The document provides information on myocardial infarction (MI or heart attack) including definitions, causes, pathophysiology, clinical manifestations, diagnostic tests, treatment, nursing management, and patient education. It defines MI as myocardial cell death due to prolonged ischemia. The main cause is sudden blockage of the coronary artery by a blood clot, causing irreversible damage to heart muscle. Clinical manifestations may include chest pain, shortness of breath, nausea, and changes in vital signs. Diagnostic tests include electrocardiogram, cardiac enzymes, and echocardiogram. Treatment focuses on reperfusion, reducing oxygen demand on the heart, and preventing complications. Nursing management involves monitoring for complications, relieving symptoms, promoting perfusion and respiratory function
The inflammation of the heart muscles, such as myocarditis, the membrane sac which surrounds the heart called as pericarditis, and the inner lining of the heart or the myocardium, heart muscle as endocarditis are known as the inflammatory heart diseases.
The Coronary Artery Disease
Also called: CAD, Coronary arteriosclerosis, Coronary atherosclerosis.
Coronary artery disease (CAD) is the most common type of heart disease. It is the leading cause of death in the United States in both men and women.
CAD happens when the arteries that supply blood to heart muscle become hardened and narrowed. This is due to the buildup of cholesterol and other material, called plaque, on their inner walls. This buildup is called atherosclerosis. As it grows, less blood can flow through the arteries. As a result, the heart muscle can't get the blood or oxygen it needs. This can lead to chest pain (angina) or a heart attack. Most heart attacks happen when a blood clot suddenly cuts off the hearts' blood supply, causing permanent heart damage.
Over time, CAD can also weaken the heart muscle and contribute to heart failure and arrhythmias. Heart failure means the heart can't pump blood well to the rest of the body. Arrhythmias are changes in the normal beating rhythm of the heart.
Treatments and Therapies
Angioplasty: MedlinePlus Health Topic From the National Institutes of Health (National Library of Medicine)
Also in Spanish
Cardiac Procedures and Surgeries (American Heart Association)
Coronary Artery Bypass Surgery: MedlinePlus Health Topic From the National Institutes of Health (National Library of Medicine)
Also in Spanish
Coronary Artery Disease: Angioplasty or Bypass Surgery? (Mayo Foundation for Medical Education and Research)
Also in Spanish
Coronary Artery Revascularization in Patients with Diabetes Mellitus (American Heart Association)
What Is a Stent? From the National Institutes of Health (National Heart, Lung, and Blood Institute)
Heart failure, sometimes known as congestive heart failure, occurs when your heart muscle doesn't pump blood as well as it should. Certain conditions, such as narrowed arteries in your heart (coronary artery disease) or high blood pressure, gradually leave your heart too weak or stiff to fill and pump efficiently.
It contains meaning, pathophysiology, types, risk factors, lab and diagnostic procedures and tests, Rx goals, appropriate medications for ANGINA PECTORIS ..... Enjoy and Learn from it!!!!
A blockage of blood flow to the heart muscle.
A heart attack is a medical emergency. A heart attack usually occurs when a blood clot blocks blood flow to the heart. Without blood, tissue loses oxygen and dies.
Symptoms include tightness or pain in the chest, neck, back or arms, as well as fatigue, lightheadedness, abnormal heartbeat and anxiety. Women are more likely to have atypical symptoms than men.
Treatment ranges from lifestyle changes and cardiac rehabilitation to medication, stents and bypass surgery.
The inflammation of the heart muscles, such as myocarditis, the membrane sac which surrounds the heart called as pericarditis, and the inner lining of the heart or the myocardium, heart muscle as endocarditis are known as the inflammatory heart diseases.
The Coronary Artery Disease
Also called: CAD, Coronary arteriosclerosis, Coronary atherosclerosis.
Coronary artery disease (CAD) is the most common type of heart disease. It is the leading cause of death in the United States in both men and women.
CAD happens when the arteries that supply blood to heart muscle become hardened and narrowed. This is due to the buildup of cholesterol and other material, called plaque, on their inner walls. This buildup is called atherosclerosis. As it grows, less blood can flow through the arteries. As a result, the heart muscle can't get the blood or oxygen it needs. This can lead to chest pain (angina) or a heart attack. Most heart attacks happen when a blood clot suddenly cuts off the hearts' blood supply, causing permanent heart damage.
Over time, CAD can also weaken the heart muscle and contribute to heart failure and arrhythmias. Heart failure means the heart can't pump blood well to the rest of the body. Arrhythmias are changes in the normal beating rhythm of the heart.
