This document provides information on ankle and foot fractures. It begins by describing ankle anatomy including the bones and ligaments. It then discusses common types of ankle fractures like Pott's fractures and classifications like the Weber system. Foot anatomy and fractures of bones like the calcaneus and talus are also outlined. Treatment approaches including casting, splinting, and surgery are described for different types and severities of ankle and foot fractures. Complications are noted. The document is a comprehensive overview of ankle and foot fracture anatomy, mechanisms of injury, classifications, clinical presentation, investigations, and management.
DR. GIRISH MOTWANI
Consultant Foot & Ankle surgeon (Paediatric & Adult)
1)Sushrut Hospital, Research Centre & PostGraduate Institute of Orthopaedics, Nagpur west
2)Aman hospital,Nagpur east
3)South point clinic, Nagpur south
DR. GIRISH MOTWANI
Consultant Foot & Ankle surgeon (Paediatric & Adult)
1)Sushrut Hospital, Research Centre & PostGraduate Institute of Orthopaedics, Nagpur west
2)Aman hospital,Nagpur east
3)South point clinic, Nagpur south
High tibial osteotomy (HTO) is a common and widely accepted procedure in orthopaedic surgery. In the literature, we find descriptions of the technique dating back to the 50s, with Jackson (Jackson, 1958). However, it was not until the 70s, with the publications of Conventry (Coventry, 1969 and 1973) and Insall (Insall, 1975), that proximal tibial osteotomy became common practice as a treatment option for medial compartment osteoarthritis of the knee usually associated to varus deformity. At that time, closing wedge osteotomies were performed, despite the greater technical difficulty and risks involved, as there were no fixation materials available that could enable opening wedge osteotomy. Only after the development of medial wedge plate fixation that opening wedge osteotomy became applicable (Puddu, 2004).
The goals of HTO are:
1. To reduce knee pain by transferring weight-bearing loads to the relatively unaffected compartment;
2. To increase the life span of the knee joint, by slowing or stopping the destruction of the medial joint compartment. This could delay the need of a joint replacement.
Medial patellofemoral ligament reconstruction ---- an update on techniques used. This lecture was taken by me at Trinity Arthroscopy Course, Chandigarh.
MCL. LCL.ALL injuries
To understand the relevant anatomy of the side ligaments of the knee
To study the mechanism of injury of each ligament and how to diagnose such injury
To highlight the different treatment options in acute or chronic situations
High tibial osteotomy (HTO) is a common and widely accepted procedure in orthopaedic surgery. In the literature, we find descriptions of the technique dating back to the 50s, with Jackson (Jackson, 1958). However, it was not until the 70s, with the publications of Conventry (Coventry, 1969 and 1973) and Insall (Insall, 1975), that proximal tibial osteotomy became common practice as a treatment option for medial compartment osteoarthritis of the knee usually associated to varus deformity. At that time, closing wedge osteotomies were performed, despite the greater technical difficulty and risks involved, as there were no fixation materials available that could enable opening wedge osteotomy. Only after the development of medial wedge plate fixation that opening wedge osteotomy became applicable (Puddu, 2004).
The goals of HTO are:
1. To reduce knee pain by transferring weight-bearing loads to the relatively unaffected compartment;
2. To increase the life span of the knee joint, by slowing or stopping the destruction of the medial joint compartment. This could delay the need of a joint replacement.
Medial patellofemoral ligament reconstruction ---- an update on techniques used. This lecture was taken by me at Trinity Arthroscopy Course, Chandigarh.
MCL. LCL.ALL injuries
To understand the relevant anatomy of the side ligaments of the knee
To study the mechanism of injury of each ligament and how to diagnose such injury
To highlight the different treatment options in acute or chronic situations
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MANAGEMENT OF ATRIOVENTRICULAR CONDUCTION BLOCK.pdfJim Jacob Roy
Cardiac conduction defects can occur due to various causes.
Atrioventricular conduction blocks ( AV blocks ) are classified into 3 types.
This document describes the acute management of AV block.
