SlideShare a Scribd company logo
APPROACH TO UNCONSCIOUS
PATIENT
Dr. Tanveen Kamal
Assistant Professor
Department Of Neurosurgery
Shahabuddin Medical College & Hospital
CONSCIOUSNESS, WHAT IS IT??
Consciousness is the state of full awareness of the self and one’s relationship to the
environment.
Clinically, the level of consciousness of a patient is defined at the bedside
by the responses of the patient to the examiner.
It is clear from this definition that it is possible for a patient to be conscious yet not
responsive to the examiner.
For example,
• if the patient lacks sensory inputs,
• is paralyzed (Locked In Syndrome),
• or for psychologic reasons
decides not to respond
WHAT IS SLEEP ,THEN?
Sleep is a form of reduced consciousness in which the responsiveness of
brain systems responsible for cognitive function is globally reduced,
So that the brain does not respond readily to environmental stimuli.
Pathologic alteration of the relationships between the brain systems that
are responsible for wakefulness and sleep can impair consciousness.
A key difference between sleep and coma is that sleep is intrinsically
reversible:
sufficient stimulation will return the individual to a normal waking state
SO……WHO IS UNCONSCIOUS ???
Patients who have a sleep like appearance and remain
behaviorally unresponsive to all external stimuli are unconscious
by any definition.
• Impaired consciousness is among the most difficult and
dramatic of clinical problems.
The brain tolerates only limited physical or metabolic injury, so that
impaired consciousness is often a sign of impending irreparable
damage to the brain.
TERMS USED TO DESCRIBE
DISORDERS OF CONSCIOUSNESS
Acute Chronic
Clouding
Delirium
Obtundation
Stupor
Coma
Locked in Syndrome
Dementia
Hypersomnia
Abulic
Akinetic mutism
Minimal consciousness
Vegetative State
Brain Death
PROBLEMS WITH DIFFERENT TERMS….IT’S
CONFUSING!!
• A wide variety of systemic and intracranial problems produce
depression of conscious level.
• Accurate assessment and recording are essential to determine
deterioration or improvement in a patient’s condition
• In 1974 Teasdale and Jennett, in Glasgow, developed a system for
conscious level assessment.
• They discarded vague terms such as stupor, semi coma and deep
coma
And described conscious level in terms of
•EYE OPENING,
•VERBAL RESPONSE AND
•MOTOR RESPONSE
EYE OPENING -4 CATEGORIES
4
3
2 1
EYE OPENING -4 CATEGORIES-DESCRIPTION
Patient’s visual or eye response
 Score your patient a 4 if he/she can open their eyes spontaneously
 Score your patient a 3 if they open their eyes to verbal speech -“Mr. X”or
light touch
 Score your patient a 2 if your patient requires painful stimuli to open
their eyes
 Score your patient a 1 if their eyes do not open. Patient is unresponsive.
VERBAL RESPONSE- 5 categories
Patient’s verbal response (if intubated or tracheotomy score NT=not
testable / T=Tube)
Score your patient a 5 if patient is oriented x 3 (He or she knows who
they are, where they are, and what the date is)
Score your patient a 4 if you not they are having some confusion or
forgetfulness (unable to answer all orientation questions)
Score your patient a 3 if they are not understood and using inappropriate
words (i.e. swearing, unrelated words, aggressive)
Score your patient a 2 if they are not able to form words and just can
make sounds
Score your patient a 1 if they are unresponsive and make no noise
MOTOR RESPONSE
Motor Response -6 Categories
6
Localizing to pain
Apply a painful stimulus to the
supraorbital nerve,
e.g. rub thumb nail in the
supraorbital groove,
increasing pressure until a
response is
obtained
5
If the patient does not localize to
supraorbital pressure, apply
pressure with a pen or hard
object to the nail bed. Record
elbow flexion as
‘flexing to pain’.
4
Abnormal flexion to a painful stimulus typically involves
• Adduction of the arm,
• internal rotation of the shoulder,
• flexion of the elbow,
• pronation of the forearm and wrist flexion (known as decorticate
posturing).
3
• Abnormal extension to a painful stimulus is also known as decerebrate
posturing.
• In decerebrate posturing, the head is extended, with the arms and legs
also extended and internally rotated.
• The patient appears rigid with their teeth clenched.
• The signs can be on just one side of the body or on both sides (the signs
may only be present in the upper limbs)
2
GCS (Glasgow Coma Scale)
Examination of Unconscious patient-History
