REFRACTIVE ERRORS
10/12/2016 SAH SURENDRA
EMMETROPIA
 The parallel rays of light coming from infinity are focused at
sensitive layer of retina with the accommodation at being at
rest
 Good visual acuity (6/6 or better) at 6 m testing distance
 If accommodation amplitude is adequate ,equally good visual
acuity at the near testing distance of 40 cm
 Axial length =24mm
10/12/2016 SAH SURENDRA
CONT
10/12/2016 SAH SURENDRA
Refractive sate of eye
 Corneal refractive power
 Anterior chamber depth
 Lens refracting power
 Axial length
10/12/2016 SAH SURENDRA
AMETROPIA
 The parallel rays of light coming from infinity are focused
either in front or behind the sensitive layer of retina in one or
both meridian when accommodation at rest
 Refractive Status And Aging
o Hyperopic (infant)
o Emmetropic (10 years old)
o Myopic (25 years old)
o Hyperopic (60 years old)
o Less hyperopic (80 years
10/12/2016 SAH SURENDRA
TYPES OF REFRACTIVE ERRORS
Hypermetropia
Myopia
Astigmatism
Aphakia
pseudophakia
10/12/2016 SAH SURENDRA
HYPERMETROPIA
 The parallel rays of light coming from infinity are focused
behind the sensitive layer of retina with accommodation
being at rest
 Back focal point is behind retina
 It is also called far/long sightedness
10/12/2016 SAH SURENDRA
AETIOLOGY
Axial hypermetropia:-the antero-posterior diameter of
eye is too short & retina is too near to optical system,1mm-3D
Curvature hypermetropia:-curvature of cornea or
lens may be too small,1mm-6D
Index hypermetropia:- due to change in refractive
index of lens ,aqueous humour ,vitreous humour or diabetes
Positional hypermetropia:-if the crystalline lens is
dislocated backwards
Absence of crystalline lens:- aphakia
10/12/2016 SAH SURENDRA
CLINICAL TYPES
simple hypermetropia:- normal biological variations in
development of eyeball
Pathological hypermetropia:-outside biological
variations of development ,include:-
 Senile :- curvature &index
 Positional
 Aphakia
Functional hypermetropia:-paralysis of
accommodation -third nerve paralysis &internal
ophthaloplegia
10/12/2016 SAH SURENDRA
OPTICAL CONDITION
 Parallel rays of light come to a behind the retina
 Parallel rays of light are brought to a focus upon the
retina by increasing the refractivity by
accommodation .the normal lens becomes more
convex
 Parallel rays of light are brought to a focus upon the
retina by increasing refractivity by a convex
spectacles lens .the degree of hypermetropia is given
by power lens
10/12/2016 SAH SURENDRA
ACCOMMODATION IN HP
 Latent hypermetropia (LH):-
 This is a condition in which all or part of hypermetropia is compensated or corrected by
the tonicity of the ciliary muscle.
 Latent hypermetropia becomes manifest when the amplitude of accommodation
decreased significantly.
 Manifest hypermetropia (MH): It has two parts.
 Facultative hypermetropia (fH) – The amount of hypermetrropia that can be
corrected by the effort of accommodation.
 Absolute hypermetropia (aH) – T he amount of hypermetropia that can not be
corrected by the effort of accommodation.
 Thus the total amount of hypermetropia (tH) is the sum of
latent and manifest hypermetropia.
tH=LH+MH=LH+(fH+aH).
