This document provides an overview of cervical trauma and cervical spine injuries. It discusses anatomy, mechanisms of injury, history and examination findings, imaging, classifications of injuries including fractures and spinal cord syndromes, and indications for surgical intervention. The key points are that cervical spine injury must be considered in polytrauma patients, manual stabilization is needed in addition to collars, and imaging such as CT and MRI can help classify fractures and rule out injuries when clinical suspicion remains.
Colorado spine surgeon, Dr. Donald Corenman, M.D., D.C. (http://neckandback.com), is an expert in treating spinal cord injuries associated with a traumatic fall, sports related injury or accident. Many spine fractures include a thoracolumbar fracture, which is a break in one or more of the thoracic and lumbar vertebrae. Spine fractures can be very serious but are also treatable in many cases. This presentation on spinal cord injuries, spine fractures and thoracolumbar fractures details events that can lead to this injury, symptoms and treatment options.
Dr. Corenman is a renowned Colorado spine surgeon and also is an expert at all spine conditions and disorders including scoliosis, degenerative disc disease, spinal stenosis, sciatica, herniated disc, slipped disc and spondylolythesis. He is also a sports medicine specialist and treats athletes with traumatic sports related injuries. He recently launched his own website (http://neckandback.com) to educate patients on spine disorders and to offer second opinions to physicians and colleagues who are seeking additional information on specific spine injuries and treatment options.
Mpfl tech - MPFL Reconstruction for Patellar InstabilityDelhiArthroscopy
MPFL Rec onstruction for Patellar Instability - By Dr Shekhar Srivastav .
Surgical Technique
- Diagnostic Arthroscopy
- Look for any Osteochondral fragment
(Loose body)
- Look for any Chondral damage
- Patellar tracking though Supero-lateral portal
Post-op Protocol
Ambulation with stick and Knee Brace- 3 wks
ROM exer – Next day upto 300 and progress
Review every 2 wks,6 wks,3 mnths,6 mnths and
yearly thereafter
Post-op assessment (Crosby-Insall criteria)
Excellent- No pain,normal activity
Good- Occasional pain,discomfort
Fair/Poor- Pain,loss of flexion,recurrent
dislocation/subluxation
Worse- Pain increased,displacement more
frequent
Caution
Must avoid overtightening-
Medial instability
Medial patellar arthritis
Patellar fractures
Preexisting Chondromalacia
Details @ http://www.delhiarthroscopy.com/
Posterolateral corner injuries of knee joint Samir Dwidmuthe
Missed posterolateral corner injuries of knee joint is a common cause for failure of ACL and PCL reconstruction only next to malpositioned tunnels.
Isolated PLC injuries are uncommon, making up <2% of all acute knee ligamentous injuries. Covey JBJS 2001
Incidence of PLC injuries associated with concomitant ACL and PCL disruptions are much more common (43% to 80%). Ranawat JAAOS 2008
A recent (MRI) analysis of surgical tibialplateau fractures demonstrated an incidence of PLC injuries in 68% of cases. Gardner JOT 2005
Take home message
PLC injuries to be ruled out in every case of ACL& PCL rupture.
Neurovascular integrity to be checked in every case.
Grade I & II can be managed conservatively.
Grade III Acute- Repair.
Grade III Chronic- Anatomic PLC recon.
Beware of varus knee alignment.
Colorado spine surgeon, Dr. Donald Corenman, M.D., D.C. (http://neckandback.com), is an expert in treating spinal cord injuries associated with a traumatic fall, sports related injury or accident. Many spine fractures include a thoracolumbar fracture, which is a break in one or more of the thoracic and lumbar vertebrae. Spine fractures can be very serious but are also treatable in many cases. This presentation on spinal cord injuries, spine fractures and thoracolumbar fractures details events that can lead to this injury, symptoms and treatment options.
