1) Shock is a serious medical condition where tissue perfusion is insufficient to meet oxygen and nutrient demands due to inadequate blood flow.
2) There are four main types of shock: hypovolemic, cardiogenic, obstructive, and distributive.
3) Signs of shock in general include rapid breathing, cold and clammy skin, a weak but rapid pulse, low blood pressure, and weakness throughout the body.
Shock is a life-threatening condition with a variety of underlying causes. It is characterized by inadequate tissue perfusion that, if untreated, results in cell death. The nurse caring for the patient with shock or at risk for shock must understand the underlying mechanisms of shock and recognize its subtle as well as more obvious signs. Rapid assessment and response are essential to the patient’s recovery.
Shock is defined as a condition where systemic blood pressure is inadequate to deliver oxygen and nutrients to vital organs and cells. There are several types of shock including hypovolemic, cardiogenic, and distributive shock such as septic or anaphylactic shock. Shock progresses through initial, compensatory, progressive, and refractory stages. In the compensatory stage, the body responds through neural and hormonal responses to increase heart rate, vasoconstriction, and release of hormones like epinephrine to try to maintain blood pressure and tissue perfusion. In later stages, tissues become damaged from lack of oxygen delivery which can lead to organ failure and death if not treated. Management of shock involves maintaining airway
Simple medical student presentation about distributive shock, type and pathophysiology of each septic shock, anaphylactic shock, neurogenic shock
including management, prognosis and disposition of patient..
brief info of type of inotropes and when to start.
This document discusses shock, including its definition, stages, types, signs and symptoms, and diagnostic evaluation. Shock is defined as inadequate tissue perfusion resulting from low blood pressure and cardiac output. It discusses the initial, compensatory, progressive and irreversible stages of shock. The main types covered are hypovolemic, septic, cardiogenic, neurogenic and anaphylactic shock. For each type the causes, pathophysiology, clinical manifestations are outlined. The diagnostic evaluation of shock involves medical history, physical exam, lab tests and imaging to identify the underlying cause and guide treatment.
Shock is defined as inadequate tissue perfusion and oxygenation. There are five main types of shock: hypovolemic, cardiogenic, obstructive, distributive, and endocrine. Shock causes a decrease in cardiac output, vasoconstriction, and activation of stress responses in an attempt to maintain perfusion to vital organs. If shock persists, cellular damage occurs due to hypoxia, acidosis, and organ dysfunction. Treatment focuses on restoring circulating volume and oxygen delivery through fluid resuscitation, vasopressors, and inotropes while addressing the underlying cause. Septic shock involves an inflammatory response to infection that can lead to multiple organ dysfunction syndrome if not promptly treated with antibiotics and circulatory
This document provides an overview of different types of shock, including hypovolemic, cardiogenic, septic, anaphylactic, and neurogenic shock. It describes the pathophysiology, clinical manifestations, diagnosis, and treatment principles for each type. Hypovolemic shock is the most common and results from reduced circulating volume due to causes like bleeding or dehydration. Septic shock involves systemic inflammation and vasodilation in response to infection. Treatment focuses on fluid resuscitation, vasopressor support, source control, and antibiotics depending on the shock etiology.
This document discusses different types of shock. It classifies shock into hypovolaemic, cardiogenic, obstructive, distributive, and endocrine shock. Hypovolaemic shock is due to reduced circulating volume. Cardiogenic shock is caused by heart failure. Obstructive shock involves mechanical obstruction of cardiac filling. Distributive shock involves abnormal blood vessel dilation leading to low blood pressure and organ perfusion issues. Endocrine shock combines features of other shock types and can be caused by thyroid or adrenal issues. The document provides examples and details on the pathophysiology of each shock type.
Shock is a life-threatening condition with a variety of underlying causes. It is characterized by inadequate tissue perfusion that, if untreated, results in cell death. The nurse caring for the patient with shock or at risk for shock must understand the underlying mechanisms of shock and recognize its subtle as well as more obvious signs. Rapid assessment and response are essential to the patient’s recovery.
Shock is defined as a condition where systemic blood pressure is inadequate to deliver oxygen and nutrients to vital organs and cells. There are several types of shock including hypovolemic, cardiogenic, and distributive shock such as septic or anaphylactic shock. Shock progresses through initial, compensatory, progressive, and refractory stages. In the compensatory stage, the body responds through neural and hormonal responses to increase heart rate, vasoconstriction, and release of hormones like epinephrine to try to maintain blood pressure and tissue perfusion. In later stages, tissues become damaged from lack of oxygen delivery which can lead to organ failure and death if not treated. Management of shock involves maintaining airway
Simple medical student presentation about distributive shock, type and pathophysiology of each septic shock, anaphylactic shock, neurogenic shock
including management, prognosis and disposition of patient..
brief info of type of inotropes and when to start.
This document discusses shock, including its definition, stages, types, signs and symptoms, and diagnostic evaluation. Shock is defined as inadequate tissue perfusion resulting from low blood pressure and cardiac output. It discusses the initial, compensatory, progressive and irreversible stages of shock. The main types covered are hypovolemic, septic, cardiogenic, neurogenic and anaphylactic shock. For each type the causes, pathophysiology, clinical manifestations are outlined. The diagnostic evaluation of shock involves medical history, physical exam, lab tests and imaging to identify the underlying cause and guide treatment.
Shock is defined as inadequate tissue perfusion and oxygenation. There are five main types of shock: hypovolemic, cardiogenic, obstructive, distributive, and endocrine. Shock causes a decrease in cardiac output, vasoconstriction, and activation of stress responses in an attempt to maintain perfusion to vital organs. If shock persists, cellular damage occurs due to hypoxia, acidosis, and organ dysfunction. Treatment focuses on restoring circulating volume and oxygen delivery through fluid resuscitation, vasopressors, and inotropes while addressing the underlying cause. Septic shock involves an inflammatory response to infection that can lead to multiple organ dysfunction syndrome if not promptly treated with antibiotics and circulatory
This document provides an overview of different types of shock, including hypovolemic, cardiogenic, septic, anaphylactic, and neurogenic shock. It describes the pathophysiology, clinical manifestations, diagnosis, and treatment principles for each type. Hypovolemic shock is the most common and results from reduced circulating volume due to causes like bleeding or dehydration. Septic shock involves systemic inflammation and vasodilation in response to infection. Treatment focuses on fluid resuscitation, vasopressor support, source control, and antibiotics depending on the shock etiology.
