INITIAL ASSESSMENT OF TRAUMA PATIENTS....(INSPIRED FROM CTLS AND ATLS GUIDELI...Prerna Biswal
THIS PRESENTATION WAS MADE AT IMA HOUSE IN BHUBANESWAR,ODISHA, BY DR.NIBEDITA PANI,HOD ,DEPT. OF ANAESTHESIOLOGY AND DR.PRERNA BISWAL,PG,ANAESTHESIOLOGY,SCBMCH,CUTTACK,
Trauma is a global problem and continues to be a leading cause of disability and death.
Approximately 25% to 30% of deaths caused by trauma can be prevented when a systematic and organized approach is used.
The main goal of the initial assessment
Recognize the patient who does have life-threatening injuries
Establish treatment priorities, and
Manage them aggressively
INITIAL ASSESSMENT OF TRAUMA PATIENTS....(INSPIRED FROM CTLS AND ATLS GUIDELI...Prerna Biswal
THIS PRESENTATION WAS MADE AT IMA HOUSE IN BHUBANESWAR,ODISHA, BY DR.NIBEDITA PANI,HOD ,DEPT. OF ANAESTHESIOLOGY AND DR.PRERNA BISWAL,PG,ANAESTHESIOLOGY,SCBMCH,CUTTACK,
Trauma is a global problem and continues to be a leading cause of disability and death.
Approximately 25% to 30% of deaths caused by trauma can be prevented when a systematic and organized approach is used.
The main goal of the initial assessment
Recognize the patient who does have life-threatening injuries
Establish treatment priorities, and
Manage them aggressively
This PPT is mainly oriented to the Final yr MBBS students who are preparing for their Final exams. The Audit cycle has taken up from Bailey & Love - 24th edition.
This PPT is mainly oriented to the Final yr MBBS students who are preparing for their Final exams. The Audit cycle has taken up from Bailey & Love - 24th edition.
This is a presentation which contains basics of polytrauma management,ATLS, triage, critical decision making skills, application of Glasgow coma scale and complications of different management strategies, if not applied properly.
thoracic aortic injuries are very rare, this presentation will give a brief idea regarding the presentation of Thoracic aortic injury and its management
The Americal Association for the Surgery of Trauma - guidelines for intestinal injury- grading and a brief description of duodenal injury and few Most common Questions
Short eye Examination components - that will tell you the main headings of an eye examination in trauma victims.
drawaneeshkatiyar@gmail.com - for further communication.
These simplified slides by Dr. Sidra Arshad present an overview of the non-respiratory functions of the respiratory tract.
Learning objectives:
1. Enlist the non-respiratory functions of the respiratory tract
2. Briefly explain how these functions are carried out
3. Discuss the significance of dead space
4. Differentiate between minute ventilation and alveolar ventilation
5. Describe the cough and sneeze reflexes
Study Resources:
1. Chapter 39, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 34, Ganong’s Review of Medical Physiology, 26th edition
3. Chapter 17, Human Physiology by Lauralee Sherwood, 9th edition
4. Non-respiratory functions of the lungs https://academic.oup.com/bjaed/article/13/3/98/278874
New Directions in Targeted Therapeutic Approaches for Older Adults With Mantl...i3 Health
i3 Health is pleased to make the speaker slides from this activity available for use as a non-accredited self-study or teaching resource.
This slide deck presented by Dr. Kami Maddocks, Professor-Clinical in the Division of Hematology and
Associate Division Director for Ambulatory Operations
The Ohio State University Comprehensive Cancer Center, will provide insight into new directions in targeted therapeutic approaches for older adults with mantle cell lymphoma.