10/12/2016 SAH SURENDRA
CLINICAL PATHOLOGY
 Cornea is small, AC shallow ,closed –angle glaucoma
 Administration of dilated drugs to dilate pupil
 Developmental aberrations such as colobomata
,microphthalmos
 The retina appears to have peculiar sheen, optic neuritis,
vascular reflex
 Face asymmetrical ,dislocation of macula, divergent squint
10/12/2016 SAH SURENDRA
CLINICAL FEATURES
 SYMPTOMS:-
 Asymtomatic
 Asthenopic :-tiredness, frontal or frontotemporal headache, watering, mild
photophobia
 Defective vision with asthenopic symptoms
 Defective vision only
 The effect of ageing on vision
 Intermitted sudden blurring of vision
 Crossed – eye sensation
10/12/2016 SAH SURENDRA
CONT
Signs:-
 Size of eyeball may be normal or small as whole
 Cornea may smaller than normal
 Variation in visual acuity
 Abnormal branching of vessels , swelling of disc, retinal shine is
more than normal
 A –scan ultrasonography may reveal a short anteroposterior length
of eyeball
10/12/2016 SAH SURENDRA
COMPLICATION
Recurrent styes, blepharitis or chalazia
Accommodative convergent squint
Amblyopia
Predisposition to develop primary narrow
angle glaucoma
10/12/2016 SAH SURENDRA
TREATMENT
OPTICAL TREATMENT:-CONVEX(PLUS) LENS
 Total amount of HP-complete cycloplegic
 Manifest refractive error is small-1D or less
 Astigmatism should be fully corrected
 Exophoria -1-2D less
 Accommodative convergent squint /amblyopia-full correction
Surgery for Hp:-
 Thermokeratoplasty
 The use of excimer &Holmium lasers is under investigation
10/12/2016 SAH SURENDRA
APHAKIA
 Condition in which absence of crystaline lens from
pupillary area of eye
CAUSES:-
 Congenital absence of lens
 Surgical aphakia occurring after removal of lens
 Aphakia due to absorption of lens matter
 Traumatic extrusion of lens
 Posterior dislocation of lens in vitreous
10/12/2016 SAH SURENDRA
OPTICS OF APHAKIA
 Changes in cardinal data of the eye
 Image formation in the aphakic eye
 Visual acuity in aphakia
 Accommodation in aphakia
 Binocular vision
10/12/2016 SAH SURENDRA
CLINICAL FEATURES
SYMPTOMS:-
 Defective vision for near & distance
 Erythropsia and cyanopsia – due to entry of infrared and ultraviolet rays in
the absence of the crystalline lens
SIGNS:-
 Limbal scar in case of surgical aphakia
 Deep anterior chamber
 Iridodonesis – tremulousness of the iris due to loss of support of
lens
 Jet Black pupil
 Loss of 3rd and 4th purkinje images
 Fundus examination reveals a small hypermetropic fundus
 Retinoscopy shows high hypermetropia
10/12/2016 SAH SURENDRA
TREATMENT
SPECTACLES:-
 Spectacles should be prescribed with about +10D lens for
correction of aphakia
 It should also include correction for surgical astigmatism and
+3-4D for near vision
 Nowadays spectacles are not preferred for use in aphakia due
to its many disadvantage
 Advantages of using spectacles in aphakia
• Easy to use
• No complications
• cheap
10/12/2016 SAH SURENDRA
CONT…..
 Disadvantages
The images are magnified – about 30% – hence not useful in unilateral
aphakia as it causes diplopia
• The field of vision in decreased considerably
• Spherical and chromatic aberration of high power lenses
• Roving ring scotoma (Jack in the box phenomenon)
• Prismatic effect of the thick lenses
• High power are cosmetically not acceptable
Contact lenses
Advantages over spectacles:
• Produces less magnification
• Better field of vision
• Less chromatic and spherical aberration
• No prismatic effect
• Cosmetically more acceptable
10/12/2016 SAH SURENDRA
CONT……
Intraocular lens implantation
• The lens can be implanted in the capsular bag or in the anterior chamber
• It eliminates most of the disadvantages associated with the use of spectacles
or contact lenses
• Disadvantage include the complications associated with surgery
Refractive surgery
 Keratophakia
– a lenticule prepared from the donor cornea is placed within the
lamellae of the patient’s cornea
 Epikeratophakia
– a lenticule prepared form the donor cornea is stitched to the
patients cornea after removing the epithelium
 Hyperopic Lasik
10/12/2016 SAH SURENDRA
PSEUDOPHAKIA
 The condition of aphakia when corrected with
implantation of an IOL
Refractive status:-
 Emmetropia
 Consective myopia
 Consective ypermetropia
10/12/2016 SAH SURENDRA
SIGNS
 Surgical limbal scar may be seen
 Anterior chamber is slightly deeper than normal
 Mild iridodonesis of iris may be demonstrated
 Purkinje image test shows fours images
 Pupil is blackish in colour but when light thrown in pupillary
area shining reflexes are observed
10/12/2016 SAH SURENDRA
MYOPIA
 Shortsightness
 Lengthening of posterior part of eye
