This document provides an overview of refractive errors, including emmetropia, ametropia, and various types of refractive errors such as hyperopia, myopia, astigmatism, aphakia, and pseudophakia. It discusses the optical conditions and clinical features of each type of refractive error, as well as their causes, treatment options, and potential complications. Key information covered includes the definition of emmetropia as the eye's ability to focus light correctly on the retina, the various causes and classifications of refractive errors, and management approaches for different refractive conditions including optical corrections and refractive surgery.
The tear film constitutes Three layers :- An outermost lipid (oily) layer An aqueous (watery) layer that makes up 90% of the tear film volume; and A mucin layer that coats the corneal surface.
3. To form smooth optical surface on cornea. To keep the surface of cornea & conjunctiva moist It serve as lubricant It transfer oxygen Provide antibacterial action Wash debris out It provides a pathway for WBC in case of injury
4. Functions of lipid layer Retards evaporation of tear film Prevents the overflow of tears
5. Function of Aqueous Layer Flushes, buffers and lubricates the corneal surface Delivers oxygen and other nutrients to the corneal surface Wash out debris Delivers antibacterial enzymes and antibodies such as lysozyme.
6. Functions of Mucin Layer Spreads tears over corneal surface. Protects the cornea against foreign substances . Makes corneal surface smooth by filling in surface irregularities
The tear film constitutes Three layers :- An outermost lipid (oily) layer An aqueous (watery) layer that makes up 90% of the tear film volume; and A mucin layer that coats the corneal surface.
3. To form smooth optical surface on cornea. To keep the surface of cornea & conjunctiva moist It serve as lubricant It transfer oxygen Provide antibacterial action Wash debris out It provides a pathway for WBC in case of injury
4. Functions of lipid layer Retards evaporation of tear film Prevents the overflow of tears
5. Function of Aqueous Layer Flushes, buffers and lubricates the corneal surface Delivers oxygen and other nutrients to the corneal surface Wash out debris Delivers antibacterial enzymes and antibodies such as lysozyme.
6. Functions of Mucin Layer Spreads tears over corneal surface. Protects the cornea against foreign substances . Makes corneal surface smooth by filling in surface irregularities
Each month, join us as we highlight and discuss hot topics ranging from the future of higher education to wearable technology, best productivity hacks and secrets to hiring top talent. Upload your SlideShares, and share your expertise with the world!
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Correction of Ametropia is very basic topic in Optometry background. Hope the SlideShare may help you. This PPT will help Bachelor students (B.optoms).
This presentation is on ammetropia, types of ammetropia, causes of ammetropia, correction of ammetropia with lenses, optical aberrations, chromatic aberration, spherical aberration, coma, accommodation, changes of accommodation, anamolies of accommodation, presbyopia, management of presbyopia
Aphakia and its causes. Correction of Aphakia. Advantages and disadvantages of different corrections. Surgeries and related signs and symptoms of aphakia. Complications related to Aphakia.
Explore natural remedies for syphilis treatment in Singapore. Discover alternative therapies, herbal remedies, and lifestyle changes that may complement conventional treatments. Learn about holistic approaches to managing syphilis symptoms and supporting overall health.
The prostate is an exocrine gland of the male mammalian reproductive system
It is a walnut-sized gland that forms part of the male reproductive system and is located in front of the rectum and just below the urinary bladder
Function is to store and secrete a clear, slightly alkaline fluid that constitutes 10-30% of the volume of the seminal fluid that along with the spermatozoa, constitutes semen
A healthy human prostate measures (4cm-vertical, by 3cm-horizontal, 2cm ant-post ).
It surrounds the urethra just below the urinary bladder. It has anterior, median, posterior and two lateral lobes
It’s work is regulated by androgens which are responsible for male sex characteristics
Generalised disease of the prostate due to hormonal derangement which leads to non malignant enlargement of the gland (increase in the number of epithelial cells and stromal tissue)to cause compression of the urethra leading to symptoms (LUTS
ARTIFICIAL INTELLIGENCE IN HEALTHCARE.pdfAnujkumaranit
Artificial intelligence (AI) refers to the simulation of human intelligence processes by machines, especially computer systems. It encompasses tasks such as learning, reasoning, problem-solving, perception, and language understanding. AI technologies are revolutionizing various fields, from healthcare to finance, by enabling machines to perform tasks that typically require human intelligence.
