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Hiatal Hernia
Hiatal hernia is the protrusion of the stomach upward into the
mediastinal cavity through the esophageal hiatus of the
diaphragm.
Types of Hiatal Hernia
• 1. Sliding hiatal hernia:
• Herniation of both the stomach and the
• gastroesophageal(GE) junction into the thorax.
• 90% of esophageal hernias
• 2. Paraesophageal hiatal hernia:
• Herniation of all or part of the stomach through the
• esophageal hiatus into the thorax with an undisplaced GE
• junction
• Least common esophageal hernia (<10%)
• Type III: Type III hernias have elements of both types I and II hernias.
With progressive enlargement of the hernia through the hiatus, the
phrenoesophageal membrane stretches, displacing the gastroesophageal
junction above the diaphragm, thereby adding a sliding element to the
type II hernia.
• Type IV: Type IV hiatus hernia is associated with a large defect in the
phrenoesophageal membrane, allowing other organs, such as colon,
spleen, pancreas and small intestine to enter the hernia sac.
• The end stage of type I and type II hernias occurs when the whole
stomach migrates up into the chest by rotating 180° around its
longitudinal axis, with the cardia and pylorus as fixed points. In this
situation the abnormality is usually referred to as an intrathoracic
stomach.
PATHOPHYSIOLOGY
• Size of hiatus not fixed, narrows with increase in intraabdominal
• pressure
• Tear of Phrenoesophageal ligament :
• is a fibrous layer of connective tissue and maintains the LES
• within the abdominal cavity
• A hiatal hernia compromises reflux barrier
• Reduced LES pressure
• Reduced esophageal acid clearance
• Transient LES relaxation episodes particularly at night time
Sliding Hiatal Hernia
• Age
• Increased intra-abdominal pressure (e.g. Obesity, pregnancy,
• coughing, heavy lifting).
• Smoking
Risk factors for Sliding Hiatal Hernia
Clinical features
• Majority are asymptomatic
• Heart burn
Larger hernias frequently associated with Gastroesophageal
• reflux disease(GERD) due to decreased competence of theLower
esophageal sphincter (LES) .
Complications
• Most common complication is GERD.
• Other complications are rare and are related to reflux
• Esophagitis (dysphagia, heartburn)
• Consequences of esophagitis ( peptic stricture, Barrett’s
• esophagus, esophageal carcinoma)
• Extra-esophageal complications ( pneumonitis/
• pneumonia, asthma, cough, laryngitis)
Investigations
• Chest X-ray
• Barium swallow
• Endoscopy
• Esophageal manometry (to measure the pressure of LES)
• 24-48h esophageal pH monitoring to quantify reflux
• Gastroscopy with biopsy to rule out cancer and esophagitis
Chest X-ray
Barium swallow
Endoscopic view:
Treatment
• LIFESTYLE MODIFICATION
• Stop smoking,
• weight loss,
• elevate head of bed,
• no meals <3h prior to sleeping,
• smaller and more frequent meals,
• avoid too much alcohol, coffee, mint and fat.
• MEDICAL THERAPY
• Antacids
• H2 receptor antagonists e.g. Cimetidine
• Proton pump inhibitors e.g. Omeprazole
• Prokinetic agents e.g. Metoclopramide
SURGICAL THERAPY
Indications:
 Failure of medical therapy
 Esophageal stricture
 Severe nocturnal aspiration
 Barrett’s esophagus
• Anti-reflux procedure e.g Fundoplication
• A laparoscopic procedure in which the fundus of the stomach is
• wrapped around lower end of esophagus. The types of fundoplication
• include:
• i. The Nissen fundoplication is total (360°),
• ii. Partial fundoplication known as Thal (270° anterior),
• iii. Belsey (270° anterior transthoracic)
• iv. Dor (anterior 180-200°)
• v. Lind (300° posterior)
• vi. Toupet fundoplication (posterior 270°) are alternative procedures
• with somewhat different indications and outcomes.