 The parallel rays of light coming from infinity are focused in
front of sensitive layer of retina when accommodation is at
rest
10/12/2016 SAH SURENDRA
AETIOLOGY
Axial myopia:-the antero-posterior diameter of eye is too
long & retina is too far from optical system
Curvature myopia:-the curvature of cornea or lens may
be too great
Positional myopia:-if lens is dislocated forwards
Index myopia:-R.i of aqueous is too high ,vitreous is too
low or lens is too high
 Myopia due to excessive accommodation occurs in patients
with spasm of accommodation
10/12/2016 SAH SURENDRA
OPTICS OF MYOPIA
 Optical system of a myopic eye is too powerful for its axial length
 Image of distance object on retina is made up of circles of diffusion
formed by divergent beam
 Far point of the myopic eye is a finite point in front of the eye
 Nodal point in myopic eye is further away from retina
 Angle alpha of eye may be negative
 Accommodation in uncorrected myopes is not developed normally
10/12/2016 SAH SURENDRA
CLINICAL VARIETIES OF MYOPIA
 Congenital myopia
 Simple or developmental myopia
 Pathological or degenerative myopia
 Acquired myopia
10/12/2016 SAH SURENDRA
CONGENTICAL MYOPIA
The child is born with elongated eyes
The refraction may be upto -10D
Typical fundus changes are seen
Progression is rare
10/12/2016 SAH SURENDRA
SIMPLE MYOPIA
 Commonest clinical type
 Does not progress much after the adolescence
 May be upto -5 to -6D
 No degenerative changes are seen in the fundus, although
peripheral retinal degeneration may be seen in later life
 Associated with good vision with a good prognosis
10/12/2016 SAH SURENDRA
PATHOLOGICAL MYOPIA
 It is also called progressive or degenerative myopia
 Myopia appears in childhood &increasing steadily with age upto 25
years or beyond
 The final amount of myopia may be -15Dto -25D or more
 There are typical degenerative changes in the fundus
 Strongly hereditary & more common in female
 Prognosis is usually poor
10/12/2016 SAH SURENDRA
ACQUIRED MYOPIA
CAUSES :-
 Index myopia
 Curvatural myopia
 Positional myopia
 Consecutive myopia
 Pseudomyopia
 Space myopia
 Night myopia
 Drug –induced myopia
10/12/2016 SAH SURENDRA
CLINICAL FEATURS
SYMPTOMS:-
 Impaired distance vision
 Eye –strain
 Exophoria or divergent squint
 Delayed dark adaptation
 Sudden loss of vision
 Black floaters
SIGNS:-
 Prominent eyeball, large cornea, deep anterior chamber& a
large pupil, apparent convergent squint, degeneration of
vitreous
10/12/2016 SAH SURENDRA
COMPLICATION OF MYOPIA
Retinal tear –vitreous haemorrhage
Retinal detachment degeneration of the
vitreous
Primary open –angle glaucoma
Posterior cortical cataract
10/12/2016 SAH SURENDRA
TREATMENT
Divergent or minus lenses in spectacles or
contact lenses
Cycloplegics
Surgery
Refractive surgery
General hygiene
10/12/2016 SAH SURENDRA
ASTIGMATISM
 Condition of refraction wherein a point focus of light cannot
be formed upon the retina
 Caused by unequal refraction of light in different meridians
 Curvatures of both axes are unequal & too small is HM ASG &
both unequal & too great is MP ASG
10/12/2016 SAH SURENDRA
AETIOLOGY
Corneal astigmastism :-abnormalites of curvature of
cornea
Lenticular astigmatism:-
• Small amount of curvature astigmatism due to congenital abnormalities of
curvature of lens
• Position astigmatism due to congenital or traumatic subluxation of lens
• Refractive index of lens in different median
Retinal astigmatism due to oblique placement
of macula
10/12/2016 SAH SURENDRA
TYPES OF ASTIGMATISM
REGULAR ASTIGMASTIM:-Axis & angle
 With –the –rule :-two principal meridian are placed at right
angles to one another but vertical meridian is more curve
than horizontal
 Against –the rule :-horizontal meridian is more curved than
vertical meridian
 Oblique:-two principal meridian are not horizontal & vertical
@ right angle to one another
 Bi-oblique:-two principal meridian are not @ right angle to
each other
10/12/2016 SAH SURENDRA
CONT..
REGULAR ASTIGMATISM:-REFRACTIVE TYPES
 Simple:-one meridian focus in front & other behind retina
 Compound :-ray of light in both meridian are focused either
in front or behind retina
 Mixed:-rays of light in one meridian are focused in front &
other behind retina
10/12/2016 SAH SURENDRA
OPTICS OF REGULAR ASTIGMATISM
 Parallel rays of light are not focused on a point but form two
focal lines
 The configuration of rays refracted through astigmatism
surface is called sturm’s conoid& distance between two lines
is called focal interval of sturm
 The length of this focal interval is a measure of degree of
astigmatism
10/12/2016 SAH SURENDRA

Refractive errors by surendra

  • 1.
