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Blood
- 2. Match the functions of blood: Preventing blood loss Maintaining body temperature Maintaining pH Preventing infection Maintaining fluid volume in circulatory system Protective Regulatory
- 3. Match the functions of blood: Preventing blood loss Maintaining body temperature Maintaining pH Preventing infection Maintaining fluid volume in circulatory system Protective Regulatory
- 4. Blood is a connective tissue in which _____ is the non-living matrix and dissolved proteins become _____ strands during clotting. Formed elements, plasma Formed elements, platelets Plasma, platelets Plasma, fibrin
- 5. Blood is a connective tissue in which _____ is the non-living matrix and dissolved proteins become _____ strands during clotting. Formed elements, plasma Formed elements, platelets Plasma, platelets Plasma, fibrin
- 6. This is the percentage of erythrocytes in a blood sample, usually around 45%: Buffy coat Erythrocytes Leukocytes Hematocrit Plasma Albumin
- 7. This is the percentage of erythrocytes in a blood sample, usually around 45%: Buffy coat Erythrocytes Leukocytes Hematocrit Plasma Albumin
- 8. This large protein is the major contributor to plasma oncotic pressure: Buffy coat Erythrocytes Leukocytes Hematocrit Plasma Albumin
- 9. This large protein is the major contributor to plasma oncotic pressure: Buffy coat Erythrocytes Leukocytes Hematocrit Plasma Albumin
- 10. This is 90% water, but contains over 100 dissolved solutes: Buffy coat Erythrocytes Leukocytes Hematocrit Plasma Albumin
- 11. This is 90% water, but contains over 100 dissolved solutes: Buffy coat Erythrocytes Leukocytes Hematocrit Plasma Albumin
- 12. This thin, whitish layer of leukocytes and platelets forms between the erythrocytes and the plasma in a sample: Buffy coat Erythrocytes Leukocytes Hematocrit Plasma Albumin
- 13. This thin, whitish layer of leukocytes and platelets forms between the erythrocytes and the plasma in a sample: Buffy coat Erythrocytes Leukocytes Hematocrit Plasma Albumin
- 14. These are white blood cells: Buffy coat Erythrocytes Leukocytes Hematocrit Plasma Albumin
- 15. These are white blood cells: Buffy coat Erythrocytes Leukocytes Hematocrit Plasma Albumin
- 16. These are red blood cells: Buffy coat Erythrocytes Leukocytes Hematocrit Plasma Albumin
- 17. These are red blood cells: Buffy coat Erythrocytes Leukocytes Hematocrit Plasma Albumin
- 18. Which of the following statements about the formed elements are true? Blood cells divide in the plasma, and continually replace themselves Erythrocytes have no nuclei Platelets are cells that rupture to stop bleeding Most of the formed elements only last for a few days in the blood
- 19. Which of the following statements about the formed elements are true? Blood cells divide in the plasma, and continually replace themselves stem cells in the red marrow divide to replace blood cells Erythrocytes have no nuclei Platelets are cells that rupture to stop bleeding platelets are cell fragments Most of the formed elements only last for a few days in the blood
- 20. What is the function of spectrin in RBCs? Spectrin forms the extracellular matrix which anchors the agglutinogens that determine ABO blood typing Spectrin gives hemoglobin its three dimensional shape Spectin is the part of hemoglobin that oxygen binds to Spectrin creates a flexible endoskeleton that gives RBCs their characteristic concave disc shape and allows for compression and flexion as they navigate the capillaries
- 21. What is the function of spectrin in RBCs? Spectrin forms the extracellular matrix which anchors the agglutinogens that determine ABO blood typing Spectrin gives hemoglobin its three dimensional shape Spectin is the part of hemoglobin that oxygen binds to Spectrin creates a flexible endoskeleton that gives RBCs their characteristic concave disc shape and allows for compression and flexion as they navigate the capillaries
- 22. Which of the following make erythrocytes an ideal oxygen carrier? The small size and disc shape give erythrocytes a high surface to volume ratio ideal for gas diffusion RBCs are 97% hemoglobin RBCs generate ATP solely by anaerobic metabolism All of the above