This document discusses different types of shock. It classifies shock into hypovolaemic, cardiogenic, obstructive, distributive, and endocrine shock. Hypovolaemic shock is due to reduced circulating volume. Cardiogenic shock is caused by heart failure. Obstructive shock involves mechanical obstruction of cardiac filling. Distributive shock involves abnormal blood vessel dilation leading to low blood pressure and organ perfusion issues. Endocrine shock combines features of other shock types and can be caused by thyroid or adrenal issues. The document provides examples and details on the pathophysiology of each shock type.
This document provides information on the pathophysiology of shock. It defines shock and describes the main types: primary/initial shock, secondary/true shock. It then discusses the mechanisms and stages of shock, including compensated/non-progressive shock, decompensated progressive shock, and decompensated/irreversible shock. The management of different types of shock such as hypovolaemic shock and septic shock is also summarized.
Shock is a life-threatening condition caused by inadequate oxygen delivery to tissues. It is a leading cause of death in children and can result from trauma, infection, dehydration, or heart failure. Early recognition of shock is key, as signs like altered mental status and abnormal perfusion may be present even when vital signs are normal. Aggressive fluid resuscitation is the primary treatment for shock in the prehospital setting, with 20mL/kg boluses of normal saline or lactated Ringer's administered as rapidly as possible. Ongoing assessment of perfusion parameters like capillary refill is essential to guide care and ensure reversal of shock.
This presentation gives information about shock and it's types.
The brief information in the presentation helps you to get basic knowledge about shock.
The easy language used in the presentation makes it easy to understand the concept clearly.
The types of shock are represented in a short manner for better understanding.
Approach to hypovolemic and septic shockAhmed Bahamid
This document discusses approaches to hypovolemic and septic shock in children. It defines shock and describes the pathophysiology, stages, and management. Shock results from inadequate oxygen delivery to tissues. Initially, compensatory mechanisms attempt to maintain blood pressure, but the condition can progress to decompensated then irreversible shock without treatment. Sepsis causes an inflammatory response that can lead to organ dysfunction if uncontrolled. Early intervention is important to improve outcomes from shock.
Shock is a life-threatening condition where the body's circulatory system fails and insufficient blood flows through the body. The document defines shock and discusses its various types including hypovolemic, cardiogenic, distributive, obstructive, and anaphylactic shock. It outlines the pathophysiology, stages, signs and symptoms, management including pharmacological interventions, and nursing care of patients experiencing shock. The summary provides an overview of what shock is, its main types, and its treatment and management.
Cardiogenic shock is a low cardiac output state resulting from inadequate tissue perfusion despite adequate left ventricular filling pressures. It is usually caused by acute myocardial infarction which accounts for about 80% of cases. Clinically, it is defined by sustained hypotension with signs of hypoperfusion and a systolic blood pressure less than 90 mmHg for at least 30 minutes or the need for vasopressor/inotropic support. The mortality rate for cardiogenic shock remains high at over 80% despite advances in management. Early diagnosis and aggressive treatment including revascularization, inotropic support, and mechanical circulatory support are aimed at improving outcomes.
This document summarizes different types of shock:
1. Hypovolemic, cardiogenic, obstructive, distributive, and endocrine shock are classified. Obstructive shock involves reduced cardiac filling from mechanical obstruction. Distributive shock features vasodilation and hypotension from conditions like sepsis.
2. Specific causes of different shock types are outlined, such as tension pneumothorax in obstructive shock and anaphylaxis in distributive shock.
3. Features of early and late septic shock are compared, showing progression from warm to cold shock with worsening end-organ dysfunction over time.
This document discusses shock, including its definition, causes, stages, classifications, signs, symptoms, and treatments. Shock is defined as a clinical state of circulatory collapse caused by a reduction in cardiac output or circulating blood volume, resulting in hypotension and impaired tissue perfusion. The stages of shock include compensated, uncompensated, and irreversible. The main types of shock covered are hypovolemic, cardiogenic, distributive (septic, anaphylactic, neurogenic), and obstructive. Treatment aims to restore adequate tissue perfusion through volume expansion and vasopressor support.
Shock is a life-threatening condition that occurs when the body is not getting enough blood flow. Lack of blood flow means the cells and organs do not get enough oxygen and nutrients to function properly. Many organs can be damaged as a result.
shock
defination: shock is defined as an acute clinical syndrome characterized by hypoperfusion and severe disfunction of vital organs.
types of shock:
hypovolaemic
cardiogenic
obstructive
distructive
neurogenic
anaphylactic
septic
pathophysiology
causes
treatment
clinical features
1. Septic shock is caused by infection which releases cytokines that damage microcirculation and cause vasodilation and capillary leakage, leading to tissue hypoxia and multiple organ failure. Early, aggressive treatment of infection along with cardiovascular and organ system support is needed to prevent high mortality rates.
2. Hypovolaemic shock results from decreased blood volume due to blood loss, fluid loss, or fluid shifts. It progresses from mild to severe as compensation fails, leading to cellular changes, metabolic acidosis, and potentially multiple organ failure without timely fluid resuscitation and hemostasis.
3. Cardiogenic shock stems from heart failure to pump adequately due to causes like myocardial infarction, arrhythmias
Shock is characterized by a systemic reduction in tissue perfusion resulting in decreased oxygen delivery. There are four main types of shock: hypovolemic, cardiogenic, obstructive, and distributive. The goals of resuscitation are to increase oxygen delivery and decrease demand. Treatment involves establishing IV access, fluid resuscitation, vasopressors, inotropes, antibiotics for infection, and treating the underlying cause. Endpoints of resuscitation include restoration of blood pressure, normalization of heart rate, urine output, lactate levels, and mental status.
