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APPRACH TO HEMODYNAMIC
COLLAPSE IN ICU
Dr Abreham
Dr Mennur A
Dr Tewodros
• (SBP) to ≤90 mmHg, a decrease in SBP of ≥
20% from a baseline, a decrease
in mean arterial pressure (MAB) to ≤65
mmHg,
Initial approach undifferentiated hypotension
1 stabilize airway and secure iv access
Intubation
2 Risk stratification
Based on brief history ,PE ,ecg
• Common conditions needing lifesaving
interventions
Anaphylaxis
• Extremely rare for ketamine and etomidate
• Neuromuscular blocking agents are divalent
molecules that make it easier for anaphylaxis
to occur, even in the setting of no previous
exposure
• More common with rocuronium and
succinylcholine.
• Hypotension, inspiratory stridor, oral and
facial edema, hives
• RX
•Epinephrine
• Diphenhydramine
• Ranitidine
Tension Pneumothorax
• Consider in patient with obstructive lung
disease that has increased airway pressures.
• Use ultrasound to confirm whether a
pneumothorax is present.
Bar code/stratosphere sign
 Needle decompression, Chest tube
Cardiac Tamponade
• Elderly patients ,Pt on anticoagulants , ESRD
patients may have occult / borderline
tamponade that only declares itself after a
change to positive pressure ventilation.
 pericardiocentesis
Cardiogenic shock from myocardial infarction
•Anterior crushing chest pain, respiratory
distress, and the ECG changes with MI
•Request troponin
• Antiplatelet agents, heparin consult for
reperfusion, or balloon pump
Cardiogenic shock from acute aortic or mitral
valve insufficiency
• Patients with chest pain, hypotension, and
new low-pitched early diastolic murmur
consistent with aortic insufficiency should
undergo POC ultrasonography or
echocardiography
Acute arrhythmias can cause hypotension
• 12-lead electrocardiography.
 cardioversion
Hypovolemia
• Hypovolemia from profound shock states such
as hemorrhage, sepsis, and anaphylaxis
• Occult hypovolemia can be unmasked by the
physiologic insult that occurs when patients
are switched from spontaneous ventilation to
positive pressure ventilation.
• With the increase in intrathoracic pressure
that accompanies positive pressure ventilation
any patient with low venous pressures / low
pre-load may develop hypotension.
 Adjusting PEEP settings, and giving a
bolus of an appropriate resuscitative fluid
Dissection of the ascending aorta
• Retrograde dissection that results in acute
aortic insufficiency, pericardial tamponade, or
myocardial infarction
• Transthoracic echocardiography or POCUS
•Aortic root dilatation
•Intimal flap
Pulmonary Hypertension
• Patients with known severe pulmonary
hypertension are a nightmare to intubate.
• Hypervolemia, hypoxia, and hypercarbia are
known to worsen their right heart function.
• Fluid boluses , and may worsen LV
impingement.
Acidosis
• Patients with profound metabolic acidosis
typically have a high minute ventilation, until
they become fatigued and then their pH can
drop.
provide minute ventilation that
approximates pre-intubation levels.
8cc/kg tidal volume
high RR
Stacked breaths / Autopeep
• Patients with obstructive lung pathology who
are not completely exhaling before breath
• Decrease venous return overtime, an cause
hypotension, and eventual arrest.
Induction /sedation Agent
• Propofol
• Any sedative agent can result in post
intubation collapse
• Decreasing the dose of sedative,
• Pressors / push dose pressors ready could
help to mitigate this
Electrolytes
• Succinylcholine has a well known set of
contraindications due to the risk of
succinylcholine associated hyperkalemia. Major
burns, crush injuries, and end stage renal disease
patients are typically rather obvious.
• Occult contraindications include those patients
with histories of malignant hyperthermia,
previous stroke / spinal cord injuries, or those
with prolonged ICU stays.
