1. 1
Diagnosis & TreatmentDiagnosis & Treatment
PlanningPlanning
Dr. KAPIL SAROHA
BDS, MDS
Orthodontics and dentofacial orthopaedics
www.drdentiste.comSaturday, February 11, 2017
2. 2
“The first step towards cure is to
know what the disease is......”
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3. 3
A century ago EDWARD. H.
ANGLE rightly said:
“In studying a case of malocclusion, give no
thought to the
– methods of treatment or appliances
until the case shall be classified with all
peculiarities and variations from the normal in
– type, occlusion and
– facial lines that have been thoroughly
comprehended.
Then the requirements and proper plan of
treatment become apparent”.
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4. 4
Human head is the mostmost
complicatedcomplicated anatomical complex in
all creation.
The interrelationships are infinite
and the causes and effects of these
relationships are almost
imponderable.
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5. 5
A thorough understanding of the
normal variations in the
– Growth and development of
dentofacial structures,
– Their anatomical fit into each other
and
– Their reaction to intrinsic and extrinsic
factors /stimuli (genetic and
environmental) itself is
Orthodontic diagnosis.
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7. 7
The goal of the diagnostic process is
to produce a complete description of
the patient’s problems and make a
problem list.
To obtain the problem list, a
collection of relevant information is
required. This collection is called a
database.
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8. 8
Mechano-Mechano-
therapytherapy
Diagnosis & Treatment
Planning - Steps
Patient History
Clinical
Examination
Analysis of
Diagnostic Records
Classification Problem List
= Diagnosis
Treat pathology
(caries, gingivitis etc.)
Treat pathology
(caries, gingivitis etc.)
Problems
in
priority
order
A
B
C
D
Possible
solution to
individual
problems
OptimalOptimal
TreatmentTreatment
PlanPlan
Data
Base
A
B
C
D
9. 9
Patient history, &Patient history, &
interview datainterview data
1. Family history
2. Motivation of patient for treatment
Internal
External
3. Reasons for taking treatment
Functional
Hygiene
Esthetics
Speech
4. Pubertal status
5. Prenatal History
1. Health of mother during pregnancy
Diseases : Bacterial / Viral
Medication
Radiation
Trauma
Patient history, &Patient history, &
interview datainterview data
1. Family history
2. Motivation of patient for treatment
Internal
External
3. Reasons for taking treatment
Functional
Hygiene
Esthetics
Speech
4. Pubertal status
5. Prenatal History
1. Health of mother during pregnancy
Diseases : Bacterial / Viral
Medication
Radiation
Trauma
Clinical Examination:Clinical Examination:
1. General examination
2. Extraoral
• Head shape
• Frontal symmetry
• Profile convexity
• Facial divergent
• Lip competency
• Incisor visibility,
3. Functional
• Mastication
• Deglutition
• Speech
• TMJ
4. Intraoral
• Hard tissues
• Soft tissues
Clinical Examination:Clinical Examination:
1. General examination
2. Extraoral
• Head shape
• Frontal symmetry
• Profile convexity
• Facial divergent
• Lip competency
• Incisor visibility,
3. Functional
• Mastication
• Deglutition
• Speech
• TMJ
4. Intraoral
• Hard tissues
• Soft tissues
Analysis ofAnalysis of
diagnostic records:diagnostic records:
1. Study Models
• Upper
• Lower
2. Radiographs
• Lateral Ceph.
• OPG
• A-P Ceph.
• IOPA
• Hand Wrist
• Occlusal
3. Photographs
• Extra-oral (3 + 2 smiling)
• Intra-oral (5)
Analysis ofAnalysis of
diagnostic records:diagnostic records:
1. Study Models
• Upper
• Lower
2. Radiographs
• Lateral Ceph.
• OPG
• A-P Ceph.
• IOPA
• Hand Wrist
• Occlusal
3. Photographs
• Extra-oral (3 + 2 smiling)
• Intra-oral (5)
Problem
List
Problem
List
Pathology:
1.
G
ingiva
(Attached
gingiva)
2.
