SlideShare a Scribd company logo
Normal Labor &
Delivery
Introduction..
Labor :
 regular uterine contractions of sufficient frequency, intensity, and duration that
lead to progressive cervical dilatation, effacement and descent of presenting part.
 3 Ps of labour: powers, the passages and the passenger.
 If any of the 3Ps are unfavourable, labour is likely to be abnormal resulting in the
need for intervention
Maternal Anatomy
• The pelvic inlet:
 Transverse diameter is 13.5 cm >
Anterior–posterior (A–P) diameter is
11.0 cm.
 The fetal head enters the pelvis
orientated in a transverse position in
keeping with the wider transverse
diameter.
 The angle of the inlet is normally 60°
Maternal Anatomy
• The Midpelvis
 It is almost round, as the transverse and A-P are
similar at 12cm.
 The ischial spines are palpable vaginally and are used
as important landmarks to:
1. To assess the descent of the presenting part on
vaginal examination (e.g. station 0 is at the level
of the ischial spines)
2. To provide a local anaesthetic pudendal nerve
block.
Maternal Anatomy
• The Pelvic Outlet
 The transverse is the widest diameter at
the inlet, but at the outlet it is the AP
diameter
 The fetal head must rotate from a
transverse to an AP position as it passes
through the pelvis
Pelvic Shapes
Fetal Anatomy
At the time of labour, the sutures
joining the bones of the vault are
soft, un-ossified membranes.
The sutures of the fetal face and
the skull base are firmly united.
Fetal Anatomy
Fetal skull moulding:
 Sutures are not fixed which allows the bones
to move together and even to overlap.
 The parietal bones usually slide over the
frontal and occipital bones
 Moulding reduces the diameters of the fetal
head and encourages progress through the
bony pelvis, while still protecting the
underlying brain
The diameters of the skull
Physiology of Labor
• The cervix: softens, shortens, thins out (effacement) and dilates for
labour to progress.
• The uterus: changes from a state of relaxation to an active state of regular
contractions to facilitate transit of the fetus through the birth canal.
• Each contraction must be followed by a resting phase in order to maintain
placental blood flow and adequate perfusion of the fetus.
• The pressure of the presenting part on the pelvic floor muscles produces
a maternal urge to push.
Physiology of Labor
 The uterus
• Uterine contractions happens in response to an increase in intracellular
calcium.
• Prostaglandins and oxytocin increase intracellular free calcium ions.
• Progressive shortening of the uterine smooth muscle cells is called
retraction and occurs in the cells of the upper part of the uterus
• Retraction results in the development of the thicker, actively contracting
‘upper segment’ and the lower segment of the uterus becomes thinner
and more stretched
• Contraction intervals of 2–4
minutes (i.e. 2 in 10 increasing to
4–5 in 10 in advanced labour)
• Their duration varies from 30 to 60
seconds or longer.
• The intensity or amplitude of the
intrauterine pressure during a
contraction averages between 30
and 60 mmHg. ( 200 MVU)
Physiology of Labor
 The cervix
• Under the influence of prostaglandins, there is an increase in proteolytic
activity and a reduction in collagen and elastin.
• Dermatan sulphate is replaced by the more hydrophilic hyaluronic acid,
which results in an increase in water content of the cervix.
• Causing cervical softening or ‘ripening’, so that when contractions begin
the processes of effacement and dilatation start.
Diagnosis of labor
The onset of labour can be defined as the presence of strong regular
painful contractions resulting in progressive cervical change
Loss of a ‘show’ or spontaneous rupture of the membranes (SROM) does
not define the onset of labour
Diagnosis of true labor pain
 History
• A history of regular painful uterine contraction in every 5- 8
min, accompanied by the history of a bloody show or
spontaneous rupture of membrane
 Physical examination
• Reduction of interval between uterine contractions
• Abdominal pain of increasing intensity
• Cervical effacement (≥ 50%)
• Cervical dilation (≥ 2 cm)
Normal labor
Spontaneous onset
Single cephalic presentation.
37-42 weeks of gestation
No artificial interventions.
Unassisted spontaneous vaginal delivery.
Duration of <12 hours in nulliparous women, and <8 hours in multiparous women.
Stages Of Labor
 There are three stages of labor:
• The first stage (stage of dilatation of the cervix) is from the onset of true labor
(regular uterine contractions) to complete dilatation of the cervix
• The second stage (stage of fetal delivery ) is from complete dilatation of the
cervix to the birth of the baby
• The third stage (stage of placental delivery) is from the birth of the baby to
delivery of the placenta.
Stages Of Labor
First stage
• It is the stage of cervical dilatation.
• Starts with the onset of true labor pain and ends with full dilatation of
the cervix i.e. 10 cm in diameter.
• It takes about 10-14 hours in primigravida and about 6-8 hours in
multipara
Phases of cervical dilatation
Latent phase:
• This is the first 3 cm of cervical dilatation which is slow takes about 8 hours
in nullipara and 4 hours in multipara.
Active phase:
• The time between the end of the latent phase (3–4 cm dilatation) and full
cervical dilatation (10 cm)
• Lasting between 2 and 6 hours, shorter in multiparous women
• Cervical dilatation during the active phase occurs typically at 1 cm/hour
or more
• It is considered abnormal if it occurs at less than 1 cm in 2 hours.
Stages Of Labor
Second stage
• It is the stage of expulsion of the fetus.
• Begins with full cervical dilatation and ends with the delivery of the
fetus.
• Its duration is about 50 minutes in primigravida and 20minutes in
multipara
• considered prolonged if more than one hour in multiparas and more than
2 hours in primigravidea
Phases Of Second stage of Labor
 The ‘passive phase’
• The time between full dilatation and the onset of involuntary expulsive
contractions
• There is no maternal urge to push and the fetal head is still relatively high in the
pelvis.
 The second phase ‘active second stage’.
• There is a maternal urge to push because the fetal head is low (often visible),
causing a reflex need to ‘bear down’
• normal active second stage should last no longer than 2 hours in a nulliparous
woman and 1 hour in multipara
Stages Of Labor
 Third stage
• It is the stage of expulsion of the placenta and membranes.
• Begins after delivery of the fetus and ends with expulsion of the placenta
and membranes.
• Its duration is about 10-20 minutes in both primi and multipara. normally
not exceed 30 minutes with active management of third stage
Duration of Labor
 Prolonged Labour
• labour lasting longer than 12 hours in nulliparous women and 8 hours in