Hiatal hernia
Hiatal hernia
Hiatal hernia
Hiatal hernia
Hiatal hernia
Hiatal hernia
Hiatal hernia

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Hiatal hernia

  • 1. Hiatal Hernia Hiatal hernia is the protrusion of the stomach upward into the mediastinal cavity through the esophageal hiatus of the diaphragm.
  • 2. Types of Hiatal Hernia • 1. Sliding hiatal hernia: • Herniation of both the stomach and the • gastroesophageal(GE) junction into the thorax. • 90% of esophageal hernias • 2. Paraesophageal hiatal hernia: • Herniation of all or part of the stomach through the • esophageal hiatus into the thorax with an undisplaced GE • junction • Least common esophageal hernia (<10%)
  • 3. • Type III: Type III hernias have elements of both types I and II hernias. With progressive enlargement of the hernia through the hiatus, the phrenoesophageal membrane stretches, displacing the gastroesophageal junction above the diaphragm, thereby adding a sliding element to the type II hernia. • Type IV: Type IV hiatus hernia is associated with a large defect in the phrenoesophageal membrane, allowing other organs, such as colon, spleen, pancreas and small intestine to enter the hernia sac. • The end stage of type I and type II hernias occurs when the whole stomach migrates up into the chest by rotating 180° around its longitudinal axis, with the cardia and pylorus as fixed points. In this situation the abnormality is usually referred to as an intrathoracic stomach.
  • 4. PATHOPHYSIOLOGY • Size of hiatus not fixed, narrows with increase in intraabdominal • pressure • Tear of Phrenoesophageal ligament : • is a fibrous layer of connective tissue and maintains the LES • within the abdominal cavity • A hiatal hernia compromises reflux barrier • Reduced LES pressure • Reduced esophageal acid clearance • Transient LES relaxation episodes particularly at night time
  • 5. Sliding Hiatal Hernia • Age • Increased intra-abdominal pressure (e.g. Obesity, pregnancy, • coughing, heavy lifting). • Smoking Risk factors for Sliding Hiatal Hernia
  • 6. Clinical features • Majority are asymptomatic • Heart burn Larger hernias frequently associated with Gastroesophageal • reflux disease(GERD) due to decreased competence of theLower esophageal sphincter (LES) .
  • 7. Complications • Most common complication is GERD. • Other complications are rare and are related to reflux • Esophagitis (dysphagia, heartburn) • Consequences of esophagitis ( peptic stricture, Barrett’s • esophagus, esophageal carcinoma) • Extra-esophageal complications ( pneumonitis/ • pneumonia, asthma, cough, laryngitis)
  • 8. Investigations • Chest X-ray • Barium swallow • Endoscopy • Esophageal manometry (to measure the pressure of LES) • 24-48h esophageal pH monitoring to quantify reflux • Gastroscopy with biopsy to rule out cancer and esophagitis
  • 12. Treatment • LIFESTYLE MODIFICATION • Stop smoking, • weight loss, • elevate head of bed, • no meals <3h prior to sleeping, • smaller and more frequent meals, • avoid too much alcohol, coffee, mint and fat.
  • 13. • MEDICAL THERAPY • Antacids • H2 receptor antagonists e.g. Cimetidine • Proton pump inhibitors e.g. Omeprazole • Prokinetic agents e.g. Metoclopramide SURGICAL THERAPY Indications:  Failure of medical therapy  Esophageal stricture  Severe nocturnal aspiration  Barrett’s esophagus
  • 14. • Anti-reflux procedure e.g Fundoplication • A laparoscopic procedure in which the fundus of the stomach is • wrapped around lower end of esophagus. The types of fundoplication • include: • i. The Nissen fundoplication is total (360°), • ii. Partial fundoplication known as Thal (270° anterior), • iii. Belsey (270° anterior transthoracic) • iv. Dor (anterior 180-200°) • v. Lind (300° posterior) • vi. Toupet fundoplication (posterior 270°) are alternative procedures • with somewhat different indications and outcomes.