The Indian Dental Academy is the Leader in continuing dental education , training dentists in all aspects of dentistry and
offering a wide range of dental certified courses in different formats.for more details please visit
www.indiandentalacademy.com
In prosthodontics, replacing the missing, without affecting the other components of the masticatory system has two main reference the maximum intercupation and the centric relation.
In this lecture discussion of centric relation as reference is exposed.
Neutral Zone
In dentistry, the neutral zone refers to that space in the oral cavity where the forces exerted by the musculature.of.the tongue are equal and balanced with the.forces exerted by the buccinators.muscle of the cheek.laterally and the orbicularis.oris muscle anteriorly
The Indian Dental Academy is the Leader in continuing dental education , training dentists in all aspects of dentistry and
offering a wide range of dental certified courses in different formats.for more details please visit
www.indiandentalacademy.com
In prosthodontics, replacing the missing, without affecting the other components of the masticatory system has two main reference the maximum intercupation and the centric relation.
In this lecture discussion of centric relation as reference is exposed.
Neutral Zone
In dentistry, the neutral zone refers to that space in the oral cavity where the forces exerted by the musculature.of.the tongue are equal and balanced with the.forces exerted by the buccinators.muscle of the cheek.laterally and the orbicularis.oris muscle anteriorly
the aims of orthodontics is to treat protruded teeth to prevent trauma . crowded teeth help initiation of caries so their treatment is indicated by orthodontics
The Indian Dental Academy is the Leader in continuing dental education , training dentists in all aspects of dentistry and
offering a wide range of dental certified courses in different formats.for more details please visit
www.indiandentalacademy.com
Types of malocclusion
Malocclusion can occur singly or in combination as follows:
1- Dental mal-relationship.
2- Dento-alveolar, involving the teeth and alveolar process.
3- Dental arch mal- relationship
4- Basal arch discrepancy
5- Cranio-facial abnormalities.
1- Dental mal relationship
Including crowding, spacing, ectopism and other local malposition of the teeth that not affect the arch size, relationship and growth.
Causes of dental malposition:
1- genetic factors.
2- Prolonged retention or early loss of primary teeth.
3- Delayed eruption of permanent teeth.
4- Supernumerary teeth, missing teeth, either congenital or due to extractions.
5- Ectopic eruption and abnormal tooth morphology.
6- Abnormal development of the teeth.
Frequency:
=The most frequently malposed teeth in permanent dentition are the 3rd molars, maxillary lateral incisors, mandibular incisors, 2nd premolars and 2nd molars, the less frequently malposed teeth are the 1st molars and 1st premolars.
= spacing is predominating in deciduous dentition while crowding is common in permanent dentition.
= Irregularities due to local pot natal causes will manifest themselves clinically and will requires corrective treatment.
= rotation of the teeth in most cases is a sequlea of crowding but sometimes occurs with spacing due to loss of proximal contact between the erupting and adjacent teeth.
= crowding and spacing of the same arch may be expected in deciduous dentition but if occurs in permanent dentition, it will represent a symptoms of tooth shifting and detective eruption due to local interfere.
Spacing of permanent teeth:
In the permanent dentition, spacing in the maxillary arch is usually localized from canine to canine.
The median diastema in maxillary arch may be caused by:
1- Abnormal labial Frenum or presence of mesiodense
2- Dwarfed or congenitally absent lateral incisors.
3- Or as a part of generalized spacing.
Spacing in the mandibular arch is less common but may occurs due to:
1- Abnormal large tongue and bi dental protrusion.
2- Abnormal lingual Frenum ----- median diastema.
3- As a part of generalized spacing.
Loss of space or space closure:
a- In mixed dentition:
= In children with well-developed arches, there is little or no space loss after extraction of primary molars, this may be due to the cuspal interlocking of U and L 6
= There will be more space loss after extraction of E then after extraction of D
= loss of UE before the eruption of UB causes the U6 to erupt far mesially toward UD occupying the space required for eruption of U5------ impaction of U5.
= in the mandibular arch the forward shifting of L6 is less great and space loss is less marked.
