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MCQs Revision Biochemistry for
NEET Jan - 2018
DR. SMILY PRUTHI PAHWA
(MD-BIOCHEMISTRY)
See video on Youtube……….
By Tulip academy of Medical sciences
Or
See video in mobile app – Search : tulip academy of medical sciences
Watch Video on YouTube :
https://www.youtube.com/watch?v=D0raz0zTkaA
Our WebSite https://tamsmed.com
Q1 . Kinks in alpha structure are formed by
which amino acid :
1) Glycine
2) Lysine
3) Methionine
4) Glutamate
Our WebSite https://tamsmed.com
Q1 . Kinks in alpha structure are formed by
which amino acid :
1) Glycine
2) Lysine
3) Methionine
4) Glutamate
Our WebSite https://tamsmed.com
Q2. A patient’s lipid profile was done. His TGs
levels were found to be 200 mg/dl, Total
cholesterol was 300 mg/dl & HDL = 40 mg/dl.
Calculate LDL:
a. 220
b. 260
c. 60
d. 100
Our WebSite https://tamsmed.com
FRIEDWALD’S EQUATION
• Total cholesterol = HDL + LDL + VLDL
Our WebSite https://tamsmed.com
FRIEDWALD’S EQUATION
• Total cholesterol = HDL + LDL + VLDL
• LDL = Total cholesterol – HDL – TG/5
= 300 – 40 – (200/5)
= 300 -40 -40
= 300-80
= 220 mg/dl
Our WebSite https://tamsmed.com
• In mmol/L  divide by 2.2
• LDL chol = total chol – HDL chol – TG/2.2
Our WebSite https://tamsmed.com
Q2. A patient’s lipid profile was done. His TGs
levels were found to be 200 mg/dl, Total
cholesterol was 300 mg/dl & HDL = 40 mg/dl.
Calculate LDL:
a. 220
b. 260
c. 60
d. 100
Our WebSite https://tamsmed.com
Q3. Lipoprotein lipase is activated by:
a. Apo C-I
b. Apo C-II
c. Apo C-III
d. Apo A-I
Our WebSite https://tamsmed.com
Q3. Lipoprotein lipase is activated by:
a. Apo C-I
b. Apo C-II
c. Apo C-III
d. Apo A-I
Our WebSite https://tamsmed.com
Role of Apoproteins in Lipoproteins
1. Structural Role
2. Enzyme Cofactors :
Apo C-II  activate Lipoprotein Lipase
Apo C-III  inhibits Lipoprotein Lipase
Apo A-I  activate LCAT
Apo A-II  inhibits LCAT
3. Ligands for Receptors
Our WebSite https://tamsmed.com
Ligands for Receptors :
• Apo E  ligand for remnants
• Apo B100 & Apo E  LDL-Receptors
• Apo A1  HDL-Receptors
Our WebSite https://tamsmed.com
Q4. Tom cat urine smell is seen in:
a) Hawkinsinuria
b) Multiple Carboxylase defect
c) G6PD
d) Phenylketonuria
Our WebSite https://tamsmed.com
Q4. Tom cat urine smell is seen in:
a) Hawkinsinuria
b) Multiple Carboxylase defect
c) G6PD
d) Phenylketonuria
Our WebSite https://tamsmed.com
• Tom cat urine odour - Multiple Carboxylase
defect
• Mousy/Musty odour - Phenylketonuria
• Burnt sugar like odour - MSUD
• Swimming pool odour - Hawkinsinuria
Our WebSite https://tamsmed.com
Q5. Odd chain fatty acids can form glucose by
which pathway ?
a) Propionyl CoA
b) Glycerol
c) Acetyl CoA entering TCA cycle
d) Lactate
Our WebSite https://tamsmed.com
Q5. Odd chain fatty acids can form glucose by
which pathway ?
