Rabies is a viral disease that causes acute inflammation of the brain. It is transmitted to humans through bites or scratches from infected animals like dogs. Once symptoms develop, rabies is nearly always fatal. Treatment involves thorough wound cleaning, rabies immunoglobulin, and rabies vaccines. Prevention focuses on vaccinating animals and using post-exposure prophylaxis for people exposed through bites. There is no cure for rabies once symptoms appear.
This ppt is About Rabies epidemiology and treatment .
This is done by using Park book 24th edition of PSM .
This presentation is presented in academics of Master of public health in Christian medical college .
One more Important thing is that that zareb regime (intramuscular ) is not practiced . We try to make this ppt lucid. and the statistics is used in the presentation is upto 27 june 2018
This ppt is About Rabies epidemiology and treatment .
This is done by using Park book 24th edition of PSM .
This presentation is presented in academics of Master of public health in Christian medical college .
One more Important thing is that that zareb regime (intramuscular ) is not practiced . We try to make this ppt lucid. and the statistics is used in the presentation is upto 27 june 2018
This ppt contains all information about epidemiology of Diptheria. It is useful for students of medical field learning preventive and social medicine, Swasthavritta (Ayurved), nursing and everyone who is interested in knowing about it.
Rabies is a viral disease that causes acute encephalitis
(inflammation of the brain) in warm blooded animals. Rabies is a zoonotic disease (a disease that is transmitted to humans from animals) that is caused by a virus
NDWC Chennai 2013 - The One Health approach towards Rabies elimination in Asi...Dogs Trust
Dr Abdul Rahman's presentation on ''The One Health approach towards Rabies elimination in Asia'' at the National Dog Welfare Conference, Chennai India 27th and 28th February 2013.
Bovine tuberculosis epidemiology & control in indiaBhoj Raj Singh
Tuberculosis in India is in hyperendemic state both in human and animals. No DOTS can help in control of human tuberculosis unless tuberculosis is controlled in animals. Control of tuberculosis in animals is a far reacheachable dream in India and thus the Tuberculosis will persist in India till the dooms day.
This document is the summary of my thesis work carried out at Indian Veterinary Research Institute, Izatnagar, India (2001)
I am sorry, I cant provide a download option for this
This ppt contains all information about epidemiology of Diptheria. It is useful for students of medical field learning preventive and social medicine, Swasthavritta (Ayurved), nursing and everyone who is interested in knowing about it.
Rabies is a viral disease that causes acute encephalitis
(inflammation of the brain) in warm blooded animals. Rabies is a zoonotic disease (a disease that is transmitted to humans from animals) that is caused by a virus
NDWC Chennai 2013 - The One Health approach towards Rabies elimination in Asi...Dogs Trust
Dr Abdul Rahman's presentation on ''The One Health approach towards Rabies elimination in Asia'' at the National Dog Welfare Conference, Chennai India 27th and 28th February 2013.
Bovine tuberculosis epidemiology & control in indiaBhoj Raj Singh
Tuberculosis in India is in hyperendemic state both in human and animals. No DOTS can help in control of human tuberculosis unless tuberculosis is controlled in animals. Control of tuberculosis in animals is a far reacheachable dream in India and thus the Tuberculosis will persist in India till the dooms day.
This document is the summary of my thesis work carried out at Indian Veterinary Research Institute, Izatnagar, India (2001)
I am sorry, I cant provide a download option for this
overview of rabies and rabies post exposure administration.pptxBirhanu Hurisa
Rabies is neglected disease with 100% case fatality. Rabies kills more than 55,000 people each year in which 99% of the deaths are from developing countries. Rabies has been recognized in Ethiopia for centuries, and the disease has become endemic since the early 17th century. By estimation, 2771 to 10,000 people die of rabies each year in Ethiopia.
The only vaccine available for human rabies post-exposure prophylaxis is the obsolete sheep brain-based Fermi vaccine, and has been manufactured since early 1950s in Ethiopia. According to Ethiopian Ministry of Health, the annual performance report, the maximum manufacturing capacity for the Nervous Tissue Vaccine (NTV) of the country is about 32,000 doses, which is far below demand. The NTV is immunogenic, more reactogenic, life threatening and caused neurological adverse reactions in 0.3-0.8 per 1000 vaccinated people.
