Heat stroke occurs when the body's temperature regulation system fails and body temperature rises to dangerous levels. It represents a failure of the body's ability to maintain thermoregulatory homeostasis. Symptoms include headache, nausea, confusion and loss of consciousness. Treatment involves rapid cooling of the body, typically using evaporative cooling techniques, to lower the core temperature and prevent irreversible organ damage. Aggressive rehydration and treatment of complications such as seizures, arrhythmias or hypotension are also important for management. Rapid cooling is crucial to improving outcomes in heat stroke patients.
A brief yet comprehensive description of a very common problem faced in KSA especially during hajj season. It is meant to enhance the awareness among ER and ICU physicians.
Basic data about heat stroke uncluding: Definition, forms, exertional and non exertional, epidemiology, risk factors, characteristics, ettiology, pathophysiology, clinical presentation in all body systems, management, cooling tools, assisting procedures, complications, prevention, and patient education
A brief yet comprehensive description of a very common problem faced in KSA especially during hajj season. It is meant to enhance the awareness among ER and ICU physicians.
Basic data about heat stroke uncluding: Definition, forms, exertional and non exertional, epidemiology, risk factors, characteristics, ettiology, pathophysiology, clinical presentation in all body systems, management, cooling tools, assisting procedures, complications, prevention, and patient education
Heat related illnesses simply explained, spectrum of hyper and hypothermia related clinical scenarios with symptoms, diagnosis, management and prognosis.
This simple Presentation highlights Sunstroke by giving:
General description about Sunstroke.
Symptoms of sunstroke.
Medications the raise the risk of Sunstroke
How to prevén and treat Sunstroke.
What to wear to avoid Sunstroke.
Heat related illnesses simply explained, spectrum of hyper and hypothermia related clinical scenarios with symptoms, diagnosis, management and prognosis.
This simple Presentation highlights Sunstroke by giving:
General description about Sunstroke.
Symptoms of sunstroke.
Medications the raise the risk of Sunstroke
How to prevén and treat Sunstroke.
What to wear to avoid Sunstroke.
Artificial respiration, the act of simulating respiration, which provides for the overall exchange of gases in the body by pulmonary ventilation, external respiration and internal respiration
First aid is the provision of initial care for an illness or injury. It is usually performed by non-expert, but trained personnel to a sick or injured person until definitive medical treatment can be accessed. Certain self-limiting illnesses or minor injuries may not require further medical care past the first aid intervention. It generally consists of a series of simple and in some cases, potentially life-saving techniques that an individual can be trained to perform with minimal equipment.
Blunt traumatic injury of the innominate artery resulting in a stroke – A rar...Apollo Hospitals
Blunt traumatic injury of innominate artery is uncommon and has been reported only in 132 cases. In the literature there has been a solitary case report of a stroke resulting from an innominate artery injury. We present a case of traumatic injury of the innominate artery resulting in an ischemic stroke.
Are you doing everything you can to minimize your workers’ exposure to heat-related illnesses? Does your company have a heat illness prevention program in place? We’ve go you covered with these tips and guidelines for keeping your workers safe and productive during these hot summer days.
Homeless people are vulnerable to the extremes of weather and exposure. Please help the homeless by donating to our #circleofwarmth campaign. https://info.miramedgs.com/CIRCLEOFWARMTHDONATETODAY
Exercising in hot and cold environments can have different effects on the body. It's important to consider factors like hydration, clothing, and duration of exercise when working out in extreme temperatures.
Role of hypothalamus in regulation of body temperatureSaad Salih
Thermoregulation is a process that allows your body to maintain its core internal temperature. All thermoregulation mechanisms are designed to return your body to homeostasis. This is a state of equilibrium.
A healthy internal body temperature falls within a narrow window. The average person has a baseline temperature between 98°F (37°C) and 100°F (37.8°C). Your body has some flexibility with temperature. However, if you get to the extremes of body temperature, it can affect your body’s ability to function. For example, if your body temperature falls to 95°F (35°C) or lower, you have “hypothermia.” This condition can potentially lead to cardiac arrest, brain damage, or even death. If your body temperature rises as high as 107.6°F (42 °C), you can suffer brain damage or even death.
Many factors can affect your body’s temperature, such as spending time in cold or hot weather conditions.
Factors that can raise your internal temperature include:
fever
exercise
digestion
Factors that can lower your internal temperature include:
drug use
alcohol use
metabolic conditions, such as an under-functioning thyroid gland
Your hypothalamus is a section of your brain that controls thermoregulation. When it senses your internal temperature becoming too low or high, it sends signals to your muscles, organs, glands, and nervous system. They respond in a variety of ways to help return your temperature to normal.