ARTIFICIAL INTELLIGENCE IN HEALTHCARE.pdfAnujkumaranit
Artificial intelligence (AI) refers to the simulation of human intelligence processes by machines, especially computer systems. It encompasses tasks such as learning, reasoning, problem-solving, perception, and language understanding. AI technologies are revolutionizing various fields, from healthcare to finance, by enabling machines to perform tasks that typically require human intelligence.
Explore natural remedies for syphilis treatment in Singapore. Discover alternative therapies, herbal remedies, and lifestyle changes that may complement conventional treatments. Learn about holistic approaches to managing syphilis symptoms and supporting overall health.
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TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Verified Chapters 1 - 19, Complete Newest Version.pdf
New Directions in Targeted Therapeutic Approaches for Older Adults With Mantl...i3 Health
i3 Health is pleased to make the speaker slides from this activity available for use as a non-accredited self-study or teaching resource.
This slide deck presented by Dr. Kami Maddocks, Professor-Clinical in the Division of Hematology and
Associate Division Director for Ambulatory Operations
The Ohio State University Comprehensive Cancer Center, will provide insight into new directions in targeted therapeutic approaches for older adults with mantle cell lymphoma.
STATEMENT OF NEED
Mantle cell lymphoma (MCL) is a rare, aggressive B-cell non-Hodgkin lymphoma (NHL) accounting for 5% to 7% of all lymphomas. Its prognosis ranges from indolent disease that does not require treatment for years to very aggressive disease, which is associated with poor survival (Silkenstedt et al, 2021). Typically, MCL is diagnosed at advanced stage and in older patients who cannot tolerate intensive therapy (NCCN, 2022). Although recent advances have slightly increased remission rates, recurrence and relapse remain very common, leading to a median overall survival between 3 and 6 years (LLS, 2021). Though there are several effective options, progress is still needed towards establishing an accepted frontline approach for MCL (Castellino et al, 2022). Treatment selection and management of MCL are complicated by the heterogeneity of prognosis, advanced age and comorbidities of patients, and lack of an established standard approach for treatment, making it vital that clinicians be familiar with the latest research and advances in this area. In this activity chaired by Michael Wang, MD, Professor in the Department of Lymphoma & Myeloma at MD Anderson Cancer Center, expert faculty will discuss prognostic factors informing treatment, the promising results of recent trials in new therapeutic approaches, and the implications of treatment resistance in therapeutic selection for MCL.
Target Audience
Hematology/oncology fellows, attending faculty, and other health care professionals involved in the treatment of patients with mantle cell lymphoma (MCL).
Learning Objectives
1.) Identify clinical and biological prognostic factors that can guide treatment decision making for older adults with MCL
2.) Evaluate emerging data on targeted therapeutic approaches for treatment-naive and relapsed/refractory MCL and their applicability to older adults
3.) Assess mechanisms of resistance to targeted therapies for MCL and their implications for treatment selection
Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
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Tom Selleck Health: A Comprehensive Look at the Iconic Actor’s Wellness Journeygreendigital
Tom Selleck, an enduring figure in Hollywood. has captivated audiences for decades with his rugged charm, iconic moustache. and memorable roles in television and film. From his breakout role as Thomas Magnum in Magnum P.I. to his current portrayal of Frank Reagan in Blue Bloods. Selleck's career has spanned over 50 years. But beyond his professional achievements. fans have often been curious about Tom Selleck Health. especially as he has aged in the public eye.
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Introduction
Many have been interested in Tom Selleck health. not only because of his enduring presence on screen but also because of the challenges. and lifestyle choices he has faced and made over the years. This article delves into the various aspects of Tom Selleck health. exploring his fitness regimen, diet, mental health. and the challenges he has encountered as he ages. We'll look at how he maintains his well-being. the health issues he has faced, and his approach to ageing .
Early Life and Career
Childhood and Athletic Beginnings
Tom Selleck was born on January 29, 1945, in Detroit, Michigan, and grew up in Sherman Oaks, California. From an early age, he was involved in sports, particularly basketball. which played a significant role in his physical development. His athletic pursuits continued into college. where he attended the University of Southern California (USC) on a basketball scholarship. This early involvement in sports laid a strong foundation for his physical health and disciplined lifestyle.
