This document provides information about ill-fitting denture induced lesions. It discusses the purpose of the presentation which is to describe the etiology, clinical features, management, and differential diagnosis of various denture induced lesions. It then covers various specific lesions in detail, including traumatic ulcer, cheek bite, inflammatory hyperplasia, fibrous papillary hyperplasia, chronic atrophic candidiasis (denture stomatitis), contact allergy, malignancy, angular cheilitis, and palatal perforation. For each lesion, it discusses causes, clinical features, and management.
1. Dental pulp diseases include pulpitis, which can be acute or chronic. Acute pulpitis is reversible or irreversible, while chronic pulpitis can be closed or open.
2. Periapical diseases result from pulp necrosis and include acute or chronic apical periodontitis, periapical abscesses, cysts, and osteomyelitis. Chronic apical periodontitis often forms a periapical granuloma.
3. Symptoms, causes, histological features, radiographic features and treatments are described for each condition. Physical, chemical and microbial factors can all contribute to pulp and periapical diseases.
Oral lichen planus is a common chronic mucocutaneous disease of unknown etiology that may undergo malignant transformation. It typically presents as white reticulated lines on the oral mucosa but can also appear as erosive, atrophic, bullous or other lesions. A confirmed diagnosis requires characteristic histopathology. While there is no cure, treatment focuses on managing symptoms like pain with topical or systemic corticosteroids and maintaining oral hygiene to reduce cancer risks.
Vestibuloplasty is a surgical procedure to deepen the vestibule by uncovering existing bone and repositioning overlying soft tissues. There are several techniques for vestibuloplasty including submucosal vestibuloplasty, secondary epithelialization techniques, and grafting vestibuloplasty. Recent advances include the use of collagen matrix grafts like Geistlich Mucograft which integrate well and promote soft tissue regeneration as an alternative to harvesting autologous grafts.
This document discusses red and white lesions of the oral cavity, focusing on oral candidiasis. It describes the various types of oral candidiasis including pseudomembranous, erythematous, chronic plaque-type, and median rhomboid glossitis. Predisposing factors, clinical findings, diagnosis, treatment with antifungal medications or surgery, and prognosis are summarized for each type. Chronic hyperplastic candidiasis may require long-term antifungal therapy or surgery due to risk of recurrence. Overall prognosis is generally good if predisposing factors can be addressed.
This document discusses radicular cysts, which are the most common inflammatory cysts in the oral cavity. Radicular cysts arise from epithelial residues in the periodontal ligament as a result of periapical periodontitis following pulp necrosis. They are usually asymptomatic but can cause swelling and bone resorption as they expand. The cyst forms from the proliferation of epithelial cell rests in the granulation tissue surrounding the apex of an infected tooth. Histologically, they are lined by stratified squamous epithelium and surrounded by fibrous connective tissue that may contain cholesterol crystals. Treatment involves root canal therapy or extraction with curettage of the cyst lining.
This document provides information on vesiculobulllous lesions, including their classification, pathophysiology, diagnosis, types of pemphigus, clinical features, pathogenesis, histopathology, confirmatory diagnosis, differential diagnosis, and treatment. It discusses conditions like pemphigus vulgaris, pemphigoid, paraneoplastic pemphigus. Pemphigus vulgaris involves autoantibodies against desmogleins 1 and 3, causing blistering in skin and mucosa. Paraneoplastic pemphigus is associated with neoplasms and involves multiple organs. Diagnosis involves biopsy, DIF, and ELISA to detect specific autoantibodies.
Vestibuloplasty is a surgical procedure to deepen the oral vestibule by changing the attachments of the soft tissue. There are several types of vestibuloplasty procedures, including mucosal advancement, secondary epithelization, and grafting. Mucosal advancement involves undermining and advancing the oral mucosa, while secondary epithelization uses the oral mucosa to line one side and allows the other side to heal through epithelization. Grafting can use skin, mucosa, or dermis grafts to line the extended vestibule. The document discusses techniques for each type of vestibuloplasty procedure.
This document describes vesiculo-bullous lesions, which present clinically as vesicles or bullae that often rupture early, appearing as ulcerated or erosive areas. Some key points:
- Vesicles are fluid-filled lesions less than 1 cm, while bullae contain fluid and are over 1 cm.
- Causes include trauma, infection, autoimmunity, and genetic factors.
- Examples described include herpes simplex infection, varicella zoster infection, and hand foot and mouth disease. Clinical features, pathogenesis, management are provided for each. Classification is discussed based on acute vs chronic presentation, clinical presentation, and histopathological location.
1. Dental pulp diseases include pulpitis, which can be acute or chronic. Acute pulpitis is reversible or irreversible, while chronic pulpitis can be closed or open.
2. Periapical diseases result from pulp necrosis and include acute or chronic apical periodontitis, periapical abscesses, cysts, and osteomyelitis. Chronic apical periodontitis often forms a periapical granuloma.
3. Symptoms, causes, histological features, radiographic features and treatments are described for each condition. Physical, chemical and microbial factors can all contribute to pulp and periapical diseases.
Oral lichen planus is a common chronic mucocutaneous disease of unknown etiology that may undergo malignant transformation. It typically presents as white reticulated lines on the oral mucosa but can also appear as erosive, atrophic, bullous or other lesions. A confirmed diagnosis requires characteristic histopathology. While there is no cure, treatment focuses on managing symptoms like pain with topical or systemic corticosteroids and maintaining oral hygiene to reduce cancer risks.
Vestibuloplasty is a surgical procedure to deepen the vestibule by uncovering existing bone and repositioning overlying soft tissues. There are several techniques for vestibuloplasty including submucosal vestibuloplasty, secondary epithelialization techniques, and grafting vestibuloplasty. Recent advances include the use of collagen matrix grafts like Geistlich Mucograft which integrate well and promote soft tissue regeneration as an alternative to harvesting autologous grafts.
This document discusses red and white lesions of the oral cavity, focusing on oral candidiasis. It describes the various types of oral candidiasis including pseudomembranous, erythematous, chronic plaque-type, and median rhomboid glossitis. Predisposing factors, clinical findings, diagnosis, treatment with antifungal medications or surgery, and prognosis are summarized for each type. Chronic hyperplastic candidiasis may require long-term antifungal therapy or surgery due to risk of recurrence. Overall prognosis is generally good if predisposing factors can be addressed.