Pulmonary Thromboembolism - etilogy, types, medical- Surgical and nursing man...VarunMahajani
Disruption of blood supply to lung alveoli due to blockage of one or more pulmonary blood vessels is called as Pulmonary thromboembolism. In this presentation we will discuss its causes, types and its management in depth.
Report Back from SGO 2024: What’s the Latest in Cervical Cancer?bkling
Are you curious about what’s new in cervical cancer research or unsure what the findings mean? Join Dr. Emily Ko, a gynecologic oncologist at Penn Medicine, to learn about the latest updates from the Society of Gynecologic Oncology (SGO) 2024 Annual Meeting on Women’s Cancer. Dr. Ko will discuss what the research presented at the conference means for you and answer your questions about the new developments.
Flu Vaccine Alert in Bangalore Karnatakaaddon Scans
As flu season approaches, health officials in Bangalore, Karnataka, are urging residents to get their flu vaccinations. The seasonal flu, while common, can lead to severe health complications, particularly for vulnerable populations such as young children, the elderly, and those with underlying health conditions.
Dr. Vidisha Kumari, a leading epidemiologist in Bangalore, emphasizes the importance of getting vaccinated. "The flu vaccine is our best defense against the influenza virus. It not only protects individuals but also helps prevent the spread of the virus in our communities," he says.
This year, the flu season is expected to coincide with a potential increase in other respiratory illnesses. The Karnataka Health Department has launched an awareness campaign highlighting the significance of flu vaccinations. They have set up multiple vaccination centers across Bangalore, making it convenient for residents to receive their shots.
To encourage widespread vaccination, the government is also collaborating with local schools, workplaces, and community centers to facilitate vaccination drives. Special attention is being given to ensuring that the vaccine is accessible to all, including marginalized communities who may have limited access to healthcare.
Residents are reminded that the flu vaccine is safe and effective. Common side effects are mild and may include soreness at the injection site, mild fever, or muscle aches. These side effects are generally short-lived and far less severe than the flu itself.
Healthcare providers are also stressing the importance of continuing COVID-19 precautions. Wearing masks, practicing good hand hygiene, and maintaining social distancing are still crucial, especially in crowded places.
Protect yourself and your loved ones by getting vaccinated. Together, we can help keep Bangalore healthy and safe this flu season. For more information on vaccination centers and schedules, residents can visit the Karnataka Health Department’s official website or follow their social media pages.
Stay informed, stay safe, and get your flu shot today!
MANAGEMENT OF ATRIOVENTRICULAR CONDUCTION BLOCK.pdfJim Jacob Roy
Cardiac conduction defects can occur due to various causes.
Atrioventricular conduction blocks ( AV blocks ) are classified into 3 types.
This document describes the acute management of AV block.
New Directions in Targeted Therapeutic Approaches for Older Adults With Mantl...i3 Health
i3 Health is pleased to make the speaker slides from this activity available for use as a non-accredited self-study or teaching resource.
This slide deck presented by Dr. Kami Maddocks, Professor-Clinical in the Division of Hematology and
Associate Division Director for Ambulatory Operations
The Ohio State University Comprehensive Cancer Center, will provide insight into new directions in targeted therapeutic approaches for older adults with mantle cell lymphoma.
STATEMENT OF NEED
Mantle cell lymphoma (MCL) is a rare, aggressive B-cell non-Hodgkin lymphoma (NHL) accounting for 5% to 7% of all lymphomas. Its prognosis ranges from indolent disease that does not require treatment for years to very aggressive disease, which is associated with poor survival (Silkenstedt et al, 2021). Typically, MCL is diagnosed at advanced stage and in older patients who cannot tolerate intensive therapy (NCCN, 2022). Although recent advances have slightly increased remission rates, recurrence and relapse remain very common, leading to a median overall survival between 3 and 6 years (LLS, 2021). Though there are several effective options, progress is still needed towards establishing an accepted frontline approach for MCL (Castellino et al, 2022). Treatment selection and management of MCL are complicated by the heterogeneity of prognosis, advanced age and comorbidities of patients, and lack of an established standard approach for treatment, making it vital that clinicians be familiar with the latest research and advances in this area. In this activity chaired by Michael Wang, MD, Professor in the Department of Lymphoma & Myeloma at MD Anderson Cancer Center, expert faculty will discuss prognostic factors informing treatment, the promising results of recent trials in new therapeutic approaches, and the implications of treatment resistance in therapeutic selection for MCL.
