Dentist in pune. (BDS. MDS) - Dr. Amit T. Suryawanshi. Seminar-Canine Impaction.
Email ID- amitsuryawanshi999@gmail.com
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This seminar explains about the development, relations, ligaments, various attachments, vascular and nervous supply and various surgical approaches and its modifications to TMJ
This seminar explains about the development, relations, ligaments, various attachments, vascular and nervous supply and various surgical approaches and its modifications to TMJ
GIANT CELL LESIONS OF THE JAW
CONTENTS
INTRODUCTION
DEFINITION
CLASSIFICATION
CONCLUSION
REFERENCE
The term Giant cell is derived from a Latin word,” giges; huge and cella; storeroom.
It is defined as an abnormally large tissue cell which often contains more than one nucleus and sometimes may appear as a merger of several normal cells.
CLASSIFICATION OF GIANT CELL LESION
According To Paul Auclair et al
1. Entities in which giant cells are the predominant histologic finding and form the basis of their recognition:
Central giant cell granuloma.
Giant cell tumour of bone.
Aneurismal bone cyst.
Cherubism.
Brown tumour of hyperparathyroidism.
II. Lesions containing giant cells
I. Infectious diseases
Bacterial - Tuberculosis ,Leprosy ,Syphilis ,Actinomycosis ,Cat scratch disease
Viral -Herpes ,Measles
Mycotic -Histoplasmosis ,Blastomycosis
II .Inflammatory diseases of unknown origin
Wegener’s granulomatosis
III. Metabolic
Histiocytosis X
IV. Neoplastic
Benign - Giant cell fibroma ,Osteoblastoma
Malignant - Chondrosarcoma ,Hodgkin’s disease , Burkitt’s lymphoma.
LESIONS CONTAININGMULTINUCLEATED GIANT CELLS
Giant Cell Granuloma
Giant Cell Tumor
Hyperparathyroidism (HPT)
Cherubism
Aneurysmal Bone Cyst.
CENTRAL GIANT CELL GRANULOMA
Benign proliferation of fibroblasts and multinucleated giant cells
Clinical and Radiographic Features
most often found in children and young adults, with up to 75% of cases occurring before 30 years of age.
Females are affected twice as frequently as males.
Lesions are more common in the anterior portions of the jaws, and mandibular lesions frequently cross the midline.
Most giant cell granulomas of the jaws are asymptomatic and first come to attention during a routine radiographic examination or as a result of painless expansion of the affected bone.
A minority of cases, however, may be associated with pain, paresthesia, or perforation of the cortical bone plate, occasionally resulting in ulceration of the mucosal surface by the underlying lesion.
RADIOGRAPHIC FEATURES
appear as radiolucent defects, which may be unilocular or multilocular.
The defect is usually well delineated, but the margins are generally noncorticated .
The lesion may vary from a 5 X 5 mm incidental radiographic finding to a destructive lesion greater than 10 cm in size.
The radiographic findings are not specifically diagnostic.
Small unilocular lesions may be confused with periapical granulomas or cysts.
Multilocular giant cell lesions cannot be distinguished radiographically from ameloblastomas or other multilocular lesions.
Histopathologic Features
presence of few to many multinucleated giant cells in a background of ovoid to spindle shaped mesenchymal cells.
There is evidence that these giant cell s re resent osteoclasts, although others suggest the cells may be aligned more closely with macrophages.
The giant cells may be aggregated focally in the lesional tissue or may be present diffusely throughout the lesion.
Dr. Ahmed M. Adawy
Professor Emeritus, Dep. Oral & Maxillofacial Surg.
Former Dean, Faculty of Dental Medicine
Al-Azhar University
The condition of being prognathic indicates abnormal forward projection of one or of both jaws beyond the established normal relationship with the cranial base. The skeletal manifestation can be due to mandibular anterior positioning (prognathism) or growth excess (macrognathia), maxillary posterior positioning (retrognathism) or growth deficiency (micrognathia), or a combination of both. The prevalence of mandibular prognathism, the etiologic factors, evaluation of patients, and treatment modalities are presented.
The presentation deals with the basics required for studying TMJ ankylosis. The text has been simplified and presented. It is well supported with illustrations.
Suggestions and feedback will be well appreciated. :)
GIANT CELL LESIONS OF THE JAW
CONTENTS
INTRODUCTION
DEFINITION
CLASSIFICATION
CONCLUSION
REFERENCE
The term Giant cell is derived from a Latin word,” giges; huge and cella; storeroom.
It is defined as an abnormally large tissue cell which often contains more than one nucleus and sometimes may appear as a merger of several normal cells.
CLASSIFICATION OF GIANT CELL LESION
According To Paul Auclair et al
1. Entities in which giant cells are the predominant histologic finding and form the basis of their recognition:
Central giant cell granuloma.
Giant cell tumour of bone.
Aneurismal bone cyst.
Cherubism.
Brown tumour of hyperparathyroidism.
II. Lesions containing giant cells
I. Infectious diseases
Bacterial - Tuberculosis ,Leprosy ,Syphilis ,Actinomycosis ,Cat scratch disease
Viral -Herpes ,Measles
Mycotic -Histoplasmosis ,Blastomycosis
II .Inflammatory diseases of unknown origin
Wegener’s granulomatosis
III. Metabolic
Histiocytosis X
IV. Neoplastic
Benign - Giant cell fibroma ,Osteoblastoma
Malignant - Chondrosarcoma ,Hodgkin’s disease , Burkitt’s lymphoma.
LESIONS CONTAININGMULTINUCLEATED GIANT CELLS
Giant Cell Granuloma
Giant Cell Tumor
Hyperparathyroidism (HPT)
Cherubism
Aneurysmal Bone Cyst.
CENTRAL GIANT CELL GRANULOMA
Benign proliferation of fibroblasts and multinucleated giant cells
Clinical and Radiographic Features
most often found in children and young adults, with up to 75% of cases occurring before 30 years of age.
Females are affected twice as frequently as males.