This document discusses radicular cysts, which are the most common inflammatory cysts in the oral cavity. Radicular cysts arise from epithelial residues in the periodontal ligament as a result of periapical periodontitis following pulp necrosis. They are usually asymptomatic but can cause swelling and bone resorption as they expand. The cyst forms from the proliferation of epithelial cell rests in the granulation tissue surrounding the apex of an infected tooth. Histologically, they are lined by stratified squamous epithelium and surrounded by fibrous connective tissue that may contain cholesterol crystals. Treatment involves root canal therapy or extraction with curettage of the cyst lining.
This document provides information on vesiculobulllous lesions, including their classification, pathophysiology, diagnosis, types of pemphigus, clinical features, pathogenesis, histopathology, confirmatory diagnosis, differential diagnosis, and treatment. It discusses conditions like pemphigus vulgaris, pemphigoid, paraneoplastic pemphigus. Pemphigus vulgaris involves autoantibodies against desmogleins 1 and 3, causing blistering in skin and mucosa. Paraneoplastic pemphigus is associated with neoplasms and involves multiple organs. Diagnosis involves biopsy, DIF, and ELISA to detect specific autoantibodies.
Vestibuloplasty is a surgical procedure to deepen the oral vestibule by changing the attachments of the soft tissue. There are several types of vestibuloplasty procedures, including mucosal advancement, secondary epithelization, and grafting. Mucosal advancement involves undermining and advancing the oral mucosa, while secondary epithelization uses the oral mucosa to line one side and allows the other side to heal through epithelization. Grafting can use skin, mucosa, or dermis grafts to line the extended vestibule. The document discusses techniques for each type of vestibuloplasty procedure.
This document describes vesiculo-bullous lesions, which present clinically as vesicles or bullae that often rupture early, appearing as ulcerated or erosive areas. Some key points:
- Vesicles are fluid-filled lesions less than 1 cm, while bullae contain fluid and are over 1 cm.
- Causes include trauma, infection, autoimmunity, and genetic factors.
- Examples described include herpes simplex infection, varicella zoster infection, and hand foot and mouth disease. Clinical features, pathogenesis, management are provided for each. Classification is discussed based on acute vs chronic presentation, clinical presentation, and histopathological location.
Fixed partial dentures transmit forces through the abutments to the periodontium. Failures are due to poor engineering, the use of improper materials, inadequate tooth preparation, and faulty fabrication. Of particular concern to prosthodontist is the selection of teeth for abutments. They must recognize the forces developed by the oral mechanism, and resistance.
Successful selection of abutments for fixed partial dentures requires sensitive diagnostic ability. Thorough knowledge of anatomy, ceramics, the chemistry and physics of dental materials, metallurgy, Periodontics, phonetics, physiology, radiology and the mechanics of oral function is fundamental.
benign and malignant tumors of connective tissue originmadhusudhan reddy
This document discusses various connective tissue tumors that can occur in the oral cavity. It describes benign fibrous lesions like fibroma and giant cell fibroma. It also discusses benign adipose tissue lesions like lipoma. Various benign vascular lesions are described, including hemangiomas and lymphangiomas. Finally, it summarizes benign bone tissue tumors like osteoma and osteoid osteoma. For each lesion, the clinical features, histopathology, radiographic appearance, and treatment are summarized.
The presentation explain white lesions in oral cavity and the classification the demonstrate the etiology, histopathology, diagnosis and treatment for each one.
This document discusses and compares different types of non-odontogenic (not related to teeth) cysts. It separates them into developmental and inflammatory cysts. Developmental cysts form due to epithelial cell remnants becoming trapped during embryonic development, while inflammatory cysts form due to duct obstruction or trauma. Some examples of developmental cysts mentioned are nasopalatine duct cysts, median palatal cysts, and dermoid cysts. Inflammatory cysts include mucoceles, ranulas caused by salivary gland duct obstruction, and retention cysts of the maxillary sinus. The document provides details on pathogenesis, clinical features, histopathology, diagnosis and treatment of several of these cyst types.
The document defines and describes periodontal pockets. It notes that periodontal pockets can be classified based on their location relative to the alveolar bone as either suprabony or infrabony. Suprabony pockets have bone loss horizontally while infrabony pockets have bone loss vertically. The document also discusses the pathogenesis of pocket formation, clinical features, histopathology, and diagnosis and probing of periodontal pockets.
This document discusses methods for diagnosing dental caries. It begins with definitions of caries and outlines ideal requirements for diagnostic methods. Traditional methods discussed include visual examination, tactile probing, tooth separation, dental floss, and radiography using techniques like intraoral periapical films and bitewing films. Advanced diagnostic tests mentioned include digital radiography, intraoral cameras, laser fluorescence. Recent advances discussed are terahertz imaging, optical coherence tomography, and cone beam computed tomography.
This document discusses the management of Class IV Ellis fractures, which involve trauma to teeth resulting in pulp involvement or loss of crown structure. It describes the emergency treatment, which may include reinserting an avulsed tooth. Intermediate treatment options for exposed or necrotic pulps include pulpectomy, apexification, or extraction. Pulpectomy involves conventional root canal treatment to eliminate infection, while apexification induces formation of new tissue at the root end in immature teeth. The document also discusses crown fractures involving enamel, dentin, or both, and the treatment options depending on the extent of injury and pulp involvement. These may include protective dressings, temporary crowns, or reattachment of fragments.
Dentinogenesis imperfecta is a hereditary condition that affects the formation of dentin in both primary and permanent teeth. It is classified into two main types - dentinogenesis imperfecta type 1 and type 2. Type 1 is caused by mutations in the DSPP gene and affects only the teeth. Type 2 may be caused by mutations in two tightly linked genes and is characterized by multiple pulp exposures and shell-like teeth. Treatment aims to prevent wear of enamel and dentin through full coverage restorations.
This presentation specifically deals with the maxillary and mandibular Major connectors used in a cast partial denture. it also mentions the uses, advantages and disadvantages of each,
Denture Induced Stomatitis, also known as denture sore mouth, is a common condition affecting up to 70% of denture wearers, characterized by erythema and swelling of the palate mucosa in contact with the dentures. It is caused mainly by Candida albicans accumulating on dentures due to poor oral and denture hygiene. Treatment involves improving hygiene, removing dental plaque from dentures and mucosa, and using antifungal medications like nystatin drops or miconazole gel.