Frenum
(Lab. / Ling.)
3.
Tonsils
/ Adenoids
4.
Tongue
5.
Dental Caries
Developm
ental Problem
s:
1.
Profile
and
Esthetics
•
Profile
(Convex, Straight, Concave)
•
Frontal ( Sym
m
etrical, Asym
m
etry)
•
Lips
2.
Alignm
ent
•
Upper (Crow
ding
/ Spacing)
•
Low
er (Crow
ding
/ Spacing)
3.
A-P
•
Skeleton
(Class
I, II, III)
•
Dental ( Class
I, II, III)
4.
Vertical
•
Skeleton
(VG
P
/Average
/ HG
P)
•
Dental (Deep
Bite
/ Norm
al / O
pen
Bite
5.
Transverse
•
Skeleton
(W
ide
/ Norm
al / Narrow
)
•
Dental (W
ide
/ Norm
al / Narrow
)
Problem
List
Problem
List
Pathology:
1.
G
ingiva
(Attached
gingiva)
2.
Frenum
(Lab. / Ling.)
3.
Tonsils
/ Adenoids
4.
Tongue
5.
Dental Caries
Developm
ental Problem
s:
1.
Profile
and
Esthetics
•
Profile
(Convex, Straight, Concave)
•
Frontal ( Sym
m
etrical, Asym
m
etry)
•
Lips
2.
Alignm
ent
•
Upper (Crow
ding
/ Spacing)
•
Low
er (Crow
ding
/ Spacing)
3.
A-P
•
Skeleton
(Class
I, II, III)
•
Dental ( Class
I, II, III)
4.
Vertical
•
Skeleton
(VG
P
/Average
/ HG
P)
•
Dental (Deep
Bite
/ Norm
al / O
pen
Bite
5.
Transverse
•
Skeleton
(W
ide
/ Norm
al / Narrow
)
•
Dental (W
ide
/ Norm
al / Narrow
)
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11. 11
If all the structures of the craniofacial
complex like the skeletal units , the
dentition and the soft tissue components
grow in harmony , then the result would be
a good occlusion with a well balanced face.
But the human face like most of our other
specialized anatomic parts, certainly has its
share of variations.
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13. 13
Since Class II malocclusion is
recognized easily by health
professionals as well as by patients
and their families, especially in cases
of excessive over jet, the correction
of class II problems may constitute
more than half of the treatmenthalf of the treatment
protocolprotocol in a typical orthodontic
practice.
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14. 14
It is interesting to note that the
process of evolution in orthodontic
diagnosis and treatment planningdiagnosis and treatment planning
has been gradual.
Now, let us trace through historylet us trace through history, the
changing perceptions on the etiology
of class II malocclusion.
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15. 15
For decades together class II was
erroneously considered a purely sagittalsagittal
problem.problem.
Pioneered by Dr. Angle’s classificationDr. Angle’s classification of
malocclusion based on anteroposterior
relationship of first molarfirst molar, probably
thousands of class II of all hues and
varities were treated as basically sagittal
discrepancies, often with disastrousdisastrous
results.
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16. 16
It was not the orthodontists alone
who were guilty of nescience, but
even the surgeons jumped onto the
bandwagon and restricted
themselves to sagittal correction of
what was actually a problem
involving more than one plane.
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17. 17
The Angle system of classification
still remains at the core of
orthodontic diagnosis a century after
its development, even though this
classification scheme is not sensitive
to imbalances in the vertical and
transverse dimensions.
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18. 18
First, let us see, how malocclusions
such as Class II develop as sagittal
discrepancy.
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24. 24
Can also be
because of
decreased cranial
flexure, the
posterior positioning
of glenoid fossa
which neutralizes
the horizontal
growth of mandible
ending up in Class
II. www.drdentiste.com
47. 47
VERTICAL
DISCREPANCY
With the passage of time, inevitably there
was gain of knowledge and wisdom and
the focus now began to shift towards other
etiologic possibilities of class II
malocclusion
It was schudy in 1964, who brought into
focus the vertical dysplasia causing and
affecting the class II malocclusion.
