This document discusses inferior epigastric vessels injury during abdominal procedures. It begins by describing the various vessels of the anterior abdominal wall including the superficial and deep epigastric vessels and circumflex iliac vessels. It then discusses the surface anatomy and course of the deep epigastric vessels, specifically the superior and inferior vessels. The document notes that injury to these vessels most commonly occurs during laparoscopic trocar insertion and provides the incidence of such injuries. It outlines ways to prevent vessel injuries including internal and external identification techniques during surgery. Finally, it describes the management and treatment approaches if an injury does occur, such as tamponade with a Foley catheter, vessel ligation, or suturing.
Difficult Laparoscopic Cholecystectomy-When and Where is the Need to Convert?Apollo Hospitals
Laparoscopic cholecystectomy has now become the treatment of choice for the gall bladder stone. With increasing experience, surgeon has started to take more difficult cases which were considered relative contra indications for laparoscopic removal of gall bladder few years back.
We conducted this study at our hospital and included all laparoscopic cholecystectomy done from May'08 to January'10. Total time taken in surgery, conversion rate and complication rate were analysed. Factors making laparoscopic cholecystectomy difficult were also analysed. We defined difficult laparoscopic cholecystectomy when we found -dense fibrotic adhesions in and around Callot's triangle, gangrenous gall bladder, empyma, large stone impacted at gall bladder neck, contracted gall bladder, Mirrizi's syndrome, h/o biliary pancreatitis, CBD stones, acute cholecystitis of <72 hrs duration.
Out of 206 cases done during above period, 56 cases were considered difficult. Only two cases were converted to open.
With growing experience and technical advancement surgery can be completed in most of the difficult cases. This is important because recently it is shown in literature that laparoscopic cholecystectomy is associated with less morbidity than open method irrespective of duration of the surgery.
Difficult Laparoscopic Cholecystectomy-When and Where is the Need to Convert?Apollo Hospitals
Laparoscopic cholecystectomy has now become the treatment of choice for the gall bladder stone. With increasing experience, surgeon has started to take more difficult cases which were considered relative contra indications for laparoscopic removal of gall bladder few years back.
We conducted this study at our hospital and included all laparoscopic cholecystectomy done from May'08 to January'10. Total time taken in surgery, conversion rate and complication rate were analysed. Factors making laparoscopic cholecystectomy difficult were also analysed. We defined difficult laparoscopic cholecystectomy when we found -dense fibrotic adhesions in and around Callot's triangle, gangrenous gall bladder, empyma, large stone impacted at gall bladder neck, contracted gall bladder, Mirrizi's syndrome, h/o biliary pancreatitis, CBD stones, acute cholecystitis of <72 hrs duration.
Out of 206 cases done during above period, 56 cases were considered difficult. Only two cases were converted to open.
With growing experience and technical advancement surgery can be completed in most of the difficult cases. This is important because recently it is shown in literature that laparoscopic cholecystectomy is associated with less morbidity than open method irrespective of duration of the surgery.
Bladder injuries are rare. But when present in cases of polytrauma they pose both a diagnostic as well as surgical challenge to the attending surgeon. Understanding the mechanisms underlying bladder injuries is pivotal in developing a diagnostic algorithm in order to avoid missing of any urologic injury. Once the extent and site of damage is diagnosed then prompt surgical intervention is the mainstay of treatment. The pathophysiology and management of bladder injuries is discussed in this paper.
colon anatomy, anatomy of large intestine, anatomy of large bowel, histology of large intestine, large intestine, histology, colon, appendices epiploica, taenia coli, haustrautions, ilio caecal valve
Groin swellings is one of the common problems in Surgery. Common causes are Inguinal hernia, femoral hernia and Undescended testis. In this slide presentation I discuss the applied anatomy of the groin region.
The incidence of biliary injury after laparoscopic cholecystectomy (LC) has shown a declining trend though it may still be twice that as with open cholecystectomy. Major biliary or vasculobiliary injury is associated with significant morbidity. As prevention is the best strategy, the concept of a culture of safe cholecystectomy has been recently introduced to educate surgeons and apprise them of basic tenets of safe performance of LC. Various aspects of safe cholecystectomy include: (1) thorough knowledge of relevant anatomy, various anatomical landmarks, and anatomical variations; (2) an understanding of the mechanisms involved in biliary/vascular injury, the most important being the misidentification injury; (3) identification of various preoperative and intraoperative predictors of difficult cholecystectomy; (4) proper gallbladder retraction; (5) safe use of various energy devices; (6) understanding the critical view of safety, including its doublet view and documentation; (7) awareness of various error traps (e.g., fundus first technique); (8) use of various bailout strategies (e.g., subtotal cholecystectomy) in difficult gallbladder cases; (9) use of intraoperative imaging techniques (e.g., intraoperative cholangiogram) to ascertain correct anatomy; and (10) understanding the concept of time-out. Surgeons should be facile with these aspects of this culture of safety in cholecystectomy in an attempt to reduce the incidence of biliary/vascular injury during LC.
