Glaucoma is a group of eye disorders characterized by abnormally high intraocular pressure that can damage the optic nerve and result in vision loss. The document defines glaucoma and identifies primary open-angle glaucoma and primary angle-closure glaucoma as the two most common types. Risk factors include aging, genetics, eye injuries or surgeries, and certain medical conditions. Treatment involves medications or surgery to reduce pressure in the eye in order to prevent further vision loss from optic nerve damage.
BLEPHARITIS
Blepharitis is a chronic inflammation of the lid margins.
Etiology
1. It follows chronic conjunctivitis due to Staphyloccocus in
debilitated children usually who are living in poor hygienic
conditions.
2. Parasites such as Demodex folliculorum, Phthiriasis
palpebrarum, crab louse, head louse also cause blepharitis.
A stye is an infection (abcess) of one of the small oil producing glands lining the eyelid, usually caused by the bacteria that are normally found along the eyelids.
A stye can occur on either the upper or lower eyelid.
There are two types of styes, internal and external hordeola.
An internal hordeolum (stye) is a bacterial infection of the meibomian glands inside the eyelids.
Internal styes tend to be more severe and occur a little less often than an external hordeolum.
An external hordeolum (stye) is a bacterial infection of the Glands of Zeis and/or Glands of Moll inside the eyelids.
This type of stye is more superficial and tends to heal more readily.
It is a chronic lipogranulomatous inflammatory lesion caused by blockage of meibomian gland orifices and stagnation of sebaceous secretion.
Patient with acne rosacea or seborhoeic dermatitis are at increased risk of chalazion formation which may be multiple or recurrent.
If it is recurrent, one should think of sebaceous gland carcinoma
GLAUCOMA
,dignosis , types of glaucoma , risk factors oo glaucoma and treatment , the clasis of drugs that use in treatment of glaucoma.
prepared by : Hardi Sdiq
university of sullaimani
collage of pharmacy
BLEPHARITIS
Blepharitis is a chronic inflammation of the lid margins.
Etiology
1. It follows chronic conjunctivitis due to Staphyloccocus in
debilitated children usually who are living in poor hygienic
conditions.
2. Parasites such as Demodex folliculorum, Phthiriasis
palpebrarum, crab louse, head louse also cause blepharitis.
A stye is an infection (abcess) of one of the small oil producing glands lining the eyelid, usually caused by the bacteria that are normally found along the eyelids.
A stye can occur on either the upper or lower eyelid.
There are two types of styes, internal and external hordeola.
An internal hordeolum (stye) is a bacterial infection of the meibomian glands inside the eyelids.
Internal styes tend to be more severe and occur a little less often than an external hordeolum.
An external hordeolum (stye) is a bacterial infection of the Glands of Zeis and/or Glands of Moll inside the eyelids.
This type of stye is more superficial and tends to heal more readily.
It is a chronic lipogranulomatous inflammatory lesion caused by blockage of meibomian gland orifices and stagnation of sebaceous secretion.
Patient with acne rosacea or seborhoeic dermatitis are at increased risk of chalazion formation which may be multiple or recurrent.
If it is recurrent, one should think of sebaceous gland carcinoma
GLAUCOMA
,dignosis , types of glaucoma , risk factors oo glaucoma and treatment , the clasis of drugs that use in treatment of glaucoma.
prepared by : Hardi Sdiq
university of sullaimani
collage of pharmacy
When parallel rays of light enter the eye ((with accommodation relaxed) and do) and do not come to a single point focus on or near the retina.
Types of Astigmatism:
Sign & Symptoms:
Management:
Vitreous hemorrhage is the extravasation, or leakage, of blood into the areas in and around the vitreous humor of the eye.[1] The vitreous humor is the clear gel that fills the space between the lens and the retina of the eye. A variety of conditions can result in blood leaking into the vitreous humor, which can cause impaired vision, floaters, and photopsia.
It's an indepth presentation by Dr. Shah-Noor Hassan.
This presentation describes the background of the cornea and the corneal diseases in general, also it describes in detailed manner how to manage the corneal ulcer with its different causes
Glaucoma is an eye disease that is often associated with elevated intraocular pressure, in which damage to the eye (optic) nerve can lead to loss of vision and even blindness. Glaucoma is the leading cause of irreversible blindness in the world.
When parallel rays of light enter the eye ((with accommodation relaxed) and do) and do not come to a single point focus on or near the retina.