In this lecture I explain in step-by-step fashion the basics of Measurement of Periodontal Attachment Loss. a photo guide is attached to the guide to aid in better understanding of the topic
Oral lichen planus is a chronic inflammatory disease that affects the oral mucosa. It is characterized by white striations (Wickham's striae) and varies in appearance from reticular to erythematous or ulcerative lesions. The cause is unknown but involves a cell-mediated immune response. Treatment focuses on reducing symptoms and includes topical corticosteroids, immunosuppressants, or retinoids. Malignant transformation may rarely occur so follow-up is important.
This document discusses odontogenic keratocysts (OKCs), a type of jaw cyst. It covers the classification, causes, histopathology, clinical features, radiographic features, differential diagnosis, treatment principles, and surgical treatment options for OKCs. OKCs most commonly occur in the mandibular molar and ramus areas and are often radiolucent and multilocular in appearance on radiographs. Treatment options include wide surgical excision or marsupialization to prevent recurrence of these cysts which have a high rate of recurrence compared to other jaw cysts.
A traumatic bone cyst is a lesion that forms within bone, often in the mandible, that lacks an epithelial lining. It is believed to result from trauma that causes a hematoma within the bone that fails to organize, leaving an empty cavity. Teenagers are most commonly affected, presenting with swelling or pain. Radiographs show a well-delineated radiolucent area with scalloped borders between tooth roots. Histopathology finds minimal fibrous tissue without an epithelial component.
Dental caries leads to changes in the enamel and dentin, including tubular sclerosis, reactionary dentine, and dead tracts formation. Left untreated, dental caries can cause pulpitis, which involves inflammation of the pulp and can be reversible, acute, or chronic. Pulpitis and pulp necrosis can further lead to apical periodontitis, periapical abscess, osteomyelitis, or cellulitis as the infection spreads. Advanced stages include formation of a periapical cyst, bone destruction, and soft tissue infection.
Oral mucosal lesions in denture wearersAamir Godil
The document discusses oral mucosal lesions that can occur in denture wearers. It describes several types of denture-related mucosal lesions (DMLs) such as traumatic ulcers, denture-induced stomatitis, and denture hyperplasia. It also discusses non-denture related lesions including fissured tongue and lichen planus. A statistical analysis found the most common DMLs were traumatic ulcers and denture stomatitis. Complete denture wearers had higher rates of DMLs while partial denture wearers saw more stomatitis. The document provides details on clinical presentation and management of several specific oral lesions.
The document provides information about Pindborg tumor, also known as calcifying epithelial odontogenic tumor (CEOT). It defines CEOT as a locally invasive epithelial odontogenic neoplasm characterized by the presence of amyloid material that may become calcified. The document discusses the pathogenesis, histopathological features including epithelial cells, amyloid-like material and calcific deposits, immunohistochemical findings, differential diagnosis and treatment of CEOT. It also mentions the recurrence rate of CEOT is typically 10-15% but can be higher in certain variants.
In this lecture I explain in step-by-step fashion the basics of Endodontic Diagnosis: Pulp Vitality Tests. a photo guide is attached to the guide to aid in better understanding of the topic
This document discusses mucosal pathologies that can result from oral prostheses. It describes how removable and fixed prostheses can disrupt the normal oral environment and initiate pathological conditions through mechanical irritation, microbial plaque accumulation, allergic reactions, and other factors. Common mucosal pathologies discussed include denture stomatitis, flabby ridge, denture irritation hyperplasia, traumatic ulcers, and angular chelitis. The document examines the clinical presentations, histology, causes, and suggested solutions for each condition.
La hiperplasia epitelial focal o enfermedad de Heck es una neoplasia benigna inusual de la mucosa bucal que afecta principalmente a niños y adolescentes de bajos recursos y de ciertos grupos étnicos como los esquimales. Se caracteriza por la presencia de pequeñas pápulas blanquecinas en la boca, especialmente en el labio inferior. Generalmente no requiere tratamiento, pero cuando hay problemas estéticos se pueden extirpar quirúrgicamente o tratar con láser o crioterapia.
Fixed partial dentures transmit forces through the abutments to the periodontium. Failures are due to poor engineering, the use of improper materials, inadequate tooth preparation, and faulty fabrication. Of particular concern to prosthodontist is the selection of teeth for abutments. They must recognize the forces developed by the oral mechanism, and resistance.
Successful selection of abutments for fixed partial dentures requires sensitive diagnostic ability. Thorough knowledge of anatomy, ceramics, the chemistry and physics of dental materials, metallurgy, Periodontics, phonetics, physiology, radiology and the mechanics of oral function is fundamental.
benign and malignant tumors of connective tissue originmadhusudhan reddy
This document discusses various connective tissue tumors that can occur in the oral cavity. It describes benign fibrous lesions like fibroma and giant cell fibroma. It also discusses benign adipose tissue lesions like lipoma. Various benign vascular lesions are described, including hemangiomas and lymphangiomas. Finally, it summarizes benign bone tissue tumors like osteoma and osteoid osteoma. For each lesion, the clinical features, histopathology, radiographic appearance, and treatment are summarized.
The presentation explain white lesions in oral cavity and the classification the demonstrate the etiology, histopathology, diagnosis and treatment for each one.
This document discusses and compares different types of non-odontogenic (not related to teeth) cysts. It separates them into developmental and inflammatory cysts. Developmental cysts form due to epithelial cell remnants becoming trapped during embryonic development, while inflammatory cysts form due to duct obstruction or trauma. Some examples of developmental cysts mentioned are nasopalatine duct cysts, median palatal cysts, and dermoid cysts. Inflammatory cysts include mucoceles, ranulas caused by salivary gland duct obstruction, and retention cysts of the maxillary sinus. The document provides details on pathogenesis, clinical features, histopathology, diagnosis and treatment of several of these cyst types.
The document defines and describes periodontal pockets. It notes that periodontal pockets can be classified based on their location relative to the alveolar bone as either suprabony or infrabony. Suprabony pockets have bone loss horizontally while infrabony pockets have bone loss vertically. The document also discusses the pathogenesis of pocket formation, clinical features, histopathology, and diagnosis and probing of periodontal pockets.
This document discusses methods for diagnosing dental caries. It begins with definitions of caries and outlines ideal requirements for diagnostic methods. Traditional methods discussed include visual examination, tactile probing, tooth separation, dental floss, and radiography using techniques like intraoral periapical films and bitewing films. Advanced diagnostic tests mentioned include digital radiography, intraoral cameras, laser fluorescence. Recent advances discussed are terahertz imaging, optical coherence tomography, and cone beam computed tomography.