- 23. Which of the following make erythrocytes an ideal oxygen carrier? The small size and disc shape give erythrocytes a high surface to volume ratio ideal for gas diffusion RBCs are 97% hemoglobin RBCs generate ATP solely by anaerobic metabolism All of the above
- 24. How many molecules of oxygen can each hemoglobin molecule transport? 1 2 4 8
- 25. How many molecules of oxygen can each hemoglobin molecule transport? 1 2 4 8
- 26. This is hemoglobin with oxygen bound: Heme Globin Oxyhemoglobin Deoxyhemoglobin Carbaminohemoglobin
- 27. This is hemoglobin with oxygen bound: Heme Globin Oxyhemoglobin Deoxyhemoglobin Carbaminohemoglobin
- 28. This is hemoglobin after offloading oxygen, reduced hemoglobin: Heme Globin Oxyhemoglobin Deoxyhemoglobin Carbaminohemoglobin
- 29. This is hemoglobin after offloading oxygen, reduced hemoglobin: Heme Globin Oxyhemoglobin Deoxyhemoglobin Carbaminohemoglobin
- 30. This red pigment contains the iron that binds to oxygen: Heme Globin Oxyhemoglobin Deoxyhemoglobin Carbaminohemoglobin
- 31. This red pigment contains the iron that binds to oxygen: Heme Globin Oxyhemoglobin Deoxyhemoglobin Carbaminohemoglobin
- 32. This is hemoglobin with carbon dioxide bound: Heme Globin Oxyhemoglobin Deoxyhemoglobin Carbaminohemoglobin
- 33. This is hemoglobin with carbon dioxide bound: Heme Globin Oxyhemoglobin Deoxyhemoglobin Carbaminohemoglobin
- 34. Two alpha and two beta chains of this are in each hemoglobin: Heme Globin Oxyhemoglobin Deoxyhemoglobin Carbaminohemoglobin
- 35. Two alpha and two beta chains of this are in each hemoglobin: Heme Globin Oxyhemoglobin Deoxyhemoglobin Carbaminohemoglobin
- 36. Erythrocyte production begins when hemocytoblasts transform into these: Hematopoeisis Erythropoiesis Erythroblast Reticulocyte Erythropoietin
- 37. Erythrocyte production begins when hemocytoblasts transform into these: Hematopoeisis Erythropoiesis Erythroblast Reticulocyte Erythropoietin
- 38. This immature RBC has ejected its nucleus and organelles, but still contains ribosomes: Hematopoeisis Erythropoiesis Erythroblast Reticulocyte Erythropoietin
- 39. This immature RBC has ejected its nucleus and organelles, but still contains ribosomes: Hematopoeisis Erythropoiesis Erythroblast Reticulocyte Erythropoietin
- 40. This is the process of RBC production, which takes place in the red marrow: Hematopoeisis Erythropoiesis Erythroblast Reticulocyte Erythropoietin
- 41. This is the process of RBC production, which takes place in the red marrow: Hematopoeisis Erythropoiesis Erythroblast Reticulocyte Erythropoietin
- 42. This hormone, produced in the kidneys stimulates erythrocyte production: Hematopoeisis Erythropoiesis Erythroblast Reticulocyte Erythropoietin
- 43. This hormone, produced in the kidneys stimulates erythrocyte production: Hematopoeisis Erythropoiesis Erythroblast Reticulocyte Erythropoietin
- 44. Which of the following statements about EPO production are true? EPO is produced mainly in the kidneys Hypoxia leads to insufficient degrading of hypoxia inducible factor (HIF), which accelerates synthesis of EPO Anything that leads to hypoxia or reduced oxygen transport can trigger EPO production All of these are true
- 45. Which of the following statements about EPO production are true? EPO is produced mainly in the kidneys Hypoxia leads to insufficient degrading of hypoxia inducible factor (HIF), which accelerates synthesis of EPO Anything that leads to hypoxia or reduced oxygen transport can trigger EPO production All of these are true
- 46. Iron is stored in cells as _____ and _____, and transported in the blood as _____. Transferrin, ferritin, hemosiderin Ferritin, hemosiderin, transferrin Hemosiderin, transferrin, ferritin
- 47. Iron is stored in cells as _____ and _____, and transported in the blood as _____. Transferrin, ferritin, hemosiderin Ferritin, hemosiderin, transferrin Hemosiderin, transferrin, ferritin
- 48. Erythrocytes have a life span of ____ days: 60-90 90-100 100-120 120-140