Bladder injuries are rare. But when present in cases of polytrauma they pose both a diagnostic as well as surgical challenge to the attending surgeon. Understanding the mechanisms underlying bladder injuries is pivotal in developing a diagnostic algorithm in order to avoid missing of any urologic injury. Once the extent and site of damage is diagnosed then prompt surgical intervention is the mainstay of treatment. The pathophysiology and management of bladder injuries is discussed in this paper.
colon anatomy, anatomy of large intestine, anatomy of large bowel, histology of large intestine, large intestine, histology, colon, appendices epiploica, taenia coli, haustrautions, ilio caecal valve
Groin swellings is one of the common problems in Surgery. Common causes are Inguinal hernia, femoral hernia and Undescended testis. In this slide presentation I discuss the applied anatomy of the groin region.
The incidence of biliary injury after laparoscopic cholecystectomy (LC) has shown a declining trend though it may still be twice that as with open cholecystectomy. Major biliary or vasculobiliary injury is associated with significant morbidity. As prevention is the best strategy, the concept of a culture of safe cholecystectomy has been recently introduced to educate surgeons and apprise them of basic tenets of safe performance of LC. Various aspects of safe cholecystectomy include: (1) thorough knowledge of relevant anatomy, various anatomical landmarks, and anatomical variations; (2) an understanding of the mechanisms involved in biliary/vascular injury, the most important being the misidentification injury; (3) identification of various preoperative and intraoperative predictors of difficult cholecystectomy; (4) proper gallbladder retraction; (5) safe use of various energy devices; (6) understanding the critical view of safety, including its doublet view and documentation; (7) awareness of various error traps (e.g., fundus first technique); (8) use of various bailout strategies (e.g., subtotal cholecystectomy) in difficult gallbladder cases; (9) use of intraoperative imaging techniques (e.g., intraoperative cholangiogram) to ascertain correct anatomy; and (10) understanding the concept of time-out. Surgeons should be facile with these aspects of this culture of safety in cholecystectomy in an attempt to reduce the incidence of biliary/vascular injury during LC.
Minimal access surgery (MAS) a new surgical and
interventional approach, was called by different name and
one of the popular is minimally invasive surgery. However,
unique complications are associated with gaining access
to the abdomen for laparoscopic surgery. The technique
of first entry inside the human body with telescope and
instruments is called access technique. The hallmark of the
new approaches is the reduction in the trauma of access.
The technique for access to the peritoneal cavity, choice of
access technique, placement locations, and port placement
is very important in MAS. Technique of access is different for
different minimal access surgical procedures. Thoracoscopy,
retroperitoneoscopy, axilloscopy, and arthroscopy all have
different ways of access. In this chapter, we will discuss
various abdominal access techniques.
It is important to know that approximately 20% of
laparoscopic complications are caused at the time of initial
access. Developing access skill is one of the important
achievements for the surgeon practicing MAS. First entry or
access in laparoscopy is of two types: (1) closed access and
(2) open access.
World's Most Popular Hands-On Laparoscopic Training Instituteraja766604
World Laparoscopy Hospital is a well-known and highly respected international training center for laparoscopic surgery. It offers a comprehensive laparoscopic surgery training course for general surgeons, gynecologists, and urologists. The training program is designed to provide both basic and advanced theoretical and practical experience to the candidates.
The laparoscopic surgery training course at World Laparoscopy Hospital is completely candidate-centered, with an emphasis on practical laparoscopic surgical problems encountered while operating on patients. The training takes place within an ultramodern laparoscopic HD wet operating room, followed by live exposure of live laparoscopic surgery in the operation theater with expert consultants.
The laparoscopic training program is affiliated with a Government-recognized university, and upon completion of the course, candidates receive a Laparoscopic Fellowship and Diploma Certificate issued by a UGC recognized university and the World Association of Laparoscopic Surgeons.
https://www.laparoscopyhospital.com/SERV01.HTM
Minimal access surgery (MAS) a new surgical and interventional approach, was called by different name and one of the popular is minimally invasive surgery. However,unique complications are associated.