a) Propionyl CoA
b) Glycerol
c) Acetyl CoA entering TCA cycle
d) Lactate
Our WebSite https://tamsmed.com
Our WebSite https://tamsmed.com
Q6. Muscle cannot
maintain blood glucose
because of deficiency of :
a) Glucose-6-
phosphatase
b) Glycogen
phosphorylase
c) Hexokinase
d) Phospho-gluco-
mutase
Q7. Muscle cannot
make use of glycogen
because of deficiency
of :
a) Glucose-6-
phosphatase
b) Glycogen
phosphorylase
c) Hexokinase
d) Phospho-gluco-
mutase
Our WebSite https://tamsmed.com
Q6. Muscle cannot
maintain blood glucose
because of deficiency of :
a) Glucose-6-
phosphatase
b) Glycogen
phosphorylase
c) Hexokinase
d) Phospho-gluco-
mutase
Q7. Muscle cannot
make use of glycogen
because of deficiency
of :
a) Glucose-6-
phosphatase
b) Glycogen
phosphorylase
c) Hexokinase
d) Phospho-gluco-
mutase
Our WebSite https://tamsmed.com
Q8. Malate shuttle is important in :
a) Glycogenesis
b) Glycolysis
c) Gluconeogenesis
d) Glycogenolysis
Our WebSite https://tamsmed.com
Q8. Malate shuttle is important in :
a) Glycogenesis
b) Glycolysis
c) Gluconeogenesis
d) Glycogenolysis
Our WebSite https://tamsmed.com
Q9. During prolonged starvation, rate of
gluconeogenesis depends on :
a) Increased alanine levels in liver
b) Decreased cGMP levels in liver
c) ADP in liver
d) Decreased essential fatty acids in liver
Our WebSite https://tamsmed.com
Q9. During prolonged starvation, rate of
gluconeogenesis depends on :
a) Increased alanine levels in liver
b) Decreased cGMP levels in liver
c) ADP in liver
d) Decreased essential fatty acids in liver
Our WebSite https://tamsmed.com
Q10. Which of the following is a Pallindrome ?
a) GGCC
b) GTCC
c) TAAT
d) TTCG
Our WebSite https://tamsmed.com
Q10. Which of the following is a Pallindrome ?
a) GGCC
b) GTCC
c) TAAT
d) TTCG
Our WebSite https://tamsmed.com
Download App for for more videos
https://tamsmed.com/biochemistry.php
Our WebSite https://tamsmed.com

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Neet PG MCQ Biochemistry 2018

  • 1. MCQs Revision Biochemistry for NEET Jan - 2018 DR. SMILY PRUTHI PAHWA (MD-BIOCHEMISTRY) See video on Youtube………. By Tulip academy of Medical sciences Or See video in mobile app – Search : tulip academy of medical sciences Watch Video on YouTube : https://www.youtube.com/watch?v=D0raz0zTkaA Our WebSite https://tamsmed.com
  • 2. Q1 . Kinks in alpha structure are formed by which amino acid : 1) Glycine 2) Lysine 3) Methionine 4) Glutamate Our WebSite https://tamsmed.com
  • 3. Q1 . Kinks in alpha structure are formed by which amino acid : 1) Glycine 2) Lysine 3) Methionine 4) Glutamate Our WebSite https://tamsmed.com
  • 4. Q2. A patient’s lipid profile was done. His TGs levels were found to be 200 mg/dl, Total cholesterol was 300 mg/dl & HDL = 40 mg/dl. Calculate LDL: a. 220 b. 260 c. 60 d. 100 Our WebSite https://tamsmed.com
  • 5. FRIEDWALD’S EQUATION • Total cholesterol = HDL + LDL + VLDL Our WebSite https://tamsmed.com
  • 6. FRIEDWALD’S EQUATION • Total cholesterol = HDL + LDL + VLDL • LDL = Total cholesterol – HDL – TG/5 = 300 – 40 – (200/5) = 300 -40 -40 = 300-80 = 220 mg/dl Our WebSite https://tamsmed.com
  • 7. • In mmol/L  divide by 2.2 • LDL chol = total chol – HDL chol – TG/2.2 Our WebSite https://tamsmed.com
  • 8. Q2. A patient’s lipid profile was done. His TGs levels were found to be 200 mg/dl, Total cholesterol was 300 mg/dl & HDL = 40 mg/dl. Calculate LDL: a. 220 b. 260 c. 60 d. 100 Our WebSite https://tamsmed.com