Types of Astigmatism:
Sign & Symptoms:
Management:
Vitreous hemorrhage is the extravasation, or leakage, of blood into the areas in and around the vitreous humor of the eye.[1] The vitreous humor is the clear gel that fills the space between the lens and the retina of the eye. A variety of conditions can result in blood leaking into the vitreous humor, which can cause impaired vision, floaters, and photopsia.
It's an indepth presentation by Dr. Shah-Noor Hassan.
This presentation describes the background of the cornea and the corneal diseases in general, also it describes in detailed manner how to manage the corneal ulcer with its different causes
Glaucoma is an eye disease that is often associated with elevated intraocular pressure, in which damage to the eye (optic) nerve can lead to loss of vision and even blindness. Glaucoma is the leading cause of irreversible blindness in the world.
Glaucoma types, Pathogenesis, Diagnosis and TreatmentPranatiChavan
Glaucomas are ocular disorders characterized by changes in the optic nerve head (optic disk) and by loss of visual sensitivity and field.
There are two major types of glaucoma: open-angle glaucoma, which accounts for most cases and closed-angle glaucoma.
Glaucoma presentation for ophthalmology course, presented as a student seminar. Class location: ophthalmology unit, An-Najah National University Hospital.
BRONCHIAL ASTHMA
ntroduction
Definition
Etiological factors
Pathophysiology
Types of asthma
Clinical manifestation Restlessness Wheezing or crackles Absent or diminished lung sounds Hyper resonance Use of accessory muscles for breathing Tachypnea with hyperventilation
Clinical manifestation
Diagnostic evaluation
Bronchoprovocation Testing: Testing that is done to identify inhaled allergens; mucous membranes are directly exposed to suspected allergen in increasing amounts. Skin Testing: Done to identify specific allergens. Exercise Challenges: Exercise is used to identify the occurrence of exercise-induced bronchospasm. Radio allergosorbent Test: Blood test used to identify a specific allergen. Chest Radiograph: May show hyper expansion of the airways.
Managemnet
Goal- Promote bronchodilationn Reduce inflammation Remove secretions Prevent ongoing symptoms Prevent asthma attack Maintain normal lung function Avoid triggers
Pharmacological therapy 1. Long term control medication- Inhaled corticosteroid Leukotriene modifiers Long acting beta agonist Methylxanthines Combine inhaler
2 Quick relief medication Short acting beta agonist Anticholinergic Oral or I/V corticosteroid
3 Bronchial thermoplasty- Form severe asthma that does not respond to medication
Non- pharmacological
Oxygen therapy Postural drainage & chest physiotherapy Coughing & deep breathing exercise Avoidance of allergen relaxation technique acupuncture
Prevention
Patients with recurrent asthma should undergo tests to identify the substances that precipitate the symptoms. Possible causes are dust, dust mites, roaches, certain types of cloth, pets, horses, detergents, soaps, certain foods, molds, and pol- lens. If the attacks are seasonal, pollens can be strongly sus- pected. Patients are instructed to avoid the causative agents whenever possible.
Complications Complications of asthma may include status asthmaticus, respiratory failure, pneumonia, and atelectasis. Airway obstruction, particularly during acute asthmatic episodes, often results in hypoxemia, requiring the administration of oxygen and the monitoring of pulse oximetry and arterial blood gases. Fluids are administered, because people with asthma are frequently dehydrated from diaphoresis and in- sensible fluid loss with hyperventilation.
Nursing diagnosis
Impaired gas exchange r/t altered oxygen supply Ineffective airway clearance r/t bronchospasm & obstruction from narrow lumen Ineffective breathing pattern r/t bronchospasm Risk for increasing attack of r
espiratory distress r/t exposure to allergens
Explore natural remedies for syphilis treatment in Singapore. Discover alternative therapies, herbal remedies, and lifestyle changes that may complement conventional treatments. Learn about holistic approaches to managing syphilis symptoms and supporting overall health.
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Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
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The prostate is an exocrine gland of the male mammalian reproductive system
It is a walnut-sized gland that forms part of the male reproductive system and is located in front of the rectum and just below the urinary bladder
Function is to store and secrete a clear, slightly alkaline fluid that constitutes 10-30% of the volume of the seminal fluid that along with the spermatozoa, constitutes semen
A healthy human prostate measures (4cm-vertical, by 3cm-horizontal, 2cm ant-post ).