Until then, investigators had never
explored the vertical dimension of the
posterior aspect of the face. But here were
the secrets to be found.
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49. 49
Rotations of Mandible
The rotationrotation of the mandible due to
vertical growth discrepancies also
has to be distinguished.
3 www.drdentiste.com
52. 52
Excess Condylar Growth
Excessive condylar growth causes forward rotation
of the mandible leading to a class II deep bite
situation.
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53. 53
Now the concept changed such that when
facial morphology indicated that vertical
growth had been excessive or that
condylar growth had been deficient, the
plan was to inhibit the downward growth of
the maxillary molars.
When it is determined that vertical growth
is deficient, the choice is to stimulate the
vertical growth of the alveolar processes.
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54. 54
This quantum shift in knowledge
about the causative factors of class II
malocclusion brought into light an
entirely new gamut of treatment
possibilities.
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55. 55
Now let us look at some class II
cases with predominant verticalvertical
discrepancydiscrepancy and their treatment
options.
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78. 78
TRANSVERSE
DISCREPANCY
It has only been during the last two
decades or so that the role of
transverse dimensiontransverse dimension has been a
topic of interest to the typical
practicing orthodontist.
Until then it was a classical
illustration of, “the eyes cannot see
what the mind does not know.”
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79. 79
Many class II malocclusions, when
evaluated clinically have no obvious
maxillary constriction.
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80. 80
When a set of study models of the patient
are “hand articulated", how-ever, it
becomes obvious that when the dental
casts are placed with the posterior
dentition in a Class I relationship, a
unilateral or a bilateral cross bitecross bite is
produced.
This indicates the presence of maxillarymaxillary
constrictionconstriction as a component of class II
malocclusion.
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81. 81
FOOT AND SHOE MECHANISM
Richen Bach and Taatz in 1971 used the
example of a foot and a shoe, with the foot
representing the mandible and the shoe
representing the maxilla.
If the shoe is too narrow, it is impossible
for the foot to slide fully into the shoe. By
widening the shoe, the foot slides forward
into its usual position.
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82. 82
When treating in the mixed dentition,
the first step in the treatment of mild
to moderate Class II malocclusions
characterized, by mild mandibular
skeletal retraction and maxillary
constriction may be expansion of
maxilla.
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83. 83
The patients can be left in a over
expanded position with contacts still
being maintained between the upper
lingual cusps and lower buccal cusps
of the posterior teeth.
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84. 84
Widening the maxilla often leads to
a spontaneous forward posturing of
the mandible during the retention
period.
After 6 to 12 months, the
spontaneous correction of the class
II relationship can be seen in many
mild to moderate class II patients.
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85. 85
The net result of this change in
outlook has been a reduction in the
number of functional jaw orthopedic
appliances that now are used in the
treatment of mild to moderate class II
malocclusion.
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100. 100
During the1940’s and 50’s
mandibular prognathism was
believed to be the sole etiological
cause for Class III malocclusions.
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101. 101
All clinical efforts were concentrated
in correcting the mandibular
prognathism using Chin cup therapy
or surgical correction by mandibular
set back was the only alternative
practiced.
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102. 102
A lack of a clear understanding of the
underlying etiology often compounded
by adressal of wrong treatment
objectives resulting in disastrous
treatment results often accentuating the
problem rather than solving it.
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103. 103
With the advent of newer diagnostic
aids such as cephalometrics
identification of the role of maxilla in
the development class III
malocclusion came into picture
completely revolutionizing the
present treatment philosophy.
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136. 136
Conclusion
But as the philosopher Fredrick
Jensen has said, “What we think we
know today shatter the errors and
blunders of yesterday and is
tomorrow discarded as worthless.
So we go from larger mistakes to
small mistakes so long as we do not
loose courage. This is true of all
therapy, no method is final”.
137. 137
Thus even with tremendous progress
in basic research and mind boggling
improvement in appliance systems,
class II & III malocclusion has still
remained an enigma
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