Lesions are more common in the anterior portions of the jaws, and mandibular lesions frequently cross the midline.
Most giant cell granulomas of the jaws are asymptomatic and first come to attention during a routine radiographic examination or as a result of painless expansion of the affected bone.
A minority of cases, however, may be associated with pain, paresthesia, or perforation of the cortical bone plate, occasionally resulting in ulceration of the mucosal surface by the underlying lesion.
RADIOGRAPHIC FEATURES
appear as radiolucent defects, which may be unilocular or multilocular.
The defect is usually well delineated, but the margins are generally noncorticated .
The lesion may vary from a 5 X 5 mm incidental radiographic finding to a destructive lesion greater than 10 cm in size.
The radiographic findings are not specifically diagnostic.
Small unilocular lesions may be confused with periapical granulomas or cysts.
Multilocular giant cell lesions cannot be distinguished radiographically from ameloblastomas or other multilocular lesions.
Histopathologic Features
presence of few to many multinucleated giant cells in a background of ovoid to spindle shaped mesenchymal cells.
There is evidence that these giant cell s re resent osteoclasts, although others suggest the cells may be aligned more closely with macrophages.
The giant cells may be aggregated focally in the lesional tissue or may be present diffusely throughout the lesion.
Dr. Ahmed M. Adawy
Professor Emeritus, Dep. Oral & Maxillofacial Surg.
Former Dean, Faculty of Dental Medicine
Al-Azhar University
The condition of being prognathic indicates abnormal forward projection of one or of both jaws beyond the established normal relationship with the cranial base. The skeletal manifestation can be due to mandibular anterior positioning (prognathism) or growth excess (macrognathia), maxillary posterior positioning (retrognathism) or growth deficiency (micrognathia), or a combination of both. The prevalence of mandibular prognathism, the etiologic factors, evaluation of patients, and treatment modalities are presented.
The presentation deals with the basics required for studying TMJ ankylosis. The text has been simplified and presented. It is well supported with illustrations.
Suggestions and feedback will be well appreciated. :)
A seminar prepared during my omfs posting hours. Short points are added for easiness to study and bihart. Reference taken from Balaji and Neelima Anil Malik
Le fort fracture by Dr. Amit T. Suryawanshi, Oral Surgeon, Pune All Good Things
Hi. This is Dr. Amit T. Suryawanshi. Oral & Maxillofacial surgeon from Pune, India. I am here on slideshare.com to share some of my own presentations presented at various levels in the field of OMFS. Hope this would somehow be helpful to you making your presentations. All the best.
Le fort fracture by Dr. Amit Suryawanshi .Dentist in Kolhapur (MDS). Oral &...All Good Things
Description:
Hi. This is Dr. Amit T. Suryawanshi. Dentist in Kolhapur (MDS) Oral & Maxillofacial surgeon from Kolhapur, India. I am here on slideshare.com to share some of my own presentations presented at various levels in the field of OMFS. Hope this would somehow be helpful to you making your presentations. All the best & your replies are welcomed!
Its a Clinical Presentation of Midface fractures-specifically, Lefort fractures. Classification, Anatomical Landmarks, Clinical Features, Diagnosis & Management protocols are discussed.
Explore natural remedies for syphilis treatment in Singapore. Discover alternative therapies, herbal remedies, and lifestyle changes that may complement conventional treatments. Learn about holistic approaches to managing syphilis symptoms and supporting overall health.
The prostate is an exocrine gland of the male mammalian reproductive system
It is a walnut-sized gland that forms part of the male reproductive system and is located in front of the rectum and just below the urinary bladder
Function is to store and secrete a clear, slightly alkaline fluid that constitutes 10-30% of the volume of the seminal fluid that along with the spermatozoa, constitutes semen
A healthy human prostate measures (4cm-vertical, by 3cm-horizontal, 2cm ant-post ).
It surrounds the urethra just below the urinary bladder. It has anterior, median, posterior and two lateral lobes
It’s work is regulated by androgens which are responsible for male sex characteristics
Generalised disease of the prostate due to hormonal derangement which leads to non malignant enlargement of the gland (increase in the number of epithelial cells and stromal tissue)to cause compression of the urethra leading to symptoms (LUTS
micro teaching on communication m.sc nursing.pdfAnurag Sharma
Microteaching is a unique model of practice teaching. It is a viable instrument for the. desired change in the teaching behavior or the behavior potential which, in specified types of real. classroom situations, tends to facilitate the achievement of specified types of objectives.
Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
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Lung Cancer: Artificial Intelligence, Synergetics, Complex System Analysis, S...Oleg Kshivets
RESULTS: Overall life span (LS) was 2252.1±1742.5 days and cumulative 5-year survival (5YS) reached 73.2%, 10 years – 64.8%, 20 years – 42.5%. 513 LCP lived more than 5 years (LS=3124.6±1525.6 days), 148 LCP – more than 10 years (LS=5054.4±1504.1 days).199 LCP died because of LC (LS=562.7±374.5 days). 5YS of LCP after bi/lobectomies was significantly superior in comparison with LCP after pneumonectomies (78.1% vs.63.7%, P=0.00001 by log-rank test). AT significantly improved 5YS (66.3% vs. 34.8%) (P=0.00000 by log-rank test) only for LCP with N1-2. Cox modeling displayed that 5YS of LCP significantly depended on: phase transition (PT) early-invasive LC in terms of synergetics, PT N0—N12, cell ratio factors (ratio between cancer cells- CC and blood cells subpopulations), G1-3, histology, glucose, AT, blood cell circuit, prothrombin index, heparin tolerance, recalcification time (P=0.000-0.038). Neural networks, genetic algorithm selection and bootstrap simulation revealed relationships between 5YS and PT early-invasive LC (rank=1), PT N0—N12 (rank=2), thrombocytes/CC (3), erythrocytes/CC (4), eosinophils/CC (5), healthy cells/CC (6), lymphocytes/CC (7), segmented neutrophils/CC (8), stick neutrophils/CC (9), monocytes/CC (10); leucocytes/CC (11). Correct prediction of 5YS was 100% by neural networks computing (area under ROC curve=1.0; error=0.0).
