The document discusses the different types of shock: hypovolemic, cardiogenic, circulatory (septic, neurogenic, anaphylactic), and endocrine shock. It provides details on the causes, pathophysiology, clinical manifestations, medical management, and nursing management of each type of shock. The primary types covered are hypovolemic, cardiogenic, septic, neurogenic, and anaphylactic shock.

1. TYPES
OF SHOCK
MS. SAHELI CHAKRABORTY
2ND YEAR MSC NURSING
RINER, BANGALORE

2. HYPOVOLEMIC
SHOCK
CARDIOGENIC
SHOCK
CIRCULATORY
SHOCK
SEPTIC
SHOCK
NEUROGENIC
SHOCK
ANAPHYLACTIC
SHOCK
TYPES
OF
SHOCK

3. 1. HYPOVOLEMIC SHOCK :-
 This is the most common type of shock and based
on the insufficient circulatory volume.
 Primary cause is loss of fluid from circulation from
either an internal or external source.
 Hypovolemic shock occurs when there is a
reduction in intravascular volume by 15% to 30%
which represents a loss of 750 ml to 1500 ml of
blood in a 70 kg person.

4. RISK FACTORS FOR HYPOVOLEMIC SHOCK
1. EXTERNAL :
 Trauma
 Surgery
 Vomiting
 Diarrhoea
 Diuretics
 Diabetes Insipidus
 External bleeding
2. INTERNAL:
 Haemorrhage
 Burns
 Ascites
 Peritonitis
 Dehydration
 Internal bleeding

5. PATHOPHYSIOLOGY OF HYPOVOLEMIC
SHOCK:
Due to etiological factor
Loss of body fluids and bloods
Decrease cardiac output
Hypo perfusion
Compensatory mechanism

6. Norepinephrine and Renin angiotensin and
Epinephrine aldosterone stimulation
Increase heart rate ADH release.
and vascular resistance
Intracellular fluid shift
to intravascular space
Increase blood volume
Increase cardiac output

7. Compensatory mechanism fails
Decrease cardiac output
Decrease blood pressure
Decrease perfusion to the vital organs
Multisystem organ failure

8. MEDICAL MANAGEMENT
1. Treatment of the underlying cause
2. Fluid and blood replacement
 Crystalloids- 0.9% sodium chloride
 Ringer lactate solution
 Hypertonic saline
 Colloids (Albumin, Dextran)
3. Redistribution of fluid
4. Pharmacologic therapy
(Vasoactive agents, Insulin therapy, Desmopressin,
Antidiarrhoeal, Antiemetics).

9. NURSING MANAGEMENT
 Primary prevention of shock is an essential focus of
nursing care.
1. Administering blood and fluids safety:
 Administration of blood transfusion.
 Blood specimens, cross matching of the blood should
be done.
 Patient should be monitored for cardiovascular
overload
 Hemodynamic pressure, vital signs, arterial blood
gases, serum lactate levels, hematocrit and
hemoglobin level need to be monitored.

10.  Maintain temperature
 Observing jugular venous pressure for distension
 The nurse must monitor cardiac and respiratory
status
2. Implementing other measures
 Oxygen is administered.
 Patient may be irritated and apprehensive so
frequent explanations about the patient’s condition
may reduce some of the patient’s fear and anxiety.

11. 2. CARDIOGENIC SHOCK
 Cardiogenic shock occurs when the heart’s ability to
contract and to pump blood is impaired and the
supply of oxygen is inadequate for the heart and
tissues.
 Coronary cardiogenic shock is more common than the
non coronary cardiogenic shock.
 Non coronary causes of cardiogenic shock are the
conditions that stress the myocardium such as severe
hypoxemia, acidosis, hypoglycaemia, hypocalcaemia,
tension pneumothorax, cardiomyopathies, valvular
damage, cardiac temponade, dysrhythmias.

12. CLINICAL MANIFESTATION:
 Distented jugular vein due to increased jugular venous
pressure.
 Absent pulse due to tachyarrhythmia
 Anginal pain
 Dysrhythmias.
 Complain of fatigue
 Express feelings of doom
 Signs of hemodynamic instability.

13. PATHOPHYSIOLOGY
1. Myocardial infarction, arrhythmias, congestive heart
failure, cardiac myopathy, cardiac valve problem
2. Impaired heart’s ability to contract and pump blood.
Decrease stroke volume Increase pulmonary
pressure
Decrease cardiac output
pulmonary edema
Decrease oxygenation to the tissues.

