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HELMINTHIOSIS
INTRODUCTION
• Helminthic infestations contribute significantly to
global burden of disease.
• Humans are the primary hosts for the most of
helminthic infections.
• Most worms produce in human sexually by producing
eggs and larvae.
• These pass out of body and infect the secondary host.
• Immature forms invade humans via skin or GIT and
mature to adult worms with characteristic tissue
distribution.
2
PATHOGENESIS
• Hookworm suck‐0.03‐0.15ml/worm/day
• Mechanical interference‐intestinal obstruction
‐hydatid cyst
• Round worms deprive the host of the nutrients
‐cysticercoids
• Hypersensitivity reaction‐tissue damage
3
MODE OF INFECTION
• Through ingestion of fecal contaminated material.
e.g. A.lumbricoides, enterobius vermicularis,
H.nana,T.solium/T.saginata
• By piercing the skin.
e.g Hookworm, Schistosomiasis, Strongaloides
• By ingestion through intermediate host.
e.g. Diphyllobothrium latum, Dracunculosis medinensi
4
CLINICAL FEATURES
• Abdominal pain – Ascaris, Hook worm
• Cough – Ascaris, P.Westermani
• Diarrhea – Schistosoma
• Dysentery – E.Coli, Schistosoma
• Dysuria – Entrobius
• Headache –T.Solium
• Pruritus – Entrobius, Filaria, Hookworm
5
INVESTIGATIONS
•Gastrointestinal Worms
• Sedimentation method
•Stool for Ova & Cyst
• Concentration method
6
•Anal Swab
•Stool Culture
•Systemic Worms
Peripheral smear –Thik,Thin
DEC Provocation test‐ Microfilaria
PREVENTIVE MEASURES
7
• Proper disposal of feces
• Personal protection
‐food
‐water
‐skin
• Diminishing the reservoir of infection
• Health education: Personal Hygiene
• Personal hygiene:
• Wash hands after defecation
• Nail cutting
• Daily bath
• Avoid scratching perianal region
• IEC

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Helminthi I.S.M CENTRAL CAMPUS

  • 2. INTRODUCTION • Helminthic infestations contribute significantly to global burden of disease. • Humans are the primary hosts for the most of helminthic infections. • Most worms produce in human sexually by producing eggs and larvae. • These pass out of body and infect the secondary host. • Immature forms invade humans via skin or GIT and mature to adult worms with characteristic tissue distribution. 2
  • 3. PATHOGENESIS • Hookworm suck‐0.03‐0.15ml/worm/day • Mechanical interference‐intestinal obstruction ‐hydatid cyst • Round worms deprive the host of the nutrients ‐cysticercoids • Hypersensitivity reaction‐tissue damage 3
  • 4. MODE OF INFECTION • Through ingestion of fecal contaminated material. e.g. A.lumbricoides, enterobius vermicularis, H.nana,T.solium/T.saginata • By piercing the skin. e.g Hookworm, Schistosomiasis, Strongaloides • By ingestion through intermediate host. e.g. Diphyllobothrium latum, Dracunculosis medinensi 4
  • 5. CLINICAL FEATURES • Abdominal pain – Ascaris, Hook worm • Cough – Ascaris, P.Westermani • Diarrhea – Schistosoma • Dysentery – E.Coli, Schistosoma • Dysuria – Entrobius • Headache –T.Solium • Pruritus – Entrobius, Filaria, Hookworm 5
  • 6. INVESTIGATIONS •Gastrointestinal Worms • Sedimentation method •Stool for Ova & Cyst • Concentration method 6 •Anal Swab •Stool Culture •Systemic Worms Peripheral smear –Thik,Thin DEC Provocation test‐ Microfilaria
  • 7. PREVENTIVE MEASURES 7 • Proper disposal of feces • Personal protection ‐food ‐water ‐skin • Diminishing the reservoir of infection • Health education: Personal Hygiene • Personal hygiene: • Wash hands after defecation • Nail cutting • Daily bath • Avoid scratching perianal region • IEC