This document provides guidance on performing an intraoral examination. It discusses examining the lips, labial mucosa, buccal mucosa, tongue, hard palate, soft palate, floor of mouth, and periodontium. For each structure, it describes the clinical examination steps of inspection and palpation to identify any abnormalities, lesions, or signs of disease. Common pathological conditions are also listed for each intraoral site. The document emphasizes performing a thorough intraoral soft tissue and periodontal examination to diagnose any oral diseases or issues.
1. Extraoral examination involves visual examination of the face, jaw, and lymph nodes to assess facial symmetry, lip seal, the temporomandibular joint, and profile.
2. Examination of the lymph nodes helps evaluate for enlargement which can suggest underlying pathology, while examination of the temporomandibular joint assesses sounds and tenderness that may indicate internal abnormalities.
3. Assessment of the facial profile and lip seal can help identify malocclusions and large masses that may be present intraorally.
principles of instrumentation of hand instrumentsfiza shameem
This document discusses principles of periodontal instrumentation. It covers topics like accessibility, visibility, instrument condition, maintaining a clean field, instrument stabilization, and activation techniques. For accessibility, it discusses patient and operator positioning. For visibility, it discusses illumination, retraction, and use of mirrors. It emphasizes the importance of sharp, clean instruments and using suction and air to maintain a clear field. It describes different instrument grasps and finger rests for stabilization. Finally, it outlines techniques for instrument adaptation, angulation, pressure, and different stroke types like exploratory, scaling, and root planing.
This document discusses pulp polyps and gingival polyps. Pulp polyps are caused by irreversible pulpitis and the inflammation of the dental pulp due to extensive decay. They typically occur in molars and present as a reddish granulation tissue mass in the pulp chamber. Gingival polyps are localized enlargements of the gums that can be caused by inflammation, medications, systemic diseases, or neoplasms. They present as red, swollen, tender gums that may bleed and cover the teeth. Both conditions are usually diagnosed based on their appearance and location. Pulp polyps require endodontic therapy or extraction, while gingival polyps can be treated by removing the cause of enlargement
This document provides information on gingival and periodontal diseases. It discusses various types of gingival diseases like gingivitis, acute gingival diseases, gingival enlargement. It also covers different types of periodontal diseases like chronic periodontitis, aggressive periodontitis. Periodontal diseases are infectious diseases that cause destruction of tissues supporting the teeth. Nearly 75% of adults suffer from some form of periodontal disease. Early detection and treatment can help most people keep their teeth for life.
The gingiva is the mucosa that covers the alveolar bone and surrounds the necks of teeth. It consists of marginal gingiva, attached gingiva, and interdental gingiva. The marginal gingiva forms a collar around each tooth. The attached gingiva is firm and resilient, attaching the gingiva tightly to the underlying bone. The interdental gingiva occupies the spaces between teeth. Microscopically, the gingiva contains keratinized oral epithelium, non-keratinized sulcular epithelium, and junctional epithelium that extends along the tooth surface. It has a dense connective tissue layer supplied by blood vessels and nerves.
This document provides an overview of abscesses of the periodontium, specifically focusing on periodontal abscesses. It defines a periodontal abscess and classifies them based on location, course, number, affected tissue, and cause. Periodontal abscesses are most prevalent in molar sites and those with pre-existing periodontal pockets. They can be caused by factors like untreated periodontitis, foreign bodies, or changes after periodontal procedures or antibiotics. The pathogenesis involves bacterial entry triggering an inflammatory response that leads to tissue destruction and pus formation.
Leukoplakia is a white patch or plaque that develops in the mouth and cannot be wiped away. It affects 1.5-12% of the population, usually those over age 40, and prevalence increases with age. Leukoplakia has various clinical forms and ranges in appearance from flat and uniform to raised or irregular patches. A biopsy is needed to examine the tissue for signs of dysplasia or oral cancer. While most leukoplakia is harmless, some may develop into cancer over time, so prevention focuses on lifestyle changes like quitting smoking and reducing alcohol.
This document provides guidance on performing an intraoral examination. It discusses examining the lips, labial mucosa, buccal mucosa, tongue, hard palate, soft palate, floor of mouth, and periodontium. For each structure, it describes the clinical examination steps of inspection and palpation to identify any abnormalities, lesions, or signs of disease. Common pathological conditions are also listed for each intraoral site. The document emphasizes performing a thorough intraoral soft tissue and periodontal examination to diagnose any oral diseases or issues.
1. Extraoral examination involves visual examination of the face, jaw, and lymph nodes to assess facial symmetry, lip seal, the temporomandibular joint, and profile.
2. Examination of the lymph nodes helps evaluate for enlargement which can suggest underlying pathology, while examination of the temporomandibular joint assesses sounds and tenderness that may indicate internal abnormalities.
3. Assessment of the facial profile and lip seal can help identify malocclusions and large masses that may be present intraorally.
principles of instrumentation of hand instrumentsfiza shameem
This document discusses principles of periodontal instrumentation. It covers topics like accessibility, visibility, instrument condition, maintaining a clean field, instrument stabilization, and activation techniques. For accessibility, it discusses patient and operator positioning. For visibility, it discusses illumination, retraction, and use of mirrors. It emphasizes the importance of sharp, clean instruments and using suction and air to maintain a clear field. It describes different instrument grasps and finger rests for stabilization. Finally, it outlines techniques for instrument adaptation, angulation, pressure, and different stroke types like exploratory, scaling, and root planing.