Rabies is neglected disease with 100% case fatality. Rabies kills more than 55,000 people each year in which 99% of the deaths are from developing countries. Rabies has been recognized in Ethiopia for centuries, and the disease has become endemic since the early 17th century. By estimation, 2771 to 10,000 people die of rabies each year in Ethiopia.
The only vaccine available for human rabies post-exposure prophylaxis is the obsolete sheep brain-based Nervous Tissue vaccine (Fermi vaccine) and has been manufactured since early 1950s in Ethiopia. According to Ethiopian Ministry of Health, the annual performance report, the maximum manufacturing capacity for the Nervous Tissue Vaccine (NTV) of the country is about 32,000 doses, which is far below demand. The NTV is immunogenic, more reactogenic, life threatening and caused neurological adverse reactions in vaccinated people.
An introduction to Rhabdoviridae.Rabies is a viral disease that causes acute inflammation of the brain in humans and other mammals. Early symptoms can include fever and tingling at the site of exposure. These symptoms are followed by one or more of the following symptoms: violent movements, uncontrolled excitement, fear of water, an inability to move parts of the body, confusion, and loss of consciousness. Once symptoms appear, the result is nearly always death. The time period between contracting the disease and the start of symptoms is usually one to three months; however, this time period can vary from less than one week to more than one year. The time is dependent on the distance the virus must travel to reach the central nervous system.
RABIES-A fatal but preventable viral disease is explained in detail (with exclusive pictures) in this PowerPoint presentation.
It also includes the "updates on prevention and control strategy" and "Zero by 2030-Rabies Elimination Strategy"
This was presented at seminar hall, Department of Community Medicine, IMS, Banaras Hindu University as a part of PG seminar.
(The video by Lancet included in this may not be played in this slideshare platform...one can access youtube for the same)
Rabies Clinical Disease Mangement By Waqas Siddiqe.pptxwaqassiddiqe
Rabies pathophysiology and disease management includes therapies and adverse effects and drug interactions.
Content taken from published articles and books valid and accurate content.
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Poliovirus is a picornaviridae. it has 3 wildtypes, Wildtype 2 has been eradicated from the world. All countries have been declared polio free except Pakistan, Afghanistan and Nigeria. Global Polio Eradication Initiative has been discussed.
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CRISPR-Cas9, a revolutionary gene-editing tool, holds immense potential to reshape medicine, agriculture, and our understanding of life. But like any powerful tool, it comes with ethical considerations.
Unveiling CRISPR: This naturally occurring bacterial defense system (crRNA & Cas9 protein) fights viruses. Scientists repurposed it for precise gene editing (correction, deletion, insertion) by targeting specific DNA sequences.
The Promise: CRISPR offers exciting possibilities:
Gene Therapy: Correcting genetic diseases like cystic fibrosis.
Agriculture: Engineering crops resistant to pests and harsh environments.
Research: Studying gene function to unlock new knowledge.
The Peril: Ethical concerns demand attention:
Off-target Effects: Unintended DNA edits can have unforeseen consequences.
Eugenics: Misusing CRISPR for designer babies raises social and ethical questions.
Equity: High costs could limit access to this potentially life-saving technology.
The Path Forward: Responsible development is crucial:
International Collaboration: Clear guidelines are needed for research and human trials.
Public Education: Open discussions ensure informed decisions about CRISPR.
Prioritize Safety and Ethics: Safety and ethical principles must be paramount.
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Deep Leg Vein Thrombosis (DVT): Meaning, Causes, Symptoms, Treatment, and Mor...The Lifesciences Magazine
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Alongside the 77th World Health Assembly in Geneva on 28 May 2024, we launched the second version of our Index, allowing us to track progress and give new insights into what needs to be done to keep populations healthier for longer.