Target Audience
Hematology/oncology fellows, attending faculty, and other health care professionals involved in the treatment of patients with mantle cell lymphoma (MCL).
Learning Objectives
1.) Identify clinical and biological prognostic factors that can guide treatment decision making for older adults with MCL
2.) Evaluate emerging data on targeted therapeutic approaches for treatment-naive and relapsed/refractory MCL and their applicability to older adults
3.) Assess mechanisms of resistance to targeted therapies for MCL and their implications for treatment selection
micro teaching on communication m.sc nursing.pdfAnurag Sharma
Microteaching is a unique model of practice teaching. It is a viable instrument for the. desired change in the teaching behavior or the behavior potential which, in specified types of real. classroom situations, tends to facilitate the achievement of specified types of objectives.
Ethanol (CH3CH2OH), or beverage alcohol, is a two-carbon alcohol
that is rapidly distributed in the body and brain. Ethanol alters many
neurochemical systems and has rewarding and addictive properties. It
is the oldest recreational drug and likely contributes to more morbidity,
mortality, and public health costs than all illicit drugs combined. The
5th edition of the Diagnostic and Statistical Manual of Mental Disorders
(DSM-5) integrates alcohol abuse and alcohol dependence into a single
disorder called alcohol use disorder (AUD), with mild, moderate,
and severe subclassifications (American Psychiatric Association, 2013).
In the DSM-5, all types of substance abuse and dependence have been
combined into a single substance use disorder (SUD) on a continuum
from mild to severe. A diagnosis of AUD requires that at least two of
the 11 DSM-5 behaviors be present within a 12-month period (mild
AUD: 2–3 criteria; moderate AUD: 4–5 criteria; severe AUD: 6–11 criteria).
The four main behavioral effects of AUD are impaired control over
drinking, negative social consequences, risky use, and altered physiological
effects (tolerance, withdrawal). This chapter presents an overview
of the prevalence and harmful consequences of AUD in the U.S.,
the systemic nature of the disease, neurocircuitry and stages of AUD,
comorbidities, fetal alcohol spectrum disorders, genetic risk factors, and
pharmacotherapies for AUD.
Tom Selleck Health: A Comprehensive Look at the Iconic Actor’s Wellness Journeygreendigital
Tom Selleck, an enduring figure in Hollywood. has captivated audiences for decades with his rugged charm, iconic moustache. and memorable roles in television and film. From his breakout role as Thomas Magnum in Magnum P.I. to his current portrayal of Frank Reagan in Blue Bloods. Selleck's career has spanned over 50 years. But beyond his professional achievements. fans have often been curious about Tom Selleck Health. especially as he has aged in the public eye.
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Introduction
Many have been interested in Tom Selleck health. not only because of his enduring presence on screen but also because of the challenges. and lifestyle choices he has faced and made over the years. This article delves into the various aspects of Tom Selleck health. exploring his fitness regimen, diet, mental health. and the challenges he has encountered as he ages. We'll look at how he maintains his well-being. the health issues he has faced, and his approach to ageing .
Early Life and Career
Childhood and Athletic Beginnings
Tom Selleck was born on January 29, 1945, in Detroit, Michigan, and grew up in Sherman Oaks, California. From an early age, he was involved in sports, particularly basketball. which played a significant role in his physical development. His athletic pursuits continued into college. where he attended the University of Southern California (USC) on a basketball scholarship. This early involvement in sports laid a strong foundation for his physical health and disciplined lifestyle.
Transition to Acting
Selleck's transition from an athlete to an actor came with its physical demands. His first significant role in "Magnum P.I." required him to perform various stunts and maintain a fit appearance. This role, which he played from 1980 to 1988. necessitated a rigorous fitness routine to meet the show's demands. setting the stage for his long-term commitment to health and wellness.