Treatments and Therapies
Angioplasty: MedlinePlus Health Topic From the National Institutes of Health (National Library of Medicine)
Also in Spanish
Cardiac Procedures and Surgeries (American Heart Association)
Coronary Artery Bypass Surgery: MedlinePlus Health Topic From the National Institutes of Health (National Library of Medicine)
Also in Spanish
Coronary Artery Disease: Angioplasty or Bypass Surgery? (Mayo Foundation for Medical Education and Research)
Also in Spanish
Coronary Artery Revascularization in Patients with Diabetes Mellitus (American Heart Association)
What Is a Stent? From the National Institutes of Health (National Heart, Lung, and Blood Institute)
Heart failure, sometimes known as congestive heart failure, occurs when your heart muscle doesn't pump blood as well as it should. Certain conditions, such as narrowed arteries in your heart (coronary artery disease) or high blood pressure, gradually leave your heart too weak or stiff to fill and pump efficiently.
It contains meaning, pathophysiology, types, risk factors, lab and diagnostic procedures and tests, Rx goals, appropriate medications for ANGINA PECTORIS ..... Enjoy and Learn from it!!!!
A blockage of blood flow to the heart muscle.
A heart attack is a medical emergency. A heart attack usually occurs when a blood clot blocks blood flow to the heart. Without blood, tissue loses oxygen and dies.
Symptoms include tightness or pain in the chest, neck, back or arms, as well as fatigue, lightheadedness, abnormal heartbeat and anxiety. Women are more likely to have atypical symptoms than men.
Treatment ranges from lifestyle changes and cardiac rehabilitation to medication, stents and bypass surgery.
Chest pain Case Presentation with managementMuqtasidkhan
CASE presentation of chest pain types, causes, investigations, management. cardiac vs non cardiac pain. life threatening chest pain. MI, ACS, PNEUMOTHORAX, PE, GERD, AORTIC DISSECTION.
shock is the state of insufficient blood flow to the tissues of the body .it contains introduction, definition, stages of shock, types of shock, diagnostic evaluation, prognosis ,prevention, care for each stage.
A myocardial infarction (commonly called a heart attack) is an extremely dangerous condition caused by a lack of blood flow to your heart muscle. The lack of blood flow can occur because of many different factors but is usually related to a blockage in one or more of your heart's arteries.
Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
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micro teaching on communication m.sc nursing.pdfAnurag Sharma
Microteaching is a unique model of practice teaching. It is a viable instrument for the. desired change in the teaching behavior or the behavior potential which, in specified types of real. classroom situations, tends to facilitate the achievement of specified types of objectives.
- Video recording of this lecture in English language: https://youtu.be/lK81BzxMqdo
- Video recording of this lecture in Arabic language: https://youtu.be/Ve4P0COk9OI
- Link to download the book free: https://nephrotube.blogspot.com/p/nephrotube-nephrology-books.html
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Knee anatomy and clinical tests 2024.pdfvimalpl1234
This includes all relevant anatomy and clinical tests compiled from standard textbooks, Campbell,netter etc..It is comprehensive and best suited for orthopaedicians and orthopaedic residents.
Lung Cancer: Artificial Intelligence, Synergetics, Complex System Analysis, S...Oleg Kshivets
RESULTS: Overall life span (LS) was 2252.1±1742.5 days and cumulative 5-year survival (5YS) reached 73.2%, 10 years – 64.8%, 20 years – 42.5%. 513 LCP lived more than 5 years (LS=3124.6±1525.6 days), 148 LCP – more than 10 years (LS=5054.4±1504.1 days).199 LCP died because of LC (LS=562.7±374.5 days). 5YS of LCP after bi/lobectomies was significantly superior in comparison with LCP after pneumonectomies (78.1% vs.63.7%, P=0.00001 by log-rank test). AT significantly improved 5YS (66.3% vs. 34.8%) (P=0.00000 by log-rank test) only for LCP with N1-2. Cox modeling displayed that 5YS of LCP significantly depended on: phase transition (PT) early-invasive LC in terms of synergetics, PT N0—N12, cell ratio factors (ratio between cancer cells- CC and blood cells subpopulations), G1-3, histology, glucose, AT, blood cell circuit, prothrombin index, heparin tolerance, recalcification time (P=0.000-0.038). Neural networks, genetic algorithm selection and bootstrap simulation revealed relationships between 5YS and PT early-invasive LC (rank=1), PT N0—N12 (rank=2), thrombocytes/CC (3), erythrocytes/CC (4), eosinophils/CC (5), healthy cells/CC (6), lymphocytes/CC (7), segmented neutrophils/CC (8), stick neutrophils/CC (9), monocytes/CC (10); leucocytes/CC (11). Correct prediction of 5YS was 100% by neural networks computing (area under ROC curve=1.0; error=0.0).