CONCLUSIONS: 5YS of LCP after radical procedures significantly depended on: 1) PT early-invasive cancer; 2) PT N0--N12; 3) cell ratio factors; 4) blood cell circuit; 5) biochemical factors; 6) hemostasis system; 7) AT; 8) LC characteristics; 9) LC cell dynamics; 10) surgery type: lobectomy/pneumonectomy; 11) anthropometric data. Optimal diagnosis and treatment strategies for LC are: 1) screening and early detection of LC; 2) availability of experienced thoracic surgeons because of complexity of radical procedures; 3) aggressive en block surgery and adequate lymph node dissection for completeness; 4) precise prediction; 5) adjuvant chemoimmunoradiotherapy for LCP with unfavorable prognosis.
These lecture slides, by Dr Sidra Arshad, offer a quick overview of physiological basis of a normal electrocardiogram.
Learning objectives:
1. Define an electrocardiogram (ECG) and electrocardiography
2. Describe how dipoles generated by the heart produce the waveforms of the ECG
3. Describe the components of a normal electrocardiogram of a typical bipolar leads (limb II)
4. Differentiate between intervals and segments
5. Enlist some common indications for obtaining an ECG
Study Resources:
1. Chapter 11, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 9, Human Physiology - From Cells to Systems, Lauralee Sherwood, 9th edition
3. Chapter 29, Ganong’s Review of Medical Physiology, 26th edition
4. Electrocardiogram, StatPearls - https://www.ncbi.nlm.nih.gov/books/NBK549803/
5. ECG in Medical Practice by ABM Abdullah, 4th edition
6. ECG Basics, http://www.nataliescasebook.com/tag/e-c-g-basics
New Directions in Targeted Therapeutic Approaches for Older Adults With Mantl...i3 Health
i3 Health is pleased to make the speaker slides from this activity available for use as a non-accredited self-study or teaching resource.
This slide deck presented by Dr. Kami Maddocks, Professor-Clinical in the Division of Hematology and
Associate Division Director for Ambulatory Operations
The Ohio State University Comprehensive Cancer Center, will provide insight into new directions in targeted therapeutic approaches for older adults with mantle cell lymphoma.
STATEMENT OF NEED
Mantle cell lymphoma (MCL) is a rare, aggressive B-cell non-Hodgkin lymphoma (NHL) accounting for 5% to 7% of all lymphomas. Its prognosis ranges from indolent disease that does not require treatment for years to very aggressive disease, which is associated with poor survival (Silkenstedt et al, 2021). Typically, MCL is diagnosed at advanced stage and in older patients who cannot tolerate intensive therapy (NCCN, 2022). Although recent advances have slightly increased remission rates, recurrence and relapse remain very common, leading to a median overall survival between 3 and 6 years (LLS, 2021). Though there are several effective options, progress is still needed towards establishing an accepted frontline approach for MCL (Castellino et al, 2022). Treatment selection and management of MCL are complicated by the heterogeneity of prognosis, advanced age and comorbidities of patients, and lack of an established standard approach for treatment, making it vital that clinicians be familiar with the latest research and advances in this area. In this activity chaired by Michael Wang, MD, Professor in the Department of Lymphoma & Myeloma at MD Anderson Cancer Center, expert faculty will discuss prognostic factors informing treatment, the promising results of recent trials in new therapeutic approaches, and the implications of treatment resistance in therapeutic selection for MCL.
Target Audience
Hematology/oncology fellows, attending faculty, and other health care professionals involved in the treatment of patients with mantle cell lymphoma (MCL).
Learning Objectives
1.) Identify clinical and biological prognostic factors that can guide treatment decision making for older adults with MCL
2.) Evaluate emerging data on targeted therapeutic approaches for treatment-naive and relapsed/refractory MCL and their applicability to older adults
3.) Assess mechanisms of resistance to targeted therapies for MCL and their implications for treatment selection
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- Video recording of this lecture in English language: https://youtu.be/lK81BzxMqdo
- Video recording of this lecture in Arabic language: https://youtu.be/Ve4P0COk9OI
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MANAGEMENT OF ATRIOVENTRICULAR CONDUCTION BLOCK.pdfJim Jacob Roy
Cardiac conduction defects can occur due to various causes.
Atrioventricular conduction blocks ( AV blocks ) are classified into 3 types.
This document describes the acute management of AV block.
Prix Galien International 2024 Forum ProgramLevi Shapiro
June 20, 2024, Prix Galien International and Jerusalem Ethics Forum in ROME. Detailed agenda including panels:
- ADVANCES IN CARDIOLOGY: A NEW PARADIGM IS COMING
- WOMEN’S HEALTH: FERTILITY PRESERVATION
- WHAT’S NEW IN THE TREATMENT OF INFECTIOUS,
ONCOLOGICAL AND INFLAMMATORY SKIN DISEASES?
- ARTIFICIAL INTELLIGENCE AND ETHICS
- GENE THERAPY
- BEYOND BORDERS: GLOBAL INITIATIVES FOR DEMOCRATIZING LIFE SCIENCE TECHNOLOGIES AND PROMOTING ACCESS TO HEALTHCARE
- ETHICAL CHALLENGES IN LIFE SCIENCES
- Prix Galien International Awards Ceremony
Dentist in pune.(BDS. MDS) - Dr. Amit T. Suryawanshi..Zygomaticomaxillary complex fractures
1. ZYGOMATICOMAXILLARY
(ZMC) COMPLEX
FRACTURES
Dr. Amit T. Suryawanshi
Dentist, Oral and Maxillofacial Surgeon
Pune, India
Contact details :
Email ID - amitsuryawanshi999@gmail.com
Mobile No - 9405622455
3. INTRODUCTION
• Most common facial fractures, the second in
frequency after nasal fractures.
• The high incidence relates to the zygoma’s
prominent position within the facial skeleton.
• Male predilection, 4:1 over females.
• Peak incidence- 2nd and 3rd decades of life.
