The document discusses the evaluation and management of facial nerve palsy. It begins with an introduction and overview of causes, evaluation of nerve function, and goals of management. It then discusses factors governing the timing and treatment of facial nerve palsy, assessment and planning, and specific management techniques. Surgical options including nerve decompression, repair, grafting and transfers are outlined. Non-surgical treatments like physical therapy are also summarized.
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Tom Selleck Health: A Comprehensive Look at the Iconic Actor’s Wellness Journeygreendigital
Tom Selleck, an enduring figure in Hollywood. has captivated audiences for decades with his rugged charm, iconic moustache. and memorable roles in television and film. From his breakout role as Thomas Magnum in Magnum P.I. to his current portrayal of Frank Reagan in Blue Bloods. Selleck's career has spanned over 50 years. But beyond his professional achievements. fans have often been curious about Tom Selleck Health. especially as he has aged in the public eye.
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Introduction
Many have been interested in Tom Selleck health. not only because of his enduring presence on screen but also because of the challenges. and lifestyle choices he has faced and made over the years. This article delves into the various aspects of Tom Selleck health. exploring his fitness regimen, diet, mental health. and the challenges he has encountered as he ages. We'll look at how he maintains his well-being. the health issues he has faced, and his approach to ageing .
Early Life and Career
Childhood and Athletic Beginnings
Tom Selleck was born on January 29, 1945, in Detroit, Michigan, and grew up in Sherman Oaks, California. From an early age, he was involved in sports, particularly basketball. which played a significant role in his physical development. His athletic pursuits continued into college. where he attended the University of Southern California (USC) on a basketball scholarship. This early involvement in sports laid a strong foundation for his physical health and disciplined lifestyle.
Transition to Acting
Selleck's transition from an athlete to an actor came with its physical demands. His first significant role in "Magnum P.I." required him to perform various stunts and maintain a fit appearance. This role, which he played from 1980 to 1988. necessitated a rigorous fitness routine to meet the show's demands. setting the stage for his long-term commitment to health and wellness.
Fitness Regimen
Workout Routine
Tom Selleck health and fitness regimen has evolved. adapting to his changing roles and age. During his "Magnum, P.I." days. Selleck's workouts were intense and focused on building and maintaining muscle mass. His routine included weightlifting, cardiovascular exercises. and specific training for the stunts he performed on the show.
Selleck adjusted his fitness routine as he aged to suit his body's needs. Today, his workouts focus on maintaining flexibility, strength, and cardiovascular health. He incorporates low-impact exercises such as swimming, walking, and light weightlifting. This balanced approach helps him stay fit without putting undue strain on his joints and muscles.
Importance of Flexibility and Mobility
In recent years, Selleck has emphasized the importance of flexibility and mobility in his fitness regimen. Understanding the natural decline in muscle mass and joint flexibility with age. he includes stretching and yoga in his routine. These practices help prevent injuries, improve posture, and maintain mobilit
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Ve...kevinkariuki227
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Verified Chapters 1 - 19, Complete Newest Version.pdf
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Lung Cancer: Artificial Intelligence, Synergetics, Complex System Analysis, S...Oleg Kshivets
RESULTS: Overall life span (LS) was 2252.1±1742.5 days and cumulative 5-year survival (5YS) reached 73.2%, 10 years – 64.8%, 20 years – 42.5%. 513 LCP lived more than 5 years (LS=3124.6±1525.6 days), 148 LCP – more than 10 years (LS=5054.4±1504.1 days).199 LCP died because of LC (LS=562.7±374.5 days). 5YS of LCP after bi/lobectomies was significantly superior in comparison with LCP after pneumonectomies (78.1% vs.63.7%, P=0.00001 by log-rank test). AT significantly improved 5YS (66.3% vs. 34.8%) (P=0.00000 by log-rank test) only for LCP with N1-2. Cox modeling displayed that 5YS of LCP significantly depended on: phase transition (PT) early-invasive LC in terms of synergetics, PT N0—N12, cell ratio factors (ratio between cancer cells- CC and blood cells subpopulations), G1-3, histology, glucose, AT, blood cell circuit, prothrombin index, heparin tolerance, recalcification time (P=0.000-0.038). Neural networks, genetic algorithm selection and bootstrap simulation revealed relationships between 5YS and PT early-invasive LC (rank=1), PT N0—N12 (rank=2), thrombocytes/CC (3), erythrocytes/CC (4), eosinophils/CC (5), healthy cells/CC (6), lymphocytes/CC (7), segmented neutrophils/CC (8), stick neutrophils/CC (9), monocytes/CC (10); leucocytes/CC (11). Correct prediction of 5YS was 100% by neural networks computing (area under ROC curve=1.0; error=0.0).