This document discusses the management of Class IV Ellis fractures, which involve trauma to teeth resulting in pulp involvement or loss of crown structure. It describes the emergency treatment, which may include reinserting an avulsed tooth. Intermediate treatment options for exposed or necrotic pulps include pulpectomy, apexification, or extraction. Pulpectomy involves conventional root canal treatment to eliminate infection, while apexification induces formation of new tissue at the root end in immature teeth. The document also discusses crown fractures involving enamel, dentin, or both, and the treatment options depending on the extent of injury and pulp involvement. These may include protective dressings, temporary crowns, or reattachment of fragments.
Dentinogenesis imperfecta is a hereditary condition that affects the formation of dentin in both primary and permanent teeth. It is classified into two main types - dentinogenesis imperfecta type 1 and type 2. Type 1 is caused by mutations in the DSPP gene and affects only the teeth. Type 2 may be caused by mutations in two tightly linked genes and is characterized by multiple pulp exposures and shell-like teeth. Treatment aims to prevent wear of enamel and dentin through full coverage restorations.
This presentation specifically deals with the maxillary and mandibular Major connectors used in a cast partial denture. it also mentions the uses, advantages and disadvantages of each,
Denture Induced Stomatitis, also known as denture sore mouth, is a common condition affecting up to 70% of denture wearers, characterized by erythema and swelling of the palate mucosa in contact with the dentures. It is caused mainly by Candida albicans accumulating on dentures due to poor oral and denture hygiene. Treatment involves improving hygiene, removing dental plaque from dentures and mucosa, and using antifungal medications like nystatin drops or miconazole gel.
In this lecture I explain in step-by-step fashion the basics of Measurement of Periodontal Attachment Loss. a photo guide is attached to the guide to aid in better understanding of the topic
Oral lichen planus is a chronic inflammatory disease that affects the oral mucosa. It is characterized by white striations (Wickham's striae) and varies in appearance from reticular to erythematous or ulcerative lesions. The cause is unknown but involves a cell-mediated immune response. Treatment focuses on reducing symptoms and includes topical corticosteroids, immunosuppressants, or retinoids. Malignant transformation may rarely occur so follow-up is important.
This document discusses odontogenic keratocysts (OKCs), a type of jaw cyst. It covers the classification, causes, histopathology, clinical features, radiographic features, differential diagnosis, treatment principles, and surgical treatment options for OKCs. OKCs most commonly occur in the mandibular molar and ramus areas and are often radiolucent and multilocular in appearance on radiographs. Treatment options include wide surgical excision or marsupialization to prevent recurrence of these cysts which have a high rate of recurrence compared to other jaw cysts.
A traumatic bone cyst is a lesion that forms within bone, often in the mandible, that lacks an epithelial lining. It is believed to result from trauma that causes a hematoma within the bone that fails to organize, leaving an empty cavity. Teenagers are most commonly affected, presenting with swelling or pain. Radiographs show a well-delineated radiolucent area with scalloped borders between tooth roots. Histopathology finds minimal fibrous tissue without an epithelial component.
Dental caries leads to changes in the enamel and dentin, including tubular sclerosis, reactionary dentine, and dead tracts formation. Left untreated, dental caries can cause pulpitis, which involves inflammation of the pulp and can be reversible, acute, or chronic. Pulpitis and pulp necrosis can further lead to apical periodontitis, periapical abscess, osteomyelitis, or cellulitis as the infection spreads. Advanced stages include formation of a periapical cyst, bone destruction, and soft tissue infection.
Oral mucosal lesions in denture wearersAamir Godil
The document discusses oral mucosal lesions that can occur in denture wearers. It describes several types of denture-related mucosal lesions (DMLs) such as traumatic ulcers, denture-induced stomatitis, and denture hyperplasia. It also discusses non-denture related lesions including fissured tongue and lichen planus. A statistical analysis found the most common DMLs were traumatic ulcers and denture stomatitis. Complete denture wearers had higher rates of DMLs while partial denture wearers saw more stomatitis. The document provides details on clinical presentation and management of several specific oral lesions.
The document provides information about Pindborg tumor, also known as calcifying epithelial odontogenic tumor (CEOT). It defines CEOT as a locally invasive epithelial odontogenic neoplasm characterized by the presence of amyloid material that may become calcified. The document discusses the pathogenesis, histopathological features including epithelial cells, amyloid-like material and calcific deposits, immunohistochemical findings, differential diagnosis and treatment of CEOT. It also mentions the recurrence rate of CEOT is typically 10-15% but can be higher in certain variants.
In this lecture I explain in step-by-step fashion the basics of Endodontic Diagnosis: Pulp Vitality Tests. a photo guide is attached to the guide to aid in better understanding of the topic
This document discusses mucosal pathologies that can result from oral prostheses. It describes how removable and fixed prostheses can disrupt the normal oral environment and initiate pathological conditions through mechanical irritation, microbial plaque accumulation, allergic reactions, and other factors. Common mucosal pathologies discussed include denture stomatitis, flabby ridge, denture irritation hyperplasia, traumatic ulcers, and angular chelitis. The document examines the clinical presentations, histology, causes, and suggested solutions for each condition.
La hiperplasia epitelial focal o enfermedad de Heck es una neoplasia benigna inusual de la mucosa bucal que afecta principalmente a niños y adolescentes de bajos recursos y de ciertos grupos étnicos como los esquimales. Se caracteriza por la presencia de pequeñas pápulas blanquecinas en la boca, especialmente en el labio inferior. Generalmente no requiere tratamiento, pero cuando hay problemas estéticos se pueden extirpar quirúrgicamente o tratar con láser o crioterapia.
Este documento presenta un resumen de diferentes lesiones hiperplásicas de tejido fibroso bucal, incluyendo la hiperplasia fibrosa focal, la hiperplasia fibrosa inflamatoria, el fibroma osificante periférico y el granuloma periférico de células gigantes. Describe las características clínicas, etiología, histología y tratamiento de cada lesión.
Este documento describe varias lesiones hiperplásicas y ulcerativas, incluyendo hiperplasia fibrosa, epulis congénito del recién nacido, granuloma periférico de células gigantes, epulis granulomatoso, granuloma piógeno y fibroma odontogénico periférico. Cada lesión se describe en términos de su etiología, presentación clínica, diagnóstico, tratamiento y pronóstico. El documento proporciona información valiosa sobre estas afecciones para ayudar
The document discusses the benefits of exercise for mental health. Regular physical activity can help reduce anxiety and depression and improve mood and cognitive function. Exercise causes chemical changes in the brain that may help protect against mental illness and improve symptoms for those who already suffer from conditions like depression and anxiety.