This document summarizes the key aspects of shock. Shock occurs when there is inadequate oxygen delivery to tissues, which can lead to cellular damage and death if left untreated. The main causes of shock discussed are hypovolaemic, septic, and cardiogenic shock. Signs and symptoms of shock include low blood pressure, fast heart rate, pale skin, confusion, and low urine output. Management involves identifying the type and stage of shock to guide fluid resuscitation and drug therapy aimed at restoring adequate circulation and oxygen delivery to tissues.
This document defines shock and describes the different types of shock. It explains the pathology of shock as decreased blood volume leading to reduced cardiac output and blood pressure failure. It outlines the compensatory mechanisms the body employs in response to shock like increased adrenaline and fluid shifts. The clinical features of mild, moderate and severe shock are provided based on percentage of blood volume lost. Treatment focuses on resuscitation, bleeding control, fluid replacement and various drugs to support the heart rate, contractions and vascular tone.
Shock is characterized by the body's inability to meet metabolic demands. It can progress from compensated to decompensated stages. Septic shock involves a systemic inflammatory response to infection. Initial treatment of shock involves airway support, rapid fluid resuscitation, and cardiovascular support with inotropes if needed. Goals are to optimize perfusion and treat the underlying cause. Children may require earlier intubation and larger fluid volumes. Physical exam findings and endpoints guide management more than labs. Early antibiotics and source control are also important in septic shock.
1) Shock is a condition where the cardiovascular system fails to adequately perfuse tissues due to impaired cardiac pump function, circulatory issues, or low blood volume.
2) The main types of shock are hypovolemic (low blood volume), cardiogenic (impaired heart function), and distributive (blood vessel problems).
3) Hypovolemic shock results from internal or external fluid loss leading to decreased circulating volume and tissue perfusion. Cardiogenic shock occurs due to impaired left ventricular pumping ability despite normal blood volume.
The document discusses shock, including its pathophysiology, classification, severity, consequences, and resuscitation. It aims to provide medical students an understanding of shock. Shock is defined as a systemic state of low tissue perfusion inadequate for cellular respiration. Different types of shock are classified including hypovolemic, cardiogenic, obstructive, distributive, and endocrine shock. The severity of shock ranges from compensated to decompensated to multiple organ failure. Consequences include cellular damage, organ dysfunction, and death. Resuscitation focuses first on fluid therapy and addressing hypovolemia, with vasopressors and inotropes as adjuncts depending on the shock type.
This document discusses shock in pediatric patients. It defines shock and describes the different types including hypovolemic, distributive, cardiogenic, obstructive, and dissociative shock. It explains how oxygen delivery works and the factors that affect it. The goals of shock management are to increase oxygen delivery and decrease demand. Treatment involves optimizing preload, contractility, afterload, oxygen content and cardiac output. Initial management follows an ABC approach of securing the airway, providing adequate breathing and circulation. Fluid resuscitation is a key part of treatment, and choices between crystalloids and colloids are discussed. Inotropic support may be needed to optimize cardiac function in shock.
Approach to Shock for Seminar Jan.2024.pptxtowinb34
In this lecture you will be able to retrieve this number after you close this window until you have printed this confirmation page or made a record of the confirmation number before closing this window until you have printed
1. Shock is a serious medical condition where insufficient blood flow reaches the tissues due to problems with the heart, blood vessels, or blood loss.
2. Shock progresses through initial, compensatory, progressive, and refractory stages and is classified as hypovolemic, cardiogenic, distributive, or obstructive.
3. Treatment involves identifying the underlying cause, restoring blood volume, increasing cardiac output, treating infection, and removing obstructions. Nursing care focuses on monitoring vital signs, giving IV fluids and medications, managing airway and comfort measures.
This document provides information on the pathophysiology of shock. It defines shock and describes the main types: primary/initial shock, secondary/true shock. It then discusses the mechanisms and stages of shock, including compensated/non-progressive shock, decompensated progressive shock, and decompensated/irreversible shock. The management of different types of shock such as hypovolaemic shock and septic shock is also summarized.
Shock is a life-threatening condition caused by inadequate oxygen delivery to tissues. It is a leading cause of death in children and can result from trauma, infection, dehydration, or heart failure. Early recognition of shock is key, as signs like altered mental status and abnormal perfusion may be present even when vital signs are normal. Aggressive fluid resuscitation is the primary treatment for shock in the prehospital setting, with 20mL/kg boluses of normal saline or lactated Ringer's administered as rapidly as possible. Ongoing assessment of perfusion parameters like capillary refill is essential to guide care and ensure reversal of shock.
This presentation gives information about shock and it's types.
The brief information in the presentation helps you to get basic knowledge about shock.
The easy language used in the presentation makes it easy to understand the concept clearly.
The types of shock are represented in a short manner for better understanding.
Approach to hypovolemic and septic shockAhmed Bahamid
This document discusses approaches to hypovolemic and septic shock in children. It defines shock and describes the pathophysiology, stages, and management. Shock results from inadequate oxygen delivery to tissues. Initially, compensatory mechanisms attempt to maintain blood pressure, but the condition can progress to decompensated then irreversible shock without treatment. Sepsis causes an inflammatory response that can lead to organ dysfunction if uncontrolled. Early intervention is important to improve outcomes from shock.
Shock is a life-threatening condition where the body's circulatory system fails and insufficient blood flows through the body. The document defines shock and discusses its various types including hypovolemic, cardiogenic, distributive, obstructive, and anaphylactic shock. It outlines the pathophysiology, stages, signs and symptoms, management including pharmacological interventions, and nursing care of patients experiencing shock. The summary provides an overview of what shock is, its main types, and its treatment and management.