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Dr Hans Groth, Chairman of the Board, World Demographic & Ageing Forum
Professor Ilona Kickbusch, Founder and Chair, Global Health Centre, Geneva Graduate Institute and co-chair, World Health Summit Council
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Defecation
Normal defecation begins with movement in the left colon, moving stool toward the anus. When stool reaches the rectum, the distention causes relaxation of the internal sphincter and an awareness of the need to defecate. At the time of defecation, the external sphincter relaxes, and abdominal muscles contract, increasing intrarectal pressure and forcing the stool out
The Valsalva maneuver exerts pressure to expel faeces through a voluntary contraction of the abdominal muscles while maintaining forced expiration against a closed airway. Patients with cardiovascular disease, glaucoma, increased intracranial pressure, or a new surgical wound are at greater risk for cardiac dysrhythmias and elevated blood pressure with the Valsalva maneuver and need to avoid straining to pass the stool.
Normal defecation is painless, resulting in passage of soft, formed stool
CONSTIPATION
Constipation is a symptom, not a disease. Improper diet, reduced fluid intake, lack of exercise, and certain medications can cause constipation. For example, patients receiving opiates for pain after surgery often require a stool softener or laxative to prevent constipation. The signs of constipation include infrequent bowel movements (less than every 3 days), difficulty passing stools, excessive straining, inability to defecate at will, and hard feaces
IMPACTION
Fecal impaction results from unrelieved constipation. It is a collection of hardened feces wedged in the rectum that a person cannot expel. In cases of severe impaction the mass extends up into the sigmoid colon.
DIARRHEA
Diarrhea is an increase in the number of stools and the passage of liquid, unformed feces. It is associated with disorders affecting digestion, absorption, and secretion in the GI tract. Intestinal contents pass through the small and large intestine too quickly to allow for the usual absorption of fluid and nutrients. Irritation within the colon results in increased mucus secretion. As a result, feces become watery, and the patient is unable to control the urge to defecate. Normally an anal bag is safe and effective in long-term treatment of patients with fecal incontinence at home, in hospice, or in the hospital. Fecal incontinence is expensive and a potentially dangerous condition in terms of contamination and risk of skin ulceration
HEMORRHOIDS
Hemorrhoids are dilated, engorged veins in the lining of the rectum. They are either external or internal.
FLATULENCE
As gas accumulates in the lumen of the intestines, the bowel wall stretches and distends (flatulence). It is a common cause of abdominal fullness, pain, and cramping. Normally intestinal gas escapes through the mouth (belching) or the anus (passing of flatus)
FECAL INCONTINENCE
Fecal incontinence is the inability to control passage of feces and gas from the anus. Incontinence harms a patient’s body image
PREPARATION AND GIVING OF LAXATIVESACCORDING TO POTTER AND PERRY,
An enema is the instillation of a solution into the rectum and sig
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3. DEFINITION
Rabies is a viral disease
that causes
acute encephalitis (inflamm
ation of the brain) in warm-
blooded animals
Rabies is a zoonotic
disease (a disease that is
transmitted to humans from
animals) that is caused by a
virus
4.
5. INTRODUCTION
The disease infects domestic and wild
animals, and is spread to people through
close contact with infected saliva via bites or
scratches.
Dogs are the source of 99% of human rabies
deaths
Once symptoms of the disease develop,
rabies is nearly always fatal.
8. SOURCE OF INFECTION
The source of infection to man is the saliva of
rabid animals.
In dogs & cats, the virus may be present in
the saliva for 3-4 days before the clinical
onset & during the course of illness till death.
Cause > 90% of the
Human cases
3 – 5% of Human
cases
9. INCUBATION PERIOD
It is highly variable in man, commonly 3-8
weeks following exposure.
The closer the bite to the brain, the shorter the
incubation.
Rabies virus travels 1 cm per day.
10.
11. PATHOGENESIS
Live virus Epidermis, Mucus membrane
Peripheral nerve
CNS ( gray matter )
Other tissue (salivary glands,…)
centripetally
centrifugally
12.
13. SIGN & SYMPTOMS
Bizarre behavior.