4. INTRODUCTION
• Zygomatic injury is mostly due to altercations
followed by motor vehicle accidents.
• In zygomatic fractures caused by altercations,
the left zygoma is most commonly affected.
• The zygoma plays an important role in facial
contour. Disruption of the zygomatic position
creates impairment of ocular and mandibular
function.
5. INTRODUCTION
• The zygoma or malar complex forms the
central support of the cheek and is a strong
buttress of the lateral and middle third of the
facial skeleton.
• It is for this reason, the zygoma is frequently
fractured, either alone or in combination with
other bony structures of the midface.
6. ANATOMY
• The zygoma, a major
buttress of the facial
skeleton, is the principal
structure of lateral
midface.
7. ANATOMY
• Zygoma is roughly quadrilateral in shape, with
an outer convex (cheek) surface and an inner
concave (temporal) surface.
• It forms the point of greatest prominence of
the cheek.
• Resembles a four sided pyramid, which has
temporal, orbital, maxillary and frontal
processes.
8. ANATOMY
• Zygoma articulates with four bones- the
frontal, sphenoid, maxillary and temporal.
• Body of the zygoma extensively articulates
with the maxilla along the anterior maxilla and
along the orbital floor.
• It forms the superolateral aspect and part of
the superoanterior aspect of the maxillary
sinus.
10. ANATOMY
• The frontal process is thick and triangular in
cross section, with facial, orbital and temporal
surfaces.
• The temporal process is flat and projects
posteriorly to articulate with the zygomatic
process of the temporal bone.
• The combination of the two makes up the
zygomatic arch.
11. ANATOMY
• The zygomaticotemporal articulation is a very
thin, delicate connection, which fractures
frequently.
• The zygoma provides origin to a major portion
of the masseter muscle along the body and
temporal surface.
• The temporal fascia also attaches along the
arch and the temporal process.
13. SURGICAL ANATOMY
‘the malar bone represents a strong bone on
fragile supports, and it is for this reason that,
though the bone is rarely broken, the four
processes – frontal, orbital, maxillary and
zygomatic – are frequent sites of fracture.’
-- H.D.Gillies, T.P. Kilner and D. Stone, 1927
14. SURGICAL ANATOMY
• 2 zygomatic bones &
their temporal processes
• Zygomatic processes of
temporal bones
Form part of the middle
third of the facial
skeleton which may be
fractured following
trauma.
15. SURGICAL ANATOMY
• Fractures involving the orbit may give rise to
alteration in the position of the globe of the
eye.
• The level of the globe is normally maintained
by the suspensory ligament of Lockwood
which passes from its medial attachment on
the lacrimal bone to be inserted laterally into
the Whitnall’s tubercle.
16. SURGICAL ANATOMY
• Zygomatic and Le fort III fractures commonly
result in drop in the level of the globe of the
eye.
• As the globe of the eye drops, the upper
eyelid follows downwards giving rise to the
physical sign ‘hooding of the eye’.
17. FRACTURE PATTERNS
• The fracture pattern of any bone depends
mainly on the direction and magnitude of the
force.
• The fracture lines pass through the areas of
greatest weakness of a bone or between
bones.
20. FRACTURE PATTERNS
• The inferior orbital fissure is the key to
remember the usual lines of ZMC fractures.
• Three fracture lines extend from the inferior
orbital fissure in an anteromedial, a
superolateral, and an inferior direction.
21. FRACTURE PATTERNS
1. One fracture extends from the inferior orbital
fissure anteromedially along the orbital floor
mostly through the orbital process of the
maxilla.
22. FRACTURE PATTERNS
2. Second line of fracture from the inferior
orbital fissure runs inferiorly through the
posterior(infratemporal) aspect of maxilla
and joins the fracture from the anterior
aspect of maxilla, under the zygomatico
maxillary buttress.
23. FRACTURE PATTERNS
3. Third line of fracture extends superiorly
from the inferior orbital fissure along the
lateral orbital wall posterior to the rim,
usually separating the zygomaticosphenoid
suture.
24. CLASSIFICATION
• Knight and North (1961)
• Rowe and Killey (1968)
• Yanagisawa ( 1973)
• Larsen and Thomson (1978)
• Rowe and Williams (1985)
• Poswillo (1988)
25. Knight and North (1961)
Based on the direction of displacement on a
Water’s view radiograph,
• Group I – Non displaced fractures
• Group II – Arch fractures
• Group III – Unrotated fractures
• Group IV – Medially rotated fractures
• Group V - Laterally rotated fractures
• Group VI – Complex fractures
27. ROWE AND KILLEY (1968)
• Type I – no significant displacement
• Type II – fracture of the arch
• Type III – rotation around a vertical axis
• Type IV – rotation around a longitudinal axis
• Type V – displacement of the complex en bloc
• Type VI – displacement of orbito-antral partition
• Type VII – displacement of the orbital rim
segments
• Type VIII – Complex comminuted fractures
28. Yanagisawa ( 1973)
• GROUPS I & II - Unchanged
• GROUP III - Medial or lateral rotation
around a vertical axis
• GROUP IV - Medial or lateral rotation
around a longitudinal axis
• GROUP V - Medial or lateral
displacement without rotation
• GROUP VI - Isolated arch fracture
• GROUP VII - All complex fractures
29. Larsen and Thomson (1978)
• GROUP I – Non displaced fractures
requiring no treatment
• GROUP II – All fractures requiring
treatment
30. ROWE AND WILLIAMS (1985)
• Fractures stable after elevation
a) Arch only (medially displaced)
b) Rotation around the vertical axis
i) medially
ii) laterally
• Fractures unstable after elevation
a) Arch only (inferiorly displaced)
b) Rotation around horizontal axis
i) medially
ii) laterally
c) Dislocations en bloc
i) inferiorly
ii) medially
iii) postero - laterally
d) Communited fractures
32. POSWILLO’S CLASIFICATION
• Inward and downward
displacement
• Inward and posterior
displacement
• Outward displacement
of the zygomatic complex
• Communition
• Fracture of the arch alone
33. SIGNS & SYMPTOMS
• Periorbital Ecchymosis and Edema
• Flattening of the malar prominence
• Flattening over the zygomatic arch
• Pain
34. SIGNS & SYMPTOMS
• Ecchymosis of the maxillary buccal sulcus
• Deformity at the zygomatic buttress of
the maxilla
• Deformity of the orbital margin
• Trismus
35. SIGNS & SYMPTOMS
• Abnormal nerve sensibility
• Epistaxis
• Subconjunctival Ecchymoses
• Crepitation from air emphysema
36. SIGNS & SYMPTOMS
• Displacement of the palpebral fissure
• Unequal pupillary levels
• Diplopia
• Enophthalmos
37. RADIOLOGIC EXAMINATION
• Plain films and Computed Tomography have
their place in determining the type, location,
magnitude, and direction of displacement of
zygomatic fractures.