multiparous women.
 Precipitous labour
• defined as expulsion of the fetus within less than 3 hours of the onset of
regular contractions.
Mechanisms of labor
 Fetal lie :
The relation of the fetal long axis to that of the mother
• Longitudinal
• Transverse
• Oblique
• A longitudinal lie is present in more than 99% of labors at term.
• Predisposing factors for transverse fetal position include
multiparity, placenta previa, hydramnios
Mechanisms of labor
 Fetal presentation :
• The portion of the fetal body that is either foremost within the birth
canal or in the closest proximity to it.
• Cephalic 96.8%
• breech 2.7 %
• Shoulder 0.3%
• Compound 0.1%
Mechanisms of labor
 Cephalic presentation
• Well flexed head (vertex or occiput presentation)
• Sinciput presentation (Military ) === transient
• Brow presentation 0.01 % === transient
• Face presentation 0.05%
• As labor progresses, sinciput and brow presentations almost always convert into
vertex or face presentations by neck flexion or extension, respectively.
 Fetal Attitude:
 Position of head with regard to
fetal spine (ie: degree of flexion or
extension)
• A - suboccipitobragmatic (vertex)
• B - occipitofrontal (military)
• C - mentovertical (brow )
• D - submintobragmatic (face)
** OP with occipitofrontal 11.5
Or suboccipitofrontal 10
Breech presentation
 Incidence:
• 25 % 28 weeks GA
• 17% 30 weeks GA
• 11% 32 weeks GA
• 2.7 % term
Presenting parts diameter in cephalic
presentation
Mechanisms of labor
Fetal position
• Relation ship of chosen portion of the fetal presenting part to the right or
left side of the birth canal (ROA, LOA , ROP, RMA…..)
• DOMINATOPRS:
 Vertex
 Face
 Breech
 Shoulder
• Occiput
• Mentum
• Sacrum
• Acromiun
Diagnosis Of Fetal Presentation & Position
• Abdominal palpation
• Vaginal examination:
- Presenting part
- Position
- Dilatation
- Effacement
- Station
- Pelvimetry
• Auscultation : arounf the umbilicus in longitdunal lie , below if cephalic and above if breech
Vaginal Examination
Head station :
Relationship of presenting
part to the ischial spine
Bishop score
The Mechanism Of Labor (OA)
 Cardinal movements of labor
1. Engagement
2. Descent
3. Flexion
4. Internal Rotation
5. Extension
6. Restitution And External Rotation
7. Expulsion
The Mechanism Of Labor
 Engagement
• Passage of widest diameter of
presenting(in transverse position) part
to level below the plane of the pelvic
inlet
• Occurs earlier in nulliparous women
(36 wks)
The Mechanism Of Labor
 Descent
• Descent of the fetal head is needed before
flexion, internal rotation and extension can
occur
 Flexion
• Occurs passively As the
• head descends into the narrower midpelvis
The Mechanism Of Labor
 Internal Rotation
 Rotation of presenting part from original position (transverse) to
anteroposterior position
 If the head is well flexed, the occiput will be the leading point
The Mechanism Of Labor
 Extension
The well-flexed head now extends
and the occiput escapes from
underneath the symphysis pubis and
distends the vulva.
This is known as ‘crowning’ of the
head.
 Restitution
This slight rotation of the occiput
through one eighth
of the circle
The Mechanism Of Labor
 External rotation
The shoulders have to rotate into the direct AP
plane, Then the occiput rotates through a
further one-eighth of a circle to the transverse
position.
The Mechanism Of Labor
 Expulsion
The anterior shoulder is under the
symphysis pubis and delivers first, and
the posterior shoulder delivers
subsequently.
In occipito-posterior
Mechanism of labour is identical to OT &
anterior varieties
The occiput rotate to the symphysis pubis
through 135º instead of 90º or 45º
If rotation does not occur direct occiput
post or
Partial rotation transverse arrest
Management In First Stage
 Vaginal examinations are usually performed every 4 hours to determine
when the active phase has been reached
 Laboring woman should be allowed to assume the position she finds most
comfortable— this will be lateral recumbency most of the time
 Sips of clear liquids, occasional ice chips are permitted
Management In Second Stage
 The first sign of the second stage is likely to be an urge to push.
 In all cases the baby should be delivered within 4 hours of reaching full
dilatation
 Once the head has crowned, the perineum should be supported the
woman should be discouraged from bearing down by telling her to take
rapid, shallow breaths
 To aid delivery of the shoulders, there should be gentle traction on the
head downwards and forwards until the anterior shoulder appears
beneath the pubis
Fetal Care
Fetal monitoring:
• Low risk : every 30 min in first stage and 15 min in second stage
• High risk : every 15 min in first stage and every 5 min in the second stage
or continuous
Episiotomy
It is a second degree pereneal tear man made by scissors
Types: Midline, medio-lateral and lateral, Mediolateral is commonly used.
Indication: complicated vaginal delivery (breech, shoulder dystocia, forceps,
vacuum), scarring from female genital cutting or poorly healed third or fourth
degree tears and fetal distress.
Episiotomy
Wait to perform episiotomy until: the perineum is thinned out; and 3-4 cm of
the baby's head is visible during a contraction
Complications: Hematoma, infection and rarly necrotizing fasciitis
Episiotomy should be restricted to an indication
Management In Third Stage
Management In Third Stage
 Management of the third stage can be ‘active’ or ‘physiological’.
 Physiological management
• Is where the placenta is delivered by maternal effort and no uterotonic drugs are
given to assist this process
 Active management
• When the signs of placental separation are recognized, controlled cord traction is
used to expedite delivery of the placenta.
Management In Third Stage
Oxytocin dose is 10 IU, intramuscularly. with intravenous access in place, 10-20 IU is
placed in 500-1000 mL of crystalloid and run quickly. With cesarean deliveries, 5 IU is
administered as an intravenous bolus, followed by a similar infusion.
Ergometrine dose: is 0.2-0.25 mg, some used 0.5 mg ; IM or IV.
Syntometrine (contains 0.5 mg of ergometrine with 5 IU of oxytocin);
Oxytocin and ergometrine acts within 30 seonds if given IV and oxytocin acts after 3
mints while ergometrine after 6-8 mints if given IM
Management In Third Stage
Advantages of Active Management
 Advantage:
• reduction of the blood loss (60% reduction of blood loss )
 Disadvantages:
• Constriction ring may occur with retention of the placenta.
• Avulsion of the cord if undue pressure is applied.
• Inversion of the uterus if fundus is pressed while the uterus is lax.
Placental Examination
Examination of the placenta & membranes:
• by exploring it on a plain surface to be sure that it is complete. If there is missed
part, exploration of the uterus is done under general anaesthesia.