This document discusses pulp polyps and gingival polyps. Pulp polyps are caused by irreversible pulpitis and the inflammation of the dental pulp due to extensive decay. They typically occur in molars and present as a reddish granulation tissue mass in the pulp chamber. Gingival polyps are localized enlargements of the gums that can be caused by inflammation, medications, systemic diseases, or neoplasms. They present as red, swollen, tender gums that may bleed and cover the teeth. Both conditions are usually diagnosed based on their appearance and location. Pulp polyps require endodontic therapy or extraction, while gingival polyps can be treated by removing the cause of enlargement
This document provides information on gingival and periodontal diseases. It discusses various types of gingival diseases like gingivitis, acute gingival diseases, gingival enlargement. It also covers different types of periodontal diseases like chronic periodontitis, aggressive periodontitis. Periodontal diseases are infectious diseases that cause destruction of tissues supporting the teeth. Nearly 75% of adults suffer from some form of periodontal disease. Early detection and treatment can help most people keep their teeth for life.
The gingiva is the mucosa that covers the alveolar bone and surrounds the necks of teeth. It consists of marginal gingiva, attached gingiva, and interdental gingiva. The marginal gingiva forms a collar around each tooth. The attached gingiva is firm and resilient, attaching the gingiva tightly to the underlying bone. The interdental gingiva occupies the spaces between teeth. Microscopically, the gingiva contains keratinized oral epithelium, non-keratinized sulcular epithelium, and junctional epithelium that extends along the tooth surface. It has a dense connective tissue layer supplied by blood vessels and nerves.
This document provides an overview of abscesses of the periodontium, specifically focusing on periodontal abscesses. It defines a periodontal abscess and classifies them based on location, course, number, affected tissue, and cause. Periodontal abscesses are most prevalent in molar sites and those with pre-existing periodontal pockets. They can be caused by factors like untreated periodontitis, foreign bodies, or changes after periodontal procedures or antibiotics. The pathogenesis involves bacterial entry triggering an inflammatory response that leads to tissue destruction and pus formation.
Leukoplakia is a white patch or plaque that develops in the mouth and cannot be wiped away. It affects 1.5-12% of the population, usually those over age 40, and prevalence increases with age. Leukoplakia has various clinical forms and ranges in appearance from flat and uniform to raised or irregular patches. A biopsy is needed to examine the tissue for signs of dysplasia or oral cancer. While most leukoplakia is harmless, some may develop into cancer over time, so prevention focuses on lifestyle changes like quitting smoking and reducing alcohol.
This document discusses several pathologies that can affect the jaws, including:
1. The adenomatoid odontogenic tumor, which presents as a swelling in young patients around unerupted teeth and consists of epithelial cells and calcifications.
2. The calcifying epithelial odontogenic tumor, which occurs in the mandible or maxilla as a radiolucent lesion containing radiopacities from calcification.
3. Odontomas, which are hamartomas containing dental tissues like enamel and dentin that appear as radiopaque masses and require conservative excision.
This document discusses dental diagnosis procedures, including:
- Provisional diagnosis is an initial diagnosis based on clinical impression without tests, from which further investigation is planned. Differential diagnosis lists conditions resembling the clinical diagnosis but differing in at least one feature.
- Investigations like blood tests, imaging, and biopsies help confirm the clinical diagnosis, detect suspected illnesses, and modify the treatment plan.
- The final diagnosis is made after investigations and identifies the primary complaint and any other issues, allowing suitable treatment to be planned.
This document discusses various developmental disturbances that can affect the tongue, including microglossia (small tongue), macroglossia (large tongue), ankyloglossia (tongue tie), cleft tongue, fissured tongue, median rhomboid glossitis (reddish patch on dorsal tongue), benign migratory glossitis (geographic tongue), hairy tongue, lingual varices (dilated veins on tongue), and lingual thyroid nodule (thyroid tissue on tongue). Many of these conditions can cause difficulties with speech, swallowing, or irritation of the tongue. Treatment may include surgery, antifungal medications, or thyroid hormone supplements.
Leukoplakia is a precancerous white lesion that occurs on mucous membranes in the mouth or other areas. It is caused by thickening of the keratin layer in the epithelium, which makes the tissue appear white. Tobacco use is the most common cause. Leukoplakia may appear as a homogeneous white patch or have mixed white and red areas (erythroleukoplakia), and the most common sites are the inside of the cheeks, gums, and tongue. A biopsy is needed to examine the tissue for signs of dysplasia and rule out other causes. While most cases remain benign, leukoplakia has an increased risk of transforming into oral cancer
Periodontitis is a chronic, slowly progressing disease which mainly results in the destruction of tooth supporting apparatus. Earlier it was classified as Chronic and Aggressive periodontitis with different clinical features and etiology. Current classification ( 2017) of periodontal disease involves periodontitis with is further divided into 4 stages and 3 grades depending on severity and rate of disease progression respectively. Diabetes meelitus and smoking are the validated risk factors for the progression of periodontitis.
