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Prof.G.S.Patnaik
CLUB FOOT
INTRODUCTION
 Clubfoot is a condition in which one or both feet are twisted into
an abnormal position at birth.
 Common birth defect
 Other terms Giles Smith Syndrome, congenital talipes
aquinovarus (CTEV)
 The condition is also known as talipes. It is a general term used
to describe a range of unusual positions of the foot.
 Present at birth and affects the foot and/or ankle.
 There is no known cause for clubfoot
 Most common in male children as it is in female children.
 Most type of clubfoot is present at birth which can happen in one
foot or in both feet. In almost half of affected infants, both feet are
involved. Although clubfoot is painless in a baby,
 Approximately 50% cases of clubfoot are bilateral
CAUSES
 Family history of clubfoot.
 Position of the baby in the uterus.
 Increased occurrences in those children with neuromuscular
disorders, such as cerebral palsy and spina bifida.
 Amniotic Band Syndrome
 Oligohydramnios
MANIFESTATION
 Fixed plantar Flexion of the ankle, characterized by the drawn up
position of the heel and inability to bring the foot to a plantigrade
(flat) standing position. This is caused by a tight Achilles tendon.
 Adduction, or turning in of the heel or hind foot.
 Adduction turning under of the forefoot and mid foot giving the
foot a kidney-shaped appearance.
 Abnormal slightly smaller size of foot & calf muscles.
 The heel cord (Achilles tendon) is tight causing the heel to be
drawn up toward the leg.
TYPES
 Structural TEV is caused by genetic factors, such as Edwards
syndrome, a genetic defect with three copies of chromosome 18.
Growth arrests at roughly 9 weeks and compartment syndrome of
the affect limb are also causes of Structural TEV. Genetic
influences increase dramatically with family history.
 Postural TEV could be caused by external influences in the final tr
imester such as intrauterine compression from oligohydramnios
or from amniotic band syndrome. However, this is countered by
findings that TEV does not occur more frequently than usual
when the intrauterine space is restricted.
PATHOPHYSIOLOGY
Predisposing Factors:
Family history of clubfoot.
Position of the baby in the uterus.
Increased occurrences in those
children with neuromuscular
disorders, such as cerebral palsy
and spina bifida.
Amniotic Band Syndrome
Oligohydramnios
Distal limb amniotic
banding
Amnion forms
constrictive bands
around a limb in utero
Cutting off the
circulation to the limb
Defective cartiliganious
anlage of the talus
Resulting in further
abnormal or arrested
development
Arrest of the fetal
development in the
fibular stage
DIAGNOSTIC PROCEDURES
 Ultrasonography
 X-ray
 CT-scan
ULTRASONOGRAPHY
X-RAY
CT-SCAN
TREATMENT
 Stretching and casting (Ponseti Method)
 Stretching and taping (French Method)
 Surgery
 Brace
FUNCTIONAL TREATMENT
 Stretching and casting (Ponseti method). This
treatment entails manipulating the foot into a
correct position and then placing it in a cast to
maintain that position. Repositioning and recasting
occurs every week for several weeks. After the
shape of the foot is realigned, it's maintained
through stretching exercises, special shoes or
splinting with braces at night for up to three years.
For this method to work effectively, you'll need to
apply your child's braces according to your doctor's
specifications so that the foot doesn't return to its
original position.
CASTING / PONSETI METHOD
FUNCTIONAL METHOD
 Stretching and taping (French method). This
approach involves daily manipulation of the foot,
followed by the use of adhesive tape to maintain
the correct position until the next day. After two
months, treatments are reduced to three times
each week until the baby is 6 months old. Once the
foot's shape is corrected, parents continue to
perform daily exercises and use night splints until
their baby is walking age. This method requires
commitment to very frequent appointments for six
months. Some providers combine the French
method and the Ponseti method
TAPING / FRENCH METHOD
BRACE
 Dennis Brown Brace- used when long leg
cast is removed after 3 weeks of treatment. The bar
is fit shoulder width apart and worn full time for the
1st 2months
DENNIS BROWNE BRACE
PHARMACOLOGIC MANAGEMENTS
 NSAIDS (Ibuprofen)
Management for pain
NURSING RESPONSIBILITIES
 Review the pathology, prognosis and future expectations to
mothers to provideknowledge base from which parents can make
informed choice.
 Discuss deformity and expected treatment in terms the parents
can understandto rule out misconceptions and to provide
information about the deformity.
 Encourage parents to hold and play with child and participate in
care to promotebonding.
 Assess and teach parent to assess for signs of excessive
pressure on
skin,redness, excoriation because these signs require immediate
evaluation andintervention.
 Elevate the extremity to promote venous return and prevents
edema.
 Check the toes every 1-2 hours for temperature, color, sensation,
motion, andcapillary refill time.
 Stimulate movement of toes to promote circulation.
 Insert plastic petals over the top edges of a new cast while it is
still wet to keepurine from soaking and softening the cast.
 Provide comfort measures such as soft music, pacifier, teething
ring, or rockingto promote relaxation and may enhance patients
coping abilities by refocusingattention.
 When the Kite casting method is being used, check circulatory
status frequently.Circulation maybe impaired because of
increased pressure on tissues and
bloodvessels. The equines correction specially places considerabl
e strain onligaments, blood vessels, and tendons.
 Discuss the importance of physical therapist to enhance mobility
NURSING DIAGNOSIS
1. Risk for disproportionate growth related to
congenital disorders.
2. Impaired physical mobility related to
musculoskeletal impairment.