CDSCO and Phamacovigilance {Regulatory body in India}NEHA GUPTA
The Central Drugs Standard Control Organization (CDSCO) is India's national regulatory body for pharmaceuticals and medical devices. Operating under the Directorate General of Health Services, Ministry of Health & Family Welfare, Government of India, the CDSCO is responsible for approving new drugs, conducting clinical trials, setting standards for drugs, controlling the quality of imported drugs, and coordinating the activities of State Drug Control Organizations by providing expert advice.
Pharmacovigilance, on the other hand, is the science and activities related to the detection, assessment, understanding, and prevention of adverse effects or any other drug-related problems. The primary aim of pharmacovigilance is to ensure the safety and efficacy of medicines, thereby protecting public health.
In India, pharmacovigilance activities are monitored by the Pharmacovigilance Programme of India (PvPI), which works closely with CDSCO to collect, analyze, and act upon data regarding adverse drug reactions (ADRs). Together, they play a critical role in ensuring that the benefits of drugs outweigh their risks, maintaining high standards of patient safety, and promoting the rational use of medicines.
Basavarajeeyam is an important text for ayurvedic physician belonging to andhra pradehs. It is a popular compendium in various parts of our country as well as in andhra pradesh. The content of the text was presented in sanskrit and telugu language (Bilingual). One of the most famous book in ayurvedic pharmaceutics and therapeutics. This book contains 25 chapters called as prakaranas. Many rasaoushadis were explained, pioneer of dhatu druti, nadi pareeksha, mutra pareeksha etc. Belongs to the period of 15-16 century. New diseases like upadamsha, phiranga rogas are explained.
New Drug Discovery and Development .....NEHA GUPTA
The "New Drug Discovery and Development" process involves the identification, design, testing, and manufacturing of novel pharmaceutical compounds with the aim of introducing new and improved treatments for various medical conditions. This comprehensive endeavor encompasses various stages, including target identification, preclinical studies, clinical trials, regulatory approval, and post-market surveillance. It involves multidisciplinary collaboration among scientists, researchers, clinicians, regulatory experts, and pharmaceutical companies to bring innovative therapies to market and address unmet medical needs.
These simplified slides by Dr. Sidra Arshad present an overview of the non-respiratory functions of the respiratory tract.
Learning objectives:
1. Enlist the non-respiratory functions of the respiratory tract
2. Briefly explain how these functions are carried out
3. Discuss the significance of dead space
4. Differentiate between minute ventilation and alveolar ventilation
5. Describe the cough and sneeze reflexes
Study Resources:
1. Chapter 39, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 34, Ganong’s Review of Medical Physiology, 26th edition
3. Chapter 17, Human Physiology by Lauralee Sherwood, 9th edition
4. Non-respiratory functions of the lungs https://academic.oup.com/bjaed/article/13/3/98/278874
Lung Cancer: Artificial Intelligence, Synergetics, Complex System Analysis, S...Oleg Kshivets
RESULTS: Overall life span (LS) was 2252.1±1742.5 days and cumulative 5-year survival (5YS) reached 73.2%, 10 years – 64.8%, 20 years – 42.5%. 513 LCP lived more than 5 years (LS=3124.6±1525.6 days), 148 LCP – more than 10 years (LS=5054.4±1504.1 days).199 LCP died because of LC (LS=562.7±374.5 days). 5YS of LCP after bi/lobectomies was significantly superior in comparison with LCP after pneumonectomies (78.1% vs.63.7%, P=0.00001 by log-rank test). AT significantly improved 5YS (66.3% vs. 34.8%) (P=0.00000 by log-rank test) only for LCP with N1-2. Cox modeling displayed that 5YS of LCP significantly depended on: phase transition (PT) early-invasive LC in terms of synergetics, PT N0—N12, cell ratio factors (ratio between cancer cells- CC and blood cells subpopulations), G1-3, histology, glucose, AT, blood cell circuit, prothrombin index, heparin tolerance, recalcification time (P=0.000-0.038). Neural networks, genetic algorithm selection and bootstrap simulation revealed relationships between 5YS and PT early-invasive LC (rank=1), PT N0—N12 (rank=2), thrombocytes/CC (3), erythrocytes/CC (4), eosinophils/CC (5), healthy cells/CC (6), lymphocytes/CC (7), segmented neutrophils/CC (8), stick neutrophils/CC (9), monocytes/CC (10); leucocytes/CC (11). Correct prediction of 5YS was 100% by neural networks computing (area under ROC curve=1.0; error=0.0).