STATEMENT OF NEED
Mantle cell lymphoma (MCL) is a rare, aggressive B-cell non-Hodgkin lymphoma (NHL) accounting for 5% to 7% of all lymphomas. Its prognosis ranges from indolent disease that does not require treatment for years to very aggressive disease, which is associated with poor survival (Silkenstedt et al, 2021). Typically, MCL is diagnosed at advanced stage and in older patients who cannot tolerate intensive therapy (NCCN, 2022). Although recent advances have slightly increased remission rates, recurrence and relapse remain very common, leading to a median overall survival between 3 and 6 years (LLS, 2021). Though there are several effective options, progress is still needed towards establishing an accepted frontline approach for MCL (Castellino et al, 2022). Treatment selection and management of MCL are complicated by the heterogeneity of prognosis, advanced age and comorbidities of patients, and lack of an established standard approach for treatment, making it vital that clinicians be familiar with the latest research and advances in this area. In this activity chaired by Michael Wang, MD, Professor in the Department of Lymphoma & Myeloma at MD Anderson Cancer Center, expert faculty will discuss prognostic factors informing treatment, the promising results of recent trials in new therapeutic approaches, and the implications of treatment resistance in therapeutic selection for MCL.
Target Audience
Hematology/oncology fellows, attending faculty, and other health care professionals involved in the treatment of patients with mantle cell lymphoma (MCL).
Learning Objectives
1.) Identify clinical and biological prognostic factors that can guide treatment decision making for older adults with MCL
2.) Evaluate emerging data on targeted therapeutic approaches for treatment-naive and relapsed/refractory MCL and their applicability to older adults
3.) Assess mechanisms of resistance to targeted therapies for MCL and their implications for treatment selection
- Video recording of this lecture in English language: https://youtu.be/lK81BzxMqdo
- Video recording of this lecture in Arabic language: https://youtu.be/Ve4P0COk9OI
- Link to download the book free: https://nephrotube.blogspot.com/p/nephrotube-nephrology-books.html
- Link to NephroTube website: www.NephroTube.com
- Link to NephroTube social media accounts: https://nephrotube.blogspot.com/p/join-nephrotube-on-social-media.html
Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
These lecture slides, by Dr Sidra Arshad, offer a quick overview of physiological basis of a normal electrocardiogram.
Learning objectives:
1. Define an electrocardiogram (ECG) and electrocardiography
2. Describe how dipoles generated by the heart produce the waveforms of the ECG
3. Describe the components of a normal electrocardiogram of a typical bipolar leads (limb II)
4. Differentiate between intervals and segments
5. Enlist some common indications for obtaining an ECG
Study Resources:
1. Chapter 11, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 9, Human Physiology - From Cells to Systems, Lauralee Sherwood, 9th edition
3. Chapter 29, Ganong’s Review of Medical Physiology, 26th edition
4. Electrocardiogram, StatPearls - https://www.ncbi.nlm.nih.gov/books/NBK549803/
5. ECG in Medical Practice by ABM Abdullah, 4th edition
6. ECG Basics, http://www.nataliescasebook.com/tag/e-c-g-basics
Ethanol (CH3CH2OH), or beverage alcohol, is a two-carbon alcohol
that is rapidly distributed in the body and brain. Ethanol alters many
neurochemical systems and has rewarding and addictive properties. It
is the oldest recreational drug and likely contributes to more morbidity,
mortality, and public health costs than all illicit drugs combined. The
5th edition of the Diagnostic and Statistical Manual of Mental Disorders
(DSM-5) integrates alcohol abuse and alcohol dependence into a single
disorder called alcohol use disorder (AUD), with mild, moderate,
and severe subclassifications (American Psychiatric Association, 2013).
In the DSM-5, all types of substance abuse and dependence have been
combined into a single substance use disorder (SUD) on a continuum
from mild to severe. A diagnosis of AUD requires that at least two of
the 11 DSM-5 behaviors be present within a 12-month period (mild
AUD: 2–3 criteria; moderate AUD: 4–5 criteria; severe AUD: 6–11 criteria).
The four main behavioral effects of AUD are impaired control over
drinking, negative social consequences, risky use, and altered physiological
effects (tolerance, withdrawal). This chapter presents an overview
of the prevalence and harmful consequences of AUD in the U.S.,
the systemic nature of the disease, neurocircuitry and stages of AUD,
comorbidities, fetal alcohol spectrum disorders, genetic risk factors, and
pharmacotherapies for AUD.