Cardiogenic shock is a low cardiac output state resulting from inadequate tissue perfusion despite adequate left ventricular filling pressures. It is usually caused by acute myocardial infarction which accounts for about 80% of cases. Clinically, it is defined by sustained hypotension with signs of hypoperfusion and a systolic blood pressure less than 90 mmHg for at least 30 minutes or the need for vasopressor/inotropic support. The mortality rate for cardiogenic shock remains high at over 80% despite advances in management. Early diagnosis and aggressive treatment including revascularization, inotropic support, and mechanical circulatory support are aimed at improving outcomes.
This document summarizes different types of shock:
1. Hypovolemic, cardiogenic, obstructive, distributive, and endocrine shock are classified. Obstructive shock involves reduced cardiac filling from mechanical obstruction. Distributive shock features vasodilation and hypotension from conditions like sepsis.
2. Specific causes of different shock types are outlined, such as tension pneumothorax in obstructive shock and anaphylaxis in distributive shock.
3. Features of early and late septic shock are compared, showing progression from warm to cold shock with worsening end-organ dysfunction over time.
This document discusses shock, including its definition, causes, stages, classifications, signs, symptoms, and treatments. Shock is defined as a clinical state of circulatory collapse caused by a reduction in cardiac output or circulating blood volume, resulting in hypotension and impaired tissue perfusion. The stages of shock include compensated, uncompensated, and irreversible. The main types of shock covered are hypovolemic, cardiogenic, distributive (septic, anaphylactic, neurogenic), and obstructive. Treatment aims to restore adequate tissue perfusion through volume expansion and vasopressor support.
Shock is a life-threatening condition that occurs when the body is not getting enough blood flow. Lack of blood flow means the cells and organs do not get enough oxygen and nutrients to function properly. Many organs can be damaged as a result.
shock
defination: shock is defined as an acute clinical syndrome characterized by hypoperfusion and severe disfunction of vital organs.
types of shock:
hypovolaemic
cardiogenic
obstructive
distructive
neurogenic
anaphylactic
septic
pathophysiology
causes
treatment
clinical features
1. Septic shock is caused by infection which releases cytokines that damage microcirculation and cause vasodilation and capillary leakage, leading to tissue hypoxia and multiple organ failure. Early, aggressive treatment of infection along with cardiovascular and organ system support is needed to prevent high mortality rates.
2. Hypovolaemic shock results from decreased blood volume due to blood loss, fluid loss, or fluid shifts. It progresses from mild to severe as compensation fails, leading to cellular changes, metabolic acidosis, and potentially multiple organ failure without timely fluid resuscitation and hemostasis.
3. Cardiogenic shock stems from heart failure to pump adequately due to causes like myocardial infarction, arrhythmias
Shock is characterized by a systemic reduction in tissue perfusion resulting in decreased oxygen delivery. There are four main types of shock: hypovolemic, cardiogenic, obstructive, and distributive. The goals of resuscitation are to increase oxygen delivery and decrease demand. Treatment involves establishing IV access, fluid resuscitation, vasopressors, inotropes, antibiotics for infection, and treating the underlying cause. Endpoints of resuscitation include restoration of blood pressure, normalization of heart rate, urine output, lactate levels, and mental status.
This document summarizes the key aspects of shock. Shock occurs when there is inadequate oxygen delivery to tissues, which can lead to cellular damage and death if left untreated. The main causes of shock discussed are hypovolaemic, septic, and cardiogenic shock. Signs and symptoms of shock include low blood pressure, fast heart rate, pale skin, confusion, and low urine output. Management involves identifying the type and stage of shock to guide fluid resuscitation and drug therapy aimed at restoring adequate circulation and oxygen delivery to tissues.
This document defines shock and describes the different types of shock. It explains the pathology of shock as decreased blood volume leading to reduced cardiac output and blood pressure failure. It outlines the compensatory mechanisms the body employs in response to shock like increased adrenaline and fluid shifts. The clinical features of mild, moderate and severe shock are provided based on percentage of blood volume lost. Treatment focuses on resuscitation, bleeding control, fluid replacement and various drugs to support the heart rate, contractions and vascular tone.
Shock is characterized by the body's inability to meet metabolic demands. It can progress from compensated to decompensated stages. Septic shock involves a systemic inflammatory response to infection. Initial treatment of shock involves airway support, rapid fluid resuscitation, and cardiovascular support with inotropes if needed. Goals are to optimize perfusion and treat the underlying cause. Children may require earlier intubation and larger fluid volumes. Physical exam findings and endpoints guide management more than labs. Early antibiotics and source control are also important in septic shock.
1) Shock is a condition where the cardiovascular system fails to adequately perfuse tissues due to impaired cardiac pump function, circulatory issues, or low blood volume.
2) The main types of shock are hypovolemic (low blood volume), cardiogenic (impaired heart function), and distributive (blood vessel problems).
3) Hypovolemic shock results from internal or external fluid loss leading to decreased circulating volume and tissue perfusion. Cardiogenic shock occurs due to impaired left ventricular pumping ability despite normal blood volume.
The document discusses shock, including its pathophysiology, classification, severity, consequences, and resuscitation. It aims to provide medical students an understanding of shock. Shock is defined as a systemic state of low tissue perfusion inadequate for cellular respiration. Different types of shock are classified including hypovolemic, cardiogenic, obstructive, distributive, and endocrine shock. The severity of shock ranges from compensated to decompensated to multiple organ failure. Consequences include cellular damage, organ dysfunction, and death. Resuscitation focuses first on fluid therapy and addressing hypovolemia, with vasopressors and inotropes as adjuncts depending on the shock type.
This document discusses shock in pediatric patients. It defines shock and describes the different types including hypovolemic, distributive, cardiogenic, obstructive, and dissociative shock. It explains how oxygen delivery works and the factors that affect it. The goals of shock management are to increase oxygen delivery and decrease demand. Treatment involves optimizing preload, contractility, afterload, oxygen content and cardiac output. Initial management follows an ABC approach of securing the airway, providing adequate breathing and circulation. Fluid resuscitation is a key part of treatment, and choices between crystalloids and colloids are discussed. Inotropic support may be needed to optimize cardiac function in shock.