- 49. Erythrocytes have a life span of ____ days: 60-90 90-100 100-120 120-140
- 50. This can result from hemorrhage, dietary deficiency, or impaired iron absorption: Hemorrhagic anemia Iron deficiency anemia Pernicious anemia Renal anemia Aplastic anemia Thalassemia Sickle cell anemia Hemolytic anemia
- 51. This can result from hemorrhage, dietary deficiency, or impaired iron absorption: Hemorrhagic anemia Iron deficiency anemia Pernicious anemia Renal anemia Aplastic anemia Thalassemia Sickle cell anemia Hemolytic anemia
- 52. In this condition, an absent or faulty globin chain leads to hemoglobin deficient RBCs: Hemorrhagic anemia Iron deficiency anemia Pernicious anemia Renal anemia Aplastic anemia Thalassemia Sickle cell anemia Hemolytic anemia
- 53. In this condition, an absent or faulty globin chain leads to hemoglobin deficient RBCs: Hemorrhagic anemia Iron deficiency anemia Pernicious anemia Renal anemia Aplastic anemia Thalassemia Sickle cell anemia Hemolytic anemia
- 54. In this condition, an amino acid substitution causes beta globin chains to link together in low oxygen conditions: Hemorrhagic anemia Iron deficiency anemia Pernicious anemia Renal anemia Aplastic anemia Thalassemia Sickle cell anemia Hemolytic anemia
- 55. In this condition, an amino acid substitution causes beta globin chains to link together in low oxygen conditions: Hemorrhagic anemia Iron deficiency anemia Pernicious anemia Renal anemia Aplastic anemia Thalassemia Sickle cell anemia Hemolytic anemia
- 56. In this disease of the elderly, inability to absorb B12 leads prevents mitosis in developing erythrocytes: Hemorrhagic anemia Iron deficiency anemia Pernicious anemia Renal anemia Aplastic anemia Thalassemia Sickle cell anemia Hemolytic anemia
- 57. In this disease of the elderly, inability to absorb B12 leads prevents mitosis in developing erythrocytes: Hemorrhagic anemia Iron deficiency anemia Pernicious anemia Renal anemia Aplastic anemia Thalassemia Sickle cell anemia Hemolytic anemia
- 58. This is caused by a lack of EPO, leading to insufficient numbers of erythrocytes: Hemorrhagic anemia Iron deficiency anemia Pernicious anemia Renal anemia Aplastic anemia Thalassemia Sickle cell anemia Hemolytic anemia
- 59. This is caused by a lack of EPO, leading to insufficient numbers of erythrocytes: Hemorrhagic anemia Iron deficiency anemia Pernicious anemia Renal anemia Aplastic anemia Thalassemia Sickle cell anemia Hemolytic anemia
- 60. This can be due to rapid or chronic blood loss: Hemorrhagic anemia Iron deficiency anemia Pernicious anemia Renal anemia Aplastic anemia Thalassemia Sickle cell anemia Hemolytic anemia
- 61. This can be due to rapid or chronic blood loss: Hemorrhagic anemia Iron deficiency anemia Pernicious anemia Renal anemia Aplastic anemia Thalassemia Sickle cell anemia Hemolytic anemia
- 62. This results from destruction or damage to the red bone marrow, such as from chemotherapy: Hemorrhagic anemia Iron deficiency anemia Pernicious anemia Renal anemia Aplastic anemia Thalassemia Sickle cell anemia Hemolytic anemia
- 63. This results from destruction or damage to the red bone marrow, such as from chemotherapy: Hemorrhagic anemia Iron deficiency anemia Pernicious anemia Renal anemia Aplastic anemia Thalassemia Sickle cell anemia Hemolytic anemia
- 64. This occurs when RBCs lyse prematurely due to hemoglobin abnormalities, bateria, parasites, or mismatched transfusions: Hemorrhagic anemia Iron deficiency anemia Pernicious anemia Renal anemia Aplastic anemia Thalassemia Sickle cell anemia Hemolytic anemia
- 65. This occurs when RBCs lyse prematurely due to hemoglobin abnormalities, bateria, parasites, or mismatched transfusions: Hemorrhagic anemia Iron deficiency anemia Pernicious anemia Renal anemia Aplastic anemia Thalassemia Sickle cell anemia Hemolytic anemia
- 66. Which of the following can lead to polycythemia? Increased EPO production from high altitude Blood doping (infusion of additional erythrocytes) Prolonged hypoxia, such as from CHF Hemostatic mechanisms triggered by anemia