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Pulmonary Thromboembolism - etilogy, types, medical- Surgical and nursing man...VarunMahajani
Disruption of blood supply to lung alveoli due to blockage of one or more pulmonary blood vessels is called as Pulmonary thromboembolism. In this presentation we will discuss its causes, types and its management in depth.
Flu Vaccine Alert in Bangalore Karnatakaaddon Scans
As flu season approaches, health officials in Bangalore, Karnataka, are urging residents to get their flu vaccinations. The seasonal flu, while common, can lead to severe health complications, particularly for vulnerable populations such as young children, the elderly, and those with underlying health conditions.
Dr. Vidisha Kumari, a leading epidemiologist in Bangalore, emphasizes the importance of getting vaccinated. "The flu vaccine is our best defense against the influenza virus. It not only protects individuals but also helps prevent the spread of the virus in our communities," he says.
This year, the flu season is expected to coincide with a potential increase in other respiratory illnesses. The Karnataka Health Department has launched an awareness campaign highlighting the significance of flu vaccinations. They have set up multiple vaccination centers across Bangalore, making it convenient for residents to receive their shots.
To encourage widespread vaccination, the government is also collaborating with local schools, workplaces, and community centers to facilitate vaccination drives. Special attention is being given to ensuring that the vaccine is accessible to all, including marginalized communities who may have limited access to healthcare.
Residents are reminded that the flu vaccine is safe and effective. Common side effects are mild and may include soreness at the injection site, mild fever, or muscle aches. These side effects are generally short-lived and far less severe than the flu itself.
Healthcare providers are also stressing the importance of continuing COVID-19 precautions. Wearing masks, practicing good hand hygiene, and maintaining social distancing are still crucial, especially in crowded places.
Protect yourself and your loved ones by getting vaccinated. Together, we can help keep Bangalore healthy and safe this flu season. For more information on vaccination centers and schedules, residents can visit the Karnataka Health Department’s official website or follow their social media pages.
Stay informed, stay safe, and get your flu shot today!
Lung Cancer: Artificial Intelligence, Synergetics, Complex System Analysis, S...Oleg Kshivets
RESULTS: Overall life span (LS) was 2252.1±1742.5 days and cumulative 5-year survival (5YS) reached 73.2%, 10 years – 64.8%, 20 years – 42.5%. 513 LCP lived more than 5 years (LS=3124.6±1525.6 days), 148 LCP – more than 10 years (LS=5054.4±1504.1 days).199 LCP died because of LC (LS=562.7±374.5 days). 5YS of LCP after bi/lobectomies was significantly superior in comparison with LCP after pneumonectomies (78.1% vs.63.7%, P=0.00001 by log-rank test). AT significantly improved 5YS (66.3% vs. 34.8%) (P=0.00000 by log-rank test) only for LCP with N1-2. Cox modeling displayed that 5YS of LCP significantly depended on: phase transition (PT) early-invasive LC in terms of synergetics, PT N0—N12, cell ratio factors (ratio between cancer cells- CC and blood cells subpopulations), G1-3, histology, glucose, AT, blood cell circuit, prothrombin index, heparin tolerance, recalcification time (P=0.000-0.038). Neural networks, genetic algorithm selection and bootstrap simulation revealed relationships between 5YS and PT early-invasive LC (rank=1), PT N0—N12 (rank=2), thrombocytes/CC (3), erythrocytes/CC (4), eosinophils/CC (5), healthy cells/CC (6), lymphocytes/CC (7), segmented neutrophils/CC (8), stick neutrophils/CC (9), monocytes/CC (10); leucocytes/CC (11). Correct prediction of 5YS was 100% by neural networks computing (area under ROC curve=1.0; error=0.0).