Approach to Shock for Seminar Jan.2024.pptxtowinb34
In this lecture you will be able to retrieve this number after you close this window until you have printed this confirmation page or made a record of the confirmation number before closing this window until you have printed
1. Shock is a serious medical condition where insufficient blood flow reaches the tissues due to problems with the heart, blood vessels, or blood loss.
2. Shock progresses through initial, compensatory, progressive, and refractory stages and is classified as hypovolemic, cardiogenic, distributive, or obstructive.
3. Treatment involves identifying the underlying cause, restoring blood volume, increasing cardiac output, treating infection, and removing obstructions. Nursing care focuses on monitoring vital signs, giving IV fluids and medications, managing airway and comfort measures.
This document provides an overview of shock, including its definition, types, causes, pathophysiology, signs and symptoms, monitoring, and treatment. It defines shock as acute circulatory failure characterized by microcirculation dysfunction and inadequate blood flow to vital organs. The main types of shock discussed are hypovolemic, cardiogenic, septic, neurogenic, and anaphylactic shock. For each type, the causes, pathophysiology, signs, monitoring parameters, and general treatment approaches are outlined. The document emphasizes the importance of early recognition and monitoring in shock patients.
This document discusses shock, its causes, pathophysiology, classification, stages, signs and symptoms, diagnosis, treatment and nursing management. Shock is defined as a life-threatening condition characterized by inadequate tissue perfusion. It can result from reduced blood volume, heart malfunction, lung issues, or other causes. Shock progresses through compensatory, progressive and irreversible stages. Treatment involves restoring blood volume, increasing cardiac output, treating underlying causes, and supportive nursing care.
This document discusses pericardial diseases, focusing on pericarditis, pericardial effusion, and cardiac tamponade. It defines the pericardium and its functions. It describes the symptoms, signs, and diagnostic criteria for acute pericarditis. Causes of pericarditis include infections, autoimmune disorders, neoplasms, radiation, renal failure, and trauma. Treatment involves NSAIDs, colchicine, or steroids depending on severity and recurrence risk. Pericardial effusion and tamponade can develop as complications, requiring drainage procedures or surgery.
Hemodynamic derangements and shock can be classified in several ways. Shock is defined as inadequate tissue perfusion resulting in cellular oxygen debt. There are four main types of shock based on hemodynamic parameters: hemorrhagic, cardiogenic, distributive, and obstructive. Etiologically, shock can be caused by hemorrhage, cardiac issues like myocardial infarction, sepsis/systemic inflammatory response syndrome, or anaphylaxis due to peripheral vasodilation. If not treated, shock can progress to organ dysfunction and failure due to hypoxic injury. Management involves identifying the cause, restoring intravascular volume and blood pressure, and treating the underlying condition.
This document provides an overview of shock in children, including:
1. Definitions of shock and the pathophysiology involving reduced tissue perfusion and oxygen delivery.
2. The epidemiology and classifications of different shock types, including hypovolemic, distributive, cardiogenic, and obstructive shock.
3. Details on the causes, signs, symptoms, and stages of specific shock types like septic, hemorrhagic, and cardiogenic shock.
4. The goals of evaluating and managing shock in children, including rapid assessment of appearance, breathing, circulation, history, and physical exam findings.
Shock is a life-threatening condition where tissues do not receive adequate oxygen due to low blood flow. There are three stages of shock: compensatory, progressive, and refractory. Shock can be classified as hypovolemic, cardiogenic, neurogenic, anaphylactic, or septic depending on the cause. Treatment focuses on restoring adequate circulation through fluid resuscitation, vasopressors, or inotropes while also treating the underlying cause of shock.
This document provides information on shock, including its definition, physiology, pathophysiology, types, symptoms, signs, management, and treatment. It defines shock as inadequate perfusion leading to inadequate oxygen delivery to tissues. The stages of shock are described as initial, compensatory, progressive, and irreversible. Types of shock include cardiogenic, hypovolemic, neurogenic, septic, anaphylactic, and obstructive shock. Signs and symptoms result from cellular hypoperfusion and include restlessness, tachycardia, decreased consciousness, nausea, and decreased urine output. Management involves treating the underlying cause, giving oxygen, intravenous fluids, and vasopressors if needed. The goal of treatment is
1. Shock is defined as inadequate tissue perfusion to meet metabolic needs due to issues with cardiac performance, vascular performance, or cellular function.
2. The main types of shock are hypovolemic, cardiogenic, obstructive, and distributive. Clinical signs include low blood pressure, fast heart rate, pale skin, confusion and loss of consciousness.
3. Treatment of shock focuses on identifying the type, treating the underlying cause, restoring circulating volume with fluids, and supporting vital organ function with vasopressors or inotropes as needed. The goal is to restore adequate perfusion to prevent multiple organ dysfunction syndrome.
SHOCK in dentistry causes and its management20103308
Shock is defined as a state of circulatory failure where there is inadequate tissue perfusion resulting in lack of oxygen delivery. It can be caused by conditions such as hemorrhage, infection, heart failure, etc. The main signs are low blood pressure and heart rate abnormalities. Treatment focuses on restoring adequate circulation through fluid resuscitation, treating the underlying cause, and supporting vital organ function. Prompt management is important to prevent multiple organ failure and death.
This document discusses pericardial diseases, including pericarditis. Pericarditis is an inflammation of the pericardium that can be acute or chronic. Acute pericarditis is commonly caused by viral or bacterial infections, while chronic pericarditis can lead to constrictive pericarditis. The diagnosis of pericarditis is based on symptoms like chest pain and pericardial rub on examination. Electrocardiogram may show ST elevation and inflammatory markers are often elevated. Echocardiogram can detect pericardial effusions. Causes include infections, autoimmune diseases, tumors, and renal failure. Tuberculous pericarditis remains an important cause
This document discusses various cardiac emergencies including myocardial infarction, angina pectoris, congestive cardiac failure, sudden cardiac arrest, cardiac tamponade, and cardiogenic shock. It provides details on the causes, symptoms, diagnostic measures, and management of these conditions. Specifically, it describes angina pectoris as chest pain caused by partial blockage of the coronary arteries, acute myocardial infarction as damage to the heart muscle caused by a blockage of blood supply, and cardiac tamponade as a life-threatening condition where fluid builds up in the space between the heart muscle and the sac surrounding the heart. The document emphasizes the importance of early recognition, CPR, defibrillation and medications in managing cardiac arrest and
This document provides an overview of the principles of shock management. It defines shock and describes its causes, including hypovolemic, cardiogenic, obstructive, distributive, and endocrine shock. The pathophysiology of shock is explained at the cellular, microvascular, and systemic levels. The stages of shock - non-progressive, progressive decompensated, and decompensated - are outlined. Signs and symptoms of shock are provided. Finally, the document discusses the general management of shock, which aims to improve oxygen delivery and utilization to prevent organ injury through restoration of perfusion and supportive care.