Hypothermia in Trauma Victims:- complication and its preventionHASSAN RASHID
Hypothermia is an important confounding factor in the severity and outcome of a trauma patient.
In this seminar, we have discussed the complication of hypothermia in trauma victims and also how to prevent it and associated harmful effects.
Lung Cancer: Artificial Intelligence, Synergetics, Complex System Analysis, S...Oleg Kshivets
RESULTS: Overall life span (LS) was 2252.1±1742.5 days and cumulative 5-year survival (5YS) reached 73.2%, 10 years – 64.8%, 20 years – 42.5%. 513 LCP lived more than 5 years (LS=3124.6±1525.6 days), 148 LCP – more than 10 years (LS=5054.4±1504.1 days).199 LCP died because of LC (LS=562.7±374.5 days). 5YS of LCP after bi/lobectomies was significantly superior in comparison with LCP after pneumonectomies (78.1% vs.63.7%, P=0.00001 by log-rank test). AT significantly improved 5YS (66.3% vs. 34.8%) (P=0.00000 by log-rank test) only for LCP with N1-2. Cox modeling displayed that 5YS of LCP significantly depended on: phase transition (PT) early-invasive LC in terms of synergetics, PT N0—N12, cell ratio factors (ratio between cancer cells- CC and blood cells subpopulations), G1-3, histology, glucose, AT, blood cell circuit, prothrombin index, heparin tolerance, recalcification time (P=0.000-0.038). Neural networks, genetic algorithm selection and bootstrap simulation revealed relationships between 5YS and PT early-invasive LC (rank=1), PT N0—N12 (rank=2), thrombocytes/CC (3), erythrocytes/CC (4), eosinophils/CC (5), healthy cells/CC (6), lymphocytes/CC (7), segmented neutrophils/CC (8), stick neutrophils/CC (9), monocytes/CC (10); leucocytes/CC (11). Correct prediction of 5YS was 100% by neural networks computing (area under ROC curve=1.0; error=0.0).
CONCLUSIONS: 5YS of LCP after radical procedures significantly depended on: 1) PT early-invasive cancer; 2) PT N0--N12; 3) cell ratio factors; 4) blood cell circuit; 5) biochemical factors; 6) hemostasis system; 7) AT; 8) LC characteristics; 9) LC cell dynamics; 10) surgery type: lobectomy/pneumonectomy; 11) anthropometric data. Optimal diagnosis and treatment strategies for LC are: 1) screening and early detection of LC; 2) availability of experienced thoracic surgeons because of complexity of radical procedures; 3) aggressive en block surgery and adequate lymph node dissection for completeness; 4) precise prediction; 5) adjuvant chemoimmunoradiotherapy for LCP with unfavorable prognosis.
These simplified slides by Dr. Sidra Arshad present an overview of the non-respiratory functions of the respiratory tract.
Learning objectives:
1. Enlist the non-respiratory functions of the respiratory tract
2. Briefly explain how these functions are carried out
3. Discuss the significance of dead space
4. Differentiate between minute ventilation and alveolar ventilation
5. Describe the cough and sneeze reflexes
Study Resources:
1. Chapter 39, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 34, Ganong’s Review of Medical Physiology, 26th edition
3. Chapter 17, Human Physiology by Lauralee Sherwood, 9th edition
4. Non-respiratory functions of the lungs https://academic.oup.com/bjaed/article/13/3/98/278874
Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
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ARTIFICIAL INTELLIGENCE IN HEALTHCARE.pdfAnujkumaranit
Artificial intelligence (AI) refers to the simulation of human intelligence processes by machines, especially computer systems. It encompasses tasks such as learning, reasoning, problem-solving, perception, and language understanding. AI technologies are revolutionizing various fields, from healthcare to finance, by enabling machines to perform tasks that typically require human intelligence.
Basavarajeeyam is an important text for ayurvedic physician belonging to andhra pradehs. It is a popular compendium in various parts of our country as well as in andhra pradesh. The content of the text was presented in sanskrit and telugu language (Bilingual). One of the most famous book in ayurvedic pharmaceutics and therapeutics. This book contains 25 chapters called as prakaranas. Many rasaoushadis were explained, pioneer of dhatu druti, nadi pareeksha, mutra pareeksha etc. Belongs to the period of 15-16 century. New diseases like upadamsha, phiranga rogas are explained.
