The document provides details on examining the nose, throat, and related structures. It begins with an overview of the nose examination, including inspecting the external nose and nasal cavity. This is followed by descriptions of anterior and posterior rhinoscopy to examine the nasal passages. Examination of the paranasal sinuses and oral cavity is also outlined. Specific structures of the throat, such as the tonsils, salivary glands, and larynx are then discussed. Examination techniques including inspection, palpation, and specific tests are described for each anatomical area.
In this PPT description of various basic instruments, anterior rhinoscopy, Posterior rhinoscopy, septum examination, nasal valve patency examination, paranasal sinus examination, etc.
Inflammation of the mucosa of sinuses associated with inflammation of the nasal mucosa is called rhinosinusitis (RS).
CLASSIFICATION:
• Acute RS: Symptoms lasting for less than 4 weeks with complete resolution.
• Subacute RS: Duration 4-12 weeks.
• Chronic RS: Duration ~ 12 weeks.
• Recurrent RS: Four or more episodes of RS per year; each lasting for 7-10 days or more with complete resolution in between the episodes.
• Nasal obstruction.
• Nasal discharge/congestion, anterior, or posterior in the form of postnasal drip.
• Facial pain or pressure.
• Alteration in the sense of smell, hyposmia or anosmia.
• Other symptoms include cough, fever, halitosis, fatigue, dental pain, pharyngitis, headache or ear fullness.
In this PPT description of various basic instruments, anterior rhinoscopy, Posterior rhinoscopy, septum examination, nasal valve patency examination, paranasal sinus examination, etc.
Inflammation of the mucosa of sinuses associated with inflammation of the nasal mucosa is called rhinosinusitis (RS).
CLASSIFICATION:
• Acute RS: Symptoms lasting for less than 4 weeks with complete resolution.
• Subacute RS: Duration 4-12 weeks.
• Chronic RS: Duration ~ 12 weeks.
• Recurrent RS: Four or more episodes of RS per year; each lasting for 7-10 days or more with complete resolution in between the episodes.
• Nasal obstruction.
• Nasal discharge/congestion, anterior, or posterior in the form of postnasal drip.
• Facial pain or pressure.
• Alteration in the sense of smell, hyposmia or anosmia.
• Other symptoms include cough, fever, halitosis, fatigue, dental pain, pharyngitis, headache or ear fullness.
Nasal polyps are soft, painless, noncancerous growths on the lining of your nasal passages or sinuses. They hang down like teardrops or grapes. They result from chronic inflammation and are associated with asthma, recurring infection, allergies, drug sensitivity or certain immune disorders.
Adenoids
Definition
The adenoids are enlarged and hypertrophied nasopharyngeal tonsils, sufficient to produce symptoms
It is disease of infancy and childhood.
Adenoids are subjected to physiological enlargement in childhood hence nasopharyngeal tonsils are commonly called Adenoids.
Nasopharyngeal Tonsil
Single pyramidal mass of sub-epithelial lymphoid tissue, present in nasopharynx at the junction of its roof and posterior wall.
The pharyngeal tonsil is composed of vertical ridges of lymphoid tissues separated by deep cleft and covered by Pseudostraitified ciliated columinar epithelium.
The free surface has 6 folds
It has no capsule
These lymphoid tissues consits of T and B lymphocytes.
It forms roof of waledeyer’s ring.
Can't normally see them because they are above and behind the uvula.
Arterial Supply
Ascending branch of facial artery
Ascending pharyngeal branch of external carotid
Pharyngeal branch of third part of maxillary artery.
Ascending cervical branch of inferior thyroid artery of thyrocervial trunk
Development
Adenoids begin forming in 3rd month of fetal development
Glandular primordia on posterior pharynx are infiltrated by lymphocytes.
Covered by pseudostratified ciliated epithelium
Fully formed by 7 month
Growth
They are not visible on X-ray in infants under age of one month.
50% of cases, it is visible at 6 month.
At the age of 2 years undergo hypertrophy and hyperplasia.
Can become nearly the size of a Table Tennis ball
Hypertrophy continues up to puberty (12 years)
Then, undergoes atrophy after puberty
Finally disappears in adults
Why does adenoid physiologically enlarge?