= the order of eruption of permanent teeth has some effect on determining the space closure. For example: the maxillary canine may block out labially because it erupts after U4, the mandibular 2nd bicuspids may block out lingually because it is the last tooth erupt in the front of L6.
b- In permanent
Factors affect occlusal development
Prepared by:
Dr Mohammed Alruby
المحايد هو شخص لم ينصر الباطل ولكن من المؤكد انه خذل الحق
Factors affect occlusal development
General factors:
1- Skeletal
2- Muscular
3- Dental
Local factors:
1- Aberrant developmental position of teeth
2- Presence of supernumerary teeth
3- Hypodontia- congenital absence of certain teeth
4- Effects of certain habits activities
5- Localized soft tissue anomalies
Skeletal factors affect occlusal development
= Any pathology condition affecting growth of the jaws is likely to have marked effect on the occlusion of the teeth
= As the teeth are set in the jaws, the relationship of the jaws to each other will have a large influence on the relationship of the dental arches.
= jaws relationship can be considered under three headings:
1- Jaws relationship to cranial base:
By relating the jaws to anterior cranial base as it anterior or posterior in position
2- Jaws relation to each other:
Class I: upper and lower in normal relation together
Class II: lower posterior to upper
Class III: in which lower arch forward in occlusion to upper
Variation in skeletal relationship may be due to:
- Variation in jaws size
- Variation in position of jaws to cranial base
Both these variations can affect the type of occlusion as in cases of skeletal I or II or III
=== vertical relationship of upper and lower also affect the occlusion, mandible with high gonial angle tends to produce a longer vertical dimension of the face. Mandible with low gonial angle tends to produce a shorter vertical dimension of the face
3- Alveolar bone in relation to basal bone:
Skeletal relationship refers to basal bone
Alveolar bone supported by basal bone
Alveolar bone supports the teeth
Muscle factors affect occlusal development
= the muscle of tongue, cheeks are of particular importance in guiding the teeth into their final position
= variation in muscle form and function can affect the position and occlusion of the teeth
Lips:
= Lips are usually brought together during swallowing and speech movements, if they are insufficient size so need extra force to close together with more muscle contraction that have high effect on the teeth related to it during eruption
=the lower lip tends to be further back than the upper as in skeletal class II and further forward as in class II, this not only increase the difficulty to put the lips together but also may cause the lower lip to modify the eruptive path of the upper incisors
= the lower lip plays more part than upper lip in functional movement and governing the position of incisors teeth
Tongue:
Tongue size: if tongue large size so it not has adequate space between arches so it fills space between upper and dental arches so prevent full vertical development of dento-alveolar structure
Tongue position: normally tongue lateral border touch the lingual cusps of molars and premolars
Tip of tongue: rest at the lingual surface between the lower in
etiology of malocclusion for general practitioners.docxDr.Mohammed Alruby
Etiology of Malocclusion
For general practitioners
Prepared by
Dr. M Alruby
Etiology in orthodontics is the study of actual causes of dento – facial abnormalities.
Malocclusion is the condition where there is a deviation from the usual or accepted relationship, dental malocclusion exists when the individual teeth within one or both jaws are abnormally related to each other, this condition may be limited to a couple of teeth or involving the majority of teeth present.
Development of normal dentition and occlusion depends on a number of interrelated factors that include the dento alveolar, skeletal and the neuromuscular factors. Thus localization of the possible etiology may be a very difficult task.
A- Extrinsic factors:
1- Evolution:
With evolution, the jaws become smaller, reduction in the number and size of teeth and diminution of jaw projection together with increased in vertical height of the face and there is retrognathic tendency in man as he ascends the evolutionary scale.
2- Heredity:
Transmission of dento facial characteristic through generation by genes. The child is a product of parents who have dissimilar genetic material. Thus the child may inherit conflicting traits from both the parents resulting in abnormalities of the dentofacial region. Another reason attributed for genetically determined malocclusion is the racial, ethnic and regional intermixer, which might have led to uncoordinated inheritance of teeth and jaws.