• Questioning relatives, friends or the ambulance team is an essential part
of the assessment of the unconscious patient
• Has the patient sustained a head injury – leading to admission, or in the
preceding weeks?
• Did the patient collapse suddenly?-Indicating that an acute problem is
more likely
• Did limb twitching occur?-Explain
• Have symptoms occurred in the preceding weeks?
• Has the patient suffered a previous illness?-DM/HTN/IHD
• Does the patient take medication?-Such as Antiplatelets/Anticoagulants
GENERAL EXAMINATION
Lack of patient co-operation does not limit general examination and this
may reveal important diagnostic signs.
In addition to those features, also look for
• signs of head injury,
• needle marks on the arm and
• evidence of tongue biting.
• Also note the smell of alcohol- beware of attributing the patient’s clinical
state solely to alcohol excess
NEUROLOGICAL EXAMINATION
• Conscious level: This assessment is of major importance. It
not only serves as an immediate prognostic guide, but also
provides a baseline with which future examinations may be
compared.
• Assess conscious level as described previously in terms
of eye opening,
verbal response and
motor response
• It is important to avoid the tendency to simply quote the patient’s
total score. This can be misleading.
• Describing the conscious level in terms of the actual responses
i.e.
• ‘no eye opening, no verbal response and extending’, avoids
any confusion over numbers
ADDITIONAL EXAMINATIONS
•Pupil response
•Funduscopy-if possible
•Corneal reflex
– tone
•Limb – reflexes
•Plantar response
IMPORTANCE OF PUPIL EXAMINATION
• The pupil size should be recorded in millimeters and the reactivity
documented as present, sluggish or absent
• UNCAL HERNIATION can compress the third nerve, compromising the
parasympathetic supply to the pupil.
• Unopposed sympathetic activity produces a sluggish enlarged pupil,
progressing to fixed and dilated under continued compression
Examination of EYE Movements- MUST Exclude
Cervical injury first
VESTIBULO-OCULAR REFLEX
Method: Water at 30°C irrigated into the external auditory meatus. Nystagmus
usually develops after a 20 second delay and lasts for more than a minute
The test is repeated after 5 minutes with water at 44°C.
Cold water effectively reduces the vestibular output from one side, creating an
imbalance and producing eye drift towards the irrigated ear.
Rapid corrective movements result in ‘nystagmus’ to the opposite ear. Hot water
(44°) reverses the convection current, increases the vestibular output and changes
the direction of nystagmus.
• Nystagmus is a condition in which the eyes make repetitive,
uncontrolled movements.
• These movements often result in reduced vision and depth
perception and can affect balance and coordination. These
involuntary eye movements can occur from side to side, up and
down, or in a circular pattern.
VISUAL FIELDS
In the unco-operative
patient, the examiner may
detect a field defect when
‘menacing’ from one side
fails to produce a ‘blink’
FACIAL WEAKNESS
Facial weakness
Failure to ‘grimace’
on one side in
response to bilateral
supraorbital pain
indicates a facial
weakness.
LIMB WEAKNESS
Detect by comparing the response in
the limbs to painful stimuli. If pain
produces an asymmetric response,
then limb weakness is present.
(If the patient ‘localises’ with one
arm, hold this down and retest to
ensure that a similar response
cannot be elicited
from the other limb.)
LOWER LIMBS
• Pain stimulus applied to the toe nails or Achilles tendon similarly tests power in
the lower limbs.
• Variation in tone,
• reflexes or
• plantar responses between each side also indicates a focal deficit.
• In practice, if the examiner fails to detect a difference in response to painful
stimuli, these additional features seldom provide convincing evidence
• Patients who do not meet all the discharge criteria will need admission for a further
period of observation and/or brain imaging.
GUIDELINES FOR COMPUTED TOMOGRAPHY (CT) IN HEAD INJURY.
Indications for CT imaging in head injury within 1 hour
● GCS <13 at any point
● GCS <15 at 2 hours
● Focal neurological deficit
● Suspected open, depressed or basal skull fracture
● More than one episode of vomiting
● Post-traumatic seizure
Indications for CT imaging within 8 hours
● Age >65
● Coagulopathy (e.g. aspirin, warfarin or rivaroxaban use)
● Dangerous mechanism of injury (e.g. fall from a height, RTA)
● Retrograde amnesia >30 minutes
QUESTIONS???
APPROACH TO UNCONSCIOUS PATIENT.pptx