Agitation
Seizures.
Difficulty in drinking.
Patients will be able to eat solids
Afraid of water - Hydrophobia.
Even sight or sound of water
disturbs the patient.
But suffer with intense thirst.
Spasms of Pharynx produces choking
Death in 1 -6 days.
Respiratory arrest / Death / Some may
survive.
14. CONTI….
Headache, fever, sore throat
Nervousness, confusion
Pain or tingling at the site of the bite
Hallucinations
Seeing things that are not really there
Hydrophobia
“Fear of water" due to spasms in the throat
Paralysis
Unable to move parts of the body
Coma and death
19. PRE EXPOSURE PROPHYLAXIS
Provided to subjects at risk before
occupational or vocational exposure
to rabies.
Subjects include diagnosticians,
laboratory & vaccine workers,
veterinarians, cavers, etc.
Simplifies post exposure
management.
Only vaccines used.
20. PEP (POST EXPOSURE PROPHYLAXIS)
Provided to subjects after rabies
exposure.
Consists of wound care, rabies immune
globulin, and vaccine.
Cleansing
Chemical Treatment
Suturing
Anti-Rabies Serum
Antibiotics & anti-tetanus measure
Observe the animal for 10 days.
21.
22. CONT…
Wash lesions well with soap
and water (tetanus booster)
Infiltrate rabies immune
globulin (20 IU/kg) into and
around the margin of the
bites.
Administer vaccine on days
0,3,7,14, and 28. (90)
29. TREATMENT
POST-EXPOSURE TREATMENT (PET)
A. LOCAL WOUND TREATMENT
- Wash with soap/detergent and water
preferably for 10 mins.
- Apply alcohol, povidone iodine/ any
antiseptic
- Anti-Tetanus
*Avoid suturing wounds
*Don’t apply ointment, cream/ wound dressing
30. ANTIMICROBIAL
- Amoxicillin
- Cloxacillin
- Cefuroxime
*For those instances where there’s no
obvious signs of infection( Amoxicillin as
prophylaxis )
***Educate the public simple local wound
treatment & warn not to use procedures
that may further contaminate the wounds
32. VACCINATION
(INTRADERMAL SCHEDULE)
Day of
Immunization
PVRV/PCECV Site
DAY 0 0.1 ml L & R deltoids/
anterolateral thighs of
infants
DAY 3 0.1 ml L & R deltoids/
anterolateral thighs of
infants
DAY 7 0.1 ml L & R deltoids /
anterolateral thighs of
infants
DAY 28/30 0.1 ml L & R deltoids/
anterolateral thighs of
infants
33. INTRAMUSCULAR SCHEDULE
Day of
Immunization
PVRV PCECV Site
Day 0 0.5 ml 1.0 ml One deltoid/
anterolateral
thigh of infants
Day 3 0.5 ml 1.0 ml Same
Day 7 0.5 ml 1.0 ml Same
Day 14 0.5 ml 1.0 ml Same
Day 28 0.5 ml 1.0 ml same
34. MANAGEMENT OF RABIES PATIENT
Once symptoms start, treatment should center
on comfort care, using sedation & avoidance of
intubation & life support measures once
diagnosis is certain
1. MEDICATIONS
a. Diazepam
b. Midazolam
c. Haloperidol + Dipenhydramine
35. 2. SUPPORTIVE CARE
- Pts w/ confirmed rabies should receive
adequate sedation & comfort care in an
appropriate medical facility.
a. Once rabies diagnosis has been confirmed,
invasive procedures must be avoided
b. Provide suitable emotional and physical
support
c. Discuss & provide important info. to
relatives concerning transmission of dse. &
indication for PET of contacts
d. Honest gentle communication concerning
prognosis should be provided to relatives of
pt
36. 3. INFECTION CONTROL
a. Patient should be admitted in a quiet,
draft-free, isolation room
b. HLCR workers & relatives in contact w/
pt should wear proper personal
protective equipment (gown, gloves,
mask, goggles)
4. DISPOSAL OF DEAD BODIES