There are three types of transmission of malocclusion from the stand point of genetics:
1- Repetitive: the recurrence of single dentofacial deviation within the immediate family.
2- Discontinuous: a tendency for a malocclusion trait to reappear within the family over several generations.
3- Variable: the occurrence of different but related types of malocclusion within several generation of the same family.
Dental defect of genetic origin include the following:
= Crowding and spacing of teeth.
= Size and characteristic of soft tissue including muscles and frenum.
= Macrognathia and micrognathia.
= Macrodontia and microdontia.
= Oligodontia.
= Tooth shape variations.
= Median diastemas.
= upper face height, nose height, and bigonial width.
= Bimaxillary protrusion.
4- Congenital:
Those are deformities of hereditary or non-hereditary origin but exciting at birth.
The congenital abnormalities that cause malocclusion:
= Cleft lip and palate:
lack of fusion between the two palatal processes to each other. From one third to one half of all cleft palate children have familial history of this deformity.
As with the non-cleft child, palatal, pharyngeal and perioral musculature is well developed at birth to meet the demand of suckling, deglutition and mastication. While the complete unilateral or complete bilateral cleft break the continuity of the upper lip and disturbs the functional pattern and significantly reduce the restraining effect of the buccinators mechanism that pro
Similar to Malocclusion & Orthodontic Treatment (20)
Extended totally extraperitoneal repair (eTEP) is a novel technique that was first introduced by Jorge Daes in 2012 to address difficult inguinal hernias.
It's my c.v and I am looking to start a career in a reputed organization and my motive will be delivering quality healthcare services it’s a passion and motivation.
Prix Galien International 2024 Forum ProgramLevi Shapiro
June 20, 2024, Prix Galien International and Jerusalem Ethics Forum in ROME. Detailed agenda including panels:
- ADVANCES IN CARDIOLOGY: A NEW PARADIGM IS COMING
- WOMEN’S HEALTH: FERTILITY PRESERVATION
- WHAT’S NEW IN THE TREATMENT OF INFECTIOUS,
ONCOLOGICAL AND INFLAMMATORY SKIN DISEASES?
- ARTIFICIAL INTELLIGENCE AND ETHICS
- GENE THERAPY
- BEYOND BORDERS: GLOBAL INITIATIVES FOR DEMOCRATIZING LIFE SCIENCE TECHNOLOGIES AND PROMOTING ACCESS TO HEALTHCARE
- ETHICAL CHALLENGES IN LIFE SCIENCES
- Prix Galien International Awards Ceremony
Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
New Directions in Targeted Therapeutic Approaches for Older Adults With Mantl...i3 Health
i3 Health is pleased to make the speaker slides from this activity available for use as a non-accredited self-study or teaching resource.
This slide deck presented by Dr. Kami Maddocks, Professor-Clinical in the Division of Hematology and
Associate Division Director for Ambulatory Operations
The Ohio State University Comprehensive Cancer Center, will provide insight into new directions in targeted therapeutic approaches for older adults with mantle cell lymphoma.
STATEMENT OF NEED
Mantle cell lymphoma (MCL) is a rare, aggressive B-cell non-Hodgkin lymphoma (NHL) accounting for 5% to 7% of all lymphomas. Its prognosis ranges from indolent disease that does not require treatment for years to very aggressive disease, which is associated with poor survival (Silkenstedt et al, 2021). Typically, MCL is diagnosed at advanced stage and in older patients who cannot tolerate intensive therapy (NCCN, 2022). Although recent advances have slightly increased remission rates, recurrence and relapse remain very common, leading to a median overall survival between 3 and 6 years (LLS, 2021). Though there are several effective options, progress is still needed towards establishing an accepted frontline approach for MCL (Castellino et al, 2022). Treatment selection and management of MCL are complicated by the heterogeneity of prognosis, advanced age and comorbidities of patients, and lack of an established standard approach for treatment, making it vital that clinicians be familiar with the latest research and advances in this area. In this activity chaired by Michael Wang, MD, Professor in the Department of Lymphoma & Myeloma at MD Anderson Cancer Center, expert faculty will discuss prognostic factors informing treatment, the promising results of recent trials in new therapeutic approaches, and the implications of treatment resistance in therapeutic selection for MCL.
