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 Genu varum ( bow leg )
 Normal development of lower
extremities:
 The uterin space during gestation forces the lower
extremity to lie in a “Buddha” position with flexion
of the hips and knees and internal rotation of the
tibia and feet.
 This position causes contracture of the medial knee
capsule, especially of the posterior oblique
ligament
 Depending on the residual tightness of this
capsular/ligamentous contracture at the onset of
walking, varying amounts of bowleggedness will
still be clinically appreciated.
 Over the course of time, these contractures stretch,
and spontaneous resolution of this “physiologic”
bowing is seen.
Genu varum and medial tibial torsion are:
 Normal in newborn and infants.
 Maximal varus is present at 6 to 12 ms of age.
 With normal growth, the lower limbs gradually
straighten with a zero Tibio femoral angle by 18 to
24 months of age. (when the infant begins to stand
and walk).
 Knees gradually drift into valgus (knock knee).

7
I. Physiologic
II.Pathologic
Differential diagnosis of genu
varum:

I. Physiologic

A. Blount’s disease
B. Hypophosphatemic or nutritional rickets
C. Posttraumatic
D. Postinfectious
E. Congenital deformities
F. Focal fibrocartilaginous dysplasia
G. Metaphyseal chondrodysplasia
H. Fibrous dysplasia
I. Osteogenesis imperfecta
J. Renal osteodystrophy
genu varum in the older child

Bowlegs after 2 years of age are considered
abnormal.
Regardless of the type, the bowing becomes most
pronounced during the 2nd year of life, when the
child starts ambulating.

10
 When genu varum occurs concurrent
with rotational abnormalities such
as internal tibial torsion, the gross
clinical appearance of the bowlegs
is greatly exaggerated.
 Pathologic genu varum:
 Focal and systemic conditions may lead to the
deformity.
 This can affect a specific region in the knee, or the
bone , with multiple sites of deformities.
 Pathologic deformities tend to occur more
unilaterally.

 Clinically they also present with a lateral thrust
due to varus instability at the knee.
14
 The stature and nutritional status of the child ,

 Developmental milestones,
 Other nutritional or medical problems.
 History of trauma or infections

 Exogenous metal intoxication , (lead and
fluoride).
 Physiologic genu varum improves with age
growth , whereas pathologic bowing of the legs
increases with skeletal growth.

 Limb deformities and presence of short stature
may indicate the possibility of bone dysplasia or a
generalized growth disorder.
It seems important to ask the parents about:
1. When they first noticed the deformity .
2. Were the legs bowed at birth and in infancy, or did
the bowlegs develop later on when the child started
walking?
3. Is the deformity improving, staying the same, or
increasing in severity?
4. When did the child begin to stand and walk?
17
 Suggests the possibility of vitamin D refractory
(hypo-phosphatemic) rickets or bone dysplasia,
( achondroplasia or metaphyseal dysplasia) .

19
 First assessed from the back of the standing child,
then with the child supine.
 For instability, which on ambulation manifests as a
lateral thrust.
 Performed with the medial malleoli
in contact,
 Done in supine.
 Greater than 6 cm is abnormal.
 Ruling out the deformity of the feet
.
 Measured using a goniometer.
In physiologic G. V. there is a gentle curve
involving both the thigh and the leg .

24
 In Blount’s disease it is commonly at the proximal
tibial metaphysis with an acute medial angulation
immediately below the knee .
 In the very rare distal femoral vara the site of
angulation is in the distal femoral metaphysis.

 When the lower tibiae are the sites of varus
angulation, the upper tibial segment is straight and
the lower segment angulated.
26
The foot progression angle may be medial or
normal.

27
In physiologic genu varum it is usually bilateral
and symmetric,
Blount’s disease it may be unilateral or bilateral ,
and asymmetric.