CONCLUSIONS: 5YS of LCP after radical procedures significantly depended on: 1) PT early-invasive cancer; 2) PT N0--N12; 3) cell ratio factors; 4) blood cell circuit; 5) biochemical factors; 6) hemostasis system; 7) AT; 8) LC characteristics; 9) LC cell dynamics; 10) surgery type: lobectomy/pneumonectomy; 11) anthropometric data. Optimal diagnosis and treatment strategies for LC are: 1) screening and early detection of LC; 2) availability of experienced thoracic surgeons because of complexity of radical procedures; 3) aggressive en block surgery and adequate lymph node dissection for completeness; 4) precise prediction; 5) adjuvant chemoimmunoradiotherapy for LCP with unfavorable prognosis.
NVBDCP.pptx Nation vector borne disease control programSapna Thakur
NVBDCP was launched in 2003-2004 . Vector-Borne Disease: Disease that results from an infection transmitted to humans and other animals by blood-feeding arthropods, such as mosquitoes, ticks, and fleas. Examples of vector-borne diseases include Dengue fever, West Nile Virus, Lyme disease, and malaria.
ARTIFICIAL INTELLIGENCE IN HEALTHCARE.pdfAnujkumaranit
Artificial intelligence (AI) refers to the simulation of human intelligence processes by machines, especially computer systems. It encompasses tasks such as learning, reasoning, problem-solving, perception, and language understanding. AI technologies are revolutionizing various fields, from healthcare to finance, by enabling machines to perform tasks that typically require human intelligence.
Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
Adv. biopharm. APPLICATION OF PHARMACOKINETICS : TARGETED DRUG DELIVERY SYSTEMSAkankshaAshtankar
MIP 201T & MPH 202T
ADVANCED BIOPHARMACEUTICS & PHARMACOKINETICS : UNIT 5
APPLICATION OF PHARMACOKINETICS : TARGETED DRUG DELIVERY SYSTEMS By - AKANKSHA ASHTANKAR
Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
2. AGENDA
VESSELS OF ANTERIOR ABDOMINAL WALL
SUPERFICIAL EPIGASTRIC VESSELS
CIRCUMFLEX ILIAC VESSELS
DEEP EPIGASTRIC VESSELS
SURFACE ANATOMY
INCIDENCE AND CAUSES OF INJURY
PREVENTION OF INJURY
MANAGEMENT OF INJURY
MISSED INJURY
3. VESSELS OF ANTERIOR ABDOMINAL WALL
Epigastric vessels ( Superficial and deep )
Circumflex iliac vessels ( Superficial and deep )
4. SUPERFICIAL EPIGASTRIC VESSELS
Origin : Femoral artery
Course : Through superficial fascia toward the umbilicus
Identified by intra-abdominal transillumination to avoid their injury
5. CIRCUMFLEX ILIAC VESSELS
Superficial circumflex iliac originate from external iliac artery.
Deep circumflex iliac originate from femoral artery.
7. SUPERIOR DEEP EPIGASTRIC VESSELS
Origin: Internal mammary artery
Course: Descends through the thorax into the rectus
muscle accompanied by 2 veins.
Termination : Anastomose with inferior epigastric vessels
at the level of umbilicus.
8. INFERIOR DEEP EPIGASTRIC VESSELS
Origin: External iliac artery just above the inguinal ligament.
Course: It ascends obliquely and medially along the medial margin of the
deep inguinal ring, pierces the transversalis fascia, and passes in front of
the arcuate line, runs between the rectus abdominis and posterior rectus
sheath.
Termination : By piercing the rectus abdominis at the umbilical level, it
divides into numerous branches and anastomoses with the superior
epigastric artery.
9. SURFACE ANATOMY
The course of the Inferior epigastric artery (IEA) is highly variable.
Distance of IEA from the midline:
At pubic symphysis : The shortest distance of IEA from the midline at this level was 1.2 cm on the left and 3.5
cm on the right; the farthest distance was 6.9 cm on the left and 6.8 cm on the right.
At the level of anterior superior iliac spines (ASIS) : The median distance of IEA from midline at this level was
4.9 cm on the left and 4.7 cm on the right
At the level of umbilicus: The mean distance of IEA from midline was 3.1 cm on the left and 3.4 cm on the
right.
10. INCIDENCE OF INJURY
0.3% to 2.5% during operative laparoscopy
Recognition of vessel injury :
Observing blood dripping down the cannula
Abdominal wall discoloration or development of a haematoma around or near the incision.