Fitness Regimen
Workout Routine
Tom Selleck health and fitness regimen has evolved. adapting to his changing roles and age. During his "Magnum, P.I." days. Selleck's workouts were intense and focused on building and maintaining muscle mass. His routine included weightlifting, cardiovascular exercises. and specific training for the stunts he performed on the show.
Selleck adjusted his fitness routine as he aged to suit his body's needs. Today, his workouts focus on maintaining flexibility, strength, and cardiovascular health. He incorporates low-impact exercises such as swimming, walking, and light weightlifting. This balanced approach helps him stay fit without putting undue strain on his joints and muscles.
Importance of Flexibility and Mobility
In recent years, Selleck has emphasized the importance of flexibility and mobility in his fitness regimen. Understanding the natural decline in muscle mass and joint flexibility with age. he includes stretching and yoga in his routine. These practices help prevent injuries, improve posture, and maintain mobilit
Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
2. EMMETROPIA
The parallel rays of light coming from infinity are focused at
sensitive layer of retina with the accommodation at being at
rest
Good visual acuity (6/6 or better) at 6 m testing distance
If accommodation amplitude is adequate ,equally good visual
acuity at the near testing distance of 40 cm
Axial length =24mm
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4. Refractive sate of eye
Corneal refractive power
Anterior chamber depth
Lens refracting power
Axial length
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5. AMETROPIA
The parallel rays of light coming from infinity are focused
either in front or behind the sensitive layer of retina in one or
both meridian when accommodation at rest
Refractive Status And Aging
o Hyperopic (infant)
o Emmetropic (10 years old)
o Myopic (25 years old)
o Hyperopic (60 years old)
o Less hyperopic (80 years
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7. HYPERMETROPIA
The parallel rays of light coming from infinity are focused
behind the sensitive layer of retina with accommodation
being at rest
Back focal point is behind retina
It is also called far/long sightedness
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8. AETIOLOGY
Axial hypermetropia:-the antero-posterior diameter of
eye is too short & retina is too near to optical system,1mm-3D
Curvature hypermetropia:-curvature of cornea or
lens may be too small,1mm-6D
Index hypermetropia:- due to change in refractive
index of lens ,aqueous humour ,vitreous humour or diabetes
Positional hypermetropia:-if the crystalline lens is
dislocated backwards
Absence of crystalline lens:- aphakia
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9. CLINICAL TYPES
simple hypermetropia:- normal biological variations in
development of eyeball
Pathological hypermetropia:-outside biological
variations of development ,include:-
Senile :- curvature &index
Positional
Aphakia
Functional hypermetropia:-paralysis of
accommodation -third nerve paralysis &internal
ophthaloplegia
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10. OPTICAL CONDITION
Parallel rays of light come to a behind the retina
Parallel rays of light are brought to a focus upon the
retina by increasing the refractivity by
accommodation .the normal lens becomes more
convex
Parallel rays of light are brought to a focus upon the
retina by increasing refractivity by a convex
spectacles lens .the degree of hypermetropia is given
by power lens
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11. ACCOMMODATION IN HP
Latent hypermetropia (LH):-
This is a condition in which all or part of hypermetropia is compensated or corrected by
the tonicity of the ciliary muscle.
Latent hypermetropia becomes manifest when the amplitude of accommodation
decreased significantly.
Manifest hypermetropia (MH): It has two parts.
Facultative hypermetropia (fH) – The amount of hypermetrropia that can be
corrected by the effort of accommodation.
Absolute hypermetropia (aH) – T he amount of hypermetropia that can not be
corrected by the effort of accommodation.
Thus the total amount of hypermetropia (tH) is the sum of
latent and manifest hypermetropia.
tH=LH+MH=LH+(fH+aH).