CONCLUSIONS: 5YS of LCP after radical procedures significantly depended on: 1) PT early-invasive cancer; 2) PT N0--N12; 3) cell ratio factors; 4) blood cell circuit; 5) biochemical factors; 6) hemostasis system; 7) AT; 8) LC characteristics; 9) LC cell dynamics; 10) surgery type: lobectomy/pneumonectomy; 11) anthropometric data. Optimal diagnosis and treatment strategies for LC are: 1) screening and early detection of LC; 2) availability of experienced thoracic surgeons because of complexity of radical procedures; 3) aggressive en block surgery and adequate lymph node dissection for completeness; 4) precise prediction; 5) adjuvant chemoimmunoradiotherapy for LCP with unfavorable prognosis.
Prix Galien International 2024 Forum ProgramLevi Shapiro
June 20, 2024, Prix Galien International and Jerusalem Ethics Forum in ROME. Detailed agenda including panels:
- ADVANCES IN CARDIOLOGY: A NEW PARADIGM IS COMING
- WOMEN’S HEALTH: FERTILITY PRESERVATION
- WHAT’S NEW IN THE TREATMENT OF INFECTIOUS,
ONCOLOGICAL AND INFLAMMATORY SKIN DISEASES?
- ARTIFICIAL INTELLIGENCE AND ETHICS
- GENE THERAPY
- BEYOND BORDERS: GLOBAL INITIATIVES FOR DEMOCRATIZING LIFE SCIENCE TECHNOLOGIES AND PROMOTING ACCESS TO HEALTHCARE
- ETHICAL CHALLENGES IN LIFE SCIENCES
- Prix Galien International Awards Ceremony
Recomendações da OMS sobre cuidados maternos e neonatais para uma experiência pós-natal positiva.
Em consonância com os ODS – Objetivos do Desenvolvimento Sustentável e a Estratégia Global para a Saúde das Mulheres, Crianças e Adolescentes, e aplicando uma abordagem baseada nos direitos humanos, os esforços de cuidados pós-natais devem expandir-se para além da cobertura e da simples sobrevivência, de modo a incluir cuidados de qualidade.
Estas diretrizes visam melhorar a qualidade dos cuidados pós-natais essenciais e de rotina prestados às mulheres e aos recém-nascidos, com o objetivo final de melhorar a saúde e o bem-estar materno e neonatal.
Uma “experiência pós-natal positiva” é um resultado importante para todas as mulheres que dão à luz e para os seus recém-nascidos, estabelecendo as bases para a melhoria da saúde e do bem-estar a curto e longo prazo. Uma experiência pós-natal positiva é definida como aquela em que as mulheres, pessoas que gestam, os recém-nascidos, os casais, os pais, os cuidadores e as famílias recebem informação consistente, garantia e apoio de profissionais de saúde motivados; e onde um sistema de saúde flexível e com recursos reconheça as necessidades das mulheres e dos bebês e respeite o seu contexto cultural.
Estas diretrizes consolidadas apresentam algumas recomendações novas e já bem fundamentadas sobre cuidados pós-natais de rotina para mulheres e neonatos que recebem cuidados no pós-parto em unidades de saúde ou na comunidade, independentemente dos recursos disponíveis.
É fornecido um conjunto abrangente de recomendações para cuidados durante o período puerperal, com ênfase nos cuidados essenciais que todas as mulheres e recém-nascidos devem receber, e com a devida atenção à qualidade dos cuidados; isto é, a entrega e a experiência do cuidado recebido. Estas diretrizes atualizam e ampliam as recomendações da OMS de 2014 sobre cuidados pós-natais da mãe e do recém-nascido e complementam as atuais diretrizes da OMS sobre a gestão de complicações pós-natais.
O estabelecimento da amamentação e o manejo das principais intercorrências é contemplada.
Recomendamos muito.
Vamos discutir essas recomendações no nosso curso de pós-graduação em Aleitamento no Instituto Ciclos.
Esta publicação só está disponível em inglês até o momento.
Prof. Marcus Renato de Carvalho
www.agostodourado.com
New Drug Discovery and Development .....NEHA GUPTA
The "New Drug Discovery and Development" process involves the identification, design, testing, and manufacturing of novel pharmaceutical compounds with the aim of introducing new and improved treatments for various medical conditions. This comprehensive endeavor encompasses various stages, including target identification, preclinical studies, clinical trials, regulatory approval, and post-market surveillance. It involves multidisciplinary collaboration among scientists, researchers, clinicians, regulatory experts, and pharmaceutical companies to bring innovative therapies to market and address unmet medical needs.