CONCLUSIONS: 5YS of LCP after radical procedures significantly depended on: 1) PT early-invasive cancer; 2) PT N0--N12; 3) cell ratio factors; 4) blood cell circuit; 5) biochemical factors; 6) hemostasis system; 7) AT; 8) LC characteristics; 9) LC cell dynamics; 10) surgery type: lobectomy/pneumonectomy; 11) anthropometric data. Optimal diagnosis and treatment strategies for LC are: 1) screening and early detection of LC; 2) availability of experienced thoracic surgeons because of complexity of radical procedures; 3) aggressive en block surgery and adequate lymph node dissection for completeness; 4) precise prediction; 5) adjuvant chemoimmunoradiotherapy for LCP with unfavorable prognosis.
Lung Cancer: Artificial Intelligence, Synergetics, Complex System Analysis, S...
Atls review and burn
1. ATLS UPDATE
ACUTE BURN
MANAGEMENTDr Awaneesh Katiyar
(M.Ch. Senior Resident)
Trauma Surgery and Critical Care
All India Institute of Medical Sciences, Rishikesh, UK.
2. A 25 years male patient arrived in ED, with history
of Patient was on bike at 80km/hour and collide with
truck- at arrival he was P-124/min, BP- 80/60 , no eye
opening on pain full stimulus abnormal flexion is
noted, right leg is crushed with active bleeding from
popliteal artery and scalp is noted , Right sided
reduced air entry and paradoxical movement.
What you will do first ?
a. Intubate him
b. Give compression to stop bleed
c. Bolus of 2L lactated warm
saline.
d. Put right side chest tube.
3. Preparation
A – laryngoscope, ET tube, bougie, Suction machine drugs, c- collar
B- chest tube kit with water seal
C- warm saline, wide bore cannula,
Adjunct – Spo2 probe , BP , FAST machine , catheter, RT and
4. Triage – red / yellow / green (multiple causalities / mass causalities)
1. Life or limb – red
2. Walking wounded – green
3. Rest goes to yellow
5. Primary survey with immediate resuscitation of life threatening injuries
1. A-Secure airway / c-collar
2. B-Assess breathing n Ventilate the patient
3. C-Control bleeding and shock – 1 litre warm saline
with wide bore / tranexa1gm
4. D- neurological disability
6. Adjuncts to primary survey
Rule of 2 in trauma
1. 2- things to monitor – Spo2 / BP
2. 2 – tubes – catheter / RT
3. 2- X-rays – CXR/PXR
4. 2- Test – ABG/blood group
5. 2- machine`s – Xrays / FAST
7. Secondary survey – head to toe examination and patient history.
1. Head
2. Neck
3. Maxillofacial
4. Chest
5. Abdomen and pelvis
6. Lower extremity
7. Upper extremity
8. Spine
9. Soft tissue
10. Airway maintenance and restriction of cervical spine motion
Breathing and ventilation
Circulation with hemorrhage control
Disability ( assessment of neurologic status)
Exposure/ Environment control
11. A B C D
Airway – kills in seconds
Breathing – takes minutes
Circulation – bleeding kills in hours
Disability (neurological)– hours to days ( except brain stem)
12. A 25 years male patient arrived in ED, with history
of Patient was on bike at 80km/hour and collide with
truck- at arrival he was P-124/min, BP- 80/60 , no eye
opening on pain full stimulus abnormal flexion is
noted, right leg is crushed with active bleeding from
popliteal artery and scalp is noted , Right sided
reduced air entry and paradoxical movement.
What you will do first ?
a. Intubate him
b. Give compression to stop bleed
c. Bolus of 2L lactated warm
saline.
d. Put right side chest tube.
13.
14. 10 SECONDS
asking the patient for his or her name,
asking what happened to you?
• Able to phonate- airway is normal
• Able complete sentence – breathing normal
• Well oriented – circulation normal with no
neurological deficit
21. A 25 years male patient arrived in ED, with history
of Patient was on bike at 80km/hour and collide with
truck- at arrival he was P-124/min, BP- 80/60 , no eye
opening on pain full stimulus abnormal flexion is
noted, right leg is crushed with active bleeding from
popliteal artery and scalp is noted , Right sided
reduced air entry and paradoxical movement.