3. Impaired skin integrity related to
musculoskeletal impairment.
4. Disturbed body image related to developmental
changes.
5. Social isolation related to alterations in physical
appearance
THANK YOU FOR LISTENING

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Clubfoot prof g s patnaik

  • 2. INTRODUCTION  Clubfoot is a condition in which one or both feet are twisted into an abnormal position at birth.  Common birth defect  Other terms Giles Smith Syndrome, congenital talipes aquinovarus (CTEV)  The condition is also known as talipes. It is a general term used to describe a range of unusual positions of the foot.  Present at birth and affects the foot and/or ankle.  There is no known cause for clubfoot  Most common in male children as it is in female children.  Most type of clubfoot is present at birth which can happen in one foot or in both feet. In almost half of affected infants, both feet are involved. Although clubfoot is painless in a baby,  Approximately 50% cases of clubfoot are bilateral
  • 3.
  • 4. CAUSES  Family history of clubfoot.  Position of the baby in the uterus.  Increased occurrences in those children with neuromuscular disorders, such as cerebral palsy and spina bifida.  Amniotic Band Syndrome  Oligohydramnios
  • 5. MANIFESTATION  Fixed plantar Flexion of the ankle, characterized by the drawn up position of the heel and inability to bring the foot to a plantigrade (flat) standing position. This is caused by a tight Achilles tendon.  Adduction, or turning in of the heel or hind foot.  Adduction turning under of the forefoot and mid foot giving the foot a kidney-shaped appearance.  Abnormal slightly smaller size of foot & calf muscles.  The heel cord (Achilles tendon) is tight causing the heel to be drawn up toward the leg.
  • 6. TYPES  Structural TEV is caused by genetic factors, such as Edwards syndrome, a genetic defect with three copies of chromosome 18. Growth arrests at roughly 9 weeks and compartment syndrome of the affect limb are also causes of Structural TEV. Genetic influences increase dramatically with family history.  Postural TEV could be caused by external influences in the final tr imester such as intrauterine compression from oligohydramnios or from amniotic band syndrome. However, this is countered by findings that TEV does not occur more frequently than usual when the intrauterine space is restricted.
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  • 10. PATHOPHYSIOLOGY Predisposing Factors: Family history of clubfoot. Position of the baby in the uterus. Increased occurrences in those children with neuromuscular disorders, such as cerebral palsy and spina bifida. Amniotic Band Syndrome Oligohydramnios Distal limb amniotic banding Amnion forms constrictive bands around a limb in utero Cutting off the circulation to the limb Defective cartiliganious anlage of the talus Resulting in further abnormal or arrested development Arrest of the fetal development in the fibular stage
  • 13. X-RAY
  • 15. TREATMENT  Stretching and casting (Ponseti Method)  Stretching and taping (French Method)  Surgery  Brace
  • 16. FUNCTIONAL TREATMENT  Stretching and casting (Ponseti method). This treatment entails manipulating the foot into a correct position and then placing it in a cast to maintain that position. Repositioning and recasting occurs every week for several weeks. After the shape of the foot is realigned, it's maintained through stretching exercises, special shoes or splinting with braces at night for up to three years. For this method to work effectively, you'll need to apply your child's braces according to your doctor's specifications so that the foot doesn't return to its original position.
  • 18. FUNCTIONAL METHOD  Stretching and taping (French method). This approach involves daily manipulation of the foot, followed by the use of adhesive tape to maintain the correct position until the next day. After two months, treatments are reduced to three times each week until the baby is 6 months old. Once the foot's shape is corrected, parents continue to perform daily exercises and use night splints until their baby is walking age. This method requires commitment to very frequent appointments for six months. Some providers combine the French method and the Ponseti method
  • 19. TAPING / FRENCH METHOD
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  • 21. BRACE  Dennis Brown Brace- used when long leg cast is removed after 3 weeks of treatment. The bar is fit shoulder width apart and worn full time for the 1st 2months
  • 23. PHARMACOLOGIC MANAGEMENTS  NSAIDS (Ibuprofen) Management for pain
  • 24. NURSING RESPONSIBILITIES  Review the pathology, prognosis and future expectations to mothers to provideknowledge base from which parents can make informed choice.  Discuss deformity and expected treatment in terms the parents can understandto rule out misconceptions and to provide information about the deformity.  Encourage parents to hold and play with child and participate in care to promotebonding.  Assess and teach parent to assess for signs of excessive pressure on skin,redness, excoriation because these signs require immediate evaluation andintervention.  Elevate the extremity to promote venous return and prevents edema.  Check the toes every 1-2 hours for temperature, color, sensation, motion, andcapillary refill time.
  • 25.  Stimulate movement of toes to promote circulation.  Insert plastic petals over the top edges of a new cast while it is still wet to keepurine from soaking and softening the cast.  Provide comfort measures such as soft music, pacifier, teething ring, or rockingto promote relaxation and may enhance patients coping abilities by refocusingattention.  When the Kite casting method is being used, check circulatory status frequently.Circulation maybe impaired because of increased pressure on tissues and bloodvessels. The equines correction specially places considerabl e strain onligaments, blood vessels, and tendons.  Discuss the importance of physical therapist to enhance mobility
  • 26. NURSING DIAGNOSIS 1. Risk for disproportionate growth related to congenital disorders. 2. Impaired physical mobility related to musculoskeletal impairment. 3. Impaired skin integrity related to musculoskeletal impairment. 4. Disturbed body image related to developmental changes. 5. Social isolation related to alterations in physical appearance
  • 27. THANK YOU FOR LISTENING