Poorly develop at birth.
Grows rapidly during childhood.
Generalized lymphoid hyperplasia occurs in children
Among the first aggregative lymphoid tissues in respiratory tract.
Physiology
Part of secondary immune system
No afferent lymphatics
Exposed to inspired antigens passed through the epithelial layer
Membrane cells and antigen presenting cells are involved in transport of antigen from the surface to the lymphoid follicle
Antigen is presented to T-helper cells
T-helper cells induce B cells in germinal center to produce antibody
Secretory IgA is primary antibody produced
Involved in local immunity
Etiology
Age : 3 -12 years
Season: winter
Food: Cold, sour, oily food
General lymphoid hyperplasia
Infection in tonsils alone or associated with
Rhinitis, Sinusitis, Tonsillitis
(esp. chronic maxillary sinusitis)
Recurrent attacks of rhinitis, sinusitis or tonsillitis may causes chronic adenoid infection
Allergy of respiratory tract.
Clinical features
Symptoms occur most commonly between ages of
3-7 years.
Depending on size of adenoid mass and space
3 types
Nasal symptoms
Aural symptoms
General symptoms
Nasal symptoms
Bilateral Nasal obstruction
Mouth breathing
interfere
Nasal polyps are soft, painless, noncancerous growths on the lining of your nasal passages or sinuses. They hang down like teardrops or grapes. They result from chronic inflammation and are associated with asthma, recurring infection, allergies, drug sensitivity or certain immune disorders.
Adenoids
Definition
The adenoids are enlarged and hypertrophied nasopharyngeal tonsils, sufficient to produce symptoms
It is disease of infancy and childhood.
Adenoids are subjected to physiological enlargement in childhood hence nasopharyngeal tonsils are commonly called Adenoids.
Nasopharyngeal Tonsil
Single pyramidal mass of sub-epithelial lymphoid tissue, present in nasopharynx at the junction of its roof and posterior wall.
The pharyngeal tonsil is composed of vertical ridges of lymphoid tissues separated by deep cleft and covered by Pseudostraitified ciliated columinar epithelium.
The free surface has 6 folds
It has no capsule
These lymphoid tissues consits of T and B lymphocytes.
It forms roof of waledeyer’s ring.
Can't normally see them because they are above and behind the uvula.
Arterial Supply
Ascending branch of facial artery
Ascending pharyngeal branch of external carotid
Pharyngeal branch of third part of maxillary artery.
Ascending cervical branch of inferior thyroid artery of thyrocervial trunk
Development
Adenoids begin forming in 3rd month of fetal development
Glandular primordia on posterior pharynx are infiltrated by lymphocytes.
Covered by pseudostratified ciliated epithelium
Fully formed by 7 month
Growth
They are not visible on X-ray in infants under age of one month.
50% of cases, it is visible at 6 month.
At the age of 2 years undergo hypertrophy and hyperplasia.
Can become nearly the size of a Table Tennis ball
Hypertrophy continues up to puberty (12 years)
Then, undergoes atrophy after puberty
Finally disappears in adults
Why does adenoid physiologically enlarge?
Poorly develop at birth.
Grows rapidly during childhood.
Generalized lymphoid hyperplasia occurs in children
Among the first aggregative lymphoid tissues in respiratory tract.
Physiology
Part of secondary immune system
No afferent lymphatics
Exposed to inspired antigens passed through the epithelial layer
Membrane cells and antigen presenting cells are involved in transport of antigen from the surface to the lymphoid follicle
Antigen is presented to T-helper cells
T-helper cells induce B cells in germinal center to produce antibody
Secretory IgA is primary antibody produced
Involved in local immunity
Etiology
Age : 3 -12 years
Season: winter
Food: Cold, sour, oily food
General lymphoid hyperplasia
Infection in tonsils alone or associated with
Rhinitis, Sinusitis, Tonsillitis
(esp. chronic maxillary sinusitis)
Recurrent attacks of rhinitis, sinusitis or tonsillitis may causes chronic adenoid infection
Allergy of respiratory tract.
Clinical features
Symptoms occur most commonly between ages of
3-7 years.
