Acne vulgaris is a self-limited skin disorder of the pilosebaceous unit that primarily affects adolescents and presents with various lesions including comedones, papules, pustules, nodules and cysts on the face, trunk and chest. It is caused by four key factors: follicular hyperkeratinization, sebum accumulation, inflammation and the presence of Propionibacterium acnes bacteria. Androgens play an important role by increasing sebum production. Treatment involves topical retinoids, antibiotics and benzoyl peroxide for mild-moderate acne and oral antibiotics, isotretinoin or hormonal therapy for severe inflammatory acne.
This is a seminar conducted by 4th year medical student under supervision of a lecturer. Sorry for not attaching the references.
Information were from few textbooks, google and also from previous dermatology posting group's seminar.
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Acne is a dermatological problem affected by an estimated 65 million people worldwide. This presentation is based on the changes on the skin in relation to the problem 'acne'.
This is a seminar conducted by 4th year medical student under supervision of a lecturer. Sorry for not attaching the references.
Information were from few textbooks, google and also from previous dermatology posting group's seminar.
Hyperpigmentation on the face slide sharevanita rattan
Hyperpigmentation on the face. How to recognise the distribution of hyperpigmentation on the face and how to treat it. The classic causes of Hyperpigmentation.
Pigmentation disorders of skin dermatology revision notesTONY SCARIA
dermatology revision notes for neet pg preparation based on lecture notes with high yield topic & last minute revision notes based on previous year questions
Acne is a dermatological problem affected by an estimated 65 million people worldwide. This presentation is based on the changes on the skin in relation to the problem 'acne'.
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2. ACNE VULGARIS
SELF-LIMITED DISORDER OF PILOSEBACEOUS UNIT
PRIMARILY IN ADOLESCENTS
PLEOMORPHIC VARIETY OF LESIONS- COMEDONES,
PAPULES, PUSTULES, NODULES
GENETIC FACTORS PLAY A ROLE IN ACNE SEVERITY
MEDICATIONS LIKE CORTICOSTEROIDS, ISONIAZID,
PHENYTOIN, LITHIUM, PROGESTINS
CAN CAUSE ACNELIKE LESIONS
3. PATHOGENESIS
FOUR KEY ELEMENTS:
FOLLICULAR HYPERKERATINIZATION
SEBUM ACCUMULATION
INFLAMMATION
PRESENCE & ACTIVITY OF Propionibacterium
acnes
ANDROGENS PLAY A PIVOTAL ROLE
INCREASE SEBUM PRODUCTION & ENLARGE
SEBACEOUS GLANDS
4. CLINICAL FEATURES
OPEN & CLOSED COMEDOS, ERYTHEMATOUS
PAPULES, PUSTULES, NODULES, CYSTS &
SCARS
FACE, TRUNK & CHEST
INFLAMATORY & NON-INFLAMMATORY LESIONS
SCARRING IS A COMPLICATION OF BOTH.
5.
6.
7.
8.
9. CLINICAL VARIANTS OF ACNE
ACNE
CONGLOBATA:
severe, scarring form of
acne where large
nodules and abscesses
become confluent to
form draining sinus
tracts
11. CLINICAL VARIANTS OF ACNE
ACNE COSMETICA:
persistent, low grade form
of acne result from use of
cosmetics, moisturizers,
sunscreens This type of
acne responds particularly
well to the topical
application of tretinoin.
12. CLINICAL VARIANTS OF ACNE
ACNE MECHANICA:
results from repeated trauma
associated with sports
helmet, shoulder pads, chin
rests of violoins and violas.
13. CLINICAL VARIANTS OF ACNE
Acne fulminans :
severe, scarring form of acne
with systemic signs and
symptoms of infection and
Leukocytosis.
16. DIFFERENTIAL DIAGNOSIS
FOLLICULITIS :
PAPULES, PUSTULES,
NO COMEDOS. USUALLY
AFFECTS TRUNK &
EXTREMITIES
17. DIFFERENTIAL DIAGNOSIS
PERIORAL
DERMATITIS: LONG
TERM USE OF TOPICAL
CORTICOSTEROIDS ON
FACE CAN RESULT IN
ACNEIFORM,
ERYTHEMATOUS,
INFLAMED PINK PAPULES
ON CHIN & CHEEKS.