28
In rickets (vitamin D refractory or vitamin
deficiency) they are enlarged.

29
Torsion of the tibia should also be
routinely assessed

 Determination of the thigh-foot angle and
evaluation of the bimalleolar axis
Take radiograms when :
I. A 3 years and older and the varus deformity is
not improving or is getting worse,

II. The medial bowing is unilateral or asymmetric,
III. The angulation is acute in the proximal tibial
metaphysis immediately below the knee,
IV. The possibility of a pathologic condition.
32
 Full-length standing bilateral antero posterior
radiographs from hip to ankle should be obtained.

 The focus of the radiograph should be at the knee
with both kneecaps pointing forward.
 The growth plates of the distal femur and

proximal tibia should be considered carefully.
 The horizontal joint lines of both the knee and
ankle are tilted medially.

34
Measure the metaphyseal - diaphyseal angle.
 In the physiologic genu varum it is less than 11degrees,
whereas in tibia vara it is greater than 11 degrees .
Medial Physeal Slope

Femoral-Tibial Axis
WHEN TO REFER ?
• Pathologic deformities:





Asymmetrical.
Localized.
Progressive.
Not expected for age.

• Exaggerated physiologic deformities:
 In the vast majority of cases, genu varum will
correct with growth.

 In physiologic genu varum education and
assurance of the parents is important and just
follow its natural course by reassessing the child in
6 months.
 For the overly concerned parent, “treatment” to
expedite this natural resolution consists of daily
knee stretches .

Method for stretching the posterior oblique ligament.
The tibia is externally rotated with the knee in a 90° flexed position.
 Orthopedic shoes are not effective in its
prevention or management.
 When severe genu varum is associated with
severe medial tibial torsion and the metaphysealdiaphyseal angle is 11 degrees or greater, a Denis
Browne splint is prescribed with the feet rotated
laterally and with an 8 to 10-inch bar between the
shoes.
 This is ordinarily worn only at night for a period
not more than 3 to 6 months in order to correct
excessive medial tibial torsion .
 The brace is worn nearly full-time, especially during
walking, to minimize the valgus stress at the knee.

 The effectiveness of the brace is related to the relief of
weight bearing stresses on the medial physeal region of
the proximal tibia.
 Brace treatment is reported to be successful in 50% to
80% of the patients treated.
 The brace is worn until the deformity has been corrected
which usually takes about 1 year.
 Thus, bracing is usually not a viable option for children
over the age of 3.
 Metabolic deformities such as rickets could simply
be corrected with medical treatment, i.e. calcium
and vitamin D supplements.
 In the adolescent with severe genu varum with
marked malalignment of the mechanical axis of
the lower limbs, occasionally osteotomy of the
tibia

46
Ostéotomies
GENU VARUM
FROM INFANCY TO ADULT LIFE
NORMAL VARUS IN INFANCY

Corrects spontaneously
PATHOLOGICAL GENU VARUM
Rachitic

Blount’s disease
Osteoclasis at age of three correction and plaster
Rachitic bow legs

Tibia vara legs straight
Late rickets bialateral osteotomy
Blount disease infracondlyar osteotomy
FEMORAL BOW LEGS
Bilateral supraconylar osteotomy
O.A. G.VARUM

Standing films are
essential

High tibial ostetomy
OSTEOMYELITIS GROWTH PLATE AFFECTION
GENU VARUM
RENAL RICKETS

Age ten years
RENAL RICKETS

Pseudo-fractures,wide epiphyseal plate
OSTEOGENESIS IMPERFECTA

Sofield multiple level
osteotomies
Principles of Evaluation and
Treatment;
(1) Genu varum is physiologic until the age of 18 to
24 months, and treatment is unnecessary.
(2) In a child with normal stature and
findings compatible with physiologic
bowing, radiographic documentation
is unnecessary.
Photographs are less expensive
and just as valuable.
(3) If radiographs are deemed necessary,
full-length standing films of the entire
lower limbs

(4) Shortness of stature should signal
the likelihood that a constitutional disorder

is the cause of genu varum.
(5) Idiopathic tibia vara is the most
common pathologic cause of
bowlegs in the child.

Bracing may be effective in the early

stages, but this has not been established
by prospective controlled clinical trials.
(6) There are various types of internal

and external fixation, all of which
are satisfactory.