Gingivitis is the most common form of gingival disease and is caused by bacterial infection resulting in inflammation of the gingival tissue. The characteristics of gingivitis include redness, swelling, bleeding upon provocation, and a change in consistency but no loss of attachment or bone loss. Gingivitis can be classified as dental plaque-induced or non-plaque induced. Dental plaque-induced gingivitis can be modified by local factors, systemic factors, medications, or malnutrition. Non-plaque induced gingivitis can result from bacterial, viral, fungal infections or genetic conditions.
This document provides information on oral submucous fibrosis (OSMF), including its definition, history, pathogenesis, clinical features, and staging classifications. OSMF is a chronic disease affecting the oral cavity and sometimes pharynx, characterized by juxtaepithelial inflammatory reaction and fibrosis of the lamina propria. Chewing betel quid and areca nut releases alkaloids and tannins that cause irritation and inflammation, activating fibroblasts and increasing production and cross-linking of collagen over time, resulting in stiffness and inability to open the mouth. OSMF is considered a precancerous condition due to its association with oral cancer. Staging systems describe progression from initial symptoms to trismus and possible malignancy
This document discusses dental elevators used for tooth extractions. It describes different types of elevators based on their design, principles of use, and applications. Straight, triangular, and pick-up elevators are discussed. Elevators remove whole teeth, roots, or root fragments using lever, wedge, and wheel/axle principles. Proper technique involves supporting the jaws, directing force along the tooth axis, and using finger guards to control forces and prevent damage to adjacent tissues.
The document provides guidelines for using the Basic Periodontal Examination (BPE) screening tool to assess periodontal health. The BPE involves probing 6 sites around each tooth to measure pocket depth and assign a code for each tooth section. Codes indicate the level of care needed, from oral hygiene instruction (Code 0-1) to non-surgical treatment (Code 2-3) to referral for specialized treatment (Code 4). While not diagnostic, the BPE gives a minimum standard for initial assessment and indicates if further detailed examination is required. Practitioners should use clinical judgment and consider unique patient factors when interpreting BPE scores.
The gingiva is divided anatomically into the marginal, attached, and interdental gingiva. The marginal gingiva forms the soft tissue wall around the teeth. The attached gingiva is firmly bound to the underlying bone. The interdental gingiva occupies the spaces between teeth. Microscopically, the gingiva contains an epithelial layer and underlying connective tissue. The epithelial layer includes the sulcular, junctional, and oral epithelium. The connective tissue contains collagen, fibroblasts, and ground substance.
Tooth mobility refers to loose teeth that can move within their sockets. It is classified on a scale of 0 to 3 based on the degree of horizontal and vertical movement. Physiologic mobility of about 0.25mm is normal, while pathologic mobility over 1mm indicates loose teeth from periodontal disease or trauma. Periodontal disease is a primary cause as it leads to loss of attachment and bone supporting the teeth. Treatment involves splinting loose teeth together, replacing missing teeth, and correcting occlusal surfaces to reduce excessive forces. For advanced periodontal cases, extraction may be necessary.
Calculus forms in layers on teeth through the mineralization of dental plaque. It consists of inorganic minerals like hydroxyapatite and organic components from bacteria and saliva. Factors like diet, age, habits, and saliva composition can affect the rate of calculus formation. Calculus is classified as supragingival or subgingival based on its location relative to the gingiva. Both types consist of calcium phosphate crystals embedded in an organic matrix but subgingival calculus has a higher mineral content. Calculus formation occurs through the precipitation and accumulation of minerals within the matrix over time.
A presentation on the instructions to be given to complete denture patients at the insertion appointment. Dealing with patients can be hard at times but with a proper approach, a strong rapport can be formed with the patient.
Dr. Nael Al Masri provides a comprehensive overview of periodontal examination and diagnosis. The summary includes:
1. A periodontal examination involves assessing the patient's medical and dental history, examining plaque, gingiva, probing depths, clinical attachment levels, bleeding, bone loss, tooth mobility, and furcation involvement.
2. Key findings that help determine a diagnosis include probing depths above 3mm, clinical attachment loss, bleeding on probing, recession, furcation involvement, and tooth mobility.
3. The examination is used to establish a diagnosis, prognosis, and develop a customized treatment plan for the patient.
Taurodontism is a condition where the body of the tooth is enlarged at the expense of the root, resulting in an elongated tooth body and short roots. It is caused by a failure of the epithelial root sheath to invaginate properly during tooth development. Taurodontism can range from mild to severe and may affect deciduous or permanent molars either unilaterally or bilaterally. Radiographically, there is an extremely large pulp chamber that extends into the elongated tooth body and short, wide roots. Diagnosis is based on the characteristic rectangular tooth crown and large pulp chamber seen clinically or radiographically. No treatment is needed as taurodontism does not typically cause clinical problems.
A simple Presentation Created by me in 2008, titled Intra-oral Examination.
its light heart-ed and fun to watch...
It contains some images of the most common lesions you might face during oral examination.
This document discusses acute apical abscess, which is a severe localized inflammatory condition characterized by the formation of pus around the apex of a tooth. The most common cause is bacterial invasion of the dental pulp from tooth decay. Clinically, it presents as acute pain that is worsened by pressure, percussion or palpation. Diagnosis involves a dental examination and x-rays. Emergency management involves establishing drainage to relieve pain, either through root canals or surgical drainage. After drainage is achieved, root canal treatment should be performed to thoroughly clean and disinfect the canals and remove the source of infection. Antibiotics may be prescribed in some cases but are generally not needed if adequate drainage is established.