• Several case reports exist of patients who
have had profound hyperkalemia without
obvious risk factors. In the absence of an
alternative explanation for hypotension, and
especially in the setting of any rhythm change
consider checking a potassium level, or
empirically treating with Calcium
POCUS for shock
• RUSH
• ACES
RUSH protocol
• Step 1: The pump
• Step 2: The tank
• Step 3: The pipes
Step 1: The pump
• Effusion around the pump’’
Parasternal
long-axis
view: large
pericardial
effusion
Tamponade:
RA/RV mid diastole
collapse
IVC >21mm, <50%
change
Swinging heart
• Squeeze of the pump’’: determination of
global left ventricular function
Strong ,poor ,intermediate
• ‘‘Strain of the pump’’: assessment of right
ventricular strain
Parasternal long-axis view:
right ventricular strain.
Step 2: Evaluation of the Tank
‘‘Fullness of the tank’’: Evaluation of the
inferior cava and jugular veins for size and
collapse with inspiration
• ‘‘Leakiness of the tank’’: peritoneal collection
and pleural fluid assessment
• ‘‘Tank compromise’’: pneumothorax
• ‘‘Tank overload’’: pulmonary edema
Step 3—Evaluation of the Pipes
‘
• Rupture of the pipes’’: aortic aneurysm and
dissection
• The sensitivity of EP-performed ultrasound for
the detection of AAA ranges from 93% to
100%, with specificities approaching 100%
• Epigastrium down to the iliac bifurcation
Short-axis view: large
abdominal aortic aneurysm
A measurement of
greater than 3 cm is
abnormal and defines an
abdominal aortic
aneurysm
• Suprasternal view: aortic dissection.
Suprasternal view: aortic
dissection.
Short-axis view: aortic
dissection
• ‘‘Clogging of the pipes’’: venous
thromboembolism Bedside ultrasound for DVT
• proximal femoral ,popliteal vein rarely
internal jugular vein
lack of compressibility and visible clot

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hem dy.pptx

  • 1. APPRACH TO HEMODYNAMIC COLLAPSE IN ICU Dr Abreham Dr Mennur A Dr Tewodros
  • 2. • (SBP) to ≤90 mmHg, a decrease in SBP of ≥ 20% from a baseline, a decrease in mean arterial pressure (MAB) to ≤65 mmHg,
  • 3.
  • 4.
  • 5. Initial approach undifferentiated hypotension 1 stabilize airway and secure iv access Intubation 2 Risk stratification Based on brief history ,PE ,ecg
  • 6. • Common conditions needing lifesaving interventions
  • 7. Anaphylaxis • Extremely rare for ketamine and etomidate • Neuromuscular blocking agents are divalent molecules that make it easier for anaphylaxis to occur, even in the setting of no previous exposure • More common with rocuronium and succinylcholine.
  • 8. • Hypotension, inspiratory stridor, oral and facial edema, hives • RX •Epinephrine • Diphenhydramine • Ranitidine
  • 9. Tension Pneumothorax • Consider in patient with obstructive lung disease that has increased airway pressures. • Use ultrasound to confirm whether a pneumothorax is present.
  • 10. Bar code/stratosphere sign  Needle decompression, Chest tube
  • 11. Cardiac Tamponade • Elderly patients ,Pt on anticoagulants , ESRD patients may have occult / borderline tamponade that only declares itself after a change to positive pressure ventilation.  pericardiocentesis
  • 12.
  • 13. Cardiogenic shock from myocardial infarction •Anterior crushing chest pain, respiratory distress, and the ECG changes with MI •Request troponin • Antiplatelet agents, heparin consult for reperfusion, or balloon pump
  • 14. Cardiogenic shock from acute aortic or mitral valve insufficiency • Patients with chest pain, hypotension, and new low-pitched early diastolic murmur consistent with aortic insufficiency should undergo POC ultrasonography or echocardiography
  • 15. Acute arrhythmias can cause hypotension • 12-lead electrocardiography.  cardioversion
  • 16. Hypovolemia • Hypovolemia from profound shock states such as hemorrhage, sepsis, and anaphylaxis • Occult hypovolemia can be unmasked by the physiologic insult that occurs when patients are switched from spontaneous ventilation to positive pressure ventilation.