• This includes,
Water’s view, Submentovertex view,
Computed Tomography.
38. RADIOLOGIC EXAMINATION
• A single Water’s view is an important adjunct
to clinical examination.
• If fractures are noted, CT should be the
procedure of choice.
• Two dimensional CT is now considered the
best and most useful means of radiologic
assessment of the facial skeleton.
39. RADIOLOGIC EXAMINATION
• CT scans allow complete assessment of the
orbital floor and walls.
• For ZMC injuries, it is optional to have both
axial & coronal high resolution scans.
• The axial scan is helpful in evaluating the
medial and lateral orbital walls, and the
coronal scan defines the extent of injury to the
orbital floor.
49. IN THE LAST PART WE DISCUSSED….
• INTRODUCTION
• ANATOMY (SURGICAL ANATOMY)
• CLASSIFICATION
• SIGNS & SYMPTOMS
• CLINICAL EXAMINATION
• RADIOLOGIC EXAMINATION
51. TREATMENT
• Historical review:
• Various authors have given various treatment
modalities and techniques for the
management of ZMC fractures.
• Dating back to 1751, when Duverney stressed
the role of contraction of temporal muscle in
realigning the medial displacement of the
zygomatic arch.
52. HISTORICAL REVIEW
• Ferrier in 1825, attempted to reduce fracture
of zygomatic arch through an incision above
the arch.
• Dupuytren in 1847, discovered the important
relationship of the temporal fascia and the
muscle as a pathway to the zygomatic arch.
53. HISTORICAL REVIEW
• Gillies in 1927 emphasised the cosmetic value
of placing the incision within the hair line.
• Stroymeyer in 1844 described the
percuteneous hook technique.
• Cheyne and Burghard in 1901 discussed the
intraoral digital manipulation technique.
• Smith and Yanagisawa in 1961 stressed the
importance of cosmetic aspects of the
treatment.
54. GENERAL PRINCIPLES OF TREATMENT
• No treatment
• Indirect reduction with,
a. No fixation
b. Temporary support
c. Direct fixation
d. Indirect fixation
• Direct reduction and fixation
55. NO TREATMENT
• Cases with a minimal degree of displacement,
which following union, are considered unlikely
to result any cosmetic deformity, disturbance
of vision, persistent paraesthesia or
impairment of mandibular movement.
56. INDIRECT REDUCTION
• NO FIXATION:
• Includes procedures which do not involve
exposure of the fracture sites.
• The principle is to disimpact and reduce the
fracture by direct application of an
instrument, through an indirect approach
remote from the fracture line.
57. NO FIXATION
• The techniques which have been developed
for this operative approach, are based upon
the introduction of an instrument through,
a. the temporal fossa,
b. the upper buccal sulcus (intraoral),
c. the cheek (percutaneous),
d. the nose (transantral)
e. the eyebrow (lateral brow)
58. NO FIXATION
• Temporal fossa approach:
• This method was introduced by Gillies et al
(1927) for elevation of the zygomatic arch.
• Incision of about 2.5 cm long, made above
and parallel to the anterior branch of the
temporal artery.
59.
60.
61. NO FIXATION
• Lateral brow approach: (Dingman & Natwig
1964)
• The advantage of this technique is that the
fracture at the orbital rim is visualized directly.
• The frontozygomatic area of the lateral orbital
rim is exposed by the eyebrow incision.
• The instrument is inserted to lift the zygoma
anteriorly, laterally and superiorly.
63. NO FIXATION
• Upper buccal sulcus:
• The advantages of this technique have been
discussed by Balasubramaniam (1967) who
considers that “ less force is required by the
intraoral approach than by the extraoral,
because the force is exerted where it should
be, i.e., more at the centre of the fractured
fragment”.
64. NO FIXATION
• Upper buccal sulcus: (Keen’s approach)
• Access is gained by an incision of about 1cm in
length at the reflection of the upper buccal
sulcus immediately behind the zygomatic
buttress.
65. NO FIXATION
• Quinn in 1977 described a modification.
• This employs a lateral coronoid approach
through an incision situated over the anterior
border of ramus.
66.
67. NO FIXATION
• Percutaneous approach: (Stroymeyer 1844)
• This method consists of inserting a hook
through the skin below and behind the
zygomatic bone so that it engages the deep
aspect and allows reduction by strong
outward traction on the handle of the
instrument.
68. NO FIXATION
• Percutaneous approach:
• Poswillo advises that the exact location of the
initial stab wound for insertion is found at the
intersection of a perpendicular line dropped
from the outer canthus of the eye and a
horizontal line extended posteriorly from the
alar margin of the nostril.
69.
70. NO FIXATION
• Intranasal transantral approach: (Lothrop’s
approach 1906)
• Not common in use.
• An opening is made into the antrum below
the inferior meatus, and a curved urethral
sound introduced and manipulated so that its
tip lies on the antral aspect of the zygomatic
bone. Firm outward and upward pressure is
applied to reposition the bone.
71. TEMPORARY SUPPORT
• This may be indicated, as a supplementary
measure, under the following circumstances:
• When the zygomatic complex is unstable
following reduction,
• When there is gross comminution of the
zygomatic bone.
