major disease of guava &pomegranate and their management and his life cycle.

2. KOTWA AZAMGARH
SUBJECT – DISEASES OF FIELD AND HORTICULTURAL
CROPS AND THEIR MANAGEMENT - I
 ABHISHEK CHAUDHARY -ID.NO. A-11447/20
 ABHISHEK KUMAR GUPTA -ID.NO. A-11448/20
 ABHISHEK KUMAR PAL –ID.NO. A-11449/20
 ABHISHEK TRIPATHI -ID.NO. A-11450/20
 AJEET KUMAR -ID .NO. A-11451/20
 AJEET NISHAD -ID.NO. A-11452/20
PRESENTED TO
DR. VINIT PRATAP SINGH

3. 
1 •Wilt
2 •Anthracnose
3 •Fruit rot
DISEASES OF GUAVA

4. 
Fusarium oxysporum f.sp. psidii
GUAVA WILT

5. 
• Occurs during rainy season
• There are two types of wilts -
1. Slow wilt [1-5 month]
2. Rapid wilt [ within 15 days]
SYMPTOMS

6. 
CONTINUED
 The affected plants show
yellowing with slight leaf
curling at the terminal
branches, become reddish at
the later stage and
subsequently premature
shedding of leaves takes
place.
 Twigs become bare and fail
to bring new leaves or
flowers and eventually dry
up.
 During rainy season

7. 
 Fruits of all the affected branches
remain underdeveloped, become
hard, black and stony.
 The entire plant becomes defoliated
and dies.
 A few plants also show partial wilt.
 The finer roots show black streaks
which become prominent on
removing the bark.
CONTINUED

8. 
 The roots also show rotting at the
basal region and the bark is easily
detachable from the cortex.
CONTINUED

9. 
Mycelium- Septate, branched, light pink.
 Conidia-
1- Macro conidia – boat shaped, hyaline,
long, septate, pointed at tip.
2- Micro conidia - small, curved, unicellular
Conidiophore – simple, slender, short,
produced in sporodochium.
 Chlamydospore – spherical, thick walled
Sporodochia present.
ETIOLOGY

10. 
CONTINUED
Micro conidia
Macro conidia
Clamydospore
Sporodochium
Colony

11. 
Infection in
roots
Symptom
development
Production
of conidia
Secondary spread
through Irrigation
water, inter culture
operation
Conidia and
mycelium
survive in plant
debris
DISEASE CYCLE
Secondary cycle

12. 
 High rainfall during august/september.
 Stagnation of water in guava field for long
duration.
 Maximum and minimum temperature ranges
23-32 ℃ with 76% relative humidity
are conducive.
 Lack of timely application of suitable control
measures.
 Older plants of 3 years of age more susceptible.
EPIDEMIOLOGY

13. 
 Proper sanitation of orchard.
 Wilted plant should be uprooted, burnt and a trench
of 1.0-1.5 m should be dug around the tree trunk.
 Treats the pits with Formalin and cover the pit for 3
days and then transplant the seedling after two
weeks.
 While transplanting seedlings avoid damage to the
roots.
 Maintain proper tree vigour by timely and adequate
manuring, inter-culture and irrigation.
MANAGEMENT

14. 
 Intercropping with turmeric or marigold or Aonla.
 Soil solarization with transparent polythene sheet
during summer months.
 Application of oil cakes like, neem cake, mahua
cake, kusum cake supplemented with urea.
 Apply 6kg neem cake + 2kg gypsum per plant.
 Judicious amendments of N and Zn.
 Resistant variety: Apple guava.
 Chinese guava (P. friedshthalianum) and Phillippine
guava are recommended as resistant root stocks.
CONTINUED

15. 
Colletotrichum gloeosporioides
ANTHRACNOSE

16. 
SYMPTOMS
 Symptoms of this disease
are observed on mature
fruits on the tree.
 The characteristic symptoms
consist of sunken, dark
colored, necrotic lesions.
Under humid conditions,
the necrotic lesions become
covered with pinkish spore
masses.
 As the disease progresses,
the small sunken lesions
coalesce to form large
necrotic patches affecting
the flesh of the fruit.

17. 
 Mycelium : Septate, dark.
 The mycelium of the fungus is localized in the spot.
 Acervuli with setae arise through epidermis.
 Conidia are hyaline, single celled, vacuolate and
falcate in shape.
ETIOLOGY

18. 
DISEASE CYCLE
Infection through stomata
Symptom
development
Production of conidia
Secondary spread
through irrigation
water & wind
Infection in weed or
collateral host
Pathogen survive
in seed, soil, weed
host as conidia &
mycelium
Secondary cycle

19. 
 The pathogen survives on seed and plant debris.
 Disease spreads in the field through air-borne
conidia.
 The disease is more sever in cool and wet seasons.
 Continuous rain.
 Temperature of 28-30˚C.
 High humidity.
EPIDEMIOLOGY

20. 
 Spray Mancozeb 0.25%.
 Foliar spray of Propiconazole (0.2%) is very
effective.
 Zn, Mn, and B spray.
 Soil amendments with organic manures.
 Sanitation of orchard.
MANAGEMENT

21. 
1
•BACTERIAL BLIGHT
2
•FRUIT ROT
DISEASES OF
POMEGRANATE

22. 
Xanthomonas axonopodies pv. punicae
BACTERIAL BLIGHT

23. 
SYMPTOMS
 Appearance of one to
several small water
soaked lesion in leaves.
 Lesions become dark
colored irregular spots
with yellow halo
resulting in premature
defoliation under
severe cases.

24. 
CONTINUED
 The pathogen also infects
stem and branches
causing girdling and
cracking symptoms.
 Spots on fruits were dark
brown irregular slightly
raised with oily
appearance, which split
open with L-shaped
cracks under severe cases.

25. 
 The bacterium is aerobic, gram negative, non spore
forming, rod with size ranging from 1-2 x 0.8-1.0
micro mt.
 Monotrichous polar flagellum.
 Bacterial colonies are circular, convex with entire
margins, whitish yellow to straw yellow colored and
opaque.
ETIOLOGY

26. DISEASE CYCLE
Infection through
Wound or
Stomata
Symptom
development
Reproduction
and
multiplication
Bacteria ooze
out from
infected area
Secondary spread through
Irrigation water, wind
drawn rain, rain splashes
Infection in seeds,
weed or collateral
host
Pathogen survive
in crop debris,
seed, weed host
Secondary cycle

27. 
 Severe during August-November.
 Intermittent rains.
 High humidity and temp ranging from 20 to 27°C.
 Monocropping.
EPIDEMIOLOGY

28. 
 Sanitation.
 Disease free planting material.
 Regular pruning.
 Crop rotation.
 After each harvest, spray ethrel to cause leaf
defoliation, collect fallen leaves and destroy.
 Spray Arka microbial consortium @5%.
 Spray copper oxy chloride 0.1% and Streptocycline
500 PPM.
MANAGEMENT

29. 