This document provides an overview of several common critical medical conditions including respiratory failure, ARDS, acute MI, CHF, GI bleed, DKA, shock, and sepsis. It defines each condition and discusses signs and symptoms, causes, complications, treatments, and nursing interventions. Respiratory failure can result from ventilation-perfusion mismatching or intrapulmonary shunting. ARDS causes damage to the alveolar-capillary interface leading to pulmonary edema. Acute MI is caused by coronary artery obstruction from thrombus or plaque. CHF occurs when the heart cannot pump sufficient blood to meet metabolic needs.
shock is the state of insufficient blood flow to the tissues of the body .it contains introduction, definition, stages of shock, types of shock, diagnostic evaluation, prognosis ,prevention, care for each stage.
This document provides guidelines for the management of shock in children. It defines shock as a pathophysiological state characterized by inadequate tissue perfusion. The main types of shock are hypovolemic, distributive, cardiogenic, and obstructive. Initial evaluation of a child in shock involves assessing appearance, breathing, and circulation to identify life-threatening conditions and classify shock severity and type. Management begins with oxygen, IV access, glucose check, and fluid boluses. Further treatment depends on shock type but may include antibiotics for sepsis, vasoactive drugs for cardiogenic shock, or epinephrine for anaphylaxis. Children are monitored for response and complications, with transfer to ICU for non-responders.
This document summarizes a case study on anemia in pregnancy conducted at Muembe Ladu Maternity Hospital. The patient, a 22-year-old pregnant woman, presented with headaches, dizziness, weakness, and fatigue. Her hemoglobin level was initially 8.0 g/dl. She was diagnosed with anemia in pregnancy and prescribed iron supplements. Nursing assessments identified risks of nutritional imbalances, ineffective breathing, activity intolerance, and infection due to low hemoglobin. The patient received counseling and showed gradual improvement in symptoms and hemoglobin levels with treatment. The case study notes recommendations to improve care, such as ensuring adequate treatment duration and monitoring, as well as increasing health education and physician support at rural clinics.
A 27-year-old woman was admitted to the hospital with severe pre-eclampsia at 35 weeks and 3 days of gestation. She had a blood pressure of 180/110 mmHg, proteinuria, edema, headache, and blurred vision. She received magnesium sulfate and antihypertensive medications. She later had a cesarean section under general anesthesia and delivered twins weighing 1.8 kg and 1.6 kg. The nursing care focused on monitoring her vital signs and symptoms, managing her blood pressure and fluids, providing rest and medications, and health education. Recommendations included improving equipment, guidelines, and staff training for managing severe pre-eclampsia.
The document discusses the protozoan Entamoeba histolytica, which causes amoebiasis or amoebic dysentery. It describes the life cycle of E. histolytica, including the trophozoite, precyst, and cyst stages. People become infected through the fecal-oral route by ingesting E. histolytica cysts from contaminated food, water, or soil. In some cases, the trophozoites invade and ulcerate the colon, causing bloody diarrhea. Rarely, they spread through the bloodstream and cause abscesses in the liver.
This presentation provides information about pneumonia, including its definition, classification, causes, signs and symptoms, diagnostic tests, treatment, nursing management, preventive measures, prognosis, and complications. Pneumonia is an inflammatory process in the lungs caused most commonly by infection. It is classified based on its causes such as bacterial or viral, and the area of lung involvement. Common signs include fever, cough, and chest pain. Treatment involves antibiotics, oxygen therapy, and nursing interventions like chest physiotherapy. Preventive measures include vaccinations, smoking cessation, and reducing alcohol use. Most patients improve with treatment but elderly or very sick individuals may have longer recovery times.
This document provides information on congestive heart failure (CHF), including its definition, causes, risk factors, pathophysiology, signs and symptoms, diagnostic measures, nursing management, treatment, and prevention. CHF is defined as the heart's inability to pump enough blood to meet the body's needs. Common causes include coronary artery disease, heart attacks, valve disease, and hypertension. Diagnostic tests include echocardiograms, electrocardiograms, stress tests, and blood tests. Treatment involves medications to reduce workload on the heart and control symptoms, while prevention focuses on controlling risk factors like high blood pressure.
The document contains a nursing assessment of a female patient who was admitted to the hospital due to difficulty defecating and abdominal pain. It details her medical history, current condition, nursing diagnoses including constipation, pain, and impaired skin integrity. The assessment addresses all aspects of her health including physical, functional, psychosocial, and includes the patient's problems, priorities, and plan of care.
This document discusses respiratory failure, including its causes, types, and management. Respiratory failure occurs when inadequate gas exchange prevents normal oxygen and carbon dioxide levels in the blood. It can result from conditions affecting breathing muscles/nerves or lung tissue damage. The two main types are hypoxemic respiratory failure, where oxygen levels are too low, and hypercapnic respiratory failure, where carbon dioxide levels are too high. Management involves oxygen therapy, positioning, clearing secretions, and potentially positive pressure ventilation.
Asthma is a chronic lung disease characterized by inflammation and narrowing of the airways. Symptoms include coughing, wheezing, shortness of breath, and chest tightness. Work-related asthma develops from exposure to triggers like dusts, chemicals, or fumes on the job and accounts for up to 20% of adult asthma cases. Triggers are identified by paying attention to when and where symptoms occur. Asthma is managed through avoiding triggers, using long-term controller medications, and quick-relief rescue medications as needed. Proper treatment can control asthma symptoms but not cure the underlying condition.
