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Legg-Calve-Perthes

Disease
WaferAldulaimi / Denmark
The diseaswasdiscovered in 1910
shortlyafterdevelopment of x-ray
Epidemiology
 1/1200 /year

 usuallyhappens in youngboys 5-7 yearsold

(3-12) thataresmaller in stature and
thattend to beveryactive
Alsoseen in small activedogs
Pathoanatomy
Transient avascularnecrosis of femoral
head
Symptoms and signs
 Limping
 Pain in hip, groin or sometimes knee area
 Stiffness
 Decrease range of motion in hip
 Leg length differentiation
 Pain can vary depending on child, amount of physical

activity and how long child goes without seeking
professional help

 some muscle spasms
Diagnosis
 History , physical examination and plane x-

rays

 MR scanning not routinely
Stages
1. Necrosis : Femoral head appears smaller
on x-rays
2. Fragmentation and femoral head
collapse. (The pain and limp are worse
during this stage)
3. Reossifiction: Healing phase starts when
new bone formation begins. (The pain
decreases)

4. Remodeling :The head is completely
healed and the dead bone is now
replaced with new bone
Treatment
Main goal is the keep the femoral head
inside the socket of the hip
1. Symptomatic treatment and
physiotherapy
2. Follow up: Pain and limp. Range of
movement and x-rays
3. Containment by braces or casting ( No
longer advocated ! )

4. Containment by surgery
 Femoral varusosteotomy
 Pelvicosteotomy
 Combinedosteotomy
In case of Hinge abduction (lateral extrusion of the
femoral head resulting in the femoral head
impinging on the edge of the acetabulum with
abduction)
An abduction-extension proximal femoral
osteotomy should be considered
 Most patients with LCPD do well until the fifth

or sixth decade of life, at which time
degenerative changes in the hip joint are
common which may need THA
CLPD

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