  • 9. Q3. Lipoprotein lipase is activated by: a. Apo C-I b. Apo C-II c. Apo C-III d. Apo A-I Our WebSite https://tamsmed.com
  • 10. Q3. Lipoprotein lipase is activated by: a. Apo C-I b. Apo C-II c. Apo C-III d. Apo A-I Our WebSite https://tamsmed.com
  • 11. Role of Apoproteins in Lipoproteins 1. Structural Role 2. Enzyme Cofactors : Apo C-II  activate Lipoprotein Lipase Apo C-III  inhibits Lipoprotein Lipase Apo A-I  activate LCAT Apo A-II  inhibits LCAT 3. Ligands for Receptors Our WebSite https://tamsmed.com
  • 12. Ligands for Receptors : • Apo E  ligand for remnants • Apo B100 & Apo E  LDL-Receptors • Apo A1  HDL-Receptors Our WebSite https://tamsmed.com
  • 13. Q4. Tom cat urine smell is seen in: a) Hawkinsinuria b) Multiple Carboxylase defect c) G6PD d) Phenylketonuria Our WebSite https://tamsmed.com
  • 14. Q4. Tom cat urine smell is seen in: a) Hawkinsinuria b) Multiple Carboxylase defect c) G6PD d) Phenylketonuria Our WebSite https://tamsmed.com
  • 15. • Tom cat urine odour - Multiple Carboxylase defect • Mousy/Musty odour - Phenylketonuria • Burnt sugar like odour - MSUD • Swimming pool odour - Hawkinsinuria Our WebSite https://tamsmed.com
  • 16. Q5. Odd chain fatty acids can form glucose by which pathway ? a) Propionyl CoA b) Glycerol c) Acetyl CoA entering TCA cycle d) Lactate Our WebSite https://tamsmed.com
  • 17. Q5. Odd chain fatty acids can form glucose by which pathway ? a) Propionyl CoA b) Glycerol c) Acetyl CoA entering TCA cycle d) Lactate Our WebSite https://tamsmed.com
  • 19. Q6. Muscle cannot maintain blood glucose because of deficiency of : a) Glucose-6- phosphatase b) Glycogen phosphorylase c) Hexokinase d) Phospho-gluco- mutase Q7. Muscle cannot make use of glycogen because of deficiency of : a) Glucose-6- phosphatase b) Glycogen phosphorylase c) Hexokinase d) Phospho-gluco- mutase Our WebSite https://tamsmed.com
  • 20. Q6. Muscle cannot maintain blood glucose because of deficiency of : a) Glucose-6- phosphatase b) Glycogen phosphorylase c) Hexokinase d) Phospho-gluco- mutase Q7. Muscle cannot make use of glycogen because of deficiency of : a) Glucose-6- phosphatase b) Glycogen phosphorylase c) Hexokinase d) Phospho-gluco- mutase Our WebSite https://tamsmed.com
  • 21. Q8. Malate shuttle is important in : a) Glycogenesis b) Glycolysis c) Gluconeogenesis d) Glycogenolysis Our WebSite https://tamsmed.com
  • 22. Q8. Malate shuttle is important in : a) Glycogenesis b) Glycolysis c) Gluconeogenesis d) Glycogenolysis Our WebSite https://tamsmed.com
  • 23. Q9. During prolonged starvation, rate of gluconeogenesis depends on : a) Increased alanine levels in liver b) Decreased cGMP levels in liver c) ADP in liver d) Decreased essential fatty acids in liver Our WebSite https://tamsmed.com
  • 24. Q9. During prolonged starvation, rate of gluconeogenesis depends on : a) Increased alanine levels in liver b) Decreased cGMP levels in liver c) ADP in liver d) Decreased essential fatty acids in liver Our WebSite https://tamsmed.com
  • 25. Q10. Which of the following is a Pallindrome ? a) GGCC b) GTCC c) TAAT d) TTCG Our WebSite https://tamsmed.com
  • 26. Q10. Which of the following is a Pallindrome ? a) GGCC b) GTCC c) TAAT d) TTCG Our WebSite https://tamsmed.com
  • 27. Download App for for more videos https://tamsmed.com/biochemistry.php Our WebSite https://tamsmed.com