This document provides information on describing gingival characteristics. It discusses the normal color, size, consistency, contour, surface texture, and position of gingiva. It describes changes seen in these characteristics due to various inflammatory and non-inflammatory conditions. Treatment approaches for conditions that alter gingival characteristics are also summarized, such as procedures for depigmentation and techniques for treating gingival recession.
This document provides guidance on performing an intra-oral examination. It discusses examining different areas of the mouth including the lips, labial mucosa, buccal mucosa, buccal vestibule, tongue, hard palate, soft palate, uvula, oropharynx, floor of the mouth, and teeth. Common lesions, abnormalities, and examination techniques are described for each area. The document also provides details on evaluating the dental pulp through tests like heat, cold, and electrical pulp testing as well as using transillumination to examine for cracked teeth.
This document discusses several pathologies that can affect the jaws, including:
1. The adenomatoid odontogenic tumor, which presents as a swelling in young patients around unerupted teeth and consists of epithelial cells and calcifications.
2. The calcifying epithelial odontogenic tumor, which occurs in the mandible or maxilla as a radiolucent lesion containing radiopacities from calcification.
3. Odontomas, which are hamartomas containing dental tissues like enamel and dentin that appear as radiopaque masses and require conservative excision.
This document discusses dental diagnosis procedures, including:
- Provisional diagnosis is an initial diagnosis based on clinical impression without tests, from which further investigation is planned. Differential diagnosis lists conditions resembling the clinical diagnosis but differing in at least one feature.
- Investigations like blood tests, imaging, and biopsies help confirm the clinical diagnosis, detect suspected illnesses, and modify the treatment plan.
- The final diagnosis is made after investigations and identifies the primary complaint and any other issues, allowing suitable treatment to be planned.
This document discusses various developmental disturbances that can affect the tongue, including microglossia (small tongue), macroglossia (large tongue), ankyloglossia (tongue tie), cleft tongue, fissured tongue, median rhomboid glossitis (reddish patch on dorsal tongue), benign migratory glossitis (geographic tongue), hairy tongue, lingual varices (dilated veins on tongue), and lingual thyroid nodule (thyroid tissue on tongue). Many of these conditions can cause difficulties with speech, swallowing, or irritation of the tongue. Treatment may include surgery, antifungal medications, or thyroid hormone supplements.
Leukoplakia is a precancerous white lesion that occurs on mucous membranes in the mouth or other areas. It is caused by thickening of the keratin layer in the epithelium, which makes the tissue appear white. Tobacco use is the most common cause. Leukoplakia may appear as a homogeneous white patch or have mixed white and red areas (erythroleukoplakia), and the most common sites are the inside of the cheeks, gums, and tongue. A biopsy is needed to examine the tissue for signs of dysplasia and rule out other causes. While most cases remain benign, leukoplakia has an increased risk of transforming into oral cancer
Periodontitis is a chronic, slowly progressing disease which mainly results in the destruction of tooth supporting apparatus. Earlier it was classified as Chronic and Aggressive periodontitis with different clinical features and etiology. Current classification ( 2017) of periodontal disease involves periodontitis with is further divided into 4 stages and 3 grades depending on severity and rate of disease progression respectively. Diabetes meelitus and smoking are the validated risk factors for the progression of periodontitis.
Gingivitis is the most common form of gingival disease and is caused by bacterial infection resulting in inflammation of the gingival tissue. The characteristics of gingivitis include redness, swelling, bleeding upon provocation, and a change in consistency but no loss of attachment or bone loss. Gingivitis can be classified as dental plaque-induced or non-plaque induced. Dental plaque-induced gingivitis can be modified by local factors, systemic factors, medications, or malnutrition. Non-plaque induced gingivitis can result from bacterial, viral, fungal infections or genetic conditions.
This document provides information on oral submucous fibrosis (OSMF), including its definition, history, pathogenesis, clinical features, and staging classifications. OSMF is a chronic disease affecting the oral cavity and sometimes pharynx, characterized by juxtaepithelial inflammatory reaction and fibrosis of the lamina propria. Chewing betel quid and areca nut releases alkaloids and tannins that cause irritation and inflammation, activating fibroblasts and increasing production and cross-linking of collagen over time, resulting in stiffness and inability to open the mouth. OSMF is considered a precancerous condition due to its association with oral cancer. Staging systems describe progression from initial symptoms to trismus and possible malignancy
This document discusses dental elevators used for tooth extractions. It describes different types of elevators based on their design, principles of use, and applications. Straight, triangular, and pick-up elevators are discussed. Elevators remove whole teeth, roots, or root fragments using lever, wedge, and wheel/axle principles. Proper technique involves supporting the jaws, directing force along the tooth axis, and using finger guards to control forces and prevent damage to adjacent tissues.
The document provides guidelines for using the Basic Periodontal Examination (BPE) screening tool to assess periodontal health. The BPE involves probing 6 sites around each tooth to measure pocket depth and assign a code for each tooth section. Codes indicate the level of care needed, from oral hygiene instruction (Code 0-1) to non-surgical treatment (Code 2-3) to referral for specialized treatment (Code 4). While not diagnostic, the BPE gives a minimum standard for initial assessment and indicates if further detailed examination is required. Practitioners should use clinical judgment and consider unique patient factors when interpreting BPE scores.