- 67. Which of the following can lead to polycythemia? Increased EPO production from high altitude Blood doping (infusion of additional erythrocytes) Prolonged hypoxia, such as from CHF Hemostatic mechanisms triggered by anemia
- 68. Match the leukocytes: Neutrophils Lymphocytes Eosinophils Monocytes Basophils Granulocytes Agranulocytes
- 69. Match the leukocytes: Neutrophils Lymphocytes Eosinophils Monocytes Basophils Granulocytes Agranulocytes
- 70. These are chemically attracted to inflammation, and phagocytize bacteria and some fungi: Neutrophils Eosinophils Basophils Lymphocytes Monocytes
- 71. These are chemically attracted to inflammation, and phagocytize bacteria and some fungi: Neutrophils Eosinophils Basophils Lymphocytes Monocytes
- 72. These are active against parasitic worms that are too large to be phagocytized: Neutrophils Eosinophils Basophils Lymphocytes Monocytes
- 73. These are active against parasitic worms that are too large to be phagocytized: Neutrophils Eosinophils Basophils Lymphocytes Monocytes
- 74. These leave the bloodstream to differentiate into macrophages, where they phagocytize pathogens in the tissue: Neutrophils Eosinophils Basophils Lymphocytes Monocytes
- 75. These leave the bloodstream to differentiate into macrophages, where they phagocytize pathogens in the tissue: Neutrophils Eosinophils Basophils Lymphocytes Monocytes
- 76. These have granules of histamine that attract cause inflammation and attract other WBCs to injuries: Neutrophils Eosinophils Basophils Lymphocytes Monocytes
- 77. These have granules of histamine that attract cause inflammation and attract other WBCs to injuries: Neutrophils Eosinophils Basophils Lymphocytes Monocytes
- 78. These play a role in immunity in the lymphoid tissue: Neutrophils Eosinophils Basophils Lymphocytes Monocytes
- 79. These play a role in immunity in the lymphoid tissue: Neutrophils Eosinophils Basophils Lymphocytes Monocytes
- 80. These play a role in immunity in the lymphoid tissue: Neutrophils Eosinophils Basophils Lymphocytes Monocytes
- 81. Hematopoietic stem cells give rise to _____, which differentiate into granulocytes and _____, which give rise to agranulocytes. Lymphoid stem cells, myeloid stem cells Myeloblasts, monoblasts Monocytes and promonocytes Myeloid stem cells, lymphoid stem cells
- 82. Hematopoietic stem cells give rise to _____, which differentiate into granulocytes and _____, which give rise to agranulocytes. Lymphoid stem cells, myeloid stem cells Myeloblasts, monoblasts Monocytes and promonocytes Myeloid stem cells, lymphoid stem cells
- 83. This is a group of cancerous conditions involving overproduction of abnormal white blood cells: Leukopenia Leukemia Acute leukemia Chronic leukemia Infectious mononucleosis
- 84. This is a group of cancerous conditions involving overproduction of abnormal white blood cells: Leukopenia Leukemia Acute leukemia Chronic leukemia Infectious mononucleosis
- 85. In this condition, the unspecialized, proliferating WBCs derive from stem cells: Leukopenia Leukemia Acute leukemia Chronic leukemia Infectious mononucleosis
- 86. In this condition, the unspecialized, proliferating WBCs derive from stem cells: Leukopenia Leukemia Acute leukemia Chronic leukemia Infectious mononucleosis
- 87. This is an abnormally low WBC count due to drugs such as chemotherapy: Leukopenia Leukemia Acute leukemia Chronic leukemia Infectious mononucleosis
- 88. This is an abnormally low WBC count due to drugs such as chemotherapy: Leukopenia Leukemia Acute leukemia Chronic leukemia Infectious mononucleosis
- 89. In this group of conditions, immature WBCs proliferate out of control, filling red bone marrow and hampering production of RBCs and other cells: Leukopenia Leukemia Acute leukemia Chronic leukemia Infectious mononucleosis
- 90. In this group of conditions, immature WBCs proliferate out of control, filling red bone marrow and hampering production of RBCs and other cells: Leukopenia Leukemia Acute leukemia Chronic leukemia Infectious mononucleosis
- 91. In this condition, the abnormal, proliferating WBCs derive from committed cells: Leukopenia Leukemia Acute leukemia Chronic leukemia Infectious mononucleosis