Dr. Corenman is a renowned Colorado spine surgeon and also is an expert at all spine conditions and disorders including scoliosis, degenerative disc disease, spinal stenosis, sciatica, herniated disc, slipped disc and spondylolythesis. He is also a sports medicine specialist and treats athletes with traumatic sports related injuries. He recently launched his own website (http://neckandback.com) to educate patients on spine disorders and to offer second opinions to physicians and colleagues who are seeking additional information on specific spine injuries and treatment options.
Mpfl tech - MPFL Reconstruction for Patellar InstabilityDelhiArthroscopy
MPFL Rec onstruction for Patellar Instability - By Dr Shekhar Srivastav .
Surgical Technique
- Diagnostic Arthroscopy
- Look for any Osteochondral fragment
(Loose body)
- Look for any Chondral damage
- Patellar tracking though Supero-lateral portal
Post-op Protocol
Ambulation with stick and Knee Brace- 3 wks
ROM exer – Next day upto 300 and progress
Review every 2 wks,6 wks,3 mnths,6 mnths and
yearly thereafter
Post-op assessment (Crosby-Insall criteria)
Excellent- No pain,normal activity
Good- Occasional pain,discomfort
Fair/Poor- Pain,loss of flexion,recurrent
dislocation/subluxation
Worse- Pain increased,displacement more
frequent
Caution
Must avoid overtightening-
Medial instability
Medial patellar arthritis
Patellar fractures
Preexisting Chondromalacia
Details @ http://www.delhiarthroscopy.com/
Posterolateral corner injuries of knee joint Samir Dwidmuthe
Missed posterolateral corner injuries of knee joint is a common cause for failure of ACL and PCL reconstruction only next to malpositioned tunnels.
Isolated PLC injuries are uncommon, making up <2% of all acute knee ligamentous injuries. Covey JBJS 2001
Incidence of PLC injuries associated with concomitant ACL and PCL disruptions are much more common (43% to 80%). Ranawat JAAOS 2008
A recent (MRI) analysis of surgical tibialplateau fractures demonstrated an incidence of PLC injuries in 68% of cases. Gardner JOT 2005
Take home message
PLC injuries to be ruled out in every case of ACL& PCL rupture.
Neurovascular integrity to be checked in every case.
Grade I & II can be managed conservatively.
Grade III Acute- Repair.
Grade III Chronic- Anatomic PLC recon.
Beware of varus knee alignment.
Cervical spine clearance lecture given to 1st-year emergency medicine residents at Duke University. Covers indications for applying cervical collar, types of collars, types of imaging of the spine, and when to remove the collar.
Cervical spine trauma and spinal cord injuries by Dr Shamavu Gabriel.pptxGabriel Shamavu
PAEDIATRICS TRAUMA ADVANCED LIFE SUPPORT PRESENTATION
Cervical spine trauma and spinal cord injuries
Prepared by Dr GABRIEL KAKURU SHAMAVU, Resident in Paediatrics and child health at Kampala International University Teaching Hospital. With Mentorship of Professor Yamile Arias Ortiz. Tutor of the course of "Paediatrics Emergencies and life support". Mars 2022
These lecture slides, by Dr Sidra Arshad, offer a quick overview of physiological basis of a normal electrocardiogram.
Learning objectives:
1. Define an electrocardiogram (ECG) and electrocardiography
2. Describe how dipoles generated by the heart produce the waveforms of the ECG
3. Describe the components of a normal electrocardiogram of a typical bipolar leads (limb II)
4. Differentiate between intervals and segments
5. Enlist some common indications for obtaining an ECG
Study Resources:
1. Chapter 11, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 9, Human Physiology - From Cells to Systems, Lauralee Sherwood, 9th edition
3. Chapter 29, Ganong’s Review of Medical Physiology, 26th edition
4. Electrocardiogram, StatPearls - https://www.ncbi.nlm.nih.gov/books/NBK549803/
5. ECG in Medical Practice by ABM Abdullah, 4th edition
6. ECG Basics, http://www.nataliescasebook.com/tag/e-c-g-basics
Pulmonary Thromboembolism - etilogy, types, medical- Surgical and nursing man...VarunMahajani
Disruption of blood supply to lung alveoli due to blockage of one or more pulmonary blood vessels is called as Pulmonary thromboembolism. In this presentation we will discuss its causes, types and its management in depth.