More Related Content

What's hot

Ketamine
KetamineKetamine
Alpha adrenergic blockers
Alpha adrenergic blockersAlpha adrenergic blockers
Alpha adrenergic blockers
Rudhra Prabhakar
 
Ketamine
KetamineKetamine
Ketamine
Karunesh Kumar
 
Methylprednisolone
MethylprednisoloneMethylprednisolone
Methylprednisolone
Shilpa Garg
 
Propofol
PropofolPropofol
complications of Anesthesia.pptx
complications of Anesthesia.pptxcomplications of Anesthesia.pptx
complications of Anesthesia.pptx
Mahmood Hasan Taha
 
Morphine &amp; fentanyl nida
Morphine &amp; fentanyl nidaMorphine &amp; fentanyl nida
Morphine &amp; fentanyl nida
Nida fatima
 
Skeletal Muscle Relaxants
Skeletal Muscle RelaxantsSkeletal Muscle Relaxants
Skeletal Muscle Relaxants
Pradeep Singh Narwat
 
Penicillin Allergy
Penicillin AllergyPenicillin Allergy
Penicillin Allergy
د. أنور الموسوي
 
Propofol ppt nandini
Propofol ppt nandiniPropofol ppt nandini
Propofol ppt nandini
Dr Nandini Deshpande
 
Antiepileptics
Antiepileptics Antiepileptics
Antiepileptics
Naser Tadvi
 
Local anestheticst systemic toxicity
Local  anestheticst systemic toxicityLocal  anestheticst systemic toxicity
Local anestheticst systemic toxicity
ram krishna
 
Anaphylaxis
AnaphylaxisAnaphylaxis
Anaphylaxis
VASS Yukon
 
Metoclopramide Midication
Metoclopramide MidicationMetoclopramide Midication
Metoclopramide Midication
Omaar Faraji
 
Anti Ig E biologics and allergic diseases
Anti Ig E biologics and allergic diseasesAnti Ig E biologics and allergic diseases
Anti Ig E biologics and allergic diseases
Chulalongkorn Allergy and Clinical Immunology Research Group
 
Mannitol,afh
Mannitol,afhMannitol,afh
Induction of anaesthesia
Induction of anaesthesiaInduction of anaesthesia
Induction of anaesthesia
Parul Gupta
 
Ketamine
KetamineKetamine
Anaphylaxis
AnaphylaxisAnaphylaxis
Mannitol
MannitolMannitol

What's hot (20)

Ketamine
KetamineKetamine
Ketamine
 
Alpha adrenergic blockers
Alpha adrenergic blockersAlpha adrenergic blockers
Alpha adrenergic blockers
 
Ketamine
KetamineKetamine
Ketamine
 
Methylprednisolone
MethylprednisoloneMethylprednisolone
Methylprednisolone
 
Propofol
PropofolPropofol
Propofol
 
complications of Anesthesia.pptx
complications of Anesthesia.pptxcomplications of Anesthesia.pptx
complications of Anesthesia.pptx
 
Morphine &amp; fentanyl nida
Morphine &amp; fentanyl nidaMorphine &amp; fentanyl nida
Morphine &amp; fentanyl nida
 
Skeletal Muscle Relaxants
Skeletal Muscle RelaxantsSkeletal Muscle Relaxants
Skeletal Muscle Relaxants
 
Penicillin Allergy
Penicillin AllergyPenicillin Allergy
Penicillin Allergy
 
Propofol ppt nandini
Propofol ppt nandiniPropofol ppt nandini
Propofol ppt nandini
 
Antiepileptics
Antiepileptics Antiepileptics
Antiepileptics
 
Local anestheticst systemic toxicity
Local  anestheticst systemic toxicityLocal  anestheticst systemic toxicity
Local anestheticst systemic toxicity
 
Anaphylaxis
AnaphylaxisAnaphylaxis
Anaphylaxis
 
Metoclopramide Midication
Metoclopramide MidicationMetoclopramide Midication
Metoclopramide Midication
 
Anti Ig E biologics and allergic diseases
Anti Ig E biologics and allergic diseasesAnti Ig E biologics and allergic diseases
Anti Ig E biologics and allergic diseases
 
Mannitol,afh
Mannitol,afhMannitol,afh
Mannitol,afh
 
Induction of anaesthesia
Induction of anaesthesiaInduction of anaesthesia
Induction of anaesthesia
 
Ketamine
KetamineKetamine
Ketamine
 
Anaphylaxis
AnaphylaxisAnaphylaxis
Anaphylaxis
 
Mannitol
MannitolMannitol
Mannitol
 

Similar to APPROACH TO UNCONSCIOUS PATIENT.pptx

neurological assessment.pptx
neurological assessment.pptxneurological assessment.pptx
neurological assessment.pptx
asst professer
 
gcs(Glasgow Coma Scale) ppt to understand.pptx
gcs(Glasgow Coma Scale)  ppt to understand.pptxgcs(Glasgow Coma Scale)  ppt to understand.pptx
gcs(Glasgow Coma Scale) ppt to understand.pptx
jkm36403
 
Cmc neuro 101
Cmc neuro 101Cmc neuro 101
Cmc neuro 101
ChristinaGraciani
 
NEUROLOGIC EXAMINATION.pptx
NEUROLOGIC EXAMINATION.pptxNEUROLOGIC EXAMINATION.pptx
NEUROLOGIC EXAMINATION.pptx
Abhay Rajpoot
 
Neurological assessment.
Neurological assessment.Neurological assessment.
Neurological assessment.
Gianne Gregorio
 
Glasgow coma scale.ppt
Glasgow coma scale.pptGlasgow coma scale.ppt
Glasgow coma scale.ppt
Sumit2018
 
Neurological examination.pptx
Neurological examination.pptxNeurological examination.pptx
Neurological examination.pptx
TumpaRana
 