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12. CLINICAL PATHOLOGY
Cornea is small, AC shallow ,closed –angle glaucoma
Administration of dilated drugs to dilate pupil
Developmental aberrations such as colobomata
,microphthalmos
The retina appears to have peculiar sheen, optic neuritis,
vascular reflex
Face asymmetrical ,dislocation of macula, divergent squint
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13. CLINICAL FEATURES
SYMPTOMS:-
Asymtomatic
Asthenopic :-tiredness, frontal or frontotemporal headache, watering, mild
photophobia
Defective vision with asthenopic symptoms
Defective vision only
The effect of ageing on vision
Intermitted sudden blurring of vision
Crossed – eye sensation
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14. CONT
Signs:-
Size of eyeball may be normal or small as whole
Cornea may smaller than normal
Variation in visual acuity
Abnormal branching of vessels , swelling of disc, retinal shine is
more than normal
A –scan ultrasonography may reveal a short anteroposterior length
of eyeball
10/12/2016 SAH SURENDRA
16. TREATMENT
OPTICAL TREATMENT:-CONVEX(PLUS) LENS
Total amount of HP-complete cycloplegic
Manifest refractive error is small-1D or less
Astigmatism should be fully corrected
Exophoria -1-2D less
Accommodative convergent squint /amblyopia-full correction
Surgery for Hp:-
Thermokeratoplasty
The use of excimer &Holmium lasers is under investigation
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17. APHAKIA
Condition in which absence of crystaline lens from
pupillary area of eye
CAUSES:-
Congenital absence of lens
Surgical aphakia occurring after removal of lens
Aphakia due to absorption of lens matter
Traumatic extrusion of lens
Posterior dislocation of lens in vitreous
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18. OPTICS OF APHAKIA
Changes in cardinal data of the eye
Image formation in the aphakic eye
Visual acuity in aphakia
Accommodation in aphakia
Binocular vision
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19. CLINICAL FEATURES
SYMPTOMS:-
Defective vision for near & distance
Erythropsia and cyanopsia – due to entry of infrared and ultraviolet rays in
the absence of the crystalline lens
SIGNS:-
Limbal scar in case of surgical aphakia
Deep anterior chamber
Iridodonesis – tremulousness of the iris due to loss of support of
lens
Jet Black pupil
Loss of 3rd and 4th purkinje images
Fundus examination reveals a small hypermetropic fundus
Retinoscopy shows high hypermetropia
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20. TREATMENT
SPECTACLES:-
Spectacles should be prescribed with about +10D lens for
correction of aphakia
It should also include correction for surgical astigmatism and
+3-4D for near vision
Nowadays spectacles are not preferred for use in aphakia due
to its many disadvantage
Advantages of using spectacles in aphakia
• Easy to use
• No complications
• cheap
10/12/2016 SAH SURENDRA
21. CONT…..
Disadvantages
The images are magnified – about 30% – hence not useful in unilateral
aphakia as it causes diplopia
• The field of vision in decreased considerably
• Spherical and chromatic aberration of high power lenses
• Roving ring scotoma (Jack in the box phenomenon)
• Prismatic effect of the thick lenses
• High power are cosmetically not acceptable
Contact lenses
Advantages over spectacles:
• Produces less magnification
• Better field of vision
• Less chromatic and spherical aberration
• No prismatic effect
• Cosmetically more acceptable
10/12/2016 SAH SURENDRA
22. CONT……
Intraocular lens implantation
• The lens can be implanted in the capsular bag or in the anterior chamber
• It eliminates most of the disadvantages associated with the use of spectacles
or contact lenses
• Disadvantage include the complications associated with surgery
Refractive surgery
Keratophakia
– a lenticule prepared from the donor cornea is placed within the
lamellae of the patient’s cornea
Epikeratophakia
– a lenticule prepared form the donor cornea is stitched to the
patients cornea after removing the epithelium
Hyperopic Lasik
10/12/2016 SAH SURENDRA
23. PSEUDOPHAKIA
The condition of aphakia when corrected with
implantation of an IOL
Refractive status:-
Emmetropia
Consective myopia
Consective ypermetropia
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24. SIGNS
Surgical limbal scar may be seen
Anterior chamber is slightly deeper than normal
Mild iridodonesis of iris may be demonstrated
Purkinje image test shows fours images
Pupil is blackish in colour but when light thrown in pupillary
area shining reflexes are observed
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25. MYOPIA
Shortsightness
Lengthening of posterior part of eye
The parallel rays of light coming from infinity are focused in
front of sensitive layer of retina when accommodation is at
rest