Basavarajeeyam is a Sreshta Sangraha grantha (Compiled book ), written by Neelkanta kotturu Basavaraja Virachita. It contains 25 Prakaranas, First 24 Chapters related to Rogas& 25th to Rasadravyas.
4. Body temperature is maintained in its
normal level by the balance between
heat production and heat loss.
The range of normal body temp. is very
narrow , about 1 degree [ 36.5 -37.5].
It is controlled by the heat regulating
centre in the hypothalamus
5. Sources of body heat ?
Normal basal metabolism [BMR]
Activity of different organs [ heart, GIT.,
diaphragm,..]
Usual daily activities like daily work ,walking
, eating , exercise ext.
Absorption of heat from the surrounding
atmosphere by: conduction, radiation, and
convection .
6. Sources of body heat (con):
Absorption of heat from the surrounding
atmosphere by:
1- Conduction: [direct contact with hot object]
2- Radiation: by transformation of heat to
electro magnetic waves like sun rays.
3- Convection: contact with gas or liquid with
different temperature
7. ?Ways of heat LOSS
In expired air.
In body excreta [urine , stool ,tears , sweat…].
Convection: [Contact with cold environment]
Radiation: [responsible for loss of about
65%of body heat in normal weather at rest]
conduction.
Evaporation of sweat.
10. Heat regulating centre:
A highly specialized sensitive cells present
in the anterior hypothalamus adjusted to
normal body temp [36 -37.8] by means of
set-point.
11. Heat regulating centre:
The centre gets information about the
body temp. by means of thermo-sensors
( specialized cells located in the viscera ,
skin , spinal cord, hypothalamus).
These sensors send continuous impulses
to the anterior hypo-thalamus through
afferent fibers passing in the spinal cord
and brain stem.
12. Heat regulating centre
The temp is then compared with that of set point .
1. If less than it , heat production mechanisms are
initiated.
2. if more than it, heat loss mechanisms are
initiated.
A core temperature greater than 41°C or less
than 34°C usually indicates that the body’s ability
to thermoregulate is impaired.
14. Heat regulating centre
Heat production & heat loss mechanisms are
stimulated by orders : from the posterior
hypothalamus to :
endocrinal system
musculoskeletal system
autonomic nervous systems.
15. Mechanism of heat loss:
Through reflexes from the hypothalamus to :
1- Autonomic n. system leading to vasodilatation of
the peripheral blood vessels & sweating.[about 8
liters of blood reaches the peripheral circulation
each minute]
2- Endocrinal system leading to decreased secretion
of thyroxin & catecholamines and increased
secretion of anti-diuretic hormone & aldosterone .
3- Arrector pili muscles relaxing .
17. HEAT CRAMP
Hot environment causes profuse sweating
Na+ (sodium) lost in sweat
Lack of Na+ causes muscle cramping
C/P:
Cramps of fingers, arms, legs, abdominal muscles
Nausea
Normotensive, mild hypotension
Tachycardia
Cool, pale skin
Awake, normal body temperature
18. HEAT CRAMP
:Management
Move to cool place, rest, lie down
Give balanced salt/water solution (Electrolyte
Solution)
Salt alone leads to increased nausea,
increased water loss
Water alone leads to worsened cramping
(dilutional hyponatremia)
20. HEAT EDEMA
Heat causes the blood vessels to expand
(dilate), so body fluid moves into the
hands or legs by gravity.
21. HEAT SYNCOPE
Heat causes an increase in blood flow to the
skin and pooling of blood in the legs,
which can lead to a sudden drop in blood
pressure followed by syncope.
23. Heat Exhaustion
RISK GROUP:
People working in hot, humid
environments
Elderly, due to decreased thirst
mechanism
Hypertensive pt due to medication effects
24. Heat Exhaustion
Symptoms : similar to a viral infection:
Fatigue and weakness
Nausea and vomiting
Headache and myalgia
Dizziness
Irritability
25. Heat Exhaustion
Physical Findings:
Orthostatic pulse and blood pressure
changes
Sweating
Tachycardia
Temperature is usually less than 41°C
Normal mental status!
26. Heat Exhaustion
Treatment:
Move to cool place, stop activity, lie down
Supine, and legs elevated
Cooling
Balanced salt/water (electrolyte) solution,
or IV fluid with NaCl, if pt too nauseated to
drink.
29. DEFINITION
A core temperature ≥40°C accompanied
by CNS dysfunction in patients with
environmental heat exposure.