Transition to Acting
Selleck's transition from an athlete to an actor came with its physical demands. His first significant role in "Magnum P.I." required him to perform various stunts and maintain a fit appearance. This role, which he played from 1980 to 1988. necessitated a rigorous fitness routine to meet the show's demands. setting the stage for his long-term commitment to health and wellness.
Fitness Regimen
Workout Routine
Tom Selleck health and fitness regimen has evolved. adapting to his changing roles and age. During his "Magnum, P.I." days. Selleck's workouts were intense and focused on building and maintaining muscle mass. His routine included weightlifting, cardiovascular exercises. and specific training for the stunts he performed on the show.
Selleck adjusted his fitness routine as he aged to suit his body's needs. Today, his workouts focus on maintaining flexibility, strength, and cardiovascular health. He incorporates low-impact exercises such as swimming, walking, and light weightlifting. This balanced approach helps him stay fit without putting undue strain on his joints and muscles.
Importance of Flexibility and Mobility
In recent years, Selleck has emphasized the importance of flexibility and mobility in his fitness regimen. Understanding the natural decline in muscle mass and joint flexibility with age. he includes stretching and yoga in his routine. These practices help prevent injuries, improve posture, and maintain mobilit
2. Ankle Anatomy :
• ankle joint is a hinged synovial joint .
• Is formed by the articulation of 3 bones that
are the talus, tibia, and fibula.
• distal ends of the tibia and fibula in the lower
limb articulates with the proximal end of the
talus.
• The talus articulates inferiorly with the
calcaneus and anteriorly with the navicular.
• bones are covered by articular cartilage .
• three malleoli in ankle joint [ lateral , medial ,
posterior or 3rd malleolus ].
3. Ankle joint is supported by :
• Fibrous capsule.
• Medial ligaments [Deltoid ligament]:-
A- Superficial :
- Tibionavicular lig.
- Tibiocalcaneal lig.
- Posterior tibiotalar part lig.
B- Deep:-
- Anterior tibiotalar part lig.
• Lateral ligament:-
- Anterior talofibular lig.
- Posterior talofibular lig.
- Calcaneofibular lig.
• Ligaments that connect
the lower end of the tibia and fibula :-
- Anterior and posterior tibiofibular lig.
- Interosseous lig.
6. Muscles of the ankle:
1.Gastrocnemius muscle.
2. Soleus muscle.
•Both connect to the calcaneus by the
Achilles tendon.
•Both are involved in planter flexion.
7. 3. Tibialis anterior .
4. Tibialis posterior.
• Both are inserted in
the inner arch of the foot .
• Both are involved in INVERSION .
8. 5. Fibularis longus.
6. Fibularis brevis.
• Both inserted into
the outer arch of the foot.
• Both are involved in EVERSION.
9. Tendons:
1- Achilles Tendon: attaches the calf muscles (Gastrocnemius and Soleus) to the heel bone
(calcaneus). Help in Lifts the heel off the ground during activity
2- Posterior Tibial Tendon: attaches one of the calf muscles (the tibialis
posterior muscle) to the bones on the inside arch of the foot. It acts to
plantarflex and and invert the foot
3- Anterior tibial tendon : attach the anterior tibialis muscle to the foot. It acts to
dorsiflex and invert the foot.
4- Two peroneal tendon : pass behind the lateral malleolus and turn the foot
down and out [ peroneous longus and peroneous brevis].
10. Nerve and blood supply:
By nerves that pass through the ankle
on their way to the foot :
1- posterior tibial nerve
2- deep peroneal nerve
3- superficial peroneal nerve
By arteries that pass through the ankle
on their way to the foot :
1- Dorsalis pedis artery
2- posterior tibial artery
13. Definition : All fractures of the lower ends of the tibia and fibula involving the
ankle joint .
Incidence: ankle fracture are among the most common injuries.
Aetiology :
1. External rotation fracture [ pott’s fracture] : Commonest type , occurs due to forcible
external ( lateral ) rotation of the foot.
2. Internal rotation fracture : very RARE , occurs due to forcible internal ( medial ) rotation
of the foot .