CONCLUSIONS: 5YS of LCP after radical procedures significantly depended on: 1) PT early-invasive cancer; 2) PT N0--N12; 3) cell ratio factors; 4) blood cell circuit; 5) biochemical factors; 6) hemostasis system; 7) AT; 8) LC characteristics; 9) LC cell dynamics; 10) surgery type: lobectomy/pneumonectomy; 11) anthropometric data. Optimal diagnosis and treatment strategies for LC are: 1) screening and early detection of LC; 2) availability of experienced thoracic surgeons because of complexity of radical procedures; 3) aggressive en block surgery and adequate lymph node dissection for completeness; 4) precise prediction; 5) adjuvant chemoimmunoradiotherapy for LCP with unfavorable prognosis.
Knee anatomy and clinical tests 2024.pdfvimalpl1234
This includes all relevant anatomy and clinical tests compiled from standard textbooks, Campbell,netter etc..It is comprehensive and best suited for orthopaedicians and orthopaedic residents.
Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
- Video recording of this lecture in English language: https://youtu.be/lK81BzxMqdo
- Video recording of this lecture in Arabic language: https://youtu.be/Ve4P0COk9OI
- Link to download the book free: https://nephrotube.blogspot.com/p/nephrotube-nephrology-books.html
- Link to NephroTube website: www.NephroTube.com
- Link to NephroTube social media accounts: https://nephrotube.blogspot.com/p/join-nephrotube-on-social-media.html
2. “The human face is the organic seat of
beauty. It is the register of value in
development, a record of Experience,
whose legitimate office is to perfect the life,
a legible language to those who will study it,
of the majestic mistress, the soul."
- Farnham, Eliza
QUOTE
3. Introduction
Causes
Evaluation of nerve function.
Goals of management of FN palsy
Factors governing T/t of FN palsy
Assessment & planning
Management of facial nerve palsy
FN disorders in newborn &children
4. Facial function plays an integral part in
our everyday lives
- Smile; nonverbal communication, etc.
Facial paralysis is devastating on many
leveL
Functional
Cosmetic
Fortunately, a plethora of techniques are
available to treat the paralyzed face.
5.
6.
7.
8. In middle ear surgery, the most comman site of inury is the
tympanic segment due to high incidence of fallopian canal
dehiscence in that region. During mastoid surgery, the facial
nerve is most commanly inured at the secong genu
Skull base neoplasms & metastasis from breast and lung cancer
are comman neoplasms causing facial paralysis in adults.
Leukemia, lymphoma are the most comman etiologies in
children.
A facial palsy progressive beyond 3 weeks since onset & with no
returns of function by 6 months is considered to be caused by
tumor until proven by otherwise
If greater than 90% neural degeneration occurs with in 2 weeks,
the mastoid & tympanic segment are decompressed.
9. HISTORY is of vital importance to establish
the onset characteristics, duration and
degree of recovery.
Previous trauma, surgery or infection may
help in arriving at a diagnosis
Examination of the face at rest and
movement.
Radiolologic evaluations
Topodiagnostic & Nerve excitability tests.
10. Normal appearnce at rest
Symmetry at involuntry motion.
Restoration of oral, ocular & nasal
sphincter
No loss of other significant functions.
11. Age
Medical history.
Residual hearing.
Prior h/o ear discharge.
Segment of nerve injured.
Patient’s expectations.
Risk tolerance.
12. Cause of facial paralysis
Functional deficit/extent of paralysis
Time course/duration of paralysis
Likelihood of recovery
Other cranial nerve deficits
Patient’s life expectancy
Patient’s needs/expectations
13.
14. Psychological Trauma :- The most
significant complication is the social
isolation & these patients often succumb
to.