• When there is comminution without bone loss
of the orbital floor.
72. TEMPORARY SUPPORT
• Instability following adequate reduction could
be due to:
• Rupture of the enveloping periosteum or
attached temporal fascia.
• Comminution of the zygomatic arch.
• Loss of bone from around the zygomatic
buttress.
• Residual fibrosis when treatment is delayed.
73. TEMPORARY SUPPORT
• Temporary support is a concept which is
primarily based upon the introduction of a
pack or other material into the antrum so as
to exert counter-pressure against those forces
which tend to bring about a relapse of the
position achieved by indirect reduction.
74. TEMPORARY SUPPORT
• Since the pack will bring about repositioning
of fragments by pressure from their antral
aspect it will be evident that this selective
effect can only take place if there is absolute
stability of the remainder of the antrum and
the other elements of the zygomatic complex.
75. DIRECT FIXATION
• Indirect reduction, combined with direct
fixation following exposure of the fracture
site, provides an excellent method of
treatment.
• Direct fixation is needed when the fractures
remain unstable after indirect reduction.
76. DIRECT FIXATION
• Transosseous wiring or osteosynthesis:
• Separation at the frontozygomatic suture line
with displacement in excess of 2-3 mm, is
likely to cause detachment of the periosteal
and fascial attachments, and hence the direct
fixation becomes necessary.
77. DIRECT FIXATION
• Incisions on the face should be placed parallel
to or within the skin creases.
• It is preferable to incise the skin through the
outer end of the eyebrow. The incision should
not be at right angles to the skin, but directed
downwards at the same angle as the emerging
hairs.
78. DIRECT FIXATION
• The Gillies temporal approach is preferable if
there is separation at the suture.
• A percutaneous repositioning may be
preferred.
• Access to the temporal aspect of the
zygomatic bone can be obtained through the
frontozygomatic incision by passing a curved
elevator supraperiosteally posterior to the
frontal process of the zygomatic bone.
79. DIRECT FIXATION
• Dingman and Natvig (1964) recommend that
the holes are drilled in an anteroposterior
direction and when the external angular
process is well formed. It also enables the
wires to be placed in a figure of eight pattern
which provides better lateral stability.
80. DIRECT FIXATION
• Elevation of the zygomatic bone and
transosseous wiring or boneplating at the
frontozygomatic suture will achieve a stable
realignment of the orbital rim.
81. DIRECT FIXATION
• A more severe displacement, a dislocation of
the zygomatic complex en bloc, will result in
separation of the fracture ends at the inferior
orbital margin, with the fracture passing into
the orbital floor.
• In this type of unstable fracture it is essential
to carry out an osteosynthesis (transosseous
wiring) or microplating technique.
83. DIRECT FIXATION
• Micro-plates positioned in such a way that the
screw holes are situated well away from the
fracture sites provides a very useful
alternative to wires.
• The use of malleable micro-plating equipment
has greatly improved the management of such
cases.
84. DIRECT FIXATION
• Application of a small plate, across the fronto-zygomatic
suture will usually ensure that there
is absolute immobility, so that union can take
place. Precision alignment of the inferior
orbital margin is essential, open reduction will
be indicated.
85. DIRECT FIXATION
• Inadequate immobilisation of the fronto-zygomatic
suture where the lateral orbital wall
is displaced may lead to a loss of malar
prominence.
86. INDIRECT FIXATION
• Indirect fixation implies that the zygomatic
bone will be rigidly secured to some point
elsewhere on the facial skeleton until union
occurs.
• The required degree of firmness can only be
achieved by means of internal
(intramedullary) pins or wires or external pins
and rods which are linked together.
87. INDIRECT FIXATION
• The indirect fixation can be achieved by the
following methods:
1. Zygomatico-zygomatic (Trans-maxillary)
2. Naso-zygomatic
3. Zygomatico-palatal
4. Maxillo-zygomatic
5. Fronto-zygomatic
6. Cranio-zygomatic
88. INDIRECT FIXATION
• Indirect fixation has only limited application at
the present time in view of the greater
efficiency and comfort obtained by internal
fixation techniques.
89. COMPLICATIONS
• Infraorbital nerve disorders
• Implant extrusion, displacement and infection
• Maxillary sinusitis
• Persistent diplopia
• Enophthalmos
• Ankylosis of zygoma to coronoid process
• Malunion of the zygoma
90. CONCLUSION
• Thus, the zygomatic complex fractures are
common injuries, second in frequency after the
nasal bone fractures.
• There being a wide range of treatment modalities
and techniques for the management of
zygomaticomaxillary complex fractures.
• It is the apt judgement and knowledge of the
surgical anatomy on the part of the surgeon
enabling him to effectively manage the ZMC
fractures with the desired outcome.
91. REFERENCES
• Row and williams volume -1
• Fonseca trauma volume -2
• Peter wardbooth
• Peterson’s oral and maxillofacial surgery.
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Editor's Notes
Zygomatic or malar fractures are the terms commonly used to describe fractures that involve the lateral one third of the middle face.
Because of the impure nature of zygomatic fractures, other terms like zygomaticomaxillary complex, zygomaticomaxillary compound, zygomaticoorbital, zygomatic complex, malar, trimalar and tripod fractures have been adopted..
The later two terms are misnomers since the zygoma has not three but four processes.
The zygoma also has a narrow, weak articulation with the zygomatic crest of the greater wing of the sphenoid bone at the lateral aspect of the inf. Orbital fissure.
It forms a major portion of the lateral aspect of the floor of the orbit.
The anatomic position of the zygoma.
Lateral skull demonstrating its articulation with the temporal, frontal and maxillary bones.
Because of its thickness, the frontal process is a frequent site for wire or bone plate fixation following fracture.
The zygoma also provides attachments for the temporal and zygomatic muscles.
the strong infraorbital and lateral orbital rim provides protection to the orbital contents.
Owing to the strong buttressing nature of the zygoma and the thin bones surrounding it, most injuries involving the zygoma are accompanied by disruption of the adjacent articulating bones.