  • 2.
    EMMETROPIA  The parallelrays of light coming from infinity are focused at sensitive layer of retina with the accommodation at being at rest  Good visual acuity (6/6 or better) at 6 m testing distance  If accommodation amplitude is adequate ,equally good visual acuity at the near testing distance of 40 cm  Axial length =24mm 10/12/2016 SAH SURENDRA
  • 3.
  • 4.
    Refractive sate ofeye  Corneal refractive power  Anterior chamber depth  Lens refracting power  Axial length 10/12/2016 SAH SURENDRA
  • 5.
    AMETROPIA  The parallelrays of light coming from infinity are focused either in front or behind the sensitive layer of retina in one or both meridian when accommodation at rest  Refractive Status And Aging o Hyperopic (infant) o Emmetropic (10 years old) o Myopic (25 years old) o Hyperopic (60 years old) o Less hyperopic (80 years 10/12/2016 SAH SURENDRA
  • 6.
    TYPES OF REFRACTIVEERRORS Hypermetropia Myopia Astigmatism Aphakia pseudophakia 10/12/2016 SAH SURENDRA
  • 7.
    HYPERMETROPIA  The parallelrays of light coming from infinity are focused behind the sensitive layer of retina with accommodation being at rest  Back focal point is behind retina  It is also called far/long sightedness 10/12/2016 SAH SURENDRA
  • 8.
    AETIOLOGY Axial hypermetropia:-the antero-posteriordiameter of eye is too short & retina is too near to optical system,1mm-3D Curvature hypermetropia:-curvature of cornea or lens may be too small,1mm-6D Index hypermetropia:- due to change in refractive index of lens ,aqueous humour ,vitreous humour or diabetes Positional hypermetropia:-if the crystalline lens is dislocated backwards Absence of crystalline lens:- aphakia 10/12/2016 SAH SURENDRA
  • 9.
    CLINICAL TYPES simple hypermetropia:-normal biological variations in development of eyeball Pathological hypermetropia:-outside biological variations of development ,include:-  Senile :- curvature &index  Positional  Aphakia Functional hypermetropia:-paralysis of accommodation -third nerve paralysis &internal ophthaloplegia 10/12/2016 SAH SURENDRA
  • 10.
    OPTICAL CONDITION  Parallelrays of light come to a behind the retina  Parallel rays of light are brought to a focus upon the retina by increasing the refractivity by accommodation .the normal lens becomes more convex  Parallel rays of light are brought to a focus upon the retina by increasing refractivity by a convex spectacles lens .the degree of hypermetropia is given by power lens 10/12/2016 SAH SURENDRA
  • 11.
    ACCOMMODATION IN HP Latent hypermetropia (LH):-  This is a condition in which all or part of hypermetropia is compensated or corrected by the tonicity of the ciliary muscle.  Latent hypermetropia becomes manifest when the amplitude of accommodation decreased significantly.  Manifest hypermetropia (MH): It has two parts.  Facultative hypermetropia (fH) – The amount of hypermetrropia that can be corrected by the effort of accommodation.  Absolute hypermetropia (aH) – T he amount of hypermetropia that can not be corrected by the effort of accommodation.  Thus the total amount of hypermetropia (tH) is the sum of latent and manifest hypermetropia. tH=LH+MH=LH+(fH+aH). 10/12/2016 SAH SURENDRA
  • 12.
    CLINICAL PATHOLOGY  Corneais small, AC shallow ,closed –angle glaucoma  Administration of dilated drugs to dilate pupil  Developmental aberrations such as colobomata ,microphthalmos  The retina appears to have peculiar sheen, optic neuritis, vascular reflex  Face asymmetrical ,dislocation of macula, divergent squint 10/12/2016 SAH SURENDRA
  • 13.
    CLINICAL FEATURES  SYMPTOMS:- Asymtomatic  Asthenopic :-tiredness, frontal or frontotemporal headache, watering, mild photophobia  Defective vision with asthenopic symptoms  Defective vision only  The effect of ageing on vision  Intermitted sudden blurring of vision  Crossed – eye sensation 10/12/2016 SAH SURENDRA
  • 14.
    CONT Signs:-  Size ofeyeball may be normal or small as whole  Cornea may smaller than normal  Variation in visual acuity  Abnormal branching of vessels , swelling of disc, retinal shine is more than normal  A –scan ultrasonography may reveal a short anteroposterior length of eyeball 10/12/2016 SAH SURENDRA
  • 15.