Depending on size of adenoid mass and space
3 types
Nasal symptoms
Aural symptoms
General symptoms
Nasal symptoms
Bilateral Nasal obstruction
Mouth breathing
interfere
This is a presentation by Dada Robert in a Your Skill Boost masterclass organised by the Excellence Foundation for South Sudan (EFSS) on Saturday, the 25th and Sunday, the 26th of May 2024.
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2024.06.01 Introducing a competency framework for languag learning materials ...Sandy Millin
http://sandymillin.wordpress.com/iateflwebinar2024
Published classroom materials form the basis of syllabuses, drive teacher professional development, and have a potentially huge influence on learners, teachers and education systems. All teachers also create their own materials, whether a few sentences on a blackboard, a highly-structured fully-realised online course, or anything in between. Despite this, the knowledge and skills needed to create effective language learning materials are rarely part of teacher training, and are mostly learnt by trial and error.
Knowledge and skills frameworks, generally called competency frameworks, for ELT teachers, trainers and managers have existed for a few years now. However, until I created one for my MA dissertation, there wasn’t one drawing together what we need to know and do to be able to effectively produce language learning materials.
This webinar will introduce you to my framework, highlighting the key competencies I identified from my research. It will also show how anybody involved in language teaching (any language, not just English!), teacher training, managing schools or developing language learning materials can benefit from using the framework.
3. INTRODUCTION
Full nose examinations assess the function,
airway resistance and occasionally sense of smell.
It includes looking into the mouth and pharynx.
Common symptoms of nasal disease include:
Airway obstruction.
Rhinorrhoea (runny nose).
Sneezing.
Loss of smell (anosmia).
Facial pain caused by sinusitis.
Snoring (associated with nasal obstruction).
4. Plan of Examination
Introduce yourself
Position patient
Examination of external nose
Inspect the nasal tip, vestibule, and nasal airway.
Anterior rhinoscopy
Posterior rhinoscopy
Post nasal examination
5. Examination of External Nose
INSPECTION :
The external is observed from a distance from the
front as well as side for any signs such as:
Nasal bridge deformity- Saddle Nose/ Hump deformity
Swelling
Ulcers
Sinuses
Growths on skin
Scars
Broadening of nose
Inflammation/ cellulites
6. Contd..
External deformities of the nose like external deviation of
nose, nasal hump, depressed nasal bridge, nasal injury,
congenital anomaly, any inflammation or swelling, benign
conditions like rhinophyma are observed.
Depressed Nasal Bridge : occurs in leprosy, syphilis or
tuberculosis of nose and also in cretinism, thalassemia major.
Nasal Injury : common after road vehicular accident. Deviated
Nose : common in boxers.
Congenital Swelling : glioma is present since birth.
Benign Condition : Rhinophyma (Potato nose) is due to
hypertrophy of sebaceous glands.
Broadening of Nose : seen in antrochoanal polyp.
Frog Face Deformity : seen in juvenile nasopharyngeal
angiofibroma.
Crepitus : felt in fracture nasal bone.
7. Contd..
PALPATION : Following thorough inspection, we
proceed to palpation, where our inspectory
conclusions are verified and additional assessment is
performed.
Crepitation
Tenderness
8. Cold Spatula Test
Tongue depressor is kept about a cm. below the nostril and
the amount of fogging on the spatula is observed. If fogging is
less on one side, it indicates obstruction of that side of nasal
cavity. In summers fogging is not appreciated so this test
should be carried out in an air-conditioned room or after
dipping the spatula in cold water. The amount of fogging can
also be measured on the graphic chromium plate.
9. Anterior Rhinoscopy
The anterior rhinoscopy is carried out by Thudicum’s nasal speculum.
Vestibule is the area, which is covered by the blades of Thudicum’s nasal
speculum. Any vestibular lesion like furuncle is likely to be missed , so first
raise the tip of nose by the thumb and examine the vestibule before using
nasal speculum.
The structures visualized and examined on anterior rhinoscopy :
Nasal septum
Nasal cavity
Turbinates
Middle meatus
Colour of mucosa
Discharge
Crusts
Polyp
Mass
Bleeding
Pus
Maggots (if any)
11. Nasal Cavity
Nasal Cavity :
Roomy nasal cavity is seen in atrophic rhinitis.
Greenish crusts are seen in atrophic rhinitis.