These simplified slides by Dr. Sidra Arshad present an overview of the non-respiratory functions of the respiratory tract.
Learning objectives:
1. Enlist the non-respiratory functions of the respiratory tract
2. Briefly explain how these functions are carried out
3. Discuss the significance of dead space
4. Differentiate between minute ventilation and alveolar ventilation
5. Describe the cough and sneeze reflexes
Study Resources:
1. Chapter 39, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 34, Ganong’s Review of Medical Physiology, 26th edition
3. Chapter 17, Human Physiology by Lauralee Sherwood, 9th edition
4. Non-respiratory functions of the lungs https://academic.oup.com/bjaed/article/13/3/98/278874
- Video recording of this lecture in English language: https://youtu.be/lK81BzxMqdo
- Video recording of this lecture in Arabic language: https://youtu.be/Ve4P0COk9OI
- Link to download the book free: https://nephrotube.blogspot.com/p/nephrotube-nephrology-books.html
- Link to NephroTube website: www.NephroTube.com
- Link to NephroTube social media accounts: https://nephrotube.blogspot.com/p/join-nephrotube-on-social-media.html
Ethanol (CH3CH2OH), or beverage alcohol, is a two-carbon alcohol
that is rapidly distributed in the body and brain. Ethanol alters many
neurochemical systems and has rewarding and addictive properties. It
is the oldest recreational drug and likely contributes to more morbidity,
mortality, and public health costs than all illicit drugs combined. The
5th edition of the Diagnostic and Statistical Manual of Mental Disorders
(DSM-5) integrates alcohol abuse and alcohol dependence into a single
disorder called alcohol use disorder (AUD), with mild, moderate,
and severe subclassifications (American Psychiatric Association, 2013).
In the DSM-5, all types of substance abuse and dependence have been
combined into a single substance use disorder (SUD) on a continuum
from mild to severe. A diagnosis of AUD requires that at least two of
the 11 DSM-5 behaviors be present within a 12-month period (mild
AUD: 2–3 criteria; moderate AUD: 4–5 criteria; severe AUD: 6–11 criteria).
The four main behavioral effects of AUD are impaired control over
drinking, negative social consequences, risky use, and altered physiological
effects (tolerance, withdrawal). This chapter presents an overview
of the prevalence and harmful consequences of AUD in the U.S.,
the systemic nature of the disease, neurocircuitry and stages of AUD,
comorbidities, fetal alcohol spectrum disorders, genetic risk factors, and
pharmacotherapies for AUD.
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Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
New Directions in Targeted Therapeutic Approaches for Older Adults With Mantl...i3 Health
i3 Health is pleased to make the speaker slides from this activity available for use as a non-accredited self-study or teaching resource.
This slide deck presented by Dr. Kami Maddocks, Professor-Clinical in the Division of Hematology and
Associate Division Director for Ambulatory Operations
The Ohio State University Comprehensive Cancer Center, will provide insight into new directions in targeted therapeutic approaches for older adults with mantle cell lymphoma.