    COMPLICATION Recurrent styes, blepharitisor chalazia Accommodative convergent squint Amblyopia Predisposition to develop primary narrow angle glaucoma 10/12/2016 SAH SURENDRA
  • 16.
    TREATMENT OPTICAL TREATMENT:-CONVEX(PLUS) LENS Total amount of HP-complete cycloplegic  Manifest refractive error is small-1D or less  Astigmatism should be fully corrected  Exophoria -1-2D less  Accommodative convergent squint /amblyopia-full correction Surgery for Hp:-  Thermokeratoplasty  The use of excimer &Holmium lasers is under investigation 10/12/2016 SAH SURENDRA
  • 17.
    APHAKIA  Condition inwhich absence of crystaline lens from pupillary area of eye CAUSES:-  Congenital absence of lens  Surgical aphakia occurring after removal of lens  Aphakia due to absorption of lens matter  Traumatic extrusion of lens  Posterior dislocation of lens in vitreous 10/12/2016 SAH SURENDRA
  • 18.
    OPTICS OF APHAKIA Changes in cardinal data of the eye  Image formation in the aphakic eye  Visual acuity in aphakia  Accommodation in aphakia  Binocular vision 10/12/2016 SAH SURENDRA
  • 19.
    CLINICAL FEATURES SYMPTOMS:-  Defectivevision for near & distance  Erythropsia and cyanopsia – due to entry of infrared and ultraviolet rays in the absence of the crystalline lens SIGNS:-  Limbal scar in case of surgical aphakia  Deep anterior chamber  Iridodonesis – tremulousness of the iris due to loss of support of lens  Jet Black pupil  Loss of 3rd and 4th purkinje images  Fundus examination reveals a small hypermetropic fundus  Retinoscopy shows high hypermetropia 10/12/2016 SAH SURENDRA
  • 20.
    TREATMENT SPECTACLES:-  Spectacles shouldbe prescribed with about +10D lens for correction of aphakia  It should also include correction for surgical astigmatism and +3-4D for near vision  Nowadays spectacles are not preferred for use in aphakia due to its many disadvantage  Advantages of using spectacles in aphakia • Easy to use • No complications • cheap 10/12/2016 SAH SURENDRA
  • 21.
    CONT…..  Disadvantages The imagesare magnified – about 30% – hence not useful in unilateral aphakia as it causes diplopia • The field of vision in decreased considerably • Spherical and chromatic aberration of high power lenses • Roving ring scotoma (Jack in the box phenomenon) • Prismatic effect of the thick lenses • High power are cosmetically not acceptable Contact lenses Advantages over spectacles: • Produces less magnification • Better field of vision • Less chromatic and spherical aberration • No prismatic effect • Cosmetically more acceptable 10/12/2016 SAH SURENDRA
  • 22.
    CONT…… Intraocular lens implantation •The lens can be implanted in the capsular bag or in the anterior chamber • It eliminates most of the disadvantages associated with the use of spectacles or contact lenses • Disadvantage include the complications associated with surgery Refractive surgery  Keratophakia – a lenticule prepared from the donor cornea is placed within the lamellae of the patient’s cornea  Epikeratophakia – a lenticule prepared form the donor cornea is stitched to the patients cornea after removing the epithelium  Hyperopic Lasik 10/12/2016 SAH SURENDRA
  • 23.
    PSEUDOPHAKIA  The conditionof aphakia when corrected with implantation of an IOL Refractive status:-  Emmetropia  Consective myopia  Consective ypermetropia 10/12/2016 SAH SURENDRA
  • 24.
    SIGNS  Surgical limbalscar may be seen  Anterior chamber is slightly deeper than normal  Mild iridodonesis of iris may be demonstrated  Purkinje image test shows fours images  Pupil is blackish in colour but when light thrown in pupillary area shining reflexes are observed 10/12/2016 SAH SURENDRA
  • 25.
    MYOPIA  Shortsightness  Lengtheningof posterior part of eye  The parallel rays of light coming from infinity are focused in front of sensitive layer of retina when accommodation is at rest 10/12/2016 SAH SURENDRA
  • 26.
    AETIOLOGY Axial myopia:-the antero-posteriordiameter of eye is too long & retina is too far from optical system Curvature myopia:-the curvature of cornea or lens may be too great Positional myopia:-if lens is dislocated forwards Index myopia:-R.i of aqueous is too high ,vitreous is too low or lens is too high  Myopia due to excessive accommodation occurs in patients with spasm of accommodation 10/12/2016 SAH SURENDRA
  • 27.
