The document summarizes diseases of the conjunctiva, including different types of conjunctivitis. It describes the anatomy of the conjunctiva and its parts. The main types of conjunctivitis discussed are bacterial, viral, allergic, and chemical conjunctivitis. Bacterial conjunctivitis commonly causes purulent discharge and is usually self-limiting. Viral conjunctivitis spreads easily and causes watery discharge. Allergic conjunctivitis like vernal catarrh causes itchiness and mucoid discharge. Chemical conjunctivitis results from exposure to irritants and causes a painful, red eye.
this document is designed and serving to successfully help students, teachers or ophthalmic clinicians to deliver a sustained and effective management of conjuctiva disorders
Conjunctivitis is an inflammation or swelling of the conjunctiva. The conjunctiva is the thin transparent layer of tissue that lines the inner surface of the eyelid and covers the white part of the eye. Often called "pink eye".
this document is designed and serving to successfully help students, teachers or ophthalmic clinicians to deliver a sustained and effective management of conjuctiva disorders
Conjunctivitis is an inflammation or swelling of the conjunctiva. The conjunctiva is the thin transparent layer of tissue that lines the inner surface of the eyelid and covers the white part of the eye. Often called "pink eye".
to download this presentation from this link
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over view for the conjunctival diseases. clinical presentation treatment .
Dacryocystitis is an infection or inflammation of the nasolacrimal sac, usually accompanied by blockage of the nasolacrimal duct. Dacryocystitis can be acute or chronic and congenital or acquired.
to download this presentation from this link
https://mohmmed-ink.blogspot.com/2020/11/conjunctival-diseases-overview.html
over view for the conjunctival diseases. clinical presentation treatment .
Dacryocystitis is an infection or inflammation of the nasolacrimal sac, usually accompanied by blockage of the nasolacrimal duct. Dacryocystitis can be acute or chronic and congenital or acquired.
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Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
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Tom Selleck, an enduring figure in Hollywood. has captivated audiences for decades with his rugged charm, iconic moustache. and memorable roles in television and film. From his breakout role as Thomas Magnum in Magnum P.I. to his current portrayal of Frank Reagan in Blue Bloods. Selleck's career has spanned over 50 years. But beyond his professional achievements. fans have often been curious about Tom Selleck Health. especially as he has aged in the public eye.
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Introduction
Many have been interested in Tom Selleck health. not only because of his enduring presence on screen but also because of the challenges. and lifestyle choices he has faced and made over the years. This article delves into the various aspects of Tom Selleck health. exploring his fitness regimen, diet, mental health. and the challenges he has encountered as he ages. We'll look at how he maintains his well-being. the health issues he has faced, and his approach to ageing .
Early Life and Career
Childhood and Athletic Beginnings
Tom Selleck was born on January 29, 1945, in Detroit, Michigan, and grew up in Sherman Oaks, California. From an early age, he was involved in sports, particularly basketball. which played a significant role in his physical development. His athletic pursuits continued into college. where he attended the University of Southern California (USC) on a basketball scholarship. This early involvement in sports laid a strong foundation for his physical health and disciplined lifestyle.
Transition to Acting
Selleck's transition from an athlete to an actor came with its physical demands. His first significant role in "Magnum P.I." required him to perform various stunts and maintain a fit appearance. This role, which he played from 1980 to 1988. necessitated a rigorous fitness routine to meet the show's demands. setting the stage for his long-term commitment to health and wellness.
Fitness Regimen
Workout Routine
Tom Selleck health and fitness regimen has evolved. adapting to his changing roles and age. During his "Magnum, P.I." days. Selleck's workouts were intense and focused on building and maintaining muscle mass. His routine included weightlifting, cardiovascular exercises. and specific training for the stunts he performed on the show.
Selleck adjusted his fitness routine as he aged to suit his body's needs. Today, his workouts focus on maintaining flexibility, strength, and cardiovascular health. He incorporates low-impact exercises such as swimming, walking, and light weightlifting. This balanced approach helps him stay fit without putting undue strain on his joints and muscles.