STATEMENT OF NEED
Mantle cell lymphoma (MCL) is a rare, aggressive B-cell non-Hodgkin lymphoma (NHL) accounting for 5% to 7% of all lymphomas. Its prognosis ranges from indolent disease that does not require treatment for years to very aggressive disease, which is associated with poor survival (Silkenstedt et al, 2021). Typically, MCL is diagnosed at advanced stage and in older patients who cannot tolerate intensive therapy (NCCN, 2022). Although recent advances have slightly increased remission rates, recurrence and relapse remain very common, leading to a median overall survival between 3 and 6 years (LLS, 2021). Though there are several effective options, progress is still needed towards establishing an accepted frontline approach for MCL (Castellino et al, 2022). Treatment selection and management of MCL are complicated by the heterogeneity of prognosis, advanced age and comorbidities of patients, and lack of an established standard approach for treatment, making it vital that clinicians be familiar with the latest research and advances in this area. In this activity chaired by Michael Wang, MD, Professor in the Department of Lymphoma & Myeloma at MD Anderson Cancer Center, expert faculty will discuss prognostic factors informing treatment, the promising results of recent trials in new therapeutic approaches, and the implications of treatment resistance in therapeutic selection for MCL.
Target Audience
Hematology/oncology fellows, attending faculty, and other health care professionals involved in the treatment of patients with mantle cell lymphoma (MCL).
Learning Objectives
1.) Identify clinical and biological prognostic factors that can guide treatment decision making for older adults with MCL
2.) Evaluate emerging data on targeted therapeutic approaches for treatment-naive and relapsed/refractory MCL and their applicability to older adults
3.) Assess mechanisms of resistance to targeted therapies for MCL and their implications for treatment selection
Acute scrotum is a general term referring to an emergency condition affecting the contents or the wall of the scrotum.
There are a number of conditions that present acutely, predominantly with pain and/or swelling
A careful and detailed history and examination, and in some cases, investigations allow differentiation between these diagnoses. A prompt diagnosis is essential as the patient may require urgent surgical intervention
Testicular torsion refers to twisting of the spermatic cord, causing ischaemia of the testicle.
Testicular torsion results from inadequate fixation of the testis to the tunica vaginalis producing ischemia from reduced arterial inflow and venous outflow obstruction.
The prevalence of testicular torsion in adult patients hospitalized with acute scrotal pain is approximately 25 to 50 percent
Prix Galien International 2024 Forum ProgramLevi Shapiro
June 20, 2024, Prix Galien International and Jerusalem Ethics Forum in ROME. Detailed agenda including panels:
- ADVANCES IN CARDIOLOGY: A NEW PARADIGM IS COMING
- WOMEN’S HEALTH: FERTILITY PRESERVATION
- WHAT’S NEW IN THE TREATMENT OF INFECTIOUS,
ONCOLOGICAL AND INFLAMMATORY SKIN DISEASES?
- ARTIFICIAL INTELLIGENCE AND ETHICS
- GENE THERAPY
- BEYOND BORDERS: GLOBAL INITIATIVES FOR DEMOCRATIZING LIFE SCIENCE TECHNOLOGIES AND PROMOTING ACCESS TO HEALTHCARE
- ETHICAL CHALLENGES IN LIFE SCIENCES
- Prix Galien International Awards Ceremony
2. Spine injury must always be considered in any patients with multiple
injuries.
Any patient with trauma who is not fully conscious should be assumed
to have cervical spine injury until proved otherwise.
Collars alone are inadequate and they need to be supplemented by
manual stabilization or lateral support with blocks and forehead tape.
Collars and neck support should not be removed unless cervical spine
injury has been excluded for sure.
3. Anatomy
• 7 Cervical Vertebrae
• C1 (Atlas) is a ring which articulates with the occiput
– C1 has no body
– C1 has no spinous process
• C2 (Axis) so named because it is the pivot on which the Atlas turns to
rotate the head
– The Axis has a vertical extension, the Dens,
which articulates with C1
4.
5. Mechanisms of Injury:
- Hyperflexion - MVA, car comes to sudden stop
- Hyperextension - MVA, car struck from behind
- Compression - Head first dive in shallow water
6. causes
Motor vehicle accident and falls account for 50% and 20% of
cervical spine injuries, respectively.
Sports-related activities account for 15%:
Diving
Football
Skiing
7.