NVBDCP.pptx Nation vector borne disease control programSapna Thakur
NVBDCP was launched in 2003-2004 . Vector-Borne Disease: Disease that results from an infection transmitted to humans and other animals by blood-feeding arthropods, such as mosquitoes, ticks, and fleas. Examples of vector-borne diseases include Dengue fever, West Nile Virus, Lyme disease, and malaria.
Pulmonary Thromboembolism - etilogy, types, medical- Surgical and nursing man...VarunMahajani
Disruption of blood supply to lung alveoli due to blockage of one or more pulmonary blood vessels is called as Pulmonary thromboembolism. In this presentation we will discuss its causes, types and its management in depth.
2. Learning Outcomes
At the end of the lecture, student will be able to:
Define acute myocardial infarction (AMI).
State the etiology of AMI.
Explain the pathophysiology of AMI
State the clinical manifestations of AMI.
State the diagnostic tests that helps to confirmed the
AMI.
2
3. Cont. Learning Outcomes
At the end of the lecture, student will be able to:
Explain the treatment and management for AMI.
Explain the nursing management for patient with
AMI.
Provide health teaching on preventive measures to
patients prior to discharge.
3
7. Acute Myocardial Infarction (AMI)
Also called a heart attack.
Defines as myocardial cell death due to
prolonged ischemia.
(Malaysian Heart Assoc, 2019).
Myocardial injury is myocardial cell death due to
non ischemic causes.
7
8. Cont..
AMI is diagnosed by the rise and/fall in cardiac
troponins with at lease one value > 99 percentile
with accompanied with at lease one of the following:
clinical history consistent with chest pain of
ischemic origin
ECG changes
imaging evidence of new loss of variable
myocardium
identification of an intracoronary (IC) thrombus
by angioplasty
(Malaysian Heart Assoc, 2019).
8
9. Cont.
The main cause of AMI ---> sudden blockage
in the coronary artery ---> due to formation of
blood clot (thrombus) ---> causing irreversible
damage (necrosis) to the heart muscle.
9
13. Pathogenesis of MI
13
Myocardial necrosis begins within 20-30 minutes, mostly
starting at the sub-endocardial region (less perfuse, high
intramural pressure.
Infarct reaches its full size within 3-6 hrs; during this
period, lysis of the thrombus by streptokinase or tPA
(tissue plasminogen activator), may limit the size of the
infarct.
14. Etiology & Risk Factors
Most heart attack results from atherosclerosis
Family history CAD
Hypercholesterolemia
↑LDL ↓HDL
↑BP
Obesity
14
18. Cont.
Fever
Nausea & vomiting ---> results from
vasovagus reflexes.
Crackles sound, peripheral edema & hepatic
enlargement ---> indicates cardiac failure.
Cardiac vascular manifestations:
elevated BP and HR
BP may drop as decreased co and urinary output.
18
19. Diagnostic Tests
Clinical history consistent chest pain of ischemic
origin.
Serum Troponin I or T
Serum CK-MB (cretinine kinase-myocardial band)
12-lead ECG (ST-T wave elevated by greater than
1 mm or > in two continuous leads ---> MI)
19
20. Cont.
Echocardiogram
Serum potassium, magnesium & calcium
(imbalances & acidosis may cause change in
conduction & contractile).
20
23. Pre-Hospital Management
Patient with suspected ST segment elevation
MI (STEMI), should be given soluble or
chewable 300 mg aspirin and 300 mg
clopidogrel.
These patient should be rapidly transported to
the hospital for early initiation of reperfusion
strategies.
DO NOT BRING TO A CLINIC
23
24. Treatment & Management
The goals of medical management are to:
minimize myocardial damage, preserve myocardial
function and prevent complications lethal
dysrhythmias and cardiogenic shock:
1) Reperfusion – use of the percutaneous coronary
intervention (PCI) or thrombolytic medications.
2) Reduce myocardial oxygen demand & increase
oxygen supply with medications, oxygen therapy
& bed rest.
3) Coronary artery bypass or minimally invasive direct
coronary bypass (MIDCAB)
24
25. Cont..
Pharmacological Therapy
Nitrates (nitroglycerin) to increase oxygen supply
Anticoagulants (aspirin, heparin)
Analgesics (morphine sulfate)
Angiotensin-converting enzyme inhibitors
Beta blocker initially & a prescription to continue its
use after hospital discharge
Thrombolytics (tPA, Activase) and reteplase (rPA,
TNKase) ---> must be administered after the onset of
symptoms, generally within 3-6hours
25
28. Contraindication for Thrombolytic
Therapy
Active Bleeding
Known bleeding disorder
History of hemorrhagic stroke
Recent major surgery or trauma
Uncontrolled hypertension, CVA
Pregnancy
30. Complications Post STEMI
Important complications following STEMI:
Arrhythmias
Heart failure due to extensive myocardial damage or
mechanical complications.