    OPTICS OF MYOPIA Optical system of a myopic eye is too powerful for its axial length  Image of distance object on retina is made up of circles of diffusion formed by divergent beam  Far point of the myopic eye is a finite point in front of the eye  Nodal point in myopic eye is further away from retina  Angle alpha of eye may be negative  Accommodation in uncorrected myopes is not developed normally 10/12/2016 SAH SURENDRA
  • 28.
    CLINICAL VARIETIES OFMYOPIA  Congenital myopia  Simple or developmental myopia  Pathological or degenerative myopia  Acquired myopia 10/12/2016 SAH SURENDRA
  • 29.
    CONGENTICAL MYOPIA The childis born with elongated eyes The refraction may be upto -10D Typical fundus changes are seen Progression is rare 10/12/2016 SAH SURENDRA
  • 30.
    SIMPLE MYOPIA  Commonestclinical type  Does not progress much after the adolescence  May be upto -5 to -6D  No degenerative changes are seen in the fundus, although peripheral retinal degeneration may be seen in later life  Associated with good vision with a good prognosis 10/12/2016 SAH SURENDRA
  • 31.
    PATHOLOGICAL MYOPIA  Itis also called progressive or degenerative myopia  Myopia appears in childhood &increasing steadily with age upto 25 years or beyond  The final amount of myopia may be -15Dto -25D or more  There are typical degenerative changes in the fundus  Strongly hereditary & more common in female  Prognosis is usually poor 10/12/2016 SAH SURENDRA
  • 32.
    ACQUIRED MYOPIA CAUSES :- Index myopia  Curvatural myopia  Positional myopia  Consecutive myopia  Pseudomyopia  Space myopia  Night myopia  Drug –induced myopia 10/12/2016 SAH SURENDRA
  • 33.
    CLINICAL FEATURS SYMPTOMS:-  Impaireddistance vision  Eye –strain  Exophoria or divergent squint  Delayed dark adaptation  Sudden loss of vision  Black floaters SIGNS:-  Prominent eyeball, large cornea, deep anterior chamber& a large pupil, apparent convergent squint, degeneration of vitreous 10/12/2016 SAH SURENDRA
  • 34.
    COMPLICATION OF MYOPIA Retinaltear –vitreous haemorrhage Retinal detachment degeneration of the vitreous Primary open –angle glaucoma Posterior cortical cataract 10/12/2016 SAH SURENDRA
  • 35.
    TREATMENT Divergent or minuslenses in spectacles or contact lenses Cycloplegics Surgery Refractive surgery General hygiene 10/12/2016 SAH SURENDRA
  • 36.
    ASTIGMATISM  Condition ofrefraction wherein a point focus of light cannot be formed upon the retina  Caused by unequal refraction of light in different meridians  Curvatures of both axes are unequal & too small is HM ASG & both unequal & too great is MP ASG 10/12/2016 SAH SURENDRA
  • 37.
    AETIOLOGY Corneal astigmastism :-abnormalitesof curvature of cornea Lenticular astigmatism:- • Small amount of curvature astigmatism due to congenital abnormalities of curvature of lens • Position astigmatism due to congenital or traumatic subluxation of lens • Refractive index of lens in different median Retinal astigmatism due to oblique placement of macula 10/12/2016 SAH SURENDRA
  • 38.
    TYPES OF ASTIGMATISM REGULARASTIGMASTIM:-Axis & angle  With –the –rule :-two principal meridian are placed at right angles to one another but vertical meridian is more curve than horizontal  Against –the rule :-horizontal meridian is more curved than vertical meridian  Oblique:-two principal meridian are not horizontal & vertical @ right angle to one another  Bi-oblique:-two principal meridian are not @ right angle to each other 10/12/2016 SAH SURENDRA
  • 39.
    CONT.. REGULAR ASTIGMATISM:-REFRACTIVE TYPES Simple:-one meridian focus in front & other behind retina  Compound :-ray of light in both meridian are focused either in front or behind retina  Mixed:-rays of light in one meridian are focused in front & other behind retina 10/12/2016 SAH SURENDRA
  • 40.
    OPTICS OF REGULARASTIGMATISM  Parallel rays of light are not focused on a point but form two focal lines  The configuration of rays refracted through astigmatism surface is called sturm’s conoid& distance between two lines is called focal interval of sturm  The length of this focal interval is a measure of degree of astigmatism 10/12/2016 SAH SURENDRA