8. History
Common presentations include the following:
- Posterior neck pain on palpation of spinous processes
- Limited range of motion associated with pain
- Weakness, numbness, or paresthesias along affected nerve roots
10. Physical
- Common findings on physical examination in cervical spine injury include the
following:
- Spinal shock
Flaccidity Areflexia
Loss of anal sphincter tone Priapism
- Neurogenic shock
Hypotension Paradoxical bradycardia
Flushed, dry, and warm peripheral skin
- Associated with Paralysis of muscles of respiration
• Diaphragm invervated by C3-5
11. Management
Diagnosis and management go hand in hand
Inappropriate movement and examination can change the outcome
for the worse
Early management
Airway, Breathing and Circulation
Exclude other life-threatening injury
Properly immobilize patient
Avoid hypothermia
12. assess the patient’s neurologic status
Neurological level
- Most caudal level of motor / sensory function
- Motor and sensory may not be the same
- Sensory can vary on each side
Bony level
Site of vertebral column damage
13.
14.
15. Complete injury
No motor or sensory function below injury level
Incomplete injury
Any motor or sensory preservation below injury level
16.
17. Incomplete Spinal Cord Syndromes
Anterior Spinal Cord Syndrome
Corticospinal and spinothalamic tracts
injured
Preservation of posterior column pathway
Etiology
Anterior spinal cord trauma
Flexion of cervical spine causing cord
contusion
Thrombosis of anterior spinal artery
18.
19. Posterior Spinal Cord Syndrome
Rare condition
Injury to dorsal column
Preservation of corticospinal and spinothalamic pathways
Etiology
Penetrating trauma to posterior aspect of cord
Hyperextension injury with vertebral arch fracture
20.
21. Central Cord Syndrome
Injury affects central portion of cord
Loss of function of central fibers of corticospinal and
spinothalamic tracts
Decreased strength and pain/temperature of upper extremities
compared with lower extremities
Etiology
Hyperextension injuries
Central spinal stenosis
Disruption of normal blood flow
old age
22.
23. Brown Sequard Syndrome
Transverse hemisection of spinal cord
Ipsilateral loss of motor function, proprioceptive/vibratory
sensation
Contralateral loss of pain/temperature sensation
Etiology = Penetrating injury or Lateral cord compression
28. C-spine Film Interpretation
1. Count Vertebrae
-C1 through C7
-If T1 not seen do Swimmer’s view
2. Assess Curvature
3. Assess Vertebral Alignment (4 lines)
-ant vertebral line
-post vertebral line
-spinolaminal line
-post spinal line
31. Lateral view
Top of T1 visible
Vertebral bodies of uniform height
Odontoid intact and closely applied to C1
AP view
Spinous processes straight and spaced equally
Intervertebral spaces roughly equal
Odontoid view
Odontoid intact
Equal spaces on either side of odontoid
Lateral margins of C1 and C2 align
32.
33. Key Things to Identify
Predental space – should
be 3mm or less
35. Prevertebral soft tissue
swelling
May be due to
hematoma from a
fracture
Soft tissue swelling
may make fracture
diagnosis difficulty
36. AP View
The height of the cervical
vertebral bodies should be
approximately equal
The height of each joint space
should be roughly equal at all
levels
Spinous process should be in
midline and in good alignment
37. Odontoid View
An adequate film should include
the entire odontoid and the
lateral borders of C1-C2.
The distance from the dens to
the lateral masses of C1 should
be equal bilaterally.
The tips of lateral mass of C1
should line up with the lateral
margins of the superior articular
facet of C2.
38. JEFFERSON FRACTURE
Compression fracture of the
bony ring of C1, characterized
by lateral masses splitting and
transverse ligament tear
Mechanism: Diving into shallow
water, RTA
Best seen on Odontoid view
Signs: Displacement of the
lateral masses of vertebrae C1
beyond the margins of the body
of vertebra C2
39. Radiographic features:
the key radiographic view is the AP open mouth, which shows
displacement of the lateral masses of vertebrae C1 beyond the
margins of the body of vertebra C2. A lateral displacement of >2
mm or unilateral displacement may be indicative of a C1
fracture. CT is required to define the extent of fracture and to
detect fragments in the spinal canal.