Importance of Flexibility and Mobility
In recent years, Selleck has emphasized the importance of flexibility and mobility in his fitness regimen. Understanding the natural decline in muscle mass and joint flexibility with age. he includes stretching and yoga in his routine. These practices help prevent injuries, improve posture, and maintain mobilit
Lung Cancer: Artificial Intelligence, Synergetics, Complex System Analysis, S...Oleg Kshivets
RESULTS: Overall life span (LS) was 2252.1±1742.5 days and cumulative 5-year survival (5YS) reached 73.2%, 10 years – 64.8%, 20 years – 42.5%. 513 LCP lived more than 5 years (LS=3124.6±1525.6 days), 148 LCP – more than 10 years (LS=5054.4±1504.1 days).199 LCP died because of LC (LS=562.7±374.5 days). 5YS of LCP after bi/lobectomies was significantly superior in comparison with LCP after pneumonectomies (78.1% vs.63.7%, P=0.00001 by log-rank test). AT significantly improved 5YS (66.3% vs. 34.8%) (P=0.00000 by log-rank test) only for LCP with N1-2. Cox modeling displayed that 5YS of LCP significantly depended on: phase transition (PT) early-invasive LC in terms of synergetics, PT N0—N12, cell ratio factors (ratio between cancer cells- CC and blood cells subpopulations), G1-3, histology, glucose, AT, blood cell circuit, prothrombin index, heparin tolerance, recalcification time (P=0.000-0.038). Neural networks, genetic algorithm selection and bootstrap simulation revealed relationships between 5YS and PT early-invasive LC (rank=1), PT N0—N12 (rank=2), thrombocytes/CC (3), erythrocytes/CC (4), eosinophils/CC (5), healthy cells/CC (6), lymphocytes/CC (7), segmented neutrophils/CC (8), stick neutrophils/CC (9), monocytes/CC (10); leucocytes/CC (11). Correct prediction of 5YS was 100% by neural networks computing (area under ROC curve=1.0; error=0.0).
CONCLUSIONS: 5YS of LCP after radical procedures significantly depended on: 1) PT early-invasive cancer; 2) PT N0--N12; 3) cell ratio factors; 4) blood cell circuit; 5) biochemical factors; 6) hemostasis system; 7) AT; 8) LC characteristics; 9) LC cell dynamics; 10) surgery type: lobectomy/pneumonectomy; 11) anthropometric data. Optimal diagnosis and treatment strategies for LC are: 1) screening and early detection of LC; 2) availability of experienced thoracic surgeons because of complexity of radical procedures; 3) aggressive en block surgery and adequate lymph node dissection for completeness; 4) precise prediction; 5) adjuvant chemoimmunoradiotherapy for LCP with unfavorable prognosis.
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Disruption of blood supply to lung alveoli due to blockage of one or more pulmonary blood vessels is called as Pulmonary thromboembolism. In this presentation we will discuss its causes, types and its management in depth.
Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
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2. APPLIED ANATOMY
• The conjunctiva is a translucent mucous membrane which lines the
posterior surface of the eyelids and anterior aspect of eyeball.
• Parts of conjunctiva;
- Palpebral conjunctiva
- Bulbar conjunctiva
- Conjunctival fornix
8. CONJUNCTIVITIS
• Conjunctivitis means inflammation of the
conjunctiva.
• Conjunctivitis is the commonest single diagnosis in
eye clinics throughout Africa.
9. ALL PATIENTS WITH CONJUNCTIVITIS HAVE SOME
SYMPTOMS AND SIGNS IN COMMON
1) Pain/Discomfort - usually described as a "foreign body
sensation"
2) Redness - The conjunctiva is red and inflamed.
3) Discharge - usually sticky, but also watery, or mucous.
10. DISCHARGE
1) Watery discharge is composed of a serous exudate and tears
and occurs in acute viral or acute allergic conjunctivitis
2) Mucoid discharge is typical of chronic allergic conjunctivitis
and dry eye
3) Mucopurulent discharge typically occurs in chlamydial or
acute bacterial infection
4) Moderately purulent discharge occurs in acute bacterial
conjunctivitis
5) Severe purulent discharge is typical of gonococcal infection
12. CONJUCTIVITIS
• It can be divided into:
1) Bacterial
2) Viral
3) Allergic
4) Chlamydial
5) Chemical
6) Granulomatous
13. BACTERIAL CONJUNCTIVITIS
• Commonly caused by Staphylococcus Aureus
• The patient has a gritty eye, with a purulent discharge
• The conjunctiva is red, particularly in the lower fornix,
where a purulent discharge may be visible
• The condition is self-limiting, but can be treated with
topical antibiotics, or regular face-washing
17. BACTERIAL CONJUNCTIVITIS. (A) EYELID EDEMA AND ERYTHEMA IN SEVERE INFECTION; (B) DIFFUSE CONJUNCTIVAL INJECTION
INVOLVING THE TARSAL AND FORNICEAL CONJUNCTIVA; (C) MUCOPURULENT DISCHARGE; (D) PROFUSE PURULENT DISCHARGE; (E)
SUPERIOR CORNEAL ULCERATION; (F) GRAM STAIN SHOWS KIDNEY-SHAPED DIPLOCOCCIC
18. GONOCOCCAL CONJUNCTIVITIS
• Rare but very serious form of bacterial conjunctivitis
caused by Neisseria Gonorrhea
• It usually affects very young babies, about 3-10 days old.
