Class II Malocclusion
 EXTRA ORAL
 CONVEX PROFILE
 SHORT UPPER LIP
 INCOMPETENT LIPS
 EVERTED LOWER LIP
 DEEP MENTOLABIAL SULCUS
 HYPERACTIVE MENTALIS
 INTRAORAL FEATURES
 CLASSII MOLAR
 CLASSII CANINE
 PROCLINED UPPER ANTERIORS
 DEEPBITE
 V SHAPED UPPER ARCH
 DEEP PALATE
Prenatal factors
 Heredity -The size position and relationship of the jaws
are to a large extent the genes.
 Teratogenesis-Administration of certain drugs during
pregnancy can result in perverted or abnormal
development.
 Irradiation-Exposure of a pregnant woman to radiation
is another cause of altered development of the dento-
facial complex.
 Intrauterine fetal position.such as a hand across the face
is found to affect mandibular growth.
Natal factors
 Trauma can sometimes be induced by improper
forceps delivery.
Post natal factors
 Traumatic injury
 Long term irradiation therapy of the skeletal cranio-
facial region.
 Infectious conditions such as rheumatoid arthritis
can influence mandibular growth.
 Abnormal function such as oral
respiration,abnormal swallowing and habits such as
thumb sucking prevent normal muscle activity.
Treatment objectives
 Correction of skeletal abnormality if present.
 Reduction of overjet
 Reduction of overbite
 Correction of crowding and local irregularities
 Correction of molar relation
 Correction of posterior crossbites
 Normalizing the musculature.
Treatment of skeletal class II malocclusion
 Growth modification
 Camouflage
 Surgical correction.
Growth modification
 Class II,division 1 malocclusions are often
complicated by the presence of underlying skeletal
abnormalities. These are intercepted by functional
and orthopaedic appliances.
Camouflage
 In patients who are beyond growth, it is not possible
to undertake growth modification procedures. Thus
the underlying skeletal discrepancy can be
camouflaged by orthodontic tooth movement. This is
often done by extraction of certain teeth and moving
rest of the teeth into the space created.
Surgical correction
 In patients exhibiting severe skeletal
malrelationship,surgery may be the ideal treatment
modality. Based on the underlying skeletal pattern a
maxillary setback or a mandibular advancement is
undertaken after the completion of growth.
 Correction of deep bite.
 -use of removable anterior bite planes.
 -use of fixed appliances to intrude the upper and
lower anteriors.
 Crossbites are treated using appliances incorporating
screws or springs that expand the maxillary arch.
Features of class II division 2
 Molars Class II
 Retroclined central incisors and rarely of other
anteriors as well.
 Deep overbite
 Pleasing straight profile
 Broad square face
 Backward path of closure
 Deep mentolabial sulcus.
 Absence of abnormal muscle activity.
Treatment objectives
 Relief of gingival trauma
 Correction of incisor relationship
 Relief of crowding and local irregularities.
 Correction of buccal segement relationship.

Class ii malocclusion

  • 1.
  • 2.
     EXTRA ORAL CONVEX PROFILE  SHORT UPPER LIP  INCOMPETENT LIPS  EVERTED LOWER LIP  DEEP MENTOLABIAL SULCUS  HYPERACTIVE MENTALIS
  • 3.
     INTRAORAL FEATURES CLASSII MOLAR  CLASSII CANINE  PROCLINED UPPER ANTERIORS  DEEPBITE  V SHAPED UPPER ARCH  DEEP PALATE
  • 4.
    Prenatal factors  Heredity-The size position and relationship of the jaws are to a large extent the genes.  Teratogenesis-Administration of certain drugs during pregnancy can result in perverted or abnormal development.  Irradiation-Exposure of a pregnant woman to radiation is another cause of altered development of the dento- facial complex.  Intrauterine fetal position.such as a hand across the face is found to affect mandibular growth.
  • 5.
    Natal factors  Traumacan sometimes be induced by improper forceps delivery.
  • 6.
    Post natal factors Traumatic injury  Long term irradiation therapy of the skeletal cranio- facial region.  Infectious conditions such as rheumatoid arthritis can influence mandibular growth.  Abnormal function such as oral respiration,abnormal swallowing and habits such as thumb sucking prevent normal muscle activity.
  • 7.
    Treatment objectives  Correctionof skeletal abnormality if present.  Reduction of overjet  Reduction of overbite  Correction of crowding and local irregularities  Correction of molar relation  Correction of posterior crossbites  Normalizing the musculature.
  • 8.
    Treatment of skeletalclass II malocclusion  Growth modification  Camouflage  Surgical correction.
  • 9.
    Growth modification  ClassII,division 1 malocclusions are often complicated by the presence of underlying skeletal abnormalities. These are intercepted by functional and orthopaedic appliances.
  • 10.
    Camouflage  In patientswho are beyond growth, it is not possible to undertake growth modification procedures. Thus the underlying skeletal discrepancy can be camouflaged by orthodontic tooth movement. This is often done by extraction of certain teeth and moving rest of the teeth into the space created.
  • 11.
    Surgical correction  Inpatients exhibiting severe skeletal malrelationship,surgery may be the ideal treatment modality. Based on the underlying skeletal pattern a maxillary setback or a mandibular advancement is undertaken after the completion of growth.
  • 12.
     Correction ofdeep bite.  -use of removable anterior bite planes.  -use of fixed appliances to intrude the upper and lower anteriors.
  • 13.
     Crossbites aretreated using appliances incorporating screws or springs that expand the maxillary arch.
  • 14.
    Features of classII division 2  Molars Class II  Retroclined central incisors and rarely of other anteriors as well.  Deep overbite  Pleasing straight profile  Broad square face  Backward path of closure  Deep mentolabial sulcus.  Absence of abnormal muscle activity.
  • 15.
    Treatment objectives  Reliefof gingival trauma  Correction of incisor relationship  Relief of crowding and local irregularities.  Correction of buccal segement relationship.