The gingiva is divided anatomically into the marginal, attached, and interdental gingiva. The marginal gingiva forms the soft tissue wall around the teeth. The attached gingiva is firmly bound to the underlying bone. The interdental gingiva occupies the spaces between teeth. Microscopically, the gingiva contains an epithelial layer and underlying connective tissue. The epithelial layer includes the sulcular, junctional, and oral epithelium. The connective tissue contains collagen, fibroblasts, and ground substance.
Tooth mobility refers to loose teeth that can move within their sockets. It is classified on a scale of 0 to 3 based on the degree of horizontal and vertical movement. Physiologic mobility of about 0.25mm is normal, while pathologic mobility over 1mm indicates loose teeth from periodontal disease or trauma. Periodontal disease is a primary cause as it leads to loss of attachment and bone supporting the teeth. Treatment involves splinting loose teeth together, replacing missing teeth, and correcting occlusal surfaces to reduce excessive forces. For advanced periodontal cases, extraction may be necessary.
Calculus forms in layers on teeth through the mineralization of dental plaque. It consists of inorganic minerals like hydroxyapatite and organic components from bacteria and saliva. Factors like diet, age, habits, and saliva composition can affect the rate of calculus formation. Calculus is classified as supragingival or subgingival based on its location relative to the gingiva. Both types consist of calcium phosphate crystals embedded in an organic matrix but subgingival calculus has a higher mineral content. Calculus formation occurs through the precipitation and accumulation of minerals within the matrix over time.
A presentation on the instructions to be given to complete denture patients at the insertion appointment. Dealing with patients can be hard at times but with a proper approach, a strong rapport can be formed with the patient.
Dr. Nael Al Masri provides a comprehensive overview of periodontal examination and diagnosis. The summary includes:
1. A periodontal examination involves assessing the patient's medical and dental history, examining plaque, gingiva, probing depths, clinical attachment levels, bleeding, bone loss, tooth mobility, and furcation involvement.
2. Key findings that help determine a diagnosis include probing depths above 3mm, clinical attachment loss, bleeding on probing, recession, furcation involvement, and tooth mobility.
3. The examination is used to establish a diagnosis, prognosis, and develop a customized treatment plan for the patient.
Taurodontism is a condition where the body of the tooth is enlarged at the expense of the root, resulting in an elongated tooth body and short roots. It is caused by a failure of the epithelial root sheath to invaginate properly during tooth development. Taurodontism can range from mild to severe and may affect deciduous or permanent molars either unilaterally or bilaterally. Radiographically, there is an extremely large pulp chamber that extends into the elongated tooth body and short, wide roots. Diagnosis is based on the characteristic rectangular tooth crown and large pulp chamber seen clinically or radiographically. No treatment is needed as taurodontism does not typically cause clinical problems.
A simple Presentation Created by me in 2008, titled Intra-oral Examination.
its light heart-ed and fun to watch...
It contains some images of the most common lesions you might face during oral examination.
This document discusses acute apical abscess, which is a severe localized inflammatory condition characterized by the formation of pus around the apex of a tooth. The most common cause is bacterial invasion of the dental pulp from tooth decay. Clinically, it presents as acute pain that is worsened by pressure, percussion or palpation. Diagnosis involves a dental examination and x-rays. Emergency management involves establishing drainage to relieve pain, either through root canals or surgical drainage. After drainage is achieved, root canal treatment should be performed to thoroughly clean and disinfect the canals and remove the source of infection. Antibiotics may be prescribed in some cases but are generally not needed if adequate drainage is established.
This document provides information on describing gingival characteristics. It discusses the normal color, size, consistency, contour, surface texture, and position of gingiva. It describes changes seen in these characteristics due to various inflammatory and non-inflammatory conditions. Treatment approaches for conditions that alter gingival characteristics are also summarized, such as procedures for depigmentation and techniques for treating gingival recession.
This document provides guidance on performing an intra-oral examination. It discusses examining different areas of the mouth including the lips, labial mucosa, buccal mucosa, buccal vestibule, tongue, hard palate, soft palate, uvula, oropharynx, floor of the mouth, and teeth. Common lesions, abnormalities, and examination techniques are described for each area. The document also provides details on evaluating the dental pulp through tests like heat, cold, and electrical pulp testing as well as using transillumination to examine for cracked teeth.
This document provides an overview of principles of oral diagnosis. It discusses various signs and symptoms that may be observed during an oral examination, including finger clubbing, cyanosis, and icterus as signs of systemic diseases. It describes examining the teeth, oral mucosa, tongue, lips, palate, and gingiva for lesions, disorders, and diseases. Common oral lesions discussed include leukoplakia, candidiasis, lichen planus, and oral cancer. The document also covers examining the temporomandibular joint and radiographs that may be used, such as panoramic x-rays and MRI. Various causes of oral pigmentation are summarized as well.
OSMF Oral Sub mucous Fibrosis -Oral MedicineSarahFanny2
1. OSMF (oral submucous fibrosis) has many etiological factors including chillies, tobacco, lime, and betel nut which act as local irritants and can cause vesicle formation, ulceration, and damage of the oral mucosa.