Sequelae of wearing complete dentures/ orthodontics training coursesIndian dental academy
Indian Dental Academy: will be one of the most relevant and exciting training center with best faculty and flexible training programs for dental professionals who wish to advance in their dental practice,Offers certified courses in Dental implants,Orthodontics,Endodontics,Cosmetic Dentistry, Prosthetic Dentistry, Periodontics and General Dentistry.
La hiperplasia papilar inflamatoria es una afección benigna causada por prótesis dentales mal ajustadas que producen inflamación crónica en la bóveda palatina. Se caracteriza por la proliferación de pequeños nódulos en la bóveda palatina. El diagnóstico se basa en el examen clínico e histopatológico y se diferencia del carcinoma verrugoso. El tratamiento incluye mejorar los hábitos de higiene, estabilizar o reemplazar las prótesis dentales y en ocasiones cirugía
The document discusses the benefits of exercise for mental health. Regular physical activity can help reduce anxiety and depression and improve mood and cognitive functioning. Exercise causes chemical changes in the brain that may help boost feelings of calmness, happiness and focus.
HIPERPLASIA FIBROSA INFLAMATORIA TRATADA CON VESTIBULOPLASTIA MODIFICADA: REP...Edwin José Calderón Flores
La extirpación quirúrgica del tejido hiperplásico inflamatorio mediante vestibuloplastia modificada y la fijación de la prótesis existente con tornillo durante la cicatrización resultó en una recuperación exitosa sin evidencia de recurrencia de la lesión a los seis meses de seguimiento. La extirpación quirúrgica del tejido blando hiperplásico es un procedimiento efectivo para tratar la hiperplasia fibrosa inflamatoria y mejorar las condiciones para una nueva prótesis.
The document discusses common complaints of complete denture wearers, including discomfort/pain, looseness, difficulty adapting, altered speech, inability to eat, and issues with appearance. It provides detailed explanations of potential causes for each complaint and recommends treatments such as adjusting the occlusion, relieving pressure areas, improving border extensions, and remaking dentures if needed. The most common problems are reported as pain, looseness due to poor retention, and inability to adapt.
La estomatitis nicotínica es causada por el calor y humo del tabaco que produce hiperqueratosis en el paladar. Se presenta comúnmente en adultos masculinos que fuman y se caracteriza por eritema y elevaciones blancas en el paladar posterior. Histológicamente muestra metaplasia epidermoide y queratosis en los conductos de las glándulas salivales, con inflamación del tejido circundante. El pronóstico es favorable si se deja de fumar y se sigue el tratamiento.
La hiperqueratosis focal es una lesión blanca causada por el roce crónico de la mucosa bucal. Se observa comúnmente en áreas como los labios y bordes de la lengua. Histopatológicamente puede mostrar células inflamatorias crónicas en el tejido subyacente, pero sin cambios epiteliales displásicos. El diagnóstico y tratamiento implican identificar y eliminar la causa del roce, y realizar una biopsia solo si la causa es dudosa.
This document provides information on post-insertion instructions, problems, and solutions for patients receiving dentures. It outlines common issues patients may experience like discomfort with speaking and eating, as well as problems like denture stomatitis. Solutions for issues are discussed, such as ensuring proper denture cleaning and storage. The document stresses the importance of educating patients to have successful denture treatment and avoid frustration by addressing any complaints.
Lesiones ulcerativas hiperplasicas de la cavidad bucalCat Lunac
Este documento describe varias lesiones ulcerativas e hiperplásicas de la cavidad bucal, incluyendo hiperplasia fibrosa, epulis congénito, granuloma periférico de células gigantes, epulis granulomatoso, granuloma piógeno y fibroma odontogénico periférico. Define estas lesiones, explica sus características clínicas e histológicas, y analiza sus posibles causas y tratamientos.
Este documento resume varias lesiones orales comunes causadas por prótesis dentales mal ajustadas o fracturadas, incluyendo hiperplasia fibrosa inflamatoria, hiperplasia papilar inflamatoria, hiperqueratosis, y estomatitis de contacto. También describe tumores benignos como torus palatino, exostosis y osteoma, así como tumores malignos como osteosarcoma y tumores odontogénicos como ameloblastoma y odontoma. Para la mayoría de estas condiciones, el tratamiento incluye quitar el agente causal, mejor
The document provides instructions for the insertion appointment process for dentures. It discusses remounting the dentures on an articulator using records to ensure proper fit. It also covers adjusting the denture borders and base using pressure indicating paste and disclosing wax. The document emphasizes equilibrating the dentures in centric relation and various excursions. Patient education on denture limitations and follow-up care is also summarized.
Management of Denture fissuratum involves surgically removing excess tissues. A poorly fitting denture should be remade or relined to properly fit. The document references Prosthodontic Treatment for Edontulous Patients by Bolender for further information on treating denture fissuratum.
Epulis fissuratum is a benign lesion caused by chronic irritation from a poorly fitting denture. A 57-year-old female presented with a complaint of difficulty eating due to her broken denture. Clinical examination revealed an epulis fissuratum lesion. The lesion was surgically excised using electrocautery. Post-operatively, the patient was prescribed antibiotics and pain medication. At follow-up, the surgical site had healed well with no recurrence of the lesion.
Este documento describe diferentes tipos de lesiones blancas que pueden presentarse en la boca. Describe lesiones como queratosis friccional, queratosis de fumador, morsicatio bucarum, estomatitis nicotínica, lengua vellosa, línea alba oclusal, leucoedema, gránulos de Fordyce y liquen plano. Explica las características clínicas y el tratamiento de cada una de estas lesiones blancas. También menciona otras lesiones como leucoplasia que pueden ser premalignas y requieren bi
This document discusses common problems patients may experience after receiving removable partial or complete dentures and how to address them. Some potential early issues include pain, soreness of teeth or soft tissues, instability of the prosthesis, biting of cheeks or tongue, and speech or eating difficulties. The summary examines how to identify and resolve issues like soft tissue irritation, erythema, tooth irritation, occlusal interference, and miscellaneous complaints through examination, identification of pressure points, and making adjustments to the denture.
Denture induced lesions /orthodontic courses by Indian dental academy Indian dental academy
The Indian Dental Academy is the Leader in continuing dental education , training dentists in all aspects of dentistry and
offering a wide range of dental certified courses in different formats.for more details please visit
www.indiandentalacademy.com
The Indian Dental Academy is the Leader in continuing dental education , training dentists in all aspects of dentistry and
offering a wide range of dental certified courses in different formats.