It surrounds the urethra just below the urinary bladder. It has anterior, median, posterior and two lateral lobes
It’s work is regulated by androgens which are responsible for male sex characteristics
Generalised disease of the prostate due to hormonal derangement which leads to non malignant enlargement of the gland (increase in the number of epithelial cells and stromal tissue)to cause compression of the urethra leading to symptoms (LUTS
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Disruption of blood supply to lung alveoli due to blockage of one or more pulmonary blood vessels is called as Pulmonary thromboembolism. In this presentation we will discuss its causes, types and its management in depth.
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2. Definition
Glaucoma is a group of disorder characterized by an
abnormally increase in intra ocular pressure (more than
21mm Hg), optic nerve dystrophy & peripheral visual
field loss
The condition damages the optic nerve and can result in
blindness.
5. Primary open angle glaucoma
Open angle is the most common type of glaucoma . It is
usually bilateral, with insidious onset and slowly
progressive course without discomfort or pain.
It is result from overproduction or obstruction of the out
flow of aqueous humor through the trabecular meshwork
& causing increased intraocular pressure
6. Clinical manifestation
Mild aching in the eyes
Mild headache
Increased IOP that is > 21 mm hg
Loss of peripheral vision
Reduce visual acuity, specially at night
Failure to detect color changes
7. Primary angle closure glaucoma
It is also known as primary closed angle glaucoma,
narrow angle glaucoma, pupil block glaucoma,
acute congestive glaucoma.
It occur because of an abnormally of structure in
the front of eye. this results from obstruction to the
outflow of aqueous humor.
Obstruction caused by anatomically narrow angle
between anterior iris and posterior corneal surface,
shallow anterior chamber, a thickened iris that
cause angle closure.
8. Clinical manifestation
Pain & redness in eyes
Increased IOP between 40-
70 mm Hg
Blurred vision
Optic disk is hyperemic
Halo may be seen
around bright light
Headache
Nausea vomiting cornea
edematous
Decrease visual acuity
Photophobia
Moderate pupillary dilation
10. Management
Beta adrenergic blockers- Timolol, Betaxolol are used to decrease
aqueous humor production
Cholinergic (miotics) – pilocarpine, carbacol are used to reduce IOP by
facilitating the out flow of aqueous humor.
Carbonic anhydrase inhibitors - dorzolamide, methazolamide, or
acetazolamide to decrease the formation & secretion of aqueous humor.
Adrenergic agonist – Epinephrine to reduce IOP by improving aqueous
humor flow
Prostaglandin analogus – latanoprost to reduce IOP by increase
uveoscleral outflow
Osmotic pressure - IV mannitol
11. Surgical management
Argon Laser Trabeculoplasty – it may be used to treat
open angle glaucoma. In laser trabeculoplasty, thermal
argon laser burns are applied to the inner surface of the
trabecular meshwork to open the intra trabecular spaces &
widen the canal of schlemm, thereby increasing the out
flow of aqueous humor & decrease IOP.
Laser Iridotomy – an opening is made by the laser beam
in the iris to eliminate the pupilary block. It relieves
pressure & preserves vision by promoting outflow.
12. Cyclocryotherapy – Application of freezing probe to the
sclera over the cilliary body that destroy some of the
ciliary processes, results in the reduction production of
aqueous humor.
Cyclodialisis - Through small incision in the sclera, a
spatula type instrument is passed into the anterior
chamber, creating an opening in the angle.
13. Filtering procedure – for chronic glaucoma filtering
procedures are used to create opening or fistula in the
trabecular meshwork to drain aqueous humor from the
anterioir chamber to the subconjunctival space into bleb.
Trabeculotomy
Sclerotomy
Drainage implant or shunt
14. Instruct the client on the importance of medications to
constrict the pupils (miotics), to decrease the production of
aqueous humor (carbonic anhydrase inhibitors), and to
decrease the production of aqueous humor and IOP (beta
blockers).
Instruct the client about the need for lifelong medication
use, to wear a Medic Alert bracelet, to avoid
anticholinergic medications to prevent increased IOP, and
to contact the health care provider (HCP) before taking
medications, including over-the counter medications.
15. Instruct the client to report eye pain, halos around the
eyes, and changes in vision to the HCP.
Instruct the client that when maximal medical therapy has
failed to halt the progression of visual field loss and optic
nerve damage, surgery will be recommended.
Prepare the client for trabeculectomy as prescribed, which
allows drainage of aqueous humor into the conjunctival
spaces by the creation of an opening.