This condition represents a failure of the
body's ability to maintain thermoregulatory
homeostasis.
30. Types of heat stroke
A- Classical type:
Occurs mainly in the extremities of age.
Gradual in onset [ commonly more than 48
hours].
Dehydration is more due to prolonged
sweating.
At presentation , skin is commonly dry.
31. Types of heat stroke
B- Exertional type:
Occurs commonly in middle aged healthy
persons .
Occurs in non acclimatized persons during
exercise or hard work in hot humid
atmosphere.
Rapid onset.
Less dehydration.
32.
33. Predisposing factors:
Environmental factors: Exposure to hot
humid weather with decreased air current.
Non Acclimatization.
Epidemic heat stroke Occurs when a city
experienced a cold winter followed by a hot
humid weather in the late spring or early
summer
Wearing heavy clothes in a hot atmosphere
34. Predisposing factors:
Military personals , athletes & young people
doing exercise or hard work in hot humid
weather.
Age: Children & elderly is the most
vulnerable.
Obesity
Dehydration
35. Predisposing factors:
Chronic illness:
C.V. diseases, Diabetes M - Hemi or
quadriplegia with autonomic dysfunction
, Infection
Drugs: Anti-cholinergic drugs, major
tranquilizers , phenothiazine and neuroleptic
drugs.
36. DIFERRENCE BETWEEN FEVER
AND ?HYPERTHERMIA
Fever is an upward adjustment of the set-
point, unlike hyperthermia .
Therefore, fever does not represent a failure
of temperature control, but rather an upward
shift of the regulated temperature.
37. DIFERRENCE BETWEEN FEVER
AND ?HYPERTHERMIA
Fever usually occurs as a result of the
body's exposure to:
1-infecting micro-organisms
2-immune complexes
3-other sources of inflammation
38. Size of the problem
In 1980, heat wave in U.S. lead to 1700
deaths.
In 1998, heat wave in India leaded to 2600.
In 2003, heat wave caused 1000 deaths in
India.
About 10000 victims were lost in France only
during the heat wave of Europe in 2003.
1000 deaths in india may 2015.
39. Effects of hyperthermia on the
body
At 42 degree :
Denaturation of proteins, enzymes &
hormones.
liquefaction of lipids including the brain tissue.
oxidative phosphorelation decrease with loss
of energy sources to different tissues including
the heat regulating centre.
40. Effects on the brain
The 1st cell to be affected is the brain cell leading to:
1-Mental changes , stupor & coma.
2-Convulsions or decerebrate rigidity.
3-Quadriplegia , hemiplegia or monoplegia
4-Different brain infarctions.
5-Paralysis of centers like heat regulation center
or respiratory center leading to death.
41. Effects on the Liver
Degeneration then necrosis to the liver
cells which may lead to liver cell failure.
42. Effects on the Kidney & skeletal
muscle
Destruction of the renal cells leading to renal failure.
-Destruction of the skeletal muscle cells
[rhabdomyolysis] specially in exertional type , leading
to myoglobinuria with possibility of renal tubular obst .
and renal failure.
Precipitation of Ca.& Ph. on the destructed muscle
cells leads to hypocalcaemia & hypophosphatemia.
Also Na. inter the cells & K. go outside the cells
leading to hyperkalemia & hyponatremia
43. Effects on the cardiovascular
system
injury to the endothelial lining of the
vessels causing D.I.C.
Affects the conductive system of the heart
that may lead to different types of
arrhythmias and heart failure
45. Clinical picture
Symptoms Occurs prior to coma in the form
of:
Headache
Nausea & vomiting.
Light headedness.
Paresthesias and Change of behavior.
then syncope & coma.
46. SIGNS
Body temperature:
Must be taken rectally, usually over 41 degree,
may be cold extremities due to peripheral circulatory
failure.
Heart rate:
with dehydration or heart failure: tachycardia + weak pulse
Irregular pulse in arrhythmia & Bradycardia in heart block.
47. SIGNS
Bl.P.:
May be : low due to low output failure [dehydration]
or high output failure [high temp.]
Respiratory rate:
Deep rapid respir. usually due to high temp.
Irregular resp.[chyne stoke] in the terminal stage
Bubbling crepitation & frothy sputum in pulmonary
edema.
48. SIGNS
Skin:
Usually grey & dry . may be flushed and sweaty.
Sweat rash is usually present.
CNS:
Coma with dilated fixed pupils , convulsions ,
muscle rigidity, tremors , hemiplegia may be
present.