More Related Content

Similar to L31 Normal Labor & Delivery

First stage of labor
First stage of laborFirst stage of labor
First stage of labor
LoorthuSelviM
 
Normal Labour & Nursing Management of First stage of Labour
Normal Labour & Nursing Management of First stage of LabourNormal Labour & Nursing Management of First stage of Labour
Normal Labour & Nursing Management of First stage of Labour
Neha Parmar
 
Labour and delivery
Labour and deliveryLabour and delivery
Labour and deliveryFahad Zakwan
 
Partogram and management of 1st and 2nd stages of labor
Partogram and management of 1st and 2nd stages of laborPartogram and management of 1st and 2nd stages of labor
Partogram and management of 1st and 2nd stages of labor
Ali S. Mayali
 
PARTOGRAPH IN MANAGING LABOUR 2021.ppt
PARTOGRAPH IN MANAGING LABOUR 2021.pptPARTOGRAPH IN MANAGING LABOUR 2021.ppt
PARTOGRAPH IN MANAGING LABOUR 2021.ppt
FraviaFiridolin
 
Labor.pdf
Labor.pdfLabor.pdf
Labor.pdf
OmarOdeh23
 
The partograph
The partograph The partograph
The partograph
Maraey Menoufy Khalil
 
lab dystocia - 25.9.23 (1).pptx Dr P Ushadevi associate professor AMC VIZAG
lab dystocia - 25.9.23 (1).pptx Dr P Ushadevi associate professor AMC VIZAGlab dystocia - 25.9.23 (1).pptx Dr P Ushadevi associate professor AMC VIZAG
lab dystocia - 25.9.23 (1).pptx Dr P Ushadevi associate professor AMC VIZAG
slidesharecgr
 
Stages of labour.pptx
Stages of labour.pptxStages of labour.pptx
Stages of labour.pptx
vincenttobi1
 
Group 6 Reproductive Health Discussion.pptx
Group 6 Reproductive Health Discussion.pptxGroup 6 Reproductive Health Discussion.pptx
Group 6 Reproductive Health Discussion.pptx
YIKIISAAC
 
Stages of Normal Labor- easy explanation
Stages of Normal Labor- easy explanationStages of Normal Labor- easy explanation
Stages of Normal Labor- easy explanation
Swatilekha Das
 
Normal labor
Normal laborNormal labor
Normal labor
Salem Ahmed
 
Management of first stage labour
Management of first stage labourManagement of first stage labour
Management of first stage labour
P V GREESHMA
 
Normal physiology of labour and delivery .pptx
Normal physiology of labour and delivery .pptxNormal physiology of labour and delivery .pptx
Normal physiology of labour and delivery .pptx
Endex Tam
 
Normal labour and delivery
Normal labour and deliveryNormal labour and delivery
Normal labour and delivery
Jograjiya Gelabhai Raghubhai
 
Normal Labour/ Stages of Labour/ Mechanism of Labour
Normal Labour/ Stages of Labour/ Mechanism of LabourNormal Labour/ Stages of Labour/ Mechanism of Labour
Normal Labour/ Stages of Labour/ Mechanism of Labour
Wasim Ak
 
First stage of labor
First stage of laborFirst stage of labor
First stage of labor
Nikita Sharma
 
Normal Labor in Obstetrics
Normal Labor in ObstetricsNormal Labor in Obstetrics
Normal Labor in Obstetrics
Dr. Aryan (Anish Dhakal)
 
NORMAL LABOUR& Mechanism.pptx
NORMAL LABOUR& Mechanism.pptxNORMAL LABOUR& Mechanism.pptx
NORMAL LABOUR& Mechanism.pptx
Iram Chaudhry
 
Normal Labor and delivery , brief lecture
Normal Labor and delivery , brief lectureNormal Labor and delivery , brief lecture
Normal Labor and delivery , brief lecture
shaymadeeb
 

Similar to L31 Normal Labor & Delivery (20)

First stage of labor
First stage of laborFirst stage of labor
First stage of labor
 
Normal Labour & Nursing Management of First stage of Labour
Normal Labour & Nursing Management of First stage of LabourNormal Labour & Nursing Management of First stage of Labour
Normal Labour & Nursing Management of First stage of Labour
 
Labour and delivery
Labour and deliveryLabour and delivery
Labour and delivery
 
Partogram and management of 1st and 2nd stages of labor
Partogram and management of 1st and 2nd stages of laborPartogram and management of 1st and 2nd stages of labor
Partogram and management of 1st and 2nd stages of labor
 
PARTOGRAPH IN MANAGING LABOUR 2021.ppt
PARTOGRAPH IN MANAGING LABOUR 2021.pptPARTOGRAPH IN MANAGING LABOUR 2021.ppt
PARTOGRAPH IN MANAGING LABOUR 2021.ppt
 
Labor.pdf
Labor.pdfLabor.pdf
Labor.pdf
 
The partograph
The partograph The partograph
The partograph
 
lab dystocia - 25.9.23 (1).pptx Dr P Ushadevi associate professor AMC VIZAG
lab dystocia - 25.9.23 (1).pptx Dr P Ushadevi associate professor AMC VIZAGlab dystocia - 25.9.23 (1).pptx Dr P Ushadevi associate professor AMC VIZAG
lab dystocia - 25.9.23 (1).pptx Dr P Ushadevi associate professor AMC VIZAG
 
Stages of labour.pptx
Stages of labour.pptxStages of labour.pptx
Stages of labour.pptx
 
Group 6 Reproductive Health Discussion.pptx
Group 6 Reproductive Health Discussion.pptxGroup 6 Reproductive Health Discussion.pptx
Group 6 Reproductive Health Discussion.pptx
 
Stages of Normal Labor- easy explanation
Stages of Normal Labor- easy explanationStages of Normal Labor- easy explanation
Stages of Normal Labor- easy explanation
 
Normal labor
Normal laborNormal labor
Normal labor
 
Management of first stage labour
Management of first stage labourManagement of first stage labour
Management of first stage labour
 