2. Clinically, OSMF presents with blanching of the oral mucosa, difficulty opening the mouth due to trismus, and formation of fibrous bands. As the disease progresses, swallowing and tongue movement can be restricted.
3. Management of OSMF focuses on eliminating irritating habits and providing symptomatic relief through medications, physiotherapy, or surgical intervention in advanced cases.
The Indian Dental Academy is the Leader in continuing dental education , training dentists in all aspects of dentistry and
offering a wide range of dental certified courses in different formats.
Lichen planus is a chronic inflammatory skin and mucous membrane disease characterized by violaceous papules that may form plaques. Oral lichen planus commonly presents as striae - sharply defined white lacy patterns - but can also be erosive, atrophic, or bullous. CD8 T cells trigger apoptosis of oral epithelial cells. Treatment aims to reduce symptoms, resolve lesions, and prevent oral squamous cell carcinoma through topical corticosteroids, systemic medications, surgery or laser, with complications including infection and malignant transformation requiring careful long-term follow-up.
This document discusses clinical features of gingivitis and chronic periodontitis. It describes the signs and symptoms of gingivitis such as color changes, consistency changes, and bleeding. It also discusses the progression of inflammation from the gingiva to the supporting periodontal tissues. Finally, it outlines the characteristics, disease distribution, risk factors, and prevalence of chronic periodontitis.
Oral candidiasis and stomatitis. name - seelam Sai charitha .pptxSaicharitha15
This document discusses oral candidiasis (thrush) and stomatitis. It provides details on:
- Causes of oral candidiasis including local and general predisposing factors
- Classification of oral candidiasis into traditional categories and primary/secondary types
- Symptoms of oral candidiasis such as white spots and discomfort
- Diagnosis through examination and testing
- Treatment using antifungal medications
It also covers types of stomatitis like canker sores and cold sores, their causes from viruses or sensitivities, symptoms like pain and sores, and treatments like antiviral medication or numbing agents.
The document summarizes the development, anatomy, disorders, and diseases of the tongue. It discusses:
1. The tongue develops from swellings in the first and third branchial arches. It has various papillae including circumvallate, fungiform, and filiform papillae.
2. Common tongue disorders include anatomical abnormalities (ankyloglossia, macroglossia), infections (hairy tongue), deficiencies (iron deficiency anemia), precancers (oral submucous fibrosis, leukoplakia), and cancers (squamous cell carcinoma).
3. The document provides detailed descriptions of numerous tongue conditions and abnormalities. It covers topics like papillae types, classifications
Children have oral mucosal conditions and other head and neck medical problems which have both similarities and differences to those found in adults .
A wide variety of oral lesions and soft tissue anomalies are detected in children, but the low frequency at which many of these entities occur makes them challenging to clinically diagnose.
This document discusses gingival enlargement from multiple perspectives. It begins by defining key terms like hyperplasia and hypertrophy. It then categorizes enlargement based on location, etiology, degree, and associated conditions. Chronic inflammatory enlargement and acute conditions like gingival abscesses are explained. Drug-induced, hereditary, and condition-associated enlargements are explored. Systemic diseases that can cause enlargement like leukemia and Wegener's granulomatosis are summarized. The document concludes with an overview of neoplastic enlargements.
This document describes several normal anatomical variants and developmental anomalies that can occur in the oral cavity. It discusses conditions like linea alba, leukoedema, oral pigmentation, lingual tonsils, and Fordyce's granules as normal variants. Developmental anomalies covered include ankyloglossia, cleft lip, cleft palate, bifid tongue, double lip, torus palatinus, torus mandibularis, multiple exostoses, and fibrous developmental malformation. Each condition is briefly described in terms of its clinical presentation, incidence, treatment if needed, and differential diagnosis.
for undergraduate dental students this presentation includes essential & common disorders which related to the tongue very briefly. Though this may be very helpfull to you to as a start for further readings & studying.
13 premalignant conditions_of_oral_cavityAshish Soni
This document discusses various oral diseases including leukoplakia, erythroplakia, oral submucous fibrosis, and candidiasis. It describes the characteristics, causes, and common sites of each disease. Treatment options are provided which include surgical excision of small lesions, chemotherapy, radiation therapy, and antifungal medications depending on the specific condition. Risk factors that can contribute to oral diseases like betel nut and tobacco chewing and smoking are also presented.
This document provides information on various types of lesions and disorders that can occur on the lips. It begins with background on lip anatomy and development. It then classifies and describes different colored, ulcerative, elevated, and developmental lesions that may affect the lips. Specific disorders covered in more depth include cheilitis (inflammation of the lips), angular cheilitis, actinic cheilitis, exfoliative cheilitis, plasma cell cheilitis, and drug-induced cheilitis. Cleft lip and other congenital abnormalities are also summarized. The document provides details on causes, clinical features, diagnosis, and management for each condition.