This document discusses several benign oral lesions that can be mistaken for tumours, including tori, fibromas, pyogenic granulomas, peripheral ossifying fibromas, and lipomas. It provides details on the clinical features, locations, appearances, diagnoses, and typical treatment for each condition, emphasizing that they are non-cancerous structural variants or reactive lesions of the oral soft tissues and bones. Conservative surgical excision is usually sufficient to treat these common benign growths of the oral cavity.
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2. PURPOSE STATEMENT
• At the end of the presentation the
learners should be able to
describe etiopathogenesis, clinical
features, management &
differential diagnosis of various
denture induced lesions.
3. Serial no. Learning objectives Domain Level Criteria condition
1. Explain the
etiopathogenesis of
various denture
induced lesions.
Cognitive Must know all --
2. Explain the clinical
features of various
denture induced
lesions.
Cognitive&
psychomotor
Must know all --
3. Differentiate denture
induced lesions from
other similar lesions.
Cognitive&
psychomotor
Must know all --
4. Describe management
for denture induced
lesions.
Cognitive Must know all --
4. ILL-FITTING DENTURE INDUCED LESIONS :-
• The oral mucosa is subject to a variety of
injuries as a result of wearing of artificial
dentures. These may be manifested as
- Traumatic ulcer
- Cheek bite
-Inflammatory hyperplasia.
-Fibrous Papillary hyperplasia
-Chronic atropic candidiasis
(denture stomatitis)
-Contact allergy
-Malignancy
-Angular cheilitis
-Palatal perforation
5. 1.TRAUMATIC ULCER ( sore spots)
Clinical Features
1. Develops within a day or two after the insertion of a new denture.
2. Due to over-extension of the flanges, sequestration or spicules of
bone under the denture or roughened or “high” spot on the inner
surface of the denture.
3. These are small, painful, irregularly shaped lesions covered by a
delicate grey necrotic membrane and surrounded by an inflammatory
halo.
Management
1. Correction of underlying cause.
2. Relief of the flanges.
4. Relief of high spots
6. Cheek bite
• Causes:-
• Thin or overextended periphery (denture base material does
not provide enough support for the cheek)
• Loss of tone of cheek musculature in old patients
• Insufficient inter-arch clearance between distal part of
denture.
• Inadequate horizontal overjet in molar region .
(posterior edge-to-edge occlusion)
Treatment
• Build out thin areas, or extend the short periphery
• Trim maxillary denture buccal to tuberosity and/or from over
retromolar pad of mandibular denture
•Re-set teeth in correct relationship
• Recontour and polish buccal surface of mandibular posterior
teeth to create horizontal overjet
7. INFLAMMATORY HYPERPLASIA
The term “inflammatory hyperplasia” is used to describe a large
range of commonly occurring nodular growths of the oral
mucosa that histologically represent inflamed fibrous and
granulation tissues.
The major etiologic factor for these lesions is generally assumed
to be chronic trauma (such as that produced by ill-fitting
dentures, calculus, overhanging dental restorations, acute or
chronic tissue injury from biting, and fractured teeth), and
chronic irritants can be convincingly demonstrated in many
cases (eg, palatal papillary hyperplasia associated with aged
maxillary dentures).
With some of these lesions, (eg, pregnancy epulis and the central
giant cell tumor associated with hyperparathyroidism), the levels
of circulating hormones also undoubtedly play a role.
The majority of lesions occur on the surface of the oral mucous
membrane, where irritants are quite common.
8. If the chronic irritant is eliminated when the lesion is excised, the
majority of inflammatory hyperplasias will not recur.
This confirms the benign nature of these lesions (as would be
expected from their histologic structure).
The following are examples of inflammatory hyperplasia's:
fibrous inflammatory hyperplasias (clinical fibroma, epulis
fissuratum,
and pulp polyp)
palatal papillary hyperplasia
pyogenic granuloma
pregnancy epulis
Epulis granulomatosa
giant cell granuloma(giant cell epulis and central giant cell tumor
of the jaw)
pseudosarcomatous Fasciitis
proliferative myositis and
Pseudoepitheliomatous hyperplasia.
9. Fibrous Inflammatory Hyperplasias
and Traumatic Fibromas
Fibrous inflammatory hyperplasias may occur as either
pedunculated or sessile (broad-based) growths on any
surface of the oral mucous membrane.
They are called fibromas if they are sessile, firm, and
covered by thin squamous epithelium.
On the gingiva, a similar lesion is often referred to as
an epulis.
The majority remain small, and lesions that are > 1 cm
in diameter are rare.
10. An exception to this rule occurs with a lesion that is
associated with the periphery of illfitting dentures, the so-
called epulis fissuratum, in which the growth is often split
by the edge of the denture, one part of the lesion lying
under the denture and the other part lying between the lip
or cheek and the outer denture surface.
This lesion may extend the full length of one side of the
denture. Many such hyperplastic growths will become
less edematous and inflamed following the removal of the
associated chronic irritant, but they rarely resolve
entirely.
In the preparation of the mouth to receive dentures, these
lesions are excised to prevent further irritation and to
ensure a soft-tissue seal for the denture periphery.
11. The differential diagnosis of fibrous inflammatory
hyperplasia should include consideration of the
possibility that the lesion is a true papilloma (a
cauliflower-like mass made up of multiple fingerlike
projections of stratified squamous epithelium with a
central core of vascular connective tissue) or a small
verrucous carcinoma.
Fibrous inflammatory hyperplasias have no malignant
potential, and recurrences following excision are almost
always a result of the failure to eliminate the particular
form of chronic irritation involved.
12. The occasional report of squamous cell carcinoma
arising in an area of chronic denture irritation, however,
underlines the fact that no oral growth, even those
associated with an obvious chronic irritant, can be
assumed to be benign until proven so by histologic
study.
Thus,whenever possible, all fibrous inflammatory
hyperplasias of the oral cavity should be treated by local
excision, with microscopic examination of the excised
tissue.
13. EPULIS FISSURATUM ( inflammatory
Fibrous Hyperplassia, Denture Injury tumour,
Denture epulis.)
-It is a tumour like
hyperplasia of fibrous
connective tissue.
-Develops in association
with the flange of ill fitting
complete or partial
dentures.
14. CLINICAL FEATURES
-Appears as single or multiple folds of hyperplastic tissue
in the alveolar vestibule.