Stability: unstable
40. the lateral displacement of C1
indicates a Jefferson fracture.
42. HANGMAN’S FRACTURE
Fracture through the pedicle of C2 secondary to hyperextension
Mechanism: Hanging or hitting a dashboard
Best seen on lateral view
Signs:
Prevertebral soft tissue swelling
Avulsion of anterior inferior corner of C2 associated with
rupture of the anterior longitudinal ligament
Anterior dislocation of the C2 vertebral body
Stability: unstable
43.
44.
45. ODONTOID FRACTURE
Fracture of the odontoid (dens) process of C2
Best seen on the lateral view
Types :
Type I – Fracture through superior portion of dens (Stable)
Type II – Fracture through the base of the dens (most common,
most dangerous, prone to non-union; Unstable; requires ORIF
Type III – Fracture that extends into the body of C2 (Stable)
best prognosis
46.
47. BURST FRACTURE
Fracture of C3-C7 that results from
axial compression
CT is required for all patients to
evaluate extent of injury
Injury to spinal cord, secondary to
displacement of posterior
fragments, is common
stable
48.
49. WEDGE FRACTURE
Compression fracture resulting from flexion
Mechanism: Hyperflexion and compression
Signs:
Buckled anterior cortex
Loss of height of anterior vertebral body
Anterosuperior fracture of vertebral body
stable
50.
51. FLEXION TEARDROP FRACTURE
Posterior ligament disruption and anterior compression fracture
of the vertebral body which results from a severe flexion injury
Mechanism: hyperflexion and compression (e.g. diving into
shallow water)
Best seen on lateral view
Signs:
Prevertebral swelling associated with anterior longitudinal
ligament tear
Teardrop fragment from anterior vertebral body avulsion
fracture
Posterior vertebral body subluxation into the spinal canal
Fracture of the spinous process
unstable
52.
53.
54. BILATERAL FACET DISLOCATION
Complete anterior dislocation of the vertebral body resulting
from extreme hyperflexion injury. It is associated with a very
high risk of cord damage
Best seen on lateral view
Signs:
Complete anterior dislocation of affected vertebral body by
half or more of the vertebral body AP diameter
Disruption of the posterior ligament complex and the
anterior longitudinal ligament
unstable
55.
56. UNILATERAL FACET DISLOCATION
Facet joint dislocation and rupture of the joint ligaments
resulting from rotatory injury of the cervical vertebrae
Best seen on lateral or oblique views
Signs:
- Anterior dislocation of affected vertebral body by less than half
of the vertebral body AP diameter
- Discordant rotation above and below involved level
- Widening of the disk space
57.
58. ANTERIOR SUBLUXATION
Disruption of the posterior
ligamentous complex resulting from
hyperflexion
Difficult to diagnose because muscle
spasm may result in similar findings
on the radiograph. May be stable
initially, but it associates with 20%-
50% delayed instability
Flexion and extension views are
helpful in further evaluation.
Signs:
Loss of normal cervical lordosis
Anterior displacement of the
vertebral body
Fanning of the interspinous
distance
59. SCIWORA
Significant Cord Injury without obvious radiological abnormality
- Higher incidence in pediatric population (34.8%)
The relatively large size of the head
inherent skeletal mobility
cord vulnerable to damage
- Higher incidence above 60 yo
Posterior vertebral spurs due to spondylosis
Ligamentum flavum bulging due to loss of disc height
Risk of central cord syndrome after hyperextension injury
60. indications for surgical intervention
1. Neurologic deficit,
2. Spinal instability, and
3. Intractable pain.
61. Forget about all of this and
If you are not happy
do not discharge the patient.