A. Turbinates :
There are three turbinates. Superior turbinate, middle turbinate and
inferior turbinate. The inferior turbinate may be normal or engorged
on one side or both sides. If DNS is on left side, there is a
compensatory hypertrophy of inferior turbinate on the right side. The
middle turbinate has normal medial convexity towards the septum. If
it has convexity towards the lateral wall and concavity towards the
septum, it is called paradoxically bent middle turbinate. Superior
turbinate is not seen by Thudicum’s nasal speculum, it can be seen
only by the nasal endoscope. Inferior turbinate and septum are the
areas of the nose, which have arterio-venous anastomosis.
12. Contd..
B. Middle Meatus :
Inspection of middle meatus for any pus is done. Suppuration of the
ethmoid or maxillary sinus is indicated by a pus in the middle
meatus. When the osteomeatal complex is congested, the FESS is
needed (functional endoscopic sinus surgery). Prior rhinoscopy may
reveal bulla ethmoidalis.
C. Colour of Mucosa :
Normal nasal mucosa is pinkish red.
Red congested mucosa is seen in acute rhinitis.
Pale oedematous mucosa is seen in allergy.
Pinkish white mucosa is seen in anaemia and tuberculosis.
13. Contd..
D. Discharge :
Watery discharge is seen in allergic rhinitis.
Clear fluid discharge is found in CSF rhinorrhoea.
Mucoid discharge is seen in antrochoanal polyp.
Mucopurulent or purulent discharge is seen in chronic maxillary
sinusitis.
Blood stained discharge is seen in inverted papilloma or in
underlying malignancy.
Foul smelling unilateral nasal discharge in children is almost
diagnostic of foreign body in the nose unless until proved
otherwise.
14. Contd..
E. Crust :
Foul smelling crusts are seen in atrophic rhinitis. In summer season,
crusting occurs in the nasal cavity and the crusts are felt by the patient
due to the hot weather. Instillation of normal saline drops brings relief in
this situation.
F. Polyps :
Polyps are oedematous mucus membrane (They are not new growth).
The nasal polyps are either ethmoidal polyps or antrochoanal polyps.
15. Contd..
G. Mass :
It could be benign or malignant.
Benign mass can be juvenile nasopharyngeal angiofibroma (JNAF).
Malignant mass could be arising from nose, sinuses or from
nasopharynx.
H. Bleeding :
Observe any frank blood or clotted blood in the nasal cavity. Quite often
ulceration with clotted blood is seen in the Little’s area in hypertensive
patients.
I. Pus:
When pus is seen to flow from the middle meatus, it suggests the
suppuration of anterior group of sinuses. When pus is seen in the
olfactory cleft it signifies suppuration of posterior group of sinuses.
16. Posterior Rhinoscopy
Method :
Posterior rhinoscopy mirror is taken, the mirror side is then warmed.
(Fogging can also be prevented by dipping the mirror in savlon solution
or rubbing a dry soap on mirror or if nothing is available, rubbing the
mirror against the mucosal surface of cheek.) The patient's temperature
should not be too high and it should be checked by touching the
metallic side of the mirror on the patient's skin. The patient should be
asked to breathe through their nose, as this brings the soft palate
forward, the tongue is pressed with the tongue depressor and the
posterior rhinoscopy mirror is introduced gently without touching the
posterior pharyngeal wall otherwise it will cause gag reflex.All
structures are examined. If the patient has a strong gag reflex or is very
unco-operative, 4% or 10% xylocaine is sprayed on the throat as well as
posterior pharyngeal wall.
17. Contd..
Structures seen in Posterior Rhinoscopy:
Anteriorly
1. Posterior end of Nasal septum.
2. Posterior end of middle and inferior turbinate
3. Posterior end of superior turbinate
4. Posterior part of superior & middle meatus
5. Nasal Surface of the soft palate & the uvula on tilting the mirror
further anteriorly.
18. Contd..
Laterally:
1. Eustachian tube opening on either side with Tubal
Elevations seen behind the posterior end of inferior
turbinate.
2. Fossa of Rosenmuller behind the eustachian tube
orifice.