NEUROLOGICAL ASSESSMENT
NEUROLOGICAL ASSESSMENTNEUROLOGICAL ASSESSMENT
NEUROLOGICAL ASSESSMENT
Manikandan T
 
Unconsciousness - Advance Nursing Practice
Unconsciousness - Advance Nursing PracticeUnconsciousness - Advance Nursing Practice
Unconsciousness - Advance Nursing Practice
Jaice Mary Joy
 
Nsg care with Unconsciousness.pptx
Nsg care with Unconsciousness.pptxNsg care with Unconsciousness.pptx
Nsg care with Unconsciousness.pptx
Abhishek Joshi
 
Neuro Assessment
Neuro AssessmentNeuro Assessment
Neuro Assessment
babykian05
 
Neurologic Nursing 1
Neurologic Nursing 1Neurologic Nursing 1
Neurologic Nursing 1
Jofred Martinez
 
cek.pptx
cek.pptxcek.pptx
cek.pptx
MerryKartika3
 
Nihss
NihssNihss
Nihss
ASHMAL
 
Altered Loc
Altered LocAltered Loc
Altered Loc
mycomic
 
Neurological Examination
Neurological ExaminationNeurological Examination
Neurological Examination
kabilansilas
 
Neurological Assessment for nursing students ppt
Neurological Assessment for nursing students pptNeurological Assessment for nursing students ppt
Neurological Assessment for nursing students ppt
blessyjannu21
 
Neurological assessment ppt
Neurological assessment pptNeurological assessment ppt
Neurological assessment ppt
Manali Solanki
 
In-Service Project - Dizzy Exam
In-Service Project - Dizzy ExamIn-Service Project - Dizzy Exam
In-Service Project - Dizzy Exam
Susan Miller
 
neurological examination ppt
neurological examination pptneurological examination ppt
neurological examination ppt
kabilansilas
 

Similar to APPROACH TO UNCONSCIOUS PATIENT.pptx (20)

neurological assessment.pptx
neurological assessment.pptxneurological assessment.pptx
neurological assessment.pptx
 
gcs(Glasgow Coma Scale) ppt to understand.pptx
gcs(Glasgow Coma Scale)  ppt to understand.pptxgcs(Glasgow Coma Scale)  ppt to understand.pptx
gcs(Glasgow Coma Scale) ppt to understand.pptx
 
Cmc neuro 101
Cmc neuro 101Cmc neuro 101
Cmc neuro 101
 
NEUROLOGIC EXAMINATION.pptx
NEUROLOGIC EXAMINATION.pptxNEUROLOGIC EXAMINATION.pptx
NEUROLOGIC EXAMINATION.pptx
 
Neurological assessment.
Neurological assessment.Neurological assessment.
Neurological assessment.
 
Glasgow coma scale.ppt
Glasgow coma scale.pptGlasgow coma scale.ppt
Glasgow coma scale.ppt
 
Neurological examination.pptx
Neurological examination.pptxNeurological examination.pptx
Neurological examination.pptx
 
NEUROLOGICAL ASSESSMENT
NEUROLOGICAL ASSESSMENTNEUROLOGICAL ASSESSMENT
NEUROLOGICAL ASSESSMENT
 
Unconsciousness - Advance Nursing Practice
Unconsciousness - Advance Nursing PracticeUnconsciousness - Advance Nursing Practice
Unconsciousness - Advance Nursing Practice
 
Nsg care with Unconsciousness.pptx
Nsg care with Unconsciousness.pptxNsg care with Unconsciousness.pptx
Nsg care with Unconsciousness.pptx
 
Neuro Assessment
Neuro AssessmentNeuro Assessment
Neuro Assessment
 
Neurologic Nursing 1
Neurologic Nursing 1Neurologic Nursing 1
Neurologic Nursing 1
 
cek.pptx
cek.pptxcek.pptx
cek.pptx
 
Nihss
NihssNihss
Nihss
 
Altered Loc
Altered LocAltered Loc
Altered Loc
 
Neurological Examination
Neurological ExaminationNeurological Examination
Neurological Examination
 
Neurological Assessment for nursing students ppt
Neurological Assessment for nursing students pptNeurological Assessment for nursing students ppt
Neurological Assessment for nursing students ppt
 
Neurological assessment ppt
Neurological assessment pptNeurological assessment ppt
Neurological assessment ppt
 
In-Service Project - Dizzy Exam
In-Service Project - Dizzy ExamIn-Service Project - Dizzy Exam
In-Service Project - Dizzy Exam
 
neurological examination ppt
neurological examination pptneurological examination ppt
neurological examination ppt
 

Recently uploaded

PGx Analysis in VarSeq: A User’s Perspective
PGx Analysis in VarSeq: A User’s PerspectivePGx Analysis in VarSeq: A User’s Perspective
PGx Analysis in VarSeq: A User’s Perspective
Golden Helix
 
