ear canal. These individuals may need periodic cleaning of
the cerumen to enhance their auditory capabilities.
13. Middle-ear causes :
Congenital :
Atresia or malformation of the ossicular chain can cause conductive
hearing loss.
Eustachian tube dysfunction :
Eustachian tube dysfunction occurs commonly in the setting of a viral
upper respiratory infection (URI) or sinusitis, and it can also occur with
allergies.
Infection :
Otitis media (OM) is a common childhood disorder that also frequently
occurs in adults
14. Middle-ear causes :
Tumors :
Malignant tumors such as Langerhans cell histiocytosis (including the Letterer-Siwe
variant) or squamous cell carcinoma may cause conductive hearing loss.
Cholesteatoma :
Cholesteatoma is a growth of desquamated,
stratified,squamous epithelium within
the middle ear space.
15. Middle-ear causes :
Otosclerosis :
Otosclerosis is a bony overgrowth that involves the footplate of the stapes.
Tympanic membrane perforation :
Conductive hearing loss due to TM perforation is common. The degree of
conductive hearing loss depends upon the size and location of the
perforation. Small perforations and those located in the anterior/inferior
quadrant cause the least amount of conductive hearing loss.
Middle ear barotrauma :
Barotrauma occurs when a patient is exposed to a sudden, large change in
ambient pressure, often during diving or flying.
18. Interpreting Weber and Rinne tests: Conductive
versus sensorineural hearing loss
Weber lateralizes Rinne test
Conductive loss
Good ear AC > BC
Bad ear To bad ear BC > AC
Sensorineural loss
Good ear To good ear AC > BC
Bad ear AC > BC
19. Pneumoscopy :
Pneumoscopy is also performed to evaluate mobility of the TM.
● A nonmobile TM may occur because of fluid in the middle ear cavity, a mass in
the middle ear cavity, or a stiff or sclerotic TM.
● A hypermobile TM may indicate ossicular chain disruption.
● The TM may only move with negative pressure;
this can be caused by retraction of the TM or middle ear
with a blocked eustachian tube, resulting in negative
middle ear pressure.
20. Pure tone, air, and bone conduction testing :
Pure tone testing is commonly known as the audiogram. Hearing is tested
with both air and bone conduction.
Any difference between air and bone
conduction thresholds is known as an air/bone gap;
a gap is consistent with conductive hearing loss
21. Tympanometry :
It is an objective measure of the changes in the acoustic impedance of
the middle ear system in response to changes in air pressure.
22. :Five types of tympanograms can be seen
1. Type A: Normal tympanic membrane (TM) mobility.
2. Type B: Flat tympanogram associated with fluid or perforation (will
have large volume).
3. Type C: Negative middle ear pressure such as from a retracted TM.
4. Type AS: Very stiff noncompliant TM associated with TM sclerosis
or otosclerosis.
5. Type AD: Hypermobile usually associated with ossicular
discontinuity.