- 92. In this condition, the abnormal, proliferating WBCs derive from committed cells: Leukopenia Leukemia Acute leukemia Chronic leukemia Infectious mononucleosis
- 93. This illness caused by the Epstein-Barr virus causes excessive numbers of large, atypical lymphocytes:: Leukopenia Leukemia Acute leukemia Chronic leukemia Infectious mononucleosis
- 94. This illness caused by the Epstein-Barr virus causes excessive numbers of large, atypical lymphocytes:: Leukopenia Leukemia Acute leukemia Chronic leukemia Infectious mononucleosis
- 95. Repeated mitosis makes ______ huge, multinucleate cells, which send cytoplasmic extensions through the walls of the ______, and fragments to release _____ into the blood. Platelets, capillaries, megakaryocytes Leukocytes, sinusoid, platelets Megakaryocytes, sinusoid, platelets Leukocytes, capillaries, platelets
- 96. Repeated mitosis makes ______ huge, multinucleate cells, which send cytoplasmic extensions through the walls of the ______, and fragments to release _____ into the blood. Platelets, capillaries, megakaryocytes Leukocytes, sinusoid, platelets Megakaryocytes, sinusoid, platelets Leukocytes, capillaries, platelets
- 97. Put the steps of hemostasis in the correct order: Platelet plug formation, vascular spasm, coagulation Coagulation, vascular spasm, platelet plug formation Vascular spasm. Platelet plug formation, coagulation
- 98. Put the steps of hemostasis in the correct order: Platelet plug formation, vascular spasm, coagulation Coagulation, vascular spasm, platelet plug formation Vascular spasm. Platelet plug formation, coagulation
- 99. Which statements are true about vascular spasm? A strongly contracted artery can reduce blood flow for up to half an hour Vascular spasm can be triggered by injury to vascular smooth muscle Chemicals released by endothelial cells and platelets can trigger vascular spasms Reflexes initiated by pain receptors can trigger vascular spasms
- 100. Which statements are true about vascular spasm? A strongly contracted artery can reduce blood flow for up to half an hour Vascular spasm can be triggered by injury to vascular smooth muscle Chemicals released by endothelial cells and platelets can trigger vascular spasms Reflexes initiated by pain receptors can trigger vascular spasms
- 101. Which statements about platelet plug formation are true? Platelets adhere to the collagen fibers of connective tissue beneath injured endothelium Platelets release the aggregating agents ADP, serotonin and thromboxane Aggregation is a positive feedback cycle Aggregation is a negative feedback cycle
- 102. Which statements about platelet plug formation are true? Platelets adhere to the collagen fibers of connective tissue beneath injured endothelium Platelets release the aggregating agents ADP, serotonin and thromboxane Aggregation is a positive feedback cycle Aggregation is a negative feedback cycle
- 103. Which statements about coagulation are true? Clotting factors are chemicals released by platelets Clotting reinforces the platelet plug with fibrin threads Clotting factors circulate in the blood in inactive form Clotting factors are activated simultaneously by chemical signals from platelets
- 104. Which statements about coagulation are true? Clotting factors are chemicals released by platelets Clotting factors are plasma proteins synthesized by the liver Clotting reinforces the platelet plug with fibrin threads Clotting factors circulate in the blood in inactive form Clotting factors are activated simultaneously by chemical signals from platelets Each clotting factor activates the next clotting factor
- 105. Injury to the body triggers the ___ and ____ pathways, in a test tube, only the ___ pathway is triggered: Extrinsic & intrinsic, extrinsic Extrinsic & intrinsic, intrinsic
- 106. Injury to the body triggers the ___ and ____ pathways, in a test tube, only the ___ pathway is triggered: Extrinsic & intrinsic, extrinsic Extrinsic & intrinsic, intrinsic