14. Hypo-perfusion tissues.
Impaired cellular metabolism.

15. MEDICAL MANAGEMENT
The goals of medical management in cardiogenic shock
are to limit further myocardial damage and preserve
the healthy myocardium and to improve the cardiac
function.
1. Correction of underlying causes.
2. Initiation of first line treatment.
 Oxygenation.
 Pain control.
 Hemodynamic monitoring
 Laboratory marker monitoring.
 Fluid therapy.

16.  Pharmacologic treatment :-
 Dobutamine.
 Nitroglycerine.
 Dopamine.
 Antiarrhythmic medications.
 Other vasoactive medication.
 Mechanical assistive devices.

17. NURSING MANAGEMENT
1. Preventing cardiogenic shock
2. Monitoring hemodynamic status.
3. Administering medications and intravenous fluids.
4. Maintaining intra-aortic balloon counter pulsation.
5. Enhancing safety and comfort.

18. 3. CIRCULATORY SHOCK
 Circulatory shock occurs when blood volume pooled
in peripheral blood vessels and results in hypovolemia
which leads to inadequate tissue perfusion.
 Contractility of the heart helps the blood to return to
the heart. And the vascular tone is determined by
central regulatory mechanism (in BP regulation) and
local regulatory mechanism (in tissue demands for
oxygen and nutrient)
 Thereby circulatory shock can be caused either by a
loss of sympathetic tone or by release of biochemical
mediators from cells.

19. PATHOPHYSIOLOGY :-
1. Precipitating event
2. Vasodilation
3. Activation of inflammatory response
4. Abnormal distribution of blood volume
5. Decreased venous return
6. Decreased cardiac output
7. Decreased tissue perfusion

20. CLASSIFICATION OF CIRCULATORY
SHOCK:
1. Septic shock
2. Neurogenic shock
3. Anaphylactic shock

21. 4. SEPTIC SHOCK
 The most common type of circulatory shock, is caused
by widespread of infection.
 The incidence is more in ICU and is increasing day by
day.
 The most common cause of death.
 The incidence can be reduced by using strict aseptic
technique, thorough hand hygiene techniques.
 Interventions include prevention of central line
infection, early debriding of wounds to remove the
necrotic tissues, carrying out standard precaution,
adhering to infection control practices, prompt
cleaning and maintaining of equipment.

22. PATHOPHYSIOLOGY
1. Severe localized infection of gram negative bacili (E.
Coli, Klebsella)
2. Septicemia (invasion of bacteria into the blood stream)
3. Inflammatory response
4. Release of endo-toxin into circulation
5. Immune system releases histamine and other chemical
mediators.
Massive vasodilation Increase capillary permeability
6. Severe broncho constriction

23. 7. Decrease oxygen supply to the tissues
8. Decrease tissue perfusion
9. Shock.

24. MEDICAL MANAGEMENT
1. Fluid replacement therapy
2. Pharmacolgic therapy
 Broad spectrum antibiotic
 Drotrecogin alfa (acts as an anti-inflammatory
cytokine, it stimulates fibrinolysis, restoring balance in
the coagulation –anticoagulation homeostatic process
of the body’s inflammatory response to injury and
infection
3. Nutritional therapy ( should start first 24 hours after
ICU admission)

25. NURSING MANAGEMENT :-
 All invasive procedure must be carried out with
aseptic technique.
 IV lines, arterial and venous puncture sites , surgical
incision, traumatic wounds, urinary catheter and
pressure ulcers must be monitored for signs of
infection.
 Patients with elderly and immunosuppressive,
extensive trauma, burns, or diabetes should be given
most attention.
 Elevated temperature is common in septic shock and
increase the metabolic rate so it should not be treated
unless it reaches to the dangerous level.

26.  The nurse administers prescribed IV fluids and
medications including antibiotic agents and
vasoactive mediators to restore vascular volume.
 Blood levels of BUN, creatinine, WBC,
hemoglobin, hematocrit, platelet levels, coagulation
studies should be monitored.
 Fluid intake and output, nutritional status, daily
weights should be checked.
 Close monitoring of the serum albumin and pre-
albumin levels help determine the patients protein
requirements.