This disruption occurs because when a force is applied to the body of the zygoma, it is distributed through its four processes to the articulating adjacent bones, which are weaker than the zygoma.
Common fracture pattern in zmc injury.
Frontal view of skull showing fracture medial to zygomaticomaxillary suture and along the zygomaticosphenoid suture within the orbit.
Oblique frontal view of skull showing fractures through frontozygomatic suture and posterior to zygomaticotemporal suture.
C. Temporal view of skull showing fractures extending from the inferior orbital fissure both superiorly through the zygomaticosphenoid suture and inferiorly through the zygomatic buttress of the maxilla.
D. Inferior view of the skull showing triple fracture of the zygomatic arch.
To be noted, the orbital floor, medial orbital wall, and zygomaticomaxillary buttress are frequently comminuted in addition to all these fracture patterns.
The orbital floor and medial wall are often comminuted, creating multiple lines of fracture within the internal orbit.
The fracture frequently extends through the infraorbital rim to the facial surface of the maxilla above or even slightly medial to the infraorbital foramen.
The fracture extends from the infraorbital rim in the maxilla laterally and inferiorly under the zygomatic buttress of the maxilla.
Comminution of the infraorbital rim and bone along the anterior and lateral maxilla is common, with frequent involvement of the infraorbital foramen.
Extending superiorly, laterally, and anteriorly toward the lateral orbital rim, the fracture frequently separates the FZ suture at the lateral orbital rim.
A ZMC fracture which follows this pattern usually has one additional fracture line through the zygomatic arch.
However the variability of these fractures is great, owing to the difference in magnitude and direction of force, the amount of soft tissue covering the zygoma, and the density of the adjacent bones.
Knight and North in 1961 classified ZMC #, after observing fracture patterns on water’s view radiographs.
Rowe and killey thought that it is insufficient to classify ZMC fractures on a Water’s view alone, and recognized that the displacement of zygomatic bone might be a consequence of axial rotation around a vertical axis or en bloc displacement.
Yanagisawa in 1973 described the zmc fractures with respect to vertical and longitudinal axes, and they maintained that it is not enough to classify ZMC fractures around one axis, i.e., only vertical axis as described by Row and Killey.
Larsen and Thompson in 1978 considered stability of the fractures as a criteria while classifying them into unstable and stable fractures.
Stable or non displaced fractures requiring no treatment and Unstable or displaced fractures requiring treatment.
The most commonly accepted classification is by Rowe and Williams. They considered stability of the fractures from the treatment point of view as the criteria for classification, and classified ZMC fractures as fractures stable after elevation and fractures unstable after elevation.
Poswillo described zmc fractures as per their displacement in different directions.
Today the most favored classifications for ZMC fractures are Rowe and Williams classification and Knight & North classification.
Edema and bleeding into the loose connective tissue of the eyelids and periorbital areas. Swelling may be present in the periorbital tissue, where the eyelids may be swollen closed.
Flattening of the normal prominence in the malar area is striking. It is characteristic when there’s distraction of the frontozygomatic suture and medial rotation and/or comminution.
A characteristic indentation or loss of the normal convex curvature in the temporal area accompanies fractures of the zygomatic arch.
Severe pain is generally not a feature unless the fractured segment is mobile. Patient complains of discomfort. Palpation of the fracture site elicits a painful response.
Ecchymosis is an important sign and may occur even with a small disruption of the anterior or lateral maxilla.
Intraoral palpation of the anterior and lateral aspect of the maxilla reveals irregularities of the normally smooth contour, in the zygomatic buttress area. Crepitation from comminuted fragments of bone can also be elicited.
Fractures running through the orbital rim often result in a gap or step deformity, frequently noted in the infraorbital and lateral orbital rims when ZMC # are present.
Trismus is especially higher in isolated fractures of the zygomatic arch. Postfracture trismus is due to impingement of the translating coronoid process of the mandible on the displaced zygomatic fragments. Also due to muscle spasm secondary to impingement by the displaced fragments, esp on temporal muscle.
Mostly due to impaired sensation of the infraorbital nerve. Infraorbital nerve paresthesia is more common in fractures that are displaced. Infraorbital anesthesia occurs when the fracture through the orbital floor/ or the anterior maxilla causes tearing, shearing or compression of the infraorbital nerve.
Whenever sinus mucosa is disrupted, hemorrhage into the sinus is possible. The maxillary sinus drains into the nose via middle meatus, unilateral hemorrhage from the nose is possible.
Subconjuctival ecchymoses may accompany even a hairline crack through the orbital rim if the periosteum has been torn. Subconjunctival ecchymoses usually have no posterior limit and will be bright red owing to the ability of the oxygen to diffuse through the conjunctiva to the collection of blood.
Fracture through a sinus wall with tearing of the lining mucosa allows air to escape into the facial soft tissue if the pressure within the sinus is greater than that within the tissue. When inflation of air occurs, one can palpate crepitation, indicating subcutaneous emphysema. It produces characteristic crackling sound.
Patients with a ZMC fracture must be advised to avoid nose blowing or holding the nose when sneezing, as surgical emphysema may result.
The lateral palpebral ligament is attached to the zygomatic portion of the orbital rim. Displacement of the zygoma carries the palpebral ligament with it. When the zygoma is displaced in an inferior direction, the lateral palpebral ligament is also depressed, causing a downward slope of the fissure (antimongoloid slant).
The loss of osseous support for the orbital contents and displacement of Tenon’s capsule and suspensory ligaments of the globe permit depression of the globe, leading to unequal pupillary levels, with the involved pupil at a lower level than that of the normal side.
Diplopia means blurred vision. Monocular diplopia is blurring of vision through one eye with the other closed. Binocular diplopia is blurring of vision through both eyes.
If the zygomatic injury has produced an increase in orbital volume, by lateral and inferior displacement of the zygoma, or disruption of the medial, lateral and inferior orbital walls, or has resulted in a decrease in orbital soft tissue volume by herniation of orbital soft tissues, enophthalmos can result.