Normal physiology of labour and delivery .pptx
Normal physiology of labour and delivery .pptxNormal physiology of labour and delivery .pptx
Normal physiology of labour and delivery .pptx
 
Normal labour and delivery
Normal labour and deliveryNormal labour and delivery
Normal labour and delivery
 
Normal Labour/ Stages of Labour/ Mechanism of Labour
Normal Labour/ Stages of Labour/ Mechanism of LabourNormal Labour/ Stages of Labour/ Mechanism of Labour
Normal Labour/ Stages of Labour/ Mechanism of Labour
 
First stage of labor
First stage of laborFirst stage of labor
First stage of labor
 
Normal Labor in Obstetrics
Normal Labor in ObstetricsNormal Labor in Obstetrics
Normal Labor in Obstetrics
 
NORMAL LABOUR& Mechanism.pptx
NORMAL LABOUR& Mechanism.pptxNORMAL LABOUR& Mechanism.pptx
NORMAL LABOUR& Mechanism.pptx
 
Normal Labor and delivery , brief lecture
Normal Labor and delivery , brief lectureNormal Labor and delivery , brief lecture
Normal Labor and delivery , brief lecture
 

More from Public Health & Medical Academy

L48 Uterine Fibroids
L48 Uterine Fibroids L48 Uterine Fibroids
L48 Uterine Fibroids
Public Health & Medical Academy
 
L45 Genital tract development and Puberty & their disorders
L45 Genital tract development and Puberty & their disordersL45 Genital tract development and Puberty & their disorders
L45 Genital tract development and Puberty & their disorders
Public Health & Medical Academy
 
L44 Ovarian cancer
L44 Ovarian cancer L44 Ovarian cancer
L44 Ovarian cancer
Public Health & Medical Academy
 
L43 Screening of cervical cancer
L43 Screening of cervical cancer L43 Screening of cervical cancer
L43 Screening of cervical cancer
Public Health & Medical Academy
 
L42 Miscarriage & Ectopic
L42 Miscarriage & Ectopic L42 Miscarriage & Ectopic
L42 Miscarriage & Ectopic
Public Health & Medical Academy
 
L41 Methods of Contraception
L41 Methods of ContraceptionL41 Methods of Contraception
L41 Methods of Contraception
Public Health & Medical Academy
 
L46 Pelvic Inflammatory Disease (PID)
L46 Pelvic Inflammatory Disease (PID)L46 Pelvic Inflammatory Disease (PID)
L46 Pelvic Inflammatory Disease (PID)
Public Health & Medical Academy
 
L40 Urinary Incontinence
L40 Urinary IncontinenceL40 Urinary Incontinence
L40 Urinary Incontinence
Public Health & Medical Academy
 
L39 Gynecological infections & ulcers
L39 Gynecological infections & ulcers L39 Gynecological infections & ulcers
L39 Gynecological infections & ulcers
Public Health & Medical Academy
 
L38 Abnormal uterine bleeding
L38 Abnormal uterine bleeding L38 Abnormal uterine bleeding
L38 Abnormal uterine bleeding
Public Health & Medical Academy
 
L37 POLYCYSTIC OVARY SYNDROME
L37 POLYCYSTIC OVARY SYNDROMEL37 POLYCYSTIC OVARY SYNDROME
L37 POLYCYSTIC OVARY SYNDROME
Public Health & Medical Academy
 
L36 Premenstrual syndrome
L36 Premenstrual syndrome L36 Premenstrual syndrome
L36 Premenstrual syndrome
Public Health & Medical Academy
 
L35 Endometriosis
L35 EndometriosisL35 Endometriosis
L34 Operative delivery
L34 Operative delivery L34 Operative delivery
L34 Operative delivery
Public Health & Medical Academy
 
L33 Induction of labor
L33 Induction of labor L33 Induction of labor
L33 Induction of labor
Public Health & Medical Academy
 
L32 Abnormal labor
L32 Abnormal labor L32 Abnormal labor
L32 Abnormal labor
Public Health & Medical Academy
 
L30 Perinatal Infections part 2
L30 Perinatal Infections part 2L30 Perinatal Infections part 2
L30 Perinatal Infections part 2
Public Health & Medical Academy
 

More from Public Health & Medical Academy (20)

L50 Prolapse
L50 ProlapseL50 Prolapse
L50 Prolapse
 
L49 Menopause
L49 MenopauseL49 Menopause
L49 Menopause
 
L48 Uterine Fibroids
L48 Uterine Fibroids L48 Uterine Fibroids
L48 Uterine Fibroids
 
L47 Amenorrhea
L47 AmenorrheaL47 Amenorrhea
L47 Amenorrhea
 
L45 Genital tract development and Puberty & their disorders
L45 Genital tract development and Puberty & their disordersL45 Genital tract development and Puberty & their disorders
L45 Genital tract development and Puberty & their disorders
 
L44 Ovarian cancer
L44 Ovarian cancer L44 Ovarian cancer
L44 Ovarian cancer
 
L43 Screening of cervical cancer
L43 Screening of cervical cancer L43 Screening of cervical cancer
L43 Screening of cervical cancer
 
L42 Miscarriage & Ectopic
L42 Miscarriage & Ectopic L42 Miscarriage & Ectopic
L42 Miscarriage & Ectopic
 
L41 Methods of Contraception
L41 Methods of ContraceptionL41 Methods of Contraception
L41 Methods of Contraception
 
L46 Pelvic Inflammatory Disease (PID)
L46 Pelvic Inflammatory Disease (PID)L46 Pelvic Inflammatory Disease (PID)
L46 Pelvic Inflammatory Disease (PID)
 
L40 Urinary Incontinence
L40 Urinary IncontinenceL40 Urinary Incontinence
L40 Urinary Incontinence
 
L39 Gynecological infections & ulcers
L39 Gynecological infections & ulcers L39 Gynecological infections & ulcers
L39 Gynecological infections & ulcers
 
L38 Abnormal uterine bleeding
L38 Abnormal uterine bleeding L38 Abnormal uterine bleeding
L38 Abnormal uterine bleeding
 
L37 POLYCYSTIC OVARY SYNDROME
L37 POLYCYSTIC OVARY SYNDROMEL37 POLYCYSTIC OVARY SYNDROME
L37 POLYCYSTIC OVARY SYNDROME
 
L36 Premenstrual syndrome
L36 Premenstrual syndrome L36 Premenstrual syndrome
L36 Premenstrual syndrome
 