Target Audience
Hematology/oncology fellows, attending faculty, and other health care professionals involved in the treatment of patients with mantle cell lymphoma (MCL).
Learning Objectives
1.) Identify clinical and biological prognostic factors that can guide treatment decision making for older adults with MCL
2.) Evaluate emerging data on targeted therapeutic approaches for treatment-naive and relapsed/refractory MCL and their applicability to older adults
3.) Assess mechanisms of resistance to targeted therapies for MCL and their implications for treatment selection
Explore natural remedies for syphilis treatment in Singapore. Discover alternative therapies, herbal remedies, and lifestyle changes that may complement conventional treatments. Learn about holistic approaches to managing syphilis symptoms and supporting overall health.
Tom Selleck Health: A Comprehensive Look at the Iconic Actor’s Wellness Journeygreendigital
Tom Selleck, an enduring figure in Hollywood. has captivated audiences for decades with his rugged charm, iconic moustache. and memorable roles in television and film. From his breakout role as Thomas Magnum in Magnum P.I. to his current portrayal of Frank Reagan in Blue Bloods. Selleck's career has spanned over 50 years. But beyond his professional achievements. fans have often been curious about Tom Selleck Health. especially as he has aged in the public eye.
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Introduction
Many have been interested in Tom Selleck health. not only because of his enduring presence on screen but also because of the challenges. and lifestyle choices he has faced and made over the years. This article delves into the various aspects of Tom Selleck health. exploring his fitness regimen, diet, mental health. and the challenges he has encountered as he ages. We'll look at how he maintains his well-being. the health issues he has faced, and his approach to ageing .
Early Life and Career
Childhood and Athletic Beginnings
Tom Selleck was born on January 29, 1945, in Detroit, Michigan, and grew up in Sherman Oaks, California. From an early age, he was involved in sports, particularly basketball. which played a significant role in his physical development. His athletic pursuits continued into college. where he attended the University of Southern California (USC) on a basketball scholarship. This early involvement in sports laid a strong foundation for his physical health and disciplined lifestyle.
Transition to Acting
Selleck's transition from an athlete to an actor came with its physical demands. His first significant role in "Magnum P.I." required him to perform various stunts and maintain a fit appearance. This role, which he played from 1980 to 1988. necessitated a rigorous fitness routine to meet the show's demands. setting the stage for his long-term commitment to health and wellness.
Fitness Regimen
Workout Routine
Tom Selleck health and fitness regimen has evolved. adapting to his changing roles and age. During his "Magnum, P.I." days. Selleck's workouts were intense and focused on building and maintaining muscle mass. His routine included weightlifting, cardiovascular exercises. and specific training for the stunts he performed on the show.
Selleck adjusted his fitness routine as he aged to suit his body's needs. Today, his workouts focus on maintaining flexibility, strength, and cardiovascular health. He incorporates low-impact exercises such as swimming, walking, and light weightlifting. This balanced approach helps him stay fit without putting undue strain on his joints and muscles.
Importance of Flexibility and Mobility
In recent years, Selleck has emphasized the importance of flexibility and mobility in his fitness regimen. Understanding the natural decline in muscle mass and joint flexibility with age. he includes stretching and yoga in his routine. These practices help prevent injuries, improve posture, and maintain mobilit
Recomendações da OMS sobre cuidados maternos e neonatais para uma experiência pós-natal positiva.
Em consonância com os ODS – Objetivos do Desenvolvimento Sustentável e a Estratégia Global para a Saúde das Mulheres, Crianças e Adolescentes, e aplicando uma abordagem baseada nos direitos humanos, os esforços de cuidados pós-natais devem expandir-se para além da cobertura e da simples sobrevivência, de modo a incluir cuidados de qualidade.