(7) Treatment of genu varum secondary
to constitutional disorders must be

tailored on an individual basis.
AZK(HOORISH BALOACH)

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AZK(HOORISH BALOACH)

  • 1.
  • 2.  Genu varum ( bow leg )
  • 3.  Normal development of lower extremities:  The uterin space during gestation forces the lower extremity to lie in a “Buddha” position with flexion of the hips and knees and internal rotation of the tibia and feet.
  • 4.  This position causes contracture of the medial knee capsule, especially of the posterior oblique ligament  Depending on the residual tightness of this capsular/ligamentous contracture at the onset of walking, varying amounts of bowleggedness will still be clinically appreciated.
  • 5.  Over the course of time, these contractures stretch, and spontaneous resolution of this “physiologic” bowing is seen.
  • 6. Genu varum and medial tibial torsion are:  Normal in newborn and infants.  Maximal varus is present at 6 to 12 ms of age.  With normal growth, the lower limbs gradually straighten with a zero Tibio femoral angle by 18 to 24 months of age. (when the infant begins to stand and walk).
  • 7.  Knees gradually drift into valgus (knock knee). 7
  • 9. Differential diagnosis of genu varum: I. Physiologic A. Blount’s disease B. Hypophosphatemic or nutritional rickets C. Posttraumatic D. Postinfectious E. Congenital deformities F. Focal fibrocartilaginous dysplasia G. Metaphyseal chondrodysplasia H. Fibrous dysplasia I. Osteogenesis imperfecta J. Renal osteodystrophy
  • 10. genu varum in the older child Bowlegs after 2 years of age are considered abnormal. Regardless of the type, the bowing becomes most pronounced during the 2nd year of life, when the child starts ambulating. 10
  • 11.  When genu varum occurs concurrent with rotational abnormalities such as internal tibial torsion, the gross clinical appearance of the bowlegs is greatly exaggerated.
  • 12.  Pathologic genu varum:  Focal and systemic conditions may lead to the deformity.  This can affect a specific region in the knee, or the bone , with multiple sites of deformities.
  • 13.  Pathologic deformities tend to occur more unilaterally.  Clinically they also present with a lateral thrust due to varus instability at the knee.
  • 14. 14
  • 15.  The stature and nutritional status of the child ,  Developmental milestones,  Other nutritional or medical problems.  History of trauma or infections  Exogenous metal intoxication , (lead and fluoride).
  • 16.  Physiologic genu varum improves with age growth , whereas pathologic bowing of the legs increases with skeletal growth.  Limb deformities and presence of short stature may indicate the possibility of bone dysplasia or a generalized growth disorder.
  • 17. It seems important to ask the parents about: 1. When they first noticed the deformity . 2. Were the legs bowed at birth and in infancy, or did the bowlegs develop later on when the child started walking? 3. Is the deformity improving, staying the same, or increasing in severity? 4. When did the child begin to stand and walk? 17
  • 18.
  • 19.  Suggests the possibility of vitamin D refractory (hypo-phosphatemic) rickets or bone dysplasia, ( achondroplasia or metaphyseal dysplasia) . 19
  • 20.  First assessed from the back of the standing child, then with the child supine.
  • 21.  For instability, which on ambulation manifests as a lateral thrust.
  • 22.  Performed with the medial malleoli in contact,  Done in supine.  Greater than 6 cm is abnormal.  Ruling out the deformity of the feet .
  • 23.  Measured using a goniometer.
  • 24. In physiologic G. V. there is a gentle curve involving both the thigh and the leg . 24
  • 25.  In Blount’s disease it is commonly at the proximal tibial metaphysis with an acute medial angulation immediately below the knee .
  • 26.  In the very rare distal femoral vara the site of angulation is in the distal femoral metaphysis.  When the lower tibiae are the sites of varus angulation, the upper tibial segment is straight and the lower segment angulated. 26
  • 27. The foot progression angle may be medial or normal. 27
  • 28. In physiologic genu varum it is usually bilateral and symmetric, Blount’s disease it may be unilateral or bilateral , and asymmetric. 28