3. Abduction fracture : occurs due to fall on EVERTED foot.
4. Adduction fracture : occurs due to fall on INVERTED foot.
5. Vertical compression fracture : occurs due to fall from a height on the foot.
19. 2)- According to level of fibular fracture [ Weber classification ] :
Type A :
•Fracture of fibula below the tibiofibular syndesmosis .
•It may be associated with a fracture of the medial malleolus or tear of the medial
ligament.
Type B :
•Fibular fracture at the level of syndesmosis .
•It may be associated with tear of the anterior tibiofibular ligament or fractures of the
medial malleolus or the posterior malleolus .
Type C :
•Fibular fracture above the level of syndesmosis , which leads to disruption of the
syndesmosis, a part of the interosseous membrane and wide separation of the
tibiofibular joint.
•There may be associated fracture of the medial and third malleolus.
20.
21. Clinical picture:
• History of trauma
• immediate pain and severe pain
• deformity
• inability to move ( cannot put weight on the injured foot )
• swelling & edema
• tenderness
• bruising
22. Complications:
Commonest complications are :-
• joint complications , osteoarthritis, ankle stiffness.
• Malunion & nonunion.
• Injury of anterior & posterior tibial nerves & vessels or long & short
saphenous.
• Injury of surrounding tendons.
23. Investigation:
✓ Plain x-ray that shows :
1. Absence of the normal overlap of the lower ends of the tibia
and fibula .
2. Widening of the space between the medial malleolus and the
talus.
3. Incongruity of the saddle-shaped surface of the talus and the
tibia.
25. A. Fracture of one malleolus without displacement :
•External fixation in a below knee cast for 6 weeks ( fixation of a joint above
and a joint below the ankle ) .
B. Fracture of 2 or 3 malleoli with displacement :
•Open reduction and internal fixation are necessary to restore normal
anatomical position and to achieve normal load distribution .
•Surgery should be done within 6 hours after trauma before development of
edema or 6 days after edema subside.
•First, fibular fracture ( lateral malleolus ) should be reduced anatomically to
restore its length & fixed by plate and screws
•Then the medial malleolus is reduced and fixed with screws .
•The third malleolus is fixed by screws .
•Collateral ligaments may need surgical repair
•Tibia-fibular syndesmosis reconstruction by protection screw which
removed after 6 weeks
Treatment:
28. •` Tibial plafond : is the distal end of the
tibia including the articular surface.
•` Mechanism of injury :
High energy axial loads as the tibial
plafond is injured by the talus punching up
into it.
•` clinical picture:
- Immediate and sever pain
- Swelling
- Bruising
- Tender to the touch
- Cannot put any weight on the injured foot
- Deformity ( out of place)
pilon fracture
29. Investigation:
X-RAY :
Appears as a comminuted distal tibial
fracture extendeing into the tibial
plafond ( ankle joint )
Usually it’s not obvious by x-ray .
CT-scan :
Gives accurate definition of the
fragments.
31. Treatment :
1- Control of swelling by elevation.
2- Apply external fixation or circular
frame fixation so the blisters can be
treated.
3- Once the skin has recovered,
open reduction and fixation with
plates and screws may be done.
4- Early movement help to reduce
the oedema and prevent stiffness.
32.
33. Ligamentous injuries of the ankle :
•Mechanism of injury : Twisting of the foot.
•Pathology : Sprain or tear of the ligaments.
An inversion twist of the foot is a frequent injury which results usually in
sprain or tear of the lateral ligaments of the ankle.
Injury to the deltoid ligament by eversion twist of the foot is rare.
•Clinically : tenderness and swelling anterior and below the lateral malleolus.
Pain which is made worse by inversion of the foot.
•X-RAY : may show subluxation of the talus.
•Treatment :
A. Sprained ankle : novocaine , hydrocortisone and elastoplast strapping of the
ankle
The patient is encouraged to resume his activities
B. Torn ligaments : below knee plaster cast for 6 weeks and an Elastoplast
bandage is then applied for one month.
35. Anatomy of the foot :
IA)- Bones:
Divided into three regions..
