Acute gingival lesion
Acute gingival lesions are
(i) Acute Necrotizing Ulcerative Gingivitis
(ANUG)
(ii) Acute Herpetic Gingivostomatitis (AHGS)
(iii) Acute pericronitis
ANUG
Def:
an inflammatory destructive disease
of the gingiva which presents
characteristic signs and symptoms
OTHER TERMS USED
 VINCENT’S INFECTION
 TRENCH MOUTH
 VINCENT’S STOMATITIS
 PSEUDOMEMBRANOUS ANGINA
ETIOLOGY
(i) Systemic factors
(ii) Local factors
SYSTEMIC FACTORS
A-Psychological stress
B-Debilitating disease
C-Nutritional deficiency
LOCAL FACTORS
A-Smoking
B-Local trauma
C-Role of bacteria
CLASSIFICATION
(i) ACUTE
(ii) SUBACUTE
(iii) RECURRENT
(iv) CHRONIC
CLINICAL FEATURE
* SYMPTOMS
1-ORAL
2-EXTRAORAL
*SIGNS
1-SYSTEMIC SIGNS
2-ORAL SIGNS
SYSTEMIC SIGNS
(a) High fever
(b) Increased pulse rate
(c) Leukocytosis
(d) Loss of appetite
(e) General lassitude
(f) Insomnia , constipation
ORAL SIGNS
 Crater like depression at the crest of the
inter dental papilla extending to the
marginal gingiva
 Spontaneous gingival hemorrhage
 Fetor odor
 Increased salivation
DIFFERENTIAL DIAGNOSIS
(i) Acute necrotic gingivostomatitis
(ii) Chronic destructive periodontal disease
(iii) Gonococcal stomatitis
(iv) AIDS
(v) Ulcerative gingivitis in leukemia
(vi) Chronic desquamative gingivitis
(vii) Agranulocytosis
TREATMENT
1- AMBULATORY
Pt will have less toxic symptoms and will
walk in to the clinic by him self
2- NON-AMBULATORY
Pt will be bed ridden
TREATMENT
1- LOCAL
2- SYSTEMIC
ACUTE HERPATIC
GINGIVOSTOMATITIS
ETIOLOGY
HSV type I
CF
EXTRAORAL FEATURE
-fever
-lassitude
-lymphadenitis
-malaise
-headache
INTRAORAL FEATURE
-Generalized gingival inflammation is one of the earliest oral signs of acute
herpetic gingivostomatitis
- It’s characterized by :
- Diffuse ,erythematuos shiny involvement of gingiva and oral mucosa
- The Giniva is edematuos and easy bleeds on slightest provocation
- Gingival inflammation is followed by appearance of :
. Grayish or yellow vesicle on the gingiva ,labial and buccal mucosa ,soft
palate, pharynx, floor of the mouth and tongue
- After 24 hours –> vesicle rupture forming small painful ulcers
. The ulcers are one to two millimeter in diameter with elevated halo like
margin and yellowish or greyish white central portion
. Frank ulcer covered by pseudomembran and surrounds by erythema.
DIAGNOSIS
-CLINICAL PICTURE
-CONFIRMATORY TEST
TREATMENT
- Anesthetic mouth wash
- Antibiotic
- Antiseptic solution
- Analgesic
- Antiviral drugs
DIFFRENTIAL DIAGNOSIS
- Erythema Multiforme
- Steven’s Johnson's syndrome
- Aphthous Ulcers
- Agranulocytosis
- Acute Periocronitis
PERICRONITIS
Def
It is the inflammation of the gingiva in
relation to the crown of an incompletely
erupted tooth occurs mostly in the
mandibular third molars
TYPES
• ACUTE
• SUBACUTE
• CHRONIC
- The pt is extremely uncomfortable
- In ability to close the jaw
- Swelling of cheek
- Systemic toxic manifestations :
-fever
-leukocytosis
-malaise
COMPLICATION
- Localized pericoronal abscess
- Difficulty in swallowing when the
inflammation spread
- Involvement of the lymph node
- Ludwig's angina
ACUT GINGIVAL ABCESS
- It is a sudden acute and painful condition
- Involve only marginal and inter dental
papilla
- The abscess area will be bright red, shiny
in colure
- The duration of abscess is short (24-48
hours)
C/F
- The flap is red
- Swollen
- Ulcerated from the inner surface
- Tender
- Radiating pain to the ear , throat and floor
of the mouth
- Foul test
- Some times there will be oozing exudate
i.e. pus from gingival margin
- on percussion on adjacent teeth there will
be sensitivity or tenderness
ETIOLOGY
Foreign bodies (e.g. tooth brush bristles ,
food particles )
TREATMENT
- Drainage of the fluctuant abscess
- Antiseptic solution
- Mouthwash
- Anti inflammatory drugs
- Antibiotic
ACUTE CANDIDASES
Predisposing factor
- Prolonged use of antibiotic
- Immunosuppressive disease
CLINICALLY
- Elevated white plaque surface (coagulated
milk)
- Bleeding surface underneath
CONFIRMATORY TEST
TREATMENT
- Nystatin
- Ampholericin B

acute gingival lesion.ppt