- 107. The intrinsic pathway is so named because: It only occurs inside the body The necessary clotting factors are present within the blood Both of these
- 108. The intrinsic pathway is so named because: It only occurs inside the body The necessary clotting factors are present within the blood Both of these
- 109. The extrinsic pathway is so named because: It depends upon a tissue factor found outside the blood It only happens when clotting is triggered outside the body
- 110. The extrinsic pathway is so named because: It depends upon a tissue factor found outside the blood It only happens when clotting is triggered outside the body
- 111. This process involves actin-myosin contractions within platelets to compress the plug and draw edges of the blood vessel closer together: Anticoagulant Clot retraction Platelet derived growth factor Fibrinolysis Plasmin Plasminogen
- 112. This process involves actin-myosin contractions within platelets to compress the plug and draw edges of the blood vessel closer together: Anticoagulant Clot retraction Platelet derived growth factor Fibrinolysis Plasmin Plasminogen
- 113. These are factors that inhibit clotting: Anticoagulant Clot retraction Platelet derived growth factor Fibrinolysis Plasmin Plasminogen
- 114. These are factors that inhibit clotting: Anticoagulant Clot retraction Platelet derived growth factor Fibrinolysis Plasmin Plasminogen
- 115. Released by platelets, this stimulates fibroblasts and smooth muscle cells to divide: Anticoagulant Clot retraction Platelet derived growth factor Fibrinolysis Plasmin Plasminogen
- 116. Released by platelets, this stimulates fibroblasts and smooth muscle cells to divide: Anticoagulant Clot retraction Platelet derived growth factor Fibrinolysis Plasmin Plasminogen
- 117. This plasma protein is incorporated into clots, where it releases a natural clot buster when activated: Anticoagulant Clot retraction Platelet derived growth factor Fibrinolysis Plasmin Plasminogen
- 118. This plasma protein is incorporated into clots, where it releases a natural clot buster when activated: Anticoagulant Clot retraction Platelet derived growth factor Fibrinolysis Plasmin Plasminogen
- 119. This clot-digesting enzyme is released when endothelial cells secrete tPA in response to the presence of a clot: Anticoagulant Clot retraction Platelet derived growth factor Fibrinolysis Plasmin Plasminogen
- 120. This clot-digesting enzyme is released when endothelial cells secrete tPA in response to the presence of a clot: Anticoagulant Clot retraction Platelet derived growth factor Fibrinolysis Plasmin Plasminogen
- 121. This process removes unnecessary clots to prevent occlusion of blood vessels: Anticoagulant Clot retraction Platelet derived growth factor Fibrinolysis Plasmin Plasminogen
- 122. This process removes unnecessary clots to prevent occlusion of blood vessels: Anticoagulant Clot retraction Platelet derived growth factor Fibrinolysis Plasmin Plasminogen
- 123. A stationary clot in an unbroken vessel is a _____, when it breaks free it becomes a ____. Thrombus, embolus Embolus, thrombus
- 124. A stationary clot in an unbroken vessel is a _____, when it breaks free it becomes a ____. Thrombus, embolus Embolus, thrombus
- 125. This complication of pregnancy, septicemia, or incomplete blood transfusion causes widespread clotting and severe bleeding: Hemophilia Thrombocytopenia Impaired liver function Disseminated intravascular coagulation
- 126. This complication of pregnancy, septicemia, or incomplete blood transfusion causes widespread clotting and severe bleeding: Hemophilia Thrombocytopenia Impaired liver function Disseminated intravascular coagulation
- 127. This is a collection of bleeding disorders due to the hereditary loss of certain clotting factors: Hemophilia Thrombocytopenia Impaired liver function Disseminated intravascular coagulation
- 128. This is a collection of bleeding disorders due to the hereditary loss of certain clotting factors: Hemophilia Thrombocytopenia Impaired liver function Disseminated intravascular coagulation