L35 Endometriosis
L35 EndometriosisL35 Endometriosis
L35 Endometriosis
 
L34 Operative delivery
L34 Operative delivery L34 Operative delivery
L34 Operative delivery
 
L33 Induction of labor
L33 Induction of labor L33 Induction of labor
L33 Induction of labor
 
L32 Abnormal labor
L32 Abnormal labor L32 Abnormal labor
L32 Abnormal labor
 
L30 Perinatal Infections part 2
L30 Perinatal Infections part 2L30 Perinatal Infections part 2
L30 Perinatal Infections part 2
 

Recently uploaded

ACUTE SCROTUM.....pdf. ACUTE SCROTAL CONDITIOND
ACUTE SCROTUM.....pdf. ACUTE SCROTAL CONDITIONDACUTE SCROTUM.....pdf. ACUTE SCROTAL CONDITIOND
ACUTE SCROTUM.....pdf. ACUTE SCROTAL CONDITIOND
DR SETH JOTHAM
 
Pulmonary Thromboembolism - etilogy, types, medical- Surgical and nursing man...
Pulmonary Thromboembolism - etilogy, types, medical- Surgical and nursing man...Pulmonary Thromboembolism - etilogy, types, medical- Surgical and nursing man...
Pulmonary Thromboembolism - etilogy, types, medical- Surgical and nursing man...
VarunMahajani
 
ARTIFICIAL INTELLIGENCE IN HEALTHCARE.pdf
ARTIFICIAL INTELLIGENCE IN  HEALTHCARE.pdfARTIFICIAL INTELLIGENCE IN  HEALTHCARE.pdf
ARTIFICIAL INTELLIGENCE IN HEALTHCARE.pdf
Anujkumaranit
 
NVBDCP.pptx Nation vector borne disease control program
NVBDCP.pptx Nation vector borne disease control programNVBDCP.pptx Nation vector borne disease control program
NVBDCP.pptx Nation vector borne disease control program
Sapna Thakur
 
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Ve...
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Ve...TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Ve...
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Ve...
kevinkariuki227
 
Lung Cancer: Artificial Intelligence, Synergetics, Complex System Analysis, S...
Lung Cancer: Artificial Intelligence, Synergetics, Complex System Analysis, S...Lung Cancer: Artificial Intelligence, Synergetics, Complex System Analysis, S...
Lung Cancer: Artificial Intelligence, Synergetics, Complex System Analysis, S...
Oleg Kshivets
 
Pharynx and Clinical Correlations BY Dr.Rabia Inam Gandapore.pptx
Pharynx and Clinical Correlations BY Dr.Rabia Inam Gandapore.pptxPharynx and Clinical Correlations BY Dr.Rabia Inam Gandapore.pptx
Pharynx and Clinical Correlations BY Dr.Rabia Inam Gandapore.pptx
Dr. Rabia Inam Gandapore
 
263778731218 Abortion Clinic /Pills In Harare ,
263778731218 Abortion Clinic /Pills In Harare ,263778731218 Abortion Clinic /Pills In Harare ,
263778731218 Abortion Clinic /Pills In Harare ,
sisternakatoto
 
KDIGO 2024 guidelines for diabetologists
KDIGO 2024 guidelines for diabetologistsKDIGO 2024 guidelines for diabetologists
KDIGO 2024 guidelines for diabetologists
د.محمود نجيب
 
Ocular injury ppt Upendra pal optometrist upums saifai etawah
Ocular injury  ppt  Upendra pal  optometrist upums saifai etawahOcular injury  ppt  Upendra pal  optometrist upums saifai etawah
Ocular injury ppt Upendra pal optometrist upums saifai etawah
pal078100
 
For Better Surat #ℂall #Girl Service ❤85270-49040❤ Surat #ℂall #Girls
For Better Surat #ℂall #Girl Service ❤85270-49040❤ Surat #ℂall #GirlsFor Better Surat #ℂall #Girl Service ❤85270-49040❤ Surat #ℂall #Girls
For Better Surat #ℂall #Girl Service ❤85270-49040❤ Surat #ℂall #Girls
Savita Shen $i11
 
Alcohol_Dr. Jeenal Mistry MD Pharmacology.pdf
Alcohol_Dr. Jeenal Mistry MD Pharmacology.pdfAlcohol_Dr. Jeenal Mistry MD Pharmacology.pdf
Alcohol_Dr. Jeenal Mistry MD Pharmacology.pdf
Dr Jeenal Mistry
 
Charaka Samhita Sutra sthana Chapter 15 Upakalpaniyaadhyaya
Charaka Samhita Sutra sthana Chapter 15 UpakalpaniyaadhyayaCharaka Samhita Sutra sthana Chapter 15 Upakalpaniyaadhyaya
Charaka Samhita Sutra sthana Chapter 15 Upakalpaniyaadhyaya
Dr KHALID B.M
 
heat stroke and heat exhaustion in children
heat stroke and heat exhaustion in childrenheat stroke and heat exhaustion in children
heat stroke and heat exhaustion in children
SumeraAhmad5
 
BENIGN PROSTATIC HYPERPLASIA.BPH. BPHpdf
BENIGN PROSTATIC HYPERPLASIA.BPH. BPHpdfBENIGN PROSTATIC HYPERPLASIA.BPH. BPHpdf
BENIGN PROSTATIC HYPERPLASIA.BPH. BPHpdf
DR SETH JOTHAM
 
Ophthalmology Clinical Tests for OSCE exam
Ophthalmology Clinical Tests for OSCE examOphthalmology Clinical Tests for OSCE exam
Ophthalmology Clinical Tests for OSCE exam
KafrELShiekh University
 
New Drug Discovery and Development .....
New Drug Discovery and Development .....New Drug Discovery and Development .....
New Drug Discovery and Development .....
NEHA GUPTA
 
Hemodialysis: Chapter 3, Dialysis Water Unit - Dr.Gawad
Hemodialysis: Chapter 3, Dialysis Water Unit - Dr.GawadHemodialysis: Chapter 3, Dialysis Water Unit - Dr.Gawad
Hemodialysis: Chapter 3, Dialysis Water Unit - Dr.Gawad
NephroTube - Dr.Gawad
 