  • 17. • With the increase in intrathoracic pressure that accompanies positive pressure ventilation any patient with low venous pressures / low pre-load may develop hypotension.  Adjusting PEEP settings, and giving a bolus of an appropriate resuscitative fluid
  • 18. Dissection of the ascending aorta • Retrograde dissection that results in acute aortic insufficiency, pericardial tamponade, or myocardial infarction • Transthoracic echocardiography or POCUS •Aortic root dilatation •Intimal flap
  • 19. Pulmonary Hypertension • Patients with known severe pulmonary hypertension are a nightmare to intubate. • Hypervolemia, hypoxia, and hypercarbia are known to worsen their right heart function. • Fluid boluses , and may worsen LV impingement.
  • 20. Acidosis • Patients with profound metabolic acidosis typically have a high minute ventilation, until they become fatigued and then their pH can drop. provide minute ventilation that approximates pre-intubation levels. 8cc/kg tidal volume high RR
  • 21. Stacked breaths / Autopeep • Patients with obstructive lung pathology who are not completely exhaling before breath • Decrease venous return overtime, an cause hypotension, and eventual arrest.
  • 22. Induction /sedation Agent • Propofol • Any sedative agent can result in post intubation collapse • Decreasing the dose of sedative, • Pressors / push dose pressors ready could help to mitigate this
  • 23. Electrolytes • Succinylcholine has a well known set of contraindications due to the risk of succinylcholine associated hyperkalemia. Major burns, crush injuries, and end stage renal disease patients are typically rather obvious. • Occult contraindications include those patients with histories of malignant hyperthermia, previous stroke / spinal cord injuries, or those with prolonged ICU stays.
  • 24. • Several case reports exist of patients who have had profound hyperkalemia without obvious risk factors. In the absence of an alternative explanation for hypotension, and especially in the setting of any rhythm change consider checking a potassium level, or empirically treating with Calcium
  • 25. POCUS for shock • RUSH • ACES
  • 26. RUSH protocol • Step 1: The pump • Step 2: The tank • Step 3: The pipes
  • 27.
  • 28. Step 1: The pump • Effusion around the pump’’ Parasternal long-axis view: large pericardial effusion Tamponade: RA/RV mid diastole collapse IVC >21mm, <50% change Swinging heart
  • 29. • Squeeze of the pump’’: determination of global left ventricular function Strong ,poor ,intermediate
  • 30. • ‘‘Strain of the pump’’: assessment of right ventricular strain Parasternal long-axis view: right ventricular strain.
  • 31. Step 2: Evaluation of the Tank ‘‘Fullness of the tank’’: Evaluation of the inferior cava and jugular veins for size and collapse with inspiration
  • 32. • ‘‘Leakiness of the tank’’: peritoneal collection and pleural fluid assessment
  • 35. Step 3—Evaluation of the Pipes ‘
  • 36. • Rupture of the pipes’’: aortic aneurysm and dissection • The sensitivity of EP-performed ultrasound for the detection of AAA ranges from 93% to 100%, with specificities approaching 100% • Epigastrium down to the iliac bifurcation
  • 37. Short-axis view: large abdominal aortic aneurysm A measurement of greater than 3 cm is abnormal and defines an abdominal aortic aneurysm
  • 38. • Suprasternal view: aortic dissection. Suprasternal view: aortic dissection. Short-axis view: aortic dissection
  • 39. • ‘‘Clogging of the pipes’’: venous thromboembolism Bedside ultrasound for DVT • proximal femoral ,popliteal vein rarely internal jugular vein lack of compressibility and visible clot