This document discusses drugs used for skin and mucous membranes. It begins by describing the structure and functions of skin. It then discusses various topical drug categories including antifungals, antibacterials, anti-inflammatories, and antipruritics. Specific drugs are described in more detail including erythromycin gel, clindamycin, hydrocortisone, and betamethasone. Application methods and nursing considerations are provided for proper administration of these topical medications.
This document provides definitions and classifications of wounds. It discusses the pathophysiology of wound infection, including the signs of inflammation. It describes the management of wounds, including wound assessment, cleansing, and surgical debridement. It covers types of wound healing, factors affecting healing, and complications and their management. Some key points include classifications by degree of contamination and mechanism of injury. It discusses the cardinal signs of inflammation. It also outlines wound closure techniques and factors influencing wound healing such as nutrition, diabetes, and chronic diseases.
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4. Learning objectives’-
28/11/2014CRITICAL CARE4
At theend of thissession every leaner should
beableto understand thefollowing:-
Define shock
List thestageof shock
Describetypesof shock with its causes, sign
and symptoms.
Explain thepath physiology of shock
List sign & symptomsof theshock in general.
Mention thecomplication of shock.
DescribeManagement & treatment of shock
5. Definition
Shock isaseriousmedical condition where
thetissueperfusion isinsufficient to meet
demand for oxygen and nutrition becausethe
body isnot getting enough blood flow.
Shock often accompaniessevereinjury /
illness.
OR
Isalifethreatening condition that occurs
when thebody isnot getting enough blood
flow.
Zanzibar university
10. Types of shock
28/11/2014CRITICAL CARE10
Shock can bedivided into:
A. Hypovolemic shock
B. Cardiogenic shock
C. Obstructive shock
D. Distributiveshock
They are;-
( !) Sepsis
(2) Neurogenic (spinal shock)
(3) Anaphylaxis
(4) Electricity
11. (A). Hypovolemic shock
28/11/2014CRITICAL CARE11
Hypovolemic shock (caused by inadequate
blood volume)
Hypovolemic shock isan emergency condition in
which severeblood and fluid lossmakestheheart
unableto pump enough blood to thebody.
Thistypeof shock can causemany organsto stop
working.
12. 28/ 11/ 2014CRITICAL CARE12
Itsprimary causeishemorrhagedueto trauma
(internal and/or external), or lossof fluid from the
circulation and vomiting and diarrhea arethe
most common causein children.
With other causesincluding burns.
Cause of hypovolemic shock
13. SIGN AND SYMPTOMS
28/11/2014. CRITICAL CARE13
A rapid, weak, thread pulsedueto decreased
blood flow combined with tachycardia.
Cool, clammy skin due to stimulation of
vasoconstriction.
Rapid and shallow breathing dueto sympathetic
nervoussystem stimulation and acidosis
15. {B} Cardiogenic shock
28/11/2014CRITICAL CARE15
Cardiogenic shock isadiseasestatewherethe
heart isdamaged enough that it isunableto
supply sufficient blood to thebody.
16.
17. Sign and symptoms
28/ 11/ 2014CRITICAL CARE17
Distended jugular veins dueto increased
jugular venouspressure.
Weak or absent pulse.
Arrhythmia often
Tachycardia.
18. CAUSES OF CARDIOGENIC SHOCK
28/11/2014CRITICAL CARE18
Cardiogenic shock iscaused by thefailureof the
heart to pump effectively.
Thiscan bedueto damageto theheart muscle,
most often from alargemyocardial infarction.
Cardiomyopathy /myocarditis
Congestiveheart failure(CHF)
Cardiac valve problems.
19. OBSTUCTIVE SHOCK
Isdueto obstruction of blood flow outsideof
theheart.
Several conditionscan result in thisform of
shock.
01/09/15CRITICAL CARE19
20. Causes of obstructive shock
Cardiac tamponade in which fluid in thepericardium
preventsinflow of blood into theheart (venousreturn).
Constrictivepericarditis in which thepericardium shrinks
and hardens, issimilar in presentation.
Tension pneumothorax Through increased intrathoracic
pressure, blood flow to theheart isprevented (venous
return).
01/09/15CRITICAL CARE20
21. continue
Pulmonary embolism istheresult of a
thromboembolic incident in theblood vesselsof
thelungs and hindersthereturn of blood to the
heart.
Aortic stenosishinderscirculation by
obstructing theventricular outflow tract.
01/09/15CRITICAL CARE21
22. Sign and symptoms of
obstructive
Low blood pressure
Lack of oxygen to body tissue
Clammy skin
Cool skin.
01/09/15CRITICAL CARE22
23. {C} DISTRIBUTIVE SHOCK
28/11/2014CRITICAL CARE23
Isdueto impaired utilization of oxygen and thus
production of energy by thecell.
Distributiveshock includethefollowing;-
Septic
Anaphylactic,
Neurogenic,
Electricity
24. Causes of distributive shock
It can becaused by;-
Systematic Inflammatory ResponseSyndrome
(SIRS)
Dueto conditionsother than infection such as
pancreatitis,burns, or trauma.
01/09/15CRITICAL CARE24
25. continue
Toxic Shock Syndrome(TSS)
Anaphylaxis(A sudden ,severeallergic reaction)
Reactionsto drug or toxins.
Hepatic (Liver) insufficiency and damageto the(
CNS)
01/09/15CRITICAL CARE25
26. Sign and symptoms of
distributive
Temperature<36 OR > 38
Heart rate>90 beatsper minutes
Respiratory Rate>20bmin
01/09/15CRITICAL CARE26
27. {a} SEPTIC SHOCK
28/11/2014CRITICAL CARE27
Septic shock ( associated with infections)
Septic shock isaseriouscondition that occurs
when an overwhelming infection leadsto low
blood pressureand low blood flow.