- 129. Disease or lack of vitamin K leads to inability to produce or deficiency of clotting factors: Hemophilia Thrombocytopenia Impaired liver function Disseminated intravascular coagulation
- 130. Disease or lack of vitamin K leads to inability to produce or deficiency of clotting factors: Hemophilia Thrombocytopenia Impaired liver function Disseminated intravascular coagulation
- 131. In this condition a deficiency of platelets causes internal bleeding during normal movements: Hemophilia Thrombocytopenia Impaired liver function Disseminated intravascular coagulation
- 132. In this condition a deficiency of platelets causes internal bleeding during normal movements: Hemophilia Thrombocytopenia Impaired liver function Disseminated intravascular coagulation
- 133. These agglutinogens do not cause immediate antibody reaction during a transfusion, but do cause a reaction upon the second exposure: Antigens Agglutinogens ABO blood groups Agglutinins Rh factor
- 134. These agglutinogens do not cause immediate antibody reaction during a transfusion, but do cause a reaction upon the second exposure: Antigens Agglutinogens ABO blood groups Agglutinins Rh factor
- 135. These glycoprotein/glycolipid markers on cell membranes identify cells as self or foreign: Antigens Agglutinogens ABO blood groups Agglutinins Rh factor
- 136. These glycoprotein/glycolipid markers on cell membranes identify cells as self or foreign: Antigens Agglutinogens ABO blood groups Agglutinins Rh factor
- 137. These categories are based on the absence or presence of A and B agglutinogens: Antigens Agglutinogens ABO blood groups Agglutinins Rh factor
- 138. These categories are based on the absence or presence of A and B agglutinogens: Antigens Agglutinogens ABO blood groups Agglutinins Rh factor
- 139. Antigens on RBCs that promote clotting in other blood types: Antigens Agglutinogens ABO blood groups Agglutinins Rh factor
- 140. Antigens on RBCs that promote clotting in other blood types: Antigens Agglutinogens ABO blood groups Agglutinins Rh factor
- 141. These are preformed antibodies on RBCs that act against agglutinogens not carried on the individual’s RBCs: Antigens Agglutinogens ABO blood groups Agglutinins Rh factor
- 142. These are preformed antibodies on RBCs that act against agglutinogens not carried on the individual’s RBCs: Antigens Agglutinogens ABO blood groups Agglutinins Rh factor
- 143. This ABO blood type is the universal recipient: AB A B O
- 144. This ABO blood type is the universal recipient: AB A B O
- 145. This ABO blood type is the universal donor: AB A B O
- 146. This ABO blood type is the universal donor: AB A B O
- 147. This tests to see if the donor’s and recipient’s blood will cause agglutination during transfusion: Hemolytic disease of the newborn Transfusion reaction Autologous transfusion Cross matching
- 148. This tests to see if the donor’s and recipient’s blood will cause agglutination during transfusion: Hemolytic disease of the newborn Transfusion reaction Autologous transfusion Cross matching
- 149. During a second pregnancy, an Rh- mother may produce antibodies that attack the fetus’ RBCs: Hemolytic disease of the newborn Transfusion reaction Autologous transfusion Cross matching
- 150. During a second pregnancy, an Rh- mother may produce antibodies that attack the fetus’ RBCs: Hemolytic disease of the newborn Transfusion reaction Autologous transfusion Cross matching
- 151. Agglutination leads to RBC rupture, and hemoglobin in the bloodstream can lead to kidney shutdown: Hemolytic disease of the newborn Transfusion reaction Autologous transfusion Cross matching
- 152. Agglutination leads to RBC rupture, and hemoglobin in the bloodstream can lead to kidney shutdown: Hemolytic disease of the newborn Transfusion reaction Autologous transfusion Cross matching
- 153. A patient’s blood is pre-donated and banked prior to operations: Hemolytic disease of the newborn Transfusion reaction Autologous transfusion Cross matching
- 154. A patient’s blood is pre-donated and banked prior to operations: Hemolytic disease of the newborn Transfusion reaction Autologous transfusion Cross matching