Depression :- Patient often become by
the facial deformity.
The patient must be
encouraged to to adopt
positive outlook.
15. Group therapy has been effective in
helping patients to deal with facial
deformity.
Resultls are better when group members
are selected of the same age group &
same age.
Patient should be learned to adapt
permenant deformity in a positive way.
16. MEDICAL TREATMENT : -
a) Physical therapy
b) Pharmacological therapy
c) Psychophysical therapy
Surgical treatment : -
a) Nerve decompression - Internally or
externally.
b) Nerve anastomosis
c) Nerve grafting
Physiotherapy
17. It includes application of heat, massage
& exercises performed twice a day.
Patient is adviced to follow these steps –
a) Wet cotton towel with hot water,
wring it out, keep the hot towel on the
face until the towel cools.
b) Massage facial cream on skin
around the eyes & mouth and mid face for
few minutes.
c) Stand infront of mirror Watch face
while doing facing exercises Intact nerve
fibres activated & muscle tone maintained
18. In case of the Bell’s palsy :-
a) Oral antivirals - Acyclovir is DOC.
b) Corticosteroids
c) Eye protection
d) Follow progression with serial exams
e) Physiotherapy.
Prednisolon is steroidal drug of choice in
both Bell’s palsy & Ramsay Hunt
syndrom.
19. If the patient is seen within 2 to 3 weeks
of onset of symptoms-tab. Prednisolone
in doses of 1mg/kg/d for 10 to 14 days
has been recommended with a gradual
tapering.
Vitamins B1, B6, B12 may be
administered which helps in nerve
conduction & regeneration.
If patient is seen after 3-4 weeks, then
steroid therapy is of no use.
Non-narcotic analgesic is used to control
pain in Bell’s Palsy & mainly Ramsay Hunt
syndrom.
21. Nerve decompression - Internally or
externally
Nerve anastomosis
Nerve grafting
Due to “skip” regions & diffuse neuritis of the facial
nerve, surgical decompression is not recommended in
Ramsay Hunt syndrome.
22. A. Acute (< 3 wks)
1) Nerve
exploration/decompression
2) Nerve repair
a) Primary anastomosis
b) Cable grafting
i) Great auricular nerve
ii) Sural nerve
B. Intermediate (3 wks- 2
yrs)
1) Nerve transfer
a. Hypoglossal-facial
b. Spinal accessory-facial
c. Masseteric-facial
2) Cross face nerve grafting
using sural nerve
C. Chronic (>2 yrs)
1) Muscle transfers
a. Temporalis
b. Masseter
c. Digastrics
2) Free muscle flaps/
microneurovascular transfer
a. Gracilis
b. Latissimus dorsi
c. Serratus anterior
d. Pectoralis minor
D. Static
procedures/ancillary
procedures (can be
performed at any time period
listed above)
1) Gold weight/spring
implants
2) Slings
3) Lid procedures
23. Timing : within 72 hrs
Indication :
a) < 10% or less muscle function on ENoG,
b) with absent voluntary muscle action
potential on EMG
Method :
a) Done by removing the facial fallopian
canal all around with widening of the
canal with diamond burrs.
b) Perineural and epineural sheaths are
split open (to drain perineural or
intraneural edema / haematoma)
25. Indication :-
Tumors limited to the tympanic and/or the
mastoid segment of the facial nerve regrdless
of preoperative hearing loss
Longitudinal fractures of temporal bone (only
mastoid segmental involvement)
AOM, COM involving only tympanic segment
and genu
Isolated mastoid fracture
Infections involving the mastoid segment
Limitations :-
Limited access to geniculate ganglion
No access to labrynthine segment
26. Procedure:-
a) Mastoidectomy air cells removed from
antrum downward to mastoid tip & ridge
of the diagastric groove is defined cells
also removed from the antrum forward to
the root zygoma, until the upper edge of
the incus & prominance of bony horizantal
canal identified.
b) Landmark for the vertical mastoid portion
of FN is post tip of incus above & ant end
of diagastric groove below drilled upto
stylomastoid foramen bone between
foramen & horizantal SSC thinned & FN
nerve is approached as pink streak.