-Flange of denture fits in to the fissure between these
folds.
-Tissue is firm and fibrous.
-Some lesions appear erythematous and ulcerated.
-Size of lesion vary from localized hyperplasia's less than
1 cm in size to massive lesions involve the entire length
of vestibule.
-Anterior portion of jaws is affected more.
-Female predilection.
15. -Another lesion called fibro epithelial polyp or
leaf like denture fibroma occurs on hard
palate beneath maxillary denture.
-It is a flattened pink mass attached to palate
by narrow stalk.
-It can easily lifted up with a probe
demonstrate its pedunculated nature.
-Edge of lesion is serrated and resembles a
leaf.
-If minor salivary glands are included they
show chronic sialadenitis.
-Unusual appearing products known as
osseous and chondromatous metaplasia is a
reactive phenomenon caused by chronic
irritation by the ill fitting denture.
17. • The frequency of occurrence of epulis
fissuratum far exceeds that of any other
exophytic lesion at the periphery of dentures.
• However the possibility of malignancy must be
considered in each case: squamous and
verrucous carcinomas, minor salivary gland
tumours, metastatic tumours, osteosarcoma,
and down reaching maxillary sinus
malignancies.
18. PALATAL PAPILLARY HYPERPLASIA
Palatal papillary hyperplasia (denture papillomatosis) is a
common lesion with a characteristic clinical appearance that
develops on the hard palate in response to chronic denture
irritation in approximately 3 to 4% of denture wearers.
Full dentures in which relief areas or “suction chambers” are cut
in the palatal seating surface appear to be the strongest stimuli,
but the lesion is also seen under partial dentures, and occasional
case reports have described the lesion in patients who have
never worn dentures.
The palatal lesion is usually associated with some degree of
denture sore mouth (stomatitis) due to chronic candidal
infection, which influences the appearance of the papillary
hyperplasia.
19. When complicated by candidal infection, the lesion may
be red to scarlet, and the swollen and tightly packed
projections resemble the surface of an overripe berry.
Such lesions are friable, often bleed with minimal trauma,
and may be covered with a thin whitish exudate.
When the candidal infection is eliminated, either by
removing the denture or by topical administration of an
antifungal agent, the papillary lesion becomes little
different in color from the rest of the palate and consists
of more or less tightly packed nodular projections.
If tiny, the nodular projections simply give a feltlike
texture to that portion of the palate, and the lesion may
even pass unnoticed unless it is stroked with an
instrument or disturbed by a jet of air.
20. It was first classified by Newton (1962) according to its
clinical appearance as:
Type 1: A localized simple inflammation or pinpoint
hyperaemia
Type 2: An erythematous or generalized simple type seen as
more diffuse erythema involving a part or the entire denture
covered mucosa
Type 3: A granular type (inflammatory papillary hyperplasia)
commonly involving the central part of the hard palate and
the alveolar ridges
• Type III often is seen in association with type I or type II.
• Type III denture stomatitis involves the epithelial response
to chronic inflammatory stimulation secondary to yeast
21. Despite their sometimes bizarre clinical
appearance, these lesions have almost no
neoplastic potential, a finding that is borne
out by the absence of atypia and cellular
dysplasia in biopsy specimens.
If the alveolar ridges are surgically
prepared for new dentures, papillary
hyperplasia lesions are usually excised or
removed (by electrocautery, cryosurgery,
or laser surgery), and the old denture or a
palatal splint is used to maintain a
postoperative surgical dressing over the
denuded area.
22. If florid papillomatosis of the palate occurs
or persists in the absence of dentures, the
differential diagnosis should also consider
several granulomatous diseases that may
manifest intraorally in this fashion (eg,
infectious granulomas,Cowden disease,
and verrucous carcinoma), particularly
when the papillary lesions are white and
extend beyond the palatal vault and onto
the alveolar mucosa.
23. • Nicotine stomatitis may also feature multiple small
nodules on palate, which are reddish before
hyperkeratosis develop.
• The following observations help in the differentiation of
this condition from IPH;
1. Nicotine stomatitis on the hard palate occurs almost
exclusively in pipe smokers who do not wear full
maxillary dentures.
2. The pattern in nicotine stomatitis is linear and
angular, and the segments are flatter and broader but
less elevated.
3. The segments in nicotine stomatitis have a
characteristic red dot in their approximate center, which
is not seen in PHP.
24. DENTURE STOMATITIS(CHRONIC
ATROPHIC CANDIDIASIS)
Chronic atrophic candidiasis includes denture stomatitis
(denture sore mouth), angular cheilitis, and median rhomboid
glossitis.
Denture Stomatitis (Denture Sore Mouth)
Denture stomatitis is a common form of oral candidiasis that
manifests as a diffuse inflammation of the maxillary denture-
bearing areas and that is often (15 to 65% of cases) associated
with angular cheilitis.
At least 70% of individuals with clinical signs of denture
stomatitis exhibit fungal growth, and this condition most likely
results from yeast colonization of the oral mucosa, combined
with bacterial colonization.
25. Three progressive clinical stages of denture sore
mouth have been described;
The first stage consists of numerous palatal petechiae
The second stage displays a more diffuse erythema
involving most (if not all) of the denture- covered mucosa
The third stage includes the development of tissue
granulation or nodularity (papillary hyperplasia)
commonly involving the central areas of the hard palate
and alveolar ridges.
26. Antifungal treatment will modify the bright red
appearance of denture sore mouth and papillary
hyperplasia specifically but will not resolve the basic
papillomatous lesion, especially if the lesions have been
present for more than 1 year.
Antifungal therapy and cessation of denture wearing
usually is advisable before surgical excision since
elimination of the mucosal inflammation often reduces
the amount of tissue that needs to be excised.
Yeast attached to the denture plays an important etiologic
role in chronic atrophic candidiasis
27. The attachment of yeast to the patient’s appliances is
increased by mucus and serum and decreased by the
presence of salivary pellicle, suggesting an explanation
for the severity of candidiasis in xerostomic patients.
Rinsing the appliance with a dilute (10%) solution of
household bleach, soaking it in boric acid, or applying
nystatin cream before inserting the denture will eliminate
the yeast.
Disinfection of the appliance is an important part of the
treatment of denture sore mouth.
28. Soft liners in dentures provide a porous
surface and an opportunity for additional
mechanical locking of plaque and yeast to
the appliance.
In general, soft liners are considered to be
an additional hazard for patients who are
susceptible to oral candidiasis.
Denture sore mouth is rarely found under a
mandibular denture.