What you will do in this
patient?
If patient is having
difficult airway
26. A 25 years male patient arrived in ED, with history
of Patient was on bike at 80km/hour and collide with
truck- at arrival he was P-124/min, BP- 80/60 , no eye
opening on pain full stimulus abnormal flexion is
noted, right leg is crushed with active bleeding from
popliteal artery and scalp is noted , Right sided
reduced air entry and paradoxical movement.
What you will do for chest
?
30. Minimum 3
Maximum 15
<8 severe
9-12 moderate
>13 mild
Motor - prognostic
31. Injuries to the musculoskeletal system
are common in trauma patients. The
delayed recognition and treatment of
these injuries can result in life-
threatening hemorrhage or limb loss.
32. External bleeding
Change in pulse quality
Ankle/ brachial index
Signs of vascular injury - soft/hard
33. Stepwise approach of bleeding control.
Manual pressure , bandage pressure dressing,
manual tourniquet/ pneumatic
250mmHg upper limb / 400mmHg in lower limb.
Should be kept for 1hour.
34. Multiple injuries - intensive resuscitation and/or emergency surgery for extremity
or other injuries is not a candidate for replantation.
Re-implantation is usually performed in isolated injury.
Wash with RL – wrap with moist gauze – then moist towel – plastic bag –
insulated chest with crushed ice(avoid freezing).
35. Traumatic Rhabdomyolysis
Acute tubular necrosis – AKI
Shock further leads to death
Assessment
Amber color urine
Myo-globinuria
S. Creatine Kinase-> 10KU/L
Associated Metabolic acidosis,
hyperkalemia, hycalcemia, further
DIC.
Management
Initiate early and aggressive IV fluids.
Which is critical to renal protection
Alkalinization of urine and osmotic
diuresis
Early diagnosis
active treatment - prevent mortality
36. Increased pressure within a musculo-
fascial compartment causes ischemia
and subsequent necrosis.
Increase in compartment content
Decrease in size of compartment.
Tight dressing ,
Crush injury/ hemotoma ,
Prolong external pressure,
Reperfusion ,
Burn,
Excessive exercise.
37. Signs and symptoms
Pain greater than expected and out of proportion to the
stimulus or injury
Pain on passive stretch of the affected muscle
Tense swelling of the affected compartment
Paresthesias or altered sensation distal to the affected
compartment
Delayed diagnosis
Neurological deficit
Muscle necrosis
Ischemic contracture
Infection
Delayed healing
Untreated –
amputation
Absence of distal pulse and delayed capillary refilling
Weakness and paralysis
Compartment syndrome is a clinical diagnosis. Pressure
measurements are only an adjunct to aid in its diagnosis.
38. - Only treatment is fasciotomy
Delay in management
leads to –
myoglobenuria
AKI
sometimes death
39. The most significant difference
between burns and other injuries
is that the consequences of burn
injury are directly linked to the
extent of the inflammatory
response to the injury.
40. A
B C D EPrimary survey-
1. Stop burning process
2. Establish airway - early intubation
is the key in inhalational injury
The airway can become obstructed not only
from direct injury (e.g., inhalation injury) but
also from the massive edema resulting from the
burn injury.
41. ABLS
Signs of respiratory compromise
Decreased level of consciousness
where airway protective reflexes are
impaired
Anticipated patient transfer of large
burn with airway issue without
qualified personnel to intubate en
route
Signs of airway obstruction
Extent of the burn (total body
surface area burn > 40%–50%)
Extensive and deep facial burns
Burns inside the mouth
Significant edema or risk for
edema
Difficulty swallowing
42. Direct thermal injury to the lower airway is very rare
Exposure to superheated steam or ignition of inhaled
flammable gases.
Breathing concerns arise from three general causes:
hypoxia,
carbon monoxide poisoning,
smoke inhalation injury.
43. Inhalation injury,
Poor compliance due to
circumferential chest burns,
thoracic trauma unrelated to the
thermal injury.