Cervical & Brachial Plexus By Dr. RIG.pptx
Cervical & Brachial Plexus By Dr. RIG.pptxCervical & Brachial Plexus By Dr. RIG.pptx
Cervical & Brachial Plexus By Dr. RIG.pptx
Dr. Rabia Inam Gandapore
 
Non-respiratory Functions of the Lungs.pdf
Non-respiratory Functions of the Lungs.pdfNon-respiratory Functions of the Lungs.pdf
Non-respiratory Functions of the Lungs.pdf
MedicoseAcademics
 

Recently uploaded (20)

ACUTE SCROTUM.....pdf. ACUTE SCROTAL CONDITIOND
ACUTE SCROTUM.....pdf. ACUTE SCROTAL CONDITIONDACUTE SCROTUM.....pdf. ACUTE SCROTAL CONDITIOND
ACUTE SCROTUM.....pdf. ACUTE SCROTAL CONDITIOND
 
Pulmonary Thromboembolism - etilogy, types, medical- Surgical and nursing man...
Pulmonary Thromboembolism - etilogy, types, medical- Surgical and nursing man...Pulmonary Thromboembolism - etilogy, types, medical- Surgical and nursing man...
Pulmonary Thromboembolism - etilogy, types, medical- Surgical and nursing man...
 
ARTIFICIAL INTELLIGENCE IN HEALTHCARE.pdf
ARTIFICIAL INTELLIGENCE IN  HEALTHCARE.pdfARTIFICIAL INTELLIGENCE IN  HEALTHCARE.pdf
ARTIFICIAL INTELLIGENCE IN HEALTHCARE.pdf
 
NVBDCP.pptx Nation vector borne disease control program
NVBDCP.pptx Nation vector borne disease control programNVBDCP.pptx Nation vector borne disease control program
NVBDCP.pptx Nation vector borne disease control program
 
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Ve...
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Ve...TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Ve...
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Ve...
 
Lung Cancer: Artificial Intelligence, Synergetics, Complex System Analysis, S...
Lung Cancer: Artificial Intelligence, Synergetics, Complex System Analysis, S...Lung Cancer: Artificial Intelligence, Synergetics, Complex System Analysis, S...
Lung Cancer: Artificial Intelligence, Synergetics, Complex System Analysis, S...
 
Pharynx and Clinical Correlations BY Dr.Rabia Inam Gandapore.pptx
Pharynx and Clinical Correlations BY Dr.Rabia Inam Gandapore.pptxPharynx and Clinical Correlations BY Dr.Rabia Inam Gandapore.pptx
Pharynx and Clinical Correlations BY Dr.Rabia Inam Gandapore.pptx
 
263778731218 Abortion Clinic /Pills In Harare ,
263778731218 Abortion Clinic /Pills In Harare ,263778731218 Abortion Clinic /Pills In Harare ,
263778731218 Abortion Clinic /Pills In Harare ,
 
KDIGO 2024 guidelines for diabetologists
KDIGO 2024 guidelines for diabetologistsKDIGO 2024 guidelines for diabetologists
KDIGO 2024 guidelines for diabetologists
 
Ocular injury ppt Upendra pal optometrist upums saifai etawah
Ocular injury  ppt  Upendra pal  optometrist upums saifai etawahOcular injury  ppt  Upendra pal  optometrist upums saifai etawah
Ocular injury ppt Upendra pal optometrist upums saifai etawah
 
For Better Surat #ℂall #Girl Service ❤85270-49040❤ Surat #ℂall #Girls
For Better Surat #ℂall #Girl Service ❤85270-49040❤ Surat #ℂall #GirlsFor Better Surat #ℂall #Girl Service ❤85270-49040❤ Surat #ℂall #Girls
For Better Surat #ℂall #Girl Service ❤85270-49040❤ Surat #ℂall #Girls
 
Alcohol_Dr. Jeenal Mistry MD Pharmacology.pdf
Alcohol_Dr. Jeenal Mistry MD Pharmacology.pdfAlcohol_Dr. Jeenal Mistry MD Pharmacology.pdf
Alcohol_Dr. Jeenal Mistry MD Pharmacology.pdf
 
Charaka Samhita Sutra sthana Chapter 15 Upakalpaniyaadhyaya
Charaka Samhita Sutra sthana Chapter 15 UpakalpaniyaadhyayaCharaka Samhita Sutra sthana Chapter 15 Upakalpaniyaadhyaya
Charaka Samhita Sutra sthana Chapter 15 Upakalpaniyaadhyaya
 
heat stroke and heat exhaustion in children
heat stroke and heat exhaustion in childrenheat stroke and heat exhaustion in children
heat stroke and heat exhaustion in children
 
BENIGN PROSTATIC HYPERPLASIA.BPH. BPHpdf
BENIGN PROSTATIC HYPERPLASIA.BPH. BPHpdfBENIGN PROSTATIC HYPERPLASIA.BPH. BPHpdf
BENIGN PROSTATIC HYPERPLASIA.BPH. BPHpdf
 
Ophthalmology Clinical Tests for OSCE exam
Ophthalmology Clinical Tests for OSCE examOphthalmology Clinical Tests for OSCE exam
Ophthalmology Clinical Tests for OSCE exam
 
New Drug Discovery and Development .....
New Drug Discovery and Development .....New Drug Discovery and Development .....
New Drug Discovery and Development .....
 
Hemodialysis: Chapter 3, Dialysis Water Unit - Dr.Gawad
Hemodialysis: Chapter 3, Dialysis Water Unit - Dr.GawadHemodialysis: Chapter 3, Dialysis Water Unit - Dr.Gawad
Hemodialysis: Chapter 3, Dialysis Water Unit - Dr.Gawad
 
Cervical & Brachial Plexus By Dr. RIG.pptx
Cervical & Brachial Plexus By Dr. RIG.pptxCervical & Brachial Plexus By Dr. RIG.pptx
Cervical & Brachial Plexus By Dr. RIG.pptx
 
Non-respiratory Functions of the Lungs.pdf
Non-respiratory Functions of the Lungs.pdfNon-respiratory Functions of the Lungs.pdf
Non-respiratory Functions of the Lungs.pdf
 