Superiorly:
1. Roof of Nasopharynx.
2. Superior part of posterior pharyngeal wall.
21. Contd..
The anterior group of sinuses are
frontal sinus, anterior ethmoidal group of sinuses and maxillary
sinus. They open in middle meatus anterior to posterior in that
order.
The posterior group of sinuses are
posterior ethmoidal group of cells and sphenoid sinus. Posterior
ethmoidal cells open into the superior meatus while sphenoid
sinus opens into the sphenoethmoidal recess.
22. Contd..
Inspection:
In acute sinusitis there may be swelling and redness of the skin
overlying the maxillary sinuses.
Palpation:
The tenderness of the maxillary sinuses is elicited by applying
the firm pressure at the canine fossa.
The tenderness of ethmoid sinuses are elicited by applying
pressure at the lateral side of bridge of nose.
The tenderness of frontal sinuses is elicited by applying
pressure at the floor of frontal sinus, just above the orbit. One
must take precaution not to press the supra orbital nerve
otherwise it may cause false tenderness.
24. EXAMINATION OF ORAL CAVITY
A. INSPECTION:
1. Tongue :
Volume: Massive tongue (macroglossia) is due to lymphangioma, :
neurofibromatosis and cretinism.
Colour: Pallor is seen in anaemia. White coloured tongue is due to
leucoplakia. Blue coloured tongue is due to venous haemangioma. Red
glazed tongue when leucoplakia plaques are shed. Black or hairy tongue
is due to Aspergillus niger. Geographical tongue has convoluted pattern
and is of no significance.
Cracks or Fissure: Congenital fissuring of tongue or scrotal tongue
usually have transverse fissure and are of no pathological significance.
Syphilis cracks are usually longitudinal.
Fur: Furring of tongue is of little value. It is found in heavy smokers
and mouth breathers. A black hairy tongue is due to fungus infection
and in immunocompromised patients.
25. Contd..
Papillae: Bald Tongue - There is generalized atrophy of papillae and found
in pernicious anaemia. Geographical Tongue - is harmless anomaly
characterised by localized irregular red area of desquamated epithelium and
filiform papillae. It is surrounded by white yellow border giving the
appearance of map.
Tounge tie: It is congenitally short frenulum linguae in children
(ankyloglossia).
Swelling: Ifany swelling is present examine its site, size, shape, margin,
colour like any other swelling mentioned earlier. Any cyst may also be
present on tongue.
Ulcer: Aphthous or dental ulcers on tongue. > Carcinomatous ulcer is
usually on the margin of tongue. > Gummatous ulcer is on the dorsum. | >
Tuberculous ulcer is on the tip, side or dorsum. > Dental ulcer is on the side
of tongue where they come in contact with sharp teeth.
Mobility: Ask patient to protrude his tongue and move from side to side.
Restricted mobility is seen in advanced carcinoma. In children short
frenulum linguae may cause tongue-tie.
26. Contd..
2. Palate:
Examine for perforation, ulceration or swelling is done. If there is
any congenital cleft palate, we look for the extent whether involving
uvula, soft palate or hard palate.
3. Floor of Mouth:
4. Buccal Mucosa:
Examine for any aphthous ulcer, leucoplakia, melanosis and mucosal
hyperpigmentation, mucous cyst, papilloma, carcinoma or cheek
nibbling.
5. Gum
6. Teeth
27. Contd..
B. PALPATION:
1. Tongue:
Ideally the tongue should be kept in the oral cavity to keep the tongue
muscle relaxed. To look for induration, ulcers, swelling.
2. Palate:
Any ulcer or cyst (nasoalveolar or nasopalatine cyst) is examined
in theusual way. Alveolar abscess causes tender fluctuating
swelling close to the alveolar process.
3. Floor of Mouth:
It should be palpated bimanually. Translucency is tested if there
is anycystic swelling. Ranula is translucent but sublingual
dermoid cyst is nottranslucent. The submandibular duct is
palpated for any stone.
28. Contd..
4. Buccal Mucosa:
The mucus membrane and the cheek should be carefully palpated
to know involvement of skin of cheek by malignant growth.
30. Examination of Salivary Glands
A. INSPECTION:
Swelling:
Parotid swelling appears below, in front, and behind the lobule of ear
causing usually lifting of the lobule. It also obliterates the normal fissure
behind the ramus of mandible.