Commonest diseases and tumours of oral cavitySarab Ji
This document summarizes diseases of the oral cavity, including developmental anomalies, tumors and cysts, teeth and periodontal diseases, and epithelial cysts of the jaw. It provides details on conditions like cleft lip/palate, leukoplakia, dental caries, periodontitis, and radicular cyst. Developmental anomalies can result from genetic or environmental factors and include conditions affecting the tongue, like macroglossia and ankyloglossia. Dental caries is caused by plaque acids demineralizing enamel and dentin. Untreated caries can lead to pulpitis and periapical abscesses. Leukoplakia is a precancerous white patch caused by hyperker
Philippine Edukasyong Pantahanan at Pangkabuhayan (EPP) CurriculumMJDuyan
(𝐓𝐋𝐄 𝟏𝟎𝟎) (𝐋𝐞𝐬𝐬𝐨𝐧 𝟏)-𝐏𝐫𝐞𝐥𝐢𝐦𝐬
𝐃𝐢𝐬𝐜𝐮𝐬𝐬 𝐭𝐡𝐞 𝐄𝐏𝐏 𝐂𝐮𝐫𝐫𝐢𝐜𝐮𝐥𝐮𝐦 𝐢𝐧 𝐭𝐡𝐞 𝐏𝐡𝐢𝐥𝐢𝐩𝐩𝐢𝐧𝐞𝐬:
- Understand the goals and objectives of the Edukasyong Pantahanan at Pangkabuhayan (EPP) curriculum, recognizing its importance in fostering practical life skills and values among students. Students will also be able to identify the key components and subjects covered, such as agriculture, home economics, industrial arts, and information and communication technology.
𝐄𝐱𝐩𝐥𝐚𝐢𝐧 𝐭𝐡𝐞 𝐍𝐚𝐭𝐮𝐫𝐞 𝐚𝐧𝐝 𝐒𝐜𝐨𝐩𝐞 𝐨𝐟 𝐚𝐧 𝐄𝐧𝐭𝐫𝐞𝐩𝐫𝐞𝐧𝐞𝐮𝐫:
-Define entrepreneurship, distinguishing it from general business activities by emphasizing its focus on innovation, risk-taking, and value creation. Students will describe the characteristics and traits of successful entrepreneurs, including their roles and responsibilities, and discuss the broader economic and social impacts of entrepreneurial activities on both local and global scales.
Chapter wise All Notes of First year Basic Civil Engineering.pptxDenish Jangid
Chapter wise All Notes of First year Basic Civil Engineering
Syllabus
Chapter-1
Introduction to objective, scope and outcome the subject
Chapter 2
Introduction: Scope and Specialization of Civil Engineering, Role of civil Engineer in Society, Impact of infrastructural development on economy of country.
Chapter 3
Surveying: Object Principles & Types of Surveying; Site Plans, Plans & Maps; Scales & Unit of different Measurements.
Linear Measurements: Instruments used. Linear Measurement by Tape, Ranging out Survey Lines and overcoming Obstructions; Measurements on sloping ground; Tape corrections, conventional symbols. Angular Measurements: Instruments used; Introduction to Compass Surveying, Bearings and Longitude & Latitude of a Line, Introduction to total station.
Levelling: Instrument used Object of levelling, Methods of levelling in brief, and Contour maps.
Chapter 4
Buildings: Selection of site for Buildings, Layout of Building Plan, Types of buildings, Plinth area, carpet area, floor space index, Introduction to building byelaws, concept of sun light & ventilation. Components of Buildings & their functions, Basic concept of R.C.C., Introduction to types of foundation
Chapter 5
Transportation: Introduction to Transportation Engineering; Traffic and Road Safety: Types and Characteristics of Various Modes of Transportation; Various Road Traffic Signs, Causes of Accidents and Road Safety Measures.
Chapter 6
Environmental Engineering: Environmental Pollution, Environmental Acts and Regulations, Functional Concepts of Ecology, Basics of Species, Biodiversity, Ecosystem, Hydrological Cycle; Chemical Cycles: Carbon, Nitrogen & Phosphorus; Energy Flow in Ecosystems.
Water Pollution: Water Quality standards, Introduction to Treatment & Disposal of Waste Water. Reuse and Saving of Water, Rain Water Harvesting. Solid Waste Management: Classification of Solid Waste, Collection, Transportation and Disposal of Solid. Recycling of Solid Waste: Energy Recovery, Sanitary Landfill, On-Site Sanitation. Air & Noise Pollution: Primary and Secondary air pollutants, Harmful effects of Air Pollution, Control of Air Pollution. . Noise Pollution Harmful Effects of noise pollution, control of noise pollution, Global warming & Climate Change, Ozone depletion, Greenhouse effect
Text Books:
1. Palancharmy, Basic Civil Engineering, McGraw Hill publishers.
2. Satheesh Gopi, Basic Civil Engineering, Pearson Publishers.
3. Ketki Rangwala Dalal, Essentials of Civil Engineering, Charotar Publishing House.
4. BCP, Surveying volume 1
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2. After extra oral examination we proceed towards intra
oral examination
The structures to be examined are :-
1. Lips
2. Labial and buccal mucosa
3. Palate
4. Floor of mouth
5. Tongue
6. Gingiva
7. Teeth
3. 1.Lips
While examination of lips we will check for:
contusion pigmentation ulceration
Double lip Lip pit swelling
Angular pit cold
sore
4. Labial and Buccal mucosa
For the examination of the labial and buccal mucosa
the lip and cheek are retracted respectively and
observed in sufficient light.
The healthy mucosa appears to be smooth, shiny, pink
and moist.
Opening of Stensen’s duct can be seen opposite to 2nd
molar.