Estas diretrizes visam melhorar a qualidade dos cuidados pós-natais essenciais e de rotina prestados às mulheres e aos recém-nascidos, com o objetivo final de melhorar a saúde e o bem-estar materno e neonatal.
Uma “experiência pós-natal positiva” é um resultado importante para todas as mulheres que dão à luz e para os seus recém-nascidos, estabelecendo as bases para a melhoria da saúde e do bem-estar a curto e longo prazo. Uma experiência pós-natal positiva é definida como aquela em que as mulheres, pessoas que gestam, os recém-nascidos, os casais, os pais, os cuidadores e as famílias recebem informação consistente, garantia e apoio de profissionais de saúde motivados; e onde um sistema de saúde flexível e com recursos reconheça as necessidades das mulheres e dos bebês e respeite o seu contexto cultural.
Estas diretrizes consolidadas apresentam algumas recomendações novas e já bem fundamentadas sobre cuidados pós-natais de rotina para mulheres e neonatos que recebem cuidados no pós-parto em unidades de saúde ou na comunidade, independentemente dos recursos disponíveis.
É fornecido um conjunto abrangente de recomendações para cuidados durante o período puerperal, com ênfase nos cuidados essenciais que todas as mulheres e recém-nascidos devem receber, e com a devida atenção à qualidade dos cuidados; isto é, a entrega e a experiência do cuidado recebido. Estas diretrizes atualizam e ampliam as recomendações da OMS de 2014 sobre cuidados pós-natais da mãe e do recém-nascido e complementam as atuais diretrizes da OMS sobre a gestão de complicações pós-natais.
O estabelecimento da amamentação e o manejo das principais intercorrências é contemplada.
Recomendamos muito.
Vamos discutir essas recomendações no nosso curso de pós-graduação em Aleitamento no Instituto Ciclos.
Esta publicação só está disponível em inglês até o momento.
Prof. Marcus Renato de Carvalho
www.agostodourado.com
Lung Cancer: Artificial Intelligence, Synergetics, Complex System Analysis, S...Oleg Kshivets
RESULTS: Overall life span (LS) was 2252.1±1742.5 days and cumulative 5-year survival (5YS) reached 73.2%, 10 years – 64.8%, 20 years – 42.5%. 513 LCP lived more than 5 years (LS=3124.6±1525.6 days), 148 LCP – more than 10 years (LS=5054.4±1504.1 days).199 LCP died because of LC (LS=562.7±374.5 days). 5YS of LCP after bi/lobectomies was significantly superior in comparison with LCP after pneumonectomies (78.1% vs.63.7%, P=0.00001 by log-rank test). AT significantly improved 5YS (66.3% vs. 34.8%) (P=0.00000 by log-rank test) only for LCP with N1-2. Cox modeling displayed that 5YS of LCP significantly depended on: phase transition (PT) early-invasive LC in terms of synergetics, PT N0—N12, cell ratio factors (ratio between cancer cells- CC and blood cells subpopulations), G1-3, histology, glucose, AT, blood cell circuit, prothrombin index, heparin tolerance, recalcification time (P=0.000-0.038). Neural networks, genetic algorithm selection and bootstrap simulation revealed relationships between 5YS and PT early-invasive LC (rank=1), PT N0—N12 (rank=2), thrombocytes/CC (3), erythrocytes/CC (4), eosinophils/CC (5), healthy cells/CC (6), lymphocytes/CC (7), segmented neutrophils/CC (8), stick neutrophils/CC (9), monocytes/CC (10); leucocytes/CC (11). Correct prediction of 5YS was 100% by neural networks computing (area under ROC curve=1.0; error=0.0).
CONCLUSIONS: 5YS of LCP after radical procedures significantly depended on: 1) PT early-invasive cancer; 2) PT N0--N12; 3) cell ratio factors; 4) blood cell circuit; 5) biochemical factors; 6) hemostasis system; 7) AT; 8) LC characteristics; 9) LC cell dynamics; 10) surgery type: lobectomy/pneumonectomy; 11) anthropometric data. Optimal diagnosis and treatment strategies for LC are: 1) screening and early detection of LC; 2) availability of experienced thoracic surgeons because of complexity of radical procedures; 3) aggressive en block surgery and adequate lymph node dissection for completeness; 4) precise prediction; 5) adjuvant chemoimmunoradiotherapy for LCP with unfavorable prognosis.