(I) Hindfoot [talus and calcaneus]
( tarsals bones)
(II) Midfoot [ navicular, cuboid ,
cuneiforms]
( tarsals bones)
(III) forefoot [ metatarsals and
phalanges ]
36. A• Tarsals :
A set of seven irregularly shaped
bones .
They are situated proximally in
the foot in the ankle area.
Divided into :-
•Proximal bones: talus ,
calcaneus.
•Intermediate bones : Navicular
bone.
•Distal bones: 3 cuneiform bones
“ medial – intermediate- lateral”
and cuboid bone.
38. C• Phalanges:
The bones of the toes .
Each toe has three phalanges [
Proximal , intermediate , distal
] .
Except the big toe , which only
has two phalanges
[ proximal and distal ].
39. IB)- joint :
`• Subtalar joint : between talus and
calcaneus .
`• Talonavicular joint : between distal
talus and navicular .
`• Metatarsophalangeal joint :
articulations between the heads of the
metatarsals and the proximal
phalanges.
`• interphalangeal joint : [ proximal &
distal ]
- Proximal: between proximal phalanx
and Middle phalanx .
- Distal: Between middle phalanx and
distal phalanx .
46. `• calcaneus is a large and strong bone that forms the
back of the foot.
`• Articulates above with talus and anteriorly with
cuboid forming subtalar joint and calcaneocuboid joints.
`• Calcaneus articulate with talus by 3 facets : anterior ,
posterior and middle.
`• It has 4 processes : anterior , posterior , medial and
lateral.
`• Its main functions is : weight bearing and stability.
48. Böhler’s angle:
It Used in the assessment of intra-
articular calcaneal fractures.
Measure by used of two
intersecting lines: one drawn from
anterior process of the calcaneus
to the highest part of posterior
articular surface and a second
drawn parallel to superior point of
tuberosity.
49. Calcaneus fracture:
Ateiology : usually by trauma .
Mechanism of injury:
`• The patient falls from a height, often from a ladder, onto one or both heels.
`• The calcaneum is driven up against the talus and is split or crushed.
`• More than 20% of these patients suffer associated injuries of the spine,
pelvis or hip.
`• It may be bilateral.
50. Classification:
I) Extra-articular fractures
involve the calcaneal processes
or the posterior part of the bone
They are easy to manage and have
a good prognosis.
II) Intra-articular fractures
cleave the bone obliquely and run
into the superior articular surface
of the Subtalar joint and it’s an
indication of open reduction and
internal fixation.
51. Clinical picture:
1` The foot is painful, swollen and bruised.
2` The heel may look broad and squat.
3` Tenderness
4` Absence of normal concavity below the lateral malleolus [
BULGE BELOW THE LATERAL MALLEOLUS]
5` The subtalar joint cannot be moved but ankle movement is
possible.
6` check for signs of a compartment syndrome of the foot
(intense pain, very extensive swelling and bruising and
diminished sensation).
52. Investigation:
Plain x-ray:-
Extra-articular fractures are usually fairly obvious, Intra-articular fractures,
also, can often be identified in the plain films .
if there is displacement of the fragments, the lateral view may show
flattening of Böhler’s angle.
However, for accurate definition of intra-articular fractures, CT is essential.
With severe injuries – and especially with
bilateral fractures – it is essential to assess the knees,
the spine and the pelvis as well.
53. Complications:
I) Broadening of the heel:
This is quite common and may cause problems
with shoe fitting and walking
II) Talocalcaneal stiffness and osteoarthritis:
Displaced intra-articular fractures may lead to joint
stiffness and, eventually, osteoarthritis.
54. Treatment:
Undisplaced fractures :-
`• Leg and foot are elevatedand treated
with ice-packs until the swelling subsides .
`• The calcaneus is compressed from side
to side to correct Broadening of the heal .
`• Firm bandage is applied and the patient
is allowed on non-weightbearing crutches
for 6 weeks.
56. Postoperatively:
- The foot is lightly splinted and elevated.
- Exercises are begun as soon as pain subsides.
- After 2–3 weeks, the patient can be allowed up on non-
weightbearing crutches.
61. Talus is the most
cartilaginous surface and
articulate with:
1` Tibia to form tibiotalar joint.
2` Calcaneus to form Subtalar
joint.