27. DECOMPRESSION:-
a) When horizantal segment of FN involved,
decompression done via triangle
bounded by FN med., chorda tympani
nerve & tympanic annulus laterally &
short process of incus sup.
b) When patient’s hearing is normal & entire
horizantal seg. of FN must be
decompressed, incus must also be
disarticulated.
c) Disposable Beaver knife is used for the
decompression of the FN by slitting the
nerve sheath.
28.
29. Exposure from IAC to Tympanic segment
(for intracanalicular and labrynthine
segments)
INDICATIONS :-
a) Bells palsy,
b) Longitudinal temporal bone fractures
Advantages :-
a) No hearing impairment, even
geniculate ganglion and tympanic
segment can be decompressed, when
b) Combined with retrolabyrinthine,
transmastoid entire facial nerve can be
seen.
30. PROCEDURE :
6x8cm trap door incision above ear (with postaural incision)
4x4 cm temporalis fascia graft harvested
Anterior based temporalis musculo perisosteal flap elevated
A bone flap centered over zygoma elevated, taking care
middle meningeal artery on inner table
Dura elevated from posterior to anterior till petrous ridge,
arcuate eminence, meatal plane, and GSPN Anteriorly.
Blue lining of superior semicircular canal seen
Anterior to it IAC opened with 7th nerve anterosuperiorly (BILLS
BAR FORMS LATERAL BOUNDARY OF MEATAL FORAMEN)
Labrynthine segment followed laterally till geniculate ganglion.
Tegmen tympani removed
Tympanic segment blue lined and final layer of bone removed
with elevator and decompressed
Bone flap replaced, with temporalis fascia over it to seal the
defect.
31. Complications :-
CSF leak, CHL/SNHL, meningitis,
Bleeding from AICA, brainstem and
cerebellar infarction
Injury to AICA
32. For exposure from brainstem to IAC
ADVANTAGES :-
a) Access without inner ear sacrifice
b) Minimal cerebellar compression as compared
to suboccipital approach.
Disadvantages :-
a) 8th nerve hampers 7th visualisation and can
lead to hearing loss
b) Reduced intracranial exposure so intracranial
vascular complications very diff to manage
Complications :-
CSF leak, Hearing impairment, cerebellar
compression, vascular intracranial
complications
33. Indications :
7th and 8th nerve function already lost
Advantages :
a) Entire nerve is exposed using single
approach
b) If interposition graft is required, enough
working space is available even at the
level of brainstem
Limitations :
Hearing and balance function loss,
CSF leak, Infections
34. Facial nerve repair is the most effective
procedure to restore facial function in
patients who have suffered nerve
damage from an accident or during
surgery.
It involves
microscopic repair
of a nerve that has
been cut
35. End-to-end
anastomosis preferred
Adv - No tension
Extratemporal repair
performed < 72 hrs of
injury
Most common method
a) Group fascicular
repair
b) Epineural repair
Group fascicular
repair
36. Severed ends of nerve
exposed
Devitalized tissue/debris
removed with fine
scalpel
Small bites of epineurium
Epineural sheath
approximated with 9-0
nonabsorbable suture
Epineural repair
recommended for injury
proximal to pes anserinus
and intratemporal EPINEURAL REPAIR TECHNIQUE
37. Cable grafts
Used when defect > 17mm nerve
cannot be re-approximated without
tension
Most common nerves used are:-
a) Greater Auricular Nerve
b) Sural Nerve
c)Medial & lateral antibrachial
cutaneous nerves
c) Sensory nerves from superficial
cervical plexus
Motor nerve grafts are better than sensory nerve
38. Harvesting :-
Located on lateral surface
of SCM at the midpoint of a
line drawn between
mastoid tip and mandibular
angle.
May extend postauricular
incision or use separate
neck incision.
Ideally used for graft upto
10 cm in length.
39. Advantages:
a) Proximity to facial nerve
b) Cross-sectional area
c) Limited morbidity
Limitations:
a) Reconstruction of long defects
b) Ideal for defects < 6cm in length
Recently, the idea of nerve repair using
autogenous axially aligned freez-
thrawed skeletal muscle has been
proposed.
40. Anatomy:-
Formed by union of medial
sural cutaneous nerve and
lateral sural cutaneous
branch of peroneal nerve
Advantages :-
Diameter equal to FN dia.