Nano-gold for Cancer Therapy chemistry investigatory project
Nano-gold for Cancer Therapy chemistry investigatory projectNano-gold for Cancer Therapy chemistry investigatory project
Nano-gold for Cancer Therapy chemistry investigatory project
SIVAVINAYAKPK
 
Call Girl Pune 7339748667 Vip Call Girls Pune
Call Girl Pune 7339748667 Vip Call Girls PuneCall Girl Pune 7339748667 Vip Call Girls Pune
Call Girl Pune 7339748667 Vip Call Girls Pune
Mobile Problem
 
Can Traditional Chinese Medicine Treat Blocked Fallopian Tubes.pptx
Can Traditional Chinese Medicine Treat Blocked Fallopian Tubes.pptxCan Traditional Chinese Medicine Treat Blocked Fallopian Tubes.pptx
Can Traditional Chinese Medicine Treat Blocked Fallopian Tubes.pptx
FFragrant
 
ENVIRONMENTAL SANITATION in community setting
ENVIRONMENTAL SANITATION in community settingENVIRONMENTAL SANITATION in community setting
ENVIRONMENTAL SANITATION in community setting
ShraddhaTamshettiwar
 
Osvaldo Bernardo Muchanga-GASTROINTESTINAL INFECTIONS AND GASTRITIS-2024.pdf
Osvaldo Bernardo Muchanga-GASTROINTESTINAL INFECTIONS AND GASTRITIS-2024.pdfOsvaldo Bernardo Muchanga-GASTROINTESTINAL INFECTIONS AND GASTRITIS-2024.pdf
Osvaldo Bernardo Muchanga-GASTROINTESTINAL INFECTIONS AND GASTRITIS-2024.pdf
Osvaldo Bernardo Muchanga
 
Breast cancer: Post menopausal endocrine therapy
Breast cancer: Post menopausal endocrine therapyBreast cancer: Post menopausal endocrine therapy
Breast cancer: Post menopausal endocrine therapy
Dr. Sumit KUMAR
 
Pharmacology of Drugs for Congestive Heart Failure
Pharmacology of Drugs for Congestive Heart FailurePharmacology of Drugs for Congestive Heart Failure
Pharmacology of Drugs for Congestive Heart Failure
Dr. Nikhilkumar Sakle
 
Nutritional deficiency disorder in Child
Nutritional deficiency disorder in ChildNutritional deficiency disorder in Child
Nutritional deficiency disorder in Child
Bhavyakelawadiya
 
Hemodialysis: Chapter 6, Hemodialysis Adequacy and Dose - Dr.Gawad
Hemodialysis: Chapter 6, Hemodialysis Adequacy and Dose - Dr.GawadHemodialysis: Chapter 6, Hemodialysis Adequacy and Dose - Dr.Gawad
Hemodialysis: Chapter 6, Hemodialysis Adequacy and Dose - Dr.Gawad
NephroTube - Dr.Gawad
 
Brain specific drug delivery.pptx -Mpharm
Brain specific drug delivery.pptx -MpharmBrain specific drug delivery.pptx -Mpharm
Brain specific drug delivery.pptx -Mpharm
MuskanShingari
 
Patellar Instability: Diagnosis Management
Patellar Instability: Diagnosis  ManagementPatellar Instability: Diagnosis  Management
Patellar Instability: Diagnosis Management
Dr Nitin Tyagi
 
All about shoulder Joint ..
All about shoulder Joint .. All about shoulder Joint ..
All about shoulder Joint ..
Aswan University Hospital
 
2nd-generation Antihistaminic Part I.pptx
2nd-generation Antihistaminic Part I.pptx2nd-generation Antihistaminic Part I.pptx
2nd-generation Antihistaminic Part I.pptx
Madhumita Dixit
 
Helminthiasis or Worm infestation in Children for Nursing students
Helminthiasis or Worm infestation in Children for Nursing studentsHelminthiasis or Worm infestation in Children for Nursing students
Helminthiasis or Worm infestation in Children for Nursing students
RAJU B N
 
Allopurinol (Anti-gout drug).pptx
Allopurinol (Anti-gout drug).pptxAllopurinol (Anti-gout drug).pptx
Allopurinol (Anti-gout drug).pptx
Madhumita Dixit
 
Call Girls Kolkata 💯Call Us 🔝 7374876321 🔝 💃 Top Class Call Girl Service A...
Call Girls Kolkata   💯Call Us 🔝 7374876321 🔝 💃  Top Class Call Girl Service A...Call Girls Kolkata   💯Call Us 🔝 7374876321 🔝 💃  Top Class Call Girl Service A...
Call Girls Kolkata 💯Call Us 🔝 7374876321 🔝 💃 Top Class Call Girl Service A...
daljeetsingh9909
 
Computer in pharmaceutical research and development-Mpharm(Pharmaceutics)
Computer in pharmaceutical research and development-Mpharm(Pharmaceutics)Computer in pharmaceutical research and development-Mpharm(Pharmaceutics)
Computer in pharmaceutical research and development-Mpharm(Pharmaceutics)
MuskanShingari
 