PA water’s view, with head 27 degree angle to the vertical plane.
PA Caldwell view, the face is at a 15 degree angle to cassette.
Submentovertex OR Jug Handle view also shows the position of the zygomatic arch clearly and its displacement can be well perceived on it.
For more details, CT scan is obtained with axial and coronal 3-5 mm cuts.
Or if there is involvement of the orbital floor and walls, which cannot be perceived well on a water’s view alone, a CT scan should be considered.
Three dimensional CT scans offer no additional information beyond what is already present in two dimensional scans.
Right sided depressed zygomatic arch fracture. Submentovertex view
Left-sided ZMC fracture (yellow arrows) with fluid level (green arrow) in the maxillary sinus
Axial CT scan showing zygomatic arch fracture.
Axial CT scan demonstrating zygomaticomaxillary complex fracture on right with severe displacement
A high resoluton 3d ct scan showing zmc fracture on the left side with comminution.
USUAALLY By direct application of an instrument to the deep (temporal) aspect of the zygomatic bone.
It still remains one of the best techniques for applying a powerful and controlled force to the zygomatic bone and arch.
The temporal fascia is attached to the outer aspect of the zygomatic bone and superior border of the zygomatic arch., and beneath this layer and superficial to the temporal muscle lies a potential space or tissue plane into which a long, flat and narrow instrument can be introduced.
Advantage of rowe’s pattern elevator over bristow’s elevator.
2.5 cm long incision above and parallel to the anterior branch of the temporal artery.
The fascia is identified and incised, and a periosteal elevator is passed downwards and forwards as far as the temporal aspect of the zygomatic bone.
After entering the space between the fascia and muscle, the periosteal elevator is withdrawn until the tip comes to the fascial incision, to act as a guide for the introduction of a rows pattern zygomatic elevator.
The reduction is often accompanied by an audible click.
At the same time during elevation, the other hand palpates along the infraorbital rim and body of the zygoma.
Instruments used are dingman’s zygomatic elevator, urethral sound or a large kelly hemostat.
Elevation of the zygoma through the lateral eyebrow approach.
Disadvantage is that, it is difficult to generate a large amount of force especially in the superior direction.
A pointed curved elevator (monks pattern) can be passed upwards supraperiosteally to contact the deep or infratemporal surface of the zygomatic bone and thus enable upward, forward and outward pressure to be exerted.
This is deepened by blunt dissection in a supraperiosteal plane, following the lateral aspect of the coronoid process and the tendon of the temporal muscle until the medial aspect of the arch is reached.
A suitable elevator is then placed in position and the arch is palpated extraorally while the instrument is moved antero-posteriorly to restore the original contour.
The upper buccal sulcus approach and the position of the instrument before the application. ( taylor monk’s pattern elevator).
A pointed curved elevator (monks pattern) can be passed upwards supraperiosteally to contact the deep or infratemporal surface of the zygomatic bone and thus enable upward, forward and outward pressure to be exerted.
Various instruments have been designed for this purpose by crowe, ginestet and dautrey and poswillo.
The percutaneous bone hook is rotated downwards through about 90 degree as the point is advanced until it engages the temporal aspect of the bone.
Elevation of the ZMC can also be done with a screw inserted percutaneously. A caroll girard bone screw.
This method is applicable only to those cases where a rotation around the vertical axis has taken place in a medial direction causing a depression of the zygomatic bone in the antral cavity.
It will be necessary therefore to ensure that the orbital margins, floor and medial wall are either intact or reduced and stabilised by some form of fixation before inserting a pack or other temporary support.
i.e., in the cases of category 2 fractures.
Before deciding that there is separation at the frontozygomatic suture line, a comparison should be made with the uninjured side.
Along the langers lines. In older patients, so called crow’s foot wrinkles around the outer aspect of the eye, an incision through one of these lines about 1 cm above the outer canthus. An incision of 1.5cm is usually enough.
This is an excellent technique which provides better lateral stability.
A separation of the bone ends at the FZ suture doesn’t mean that there will be an associated separation at the fracture site in the inferior orbital margin, since a pivotal movement may have taken place along the horizontal axis between the zygomatic arch and the inferior orbital rim with depression of the zygomatic bone into the antrum.
Unlike the relatively robust bone at the frontozygomatic suture, the bone of the inferior orbital margin is thin and the antrum is in close proximity so that the drill holes must be carefully placed in relation to the fracture sites.
They should pass from about 5mm below the outer aspect of the rim obliquely upwards and backwards, whenever possible be situated behind the rim and also be situated 3-5mm away from the fracture margins. A direct wire may be used but a figure of 8 pattern provides better stability.
Wire used is 0.35 mm fine stainless steel wire, away from fracture margins.
The fragments are maintained in position by a direct and a figure of 8 wire suture.
When there is loss of bone, it will be necessary to select a stable point at the extreme limits of the comminuted segment through which a hole may be safely drilled.
After passing a 0.35mm diameter soft stainless steel wire through the hole on one side, the ends are carefully twisted to form a cable sufficient to bridge the gap.
Postoperative instability is due to a hinge rotation around the FZ suture or due to the separation of the bone ends at this site. And it may not be sufficient to control the zygomatic bone, with transosseous wiring especially if there is also a tendency to rotation and displacement at the inferior orbital margin , even after insertion of wires, when there is loss of support from the zygomatic support.
In this case, a combination of the frontozygomatic wire and an intraoral approach to the zygomatic buttress where mini plates may be attached, thereby reducing and aligning the buttress and hence the lateral orbital wall, may be of significant help.
A plate may also be applied across the frontozygomatic suture, although it is preferable to use a wire initially since a degree of mobility remains, thereby facilitating correct alignment of the zygomatic buttress when placing a miniplate across it.
However, the technique of indirect fixation does provide a means of fixation when there has been gross loss of bone in the region of the fronto-zygomatic suture and inferior orbital rim.
In all those methods, Kirschner wires are used to fix the fractures zygomatic bone to some point else where on the facial skeleton, which is stable.