Chest pain post STEMI may be due to:
reinfarction / recurrent MI
post infarct angina
pericarditis
non cardiac causes such as gastritis (epigastric pain)
30
31. Nursing Management
Obtain baseline data on current status for
comparison with ongoing status include:
history of chest pain or discomfort,
dyspnea,
palpitations,
unusual fatigue,
faintness (syncope) or
diaphoresis.
31
32. Cont..
1. Perform a complete physical assessment
---> for detecting complications & any change in
status. The examination include:
Assess level of consciousness
Evaluate chest pain (most important clinical
finding).
Assess heart sound to detect an early sign of
impending LVF.
Measure BP to determine response to pain &
treatment.
32
33. Cont..
Note narrowed pulse pressure after MI, suggesting
ineffective ventricular contraction.
Assess bowel movement. Serve laxative to prevent
straining.
Observe urinary output and check for edema; an
early sign of cardiogenic shock in hypotension with
oliguria.
Examine IV lines and sites frequently.
2. Maintain CRIB
3. Assist in ADLs
33
34. Nursing Diagnoses
1) Ineffective cardiac tissue perfusion related to
reduced coronary blood flow.
2) Risk for imbalanced fluid volume.
3) Risk for ineffective peripheral tissue perfusion
related to decreased cardiac output from left
ventricular dysfunction.
4) Death anxiety.
5) Deficient knowledge about post acute coronary
syndrome self-care.
34
35. Nursing Interventions
Relieve sign & symptoms of ischemia.
Administer oxygen to reduce pain associated with
low levels of circulating oxygen.
Assess vital signs frequently to detect
hemodynamic changes.
Position patient of Fowler’s position or put on
cardiac bed to decrease chest discomfort and
dyspnea.
35
36. Cont…
Improving Respiratory Function
Assess respiratory function to detect an early
signs of complications.
Monitor fluid volume status to prevent
overloading the heart and lungs.
Encourage patient to perform deep breathing
exercise and change position often to prevent
pooling fluid in lungs bases.
36
37. Cont…
Promoting Adequate Tissue Perfusion
Maintain patient on bed rest
rest to reduce myocardial oxygen consumption.
Check skin temperature and peripheral pulses
frequently
determine adequate tissue perfusion.
37
38. Cont…
Reduce Anxiety
Develop a therapeutic relationship with patient.
Allow patient to express feelings.
Provide information to the patient and family in an
honest and supportive manner.
Ensure a quiet environment, prevent interruptions that
disturb sleep.
Use a caring and appropriate touch, relaxation
technique, and use humor.
Provide spiritual support consistent with patient’s
beliefs.
Provide divertional therapy.
38
39. Health Teaching
Compliance with prescribed medication.
Adhere to the prescribed cardiac rehabilitation
regimen.
Assist patient with scheduling & keeping follow up
appointments for monitoring, laboratory test, ECG
and general health screening.
Advise family member to assist patient in adhere to
restrictions dietary advice.
Instruct patient to monitor for sign of complications
and seek for medical attention immediately.
39
40. Cont.
Advice patient to change life styles:
Stop smoking and alcohol intake
Regular exercise as advised at lease 3 times a
week (cardio-exercise).
Diet modifications
Stress management
Maintain ideal body weight
40
41. References
Amsterdam EA, Wenger NK, Brindis RG, et al. (2014).
AHA/ACC guideline for the management of patients with
non–ST-elevation acute coronary syndromes. J Am Coll
Cardiol. 2014;64(24):e139- 228. [PMID:25260718]
Basavanthappa. B.T. (2015). Medical Surgical Nursing (3rd ed.).
New Delhi: Jaypee.
Black, J. M., & Hawks, J. H. (2011). Medical Surgical Nursing.
(8th ed.). St. Louis, UK: Saunders.
Ignatavicius, D. D., & Workman, M. L. (2016). Medical-Surgical
Nursing: Patient-Centered Collaboration Care. (8th ed.).
Singapore: Elsevier.
https://www.malaysianheart.org/files/5cb6bf193304e.pdf
41
Early management of STEMI is directed at:
Pain relief – analgesic & oxygen therapy
Establishing early reperfusion – use of Percutaneous Coronary Intervention (PCI) or thrombolytic medications
Treatment of complications