Thebrain, heart, kidneys, and liver may not work
properly or may fail.
29. CAUSES OF SEPTIC
28/11/2014CRITICAL CARE29
Septic shock can becaused by ;-
1) Gram negative
Bacteriasuch as(among others) Escherichia
co li, Proteusspecies, klebsiela, pneumo niae which
releasean endotoxin which produces adverse
biochemical,
Immunological and occasionally neurological
effects which are harmful to the body.
30. Continue
28/ 11/ 2014CRITICAL CARE30
2) Gram-positive
Cocci such aspneumococci and
Streptococci and certain fungi aswell asGram-
positive
bacterial toxins.
Septic shock also includessomeelements of
cardiogenic shock.
31. Symptoms of septic
shock
Symptomsare:
similar to hypovolaemic shock except in thefirst
stages:
Pyrexiaand fever, or hyperthermia, dueto
overwhelming bacterial infection.
Vasodilation and increased cardiac output due
to sepsis.
01/09/15CRITICAL CARE31
35. Sign and symptoms of anaphylactic
Polyuria,
Respiratory distress, hypotension (low blood
pressure),
Encephalitis, fainting, unconsciousness,
Urticaria(hives),
Angioedema(swelling of thelips, face, neck
and throat), Tears(dueto angioedemaand stress),
Vomiting, itching,
Anxiety,
01/09/1535
CRITICAL CARE
36. (d) Electrical Shock
28/11/201436
Isthephysiological reaction or injury caused by
electric
current passing through thehuman body.
OR
An electric current that passesthrough thebody is
also called ashock.
And it can beso dangerous but electrical
shock isdifferent from themedical shock
discussed .
38. Continue
28/11/2014CRITICAL CARE38
In electric shock Oxygen isroutinely given, to the
victim and somepeopleneed to beput on a
ventilator (abreathing machine) to increasethe
amount of oxygen getting to their cells.
40. Sign and symptoms
Unconsciousness.
Burns, particulary entranceand exit burns(
wheretheelectricity entered and left thebody.)
Difficult in beathing or n breathing at all.
Sudden onset of cardiac arrest.
01/09/15CRITICAL CARE40
41. PATHOPHYSIOLOGY OF SHOCK
Cellsswitch from aerobic to anaerobic metabolism
Lactic acid production
Cell function ceasesand swells
Membranebecomesmorepermeable
Electrolytesand fluidsseepsin &out of cell.
01/09/15CRITICAL CARE41
42. continue
Na and K pump impaired
01/09/15CRITICAL CARE42
Mitochondria damage cells death
44. Sign and symptoms in general types
) Rapid respiration
2) Cold and clammy skin,
3) A weak but rapid pulse,
4) Fall in blood pressure, dueto reduction of blood
volume
5) Weaknessall over thebody.
6) Dizzy, confused, and may becomeunconscious.
Intensivethirsty dueto lossof water
01/09/1544
47. Managements and Treatments
shock
28/11/2014CRITICAL CARE47
Airway
breathing
Circulation
Treatmentsof shock alwaysshould beaimed to
correct thecauseof shock and helping
physiological compensatory mechanism to restore
thetissueperfusion.
48. continue
Themain goalsof thetreatment of cardiogenic
shock arethere-establishment of circulation to the
myocardium, minimising heart muscledamageand
improving theheart’seffectivenessasapump.
Oxygen (O2) therapy to reducestheworkload
of theheart by reducing tissuedemandsfor blood
flow.
Administration of cardiac drugs
01/09/15CRITICAL CARE48
49. Continue
Increaseheart’spumping action and decrease
work load of theheart through medication such as
Dopamine, dobutamine, epinephrine,
norepinephrine.
Nitroglycerineuseful only if cardiogenic shock
accompanied by appropriate
Dosage10mg-min 6ml-hr increaseby 10mg –
min every 5-10min to achieveoptimal effect.
01/09/15CRITICAL CARE49
50. Continue
28/11/2014CRITICAL CARE50
So dueto thisreason shock istreated dueto
under lying causesof it if whether ishemorrhagic,
or Hypovolemic or anaphylactic
In all typesof shock restoration of arterial
pressureisessential to maintain perfusion and
blood flow to thebrain.
51. continue
28/11/2014CRITICAL CARE51
Peoplein shock should betaken by ambulance
to ahospital asquickly aspossible. then, they
should bekept lying down on their back with their
feet raised about afoot higher than their head.
Administer counter-mediatorssuch asanti-
histamine.
52. GENERAL PREVENTION OF SHOCK
28/11/2014CRITICAL CARE52
Elevatethefoot of thebed to maintain agood
blood supply to thevital organ eg;- brain cells
Replacement of lost fluidsby infusion or
transfusion.
Administer oxygen if cyanosisismarked.
In caseof trauma, giveanalgesic for pain.
53. CONTINUE
Identify and control sourceof infection
Administer antibiotic
Removerisk factor for infection
01/09/15CRITICAL CARE53
55. References
28/ 11/ 2014CRITICAL CARE55
Silverman, Adam (Oct 2005). "Shock: A Common Pathway For Life-
Threatening Pediatric Illnesses And Injuries". Pediatric Emergency
Medicine Practice 2 (10).
Tintinalli, Judith E. (2010). Emergency Medicine: A Comprehensive Study
Guide (Emergency Medicine (Tintinalli)). New York: McGraw-Hill
Companies. pp. 165–172. ISBN 0-07-148480-9. Marino, Paul L.
(September 2006). The ICU Book. Lippincott Williams & Wilkins,
Philadelphia & London. ISBN 0-7817-4802-X.
Tintinalli, Judith E. (2010). Emergency Medicine: A Comprehensive Study
Guide (Emergency Medicine (Tintinalli)). New York: McGraw-Hill
Companies. pp. 174–175. ISBN 0-07-148480-9.
Tintinalli, Judith E. (2010). Emergency Medicine: A Comprehensive Study
Guide (Emergency Medicine (Tintinalli)). New York: McGraw-Hill
Companies. ISBN 0-07-148480-9.