Selective α1-Blocker.pptx
Selective α1-Blocker.pptxSelective α1-Blocker.pptx
Selective α1-Blocker.pptx
Madhumita Dixit
 
Call Girls In Mumbai +91-7426014248 High Profile Call Girl Mumbai
Call Girls In Mumbai +91-7426014248 High Profile Call Girl MumbaiCall Girls In Mumbai +91-7426014248 High Profile Call Girl Mumbai
Call Girls In Mumbai +91-7426014248 High Profile Call Girl Mumbai
Mobile Problem
 

Recently uploaded (20)

PGx Analysis in VarSeq: A User’s Perspective
PGx Analysis in VarSeq: A User’s PerspectivePGx Analysis in VarSeq: A User’s Perspective
PGx Analysis in VarSeq: A User’s Perspective
 
Nano-gold for Cancer Therapy chemistry investigatory project
Nano-gold for Cancer Therapy chemistry investigatory projectNano-gold for Cancer Therapy chemistry investigatory project
Nano-gold for Cancer Therapy chemistry investigatory project
 
Call Girl Pune 7339748667 Vip Call Girls Pune
Call Girl Pune 7339748667 Vip Call Girls PuneCall Girl Pune 7339748667 Vip Call Girls Pune
Call Girl Pune 7339748667 Vip Call Girls Pune
 
Can Traditional Chinese Medicine Treat Blocked Fallopian Tubes.pptx
Can Traditional Chinese Medicine Treat Blocked Fallopian Tubes.pptxCan Traditional Chinese Medicine Treat Blocked Fallopian Tubes.pptx
Can Traditional Chinese Medicine Treat Blocked Fallopian Tubes.pptx
 
ENVIRONMENTAL SANITATION in community setting
ENVIRONMENTAL SANITATION in community settingENVIRONMENTAL SANITATION in community setting
ENVIRONMENTAL SANITATION in community setting
 
Osvaldo Bernardo Muchanga-GASTROINTESTINAL INFECTIONS AND GASTRITIS-2024.pdf
Osvaldo Bernardo Muchanga-GASTROINTESTINAL INFECTIONS AND GASTRITIS-2024.pdfOsvaldo Bernardo Muchanga-GASTROINTESTINAL INFECTIONS AND GASTRITIS-2024.pdf
Osvaldo Bernardo Muchanga-GASTROINTESTINAL INFECTIONS AND GASTRITIS-2024.pdf
 
Breast cancer: Post menopausal endocrine therapy
Breast cancer: Post menopausal endocrine therapyBreast cancer: Post menopausal endocrine therapy
Breast cancer: Post menopausal endocrine therapy
 
Pharmacology of Drugs for Congestive Heart Failure
Pharmacology of Drugs for Congestive Heart FailurePharmacology of Drugs for Congestive Heart Failure
Pharmacology of Drugs for Congestive Heart Failure
 
Nutritional deficiency disorder in Child
Nutritional deficiency disorder in ChildNutritional deficiency disorder in Child
Nutritional deficiency disorder in Child
 
Hemodialysis: Chapter 6, Hemodialysis Adequacy and Dose - Dr.Gawad
Hemodialysis: Chapter 6, Hemodialysis Adequacy and Dose - Dr.GawadHemodialysis: Chapter 6, Hemodialysis Adequacy and Dose - Dr.Gawad
Hemodialysis: Chapter 6, Hemodialysis Adequacy and Dose - Dr.Gawad
 
Brain specific drug delivery.pptx -Mpharm
Brain specific drug delivery.pptx -MpharmBrain specific drug delivery.pptx -Mpharm
Brain specific drug delivery.pptx -Mpharm
 
Patellar Instability: Diagnosis Management
Patellar Instability: Diagnosis  ManagementPatellar Instability: Diagnosis  Management
Patellar Instability: Diagnosis Management
 
All about shoulder Joint ..
All about shoulder Joint .. All about shoulder Joint ..
All about shoulder Joint ..
 
2nd-generation Antihistaminic Part I.pptx
2nd-generation Antihistaminic Part I.pptx2nd-generation Antihistaminic Part I.pptx
2nd-generation Antihistaminic Part I.pptx
 
Helminthiasis or Worm infestation in Children for Nursing students
Helminthiasis or Worm infestation in Children for Nursing studentsHelminthiasis or Worm infestation in Children for Nursing students
Helminthiasis or Worm infestation in Children for Nursing students
 
Allopurinol (Anti-gout drug).pptx
Allopurinol (Anti-gout drug).pptxAllopurinol (Anti-gout drug).pptx
Allopurinol (Anti-gout drug).pptx
 
Call Girls Kolkata 💯Call Us 🔝 7374876321 🔝 💃 Top Class Call Girl Service A...
Call Girls Kolkata   💯Call Us 🔝 7374876321 🔝 💃  Top Class Call Girl Service A...Call Girls Kolkata   💯Call Us 🔝 7374876321 🔝 💃  Top Class Call Girl Service A...
Call Girls Kolkata 💯Call Us 🔝 7374876321 🔝 💃 Top Class Call Girl Service A...
 