Pulmonary Thromboembolism - etilogy, types, medical- Surgical and nursing man...VarunMahajani
Disruption of blood supply to lung alveoli due to blockage of one or more pulmonary blood vessels is called as Pulmonary thromboembolism. In this presentation we will discuss its causes, types and its management in depth.
Ethanol (CH3CH2OH), or beverage alcohol, is a two-carbon alcohol
that is rapidly distributed in the body and brain. Ethanol alters many
neurochemical systems and has rewarding and addictive properties. It
is the oldest recreational drug and likely contributes to more morbidity,
mortality, and public health costs than all illicit drugs combined. The
5th edition of the Diagnostic and Statistical Manual of Mental Disorders
(DSM-5) integrates alcohol abuse and alcohol dependence into a single
disorder called alcohol use disorder (AUD), with mild, moderate,
and severe subclassifications (American Psychiatric Association, 2013).
In the DSM-5, all types of substance abuse and dependence have been
combined into a single substance use disorder (SUD) on a continuum
from mild to severe. A diagnosis of AUD requires that at least two of
the 11 DSM-5 behaviors be present within a 12-month period (mild
AUD: 2–3 criteria; moderate AUD: 4–5 criteria; severe AUD: 6–11 criteria).
The four main behavioral effects of AUD are impaired control over
drinking, negative social consequences, risky use, and altered physiological
effects (tolerance, withdrawal). This chapter presents an overview
of the prevalence and harmful consequences of AUD in the U.S.,
the systemic nature of the disease, neurocircuitry and stages of AUD,
comorbidities, fetal alcohol spectrum disorders, genetic risk factors, and
pharmacotherapies for AUD.
These lecture slides, by Dr Sidra Arshad, offer a quick overview of physiological basis of a normal electrocardiogram.
Learning objectives:
1. Define an electrocardiogram (ECG) and electrocardiography
2. Describe how dipoles generated by the heart produce the waveforms of the ECG
3. Describe the components of a normal electrocardiogram of a typical bipolar leads (limb II)
4. Differentiate between intervals and segments
5. Enlist some common indications for obtaining an ECG
Study Resources:
1. Chapter 11, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 9, Human Physiology - From Cells to Systems, Lauralee Sherwood, 9th edition
3. Chapter 29, Ganong’s Review of Medical Physiology, 26th edition
4. Electrocardiogram, StatPearls - https://www.ncbi.nlm.nih.gov/books/NBK549803/
5. ECG in Medical Practice by ABM Abdullah, 4th edition
6. ECG Basics, http://www.nataliescasebook.com/tag/e-c-g-basics
Ozempic: Preoperative Management of Patients on GLP-1 Receptor Agonists Saeid Safari
Preoperative Management of Patients on GLP-1 Receptor Agonists like Ozempic and Semiglutide
ASA GUIDELINE
NYSORA Guideline
2 Case Reports of Gastric Ultrasound
2. Malocclusion:
Malocclusion is a condition in which there is a malrelationship between the arches
in any of the planes of the space or in which there are anomalies in tooth position
beyond the limit of the normal. (WALTHER & HOUSTON)
According to Angle: the key to occlusion was the maxillary 1st molar
• Class I (normal occlusion)
• Class I malocclusion
• Class II malocclusion
• Class III malocclusion
3. CLASSIFICATION
Depending upon which part of the oral & maxillofacial unit is at fault :
1. Individual tooth malposition's (DENTAL DYSPLASIA)
2. Malrelation’s of the dental arches or dento alveolar segments. (SKELETODENTAL DYSPLASIA)
3. Skeletal malrelationship (SKELETAL DYSPLASIA)
4. I. Individual tooth malposition's (DENTAL DYSPLASIA)
o These are malocclusions of individual teeth in relation to adjacent teeth with in same dental
arch.
o This disorder is also called intra arch malocclusions.
o This malocclusion includes condition like spacing or crowding with in dental arches.