50. Blood Gases:
Commonly reveals :
metabolic acidosis due to lactate
accumulation specially in exertional type.
Respiratory alkalosis may be present due to
hypercapnia.
52. LIVER ENZYMES
A.L.T. , A.S.T. & LDH:
are markedly elevated.
N.B:
the A.S.T. level is prognostic, the level of
1000 i.u./ liter or more in the first 24 hours
reflects a poor prognosis with serious brain ,
liver & renal damage and the reverse.
54. Electrolytes:
± Na
↑ K in first 24 hours.
↓ Ca due to precipitation. in the damaged
fibers.
↓ Ph due to the same reason
55. E.C.G.:
S-T segment & T wave abnormality with
varieties arrhythmias and BBB. may occur
and most of them are reversible after
cooling.
56. Electrolytes: ± Na according to the type of
dehyd. ↑ K in first 24 hours. ↓ Ca due to
precipitation. in the damaged fibers. ↓ Ph due
to the same reason. E.C.G.: S-T segment &
T wave abnormality with varieties arrhythmias
and BBB. may occur and most of them are
reversible after cooling.
58. Management
Heat stroke is one of the medical emergency
that needs rapid interference .
The seconds are precious for the patient, so
our aim is to decrease the body temp. below
the harmful level as quickly as possible to
avoid irreversible cellular damage.
Pre-hospital cooling: decrease morbidity and
mortality rate.
59. Management
COOLING
There are 2 different methods for cooling:
the aggressive cooling measures .
the slow evaporative technique.
60. A-Aggressive cooling measures
It includes :
1) Direct application. of ice on the whole
body.
2) Immersing the body in cold or iced water.
3) Application of ice in areas of great
vessels e.g. axilla , groin & front of the
neck.
61. 4) Gastric lavage with iced fluids.
5) Enema with iced fluids.
6) Peritoneal lavage with iced fluids.
7) I.V. infusion of cold fluids.
8) Inhalation of cold air.
62. Disadvantages of these methods:
The cooling rate is less [0.1degree/min.]
except in peritoneal lavage [0.55
degree/min.]
Difficult for application in comatosed
patient.
May cause shivering which increase body
temp.
63. Disadvantages of these methods:
Direct ice to the skin leads to
vasoconstriction .
-Ice enema may cause shock and sudden
death.
Peritoneal lavage may leads to peritonitis.
65. B-Slow evaporative technique
technique:
This is done by spraying the body with
water, then expose the body to strong
current of dry air
This process continues until the temp.
reaches 39 degree , then cooling must be
stopped
66. B-Slow evaporative technique
Advantages of the technique:
Faster rate of cooling [ 0.33 deg./min. ]
Easily applied for comatosed patient.
Not cause shivering or peripheral v . c.
This method must be done in specialized
center.
68. The heat stroke & heat
exhaustion center
The center is composed of: -
2 suitable rooms : * cooling room . *
observation room.
69. cooling room :
Enough number of air conditions insure a
temp. room 25- 30 (average 27) and also dry
air.
Slated beds without mattresses .
opposite to each bed one fan must be fixed
to wall to supply a horizontal current of air.
70. cooling room :
A number of suction fans in the upper part
of the wall for renewal of air & removal of
humid air
A source of tape water & ice must be in
the cooling room. - All equipments . and
emergency drugs needed for comatosed
patient must be supplied
71.
72. observation room
The second room is a neighboring
conditioned room containing normal
beds for observation for 24 h. after
cooling
73. MEDICAL TREATMENT
Assessment and management of A,B,C
Airway, breathing , circulation
Oxygen
TT of sizures:
Midazolam: 0.1- 0,2 mg/kg IV, max 4mg
TT of shivering:
chlorpromazine: 10 – 25 mg IM or Midazolam
74. MEDICAL TREATMENT
TT of hypotension:
usually result from:
Peripheral vasodilatation
Volume depletion( dehydration)
Cardiac dysfunction
Use crystalloid solution as normal saline to
maintain urine output 50 ml / h .
Excessive fluid administration may result in
pulmonary edema
75. MEDICAL TREATMENT
Antipyretic medications( acetaminophen
and ibuprofen): are ineffective and should
not be used as it may exacerbate liver
injury and coagulation disorders
76. MEDICAL TREATMENT
Treatment of end organ dysfunction:
Respiratory dysfunction
Cardiac dysfunction and arrthymia
Acute kidney injury and rhabdomyolosis
Hepatic injury
DIC