L31 Normal Labor & Delivery

  • 2. Introduction.. Labor :  regular uterine contractions of sufficient frequency, intensity, and duration that lead to progressive cervical dilatation, effacement and descent of presenting part.  3 Ps of labour: powers, the passages and the passenger.  If any of the 3Ps are unfavourable, labour is likely to be abnormal resulting in the need for intervention
  • 3. Maternal Anatomy • The pelvic inlet:  Transverse diameter is 13.5 cm > Anterior–posterior (A–P) diameter is 11.0 cm.  The fetal head enters the pelvis orientated in a transverse position in keeping with the wider transverse diameter.  The angle of the inlet is normally 60°
  • 4. Maternal Anatomy • The Midpelvis  It is almost round, as the transverse and A-P are similar at 12cm.  The ischial spines are palpable vaginally and are used as important landmarks to: 1. To assess the descent of the presenting part on vaginal examination (e.g. station 0 is at the level of the ischial spines) 2. To provide a local anaesthetic pudendal nerve block.
  • 5. Maternal Anatomy • The Pelvic Outlet  The transverse is the widest diameter at the inlet, but at the outlet it is the AP diameter  The fetal head must rotate from a transverse to an AP position as it passes through the pelvis
  • 7.
  • 8. Fetal Anatomy At the time of labour, the sutures joining the bones of the vault are soft, un-ossified membranes. The sutures of the fetal face and the skull base are firmly united.
  • 9. Fetal Anatomy Fetal skull moulding:  Sutures are not fixed which allows the bones to move together and even to overlap.  The parietal bones usually slide over the frontal and occipital bones  Moulding reduces the diameters of the fetal head and encourages progress through the bony pelvis, while still protecting the underlying brain
  • 10. The diameters of the skull
  • 11. Physiology of Labor • The cervix: softens, shortens, thins out (effacement) and dilates for labour to progress. • The uterus: changes from a state of relaxation to an active state of regular contractions to facilitate transit of the fetus through the birth canal. • Each contraction must be followed by a resting phase in order to maintain placental blood flow and adequate perfusion of the fetus. • The pressure of the presenting part on the pelvic floor muscles produces a maternal urge to push.
  • 12. Physiology of Labor  The uterus • Uterine contractions happens in response to an increase in intracellular calcium. • Prostaglandins and oxytocin increase intracellular free calcium ions. • Progressive shortening of the uterine smooth muscle cells is called retraction and occurs in the cells of the upper part of the uterus • Retraction results in the development of the thicker, actively contracting ‘upper segment’ and the lower segment of the uterus becomes thinner and more stretched
  • 13. • Contraction intervals of 2–4 minutes (i.e. 2 in 10 increasing to 4–5 in 10 in advanced labour) • Their duration varies from 30 to 60 seconds or longer. • The intensity or amplitude of the intrauterine pressure during a contraction averages between 30 and 60 mmHg. ( 200 MVU)
  • 14. Physiology of Labor  The cervix • Under the influence of prostaglandins, there is an increase in proteolytic activity and a reduction in collagen and elastin. • Dermatan sulphate is replaced by the more hydrophilic hyaluronic acid, which results in an increase in water content of the cervix. • Causing cervical softening or ‘ripening’, so that when contractions begin the processes of effacement and dilatation start.
  • 15. Diagnosis of labor The onset of labour can be defined as the presence of strong regular painful contractions resulting in progressive cervical change Loss of a ‘show’ or spontaneous rupture of the membranes (SROM) does not define the onset of labour
  • 16. Diagnosis of true labor pain  History • A history of regular painful uterine contraction in every 5- 8 min, accompanied by the history of a bloody show or spontaneous rupture of membrane  Physical examination • Reduction of interval between uterine contractions • Abdominal pain of increasing intensity • Cervical effacement (≥ 50%) • Cervical dilation (≥ 2 cm)
  • 17.
  • 18. Normal labor Spontaneous onset Single cephalic presentation. 37-42 weeks of gestation No artificial interventions. Unassisted spontaneous vaginal delivery. Duration of <12 hours in nulliparous women, and <8 hours in multiparous women.
  • 19. Stages Of Labor  There are three stages of labor: • The first stage (stage of dilatation of the cervix) is from the onset of true labor (regular uterine contractions) to complete dilatation of the cervix • The second stage (stage of fetal delivery ) is from complete dilatation of the cervix to the birth of the baby • The third stage (stage of placental delivery) is from the birth of the baby to delivery of the placenta.
  • 20. Stages Of Labor First stage • It is the stage of cervical dilatation. • Starts with the onset of true labor pain and ends with full dilatation of the cervix i.e. 10 cm in diameter. • It takes about 10-14 hours in primigravida and about 6-8 hours in multipara
  • 21. Phases of cervical dilatation Latent phase: • This is the first 3 cm of cervical dilatation which is slow takes about 8 hours in nullipara and 4 hours in multipara. Active phase: • The time between the end of the latent phase (3–4 cm dilatation) and full cervical dilatation (10 cm) • Lasting between 2 and 6 hours, shorter in multiparous women • Cervical dilatation during the active phase occurs typically at 1 cm/hour or more • It is considered abnormal if it occurs at less than 1 cm in 2 hours.
  • 22. Stages Of Labor Second stage • It is the stage of expulsion of the fetus. • Begins with full cervical dilatation and ends with the delivery of the fetus. • Its duration is about 50 minutes in primigravida and 20minutes in multipara • considered prolonged if more than one hour in multiparas and more than 2 hours in primigravidea
  • 23. Phases Of Second stage of Labor  The ‘passive phase’ • The time between full dilatation and the onset of involuntary expulsive contractions • There is no maternal urge to push and the fetal head is still relatively high in the pelvis.  The second phase ‘active second stage’. • There is a maternal urge to push because the fetal head is low (often visible), causing a reflex need to ‘bear down’ • normal active second stage should last no longer than 2 hours in a nulliparous woman and 1 hour in multipara
  • 24. Stages Of Labor  Third stage • It is the stage of expulsion of the placenta and membranes. • Begins after delivery of the fetus and ends with expulsion of the placenta and membranes. • Its duration is about 10-20 minutes in both primi and multipara. normally not exceed 30 minutes with active management of third stage
  • 25. Duration of Labor  Prolonged Labour • labour lasting longer than 12 hours in nulliparous women and 8 hours in multiparous women.  Precipitous labour • defined as expulsion of the fetus within less than 3 hours of the onset of regular contractions.