In some instances, the blood may track to a more distant site, manifesting as a pararectal or vulval mass
11. CAUSES OF INJURY
The anatomical position of the IEA subjects it to risk of injury during abdominal procedures that are close to the
artery.
laparoscopic trocar insertion
Insertion of intraabdominal drains.
Peritoneal dialysis catheter
Paracentesis
13. INTERNAL IDENTIFICATION
Direct visualisation transperitoneally is the ideal approach to
avoid inferior epigastric vessel injury during laparoscopic
accessory trocar placement, which should be inserted lateral to
the vessels at a 90° angle to the abdominal wall.
Always identify the IEA as they course along the parietal
peritoneum in the lateral umbilical fold.
The IEA are located lateral to the medial umbilical fold but medial
to the deep inguinal ring
Identify the deep inguinal ring by locating where the round
ligament enters the inguinal canal
15. INTERNAL IDENTIFICATION
Having difficulty finding the deep inferior epigastric vessels?
Look between the round ligament insertion and median
umbilical ligament!
Place your trocar lateral to round ligament insertion.
16. EXTERNAL IDENTIFICATION
If the Inferior epigastric vessels are obscured by excess tissue and can
not be easily identified.
Place the trocar approximately 8 cm lateral to the midline and 5 cm
above symphysis pubis .
These right and left abdominal area approximate McBurney’s point and
Hurd’s point, respectively.
To avoid injury to IEA, trocars can be safely inserted 8 cm away from the
midline (or) in the lateral third of the distance between the midline and a
sagittal plane running through ASIS.
17. EXTERNAL IDENTIFICATION
The medial and lateral safer zones can be drawn to reduce the risk
of IEA injury:
1) medially, within 1 cm either side of the midline
2) laterally, more than 8 cm from the midline or more than two-
thirds along the horizontal line between the midline and a sagittal
plane through the ASIS.
It should be recognised that the IEAs located in the area between
4 cm and 8 cm from the midline and 80% of IEAs had at least one
branch more than 1 mm in diameter emerging from the lateral
border of the rectus sheath.
19. MANAGEMENT OF IEA INJURY
Do not remove the trocar
Leave the trocar in place to help tamponade the vessel and aid in
localizing the site of injury.
Move the trocar into each quadrant to find a position that causes
the bleeding to stop. When the proper quadrant is found,
pressure from the portion of the trocar within the abdomen
tamponades the bleeding.
20. MANAGEMENT OF IEA INJURY
Then, grasp the vessel by non traumatic grasper through
the contralateral port below the site of injury ( towards the
origin ) to avoid retraction of the vessel and to prevent
further expansion of the hematoma
21. MANAGEMENT OF IEA INJURY
Then , you have three choices , either :
Insertion of foley’s catheter
Coagulation
suturing
22. MANAGEMENT OF IEA INJURY
A Foley catheter inserted through the port site, and the
balloon inflated in the peritoneal cavity by 30 ml water.
The balloon can then be pulled up against the bleeding
point with a resultant tamponade effect.
The catheter can be secured externally using a clamp.
Leave the catheter in place for 24-48 hours
23. MANAGEMENT OF IEA INJURY
Coagulation of the vessel by bipolar proximal and distal to
the site of injury if the vessel has not retracted to the
abdominal wall.
24. MANAGEMENT OF IEA INJURY
Suturing the vessel proximal and distal to the site
of injury.
Take the stitch by port closure needle using the
same technique like closing the port site.
Leave the suture in place for 48-72 hours.
By prolene on silicon tube or piece of gauze to
avoid necrosis of the skin. ( Like tension sutures as
in the image )
25. MANAGEMENT OF IEA INJURY
Do not forget to ensure hemostasis on decreasing the intra-
abdominal pressure.
If all these measures failed or the hematoma is enlarging, do not
hesitate to do immediate laparotomy.
26. MISSED INJURY
It is advisable to directly visualise the removal of accessory trocars, as this may reveal inadvertent bleeding
points
Bleeding may not immediately be apparent to the surgical team due to a number of factors, including
increased abdominal pressure (pneumoperitoneum) or decreased venous pressure associated with the
Trendelenburg position.
If the lacerated vessel presents postoperatively as a haematoma, initial management should be with local
compression. The temptation to open or aspirate the haematoma should be resisted, as such a manoeuvre
may inhibit the tamponade effect and could increase the risk of abscess formation.
If the haematoma continues to enlarge, however, or if there is evidence of haemodynamic compromise, wound
exploration is indicated.