29. One possible explanation for this is that
the negative pressure that forms under the
maxillary denture excludes salivary
antibody from this region, and yeast may
reproduce, undisturbed, in the space
between the denture and mucosa.
The closer adaptation of the maxillary
denture and palate may also bring the large
number of yeasts adhering to the denture
surface into contact with the mucosa.
30. • The clinical picture of denture
stomatits is rather specific; few if
any other diseases appear the same.
• Infections by other organisms,
however could be responsible for a
similar diffuse redness either alone
or I association with candida.
31. • Contact allergy to denture base
occurs occasionally.
• In such cases redness will not be
restricted to tissue under the
denture, but all mucosal surfaces in
contact with the acrylic will be red.
• Epicutaneous tests of the material
will be diagnostic .
32.
33. ANGULAR CHEILITIS (perleche,
angular cheilosis.)
Inflammation of skin and mucous membrane at angle
of mouth.
Characterised by redness, fissuring, scaling or
crustation.
ETIOLOGY
1. Nutritional deficiency- Riboflavin, Folate, Iron and
protein deficiency.
2. Reduced vertical dimension in complete dentures.
3. Candida,Streptococcus, staphylococcus infection.
4. Manifestation of HIV/ AIDS.
5. Systemic condition: Diabetis, Neutropenia
34. CLINICAL FEATURES
-Occurs as an area of redness, linear deep
fissure, peeling of skin, dry crust at the
angle of mouth.
- Symptomatic.
-Pain and bleeding at site.
-Difficulty in mouth opening.
-Occurs bilaterally.
-RHAGADES :- linear furrow or fissures
radiating from the angle of mouth are seen
in severe forms, especially in denture
wearers.
35. MANAGEMENT
1. Removal of the cause.
2. Nutritional supplement:- Vitamin B and
Iron supplements.
3. Ketoconazole: 200mg tab once daily for
1-2 weeks or
Fluconazole: 100mg tab once daily for 1-2
weeks.
4. For severe candidiasis - Fluconazole is
drug of choice.
36. Palatal Perforation
Even though it is well known about the harmful effects of suction
disk use in complete dentures, they are
still popular in rural and semi urban areas of India as a cheap
means to achieve retention in maxillary dentures.
For retention suction cups produces negative
pressure on the mucosa which it contacts.
This negative pressure induced by them has a
destructive effect on the palatal tissues.
This negative pressure reduces the blood circu
lation in the underlying tissues , which causes hypoxia
in the affected area and necrosis of tissue.
The underlying bony part may also be affected by
this leading to tissue perforation
37. • These pathological changes are more severe if the
patient is in the habit of continuously wearing the
dentures 24 hrs a day.
TREATMENT:-
• surgically close the opening and allow complete
healing using a healing plate and followed by fabrication
of new complete denture.
38.
39. Precautions to be taken by the
Dentist to avoid these problems
• There should be adequate relief .
• There should be no overextension of the
lingual flange into the lateral throat form.
• The vertical dimension must be accurate.
• There shouldn’t be any deflective occlusal
contacts.
• The denture borders should not be
overextended.
• There should be sufficient horizontal overlap of
posterior teeth.
• Note that the borders are rounded and
smooth, and the palate is highly polished and
the proper thickness.
40. CONCLUSION
Proper denture care is important for both the
health of dentures and patients mouth.
Handle dentures with great care.
To avoid accidentally dropping them,
stand over a folded towel or a full
sink of water when handling
dentures.
41. Brush and rinse dentures daily:-
•Like natural teeth, dentures must be brushed daily to remove
food and plaque.
• Brushing also helps prevent the development of permanent
stains on the dentures.
•Use a brush with soft bristles that is specifically designed for
cleaning dentures.
•Avoid using a hard-bristled brush as it can damage or wear down
dentures.
•Gently brush all surfaces of the denture and be careful not to
damage the plastic or bend attachments.
• In between brushings, rinse dentures after every meal.
42. Denture care when not being worn:-
•Dentures need to be kept moist when not being worn so
they do not dry out or lose their shape.
•When not worn, dentures should be placed in a denture
cleanser soaking solution or in water.
•However, if the denture has metal attachments, the
attachments could tarnish if placed in a soaking solution.
•Dentures should never be placed in hot water, as it can
cause them to warp.
43. Clean with a denture cleaner:-
• Hand soap or mild dishwashing liquid can be used for
cleaning dentures.
• Household cleansers and many toothpastes may be too
abrasive for dentures and should not be used.
•Also, avoid using bleach, as this may whiten the pink
portion of the denture.
•Ultrasonic cleaners can be used to care for dentures.
•These cleaners are small bathtub-like devices that contain
a cleaning solution.
44. • The denture is immersed in the tub and then
sound waves create a wave motion that
dislodges the undesirable deposits.
• Use of an ultrasonic cleaner, however, does
not replace a thorough daily brushing.
• Products with the American Dental Association
(ADA) Seal of Acceptance are recommended
since they have been evaluated for safety and
effectiveness.
45. REFERENCES
• MARTIN S. GREENBERG,MICHAEL GLICK; Burket’s oral medicine
Diagnosis & Treatment Tenth Edition
• Norman.k Wood, Paul.W.Goaz; DIFFERENTIAL DIAGNOSIS of
ORAL and MAXILLOFACIAL LESIONS;5th
edition
• B. C. Webb,C. J. Thomas et al; Candida-associated denture
stomatitis . Aetiology and management : A review. Part 2 Oral
diseases caused by candida species Australian Dental Journal
1998;43:(3):160-6
• Tatiana PEREIRA-CENCI, Altair Antoninha DEL BEL CURY, Wim
CRIELAAR, Jacob Martien TEN CATE DEVELOPMENT OF
CANDIDA-ASSOCIATED DENTURE STOMATITIS: NEW INSIGHTS;
J Appl Oral Sci. 2008;16(2):86-94
• Hrizdana Hadjieva, Mariana Dimova, S. Todorov; STOMATITIS
PROSTHETICA-A POLYETIOLOGIC DISORDER; Journal of IMAB -
Annual Proceeding (Scientific Papers) 2006, book 2
• GAUTAM BAGCHI, DEVENDRA MANDHYAN AND DILIP KUMAR
NATH; REHABILITATION OF A PATIENT WITH PALATAL PERFORATION DUE TO
PROLONGED USE OF DENTURE WITH SUCTION DISK: A CASE REPORT; (JRAAS)
27:62-64, 2012