They become infected from their mother's genital tract
during birth
• The eyelids are very swollen, and there is a profuse
purulent discharge. Within 24 hours both corneas can be
totally destroyed and the child will be permanently blind
• Gonococcal conjunctivitis can also occur in adults
19.
20. • Patients with a severe purulent conjunctivitis need an
immediate swab and gram stain of their discharge
• Treatment with half-hourly antibiotic drops, and
appropriate systemic treatment for gonorrhoea.
21.
22. • (OPhthalmia neonatorum) can be prevented by cleaning the
eyes of babies at birth
• Additional protection may be provided by giving a drop of an
antibiotic or disinfectant (such as Silver Nitrate or Povidine
Iodine) into each eye.
23. VIRAL CONJUCTIVITIS
• Inflammation of the conjuctiva caused by the virus
• The two commonest viruses causing viral conjunctivitis outbreaks are:
-Adenovirus group
-Enterovirus 70 group
• Mainly occur in epidemics (schools work place)
• Transmitted by fingers, swimming pools
• The most common cause of viral conjunctivitis is adeno virus
25. ADENOVIRUS
• Adenovirus causes severe conjunctivitis, which may last for up to
four weeks
• Can be associated with small sub-epithelial corneal opacities,
which may take up to six months to disappear
• The patient will have follicles in the upper and lower fornices,
and the corneal opacities may be visible
• There is no specific treatment
26. ENTEROVIRUS70 GROUP
• Enterovirus causes acute hemorrhagic conjunctivitis
• This is a milder form of conjunctivitis, accompanied by sub-
conjunctival hemorrhages, which make the eye look worse
than it really is
• There is no specific treatment
• The condition resolves spontaneously in 1-2 weeks.
27. PRECAUTION
• Both types of viral conjunctivitis are very infectious, and
great care must be taken to avoid transmitting the disease
to other patients or to yourself!
28. ALLERGIC CONJUNCTIVITIS
• This is the most common form of conjunctivitis
• vernal catarrh is the commonest form of allergic conjuctivitis
• This is a chronic allergic condition that lasts 5-7 years before spontaneously
resolving
• It is common in young people, but rare in anyone over 20 years old
• It is found in cooler, higher areas of Kenya
29. SIGNS AND SYMPTOMS
• Itchy eyes,
• mucoid or watery discharge
• There are two types of vernal catarrh, limbal and tarsal.
• Patients with limbal vernal catarrh have large papillae around the limbus
• Those with tarsal vernal catarrh have papillae on the upper tarsal plate
• Many patients have both, but with one or other type predominating
31. LIMBAL VERNAL DISEASE. (A) SPARSE LIMBAL PAPILLAE; (B) PAPILLAE WITH HORNER–TRANTAS DOTS; (C) EXTENSIVE
PAPILLAE; (D) EXTREMELY SEVERE INVOLVEMENT
32.
33. RX OF ALLERGIC CONJUNCTIVITIS
• Topical steroid drops make the eye feel much better, but the
disease recurs when the drops are stopped
• Long-term use of steroid drops may cause blindness
• Systemic steroids are more effective, but even more dangerous
• Unless there is corneal ulceration, oral steroids should be
avoided
34. • The patient can be reassured that the disease will get
better
• Immediate symptomatic relief can be obtained by washing
the eyes with clean cold water.
• Mast cell stabilizers drops
• Antihistamine drops
• Soothing agents
35. CHEMICAL CONJUNCTIVITIS
This is caused by exposure to an irritant chemical such as
• acids,
• tobacco
• smoke,
• snake venom, or the
• sap of certain trees- particularly Euphorbia spp.
36. PRESENTATION/RX
• There is usually hx of chemical entering the eye
• Patient has painful, watering, red eye
• If exposure took place within the preceding few hours, it
may be helpful to irrigate the conjunctival sac
• Antibiotic drops should be given to prevent secondary
infection