Computer in pharmaceutical research and development-Mpharm(Pharmaceutics)
Computer in pharmaceutical research and development-Mpharm(Pharmaceutics)Computer in pharmaceutical research and development-Mpharm(Pharmaceutics)
Computer in pharmaceutical research and development-Mpharm(Pharmaceutics)
 
Selective α1-Blocker.pptx
Selective α1-Blocker.pptxSelective α1-Blocker.pptx
Selective α1-Blocker.pptx
 
Call Girls In Mumbai +91-7426014248 High Profile Call Girl Mumbai
Call Girls In Mumbai +91-7426014248 High Profile Call Girl MumbaiCall Girls In Mumbai +91-7426014248 High Profile Call Girl Mumbai
Call Girls In Mumbai +91-7426014248 High Profile Call Girl Mumbai
 

APPROACH TO UNCONSCIOUS PATIENT.pptx

  • 1. APPROACH TO UNCONSCIOUS PATIENT Dr. Tanveen Kamal Assistant Professor Department Of Neurosurgery Shahabuddin Medical College & Hospital
  • 2. CONSCIOUSNESS, WHAT IS IT?? Consciousness is the state of full awareness of the self and one’s relationship to the environment. Clinically, the level of consciousness of a patient is defined at the bedside by the responses of the patient to the examiner. It is clear from this definition that it is possible for a patient to be conscious yet not responsive to the examiner. For example, • if the patient lacks sensory inputs, • is paralyzed (Locked In Syndrome), • or for psychologic reasons decides not to respond
  • 3. WHAT IS SLEEP ,THEN? Sleep is a form of reduced consciousness in which the responsiveness of brain systems responsible for cognitive function is globally reduced, So that the brain does not respond readily to environmental stimuli. Pathologic alteration of the relationships between the brain systems that are responsible for wakefulness and sleep can impair consciousness. A key difference between sleep and coma is that sleep is intrinsically reversible: sufficient stimulation will return the individual to a normal waking state
  • 4. SO……WHO IS UNCONSCIOUS ??? Patients who have a sleep like appearance and remain behaviorally unresponsive to all external stimuli are unconscious by any definition. • Impaired consciousness is among the most difficult and dramatic of clinical problems. The brain tolerates only limited physical or metabolic injury, so that impaired consciousness is often a sign of impending irreparable damage to the brain.
  • 5. TERMS USED TO DESCRIBE DISORDERS OF CONSCIOUSNESS Acute Chronic Clouding Delirium Obtundation Stupor Coma Locked in Syndrome Dementia Hypersomnia Abulic Akinetic mutism Minimal consciousness Vegetative State Brain Death
  • 6. PROBLEMS WITH DIFFERENT TERMS….IT’S CONFUSING!! • A wide variety of systemic and intracranial problems produce depression of conscious level. • Accurate assessment and recording are essential to determine deterioration or improvement in a patient’s condition • In 1974 Teasdale and Jennett, in Glasgow, developed a system for conscious level assessment. • They discarded vague terms such as stupor, semi coma and deep coma
  • 7. And described conscious level in terms of •EYE OPENING, •VERBAL RESPONSE AND •MOTOR RESPONSE
  • 8. EYE OPENING -4 CATEGORIES 4 3 2 1
  • 9. EYE OPENING -4 CATEGORIES-DESCRIPTION Patient’s visual or eye response  Score your patient a 4 if he/she can open their eyes spontaneously  Score your patient a 3 if they open their eyes to verbal speech -“Mr. X”or light touch  Score your patient a 2 if your patient requires painful stimuli to open their eyes  Score your patient a 1 if their eyes do not open. Patient is unresponsive.
  • 10. VERBAL RESPONSE- 5 categories Patient’s verbal response (if intubated or tracheotomy score NT=not testable / T=Tube) Score your patient a 5 if patient is oriented x 3 (He or she knows who they are, where they are, and what the date is) Score your patient a 4 if you not they are having some confusion or forgetfulness (unable to answer all orientation questions) Score your patient a 3 if they are not understood and using inappropriate words (i.e. swearing, unrelated words, aggressive) Score your patient a 2 if they are not able to form words and just can make sounds Score your patient a 1 if they are unresponsive and make no noise
  • 11. MOTOR RESPONSE Motor Response -6 Categories 6
  • 12. Localizing to pain Apply a painful stimulus to the supraorbital nerve, e.g. rub thumb nail in the supraorbital groove, increasing pressure until a response is obtained 5
  • 13. If the patient does not localize to supraorbital pressure, apply pressure with a pen or hard object to the nail bed. Record elbow flexion as ‘flexing to pain’. 4
  • 14. Abnormal flexion to a painful stimulus typically involves • Adduction of the arm, • internal rotation of the shoulder, • flexion of the elbow, • pronation of the forearm and wrist flexion (known as decorticate posturing). 3