  • 29. In rickets (vitamin D refractory or vitamin deficiency) they are enlarged. 29
  • 30. Torsion of the tibia should also be routinely assessed  Determination of the thigh-foot angle and evaluation of the bimalleolar axis
  • 31.
  • 32. Take radiograms when : I. A 3 years and older and the varus deformity is not improving or is getting worse, II. The medial bowing is unilateral or asymmetric, III. The angulation is acute in the proximal tibial metaphysis immediately below the knee, IV. The possibility of a pathologic condition. 32
  • 33.  Full-length standing bilateral antero posterior radiographs from hip to ankle should be obtained.  The focus of the radiograph should be at the knee with both kneecaps pointing forward.
  • 34.  The growth plates of the distal femur and proximal tibia should be considered carefully.  The horizontal joint lines of both the knee and ankle are tilted medially. 34
  • 35. Measure the metaphyseal - diaphyseal angle.  In the physiologic genu varum it is less than 11degrees, whereas in tibia vara it is greater than 11 degrees .
  • 37. WHEN TO REFER ? • Pathologic deformities:     Asymmetrical. Localized. Progressive. Not expected for age. • Exaggerated physiologic deformities:
  • 38.
  • 39.  In the vast majority of cases, genu varum will correct with growth.  In physiologic genu varum education and assurance of the parents is important and just follow its natural course by reassessing the child in 6 months.
  • 40.  For the overly concerned parent, “treatment” to expedite this natural resolution consists of daily knee stretches . Method for stretching the posterior oblique ligament. The tibia is externally rotated with the knee in a 90° flexed position.
  • 41.  Orthopedic shoes are not effective in its prevention or management.  When severe genu varum is associated with severe medial tibial torsion and the metaphysealdiaphyseal angle is 11 degrees or greater, a Denis Browne splint is prescribed with the feet rotated laterally and with an 8 to 10-inch bar between the shoes.
  • 42.  This is ordinarily worn only at night for a period not more than 3 to 6 months in order to correct excessive medial tibial torsion .
  • 43.  The brace is worn nearly full-time, especially during walking, to minimize the valgus stress at the knee.  The effectiveness of the brace is related to the relief of weight bearing stresses on the medial physeal region of the proximal tibia.
  • 44.  Brace treatment is reported to be successful in 50% to 80% of the patients treated.  The brace is worn until the deformity has been corrected which usually takes about 1 year.  Thus, bracing is usually not a viable option for children over the age of 3.
  • 45.  Metabolic deformities such as rickets could simply be corrected with medical treatment, i.e. calcium and vitamin D supplements.
  • 46.  In the adolescent with severe genu varum with marked malalignment of the mechanical axis of the lower limbs, occasionally osteotomy of the tibia 46
  • 48. GENU VARUM FROM INFANCY TO ADULT LIFE
  • 49. NORMAL VARUS IN INFANCY Corrects spontaneously
  • 51. Osteoclasis at age of three correction and plaster
  • 52. Rachitic bow legs Tibia vara legs straight
  • 55. FEMORAL BOW LEGS Bilateral supraconylar osteotomy
  • 56. O.A. G.VARUM Standing films are essential High tibial ostetomy
  • 57. OSTEOMYELITIS GROWTH PLATE AFFECTION GENU VARUM
  • 61. Principles of Evaluation and Treatment; (1) Genu varum is physiologic until the age of 18 to 24 months, and treatment is unnecessary.
  • 62. (2) In a child with normal stature and findings compatible with physiologic bowing, radiographic documentation is unnecessary. Photographs are less expensive and just as valuable.
  • 63. (3) If radiographs are deemed necessary, full-length standing films of the entire lower limbs (4) Shortness of stature should signal the likelihood that a constitutional disorder is the cause of genu varum.
  • 64. (5) Idiopathic tibia vara is the most common pathologic cause of bowlegs in the child. Bracing may be effective in the early stages, but this has not been established by prospective controlled clinical trials.
  • 65. (6) There are various types of internal and external fixation, all of which are satisfactory. (7) Treatment of genu varum secondary to constitutional disorders must be tailored on an individual basis.