Length : >10cm
Accessibility
Low morbidity associated
with sacrifice
Disadvantages:
Variable caliber
Often too large
Difficult to make graft
approximation
Unsightly scar
41. Donor nerves are XII CN, spinal accessory,
masseteric branch of trigeminal nerve.
Restores movement to the side of the face
that has been paralyzed.
With the stump of the 12th nerve hooked up
to the end of the 7th nerve, the face will
move when the tongue is moved.
Best outcomes from cross facial nerve grafting
obtained if the period of denervation is less than 6
months.
42. INDICATIONS:
Irreversible facial nerve injury
Intact facial musculature/distal facial nerve
Intact proximal donor nerve
Prior to distal muscle/facial nerve atrophy
Ideal if performed within a year of facial paralysis
Advantages:-
Time interval until movement
4-6 months
Avoid multiple sites of anastomosis
Mimetic-like function achievable
with practice
Disadvantages:-
Donor site morbidity
Some degree of
synkinesis
43. 1) Parotidectomy incision
extended into cervical
crease ~ 2-3 cm below
inferior border of mandible
2) Facial nerve identified and
dissected distal to pes
anserinus
3) Identify hypoglossal nerve
a) SCM retracted posteriorly
b) Dissect superiorly until
posterior belly of digastic is
identified
c) Retract digastric
superiorly and CN XII is
found inferiorly.
d) Hypoglossal is within 2-3
cm of main trunk of the
facial nerve
1) Hypoglossal nerve is dissected
anteriorly and medially into the
tongueTransect distal to ansa
hypoglossis
2) Facial nerve transected at the
stylomastoid foramen
44. Entire hypoglossal nerve
transected
40% segment of
nerve secured to
lower division
Hypoglossal
nerve
reflected
superiorly
“Split” XII – VII cranial nerve transfer
transfer
45. End to end
neurorrhaphy between
XII CN & donor cable
nerve graft ( eg.
greater auricular nerve)
which serves as a jump
graft to the main trunk
of facial nerve
Jump graft modification
46. Facial nerve can be
mobilised in its
mastoid segment
from 2nd genu distally
& rotated inferiorly to
allow direct
coaptation to the
hypoglossal nerve.
It typically requires
removal of the
mastoid tip.
Reflection of the facial
nerve out of the mastoid
bone.
47. Contralateral Facial nerve used to
reinnervate paralyzed side using a nerve
graft
Sural nerve often employed
~25-30cm of graft needed
Restitution of smile and eye blinking
obtained.
Disadvantage:-
a)2nd surgical site
b)Violation of the normal facial nerve
48. FOUR techniques
Sural nerve graft routed from
buccal branch of normal VII
to stump of paralyzed VII
Zygomaticus and buccal
branch of normal VII used to
reinnervate zygomatic and
marginal mandibular portions
respectively
4 separate grafts from
temporal, zygomatic, buccal
and marginal mandibular
divisions of normal CN VII to
corresponding divisions on
paralyzed side.
Entire lower division of normal
side grafted to main trunk on
paralyzed side.
49. INDICATION:
Congenital facial paralysis
Facial nerve interruption of at least 3
years
Loss of motor endplates
Crossover techniques not possible due to
donor nerve sacrifice
50. Often used for
reanimation of the oral
commisure.
Middle 1/3 of muscle is
best for transfer (Sherris,
2004)
51. Incision in preauricular
crease extending to sup.
temporal line
Obtain wide exposure of
temporalis muscle by
dissecting above the
SMAS
Incise down on
periosteum to elevate
muscle fibers
Harvest middle 1/3
Large tunnel created
over zygomatic arch
Orbicularis oris muscle
exposed via vermilion
border incision at oral
commissure
52. Large tunnel over zygomatic arch used
to connect oral commisure to zygomatic
arch/superior incision.
Temporalis flap detached and elevated
from its origin and tunneled to the oral
commissure.
3-0 prolene used to suture orbicularis to
temporalis at oral commissure
Overcorrection of nasolabial fold and
oral commissure
53. Used when temporalis muscle is not
opted.