27. 5. NEUROGENIC SHOCK
 In neurogenic shock vasodilation occurs as a result of a
loss of balance between parasympathetic and
sympathetic stimulation.
 Sympathetic stimulation causes vascular smooth muscle
to constrict, and parasympathetic stimulation causes
vascular smooth muscle to relax or dilate.
 In neurogenic shock the sympathetic system not able to
respond to the body stressors. Therefore neurogenic
shock are signs of parasympathetic stimulation.
 Parasympathetic stimulation causes vasodilation that
results in hypovolemic state and gradually leads to
hypotension and shock.

28. ETIOLOGY:
 Spinal cord injury
 Spinal anesthesia
 Nervous system damage.
 Depressant action of medication

29. PATHOPHYSIOLOGY
1. Spinal cord injury , anaesthesia.
2. Loss of autonomic nervous system and motor
function below the level of injury
3. Loss of sympathetic control
4. Increase sympathetic tone
5. Arterial of venous pooling
6. Dilatation of blood vessels
7. Hypotension
8. Warm, dry, flushed skin and bradycardia.
9. Decrease tissue perfusion to the vital organ
10. Multisystem organ failure.

30. MANAGEMENT :-
 Elevate the head of the bed at least 30 degree angle
when the patient receives spinal or epidural
anaesthesia. Because it helps in the prevention of the
spread of anaesthetic agent up the spinal cord.
 Carefully immobilize the patient in case of patient
with spinal cord injury.
 Supporting of cardiovascular and neurologic function.
 Apply anti-embolism stockings.
 Elevate the foot end to prevent the venous pooling of
the blood in the legs because it may increase the risk
of thrombus formation.

31.  The nurse must monitor the lower extremity pain,
redness, tenderness and warmth.
 Patient should be evaluated for deep vein thrombosis
by assessing the calf muscle pain.
 Administer heparin or low molecular weight heparin
(lovenox) as prescribed.
 Application of anti-embolism stockings or use of
pneumatic compression of the legs may prevent
thrombus formation.
 Passive range of motion of the immobile extremities
helps promote circulation.
 The nurse must monitor the patient for signs of
internal bleeding that could lead to hypovolemic
shock.

32. 6. ANAPHYLACTIC SHOCK
 Anaphylactic shock occurs rapidly and is life
threatening.
 Anaphylactic shock occurs in patients who has
already exposed to an antigen and who have
developed antibodies to it.
 Caused by an severe anaphylactic reaction to an
allergen, antigen, drug, foreign protein causing the
release of histamine which causes vasodilation
leading to hypotension and increased capillary
permeability.

33. PATHOPHYSIOLOGY
1. Antigen re-exposure.
2. Hypersensitivity antibody response
3. Activation of mast cells
4. Release of vasoactive substances such as
bradykinin, histamine.
5. Arterial vasodilation
6. Increase capillary permeability
7. Severe bronchospasm
8. Decrease oxygen supply and increase demand of
oxygen
9. Inadequate tissue perfusion
10. Shock and death.

34. MEDICAL MANAGEMENT
 Remove the causative antigen
 Emergency basic life support.
 Epinephrine is given for its vasoactive action
 Diphenydramine-to reverse the effects of histamine
thereby reducing the capillary permeability.
 Nebulized medication such as albutarol to reverse
the histamine induced bronchospasm.
 CPR in case of cardiac or respiratory arrest.
 Endotracheal intubation or tracheostomy to establish
the airway.

35. NURSING MANAGEMENT
 Check the vitals, respiration, BP and Mean arterial
pressure.
 The nurse must assess for previous reaction of the allergy
to medication, blood products, foods, contrast agents.
 Observe patient for allergic reaction while administering
the medication.
 The nurse must identify patients who are at risk for
anaphylactic reactions to contrast agents used for
diagnostic tests.
 This information need to be communicated to the
diagnostic testing site.
 After recovery from anaphylaxis the patient and family
require an explanation of the event.

36. 7. ENDOCRINE SHOCK BASED ON ENDOCRINE
DISTURBANCES
 Hypothyroidism in critically ill patient reduces
cardiac output, lead to hypotension and
respiratory insufficiency.
 Thyrotoxicosis may induce a reversible
cardiomyopathy.
 Acute adrenal insufficiency may results in shock
 Surgery and patients on corticosteroid therapy
may lead to shock in severe cases.