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26. AETIOLOGY
Axial myopia:-the antero-posterior diameter of eye is too
long & retina is too far from optical system
Curvature myopia:-the curvature of cornea or lens may
be too great
Positional myopia:-if lens is dislocated forwards
Index myopia:-R.i of aqueous is too high ,vitreous is too
low or lens is too high
Myopia due to excessive accommodation occurs in patients
with spasm of accommodation
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27. OPTICS OF MYOPIA
Optical system of a myopic eye is too powerful for its axial length
Image of distance object on retina is made up of circles of diffusion
formed by divergent beam
Far point of the myopic eye is a finite point in front of the eye
Nodal point in myopic eye is further away from retina
Angle alpha of eye may be negative
Accommodation in uncorrected myopes is not developed normally
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28. CLINICAL VARIETIES OF MYOPIA
Congenital myopia
Simple or developmental myopia
Pathological or degenerative myopia
Acquired myopia
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29. CONGENTICAL MYOPIA
The child is born with elongated eyes
The refraction may be upto -10D
Typical fundus changes are seen
Progression is rare
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30. SIMPLE MYOPIA
Commonest clinical type
Does not progress much after the adolescence
May be upto -5 to -6D
No degenerative changes are seen in the fundus, although
peripheral retinal degeneration may be seen in later life
Associated with good vision with a good prognosis
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31. PATHOLOGICAL MYOPIA
It is also called progressive or degenerative myopia
Myopia appears in childhood &increasing steadily with age upto 25
years or beyond
The final amount of myopia may be -15Dto -25D or more
There are typical degenerative changes in the fundus
Strongly hereditary & more common in female
Prognosis is usually poor
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32. ACQUIRED MYOPIA
CAUSES :-
Index myopia
Curvatural myopia
Positional myopia
Consecutive myopia
Pseudomyopia
Space myopia
Night myopia
Drug –induced myopia
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33. CLINICAL FEATURS
SYMPTOMS:-
Impaired distance vision
Eye –strain
Exophoria or divergent squint
Delayed dark adaptation
Sudden loss of vision
Black floaters
SIGNS:-
Prominent eyeball, large cornea, deep anterior chamber& a
large pupil, apparent convergent squint, degeneration of
vitreous
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34. COMPLICATION OF MYOPIA
Retinal tear –vitreous haemorrhage
Retinal detachment degeneration of the
vitreous
Primary open –angle glaucoma
Posterior cortical cataract
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35. TREATMENT
Divergent or minus lenses in spectacles or
contact lenses
Cycloplegics
Surgery
Refractive surgery
General hygiene
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36. ASTIGMATISM
Condition of refraction wherein a point focus of light cannot
be formed upon the retina
Caused by unequal refraction of light in different meridians
Curvatures of both axes are unequal & too small is HM ASG &
both unequal & too great is MP ASG
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37. AETIOLOGY
Corneal astigmastism :-abnormalites of curvature of
cornea
Lenticular astigmatism:-
• Small amount of curvature astigmatism due to congenital abnormalities of
curvature of lens
• Position astigmatism due to congenital or traumatic subluxation of lens
• Refractive index of lens in different median
Retinal astigmatism due to oblique placement
of macula
10/12/2016 SAH SURENDRA
38. TYPES OF ASTIGMATISM
REGULAR ASTIGMASTIM:-Axis & angle
With –the –rule :-two principal meridian are placed at right
angles to one another but vertical meridian is more curve
than horizontal
Against –the rule :-horizontal meridian is more curved than
vertical meridian
Oblique:-two principal meridian are not horizontal & vertical
@ right angle to one another
Bi-oblique:-two principal meridian are not @ right angle to
each other
10/12/2016 SAH SURENDRA
39. CONT..
REGULAR ASTIGMATISM:-REFRACTIVE TYPES
Simple:-one meridian focus in front & other behind retina
Compound :-ray of light in both meridian are focused either
in front or behind retina
Mixed:-rays of light in one meridian are focused in front &
other behind retina
10/12/2016 SAH SURENDRA
40. OPTICS OF REGULAR ASTIGMATISM
Parallel rays of light are not focused on a point but form two
focal lines
The configuration of rays refracted through astigmatism
surface is called sturm’s conoid& distance between two lines
is called focal interval of sturm
The length of this focal interval is a measure of degree of
astigmatism
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