  • 15. • Abnormal extension to a painful stimulus is also known as decerebrate posturing. • In decerebrate posturing, the head is extended, with the arms and legs also extended and internally rotated. • The patient appears rigid with their teeth clenched. • The signs can be on just one side of the body or on both sides (the signs may only be present in the upper limbs) 2
  • 17. Examination of Unconscious patient-History • Questioning relatives, friends or the ambulance team is an essential part of the assessment of the unconscious patient • Has the patient sustained a head injury – leading to admission, or in the preceding weeks? • Did the patient collapse suddenly?-Indicating that an acute problem is more likely • Did limb twitching occur?-Explain • Have symptoms occurred in the preceding weeks? • Has the patient suffered a previous illness?-DM/HTN/IHD • Does the patient take medication?-Such as Antiplatelets/Anticoagulants
  • 18. GENERAL EXAMINATION Lack of patient co-operation does not limit general examination and this may reveal important diagnostic signs. In addition to those features, also look for • signs of head injury, • needle marks on the arm and • evidence of tongue biting. • Also note the smell of alcohol- beware of attributing the patient’s clinical state solely to alcohol excess
  • 19. NEUROLOGICAL EXAMINATION • Conscious level: This assessment is of major importance. It not only serves as an immediate prognostic guide, but also provides a baseline with which future examinations may be compared. • Assess conscious level as described previously in terms of eye opening, verbal response and motor response
  • 20. • It is important to avoid the tendency to simply quote the patient’s total score. This can be misleading. • Describing the conscious level in terms of the actual responses i.e. • ‘no eye opening, no verbal response and extending’, avoids any confusion over numbers
  • 21. ADDITIONAL EXAMINATIONS •Pupil response •Funduscopy-if possible •Corneal reflex – tone •Limb – reflexes •Plantar response
  • 22. IMPORTANCE OF PUPIL EXAMINATION • The pupil size should be recorded in millimeters and the reactivity documented as present, sluggish or absent • UNCAL HERNIATION can compress the third nerve, compromising the parasympathetic supply to the pupil. • Unopposed sympathetic activity produces a sluggish enlarged pupil, progressing to fixed and dilated under continued compression
  • 23. Examination of EYE Movements- MUST Exclude Cervical injury first
  • 24. VESTIBULO-OCULAR REFLEX Method: Water at 30°C irrigated into the external auditory meatus. Nystagmus usually develops after a 20 second delay and lasts for more than a minute The test is repeated after 5 minutes with water at 44°C. Cold water effectively reduces the vestibular output from one side, creating an imbalance and producing eye drift towards the irrigated ear. Rapid corrective movements result in ‘nystagmus’ to the opposite ear. Hot water (44°) reverses the convection current, increases the vestibular output and changes the direction of nystagmus.
  • 25. • Nystagmus is a condition in which the eyes make repetitive, uncontrolled movements. • These movements often result in reduced vision and depth perception and can affect balance and coordination. These involuntary eye movements can occur from side to side, up and down, or in a circular pattern.
  • 26.
  • 27. VISUAL FIELDS In the unco-operative patient, the examiner may detect a field defect when ‘menacing’ from one side fails to produce a ‘blink’
  • 28. FACIAL WEAKNESS Facial weakness Failure to ‘grimace’ on one side in response to bilateral supraorbital pain indicates a facial weakness.
  • 29. LIMB WEAKNESS Detect by comparing the response in the limbs to painful stimuli. If pain produces an asymmetric response, then limb weakness is present. (If the patient ‘localises’ with one arm, hold this down and retest to ensure that a similar response cannot be elicited from the other limb.)
  • 30. LOWER LIMBS • Pain stimulus applied to the toe nails or Achilles tendon similarly tests power in the lower limbs. • Variation in tone, • reflexes or • plantar responses between each side also indicates a focal deficit. • In practice, if the examiner fails to detect a difference in response to painful stimuli, these additional features seldom provide convincing evidence
  • 31. • Patients who do not meet all the discharge criteria will need admission for a further period of observation and/or brain imaging. GUIDELINES FOR COMPUTED TOMOGRAPHY (CT) IN HEAD INJURY. Indications for CT imaging in head injury within 1 hour ● GCS <13 at any point ● GCS <15 at 2 hours ● Focal neurological deficit ● Suspected open, depressed or basal skull fracture ● More than one episode of vomiting ● Post-traumatic seizure Indications for CT imaging within 8 hours ● Age >65 ● Coagulopathy (e.g. aspirin, warfarin or rivaroxaban use) ● Dangerous mechanism of injury (e.g. fall from a height, RTA) ● Retrograde amnesia >30 minutes