3` Navicular to form
Talonavicular joint.
62. Talus fracture:
Ateiology : usually by trauma.
Mechanism of injury:
• falls from a height lead to compressing the talus between
the tibia and calcaneus .
• Hyper dorsiflexion and axial loading.
63. Clinical picture:
1• Pain in foot and ankle.
2• Swelling in foot and ankle.
3• if there’s displaced, there may be
an obvious deformity
4• inability to move.
65. Classification of talus fracture:
Hawkin’s classification:
Type I : non-displaced fracture
In type I fractures AVN is
less than 10%.
66. Type II :
Displaced (however little)
and associated
with subluxation or
dislocation of the subtalar
joint.
In type II AVN is about 30–
40%.
67. Type III :
Displaced, with dislocation of
the body of the talus from the
ankle joint ( tibiotalar
dislocation or subluxation).
In type III AVN more than 90%.
68. Type IV :
Fracture with Subtalar and
tibiotalar dislocation and
Talonavicular subluxation.
In type IV AVN more than
90%.
69. Investigation:
X-Ray :-
Is not easy to see the
fracture by x-ray
because of
unfamiliarity with the
normal appearance in
various x-ray
projections.
CT-scan is essential
70.
71. Treatment:
Undisplaced fracture:-
1` no need for reduction.
2` split plaster is done until
swelling subsides then is
replaced by complete plaster.
3` remains in plantigrade
position for 6-8 weeks .
72. Displaced fracture:
1` reduction is required : closed
reduction should be tried first , if
this fails an open reduction must be
done.
2` below knee plaster is needed for
6-12 weeks.
3` weightbearing isn’t allowed until
healing occurs.
73. Tarsometatarsal injury:
•They may be :
`- Sprains : common
`- dislocation : rare
• Mechanism of injury:
Twisting or crushing injuries.
• A fracture dislocation should always
be suspected if the patient has pain,
swelling and bruising of the foot after
an accident, even if there is no
obvious deformity.
74. Investigation :
X-ray:-
- Fracture dislocation is clear
and can’t be missed.
- Full extent of the injury is not
clear.
CT-Scan :-
Is the investigation of choice
for bony and articular injury.
MRI :-
Used to see ligamentous injury.
75. Treatment:
Undisplaced sprains: require cast immobilization
for 4–6 weeks.
Subluxation or dislocation:
-requires reduction usually closed reduction by Traction
and manipulation under anaesthesia (open
reduction is rarely needed).
-the position is then held with K-wires or screws and cast
immobilization.
-The patient is instructed to remain nonweightbearing for
6–12 weeks.
76.
77. Metatarsals fracture:
Mechanism of injury:-
1` Direct blow
2` severe twisting
3` Repetitive stress
Clinical picture:-
1` History of injury
2` pain
3` swelling
Investigation:-
X-RAY can show the fracture.
78. Treatment:
- In undisplaced or slightly
displaced fracture: A walking
plaster may be applied, and is
retained for 3 weeks
- In the severe displacement :
reduction and fixation may be
done. weightbearing is
avoided for 3 weeks and this is
followed by a further 3 weeks
in a weightbearing cast.
79. Stress injury ( march fracture) :
It’s a very tiny fracture of the metatarsal bones due to repeated
loads on the foot .
Occurs in young adults particularly army member or sport person .
80. Clinical picture:
1` painful foot after overuse.
2` tenderness.
3` tender lump is palpable in the metatarsal bone.
4` swelling.
Investigation:
- Not diagnosed by x-ray as it is normal
- A radio-isotope scan will show an area of intense
activity in the bone .
- MRI also may show stress changes in the bone.
81. Treatment:
No displacement occurs so neither reduction nor
splintage is necessary.
The forefoot may be supported and normal walking is
encouraged.
82. Fracture of phalanges:
Mechanism of injury:
A heavy object falling on the toes causing
fracture of the phalanges.
Management:
- No specific treatment and the patient
encouraged To walk in a suitably adapted boot.
- If pain is marked , the toe can be splinted by
strapping it to its neighbor for 2-3 weeks.
- If the skin is broken, it must be covered with a
sterile dressing .