  • 26. Mechanisms of labor  Fetal lie : The relation of the fetal long axis to that of the mother • Longitudinal • Transverse • Oblique • A longitudinal lie is present in more than 99% of labors at term. • Predisposing factors for transverse fetal position include multiparity, placenta previa, hydramnios
  • 27. Mechanisms of labor  Fetal presentation : • The portion of the fetal body that is either foremost within the birth canal or in the closest proximity to it. • Cephalic 96.8% • breech 2.7 % • Shoulder 0.3% • Compound 0.1%
  • 28. Mechanisms of labor  Cephalic presentation • Well flexed head (vertex or occiput presentation) • Sinciput presentation (Military ) === transient • Brow presentation 0.01 % === transient • Face presentation 0.05% • As labor progresses, sinciput and brow presentations almost always convert into vertex or face presentations by neck flexion or extension, respectively.
  • 29.  Fetal Attitude:  Position of head with regard to fetal spine (ie: degree of flexion or extension) • A - suboccipitobragmatic (vertex) • B - occipitofrontal (military) • C - mentovertical (brow ) • D - submintobragmatic (face) ** OP with occipitofrontal 11.5 Or suboccipitofrontal 10
  • 30.
  • 31. Breech presentation  Incidence: • 25 % 28 weeks GA • 17% 30 weeks GA • 11% 32 weeks GA • 2.7 % term
  • 32.
  • 33. Presenting parts diameter in cephalic presentation
  • 34. Mechanisms of labor Fetal position • Relation ship of chosen portion of the fetal presenting part to the right or left side of the birth canal (ROA, LOA , ROP, RMA…..) • DOMINATOPRS:  Vertex  Face  Breech  Shoulder • Occiput • Mentum • Sacrum • Acromiun
  • 35.
  • 36.
  • 37.
  • 38.
  • 39. Diagnosis Of Fetal Presentation & Position • Abdominal palpation • Vaginal examination: - Presenting part - Position - Dilatation - Effacement - Station - Pelvimetry • Auscultation : arounf the umbilicus in longitdunal lie , below if cephalic and above if breech
  • 40.
  • 41. Vaginal Examination Head station : Relationship of presenting part to the ischial spine
  • 43. The Mechanism Of Labor (OA)  Cardinal movements of labor 1. Engagement 2. Descent 3. Flexion 4. Internal Rotation 5. Extension 6. Restitution And External Rotation 7. Expulsion
  • 44. The Mechanism Of Labor  Engagement • Passage of widest diameter of presenting(in transverse position) part to level below the plane of the pelvic inlet • Occurs earlier in nulliparous women (36 wks)
  • 45. The Mechanism Of Labor  Descent • Descent of the fetal head is needed before flexion, internal rotation and extension can occur  Flexion • Occurs passively As the • head descends into the narrower midpelvis
  • 46. The Mechanism Of Labor  Internal Rotation  Rotation of presenting part from original position (transverse) to anteroposterior position  If the head is well flexed, the occiput will be the leading point
  • 47. The Mechanism Of Labor  Extension The well-flexed head now extends and the occiput escapes from underneath the symphysis pubis and distends the vulva. This is known as ‘crowning’ of the head.
  • 48.  Restitution This slight rotation of the occiput through one eighth of the circle The Mechanism Of Labor  External rotation The shoulders have to rotate into the direct AP plane, Then the occiput rotates through a further one-eighth of a circle to the transverse position.
  • 49. The Mechanism Of Labor  Expulsion The anterior shoulder is under the symphysis pubis and delivers first, and the posterior shoulder delivers subsequently.
  • 50. In occipito-posterior Mechanism of labour is identical to OT & anterior varieties The occiput rotate to the symphysis pubis through 135º instead of 90º or 45º If rotation does not occur direct occiput post or Partial rotation transverse arrest
  • 51. Management In First Stage  Vaginal examinations are usually performed every 4 hours to determine when the active phase has been reached  Laboring woman should be allowed to assume the position she finds most comfortable— this will be lateral recumbency most of the time  Sips of clear liquids, occasional ice chips are permitted
  • 52. Management In Second Stage  The first sign of the second stage is likely to be an urge to push.  In all cases the baby should be delivered within 4 hours of reaching full dilatation  Once the head has crowned, the perineum should be supported the woman should be discouraged from bearing down by telling her to take rapid, shallow breaths  To aid delivery of the shoulders, there should be gentle traction on the head downwards and forwards until the anterior shoulder appears beneath the pubis
  • 53.
  • 54. Fetal Care Fetal monitoring: • Low risk : every 30 min in first stage and 15 min in second stage • High risk : every 15 min in first stage and every 5 min in the second stage or continuous
  • 55. Episiotomy It is a second degree pereneal tear man made by scissors Types: Midline, medio-lateral and lateral, Mediolateral is commonly used. Indication: complicated vaginal delivery (breech, shoulder dystocia, forceps, vacuum), scarring from female genital cutting or poorly healed third or fourth degree tears and fetal distress.
  • 56. Episiotomy Wait to perform episiotomy until: the perineum is thinned out; and 3-4 cm of the baby's head is visible during a contraction Complications: Hematoma, infection and rarly necrotizing fasciitis Episiotomy should be restricted to an indication
  • 57.
  • 59. Management In Third Stage  Management of the third stage can be ‘active’ or ‘physiological’.  Physiological management • Is where the placenta is delivered by maternal effort and no uterotonic drugs are given to assist this process  Active management • When the signs of placental separation are recognized, controlled cord traction is used to expedite delivery of the placenta.
  • 61. Oxytocin dose is 10 IU, intramuscularly. with intravenous access in place, 10-20 IU is placed in 500-1000 mL of crystalloid and run quickly. With cesarean deliveries, 5 IU is administered as an intravenous bolus, followed by a similar infusion. Ergometrine dose: is 0.2-0.25 mg, some used 0.5 mg ; IM or IV. Syntometrine (contains 0.5 mg of ergometrine with 5 IU of oxytocin); Oxytocin and ergometrine acts within 30 seonds if given IV and oxytocin acts after 3 mints while ergometrine after 6-8 mints if given IM Management In Third Stage
  • 62. Advantages of Active Management  Advantage: • reduction of the blood loss (60% reduction of blood loss )  Disadvantages: • Constriction ring may occur with retention of the placenta. • Avulsion of the cord if undue pressure is applied. • Inversion of the uterus if fundus is pressed while the uterus is lax.
  • 63. Placental Examination Examination of the placenta & membranes: • by exploring it on a plain surface to be sure that it is complete. If there is missed part, exploration of the uterus is done under general anaesthesia.