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Head injury
1. Head injury
primary brain injury:damage at
time of injury
scalp
blood loss
laceration
foreign body
portal of infection
skull
liner fractures
basilar fractures
depressed fractures
perforating and penetrating
intracerebral haemorrhage
vascular injury
penetrating injury
4.6% gunshot
0.4% stab wounds
1/3 hv vascular injury
1/3 results in infection
management
wound debridement
removal of foreign body
angiography
focal
EDH=extradural haematoma
usually blunt trauma
a/w linear fracture
usually temporal 70-80%
tear of middle meningeal artery
biconvex due to attachment of dura to skull sutures
lucid interval
treatment emergency craniotomy
subdural haematoma
acute
venous tear/brain laceration
covers entire cerebral surface
srescent shaped
a/w parenchymal injury morbidity/mortality underlying brain injury
mortality 30-90%
tx early elective craniotomy
chronic
elderly and infants
50% raised ICP
Tx
early burr hole drainage in the presence
of raised ICP of lateralising signs
intracerebral haematoma
15% of fatal head injuries
a/w
contussions
diffuse axonal injuries
subdural haematoma
Tx
evacuation of haematoma in the
presence of raised ICP or marked
midline shift
contusion
laceration
subarachnoid haemorrhage
IVH=intraventricular haemorrhage
hypothalamic and pituitary
brainstem
cranial nerves
diffuse
concussion
diffuse axonal injury(DAI)
35% of all fatal head injuries
prolonged unconsciousness in the
absence of a mass lesion
basal cisterns effaced
ventricles compressed
sulci invisible
secondary injury:after initial injury
systemic
hypoxia
hypotension
hypercapnia
hyperthermia
hypoglycaemia
intracranial
brain swelling
brain shift and herniation
raised ICP
post-traumatic fits
5% adults in first week
7% children
majority in 24 hrs
2/3 have more than one seizure
can be delayed
incidence depends on severity of injury
higher incidence if
post traumatic amnesia>24 hrs
intracranial haematoma
depressed fracture
penetrating injury
intracranial infection
prevention of secondary brain
injury can prevent death and
disability
prevention of secondary injury
low risk
asymp, headache, dizziness, scalp
laceration, scalp contussion, scalp
abrasion
moderate risk
changeof consciousness, progressive
headache, alcohol/drug intoxication etc
high risk
depressed consciousness
decreasing consciousness
penetratin injury
palpable/depressed fracture
management
conservative
observation
ventilation
fluids
mannitol
steroids
ICP monitoring
maintain CPP
surgical
ventricular drain
burr holes
craniotomy
elevation skull fracture
initial assessment and
management
primary survey
A-airway and C-spine control
B-breathing and ventilation
C-circulation and ahemorrhage control
D-disability(neurological
evaluation-GCS)
E-exposure/environmental control
if GCS<8 -->intubate avoid
decrease BP and decrease O2
secondary survey
head to toe
revaluation
definitive care
glasgow coma score
post-traumatic subarachnoid
a/w 39% motality
causes severe post traumatic headache
a/w underlying brain injury and
cause major morbidity
post-concussional syndrome
behavioural change
headaches
dizziness
irritable
vertigo
mood swings
poor memory
majority recover
management
neuropsychiatric assessment
neuropsychimetric assessment
- - Mindjet