Submandibular gland swelling is present in the submandibular triangle.
Duct:
The Stensen’s duct (parotid duct) is seen on the buccal surface, opposite
to the upper 2nd molar tooth. In suppurative parotitis, pus may come out
of the duct on pressing the gland while in malignant growth, blood may
come out. We ask the patient to touch the palate by the tip of tongue, the
opening of submandibular duct (Wharton’s duct) on either side of
frenulum linguae or sublingual duct (Bartholian's duct) is seen. It may be
inflamed or swollen.
31. Contd..
B. PALPATION:
The parotid duct may be palpated on the masseter muscle by rolling
the finger across it while patient clinches the teeth by making the
muscle taut. The terminal part of duct is palpated bidigitally between
index finger in the mouth and thumb over the cheek.
The submandibular gland and duct is palpated bidigitally. A finger is
inserted inside the mouth along the groove between the alveolus and
the tongue and pressed on the floor of mouth. The finger of the other
hand is placed under the jaw. The gland and duct are palpated from
behind. This bidigital palpation helps to differentiate it from the
enlarged submandibular lymph node. The finger inside the mouth
can feel the deeper part of salivary gland but not the lymph node (as
salivary gland is situated above the mylohyoid muscle while lymph
node is situated below the muscle.)
32. Contd..
Saliva Flow Test : Test the flow of saliva by asking the
patient to suck lemon. In absence of any stone or
obstruction in the duct, saliva flows freely from the duct. If
duct is obstructed by the stone, the salivary outflow is
markedly obstructed and there is obvious swelling of the
gland.
33. EXAMINATION OF TONSIL AND PHARYNX
TONSILS
We ask the patient to open the mouth and Lack’s tongue
depressor is introduced to press the tongue. The tongue
depressor should never press the posterior part of tongue as
this causes gag reflex (due to the glossopharyngeal nerve).
Examination of the tonsil for its size, crypts, anterior pillar
and posterior pillar is done.
34. Contd..
Tonsillitis can be acute or chronic:
1. Acute Tonsillitis :
a. Acute Follicular Type: There is acute inflammation of crypts
and exudation from the crypts marks the reddened surface
with white or yellow spots.
b. Acute Parenchymatous type: There is inflammation of whole
tonsil.
c. Acute Membranous type: Another variant of follicular
tonsillitis in which exudation from the crypts may coalesce to
form the confluent membrane over the tonsil. It should be
differentiated from diphtheric tonsillitis, in which membrane
bleeds on removal. (while membranous tonsillitis does not
bleed.)
35. Contd..
2. Chronic Tonsillitis : It is the complication of acute
tonsillitis. It can be chronic follicular or chronic
parenchymatous tonsillitis.
Cardinal Signs of chronic tonsillitis :–
1. Flushing of anterior pillar
2. On pressing the anterior pillar, cheesy material comes out of
tonsil
3. Enlarged tender jugulodigastric lymph node, when there is
no other reason for it.
Out of these 3 signs, if 2 signs are present it is suggestive of
chronic tonsillitis.
36. Contd..
Chronic Fibroid Tonsillitis :
It is the condition where tonsils are buried between the
pillars. They are innocent looking, though they are not as
repeated inflammation causes more fibrosis and reduction in
size. It tends to bleed more during tonsillectomy.
37. Posterior Pharyngeal wall
Examination of any bulging at posterior pharyngeal wall.
Bulging only on one side of midline especially in children is
suggestive of acute retropharyngeal abscess. There is throaty cry
resembling quack of duck. The abscess is drained through oral cavity
as it is in the true retropharyngeal space i.e. space between the
prevertebral fascia and buccopharyngeal fascia. This space is also
known as space of Gilette.
Smooth bulging of posterior pharyngeal wall in the midline
involving both sides of midline is seen in chronic pharyngeal abscess
which is due to caries of the cervical spine. This abscess is behind or
posterior to the prevertebral fascia or space of Gilette. It is drained
from the neck at the anterior border of sternomastoid.
39. Contd..
Postnasal drip in children is due to adenoid infection while
in adults it is suggestive of sinusitis. The red granulations at
the posterior pharyngeal wall commonly called as granular
pharyngitis is usually of allergic type and is common in
those who work in dry dusty atmosphere or in chronic
smokers.