MANAGEMENT
Administer supplemental oxygen with or
without intubation.
Breathing concerns
1. Hypoxia,
2. CO poisoning,
3. smoke inhalation
injury.
44. CO POISONING
Burned in enclosed areas.
Direct measurement of carboxyhemoglobin (HbCO).
Patients with CO levels of less than 20% usually
have no physical symptoms.
Higher CO levels can result in:
headache and nausea (20%–30%)
confusion (30%–40%)
coma (40%–60%)
death (>60%)
Breathing concerns
1. Hypoxia,
2. CO poisoning,
3. smoke inhalation
injury.
Facts of CO to remember
240 times that of O2.
Half life – 4 hours at FiO2 21%
- 40 minutes FiO2 100%
45. Important Facts
1. Spo2 monitor not reliable in CO poisoning
2. Cherry red skin – rare
3. Stridor may be late – sign
4. Before transfer to burn center – always evaluate airway and consider for
intubation if needed.
5. Intubate with – appropriate size tube
(use endotracheal tubes at least 7.5 mm ID or larger in an adult and size 4.5 mm
ID ETT in a child.)
46. ABA - 2 Criteria to diagnose
1. Exposure to the combustible agent
2. Exposure of smoke to lower airway below
vocal cards – seen in bronchodcopy
Breathing concerns
1. Hypoxia,
2. CO poisoning,
3. smoke inhalation
injury.
Mortality increases to double in
inhalational injury with burn as compare
to isolated burn
high likelihood – inhalational injury
Adult >20% burn
<10/>50 years of age > 10% burn
47. Investigation
Chest x-ray and
Arterial blood gas determination.
Management –
Early airway management.
Avoid hypoxia
Breathing concerns
1. Hypoxia,
2. CO poisoning,
3. smoke inhalation
injury.
48. CIRCULATION
Polytrauma - hemorrhagic losses,
Burn - ongoing losses from capillary leak
due to inflammation.
Burn resuscitation fluids for deep partial
and full-thickness burns larger than 20%
TBSA.
TAKING CARE NOT TO OVER
RESUSCITATE
TASK
- To secure the IV Line in burn
If peripheral IV line cannot be
obtained, consider central venous
access or intraosseous infusion.
49. Goal – to maintain perfusion of the
tissue and avoid over fluid
resuscitation.
Which fluid ?
How much fluid?
Its less or over or enough!
50. All Adult or children >30 kg - Warmed isotonic lactated ringer’s solution
Childen <30 kg - D5LR
Which fluid ?
How much fluid?
Its less or over or enough!
51. Which fluid ?
How much fluid?
Its less or over or enough!
Adopted by – ABA
2ml (RL) x Kg x TBSA for 2nd and 3rd degree
Traditional parkland – avoided for concerns about over- resuscitation and
associated mortality.
Calculated fluid volume – half- 8hours half in subsequent 16 hours.
Pediatric burn patients should
begin at 3 mL/kg/% TBSA;
52. ITS LESS OR
OVER OR
ENOUGH
Which fluid ?
How much fluid?
Its less or over or enough!
55. Gently cover the wound
Do not break blisters or apply an antiseptic agent.
Remove any previously applied medication before using antibacterial topical
agents.
Do not apply cold water to a patient with extensive burns (i.e., > 10% TBSA)
A fresh burn is a clean area that must be protected from contamination.
Early diagnosis of compartment and immediate fasciotomy
Consider for adequate pain relief, antibiotics and tetanus prophylaxis.
56. CASE SCENARIO
23years male (100kg) sustained
2nd degree burn almost 80% burn
while working in the factory ?
What you will do first ?
57. Airway -
Breathing
Circulation – fluid
Exposure and wound care
58. Identify life and limb threatening injuries – immediate react on it.
ABCDE – universal
Identification and simultaneous management is the key.
Always keep in the mind –
- life is always important over limb,
- limb over disability or cost
59. THANK YOU
Or Mail me @
drawaneeshkatiyar@gmail.com
Asking questions?make you wise.