May be preferred due to avoidance of
large facial incision
Disadvantages:-
a) Less available muscle compared to
temporalis
b) Vector of pull on oral commisure is
more horizontal than
superior/oblique like temporalis
54. Vertical incision made in inferior portion
of muscle.
Anterior half of muscle is split into 2
divisions.
The 2 anterior slips of muscle are
tunneled anteriorly to reach the oral
commisure via external vermillion border
incisions.
Muscle slips are attached to lips and oral
commisure in the deep dermal layer
using suture.
55. They have potential of achieving individual
segmental contractions
› Reduction of synkinesis
Muscle flaps used are:
› Gracilis
› Latissimus dorsi
› Inferior rectus abdominus
56. Requires viable muscle and nerve
innervation
Traditionally done in 2 stages
a) 1st:- Cross-face nerve graft ~ 1 yr prior
to muscle transfer.
b) 2nd:- Muscle transfer performed after
neural ingrowth of graft.
57. 1. “Workhorse” for free
muscle transfer
2. Long, thin muscle in
medial thigh
-Good neurovasular
pedicle
1. Adductor artery
and vein
2. Anterior obturator
nerve
3. 2 stages involved:
1. Sural nerve employed
for cross-face graft
2. Gracilis muscle
transferred after 6-12
months
4. Vascular anastomosis to
the facial artery and vein
or to superficial temporal
Anterior Obturator nerve
Adductor a. & v.
58. Exposure keratitis Corneal ulceration
Corneal breakdown Blindness
Goal of treatment is to maintain Cornea
safe.
Complications of orbicularis oculi paresis-
a) Delayed blinking
b) Impairment of nasolacrimal system
c) Dry eye
d) Risk of cornea.
59. Initial treatment
a) Ophthalmic drops/ointments
b) Protective taping,
c) Occlusive moisture chambers,
d) Soft contact lenses, scleral shields
e) Tarsorrhaphy suture
Majority of patients require definitive
surgical treatment to correct chronic
impairment
60. Surgical options include:
a) Palpebral springs (Levine, May)
b) Tarsorrhaphy (McLaughlin)
c) Lid loading & shortening tech(Gold
weight, spring implant)
d) Combinations
e) Temporalis muscle transfer (Gillies)
f) Encircling the upper and lower eyelids
with silicone or fascia lata (Freeman)
Surgical T/t considered when medical T/t fails & in case of BAD
syndrome (Scott-Brown’s 6th edition)
B Bell’s phenomenon
A corneal Anaesthesia
D Dry eye
61. Advantages
a) Less visible
Disadvantages
a) Technically difficult
b) Higher risk
of extrusion
62. Horizontal mattress 5-0 nylon
Begin 3mm medial to lateral
canthus, 6mm from lid
margin
Stitch travels through gray
line to 5mm below lower lid
margin
Bolster with 3mm, 4-french
rubber catheter.
Cosmetically unappealing,
visual field affected
63. Early technique
– Incision in the supratarsal crease
– Subcutaneous pocket
– Insert weight
– Close skin
Stainless steel Gold
– High profile
– Migratory
– High rate of extrusion
– Higher density - more weight
in same size
-Malleable - conforms to the
globe-lower profile
– Lower reactivity,
Reversible,Migratory
– High rate of extrusion
64. Small incision made
several millimeters
above the upper
eyelid margin.
Tarsal plate exposed
with sharp dissection
Gold weight secured
to tarsus beneath
levator aponeurosis
using 8-0 nylon.
Wound closed in 2
layers
65. Advantages
a) Technically straightforward
b) Consistent
Disadvantages
a) less than with previous technique
b) Less Visibility
c) Less Extrusion
d) Less Mobility
67. The two main d/d possibilities are
developmental & traumatic.
The most common finding asso with
congenital facial palsy is presence of two
or more other anomalies.
Develop. b/l facial palsy is freq. incomplete
with lower portion of face less affected than
the upper part. This distinguishes it from
facial palsy due to trauma, which is rarely
B/L & equally involves upper and lower part
of the face.
68. At present, with the exception of free
muscle neurovascular transplantation,
there is no effective way to restore facial
function in conginital facial paralysis.
Delay reanimation surgical procedures
until patient reaches adolescent years.
Management directed towards
preventing complications.
Main area of concern for reanimation
is the eye.