41. Examination of Larynx
EXTERNAL EXAMINATION:
We observe the Adam’s apple. It is about 90° in men and about
120° in women. An advanced laryngeal carcinoma may distort
the shape of Adam’s apple.
Laryngeal Crepitus: We move the larynx from side to side. A
crepitus sound is felt. It is a normal sound produced by the
movement of laryngeal cartilage against the cervical spine but
absence of this sound (Boaca’s sign) may be due to the
possibility of growth between the laryngeal framework and the
cervical spine (postcricoid carcinoma).
42. Contd..
INTERNAL EXAMINATION :
Indirect Laryngoscopy:
We warm the mirror side of indirect laryngoscopy mirror,
tested on the examiner’s hand for heat and hold it like a pen
with right hand. Alternately mirror can be dipped in the
solution of savlon or rubbed against the buccal mucosa to
prevent the fogging.
We ask the patient to open the mouth, protrude his tongue.
The anterior part of tongue is grasped by the left hand. We
ask patient to breathe gently through the mouth and we place
the warm indirect laryngoscopy mirror inside the throat
gently against the anterior surface of uvula.
43. Contd..
The structures seen on indirect laryngoscopy are :
a) Anterior part of larynx-epiglottis and anterior commissure
(seen towards the top of the mirror).
b) Posterior part of the larynx - the arytenoids and the posterior
commissure (seen at the lower portion of the mirror).
44. Contd..
The patient’s right vocal cord is seen on the left side of the
mirror as the examiner looks at it. We first examine the
vallecula and the tip of epiglottis and then the ary-epiglottic
fold and the pyriform fossa on each side. Then postcricoid
region, arytenoids, false cords (ventricular folds) and vocal
cords (vocal folds) are inspected. Sometimes it is possible
to see upper few cms. of trachea. Finally movement of vocal
cords are studied by asking patient to phonate ‘ee’ and
breathe gently alternately several times. Also examination
of the colour of mucosa all around is seen.
45. Contd..
The following things are examined :
a. Epiglottis:
The normal colour of epiglottis is pinkish.
Bright red, swollen epiglottis is seen in acute laryngitis. X-ray
neck lateral view shows thumb sign.
Pale, swollen epiglottis is seen in allergic laryngitis.
46. Vocal Cords Contd..
b. Vocal Cords :
Colour : The normal vocal cords are pearly white in colour. In
acute laryngitis they are congested.
Oedema : Oedema of vocal cords is seen in Reinke’s oedema (it
is oedema of the Reinke’s space of the vocal cords).
Edge: Vocal nodules (Singer’s nodules) are seen at the junction
of anterior 1/3rd and posterior 2/3rd of the vocal cords.
The malignant growth may be seen anywhere in the vocal
cords and requires its removal and histopathological
examination.
Any solitary nodule can be solitary papilloma.
Surface : Observe for any cyst, ulcer, leucoplakic patch or
granulations.
47. Vocal Cords Contd..
Movement :
The movements of vocal cords are examined at :
gentle breathing during phonation
forced inspiration
on coughing
at rest
The movement of vocal cord is restricted in abductor or adductor
paresis, in infiltration by growth or in arthritis of crico-arytenoid
joint.
48. Vocal Cords Contd..
Position of Vocal Cords :
There are different positions of vocal cords:
Median position
Paramedian position
Cadaveric position
Gentle abduction
Full abduction
49. c. Arytenoids
d. Ary-epiglottic folds
e. Inter-arytenoids area: Congestion of this area occurs in tuberculosis of
the larynx.
f. Glottic chink: Glottic chink is reduced in vocal cord paralysis or any
malignant growth.
g. Postcricoid area: Any growth of this area along with absent laryngeal
crepitus is highly suspicious of malignancy of this area.
h. Pyriform fossa: Fullness of this area is highly suspicious of malignancy.
Pooling of saliva in both the pyriform fossa (Jackson’s sign) is
suspicious of post cricoid malignancy.
i. Upper few cm. of trachea
j. Vallecula, median glosso epiglottic fold and base of tongue is also examined
after completing examination of larynx.