They are of following type:
5. 1. MESIAL
INCLINATION
OR TIPPING
2. DISTAL
INCLINATION OR
TIPPING
3. LINGUAL
INCLINATION
OR TIPPING
4.LABIAL/BUCCAL
INCLINATION OR TIPPING 5.INFRA OCCLUSION
6.SUPRAOCCLUSION
7.ROTATIONS :-
a.Mesiolingual or distolabial b. Distolingual or mesiolabial
8.TRANSPOSITION
6. ii. Malrelation's of Dental Arches:
o These characterized by an abnormal relationship between teeth or groups of teeth of one
dental arch to another arch.
o These occur in all the three planes of space, namely –
> sagittal plane
> vertical plane
> transverse plane
7. SAGITTAL PLANE MALOCCLUSION
a. Pre normal occlusion:
Where the mandibular dental arch is
placed more posteriorly when the
teeth meet in centric occlusion.
b. Post normal occlusion:
Where the mandibular dental arch is
place more distally when the teeth
meet in centric occlusion.
9. TRANSVERS PLANE MALOCCLUSION
These includes various types of cross bites due to constriction of the
dental arches or some other reason the relationship is disturbed.
10. iii. SKELETAL MALOCCLUSIONS:
o These malocclusions are caused due to the defect in the underlying skeletal structure itself.
o The defect can be in size position or relationship b/w the jaw bones.
12. Specific causes of malocclusion:
A. Disturbances in embryological development
• Causes: range from genetic disturbances to
specific environmental insults
• Teratogens: chemical and other agents
capable of producing embryologic defects if
given at the critical time
• <1% of children who need orthodontics had
a disturbance in embryologic development
as a major contributing cause.
B. Skeletal growth disturbances
• Fetal molding and birth injuries
– Intrauterine molding: pressure against
the face
– Birth trauma to the mandible: use
forceps in delivery
– Childhood fracture of the jaw
13. C. Muscle dysfunction:
• Damage to motor nerve →underdevelopment
of that part of the face (Muscle atrophy).
• Excessive muscle contraction of neck on one
side can resist growth in the same way as
scaring after injury → facial asymmetry
D. Acromegaly:
• It is caused by anterior pituitary gland tumor
that causes excessive secretion of growth
hormones.
• Excessive growth of the mandible
class III malocclusion
E. Disturbances of dental development:
• Congenitally missing teeth
• Malformed or supernumerary teeth
• Interferences with eruption
• Ectopic eruption
• Early loss of primary teeth
• Traumatic displacement of teeth
F. Hemi mandibular hypertrophy:
• unilateral Excessive growth of the mandible
15. Genetic influence:
• Inherited in 2 major ways:
– Disproportion between the size of the teeth and the size of the jaws (Teeth vs Jaw)
– Disproportion between size or shape of the upper and lower jaws (Upper vs Lower)
16. Environmental influences:
• If a habit like thumb sucking created pressure against the teeth for more than the
threshold duration (6 hours or more per day), it certainly could move teeth.
Thumb sucking:
• During primary dentition: no influence
• If it persists beyond the time that the permanent teeth begin to erupt:
– Flared and spaced maxillary incisors
– Lingually positioned lower incisors
– Anterior open bite
– A narrow upper arch
17. A malocclusion where the molar relationship shows
the buccal groove of the mandibular first molar
distally positioned when in occlusion with the
mesiobuccal cusp of the maxillary first molar.
The mesiobuccal cusp of the maxillary first molar is
aligned with the buccal groove of the mandibular first
molar. There is alignment of the teeth, normal overbite
and overjet and coincident maxillary and mandibular
midlines.
A normal molar relationship exists but there is
crowding, misalignment of the teeth, cross bites, etc.
A malocclusion where the molar relationship shows
the buccal groove of the mandibular first molar
mesially positioned to the mesiobuccal cusp of the
maxillary first molar when the teeth are in
occlusion.