ABNORMAL FINDINGS:
COLOR: The black spots (macule) may
be seen on the mucosa due to
the deposition of melanin as a response of local
5. The red patches may be seen on the mucosa which can be
solitary or generalized.
The red patches on the mucosa can be seen due to :
1, Erythroplakia
2, Allergic reactions
3, Candidiasis (atrophic/ erythmatous)
4, Carcinoma in-situ
Similarly like red patches the white patches are also seen
which may be due to:
1, Leukoplakia
2, Leukoedema
3, Linea alba
4, white sponge nevus
6. CONSISITENCY:
The consistency of the mucosa may become fibrous
instead of smooth which may be due
1, Oral sub mucous fibrosis
2, chronic cheek bite
3, linea alba
4, Fordyce’s granules
5, Red or white lesions
6, Candidiasis
These fibrous appearing mucosa may be scrapable
or non scrapable depending upon the cause.
Other factors to be seen are the swelling, ulceration
or presence of nodules and Fordyce's granules.
7. Soft and hard palate
After the proper illumination of light we will check
for:
• Discoloration
• Swelling/fistula/tori /ulcer
• Cleft
• Perforation: usually caused by gumma
• Asymmetry of structures
• Any scar
• Papillary hyperplasia
further the palate is palpated to check for the
swelling or tenderness on the palate.
9. Floor of mouth
For the examination of the floor of mouth the patient
is asked to elevate the tongue and then the floor is
examined with proper illumination of light.
The opening of Wharton’s duct is observed.
The pooling of saliva is seen.
The floor is also seen for ulceration and swelling.
depending on the color we can find the cause of
swelling.
For e.g., if the swelling is Red : hemangioma
Blue: ranula
While examination of floor of mouth we can also
check the presence or absence of ankyloglossia.
11. Tongue
Dorsal surface is examined while keeping the
tongue at rest.
COLOR:
We will check for the presence of pigmentation
For e.g. yellow color : Jaundice
black color : Heavy smokers
SIZE
We will check for the presence of macroglossia or
microglossia.
FISSURE
We will then check for the presence of fissures on
the surface of tongue
Transverse congenital
Longitudinal syphilis
13. Varicosity of tongue is a physiological process
caused due to advancing age leading to elastolytic
degeneration of the sublingual veins.
Frenal attachment : High
Normal
14. GINGIVA
Gingiva is the part of the oral mucosa that covers the
alveolar process of the jaw and surrounds the neck
of the teeth.
It is the most important structure to be examined in
the oral cavity to check for the periodontal condition.
15. COLOR:
The normal color of the gingiva is coral pink or pale
pink
The color of the gingiva depends upon :
1. Vascular supply
2. Thickness and degree of keratinisation of
epithelium
3. Presence of pigment containing cells.
The change in color signifies the diseased gingiva
Acute: red
Chronic: bluish pink
16. Size:
The size of the gingiva depends on bulk of cellular +
intercellular elements and blood supply
The healthy gingiva fits snugly around the teeth.
The alteration in the size is the common feature of
gingival disease.
The increase in the size of gingiva is commonly
known as the gingival enlargement
They are further classified as:
1. Inflammatory enlargement : i) Acute
ii) chronic
18. Contour:
The marginal gingiva follows the scalloped outline
on the facial and lingual surfaces.
The interdental papilla are pointed and pyramidal
and also fills the space present interdentally.
The diseased marginal gingiva becomes rounded,
rolled and bulbous.
The interdental gingiva becomes bulbous, flat and
cratered
Normal gingiva
Diseased gingiva
19. Position: The level at which gingival margin is
attached to the tooth - At CEJ
Exposure of the tooth by the apical migration of the
gingiva is known as gingival recession.
Consistency: Firm and resilient except the free
marginal gingiva. It is tightly bounded to the
underlying bone.
The gingiva becomes edematous and fibrotic in
diseased condition.
20. Texture:
Stippling or the orange peel appearance is seen on
the attached gingiva on drying it with the cotton.
It appears on the gingiva after 5 years of age and
remains throughout the life in the healthy gingiva
The stippling is lost in the diseased gingiva.
21. Gingivitis
Gingivitis is defined as the inflammation of the
gingiva.
It is further classified as : Acute and Chronic
: Localized or Generalized
22. Signs and symptoms:
•Bright red or purple gums
•Spongy and swollen gums
•They are mainly painless unless pressure applied
•Easily bleed even on gentle brushing
•Loss of stippling
23. Causes:
• Build up of bacteria due to accumulation of the
plaque which lead to irritation of the gingiva.
•Poor oral hygiene
• Smoking
• Faulty prosthesis
• Malocclusion
• Breathing through mouth
• Local trauma
• Vitamin deficiency
Gingivitis is usually a reversible condition, but if they
are left untreated it may lead to the irreversible and a
severe condition mainly known as PERIODONTITIS.
24. Periodontitis
It is the inflammation of the gums and supporting
structures of the teeth.
It may also be defined as the disease involving one
or more of the four components of the peridontium
25. Signs and symptoms:
All the signs of the gingivitis are present , in addition
to:
•Bone loss
•Halitosis
•Pocket formation
•Occasional suppuration
•Tooth mobility
•Spacing between the teeth
•Gum recession
•Increased sensitivity and food impaction